Academic literature on the topic 'Chickens microbiology'

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Journal articles on the topic "Chickens microbiology"

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McCREA, B. A., K. H. TONOOKA, C. VanWORTH, E. R. ATWILL, and J. S. SCHRADER. "Colonizing Capability of Campylobacter jejuni Genotypes from Low-Prevalence Avian Species in Broiler Chickens." Journal of Food Protection 69, no. 2 (2006): 417–20. http://dx.doi.org/10.4315/0362-028x-69.2.417.

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Genetic variations in Campylobacter jejuni or host factors result in low prevalence rates among nonchicken poultry species. The objective of this study was to determine the colonizing potential, in broiler chickens, of C. jejuni that was recovered from low-prevalence avian species. Twenty-day-old Campylobacter-negative broiler chicks were inoculated by oral gavage with genetically different primary isolates of C. jejuni recovered from squab, duck, or chicken. Serial sampling and microbiologic testing of ceca were used to determine the level of colonization and the prevalence of positive chickens. All isolates were recovered from chickens by 10 days postinoculation. The C. jejuni strains recovered from challenged birds were genetically identical to the inoculated strains. By 10 days postinoculation, treatment groups inoculated with duck or control chicken isolates were 100% positive. The level of colonization by the squab isolate on day 2 postinoculation was significantly less than the duck or chicken isolates and had not colonized all birds by day 10 postinoculation.
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Volf, Jiri, Magdalena Crhanova, Daniela Karasova, et al. "Eggshell and Feed Microbiota Do Not Represent Major Sources of Gut Anaerobes for Chickens in Commercial Production." Microorganisms 9, no. 7 (2021): 1480. http://dx.doi.org/10.3390/microorganisms9071480.

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In this study, we addressed the origin of chicken gut microbiota in commercial production by a comparison of eggshell and feed microbiota with caecal microbiota of 7-day-old chickens, using microbiota analysis by 16S rRNA sequencing. In addition, we tested at which timepoint during prenatal or neonatal development it is possible to successfully administer probiotics. We found that eggshell microbiota was a combination of environmental and adult hen gut microbiota but was completely different from caecal microbiota of 7-day-old chicks. Similarly, we observed that the composition of feed microbiota was different from caecal microbiota. Neither eggshell nor feed acted as an important source of gut microbiota for the chickens in commercial production. Following the experimental administration of potential probiotics, we found that chickens can be colonised only when already hatched and active. Spraying of eggs with gut anaerobes during egg incubation or hatching itself did not result in effective chicken colonisation. Such conclusions should be considered when selecting and administering probiotics to chickens in hatcheries. Eggshells, feed or drinking water do not act as major sources of gut microbiota. Newly hatched chickens must be colonised from additional sources, such as air dust with spores of Clostridiales. The natural colonisation starts only when chickens are already hatched, as spraying of eggs or even chickens at the very beginning of the hatching process did not result in efficient colonisation.
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Makarova, Aleksandra V., Anatoly B. Vakhrameev, and Inessa A. Meftah. "Comparative characteristics of the growth and development of meategg and egg-meat chickens." Agrarian science, no. 11-12 (January 20, 2021): 29–32. http://dx.doi.org/10.32634/0869-8155-2020-343-11-29-32.

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Relevance. Recently, the number of many breeds and lines of chickens has significantly decreased, while others are under threat of extinction. Reducing the genetic resources of chicken’s worldwide limits the possibilities of future breeding programs. The study of the genetic diversity of the species allows you to more accurately manage the productive and potential of chicken breeds, the possibilities of its use for obtaining Biosafety and full-fledged food. Adding exterior estimation data to the bird breeding value forecast increases its accuracy and is relevant.Methods. The study was conducted on chickens of two experimental populations «Experimental CS» meat-egg of productivity and «Experimental LZS» egg-meat productivity from the Genetic collection of rare and endangered chicken breeds Russian Research Institute of Farm Animal Genetics and Breeding — Branch of the L.K. Ernst Federal Science Center for Animal Husbandry.Results. The highest absolute increase in live weight of chickens is observed in the period of 4-8 weeks, and the relative increase at the age of 4 weeks, regardless of the type of productivity of chickens. The results of cultivation largely depend on the intensity of growth of the bird in the early period, up to four weeks of age. The superiority of the meat-egg population in comparison with the egg-meat population was revealed in terms of breast circumference (5.0–7.7%).
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Feng, Ze-Qing, Ting Lian, Yong Huang, Qing Zhu, and Yi-Ping Liu. "Expression Pattern of Genes of RLR-Mediated Antiviral Pathway in Different-Breed Chicken Response to Marek’s Disease Virus Infection." BioMed Research International 2013 (2013): 1–9. http://dx.doi.org/10.1155/2013/419256.

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It has been known that the chicken’s resistance to disease was affected by chicken’s genetic background. And RLR-mediated antiviral pathway plays an important role in detection of viral RNA. However, little is known about the interaction of genetic background with RLR-mediated antiviral pathway in chicken against MDV infection. In this study, we adopted economic line-AA broilers and native Erlang mountainous chickens for being infected with MDV. Upon infection with MDV, the expression ofMDA-5was upregulated in two-breed chickens at 4, 7, and 21 d.p.i. It is indicated that MDA-5 might be involved in detecting MDV in chicken. Interestingly, the expression ofIRF-3andIFN-βgenes was decreased in spleen and thymus of broilers at 21 d.p.i, but it was upregulated in immune tissues of Erlang mountainous chickens. And the genome load of MDV in spleen of broiler is significantly higher than that in Erlang mountainous chickens. Meanwhile, we observed that the death of broiler mainly also occurred in this phase. Collectively, these present results demonstrated that the expression patters ofIRF-3andIFN-βgenes in chicken against MDV infection might be affected by the genetic background which sequently influence the resistance of chicken response to MDV.
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Kheimar, Ahmed, Romina Klinger, Luca D. Bertzbach, et al. "A Genetically Engineered Commercial Chicken Line Is Resistant to Highly Pathogenic Avian Leukosis Virus Subgroup J." Microorganisms 9, no. 5 (2021): 1066. http://dx.doi.org/10.3390/microorganisms9051066.

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Viral diseases remain a major concern for animal health and global food production in modern agriculture. In chickens, avian leukosis virus subgroup J (ALV-J) represents an important pathogen that causes severe economic loss. Until now, no vaccine or antiviral drugs are available against ALV-J and strategies to combat this pathogen in commercial flocks are desperately needed. CRISPR/Cas9 targeted genome editing recently facilitated the generation of genetically modified chickens with a mutation of the chicken ALV-J receptor Na+/H+ exchanger type 1 (chNHE1). In this study, we provide evidence that this mutation protects a commercial chicken line (NHE1ΔW38) against the virulent ALV-J prototype strain HPRS-103. We demonstrate that replication of HPRS-103 is severely impaired in NHE1ΔW38 birds and that ALV-J-specific antigen is not detected in cloacal swabs at later time points. Consistently, infected NHE1ΔW38 chickens gained more weight compared to their non-transgenic counterparts (NHE1W38). Histopathology revealed that NHE1W38 chickens developed ALV-J typical pathology in various organs, while no pathological lesions were detected in NHE1ΔW38 chickens. Taken together, our data revealed that this mutation can render a commercial chicken line resistant to highly pathogenic ALV-J infection, which could aid in fighting this pathogen and improve animal health in the field.
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VARABIOFF, YURI, GREGORY E. MITCHELL, and STEPHEN M. NOTTINGHAM. "Effects of Irradiation on Bacterial Load and Listeria monocytogenes in Raw Chicken." Journal of Food Protection 55, no. 5 (1992): 389–91. http://dx.doi.org/10.4315/0362-028x-55.5.389.

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After irradiation of chickens to a dose of 2.5 kGy, the decrease in the standard plate count (SPC) was similar in air and in vacuum-packaged chickens. During storage at 4°C for 15 d, the SPC increased progressively in both types of packaged chickens. At the end of the storage period, the SPC was higher in air-packaged chicken than in vacuum-packaged chickens. In irradiated chickens, Listeria monocytogenes was only recovered from the vacuum-packaged chickens after 7 d cold storage. In unirradiated chickens, L. monocytogenes proliferated similarly in both air- and vacuum-packaged chickens.
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THOMAS, COLLEEN, DANIEL J. KING, and DAVID E. SWAYNE. "Thermal Inactivation of Avian Influenza and Newcastle Disease Viruses in Chicken Meat." Journal of Food Protection 71, no. 6 (2008): 1214–22. http://dx.doi.org/10.4315/0362-028x-71.6.1214.

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Avian influenza viruses (AIV) and Newcastle disease viruses (NDV) of high pathogenicity cause severe systemic disease with high mortality in chickens and can be isolated from the meat of infected chickens. Although AIV and NDV strains of low pathogenicity are typically not present in chicken meat, virus particles in respiratory secretions or feces are possible sources of carcass contamination. Because spread of AIV and NDV is associated with movement of infected birds or their products, the presence of these viruses in chicken meat is cause for concern. This study presents thermal inactivation data for two viruses of high pathogenicity in chickens (AIV strain A/chicken/Pennsylvania/1370/1983 and NDV strain APMV-1/chicken/California/S0212676/2002) and two viruses of low pathogenicity in chickens (AIV strain A/chicken/Texas/298313/2004 and NDV strain APMV-1/chicken/Northern Ireland/Ulster/1967). Under the conditions of the assay, high-pathogenicity AIV was inactivated more slowly in meat from naturally infected chickens than in artificially infected chicken meat with a similar virus titer. In contrast, high-pathogenicity NDV was inactivated similarly in naturally and artificially infected meat. Linear regression models predicted that the current U.S. Department of Agriculture–Food Safety and Inspection Service time-temperature guidelines for cooking chicken meat to achieve a 7-log reduction of Salmonella also would effectively inactivate the AIV and NDV strains tested. Experimentally, the AIV and NDV strains used in this study (and the previously studied H5N1 high-pathogenicity AIV strain A/chicken/Korea/ES/2003) were effectively inactivated in chicken meat held at 70 or 73.9°C for less than 1 s.
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BAILEY, J. S., and D. E. COSBY. "Salmonella Prevalence in Free-Range and Certified Organic Chickens." Journal of Food Protection 68, no. 11 (2005): 2451–53. http://dx.doi.org/10.4315/0362-028x-68.11.2451.

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Many consumers assume that broiler chickens grown under traditional commercial conditions will have more Salmonella than free-range or organic chickens, which usually are less crowded, have access to outside spaces during grow out, and are fed special diets. Despite these perceptions, there is a lack of published information about the microbiological status of free-range and organic chickens. A total of 135 processed free-range chickens from four different commercial free-range chicken producers were sampled in 14 different lots for the presence of Salmonella. Overall, 9 (64%) of 14 lots and 42 (31%) of 135 of the carcasses were positive for Salmonella. No Salmonella were detected in 5 of the 14 lots, and in one lot 100% of the chickens were positive for Salmonella. An additional 53 all-natural (no meat or poultry meal or antibiotics in the feed) processed chickens from eight lots were tested; 25% of the individual chickens from 37% of these lots tested positive for Salmonella. Three lots of chickens from a single organic free-range producer were tested, and all three of the lots and 60% of the individual chickens were positive for Salmonella. The U.S. Department of Agriculture Food Safety and Inspection Service reported that commercial chickens processed from 2000 to 2003 had a Salmonella prevalence rate of 9.1 to 12.8%. Consumers should not assume that free-range or organic conditions will have anything to do with the Salmonella status of the chicken.
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ZHANG, GUODONG, LI MA, and MICHAEL P. DOYLE. "Potential Competitive Exclusion Bacteria from Poultry Inhibitory to Campylobacter jejuni and Salmonella." Journal of Food Protection 70, no. 4 (2007): 867–73. http://dx.doi.org/10.4315/0362-028x-70.4.867.

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The objective of this study was to isolate from chickens potential competitive exclusion bacteria (CE) that are inhibitory to Campylobacter jejuni or Salmonella, or to both, for subsequent development of a defined CE product for use in poultry. Adult chickens from family farms, commercial farms, and broiler chicken research centers were sampled to identify and select C. jejuni–free donor chickens. A challenge treatment, which included administering perorally 106 CFU C. jejuni per chicken and determining undetectable cecal shedding of campylobacters at 4 weeks, was important for identifying the best CE donor chickens. Screening of bacterial colonies obtained from nine donor chickens by using selective and nonselective media yielded 636 isolates inhibitory to six C. jejuni strains in vitro, with 194 isolates being strongly inhibitory. Of the 194 isolates, 145 were from ceca, and 117 were facultative anaerobic bacteria. One hundred forty-three isolates were inhibitory to six strains of Salmonella (including five different serotypes) in vitro. Of these, 41 were strongly inhibitory to all C. jejuni and Salmonella strains evaluated, and most were Lactobacillus salivarius. A direct overlay method, which involved directly applying soft agar on plates with discrete colonies from mucus scrapings of gastrointestinal tracts, was more effective in isolating CE than was the frequently practiced isolation method of picking and transferring discrete colonies and then overlaying them with soft agar. The best approach for obtaining bacteria highly inhibitory to Salmonella and C. jejuni from chickens was to isolate bacteria from ceca under anaerobic conditions. Free-range chickens from family farms were better donors of potential CE strongly inhibitory to both Salmonella and Campylobacter than were chickens from commercial farms and broiler chicken research centers.
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HARADA, TETSUYA, YASUAKI MITO, KOICHI OTSUKI, and TOSHIYUKI MURASE. "Resistance to Gentamicin and Vancomycin in Enterococcal Strains Isolated from Retail Broiler Chickens in Japan." Journal of Food Protection 67, no. 10 (2004): 2292–95. http://dx.doi.org/10.4315/0362-028x-67.10.2292.

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A total of 137 Enterococcus strains isolated from chicken meat were subjected to antimicrobial susceptibility tests. Strains with the vanC1 gene were isolated from seven of nine samples of chicken meat processed in Japan and from all chickens from China and Brazil between July 2001 and April 2002. The pulsed-field gel electrophoresis (PFGE) patterns of the isolates were distinguishable from each other, suggesting that VanC1-type vancomycin-resistant Enterococcus is preferentially colonized in broiler chickens in these countries. The incidence of high-level gentamicin resistant (HLGR) enterococci that harbored the aac(6′)-Ie-aph(2″)-Ia or aph(2′)-Id gene varied among the countries from which the chickens originated (Japan, 2 of 65; China, 11 of 43; Brazil, 6 of 29). Moreover, the PFGE patterns of the HLGR strains were distinguishable from each other, except for two strains obtained from chickens from Brazil. The results suggest that HLGR Enterococcus is highly prevalent in broiler chickens.
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Dissertations / Theses on the topic "Chickens microbiology"

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McConnell, Claire Deborah. "Effects of chicken anaemia virus on cell-mediated immune function in chickens." Thesis, Queen's University Belfast, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.317510.

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Martins, Nelson Rodrigo da Silva. "Studies of the immunoglobulin responses to viral infections of chickens." Thesis, University of Surrey, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.254977.

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Echevarría-Núñez, Lisbeth E. "Role of Surface Molecules in Campylobacter jejuni Colonization and Virulence in Chickens." Diss., The University of Arizona, 2012. http://hdl.handle.net/10150/228452.

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Campylobacter spp. is one of the two major causes of foodborne illness throughout the world. Campylobacter accounts for the most common causes of diarrheal illness caused by bacterial pathogens worldwide and in the United States. It is estimated that Campylobacter diarrheal illness affects about 2.4 million persons every year with an estimated cost of treatment and loss of productivity exceeding $1 billion annually. Previous work in our laboratory on biofilms has demonstrated the presence pilus-like surface-associated structures disseminating from the cell wall of C. jejuni isolates not expressing flagella (flaAB mutants). To further investigate this finding, bioinformatics analysis, purification and identification of genes involved in the expression of surface-associated structures as well as mutational analysis of putative genes were performed. We identified two important poultry colonization factors in C. jejuni. These studies might provide insights in understanding the pathogenesis of C. jejuni. Moreover, it will provide a new target for potential vaccine development against C. jejuni infection in poultry production system. Thus directly impacting the number of C. jejuni infection in humans.
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Law, Bibiana Felicity. "Assessment of the pathogenicity of Campylobacter jejuni from broiler chickens." Diss., The University of Arizona, 2005. http://hdl.handle.net/10150/282899.

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Sixty-three of 435 (14.5%) samples collected from broiler chickens were positive for C. jejuni. Twenty-two of 55 samples were from organic chickens (40%) and 41 of 380 samples were from conventional chickens (10.8%). Isolates were subjected to macrorestriction profiling using SmaI and analyzed for their ability to survive in macrophage cells and invade in epithelial cells. Antibiotic testing to cefaclor, ciprofloxacin, tetracycline, erythromycin, gentamicin, trimethroprim/sulfamethoxazole, and ampicillin were performed. Finally, 5 isolates of varying putative in vitro virulence traits were chosen for experimental inoculation of newborn piglets. Five piglets per isolate were tested and examined macroscopically and microscopically upon necropsy. Genotyping of isolates indicated 1 to 3 profiles per flock. Of the 22 organic isolates from chickens, only 3 (13.6%) were able to survive within macrophages. For the conventional isolates, 21 out of 41 (51.2%) were able to survive. However, the majority of isolates (90.5%) from both organic and conventional isolates were not capable of invading epithelial cells. No isolates exhibited resistance to ciprofloxacin or gentamicin. One isolate out of 63 (1.6%) was resistant to erythromycin, 52 (82.5%) to tetracycline, 28 (44.4%) to trimethroprim/sulfamethoxazole, and 6 (9.5%) to cefaclor. In terms of the piglet studies, regardless of the combination of in vitro invasion or survival results or type of flock, most piglets (16/25) in all groups exhibited hyperemia, edema, and hemorrhage in the small intestine or colon upon gross examination. Microscopic examination revealed congested mucosa and erosion of the epithelium in 10 of the 25 piglets from 4 of the 5 groups. In conclusion, this study suggests that C. jejuni isolated from broiler chickens are virulent in piglets and are probably capable of causing disease in humans. Furthermore, the results of the survival and invasion assays did not correlate with the results of the piglet studies and cannot be relied upon to predict degree of virulence. Therefore, another virulence factor is responsible for the pathogenesis, such as a toxin(s). As this is the first study to confirm putative in vitro virulence traits with an animal model, further research is recommended with the piglet model to assess pathogenicity.
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Kinsey, Yvette E. "The effect of the inclusion of probiotic micro-organisms in the diet of growing chickens." Thesis, University of Newcastle Upon Tyne, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.307940.

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Nicholas, Robin Ashley John. "Studies on the development and application of an ELISA for the detection of antibody to Salmonella enteritidis in chickens and their eggs." Thesis, Brunel University, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.306853.

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Hu, Jinxin. "Molecular and genetic studies of resistance to infection with Salmonella typhimurium in chickens." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape11/PQDD_0001/NQ44455.pdf.

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Abundo, Michael Edward Cruz. "Evaluation of sampling methods for the study of respiratory bacterial microbiota in chickens." The Ohio State University, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1574851946483897.

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Armstrong, Alexandra Edwards. "Salmonella in an Oyster Production and Small Feedlot Environment, Use of Novel Proteins Expressed by an Attenuated Salmonella Vector for the Reduction of Campylobacter Colonization in Broiler Chickens." Diss., The University of Arizona, 2012. http://hdl.handle.net/10150/228492.

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The CDC estimates that 48 million illnesses, 128,000 hospitalizations, and 3,000 deaths annually are attributable to foodborne illnesses, making their impact significant in terms of both human health and economic losses (3). Estimates vary, but it is frequently stated that Campylobacter species affect 2.4 million people annually (28). Among bacterial foodborne pathogens it is second in the US only to Salmonella, which in recent years has consistently been the most frequently reported, most likely to cause hospitalization, and deadliest foodborne bacterial illness in the US (3, 106).In order to reduce the burden of illness caused by these pathogens and improve the safety of our food supply, continued investigation of the epidemiology, transmission and interactions of these organisms with their environments is necessary. Additionally, prevention of colonization within natural reservoirs of these bacteria which contribute to contamination of foods is an important step in the reduction of the burden of foodborne illness. This work examines the relationship of Salmonella to oysters and the aquatic environment in which they are raised, the interactions of Salmonella in a small feedlot environment, and the reduction of colonization of broiler chickens by Campylobacter jejuni through vaccination with recombinant attenuated Salmonella vectors into which novel Campylobacter genes had been cloned. It was found that while Salmonella is still sporadically present on the West Coast of the US, an area where oysters were previously found to be positive for the organism, the strain which predominated in the last study of that area is reduced in prevalence. Additionally, it was found that that strain does not possess special fitness in oysters or the aquatic environments in which they are raised, though Salmonella survives in oysters and water samples longer than a representative coliform. Salmonella is also present in the small feedlot environment sampled, and animal stress appears to play a role in the shedding of the organism in that environment, leading to the potential contamination of beef carcasses during processing. Reduction of colonization by C. jejuni in broilers was achieved in the case of both vaccines, with a maximum reduction of four logs as compared to controls.
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Pisula, Anneka. "Detecting a Probiotic Product Within the Gut of Broiler Chickens." DigitalCommons@CalPoly, 2018. https://digitalcommons.calpoly.edu/theses/1921.

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As of January 2017, the U.S. poultry industry banned the use of antibiotics and now relies on alternatives such as probiotics to help protect animal health. Although probiotic use is not a new concept in the poultry industry, identifying the best combination of bacterial strains to generate an effective probiotic formula requires further investigation. This study aimed to detect a probiotic product of four bacterial strains (Pedioccoccus acidilactici, Pediococcus pentosaceus, Lactobacillus plantarum, and Bacillus subtilis) in a feeding trial with broiler chickens. Birds given the probiotic were predicted to show an improved growth performance with the probiotics colonizing the gut. Ninety-six broiler chickens were equally divided into 3 treatment and 3 control pens. During the 25-day experiment, birds were fed a starter diet (days 0-11) and a grower diet (days 12-25). Experimental birds were administered the probiotic product via the drinking water at a concentration of 3.1×104 CFU/ml. Control birds had an equivalent amount of dextrose filler added to their water supply. Feces were collected hourly on day one and daily thereafter. On days 1, 22, and 25 of the experiment, 2 birds from each pen were euthanized for gut sampling. Lumen and mucosa samples were collected from the duodenum, jejunum, ileum, and ceca. Species-specific and strain specific PCR primers were employed for probiotic detection. Wild strains of P. acidilactici, P. pentosaceus, and L. plantarum were detected in the feeds, inhibiting detection of the probiotic strains when using species-specific PCR primers. Strain-specific primers were used to detect the probiotic Pedioccoccus acidilactici and Lactobacillus plantarum strains. B. subtilis was detected in feces within one hour of probiotic administration and was predominantly detected in experimental birds only. Both P. acidilactici and L. plantarum probiotic strains were initially detected in the feces of treated birds within two hours of probiotic administration and again ten days later. Both L. plantarum and B. subtilis were seen only in treated bird gut samples. L. plantarum was predominantly detected in the ceca near the end of the small intestine. P. pentosaceus was observed more often in treated gut samples and P. acidilactici was the least commonly detected probiotic strain. All administered bacteria were rarely seen in mucosa samples. Feed-endogenous P. acidilactici and L. plantarum strains became progressively more detectable in the mucosa along the gastrointestinal tract suggesting gut colonization, however, probiotic strains did not appear to colonize the mucosa of treated birds. Although probiotic strains were no longer detected after product removal, all probiotic strains were detected in feces and gut samples during probiotic administration, suggesting the bacteria can colonize the gut. Probiotic supplementation did not result in significant differences in body weight gain, feed intake, or feed conversion ratio. However, birds growing in a more stressful environment than the carefully controlled experimental set up used here may show probiotic-related effects. This study identified that the probiotic bacteria appeared to survive the gastrointestinal tract, exhibited a transit time of 1-2 hours, could possibly colonize chickens, and localized near the end of the chicken gut.
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Books on the topic "Chickens microbiology"

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World Health Organization (WHO). Risk assessment of Campylobacter spp. in broiler chickens: Technical report. World Health Organization, 2009.

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(WHO), World Health Organization. Risk assessment of Campylobacter spp. in broiler chickens: Interpretative summary. World Health Organization, 2009.

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United States. Food Safety and Inspection Service. Science and Technology. Microbiology Division, ed. Nationwide raw ground chicken microbiological survey. U.S. Dept. of Agriculture, Food Safety and Inspection Service, Science and Technology, Microbiology Division, 1996.

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Chen, Yinghwei. Quality of fryers purchased in retail markets using microbial and sensory assessment. 1989.

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Organization, World Health, and Food and Agriculture Organization of the United Nations., eds. Risk assessments of Salmonella in eggs and broiler chickens. World Health Organization, 2002.

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Big chicken: The incredible story of how antibiotics created modern agriculture and changed the way the world eats. National Geographic Partners, LLC, 2017.

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Risk Assessments of Salmonella in Eggs And Broiler Chickens: Interpretative Summary (Microbiological Risk Assessment Series). Bernan Assoc, 2003.

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Food and Agriculture Organization of the United Nations., World Health Organization, and Joint FAO/WHO Expert Consultation on Risk Assessment of Campylobacter spp. in Broiler Chickens and Vibrio spp. in Seafood (2002 : Bangkok, Thailand), eds. Risk assessment of Campylobacter spp. in broiler chickens and Vibrio spp. in seafood: Report of a Joint FAO/WHO Expert Consultation, Bangkok, Thailand, 5-9 August 2002. World Health Organization, Food and Agriculture Organization of the United Nations, 2003.

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Harrison, Mark. Herpes simplex and zoster. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198765875.003.0023.

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This chapter describes the microbiology of herpes simplex and zoster as it applies to Emergency Medicine, and in particular the Primary FRCEM examination. The chapter outlines the key details of the clinical features, basis of diagnosis, treatment, and prevention, of herpes simplex and zoster, including herpes encephalitis, chicken pox, and shingles. This chapter is laid out exactly following the RCEM syllabus, to allow easy reference and consolidation of learning.
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World Health Organization (WHO). Risk Assessment of Campylobacter Spp. in Broiler Chickens And Vibrio Spp. in Seafood: Report Of A Joint FAO/WHO expert Consultation, Bangkok, Thailand, 5-9 August 2002 (Fao Food and Nutrition Paper). Food & Agriculture Organization of the UN (FA, 2004.

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Book chapters on the topic "Chickens microbiology"

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Wabeck, Charles J. "Microbiology of Poultry Meat Products." In Commercial Chicken Meat and Egg Production. Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0811-3_45.

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Schat, K. A. "Chicken Anemia Virus." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-70972-5_10.

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Sanchez-Garcia, F. J., and W. T. McCormack. "Chicken γδ T Cells." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-80057-3_6.

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Funk, P. E., and C. B. Thompson. "Current Concepts in Chicken B Cell Development." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-80057-3_3.

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Yulipriyanto, H., Philippe Morand, G. Tricot, and C. Aubert. "Effect of Additives on the Nitrification-Denitrification Activities During Composting of Chicken Manure." In Microbiology of Composting. Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/978-3-662-08724-4_21.

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Hino, S., and A. A. Prasetyo. "Relationship of Torque Teno Virus to Chicken Anemia Virus." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-70972-5_8.

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de Smit, M. H., and M. H. M. Noteborn. "Apoptosis-Inducing Proteins in Chicken Anemia Virus and TT Virus." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-70972-5_9.

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Kaufman, J., and H. J. Wallny. "Chicken MHC Molecules, Disease Resistance and the Evolutionary Origin of Birds." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-80057-3_12.

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Shen, Cangliang, and Yifan Zhang. "Enumeration and Identification of Staphylococcus aureus in Chicken Salads." In Food Microbiology Laboratory for the Food Science Student. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-58371-6_6.

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Simonsen, M. "Why I Think the Chicken Should Survive in Immunology and Developmental Biology." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-80057-3_1.

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Conference papers on the topic "Chickens microbiology"

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Hinton Jr, Arthur. "Inhibition of Growth of Salmonella by Native Flora of Broiler Chickens." In XII Latin American Congress on Food Microbiology and Hygiene. Editora Edgard Blücher, 2014. http://dx.doi.org/10.5151/foodsci-microal-304.

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Borsato-Moysés, Juliano, Sarah Jarschel de Camargo, Maristela da Silva do Nascimento, Neusely da Silva, and Valéria Cristina Amstalden Junqueira. "Quantification of Thermotolerant Campylobacter Spp. in Frozen Chicken Carcasses." In XII Latin American Congress on Food Microbiology and Hygiene. Editora Edgard Blücher, 2014. http://dx.doi.org/10.5151/foodsci-microal-144.

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Chow, Felipe Coser, Afonso de Liguori Oliveira, and Roseane Batitucci Passos de Oliveira. "Frozen Mechanically Deboned Chicken Meat: Compliance With Brazilian Legal Parameters." In XII Latin American Congress on Food Microbiology and Hygiene. Editora Edgard Blücher, 2014. http://dx.doi.org/10.5151/foodsci-microal-211.

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Wessling, Claudia Regina, Cibeli Viana, Vanessa Mendonça Soares, et al. "Penetration of Salmonella Enteritidis in Chicken Breasts Stored Under Refrigeration Temperatures." In XII Latin American Congress on Food Microbiology and Hygiene. Editora Edgard Blücher, 2014. http://dx.doi.org/10.5151/foodsci-microal-117.

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Hanim, C., V. A. Cahya, L. M. Yusiati, and A. Kurniawati. "Isolation and Identification of Bacteriocin-producing Bacillus Strain Isolated from the Gastrointestinal Tract of Indonesian Native Chicken (Gallus domesticus)." In 10th International Seminar and 12th Congress of Indonesian Society for Microbiology (ISISM 2019). Atlantis Press, 2021. http://dx.doi.org/10.2991/absr.k.210810.007.

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Cahya, V. A., C. Hanim, and L. M. Yusiati. "Characterization of Bacteriocin Produced by Bacillus subtilis 11A Isolated from the Gastrointestinal Tract of Indonesian Native Chicken (Gallus domesticus)." In 10th International Seminar and 12th Congress of Indonesian Society for Microbiology (ISISM 2019). Atlantis Press, 2021. http://dx.doi.org/10.2991/absr.k.210810.021.

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