Relevant bibliographies by topics / Chk 1

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Chk 1'

Author: Grafiati
Published: 4 June 2021
Last updated: 4 February 2022

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Journal articles on the topic "Chk 1"

1

Hammond, Ester M., Rachel A. Freiberg, and Amato J. Giaccia. "The roles of Chk 1 and Chk 2 in hypoxia and reoxygenation." Cancer Letters 238, no. 2 (2006): 161–67. http://dx.doi.org/10.1016/j.canlet.2005.06.029.

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Kim, Seung-Hwan, Antonia H. Holway, Suzanne Wolff, Andrew Dillin, and W. Matthew Michael. "SMK-1/PPH-4.1–mediated silencing of the CHK-1 response to DNA damage in early C. elegans embryos." Journal of Cell Biology 179, no. 1 (2007): 41–52. http://dx.doi.org/10.1083/jcb.200705182.

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During early embryogenesis in Caenorhabditis elegans, the ATL-1–CHK-1 (ataxia telangiectasia mutated and Rad3 related–Chk1) checkpoint controls the timing of cell division in the future germ line, or P lineage, of the animal. Activation of the CHK-1 pathway by its canonical stimulus DNA damage is actively suppressed in early embryos so that P lineage cell divisions may occur on schedule. We recently found that the rad-2 mutation alleviates this checkpoint silent DNA damage response and, by doing so, causes damage-dependent delays in early embryonic cell cycle progression and subsequent lethali
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Caeser, Rebecca, and Triparna Sen. "Should WEE(1) CHK(1) in on the FAM(122A)ily?" Molecular Cell 80, no. 3 (2020): 377–78. http://dx.doi.org/10.1016/j.molcel.2020.10.011.

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Boehrer, Simone, Lionel Ades, Nicolas Tajeddine, et al. "Selective Suppression of the DNA Damage Response in Acute Myeloid Leukemia Versus Myelodysplastic Syndrome." Blood 112, no. 11 (2008): 1656. http://dx.doi.org/10.1182/blood.v112.11.1656.1656.

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Abstract Background: Activation of the DNA-damage-response (DDR)-pathway in preneoplastic lesions of solid neoplasms hinders tumorigenesis and needs to be abolished for transformation, and could also underlie progression of MDS to AML. We previously showed (ASH 2007, abstr n° 2438) in a panel of myeloid cell lines, that γ-irradiation induces apoptosis and G2/M-arrest in the MDS-derived cell lines P39 and MOLM-13, but to a considerably lesser extent in AML cell lines MV4-11 and KG-1; this DDR is mediated by a concomitant increase in the phosphorylation of ATM, Chk-1 and H2AX in MDS-derived cell
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Lee, Byeong-Chel, Shalom Avraham, Akira Imamoto, and Hava Karsenty Avraham. "Identification of the nonreceptor tyrosine kinase MATK/CHK as an essential regulator of immune cells using Matk/CHK-deficient mice." Blood 108, no. 3 (2006): 904–7. http://dx.doi.org/10.1182/blood-2005-12-4885.

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Abstract Matk/CHK knockout mice were reported to show no apparent phenotypic abnormalities. This was thought to be due to the homologous kinase Csk that compensates for Matk/CHK. Here, we present the first evidence that the nonreceptor tyrosine kinase, Matk/CHK, is an important modulator of immune cell signaling. We found that the frequency of primitive hematopoietic cells, the side population c-kit+ Lin– Sca-1+ (SPKLS) cells, in Matk/CHK–/– mice was increased 2.2-fold compared with the control mice. Moreover, Matk/CHK deficiency led to significantly higher pre–B cell colony formation followin
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Yamaguchi, N., Y. Nakayama, T. Urakami, et al. "Overexpression of the Csk homologous kinase (Chk tyrosine kinase) induces multinucleation: a possible role for chromosome-associated Chk in chromosome dynamics." Journal of Cell Science 114, no. 9 (2001): 1631–41. http://dx.doi.org/10.1242/jcs.114.9.1631.

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The Csk family of non-receptor-type tyrosine kinases consists of Csk and the Csk homologous kinase Chk. Each enzyme suppresses the catalytic activity of Src family kinases by phosphorylating their C-terminal negative regulatory tyrosine residues. Ectopic and transient expression of Chk in COS-1 cells showed nuclear localization of Chk and growth inhibition. To further explore the role of Chk in cell growth, we overexpressed Chk in human immature myeloid KMT-2 cells. Chk overexpression brought about growth retardation and aberrant chromosome movement leading to multinucleation, and these events
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Ni, Zhi-Jie, Paul Barsanti, Nathan Brammeier, et al. "4-(Aminoalkylamino)-3-benzimidazole-quinolinones as potent CHK-1 inhibitors." Bioorganic & Medicinal Chemistry Letters 16, no. 12 (2006): 3121–24. http://dx.doi.org/10.1016/j.bmcl.2006.03.059.

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Bourgeois, Alice, Sébastien Bonnet, Sandra Breuils-Bonnet, et al. "Inhibition of CHK 1 (Checkpoint Kinase 1) Elicits Therapeutic Effects in Pulmonary Arterial Hypertension." Arteriosclerosis, Thrombosis, and Vascular Biology 39, no. 8 (2019): 1667–81. http://dx.doi.org/10.1161/atvbaha.119.312537.

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Boehrer, Simone, Lionel Ades, Nicolas Tajeddine, et al. "The Capacity of the Hypomethylating Agents Azacytidine and Decitabine to Induce Apoptosis and Cell Cycle Arrest Depends on the Activation of the DNA-Damage Response Pathway." Blood 112, no. 11 (2008): 1655. http://dx.doi.org/10.1182/blood.v112.11.1655.1655.

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Abstract Background: The hypomethylating agents azacytidine (AZA) and decitabine (DEC) have shown clinical efficacy in patients (pts) with MDS. There is in vitro evidence that both agents, in addition to their hypomethylating effect, also function by inducing apoptosis, cell cycle arrest and/or the activation of a DNA damage response (DDR). However, the exact contributions of those mechanisms of action and their functional interdependence remain to be defined. Methods: A panel of MDS (P39, MDS-1)- and AML (HL-60, KG-1)-derived cell lines were incubated with increasing dosages of AZA (1–2μM) an
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Nagy, Zoltan, Jun Mori, Vanesa-Sindi Ivanova, Alexandra Mazharian, and Yotis A. Senis. "Interplay between the tyrosine kinases Chk and Csk and phosphatase PTPRJ is critical for regulating platelets in mice." Blood 135, no. 18 (2020): 1574–87. http://dx.doi.org/10.1182/blood.2019002848.

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Abstract The Src family kinases (SFKs) Src, Lyn, and Fyn are essential for platelet activation and also involved in megakaryocyte (MK) development and platelet production. Platelet SFKs are inhibited by C-terminal Src kinase (Csk), which phosphorylates a conserved tyrosine in their C-terminal tail, and are activated by the receptor-type tyrosine phosphatase PTPRJ (CD148, DEP-1), which dephosphorylates the same residue. Deletion of Csk and PTPRJ in the MK lineage in mice results in increased SFK activity, but paradoxically hypoactive platelets resulting from negative feedback mechanisms, includ
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Dissertations / Theses on the topic "Chk 1"

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O'Neil, Crystal L. "Enzyme Exploitation: Manipulating Enzyme Function for Therapy, Synthesis and Natural Product Modification." The Ohio State University, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=osu1293722936.

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Ghelli, Luserna di Rorà Andrea <1987&gt. "The Inhibition of Chk1/Chk2 and Wee-1 Kinases as a Promising Therapy for the Treatment of Adult Acute Lymphoblastic Leukemia." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2016. http://amsdottorato.unibo.it/7529/.

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Due to inadequate treatments, the survival rate of adult patients with acute lymphoblastic leukemia (ALL) is still very poor. Thus there is a need to improve the efficacy of conventional therapy. In this study we evaluated the effectiveness of checkpoint kinase inhibitors (Chk-i) in single agent and in combination with different compounds conventionally used for the treatment of B-/T-ALL. We showed that Chk1 and Chk2 kinases are highly expressed and hyper-activated in tumor samples in comparison to normal tissue. On these bases we speculate that the inhibition of these kinases could mine the g
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DETIVAUD, LENAICK. "Caracterisation et purification partielle de cdk x, une proteine proche de cdk 1." Paris 6, 1999. http://www.theses.fr/1999PA066152.

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Les cdks (kinases cycline-dependantes) constituent une famille de proteines kinases dont le nombre d'elements identifies ne cesse de croitre. Ces cdks ont ete d'abord identifiees comme molecules cles du cycle cellulaire. L'implication de certaines d'entre elles dans d'autres fonctions et la decouverte de cdks et homologues dans des tissus bien differencies permettent d'envisager un spectre d'action plus large pour ces proteines kinases. Nous avons etudie une nouvelle proteine d'environ 35 kda, et l'activite hmg i kinase qui copurifie sur la resine d'affinite p15 c d k - b p-sepharose. De meme
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Schwalowsky, Lutz. "Die ferroelastische Phasenumwandlung von Verbindungen des Letovizit-Typs, [(NH4)1-xRbx]3H(SO4)2, x=0.0-0.3 [NH4 1-chi Rb chi 3H SO4 2, chi=0.0-0.3]." [S.l. : s.n.], 2000. http://www.sub.uni-hamburg.de/disse/167/diss.pdf.

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Ådna, Jostein. "Jerusalemer Tempel und Tempelmarkt im 1. Jahrhundert n. Chr. /." Wiesbaden : Harrassowitz Verl, 1999. http://catalogue.bnf.fr/ark:/12148/cb376523372.

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Texte remanié de: Diss.--Theologische Gemeindefakultät--Oslo, 1994. Titre de soutenance : Jesu Kritik am Tempel. Eine Untersuchung zum Verlauf und Sinn der sogenannten Tempelreinigung Jesu, Markus 11, 15-17 und Parallelen.<br>Bibliogr. p. 151-170. Index.
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Rohmann, Dirk. "Gewalt und politischer Wandel im 1. Jahrhundert n. Chr. /." München : H. Utz, 2006. http://catalogue.bnf.fr/ark:/12148/cb410351899.

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Ungaro, Francesca Consiglia [Verfasser], and Karl [Akademischer Betreuer] Jakobs. "Search for the Supersymmetric Partner of the Top Quark with the ATLAS Detector via $\tildet}^}_{1} \rightarrow t \widetilde{\chi}^{0}_{1}$ and $\tilde{t}^{}_{1} \rightarrow b \widetilde{\chi}^{\pm}_{1}$ Decays." Freiburg : Universität, 2015. http://d-nb.info/1119805546/34.

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Kühnen, Angela. "Die Imitatio Alexandri in der römischen Politik : 1. Jh. v. Chr. - 3. Jh. n. Chr. /." Münster : Rhema, 2008. http://catalogue.bnf.fr/ark:/12148/cb41293881k.

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Wiele, Nils. "Phononenzustandsdichten von FexPt1-x [Fe chi Pt 1 chi] gemessen mit nuklearer inelastischer Absorption von Synchrotronstrahlung." [S.l.] : [s.n.], 2001. http://deposit.ddb.de/cgi-bin/dokserv?idn=963754335.

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Tai, Helen H. "The role of Xist and CHD-1 in gene silencing." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0018/NQ57070.pdf.

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Books on the topic "Chk 1"

1

Che 1-kwa che 1-chang: Yi Mu-yŏng sosŏl. Munidang, 2007.

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Na, Un-mong. 1-chang 1-che sŏlgyo moŭmjip. Aehyangsuk Chʻulpʻanbu, 1989.

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Hŏnpŏp che 1-cho nŭn ŏnjena minju konghwaguk: Yi Chʻŏl-bŏm sijip. Hanul, 1991.

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Camilotti, Silvia, and Sara Civai. Straniero a chi? Edizioni Ca' Foscari, 2018. http://dx.doi.org/10.30687/978-88-6969-290-1.

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Questo volume raccoglie alcuni dei numerosi testi che gli studenti delle scuole secondarie di primo e secondo grado della regione Veneto hanno inviato all’Archivio Scritture Scrittrici Migranti, partecipando al concorso letterario ‘Straniero a chi? Scriviamo le migrazioni’. A emergere sono i grandi nuclei tematici della migrazione: l’alterità, condizione avvertita come propria non solo di chi è costretto a migrare, ma anche di quanti avvertono un senso di estraneità al mondo che li circonda; il trauma del viaggio, le speranze in un futuro diverso e lo scontro con una realtà durissima, sia dura
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Yoshimura, Tatsuya. Chu lian 1/2. Zheng Wen, 2002.

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Bando, Masako. Tabihate no chi: 1. Kadokawa Shoten, 2001.

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Soltakhanov, Ė. Kh. Abat: 1-chu klassana. Nokhchiĭn pachkhʹalkhan knizhni izd-vo "Zhaĭna", 1997.

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Taehan Min'guk ŭn minju konghwaguk ida: Hŏnpŏp che 1-cho sŏngnip ŭi yŏksa. Tolbegae, 2013.

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Nam-Puk ŏnŏ tongjilsŏng hoebok ŭl wihan kukche haksul hoeŭi (1st 2001 Beijing, China). Nam-Puk ŏnŏ tongjilsŏng hoebok ŭl wihan che 1-cha haksul hoeŭi nonmunjip. Kungnip Kugŏ Yŏnguwŏn, 2002.

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Cha 1% jiu ai shang ni: 1% closer. Huang guan chu ban she (Xianggang) you xian gong si, 2007.

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Book chapters on the topic "Chk 1"

1

van Roy, Frans, Volker Nimmrich, Anton Bespalov, et al. "CHK." In Encyclopedia of Signaling Molecules. Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_100262.

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Zhang, Haiying, Zhan Xiao, and Tom Sowin. "Chk1 and Chk2 as Checkpoint Targets." In Checkpoint Controls and Targets in Cancer Therapy. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60761-178-3_16.

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Vogt, J. "53 CHF Fluoromethylene." In Asymmetric Top Molecules. Part 1. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-10371-1_55.

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van Roy, Frans, Volker Nimmrich, Anton Bespalov, et al. "Cholecystokinin-1 Receptor: CCK-1 Receptor." In Encyclopedia of Signaling Molecules. Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_100265.

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Goto, Hidemasa, Hironori Inaba, and Masaki Inagaki. "Chk1." In Encyclopedia of Signaling Molecules. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4614-6438-9_101557-1.

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Beckwith, A. L. J. "4.3.1 Secondary alkyl radicals, type R1–CH2–ĊH–CH2–CH2–R2." In Inorganic Radicals, Metal Complexes and Nonconjugated Carbon Centered Radicals. Part 1. Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-48466-0_90.

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Hirota, E., K. Kuchitsu, T. Steimle, J. Vogt, and N. Vogt. "30 CHP+ Methylidynephosphine (1+)ion." In Molecules Containing No Carbon Atoms and Molecules Containing One or Two Carbon Atoms. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-540-70614-4_231.

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Gooch, Jan W. "CHC." In Encyclopedic Dictionary of Polymers. Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_2231.

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Gooch, Jan W. "CHR." In Encyclopedic Dictionary of Polymers. Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_2346.

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Gooch, Jan W. "CFK." In Encyclopedic Dictionary of Polymers. Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_2174.

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Conference papers on the topic "Chk 1"

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Maharaj, Anil, Abigail Davis, Bo Zhong, Martine F. Roussel, and Clinton F. Stewart. "Abstract 4927: Plasma and CNS pharmacokinetics of the CHK-1 inhibitor prexasertib (LY-2606368) in mice bearing orthotopic group 3 medulloblastoma." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-4927.

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Lampert, Erika J., Min-Jung Lee, Jane B. Trepel, et al. "Abstract 5000: Cell cycle checkpoint kinase (CHK)1 inhibition induces immune modulation in recurrentBRCAwild-type high-grade serous ovarian cancer (HGSOC) patients." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-5000.

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Lampert, Erika J., Min-Jung Lee, Jane B. Trepel, et al. "Abstract 5000: Cell cycle checkpoint kinase (CHK)1 inhibition induces immune modulation in recurrentBRCAwild-type high-grade serous ovarian cancer (HGSOC) patients." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-5000.

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Bendell, Johanna, Stefan Grant, Filip Janku, et al. "Abstract CT240: Checkpoint kinase (CHK) 1/2 inhibitor LY2606368 in a phase I dose-expansion study in patients with squamous cell carcinoma of the head and neck." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-ct240.

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André, Paul, and Max L. Wilson. "CHI Madness: Part 1." In the 2011 annual conference. ACM Press, 2011. http://dx.doi.org/10.1145/1978942.2167177.

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Stolze, Markus, Jürgen Koenemann, Daniela Handl, Barbara Hayes Roth, and Joseph Kramer. "Designing interactive systems for 1-to-1 e-commerce." In CHI '00 extended abstracts. ACM Press, 2000. http://dx.doi.org/10.1145/633292.633512.

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Chung, Injae. "Abstract 6: New chk1 inhibitor development & the feasibility of use of chk1 inhibitor with antimetabolite for NSCLC." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-6.

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Voinov, Vassilly, Mikhail Nikulin, and Natalie Pya. "Independent $\chi^{2}_{1}$ distributed in the limit components of some chi-squared tests." In Recent Advances in Stochastic Modeling and Data Analysis. WORLD SCIENTIFIC, 2007. http://dx.doi.org/10.1142/9789812709691_0030.

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Pietriga, Emmanuel. "Session details: Pointing 1." In CHI '11: CHI Conference on Human Factors in Computing Systems. ACM, 2011. http://dx.doi.org/10.1145/3248994.

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Agrawala, Maneesh. "Session details: Visualization 1." In CHI '09: CHI Conference on Human Factors in Computing Systems. ACM, 2009. http://dx.doi.org/10.1145/3256974.

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Reports on the topic "Chk 1"

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Sharp, Jeremy, Locke Williams, Duncan Bryant, et al. Rough River Outlet Works physical model study. Engineer Research and Development Center (U.S.), 2021. http://dx.doi.org/10.21079/11681/41043.

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The US Army Corps of Engineers, Louisville District, requested the support and assistance of the US Army Engineer Research and Development Center, Coastal and Hydraulics Laboratory (CHL), in the evaluation of the hydraulic performance of the replacement Outlet Works for Rough River Dam. To support the design effort, CHL constructed a 1:25.85 scale physical model. The proposed features of the model in the domain are the curved approach channel, intake structure, transition, curved conduit, stilling basin, concrete apron, and retreat channel. Tests performed to evaluate the hydraulic performance
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Steger, H. F., and W. S. Bowman. Ch-1 and ch-2: certified reference gold ores. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 1985. http://dx.doi.org/10.4095/307256.

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Schneider, Brandt. Role of the Cdk Inhibitor Sic 1 in Start. Defense Technical Information Center, 1998. http://dx.doi.org/10.21236/ada359281.

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Schneider, Brandt. Role of the Cdk Inhibitor Sic 1 in Start. Defense Technical Information Center, 2000. http://dx.doi.org/10.21236/ada392485.

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Schneider, Brandt. Role of the Cdk Inhibitor Sic 1 in Start. Defense Technical Information Center, 1999. http://dx.doi.org/10.21236/ada378108.

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Steger, H. F., and W. S. Bowman. Ch-1 et ch-2: minerais d'or de référence certifiés. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 1985. http://dx.doi.org/10.4095/307257.

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Armstrong, T. A., D. Bettoni, and V. Bharadwaj. Study of the {chi}{sub 1} and {chi}{sub 2} charmonium states formed in {anti p}p annihilations. Office of Scientific and Technical Information (OSTI), 1998. http://dx.doi.org/10.2172/663247.

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Wade, Elisabeth Ayn. Photodissociation of ketene: CH2CO → CH2(a1A1) + CO(v=1) rates and dynamics. Office of Scientific and Technical Information (OSTI), 1996. http://dx.doi.org/10.2172/451218.

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Ringer, Lymor. Role of p53 in cdk Inhibitor VMY-1-103-Induced Apoptosis in Prostate Cancer. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada567635.

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Ringer, Lymor. Role of p53 in cdk Inhibitor VMY-1-103-induced Apoptosis in Prostate Cancer. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada592440.

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