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1

MIYOSHI, Norimasa. "Sengo kasen gyō sei to damu kaihatsu: Tonegawa suikei ni okeru chisui, risui no kōzō tenkan." Social Science Japan Journal 20, no. 1 (February 1, 2017): 153–56. http://dx.doi.org/10.1093/ssjj/jyx004.

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2

Li, Haiying, Jia Lu, Jiang Yu, and Xinghua Wu. "Identification and Assessment of Ecological Sensitivity in Chishui River Basin Based on GIS." E3S Web of Conferences 267 (2021): 01016. http://dx.doi.org/10.1051/e3sconf/202126701016.

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In this paper, GIS and RS spatial analysis techniques were used to establish a system for identifying sensitive targets of terrestrial ecosystems in Chishui River Basin, and to identify protection targets of terrestrial ecosystems in different regions. The result shows that the assessment of habitat sensitivity in Chishui River Basin was dominated by non-sensitive areas and highly sensitive areas, which highly sensitive areas account for 60.43% of the basin area, mostly concentrated in the mountainous areas at the junction of Chishui City, Xishui County and Gulin County; non-sensitive areas account for 39.13% and are mosaically distributed with highly sensitive areas; mildly sensitive areas, moderately sensitive areas and extremely sensitive areas account for a total of about 0.5% of the basin area and are more scattered throughout the basin. The distribution is scattered. The moderately sensitive areas in the Chishui River Basin are more than 60.7% of the basin area, indicating that the habitats in the Chishui River Basin are generally sensitive and the ecologically sensitive targets are mainly wooded areas, which need to be protected.
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3

Freysz, M., and C. Doussot. "Traumi toracici chiusi." EMC - Urgenze 12, no. 2 (January 2008): 1–19. http://dx.doi.org/10.1016/s1286-9341(08)70026-7.

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4

Mirek, S., and M. Freysz. "Traumi toracici chiusi." EMC - Urgenze 22, no. 2 (June 2018): 1–15. http://dx.doi.org/10.1016/s1286-9341(18)89782-4.

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5

Saccone, Eduardo. "Con gli occhi chiusi." MLN 110, no. 1 (1995): 1–19. http://dx.doi.org/10.1353/mln.1995.0011.

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6

Jannot, Jean-René. "Les reliefs de Chiusi." Mélanges de l'École française de Rome. Antiquité, no. 122-1 (September 15, 2010): 51–72. http://dx.doi.org/10.4000/mefra.334.

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7

Córdoba, A., Francisco Aguayo González, Juan Ramón Lama Ruiz, and Ana de las Heras. "Innovation in Lean Manufacturing by Kansei-Chisei Engineering." Materials Science Forum 853 (April 2016): 48–51. http://dx.doi.org/10.4028/www.scientific.net/msf.853.48.

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This article forms part of the research for innovation in intelligent systems and sustainable production in a social approach, with the aim of formulating a neuroscientific model for the application of Kansei-Chisei Engineering in Lean manufacturing environments, in the metal-mechanical sector. In the area of ergonomics design of environments and products has developed from a practical rational approaches to design emotional solutions incorporating affective design with Kansei engineering design. This paper describes the main objectives for methodological development based on Kansei-Chisei Engineering for application to design Lean manufacturing environments, the main background, current state of art and objectives.
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8

Qiu, Liang, and Hong Juan Zhai. "An Ecological Compensation Mechanism of Chishui River Water Resources Protection and Research." Applied Mechanics and Materials 685 (October 2014): 463–67. http://dx.doi.org/10.4028/www.scientific.net/amm.685.463.

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Chishui River, a first-order south tributary of upper Yangtze River, extends cross Sichuan, Yunnan and Guizhou provinces and is famous as the “eco-river with a lot of endemic fishes”, “Beauty River with natural scenic landscapes”, “Chinese famous liquors river with Maotai” and “hero river”. According to the existing problems in Ecological Environment of the Chishui River, the ecological compensation mechanism was established and the compensation of the main watershed ecology, ecological compensation object, ecological compensation standards and ecological compensation ways were determined, for protecting the environment requirements of the rare and unique fish and Liquor industry production of Yangtze River.
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9

Kim, Irene, Betsy Hopson, Inmaculada Aban, Elias B. Rizk, Mark S. Dias, Robin Bowman, Laurie L. Ackerman, et al. "Decompression for Chiari malformation type II in individuals with myelomeningocele in the National Spina Bifida Patient Registry." Journal of Neurosurgery: Pediatrics 22, no. 6 (December 2018): 652–58. http://dx.doi.org/10.3171/2018.5.peds18160.

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OBJECTIVEThe purpose of this study was to determine the rate of decompression for Chiari malformation type II in individuals with myelomeningocele in the National Spina Bifida Patient Registry (NSBPR). In addition, the authors explored the variation in rates of Chiari II decompression across NSBPR institutions, examined the relationship between Chiari II decompression and functional lesion level of the myelomeningocele, age, and need for tracheostomy, and they evaluated for temporal trends in rates of Chiari II decompression.METHODSThe authors queried the NSBPR to identify all individuals with myelomeningocele between 2009 and 2015. Among these patients, they identified individuals who had undergone at least 1 Chiari II decompression as well as those who had undergone tracheostomy. For each participating NSBPR institution, the authors calculated the proportion of patients enrolled at that site who underwent Chiari II decompression. Logistic regression was performed to analyze the relationship between Chiari II decompression, functional lesion level, age at decompression, and history of tracheostomy.RESULTSOf 4448 individuals with myelomeningocele identified from 26 institutions, 407 (9.15%) had undergone at least 1 Chiari II decompression. Fifty-one patients had undergone tracheostomy. Logistic regression demonstrated a statistically significant relationship between Chiari II decompression and functional lesion level of the myelomeningocele, with a more rostral lesion level associated with a higher likelihood of posterior fossa decompression. Similarly, children born before 2005 and those with history of tracheostomy had a significantly higher likelihood of Chiari II decompression. There was no association between functional lesion level and need for tracheostomy. However, among those children who underwent Chiari II decompression, the likelihood of also undergoing tracheostomy increased significantly with younger age at decompression.CONCLUSIONSThe rate of Chiari II decompression in patients with myelomeningocele in the NSBPR is consistent with that in previously published literature. There is a significant relationship between Chiari II decompression and functional lesion level of the myelomeningocele, which has not previously been reported. Younger children who undergo Chiari II decompression are more likely to have undergone tracheostomy. There appears to be a shift away from Chiari II decompression, as children born before 2005 were more likely to undergo Chiari II decompression than those born in 2005 or later.
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10

Schanker, Benjamin D., Brian P. Walcott, Brian V. Nahed, Kristopher T. Kahle, Yan Michael Li, and Jean-Valery C. E. Coumans. "Familial Chiari malformation: case series." Neurosurgical Focus 31, no. 3 (September 2011): E1. http://dx.doi.org/10.3171/2011.6.focus11104.

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Chiari malformations (Types I–IV) are abnormalities of the posterior fossa that affect the cerebellum, brainstem, and the spinal cord with prevalence rates of 0.1%–0.5%. Case reports of familial aggregation of Chiari malformation, twin studies, cosegregation of Chiari malformation with known genetic conditions, and recent gene and genome-wide association studies provide strong evidence of the genetic underpinnings of familial Chiari malformation. The authors report on a series of 3 family pairs with Chiari malformation Type I: 2 mother-daughter pairs and 1 father-daughter pair. The specific genetic causes of familial Chiari malformation have yet to be fully elucidated. The authors review the literature and discuss several candidate genes. Recent advances in the understanding of the genetic influences and pathogenesis of familial Chiari malformation are expected to improve management of affected patients and monitoring of at-risk family members.
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11

Li, Fanxi, Xia Yu, Jiemei Lv, Qixin Wu, and Yanling An. "Assessment of heavy metal pollution in surface sediments of the Chishui River Basin, China." PLOS ONE 17, no. 2 (February 9, 2022): e0260901. http://dx.doi.org/10.1371/journal.pone.0260901.

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Accumulated heavy metals in surface sediments are released into the aquatic environment, causing secondary contamination of the hydrosphere, and increasing the risks to human health. To evaluate the pollution characteristics of heavy metals in the sediments of the Chishui River Basin, in the present study, the concentrations of five heavy metals in surface sediments of the Chishui River Basin in China were investigated using the geo-accumulation index, pollution load index, and potential ecological risk indexes. These indexes evaluated the degree of contamination and the influence of human activities on heavy metal levels in the basin. Cu, Zn, Cd, Hg, and As were found at concentrations of 5.12–120.40, 36.01–219.31, 0.03–1.28, 0.01–1.18, and 1.56–11.59 mg kg–1, respectively, with mean values of 37.43, 91.92, 0.25, 0.07, and 5.16 mg kg–1, respectively, in the order Zn > Cu > As > Cd > Hg. The contamination indices revealed Hg as the principal pollutant based on the spatial distribution, while Pearson’s correlation coefficients suggested that Cu, Zn, and As originated from a similar source. Hg had a different source from the other metals, whereas Cd originated from a different source compared with that of Zn, As, and Hg. This paper showed a Hg and Cd contamination in the Chishui River Basin.
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12

Katkar, A. S., Anderson H. Kuo, S. Calle, K. Gangadhar, and K. Chintapalli. "Budd-Chiari Syndrome Caused by TIPS Malposition: A Case Report." Case Reports in Medicine 2014 (2014): 1–4. http://dx.doi.org/10.1155/2014/267913.

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Budd-Chiari syndrome refers to hepatic pathology secondary to diminished venous outflow, most commonly associated with venothrombotic disease. Clinically, patients with Budd-Chiari present with hepatomegaly, ascites, abdominal distension, and pain. On imaging, Budd-Chiari syndrome is hallmarked by occluded IVC and or hepatic veins, caudate lobe enlargement, heterogeneous liver enhancement, intrahepatic collaterals, and hypervascular nodules. Etiopathological factors for Budd-Chiari syndrome include several systemic thrombotic and nonthrombotic conditions that can cause venous outflow obstruction at hepatic veins and/or IVC. While the transjugular intrahepatic portosystemic shunt (TIPS) is used as a treatment option for Budd-Chiari syndrome, Budd-Chiari syndrome is not a well-known complication of TIPS procedure. We report a case of Budd-Chiari syndrome that occurred in a transplanted cirrhotic liver from malpositioned proximal portion of the TIPS in IVC causing occlusion of the ostia of hepatic veins which was subsequently diagnosed on contrast-enhanced CT.
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13

Porcu, Manuela. "Comprensori chiusi e turismo residenziale." SOCIOLOGIA URBANA E RURALE, no. 107 (June 2015): 121–34. http://dx.doi.org/10.3280/sur2015-107009.

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14

Ferrari, P., G. Gibertini, M. Malagoli, and N. Cortesi. "I Traumi Chiusi Del Rene." Urologia Journal 54, no. 5 (October 1987): 525–32. http://dx.doi.org/10.1177/039156038705400501.

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15

Papachristou, M. D., R. E. Ward, V. Agarwal, and B. F. Branstetter. "Complex Chiari Malformation: What the Neurosurgeon Needs to Know." Neurographics 12, no. 1 (January 1, 2022): 35–42. http://dx.doi.org/10.3174/ng.2100050.

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Chiari malformation type 1 is a frequently encountered but incompletely understood morphologic variant that may present with headache and lower cranial nerve symptoms. The main surgical treatment for Chiari malformation type 1 is suboccipital decompression. Further research has identified a subset of patients with Chiari malformation type 1 with radiographic findings that predispose to suboptimal outcomes from suboccipital decompression alone. This disease process has come to be referred to as complex Chiari malformation. Here, we discuss imaging findings of complex Chiari malformation, identify craniovertebral metrics used to stratify patients, and propose a reporting structure to assist our neurosurgical colleagues in the selection of appropriate treatment.Learning Objective: To understand imaging findings and radiographic metrics associated with Complex Chiari malformation and their implication on surgical management
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16

Li, Gangping, Ying Huang, Shunyu Tang, Yuhu Song, Huimin Liang, Dehan Liu, Ling Yang, and Xiaohua Hou. "A single-center retrospective study: Clinical features of different types of Budd–Chiari syndrome in Chinese patients in the Hubei area." Vascular 26, no. 1 (July 25, 2017): 80–89. http://dx.doi.org/10.1177/1708538117718638.

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Background The characteristics and prevalence of Budd–Chiari syndrome in China remain unclear. This study aimed to analyze the clinical features of Budd–Chiari syndrome in Chinese patients in the Hubei area. Methods One-hundred and thirty patients with Budd–Chiari syndrome, admitted to Union Hospital from January 2002 to January 2011, were included in this retrospective study. Clinical features, laboratory data, imaging characteristics, and cumulative patency rates were analyzed. Results Of the 130 patients with Budd–Chiari syndrome, 77 were men (59.2%) and 53 women (40.8%). Budd–Chiari syndrome was more commonly associated with inferior vena cava block (56.9%, 74/130) than hepatic vein block (19.2%, 25/130) and combined inferior vena cava/hepatic vein block (23.9%, 31/130). The clinical features of Budd–Chiari syndrome varied based on the location of the obstruction. The incidence of bilirubin abnormality, elevated alkaline phosphatase, and γ-glutamyl peptide transferase levels was common in patients with Budd–Chiari syndrome. Liver injury was more severe in cases with combined inferior vena cava/hepatic vein block than in the other two types of Budd–Chiari syndrome. Color Doppler ultrasound imaging was better for the diagnosis of hepatic vein obstruction, while computed tomography and magnetic resonance imaging were superior in diagnosing inferior vena cava obstruction. The cumulative 1-, 5-, and 10-year patency rates were 97%, 69%, and 59%, respectively. Univariate analysis indicated that liver cirrhosis was an independent risk factor of recurrence. Conclusion The most prevalent type of Budd–Chiari syndrome is inferior vena cava obstruction in Chinese patients in the Hubei area. Different types of Budd–Chiari syndrome have diverse clinical and biochemical features, which may assist clinicians in diagnosing Budd–Chiari syndrome. Liver cirrhosis was found as an independent risk factor of recurrence.
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17

Qin, Qiang, Fubin Zhang, Fei Liu, Chunling Wang, and Huanzhang Liu. "Food Web Structure and Trophic Interactions Revealed by Stable Isotope Analysis in the Midstream of the Chishui River, a Tributary of the Yangtze River, China." Water 13, no. 2 (January 15, 2021): 195. http://dx.doi.org/10.3390/w13020195.

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Understanding energy flow and nutrient pathways is crucial to reveal the dynamics and functions of riverine ecosystems and develop appropriate conservation strategies. In this study, we utilized stable isotopes of δ13C and δ15N to examine the fundamental characteristics of trophic position, trophic niche, and carbon source for the food web in the midstream of the Chishui River, a tributary to the Yangtze River. Our results showed that stable isotope signatures among different sorts of basal resources and consumers were significantly distinguishable and that the food chain consisted of four trophic levels, indicating the multiple trophic pathways and long food chain length here. The trophic guilds of fish were classified into four categories, in which herbivorous and carnivorous fish showed greater trophic diversity and omnivorous fish had higher trophic redundancy, which meant that there was a stable trophic niche structure in the study area. Phytoplankton and periphyton presented the largest contributions to consumers, indicating that autochthonous productivity was the dominant carbon source in the midstream of the Chishui River. Since the Chishui River is still in a natural condition without any dam constructions, the autochthonous productivity, stable trophic niche structure, multiple trophic pathways and long food chain length found here demonstrate its high conservation value. Therefore, the strategy to refrain from damming on this river should persist into the future.
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18

Escalante, Andrés A. "Entrevista a Mariana Rodríguez." Review of Global Management 6, no. 1 (August 25, 2021): 35–40. http://dx.doi.org/10.19083/rgm.v6i1.1525.

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19

Hoffman, Harold J., John Neill, Kerry R. Crone, Bruce E. Hendrick, and Robin P. Humphreys. "Hydrosyringomyelia and Its Management in Childhood." Neurosurgery 21, no. 3 (September 1, 1987): 347–51. http://dx.doi.org/10.1227/00006123-198709000-00012.

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Abstract Syringomyelia, once regarded as a degenerative disease of adults, is now recognized to be a disorder usually associated with the Chiari malformation and occurring in patients of all ages. We have reviewed 47 patients with syringomyelia treated on the Neurosurgical Service at the Hospital for Sick Children during the years 1977 to 1985. Twelve of these patients had a Chiari I malformation, 30 had a Chiari II malformation, and 5 had an acquired Chiari malformation. Thirty-one of these patients were treated by decompression of the Chiari malformation and plugging of the obex, 5 were treated by a simple posterior fossa decompression, 9 were treated by shunting of the syringomyelic cavity, and 2 were treated by a combined decompression of the posterior fossa and shunting of the syrinx. The Gardner procedure (decompression of the Chiari malformation and plugging of the obex) was the procedure most commonly used in managing our group of patients and resulted in improvement in over 70% of patients.
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20

ZHENG, Xiu-Fen, Chau-Huei CHEN, and Chun-He ZHANG. "Study on Temporal Variations of Shear-Wave Splitting in the Chiayi Area, Aftershock Zone of 1999 Chichi Earthquake, Taiwan." Chinese Journal of Geophysics 51, no. 1 (January 2008): 115–24. http://dx.doi.org/10.1002/cjg2.1200.

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Udayakumaran, Suhas. "Chiari V or Chiari II plus?" Child's Nervous System 28, no. 3 (December 9, 2011): 337–38. http://dx.doi.org/10.1007/s00381-011-1654-z.

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22

Iliescu, Laura, Letitia Toma, Adriana Mercan-Stanciu, Mihaela Grumeza, Mihai Dodot, Teodora Isac, and Simona Ioanitescu. "Budd-Chiari syndrome - various etiologies and imagistic findings. A pictorial review." Medical Ultrasonography 21, no. 3 (August 31, 2019): 344. http://dx.doi.org/10.11152/mu-1921.

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Budd Chiari syndrome defines an obstruction of the hepatic venous outflow. Primary causes include pro-coagulant states resulting in venous thrombosis, while secondary Budd Chiari syndrome appears in conditions associated with extrinsic compression of the hepatic veins or tumor invasion. Clinical presentation is greatly varied, from incidentally discovered asymptomatic thrombosis to fulminant liver failure due to hepatic congestion. Abdominal ultrasonography is the key diagnostic tool of Budd Chiari syndrome. This pictorial essay aims to show the ultrasonographic aspect of Budd-Chiari syndrome associated with other medical conditions (abdominal malignancy, hematologic disorders and abdominal surgery)
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23

Feinberg, Michelle, Parker Babington, Shawn Sood, and Robert Keating. "Isolated unilateral trismus as a presentation of Chiari malformation: case report." Journal of Neurosurgery: Pediatrics 17, no. 5 (May 2016): 533–36. http://dx.doi.org/10.3171/2015.7.peds1592.

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The authors present a case of Chiari malformation manifesting as isolated trismus, describe the typical symptoms associated with Chiari malformation, and discuss the potential anatomical causes for this unique presentation. A 3-year-old boy presented with inability to open his jaw for 6 weeks with associated significant weight loss. The results of medical and radiological evaluation were negative except for Type I Chiari malformation with cerebellar tonsils 12 mm below the level of the foramen magnum. The patient underwent Chiari decompression surgery. Postoperatively, his ability to open his mouth was significantly improved, allowing resumption of a regular diet. Postoperative MRI revealed almost complete resolution of the syringobulbia. To the best of the authors' knowledge, this is the first reported case of isolated trismus from Chiari malformation with syringobulbia.
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24

Mazzola, Catherine A., and Arno H. Fried. "Revision surgery for chiari malformation decompression." Neurosurgical Focus 15, no. 3 (September 2003): 1–8. http://dx.doi.org/10.3171/foc.2003.15.3.3.

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Chiari malformations comprise four different hindbrain anomalies originally described by Hans Chiari, a professor of pathology at the German University in Prague. There are four basic Chiari malformations. The reasons for revision of Chiari malformation decompression may be for conservative or inadequate initial decompression or the development of postoperative complications. Another reason involves cases of both hindbrain herniation and syringomyelia in patients who have undergone adequate posterior fossa decompression without resolution of symptoms, signs, or radiological appearance of their syrinx cavity. Additionally, symptom recurrence has been reported in association with various types of dural grafts. Reoperation or revision surgery for patients with Chiari malformations is common and may not be due to technical error or inadequate decompression. The types of revision surgeries, their indications, and initial presentations will be reviewed.
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Colpan, Mustafa Efkan, and Zeki Sekerci. "Chiari Type I Malformation Presenting as Hemifacial Spasm: Case Report." Neurosurgery 57, no. 2 (August 1, 2005): E371. http://dx.doi.org/10.1227/01.neu.0000166688.69081.8b.

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ABSTRACT OBJECTIVE AND IMPORTANCE: We report on a patient with a Chiari I malformation presenting with right hemifacial spasm. Clinicians should consider the downward displacement of the hindbrain as a rare cause of hemifacial spasm in Chiari I malformation. CLINICAL PRESENTATION: An 18-year-old man was admitted with right hemifacial spasm. The results of the neurological examination were normal except for the facial spasm. Magnetic resonance imaging demonstrated a Chiari I malformation without syringomyelia. After surgery, the hemifacial spasm completely resolved. INTERVENTION: Posterior fossa decompression, C1 laminectomy, and duraplasty were performed. CONCLUSION: The hemifacial spasm could be attributed to compression and/or traction of the facial nerve because of downward displacement of the hindbrain in Chiari I malformation. Compression and/or traction might create irritation of the facial nerve that causes hemifacial spasm. Resolution of the hemifacial spasm after posterior fossa decompression could explain the facial nerve irritation in Chiari I malformation. Clinicians should consider Chiari malformation as a cause of hemifacial spasm and posterior fossa decompression as a potential treatment.
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Ando, Takeaki, Shannon Gehr, Melanie L. McGrath, and Adam B. Rosen. "Diagnosis of a Chiari Malformation After a Concussion in a Junior College Football Player With a History of Chronic Headaches: A Case Report." International Journal of Athletic Therapy and Training 22, no. 5 (September 2017): 21–25. http://dx.doi.org/10.1123/ijatt.2016-0103.

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The purpose of this report is to present the case of a National Junior Collegiate Athletic Association football player diagnosed with Chiari malformation postconcussion. A Chiari malformation is characterized by the cerebellum presenting below the level of the foramen. The uniqueness of this case stems from the patient’s health history, length of symptoms, and diagnosis. The effectiveness of treatment options, and the primary means to reduce the risk of catastrophic head injury in those with Chiari malformations are debatable. Clinicians should be familiar with the potential for the presence of a Chiari malformation with persistent symptoms postconcussion.
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Vurdem, Ümit Erkan, Niyazi Acer, Tolga Ertekin, Ahmet Savranlar, and Mehmet Fatih İnci. "Analysis of the Volumes of the Posterior Cranial Fossa, Cerebellum, and Herniated Tonsils Using the Stereological Methods in Patients with Chiari Type I Malformation." Scientific World Journal 2012 (2012): 1–7. http://dx.doi.org/10.1100/2012/616934.

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Objective. The aim of this study was to determine the posterior cranial fossa volume, cerebellar volume, and herniated tonsillar volume in patients with chiari type I malformation and control subjects using stereological methods.Material and Methods. These volumes were estimated retrospectively using the Cavalieri principle as a point-counting technique. We used magnetic resonance images taken from 25 control subjects and 30 patients with chiari type I malformation.Results. The posterior cranial fossa volume in patients with chiari type I malformation was significantly smaller than the volume in the control subjects (P<0.05). In the chiari type I malformation group, the cerebellar volume was smaller than the control group, but this difference was not statistically significant (P>0.05). In the chiari type I malformation group, the ratio of cerebellar volume to posterior cranial fossa volume was higher than in the control group. We also found a positive correlation between the posterior cranial fossa volume and cerebellar volume for each of the groups (r=0.865,P<0.001). The mean (±SD) herniated tonsillar volume and length were0.89±0.50 cm3and9.63±3.37 mm in the chiari type I malformation group, respectively.Conclusion. This study has shown that posterior cranial fossa and cerebellum volumes can be measured by stereological methods, and the ratio of these measurements can contribute to the evaluation of chiari type I malformation cases.
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Thuillier, Jean-Paul. "Un relief archaïque de Chiusi inédit." Bulletin de la Société Nationale des Antiquaires de France 1997, no. 1 (2001): 38–39. http://dx.doi.org/10.3406/bsnaf.2001.10145.

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29

Huntsman, Theresa. "Hellenistic Etruscan Cremation Urns from Chiusi." Metropolitan Museum Journal 49 (January 2014): 141–50. http://dx.doi.org/10.1086/680029.

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30

Fiuza, Sergio da Silva, Jorge Ferreira Kusdra, and Denise Temporim Furtado. "Caracterização química e atividade microbiana de coprólitos de Chibui bari (Oligochaeta) e do solo adjacente." Revista Brasileira de Ciência do Solo 35, no. 3 (June 2011): 723–28. http://dx.doi.org/10.1590/s0100-06832011000300007.

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Chibui bari é um oligochaeta edáfico nativo da região amazônica, considerado como minhocuçu em razão do seu grande tamanho (até 60 cm de comprimento). Seus excrementos ou coprólitos são liberados em montículos com formato de torre, com altura de até 30 cm e com matéria seca de até 2,0 kg. O objetivo desta pesquisa foi comparar a disponibilidade de nutrientes, o teor de C orgânico e a atividade microbiana dos coprólitos de Chibui bari com o solo adjacente de áreas de floresta secundária, seringal de cultivo e pastagem, localizadas na Universidade Federal do Acre - UFAC, em Rio Branco, Acre. As coletas foram realizadas considerando o delineamento em blocos casualizados, com três tratamentos, constituídos por: a) coprólitos de Chibui bari e por amostras compostas de solos retiradas no raio de 10 cm de cada coprólito coletado, nas profundidades de b) 0-10 cm e c) 10-20 cm. Os resultados foram submetidos à análise de variância, e as médias dos tratamentos, comparadas pelo teste de Tukey a 5 %. Além disso, efetuou-se análise de correlação simples entre as variáveis. Os coprólitos apresentaram maiores valores de pH, P disponível, bases trocáveis (Ca2+, Mg2+, K+), C orgânico e atividade microbiana. O teor de Al3+ foi menor nos coprólitos do que no solo na camada de 0-20 cm em 55 % (pastagem), 62 % (seringal) e 70 % (floresta). Verificaram-se, ainda, correlações positivas dos valores de C orgânico total (COT) com os de pH, P, K+, Ca2+ e Mg2+ e correlações negativas de Al3+ com COT, pH, P, K+, Ca2+ e Mg2+ nas três áreas avaliadas.
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Aslıyüce, Yasemin Özel, and Özlem Ülger. "Efficacy of Suboccipital Release and Stabilization Exercise Training in Type 1 Chiari Malformation Patient Undergoing Surgical Treatment: A Case Report." International Journal of Sport, Exercise and Health Research 5, no. 1 (May 30, 2021): 30–33. http://dx.doi.org/10.31254/sportmed.5109.

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In Chiari malformation, some symptoms are known to recur after decompression surgery. To our knowledge, this is the first case for symptomatic outcome after suboccipital release and stabilization exercises the patient with type 1 Chiari malformation who has experienced surgery. The present study aims to investigate the efficacy of suboccipital release and stabilization exercise training in a patient with Type 1 Chiari Malformation undergoing surgical treatment. A 47- year-old female patient with Chiari malformation received suboccipital release and stabilization exercise training for 10 weeks. Her symptomatic parameters were evaluated, such as pain, grip and pinch strength, deep cervical flexor strength, balance and gait. After the treatment, there were improvements in pain perception, the score of performance index of deep cervical muscles, grip force, static balance and gait parameters. Suboccipital oscillation and stabilization exercises are thought to be safe and effective in patients with Chiari malformation undergoing decompression surgery
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Willemsen, Koen, Menco J. S. Niemeyer, Netanja I. Harlianto, Said Sadiqi, Peter R. Seevinck, Ralph J. B. Sakkers, Harrie Weinans, and Bart C. H. Van der Wal. "Good long-term outcomes of the hip Chiari osteotomy in adolescents and adults with hip dysplasia: a systematic review." Acta Orthopaedica 93 (February 4, 2022): 296–302. http://dx.doi.org/10.2340/17453674.2022.2031.

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Background and purpose: The Chiari osteotomy was a regular treatment for developmental hip dysplasia before it became mostly reserved as a salvage therapy. However, the long-term survival of the Chiari osteotomy has not been systematically investigated. We investigated the survival time of the Chiari osteotomy until conversion to total hip arthroplasty (THA) in patients with primary hip dysplasia, and factors which correlated with survival, complications, and the improvement measured in radiographic parameters. Patients and methods: Studies were included when describing patients (> 16 years) with primary hip dysplasia treated with a Chiari osteotomy procedure with 8 years’ follow-up. Data on patient characteristics, indications, complications, radiographic parameters, and survival time (endpoint: conversion to THA) were extracted. Results: 8 studies were included. The average postoperative center–edge angle, acetabular head index, and Sharp angle were generally restored within the target range. 3 studies reported Kaplan-Meier survival rates varying from 96% at 10 years to 72% at 20 years’ follow-up. Negative survival factors were high age at intervention and pre-existing advanced preoperative osteoarthritis. Moreover, reported complications ranged between 0% and 28.3 %. Interpretation: The Chiari osteotomy has high reported survival rates and is capable of restoring radiographic hip parameters to healthy values. When carefully selected by young age, and a low osteoarthritis score, patients benefit from the Chiari osteotomy with satisfactory survival rates. The position of the Chiari osteotomy in relation to the periacetabular osteotomies should be further (re-)explored.
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33

Batty, Ruth, Lavanya Vitta, Elspeth H. Whitby, and Paul D. Griffiths. "Is There a Causal Relationship Between Open Spinal Dysraphism and Chiari II Deformity?" Neurosurgery 70, no. 4 (September 23, 2011): 890–99. http://dx.doi.org/10.1227/neu.0b013e318237a6c1.

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Abstract BACKGROUND: Chiari II deformity is associated with open spinal dysraphism. A causal relationship has been proposed by McLone and Knepper. This article evaluates that hypothesis. OBJECTIVE: To establish the frequency of Chiari II deformity in fetuses with open spinal dysraphism, assess whether meningocele sac neck area and volume influence the severity of posterior fossa changes, and assess whether the severity of associated findings (ventriculomegaly, amount of extracerebral CSF) are associated with Chiari II deformity. METHODS: Sixty-five fetuses with open spinal dysraphism were compared with gestationally aged matched “normal” fetuses on ultrafast MR images. Cerebellar vermis and bony posterior fossa surface area were measured on midline sagittal images. Hindbrain herniation was noted if present. In the open spinal dysraphic group, sac neck area and volume were measured. Ventriculomegaly was assessed by linear measurement of the trigone of the lateral ventricle and extracerebral CSF depth was measured maximally over the lateral surface of the cerebral hemispheres. RESULTS: Fifteen of 65 fetuses with open spinal dysraphism did not have Chiari II deformity. Neck area and volume of the sac did not correlate with the presence of Chiari II deformity or reduction in bony posterior fossa size. CONCLUSION: A relatively high proportion of fetuses with open spinal dysraphism do not have Chiari II deformity in utero. There is a lack of correlation between indicators of spinal dysraphism severity and the extent of the posterior fossa abnormality. This raises some interesting questions about the causality of the Chiari II deformity.
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34

Sarnat, Harvey B. "Regional Ependymal Upregulation of Vimentin in Chiari II Malformation, Aqueductal Stenosis, and Hydromyelia." Pediatric and Developmental Pathology 7, no. 1 (January 2004): 48–60. http://dx.doi.org/10.1007/s10024-003-2127-5.

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Vimentin, glial fibrillary acidic protein (GFAP) and S-100β protein were studied by immunocytochemistry in the ependyma of patients with Chiari II malformations, congenital aqueductal stenosis, and hydromyelia. Paraffin sections of brains and spinal cords of 16 patients were examined, 14 with Chiari II malformations, most with aqueductal stenosis and/or hydromyelia as associated features, and 2 patients with congenital aqueductal stenosis without Chiari malformation. Patients ranged in age from 20-wk gestation to 48 years. The results demonstrated: 1) in the fetus and young infant with Chiari II malformations, congenital aqueductal stenosis, and hydromyelia, vimentin is focally upregulated in the ependyma only in areas of dysgenesis and not in the ependyma throughout the ventricular system; 2) GFAP and S-100β protein are not coexpressed, indicating that the selective upregulation of vimentin is not simple maturational delay; 3) vimentin upregulation also is seen in the ependymal remnants of the congenital atretic cerebral aqueduct, not associated with Chiari malformation; 4) in the older child and adult with Chiari II malformation, vimentin overexpression in the ependyma becomes more generalized in the lateral ventricles as well, hence evolves into a nonspecific upregulation. The interpretation from these findings leads to speculation that it is unlikely that ependymal vimentin is directly involved in the pathogenesis of Chiari II malformation, but may reflect a secondary upregulation due to defective expression of another gene. This gene may be one of rhombomeric segmentation that also plays a role in defective programming of the paraxial mesoderm for the basioccipital and supraoccipital bones resulting in a small posterior fossa. This interpretation supports the hypothesis of a molecular genetic defect, rather than a mechanical cause, as the etiology of the Chiari II malformation.
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Slavkovic, Nemanja, Zoran Vukasinovic, Milan Apostolovic, and Boris Vukomanovic. "Chiari pelvic osteotomy in treatment of hip dysplasia." Srpski arhiv za celokupno lekarstvo 141, no. 9-10 (2013): 710–14. http://dx.doi.org/10.2298/sarh1310710s.

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Chiari pelvic osteotomy is a surgical procedure having been performed for almost sixty years in patients with the insuffcient coverage of the femoral head. It is most frequently used in young patients with dysplastic acetabular sockets as a part of developmental hip dysplasia. Even though performance of the Chiari osteotomy is associated with positive therapeutical results, above all, its main goal is to delay inevitable degenerative changes. Original surgical technique has been modified and improved over time. Nevertheless, the basic idea has remained unchanged - increasing of the femoral head coverage by medial displacement of the distal part of the pelvis along with capsular interpositioning. Given the complexity of operation, the complication percentage is rather low. Chiari pelvic osteotomy has lost its actuality and importance during this past six decades. The role of Chiari pelvic osteotomy has been considerably taken over by other more efficient and more lasting surgical procedures. Nonetheless, Chiari pelvic osteotomy is still present in modern orthopedic practice, above all as ?salvage? osteotomy.
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36

Polster, Sean P., Mark C. Dougherty, Hussein A. Zeineddine, Seon-Kyu Lee, and David Frim. "A Report of 2 Cases of Brainstem Hemorrhage After Suboccipital Craniectomy for Chiari Decompression." Operative Neurosurgery 14, no. 5 (July 11, 2017): E58—E62. http://dx.doi.org/10.1093/ons/opx146.

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Abstract BACKGROUND AND IMPORTANCE Decompression surgery for Chiari malformation is known to have very low procedure-related complications. There has been no report of post-Chiari malformation decompression surgery development of brainstem hemorrhage. We report 2 post-Chiari decompression surgery brainstem hemorrhage cases with 2-yr follow-up. CLINICAL PRESENTATION Two cases were reviewed in which patients underwent uncomplicated suboccipital craniectomy with expansive autologous pericranium duraplasty for Chiari decompression. Postoperatively, both patients awoke with hemibody sensory and motor deficits. Immediate postoperative magnetic resonance imaging revealed a small hemorrhage within the dorsal medulla in both cases. Follow-up imaging shows resolution along with near complete clinical recovery of deficits. CONCLUSION These cases demonstrate a rare postdecompression surgery-related complication in Chiari malformation. We hypothesize that these hemorrhages may occur from the rapid drainage of cerebrospinal fluid resulting in a loss of positive pressure, allowing a low-pressure hemorrhage to occur. Given that these hemorrhages are of low pressure, recovery is excellent.
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37

Humphrey, Emily. "Chiari malformations in adults: their surgical and nursing management." British Journal of Neuroscience Nursing 15, no. 3 (June 2, 2019): 130–36. http://dx.doi.org/10.12968/bjnn.2019.15.3.130.

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There are two categories of Chiari malformations that occur in adults: type 1 and type 2. Type 1 Chiari malformations are conditions where the bottommost parts of the back of the brain (called the cerebellar tonsils) descend below the skull and enter the spinal canal—the space around the spinal cord. In type 2 Chiari malformations, more of the brain descends below the skull. This means that, as well as the cerebellar tonsils, the lower section of the brainstem (the medulla) and the brain's lowermost fluid-filled cavity, the fourth ventricle, also descend. The descending structures cause pressure and disrupt the normal flow of fluid that circulates in the brain and spinal cord, known as cerebrospinal fluid (CSF). This article further explains the Chiari malformations, presents theories of how they are caused and describes their symptoms and complications. It also discusses Chiari malformations, theories of causation, symptoms, complications, treatment and nursing management considerations.
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PEARCE, J. M. S. "Arnold Chiari, or "Cruveilhier Cleland Chiari" malformation." Journal of Neurology, Neurosurgery & Psychiatry 68, no. 1 (January 1, 2000): 13. http://dx.doi.org/10.1136/jnnp.68.1.13.

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39

Harsha, Kamble Jayaprakash, and Jeevan S. Nair. "Chiari I Malformation Associated with Turner Syndrome." Journal of Neurosciences in Rural Practice 08, no. 02 (April 2017): 277–80. http://dx.doi.org/10.4103/0976-3147.203840.

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ABSTRACTTurner syndrome (TS) is a rare genetic disease due to the absence of one X chromosome. Patients with TS have more subtle neurological/neuropsychiatric problems, while headache is an uncommon clinical presentation which needs attention. We report a 12-year-old child presenting with typical cough headache. Her magnetic resonance imaging revealed Chiari I malformation associated with TS. To the best of our knowledge, Chiari I malformation associated with TS is not described in literature. We report the first case of TS associated with Chiari I malformation. Interestingly, Chiari I malformation is also associated with Noonan’s syndrome, which is a close morphological mimicker of TS, raising the possibility of sharing similar pathogenesis in both conditions.
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40

Shah, Amar S., Alexander T. Yahanda, Umeshkumar Athiraman, Rene Tempelhoff, and Michael R. Chicoine. "Spinal cord infarction with resultant paraplegia after Chiari I decompression: case report." Journal of Neurosurgery: Spine 32, no. 4 (April 2020): 615–21. http://dx.doi.org/10.3171/2019.10.spine19921.

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Paraplegia after posterior fossa surgery is a rare and devastating complication. The authors reviewed a case of paraplegia following Chiari decompression and surveyed the literature to identify strategies to reduce the occurrence of such events.An obese 44-year-old woman had progressive left arm pain, weakness, and numbness and tussive headaches. MRI studies revealed a Chiari I malformation and a cervicothoracic syrinx. Immediately postoperatively after Chiari decompression the patient was paraplegic, with a T6 sensory level bilaterally. MRI studies revealed equivocal findings of epidural hematoma at the site of the Chiari decompression and in the upper thoracic region. Surgical exploration of the Chiari decompression site and upper thoracic laminectomies identified possible venous engorgement, but no hematoma. Subsequent imaging suggested a thoracic spinal cord infarction. Possible explanations for the spinal cord deficit included spinal cord ischemia related to venous engorgement from prolonged prone positioning in an obese patient in the chin-tucked position. At 6.5 years after surgery the patient had unchanged fixed motor and sensory deficits.Spinal cord infarction is rare after Chiari decompression, but the risk for this complication may be increased for obese patients positioned prone for extended periods of time. Standard precautions may be insufficient and intraoperative electrophysiological monitoring may need to be considered in these patients.
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41

Papanastassiou, Alexander M., Richard B. Schwartz, and Robert M. Friedlander. "CHIARI I MALFORMATION AS A CAUSE OF TRIGEMINAL NEURALGIA." Neurosurgery 63, no. 3 (September 1, 2008): E614—E615. http://dx.doi.org/10.1227/01.neu.0000324726.93370.5c.

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ABSTRACT OBJECTIVE Trigeminal neuralgia (TN) is usually associated with vascular compression of the trigeminal nerve, but some cases are associated with central lesions such as tumors, aneurysms, or arteriovenous malformations. In this article, we report the 19th case of TN associated with Chiari I malformation and review clinical outcomes and pathophysiology. CLINICAL PRESENTATION A 63-year-old right-handed man initially presented in 1993 with left-sided lancinating facial pain in the V2 distribution of the trigeminal nerve; the pain was triggered by certain movements, tactile stimulation, or a hot shower. Magnetic resonance imaging revealed a Chiari I malformation associated with a syrinx from C1 to C3. INTERVENTION The patient underwent uncomplicated suboccipital craniectomy, C1 laminectomy, and duraplasty for Chiari decompression. Postoperatively, his pain resolved over a period of 1 year. CONCLUSION Chiari I malformation has been found to be associated with TN in 19 cases in the English-language literature. In patients refractory to medical treatment, suboccipital decompression leads to resolution of pain in about two-thirds of patients. Potential mechanisms for the pathogenesis of TN in the setting of Chiari I malformation are discussed. Chiari I malformation is important to consider as a rare cause of TN that responds to surgical therapy.
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42

Gowhari, Michel, Robert Molokie, Fernando Testai, Victor R. Gordeuk, and Olaide I. Ajayi. "A Case Series: Chiari Malformation and Sickle Cell Disease." Blood 120, no. 21 (November 16, 2012): 4772. http://dx.doi.org/10.1182/blood.v120.21.4772.4772.

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Abstract Abstract 4772 Introduction: Chiari type I malformation is defined as tonsillar herniation of at least 3 to 5 mm below the foramen magnum. The prevalence of Chiari type I malformation in the general population is estimated to be 0.1 to 0.5%. In the SIT (Silent Infarct Transfusion) Trial in children with sickle cell disease, Chiari type I malformation was found as an incidental finding in 22 of 953 (2.3%) of the individuals imaged. Thus, the incidence of Chiari type I malformation in the SIT Trial appeared to be about five times greater than in the general population (Jordan et al Pediatrics 2010). Recently, four hemoglobin SS patients aged 6–17 years with and Chiari type I malformation were reported from one institution (Chonat el al J Pediatr Hematol Oncol 2011). Here we describe six sickle cell disease patients aged 17–24 years with Chiari type I malformation from two institutions. Discussion: All of the cases had severe sickling phenotype, either hemoglobin SS or sickle/B°thalassemia. The majority of the patients were experiencing neurologic symptoms that were subsequently attributed to Chiari type I malformation. Five of the six cases were eventually evaluated by neurosurgery. Neurosurgical intervention with posterior fossa decompression was recommended in four of these individuals. To date one patient has undergone surgery with relief of symptoms. Although Chiari type I malformation is thought to be primarily a congenital disorder, it has also been associated with disorders that are non-congenital. Individuals with sickle cell disease may have a thick calvarium as a result of erythroid hyperplasia and expansion of the diploe of the skull bones. Other conditions with severe anemia and thalassemia have been associated with increased thickness of the calvarium caused by diploic erythroid hyperplasia. One can question whether the increased thickness of the calvarium in some sickle cell patients results in craniocephalic disproportion with a subsequent acquired cerebellar ectopia, hence the increased Chiari type I malformation in patients with sickle cell disease. Central nervous system involvement in sickle cell disease is common. Any patient with sickle cell disease presenting with symptoms indicative of an ongoing or impending neurological event requires an in depth investigation and entails a broad differential. Chiari malformations may need to be part of this differential diagnosis. Disclosures: No relevant conflicts of interest to declare.
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43

Wan, Michael J., Hiroshi Nomura, and Charles H. Tator. "CONVERSION TO SYMPTOMATIC CHIARI I MALFORMATION AFTER MINOR HEAD OR NECK TRAUMA." Neurosurgery 63, no. 4 (October 1, 2008): 748–53. http://dx.doi.org/10.1227/01.neu.0000325498.04975.c0.

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ABSTRACT OBJECTIVE The Chiari I malformation is a rare disorder characterized by downward herniation of the brainstem and cerebellar tonsils through the foramen magnum. Many individuals with the Chiari I malformation do not become symptomatic until adulthood, and the factors that contribute to the onset of symptoms have not been well characterized. The purpose of this systematic chart review was to determine the incidence and validity of minor head or neck trauma as precipitating factors for the onset of symptomatic Chiari I malformation. METHODS The charts of all patients seen by the senior author (CHT) between January 1985 and July 2006 were reviewed to identify patients who had presented with symptomatic Chiari I malformation after minor head or neck trauma. Specific inclusion criteria were used to determine whether the onset of symptoms could be reliably attributed to the minor trauma. RESULTS Of the 85 patients with symptomatic Chiari I malformation seen by the senior author during this time, 11 (12.9%) had a history of minor head or neck trauma preceding the onset of symptoms. Of these, there were 3 patients (3.5%) in whom the onset of symptoms could be attributed to the trauma based on strict inclusion criteria. CONCLUSION Minor head or neck trauma can precipitate the onset of symptoms in a small number of previously asymptomatic patients with Chiari I malformation. Health care professionals must be aware that neurological symptoms that persist or worsen after minor head or neck trauma could indicate an underlying Chiari I malformation.
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44

A, Sharma, K. V. Amrutha, and Abraham J. "ARNOLD CHIARI MALFORMATION: CASE SERIES." International Journal of Anatomy and Research 4, no. 1 (March 31, 2016): 2151–56. http://dx.doi.org/10.16965/ijar.2016.167.

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45

Nacci, A., S. O. Romeo, S. Berrettini, J. Matteucci, M. D. Cavaliere, V. Mancini, E. Panicucci, F. Ursino, and B. Fattori. "Stabilometric findings in patients affected by organic dysphonia before and after phonomicrosurgery." Acta Otorhinolaryngologica Italica 37, no. 4 (August 2017): 286–94. http://dx.doi.org/10.14639/0392-100x-1035.

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Lo scopo di questo studio è stato quello di dimostrare se pazienti affetti da disfonia organica presentino alterazioni posturali caratteristiche e se l’intervento di fonomicrochirurgia delle corde vocali determini modificazioni dell’assetto posturale. Sono stati esaminati 40 soggetti (22 maschi, 18 femmine; età media 32,6 ± 7,5 anni) affetti da disfonia organica (15 polipi cordali, 11 cisti da ritenzione sottomucosa, 10 noduli vocali fibrosi bilaterali e 4 edema di Reinke). L’assetto posturale è stato studiato mediante stabilometria statica in respirazione spontanea, ad occhi aperti e ad occhi chiusi, in tre diverse fasi: prima dell’intervento chirurgico, 24 ore dopo la chirurgia e 6 mesi dopo l’intervento. Le variabili prese in considerazione sono state: le coordinate del centro di pressione sul piano frontale e sagittale, la lunghezza e la superficie della traccia, la velocità media delle oscillazioni e le relative deviazioni standard, l’analisi spettrale della frequenza di oscillazione e i valori di statokinesigramma e stabilogramma. Nessun pattern patologico caratteristico veniva rilevato alla stabilometria in condizioni basali nei diversi sottogruppi di pazienti (polipi, cisti, edema di Reinke). Solo il sottogruppo di pazienti con noduli vocali bilaterali fibrosi (8/10; 80%) mostrava un lieve spostamento in avanti del centro di gravità sia ad occhi aperti che ad occhi chiusi. In tutti i sottogruppi non si verificavano modificazioni significative delle variabili stabilometriche ad occhi aperti e ad occhi chiusi, prima e dopo l’intervento chirurgico. L’uso della stabilometria statica, in questo studio, dimostra l’assenza di alterazioni posturali caratteristiche nei casi di disfonia organica ed esclude che la semplice rimozione della lesione cordale, unilaterale o bilaterale, possa determinare variazioni posturali.
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46

Weber, Peter C., and Stephen P. Cass. "Neurotologic Manifestations of Chiari 1 Malformation." Otolaryngology–Head and Neck Surgery 109, no. 5 (November 1993): 853–60. http://dx.doi.org/10.1177/019459989310900513.

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It is important for otolaryngologists to recognize Chiari malformations as part of the differential diagnosis of balance disorders because patients may initially be seen with symptoms referable to the vestibular system, including ataxia, nystagmus, or vertigo. The objective of this paper is to review the signs, symptoms, and vestibular test findings of a series of patients with Chiari 1 malformation. Six patients were identified by retrospective chart review with a diagnosis of Chiari malformation. Each patient had a complete otoneurologic examination and vestibular function testing. The results indicated that patients fell into two different vestibular test result profiles. First, patients with advanced symptoms demonstrated oculomotor dysfunction, central vestibular nystagmus, abnormal vestibular visual interaction, and abnormal tilt suppression of postrotatory nystagmus. On the other hand, a number of patients were identified with incidentally noted Chiari malformation on magentic resonance imaging scan who had a vestibular test profile consistent with peripheral vestibulopathy without signs and symptoms of central nervous system dysfunction. Guidelines are provided to help determine the extent of the group of symptoms attributable to an incidentally discovered Chiari malformation.
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47

Safin, Sh M., Kh P. Derevyanko, I. M. Gilemkhanova, and M. S. Subramaniam. "Chiari malformation type I with hearing impairment (clinical case)." Vestnik nevrologii, psihiatrii i nejrohirurgii (Bulletin of Neurology, Psychiatry and Neurosurgery), no. 10 (October 1, 2020): 15–19. http://dx.doi.org/10.33920/med-01-2010-02.

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Chiari malformation in the onset of the disease can manifest itself in a different clinical symptom complex. The literature describes isolated cases of a combination of Chiari malformation with hearing impairment. The article presents a description of a clinical case of a combination of Chiari malformation with a pontocerebellar cyst combined with unilateral hearing loss in a 34-year-old patient. As a result of the neurosurgical treatment, hearing restoration and regression of existing neurological symptoms were achieved.
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48

Carvalho, Daniela, Fernando Oikawa, Nilce Mitiko Matsuda, and Alice Tatsuko Yamada. "Budd-Chiari syndrome in association with Behçet's disease: review of the literature." Sao Paulo Medical Journal 129, no. 2 (March 2011): 107–9. http://dx.doi.org/10.1590/s1516-31802011000200009.

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The risk that patients with Behçet's disease may develop various thrombotic complications has been previously described. Although vascular complications from Budd-Chiari syndrome associated with Behçet's disease have been described, the pathogenic mechanisms are still unknown. Severe vascular complications present in Budd-Chiari syndrome associated with Behçet's disease are very common among young male adults. The objective of this study was to review the literature and present the association of Budd-Chiari syndrome with Behçet's disease.
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49

Unal, M., and C. Bagdatoglu. "Arnold-Chiari type I malformation presenting as benign paroxysmal positional vertigo in an adult patient." Journal of Laryngology & Otology 121, no. 3 (December 14, 2006): 296–98. http://dx.doi.org/10.1017/s0022215106003082.

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Arnold-Chiari malformations are a group of congenital hindbrain and spinal cord abnormalities characterized by herniation of the contents of the posterior cranial fossa caudally through the foramen magnum into the upper cervical spine. It is important to recognize Arnold-Chiari type I malformation in the differential diagnosis of adult vertigo cases. We present a 51-year-old patient with Arnold-Chiari type I malformation that was initially diagnosed as posterior semicircular canal benign paroxysmal positional vertigo.
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Schwimmer-Okike, Nneka, Johannes Niebuhr, Grit Gesine Ruth Schramek, Stefan Frantz, and Heike Kielstein. "The Presence of a Large Chiari Network in a Patient with Atrial Fibrillation and Stroke." Case Reports in Cardiology 2016 (2016): 1–3. http://dx.doi.org/10.1155/2016/4839315.

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The Chiari network is an embryological remnant found in the right atrium, mostly without any significant pathophysiological consequences. However, several cardiac associations are reported in the literature including supraventricular tachyarrhythmias. We present a case of a 96-year-old body donor with a stroke episode and intermittent atrial fibrillations. The dissection of the heart revealed the presence of an immense Chiari network with a large central thrombus. The role of a Chiari network in the pathogenesis of stroke and pulmonary embolism is discussed.
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