Relevant bibliographies by topics / Chlamydophia pneumoniae

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  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
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Academic literature on the topic 'Chlamydophia pneumoniae'

Author: Grafiati
Published: 4 June 2021
Last updated: 13 February 2022

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Journal articles on the topic "Chlamydophia pneumoniae"

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Kim, Woo Jin, Hui Young Lee, Seung-Joon Lee, et al. "Development of Protein Chip for Diagnosis of Chlamydophia Pneumoniae." Tuberculosis and Respiratory Diseases 60, no. 4 (2006): 412. http://dx.doi.org/10.4046/trd.2006.60.4.412.

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Fujita, Jiro, and Takeshi Kinjo. "Where is Chlamydophila pneumoniae pneumonia?" Respiratory Investigation 58, no. 5 (2020): 336–43. http://dx.doi.org/10.1016/j.resinv.2020.06.002.

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Berebichez-Fridman, R., R. Blachman-Braun, S. Azrad-Daniel, R. Vázquez-Campuzano, and R. Vázquez-López. "Atypical pneumonias caused by Legionella pneumophila, Chlamydophila pneumoniae and Mycoplasma pneumonia." Revista Médica Del Hospital General De México 78, no. 4 (2015): 188–95. http://dx.doi.org/10.1016/j.hgmx.2015.06.005.

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Miyashita, Naoyuki, Hiroshi Fukano, Keiji Mouri, et al. "Community-acquired pneumonia in Japan: a prospective ambulatory and hospitalized patient study." Journal of Medical Microbiology 54, no. 4 (2005): 395–400. http://dx.doi.org/10.1099/jmm.0.45920-0.

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In this study the aetiology of community-acquired pneumonia (CAP) in Japan was investigated and the incidence of causative pathogens in ambulatory and hospitalized patients was compared. In addition, the roles of Chlamydophila felis and Chlamydophila pecorum as causes of CAP were investigated. Five hundred and six patients with CAP who visited an outpatient clinic or were admitted to one of three different hospitals were enrolled in this study; 106 of them were outpatients and 400 were hospitalized patients. Among the 506 CAP cases, Mycoplasma pneumoniae was the most common pathogen found in the outpatients and Streptococcus pneumoniae was the most common in the hospitalized patients. No cases of Chlamydophila pecorum pneumonia were observed and only one patient had an antibody titre suggestive of recent infection with the feline strain of Chlamydophila. The incidence of infection with M. pneumoniae and Chlamydophila pneumoniae was higher among the outpatients than among hospitalized patients, whereas the incidence of infection with S. pneumoniae and Haemophilus influenzae was higher among the hospitalized patients. Recognition of these results will allow prompt and appropriate antimicrobial therapy to be provided using Japanese CAP guidelines.
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Sybilski, Adam J. "Atypical pathogen infection of respiratory system." Medycyna Faktów 14, no. 1 (2021): 78–81. http://dx.doi.org/10.24292/01.mf.0121.10.

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The frequency of respiratory infections, especially atypical pneumonia, is increasing significantly. Most often, atypical pneumonia is caused by Mycoplasma pneumoniae and Chlamydophila pneumoniae. Human is the only reservoir of these atypical bacteria. The infection occurs via droplets or direct contact with a sick person or convalescent. Pneumonia of the etiology of Mycoplasma pneumoniae and Chlamydophila pneumoniae most often affects children without comorbidities and is usually mild, while most patients with Legionella infection require intensive care treatment. Symptoms of mycoplasma infection can range from mild symptoms in the upper respiratory tract to pneumonia and extrapulmonary symptoms. The infection is often underdiagnosed, and patients usually do not seek medical attention and treatment. Chlamydial pneumonia is, in most cases, mild, similar to Mycoplasma pneumoniae, and tends to heal itself. The drugs of choice in the treatment of atypical pneumonia are macrolides, and because of the best compliance in children – azithromycin.
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Rosario Martín, Cristina, Blanca Navarro Cubells, and Francisco Carrión Valero. "Immune-mediated Leukopenia due to Chlamydophila pneumoniae Pneumonia." Archivos de Bronconeumología (English Edition) 51, no. 12 (2015): 663–64. http://dx.doi.org/10.1016/j.arbr.2015.10.004.

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Selvaraj, Jayaraman. "The porin AaxA protein model from Chlamydia pneumonia." Bioinformation 16, no. 10 (2020): 786–88. http://dx.doi.org/10.6026/97320630016786.

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Chlamydophila pneumoniae is an intracellular pathogen accountable for various acute respiratory infections. C. pneumoniae has a gene cluster which encodes a putative outer membrane porin (aaxA), arginine decarboxylase (CPn1032 or aaxB) and a putative cytoplasmic membrane transporter (CPn1031 or aaxC). Therefore, it is of interest to document a molecular protein model of porin AaxA from Chlamydia pneumonia to gain structure to functional insight on the protein.
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Koshkarina, Е. A., O. V. Kovalishena, N. V. Saperkin, V. V. Krasnov, Р. G. Zubarov, and O. М. Chekanina. "Assessment of current laboratory diagnosis of pneumococcal community-acquired pneumonia." Fundamental and Clinical Medicine 5, no. 4 (2020): 21–29. http://dx.doi.org/10.23946/2500-0764-2020-5-4-21-29.

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Aim. To investigate the aetiology of community-acquired pneumonia in hospitalised children and to evaluate the accuracy of the methods for its laboratory confirmation. Materials and Methods. We performed descriptive and cross-sectional epidemiological studies. Results of the rapid immunochromatographic assay (ICT) were compared with those obtained by polymerase chain reaction (PCR). Results. DNA of Streptococcus pneumoniae and Mycoplasma pneumoniae was found in 65.5% and 13.8% of the patients. Microbial associations were observed in 13.7% of patients (Mycoplasma pneumoniae + Streptococcus pneumoniae, 10.3%; Streptococcus pneumoniae + Haemophilus influenzae, 3.4%). Chlamydophila pneumoniae and SARS-CoV-2 were not detected. The cause of community-acquired pneumonia was not identified in 6.9% of the cases. A diagnostic accuracy of ICT was 27.58% and its sensitivity was relatively small (9.09%; 95% CI 1; 29), compared with a relatively high specificity (85.7%; 95% CI 42; 100). Conclusions. Rapid ICT assay must be accompanied by the PCR or other diagnostic methods for the diagnosis of pneumococcal community-acquired pneumonia in children.
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Blasi, F., P. Tarsia, and S. Aliberti. "Chlamydophila pneumoniae." Clinical Microbiology and Infection 15, no. 1 (2009): 29–35. http://dx.doi.org/10.1111/j.1469-0691.2008.02130.x.

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Burillo, Almudena, and Emilio Bouza. "Chlamydophila pneumoniae." Infectious Disease Clinics of North America 24, no. 1 (2010): 61–71. http://dx.doi.org/10.1016/j.idc.2009.10.002.

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Dissertations / Theses on the topic "Chlamydophia pneumoniae"

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Wehrl, Wolfgang. "Funktionelle Proteomanalyse von Chlamydophila pneumoniae." [S.l. : s.n.], 2004. http://www.diss.fu-berlin.de/2005/18/index.html.

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Braz, Maria Amador. "Detecção e caracterização molecular de Chlamydophila psittaci e Chlamydophila abortus em aves assintomáticas /." Araçatuba : [s.n.], 2012. http://hdl.handle.net/11449/94591.

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Orientador: Marcelo Vasconcelos Meireles
Banca: Raphael Lúcio Andreatti Filho
Banca: Gisele Fabrino Machado
Resumo: Chlamydophila psittaci é uma bactéria que causa doença respiratória ou sistêmica em aves e em seres humanos. Há ainda, alguns relatos de infecção em aves por Chlamydophila abortus, que é um agente etiológico de problemas reprodutivos em mamíferos. Em vista do risco de transmissão para humanos a partir de aves assintomáticas o objetivo deste estudo foi detectar a presença de C. psittaci e C. abortus em amostras de fezes ou suabes cloacais de aves assintomáticas. Foram colhidas 403 amostras fecais ou suabes cloacais, provenientes de aves domésticas, selvagens ou exóticas, mantidas em cativeiro ou oriundas de apreensão. As amostras foram submetidas à PCR em tempo real para C. psittaci e C. abortus, para amplificação de fragmento parcial do gene da subunidade 16S do rRNA, utilizando o SsoFast™ EvaGreen® Supermix (Bio-Rad) e análise da curva de dissociação. Para determinação do genótipo de C. psittaci, foi utilizada a hemi- nested PCR específica para o gene OMP-A, realizada nas amostras positivas pela PCR em tempo real, seguida de sequenciamento dos fragmentos amplificados. A PCR em tempo real revelou positividade em 17 (4,21%) amostras. A hemi-nested foi positiva em 2 amostras positivas pela PCR em tempo real. O genótipo A de C. psittaci foi identificado pelo sequenciamento de uma amostra amplificada pela hemi-nested PCR
Abstract: Chlamydophila psittaci is a bacterium that causes respiratory or systemic disease in birds and humans. In birds there is also some reports of infection by Chlamydophila abortus that is responsible for abortions in mammals. Owing to the risk of transmission of Chlamydophila from asymptomatic birds to humans, the objective of this study was to detect the presence of C. psittaci and C. abortus in asymptomatic birds. Four hundred and three fecal samples or cloacal swabes were collected from domestic, wild or exotic birds kept in captivity or from apprehension. The 403 samples were examined by real time PCR specific for the 16S subunit of rRNA gene using SsoFastEvaGreen®Supermix™(Bio-Rad) and melting curve analysis. Hemi- nested PCR specific for the OMP-A gene, accomplished in real-time PCR positive samples, followed by sequencing of the amplified fragments were used to determine the genotype of C. psittaci. Real-time PCR was positive in 17 (4.21%) samples. Hemi-nested PCR revealed positivity in two samples previously positive by real-time PCR. Sequencing of the fragment amplified by hemi-nested PCR allowed for the identification of genotype A of C. psittaci in one sample
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Braz, Maria Amador [UNESP]. "Detecção e caracterização molecular de Chlamydophila psittaci e Chlamydophila abortus em aves assintomáticas." Universidade Estadual Paulista (UNESP), 2012. http://hdl.handle.net/11449/94591.

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Made available in DSpace on 2014-06-11T19:27:15Z (GMT). No. of bitstreams: 0 Previous issue date: 2012-03-29Bitstream added on 2014-06-13T18:31:11Z : No. of bitstreams: 1 braz_ma_me_araca.pdf: 405940 bytes, checksum: bb0a5b6b5df0a0da63d3d467bb5e891c (MD5)
Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)
Chlamydophila psittaci é uma bactéria que causa doença respiratória ou sistêmica em aves e em seres humanos. Há ainda, alguns relatos de infecção em aves por Chlamydophila abortus, que é um agente etiológico de problemas reprodutivos em mamíferos. Em vista do risco de transmissão para humanos a partir de aves assintomáticas o objetivo deste estudo foi detectar a presença de C. psittaci e C. abortus em amostras de fezes ou suabes cloacais de aves assintomáticas. Foram colhidas 403 amostras fecais ou suabes cloacais, provenientes de aves domésticas, selvagens ou exóticas, mantidas em cativeiro ou oriundas de apreensão. As amostras foram submetidas à PCR em tempo real para C. psittaci e C. abortus, para amplificação de fragmento parcial do gene da subunidade 16S do rRNA, utilizando o SsoFast™ EvaGreen® Supermix (Bio-Rad) e análise da curva de dissociação. Para determinação do genótipo de C. psittaci, foi utilizada a hemi- nested PCR específica para o gene OMP-A, realizada nas amostras positivas pela PCR em tempo real, seguida de sequenciamento dos fragmentos amplificados. A PCR em tempo real revelou positividade em 17 (4,21%) amostras. A hemi-nested foi positiva em 2 amostras positivas pela PCR em tempo real. O genótipo A de C. psittaci foi identificado pelo sequenciamento de uma amostra amplificada pela hemi-nested PCR
Chlamydophila psittaci is a bacterium that causes respiratory or systemic disease in birds and humans. In birds there is also some reports of infection by Chlamydophila abortus that is responsible for abortions in mammals. Owing to the risk of transmission of Chlamydophila from asymptomatic birds to humans, the objective of this study was to detect the presence of C. psittaci and C. abortus in asymptomatic birds. Four hundred and three fecal samples or cloacal swabes were collected from domestic, wild or exotic birds kept in captivity or from apprehension. The 403 samples were examined by real time PCR specific for the 16S subunit of rRNA gene using SsoFastEvaGreen®Supermix™(Bio-Rad) and melting curve analysis. Hemi- nested PCR specific for the OMP-A gene, accomplished in real-time PCR positive samples, followed by sequencing of the amplified fragments were used to determine the genotype of C. psittaci. Real-time PCR was positive in 17 (4.21%) samples. Hemi-nested PCR revealed positivity in two samples previously positive by real-time PCR. Sequencing of the fragment amplified by hemi-nested PCR allowed for the identification of genotype A of C. psittaci in one sample
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Fagundes, Raquel de Queiroz. "Estudo da co-participação de infecção natural pela Chlamydophila pneumoniae e Mycoplasma pneumoniae na aterogênese experimental em coelhos." Universidade de São Paulo, 2006. http://www.teses.usp.br/teses/disponiveis/5/5131/tde-16102014-102605/.

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Fatores de risco clássicos descritos como associados ao desenvolvimento da aterosclerose nem sempre conseguem explicar uma variabilidade da evolução da doença entre um indivíduo e outro. Evidências consistentes mostram que a inflamação exerce papel crucial na patogenia da aterosclerose. Recentemente, a infecção por alguns agentes, principalmente a C. pneumoniae, tem sido progressivamente aventada como possível fator colaborador no processo de aterogênese. O Mycoplasma pneumoniae, bactéria que utiliza colesterol para sua proliferação, foi descrito no interior de placas gordurosas humanas, conjuntamente com a C.pneumoniae. Objetivo - O presente estudo tem como objetivo verificar se antígenos dos agentes infecciosos M. pneumoniae e C. pneumoniae estão presentes na parede das artérias aortas normais dos coelhos, animal que tem sido freqüentemente utilizado no estudo da aterosclerose, e se a introdução de dieta rica em colesterol leva a proliferação desses microorganismos concomitante com o desenvolvimento da placa aterosclerótica e de inflamação. Material e Métodos - Foram estudados 39 coelhos da raça Nova Zelândia, divididos em três grupos conforme a alimentação: Grupo A - dieta normal , Grupo B - dieta com 1% de colesterol por 8 semanas e Grupo C - dieta com 1% de colesterol por 12 semanas. O colesterol total e frações foram dosados bioquimicamente, após sacrifício dos animais. Avaliou-se macroscopicamente a quantidade de placas de gordura na superfície da aorta através da coloração pelo Sudan IV e microscopicamente dois segmentos transversais: um torácico e um abdominal os quais foram submetidos a reações de Imunohistoquímica para antígenos infecciosos. Resultados - Antígenos da C. pneumoniae e M. pneumoniae estiveram presentes em todos os animais estudados, porém em quantidades progressivamente maiores nos grupos A, B e C. Houve forte correlação positiva entre a quantidade de antígenos infecciosos e espessura da camada íntima, inflamação e quantidade de gordura, à histologia tanto no segmento torácico quanto no abdominal. Houve correlação negativa entre perímetro do vaso e tamanho das placas no segmento torácico, cujas placas apresentaram maior porcentagem de fibrose comparadas com o segmento abdominal, indicando que as placas dessa região apresentam mais características evolutivas para placa estável, ou seja, remodelamento negativo do vaso e capa de fibrose. A intensidade da inflamação tanto na adventícia quanto na íntima se correlacionou positivamente à quantidade de ambos os patógenos reforçando a hipótese de que esses patógenos estão influenciando no desencadeamento da inflamação e desenvolvimento de aterosclerose. Os níveis plasmáticos de colesterol total e suas frações também se correlacionaram positivamente com os agentes infecciosos. Em conclusão - A alimentação rica em colesterol levou a aumento da intensidade de antígenos de Mycoplasma pneumoniae e C. pneumoniae presentes na íntima da aorta de coelhos, em correlação com aumento da espessura intimal e inflamação na parede do vaso, favorecendo de forma intensa a hipótese de que esses agentes têm papel importante na aterogênese. A presença desses microorganismos em pequena quantidade nos animais controles reforça a idéia de que esses microorganismos são habitantes usuais de mamíferos e que fatores de risco tais como o colesterol induzem sua proliferação e o desenvolvimento da aterosclerose
Classical risk factors described to be associated with the development of atherosclerosis do not completely explain the variability of the disease occurring among the individuals. Consistent evidences show that inflammation exerts a fundamental role in the pathogenesis and severity of atherosclerosis. Recentently, some infectious agents, specially the Chlamydophila pneumoniae (C.pneumoniae), have progressively been claimed as possible factor acting in the progression of atherosclerosis. Mycoplasma pneumoniae (M.pneumoniae), a bacterium that needs cholesterol for proliferation, has been described in human fat plaques, in conjunction with C.pneumoniae. Objective - The present study has the objective of analyzing if M. pneumoniae and C. pneumoniae antigens are present at the rabbit aorta arterial walls, as the rabbit is an animal model frequently used in studies of atherosclerosis; and if the cholesterol enriched diet may cause proliferation of these microorganisms concomitantly with the development of atherosclerotic plaques and inflammation. Material and Methods - 39 aortas from New Zealand rabbits divided in three groups according to the diet were studied: Group A -normal dieta, Group B - 1% cholesterol enriched diet for 8 weeks and Group C - 1% cholesterol enriched diet for 12 weeks. The serum levels of total and fractions of cholesterol were biochemically measured after animals killed. A macroscopic study of the percentage area occupied by fat at the intimal aorta surface was performed, using the Sudan IV stain. A microscopic study was performed in two transversal segments: a thoracic and an abdominal one, which were submitted to immunohistochemistry for detection of these infectious agents. Results - C. pneumoniae and M. pneumoniae Ags were present in all studied animals, however in progressively higher amounts in groups A, B and C. There was a strong positive correlation between microscopic amounts of infectious antigens and intimal thickness, inflammation and intraplaque fat, both at the thoracic and abdominal segments. There was a negative correlation between vessel perimeter and plaque area at the thoracic segment, where the plaques presented higher percentage of fibrosis compared with the abdominal segment, suggesting that the thoracic region plaques have characteristics for progression to estabilization: negative remodeling of the vessel and fibrous cap. The quantity of Inflammation at both intima and adventitia correlated positively with these infectious agents, re-inforcing the hypothesis that infectious antigens are inducing inflammation and development of atherosclerosis. Serum levels of total cholesterol and their fractions also positively correlated with the amount of infectious antigens. In conclusion - A rich cholesterol diet caused an increase of M. pneumoniae and C.pneumoniae antigens at intima of rabbit aortas, in strong positive correlation with intimal thickness and inflammation of the aorta wall, favoring the hypothesis that these infectious agents have important role in atherogenesis. The presence of small amount of these microorganisms\' antigens at the controls re-inforces the Idea that these microorganisms are usual inhabitants of mammals and that the risk factors such as cholesterol induce their proliferation and acceleration of atherosclerogenesis.
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Förster, Stefanie [Verfasser]. "Nod1 vermittelt Endothelzellaktivierung durch Chlamydophila pneumoniae / Stefanie Förster." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2008. http://d-nb.info/1022909509/34.

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Klucken, Andrea C. [Verfasser]. "Chlamydophila pneumoniae - induzierte Aktivierung humaner Endothelzellen / Andrea C. Klucken." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2008. http://d-nb.info/1023361523/34.

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Pun, Chi-kit Patrick. "Molecular diagnosis of adenovirus, mycoplasma pneumoniae and Chlamydia pneumoniae infection in hospitalized children." Click to view the E-thesis via HKUTO, 2004. http://sunzi.lib.hku.hk/hkuto/record/B31972123.

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Maia, Irineu Luiz. "Análise da prevalência de chlamydia pneumoniae e mycoplasma pneumoniae em diferentes formas de apresentação da doença coronária obstrutiva." Universidade de São Paulo, 2006. http://www.teses.usp.br/teses/disponiveis/5/5131/tde-24012008-135308/.

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Introdução: recente estudo brasileiro detectou a presença concomitante do Mycoplasma pneumoniae e Chlamydia pneumoniae em lesões ateromatosas coronárias estáveis e instáveis. O objetivo do presente estudo foi testar a associação entre títulos sorológicos de anticorpos anti-Chlamydia pneumoniae e anti-Mycoplasma pneumoniae e as Síndromes Isquêmicas Miocárdicas Instáveis. Métodos: foram incluídos de forma prospectiva, 138 pacientes divididos em 4 grupos: 34 pacientes com Síndrome Isquêmica Miocárdica Instável com supradesnível do segmento ST, 40 pacientes com Síndrome Isquêmica Miocárdica Instável sem supradesnível ST, 30 pacientes com aterosclerose crônica assintomática e 34 doadores de sangue sem doença coronária conhecida. Nos dois primeiros grupos, as amostras sorológicas foram colhidas durante o evento agudo e com seis meses de seguimento, enquanto nos outros dois (aterosclerose crônica e controle) as mesmas foram colhidas uma única vez. Em todas as amostras foram dosados anticorpos da classe IgG anti-Chlamydia pneumoniae e anti-Mycoplasma pneumoniae utilizando a técnica de imunoflorescência indireta.. Resultados: seis meses após a internação, os pacientes com síndrome isquêmica miocárdica instável com supradesnível ST apresentaram significativa redução dos títulos sorológicos, em relação às sorologias colhidas durante o evento coronário agudo, o que ocorreu tanto com a chlamydia (307,5+47,5 versus 650+115,7 p=0,0001) quanto com o mycoplasma (21,5+3,5 versus 36,5+5 p=0,0004). O grupo sem supradesnível ST não teve variação significativa dos níveis sorológicos em seis meses de seguimento (522,6+102,7 versus 576+84,1 p=0,27) para chlamydia e 27,6+5,8 versus 27,6 + 5,8 p >0,99 para mycoplasma. Foi realizada também uma comparação entre os níveis sorológicos de todos os grupos analisados, e observou-se que os grupos com síndrome isquêmica miocárdica instável (com e sem supra ST), tiveram valores sorológicos mais elevados do que os grupos aterosclerose crônica e controle, mas as diferenças não foram significativas. Os valores para chlamydia foram: 650+115,7 (com supra), 576+84 (sem supra), 373,3+65 (aterosclerose crônica) e 343,5+57,2 (controle), p=0,083; e os valores para mycoplasma foram: 36,5+5 (com supra), 27,6+5,8 (sem supra), 23+4,3 (aterosclerose crônica) e 26,7+3,3 (controle), p=0,171. Conclusões: o presente estudo demonstra associação entre títulos de anticorpos anti-Chlamydia pneumoniae e anti-Mycoplasma pneumoniae e a instabilização da placa coronária. Demonstra ainda a normalização dos mesmos títulos em um período de até seis meses, a partir do quadro agudo.
Objective of the study: to test the association of serum titers of anti-Chlamydia pneumoniae and anti-Mycoplasma pneumoniae antibodies and Acute Coronary Syndrome (ACS). The patients were divided into 4 groups: ACS with ST-segment elevation, ACS without ST-segment elevation, chronic asymptomatic atherosclerosis and blood donors without known coronary disease. Serum samples were collected during the acute event and after six months of follow-up. Six months after the acute event, patients with ACS with St-segment elevation showed a significant decrease of serum titers, when compared to the other one. That\'s show the association between anti-Chlamydia pneumoniae and anti-Mycoplasma pneumoniae antibody titers and acute coronary syndrome.
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Kanberg, Josefine. "Development and optimization of methods for detection of Chlamydophila pneumoniae." Thesis, Uppsala University, Department of Medical Biochemistry and Microbiology, 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-9318.

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The purpose of the study was to set up a method for cultivation of C. pneumoniae from infected mouse tissue in Hep2 cells. We also evaluated a new reagent, Chlamatis, which is considered to increase the detection sensitivity of the bacterium with both PCR and cultivation. All 10 serum samples treated with Chlamatis were negative for C. pneumoniae in PCR. The cultivation of tissue was found to work without problems. The yield of the bacteria was highest at the speed of 30 Hz using homogenization with TissueLyser. Mice infected with C. pneumoniae were killed at days 4, 8, 20 and 40. The highest yields of C. pneumoniae were found at days 4 and 8 using PCR with all infected mice. The results obtained with PCR and cultivation confirmed each other to a large extent. For heart tissue, PCR positive mice were found only at days 4 and 8. The number of PCR positive lung samples confirmed to a large extent the number found by cultivation, except for mice killed after 4 days and 40 days where the results differed slightly. This indicated that the optimization of the cultivation method was successful

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Brassard, Pascal. "Identification et caractérisation de protéines immunogènes chez Chlamydophila (Chlamydia) pneumoniae." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/MQ60703.pdf.

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Books on the topic "Chlamydophia pneumoniae"

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Parker, James N., and Philip M. Parker. The official patient's sourcebook on chlamydia pneumonia. Edited by Icon Group International Inc and NetLibrary Inc. Icon Health Publications, 2002.

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John, Camm A., ed. Chronic infection, Chlamydia, and coronary heart disease. Kluwer Academic Publishers, 1999.

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Luigi, Allegra, and Blasi Francesco 1959-, eds. Chlamydia pneumoniae: The lung and the heart. Springer, 1999.

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(Editor), Luigi Allegra, and F. Blasi (Editor), eds. Chlamydia Pneumoniae: The Lung and the Heart. Springer-Verlag Telos, 1999.

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L'age-Stehr, Johanna. Chlamydia pneumoniae and Chronic Diseases: Proceedings of the State-of-the Art Workshop held at the Robert Koch-Institut Berlin on 19 and 20 March 1999. Springer, 2000.

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Chlamydia pneumoniae and Chronic Diseases: Proceedings of the State-of-the-Art Workshop held at the Robert Koch-Institut Berlin on 19 and 20 March 1999. Springer, 2011.

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(Editor), Herman Friedman, Yoshimasa Yamamoto (Editor), and Mauro Bendinelli (Editor), eds. Chlamydia pneumoniae Infection and Disease (Infectious Agents and Pathogenesis). Springer, 2004.

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Langer, Martin, and Edoardo Carretto. Diagnosis and management of atypical pneumonia. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0118.

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‘Atypical pneumonia’ is an old, but successful term, which covers respiratory infections caused by different micro-organisms, causing similar clinical symptoms, and characterized by similar antimicrobial sensitivity/resistance. Out of specific epidemic contexts, Legionella pneumophila, Mycoplasma pneumoniae, and Chlamydophila pneumonia are the micro-organisms involved, L. pneumophila being by far the most frequently involved in severe community-acquired ‘atypical’ pneumonia. It is important to suspect ‘atypical’ pneumonia on the basis of clinical presentation and the correlated extrapulmonary symptoms. Knowledge of symptoms leads to suspicion and, consequently, to timely and adequate empiric treatment and proper diagnostic work-up. Standard microbiological diagnosis is based on urinary antigen test for L. pneumophila and on serology for the other pathogens. A culture should be performed if legionellosis is suspected. NATs techniques could be the future diagnostic tests. Close collaboration with the microbiologist will improve the diagnosis.
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Book chapters on the topic "Chlamydophia pneumoniae"

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Witkiewicz, W., J. Gnus, W. Hauzer, et al. "Chlamydophila pneumoniae (Chlamydia pneumoniae) infection in patients with abdominal aortic aneurysm." In Berliner Gefäßchirurgische Reihe. Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-11719-0_6.

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Choroszy-Krol, Irena, Magdalena Frej-Madrzak, Agnieszka Jama-Kmiecik, Jolanta Sarowska, Grazyna Gosciniak, and Iwona Pirogowicz. "Incidence of Chlamydophila Pneumoniae Infection in Children During 2007–2010." In Neurobiology of Respiration. Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-6627-3_13.

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Choroszy-Krol, Irena, Magdalena Frej-Madrzak, Agnieszka Jama-Kmiecik, et al. "Detection of Chlamydophila Pneumoniae Antigens in Patients with Chronic Cough." In Neurobiology of Respiration. Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-6627-3_7.

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Jama-Kmiecik, A., M. Frej-Madrzak, G. Gosciniak, J. Sarowska, and I. Choroszy-Krol. "Detection of Chlamydophila Pneumoniae and Typical Bacteria in Patients with Chronic Cough." In Advances in Experimental Medicine and Biology. Springer International Publishing, 2015. http://dx.doi.org/10.1007/5584_2015_125.

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Kohlhoff, Stephan A., and Margaret R. Hammerschlag. "Chlamydophila pneumoniae." In Nelson Textbook of Pediatrics. Elsevier, 2011. http://dx.doi.org/10.1016/b978-1-4377-0755-7.00217-7.

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"Chlamydophila pneumoniae." In Lexikon der Infektionskrankheiten des Menschen. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-39026-8_185.

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"Chlamydia pneumoniae, Chlamydophila pneumoniae." In Encyclopedia of Medical Genomics and Proteomics. CRC Press, 2004. http://dx.doi.org/10.1081/e-emgp-120024079.

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Poppert, Sven, Udo Reischl, and Andreas Essig. "Chlamydia pneumoniae,Chlamydophila pneumoniae." In Encyclopedia of Medical Genomics and Proteomics. Informa Healthcare, 2004. http://dx.doi.org/10.3109/9780203997352.048.

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Shah, Samir S. "Chlamydophila (Chlamydia) pneumoniae." In Principles and Practice of Pediatric Infectious Disease. Elsevier, 2008. http://dx.doi.org/10.1016/b978-0-7020-3468-8.50172-3.

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HAMMERSCHLAG, MARGARET R., STEPHAN A. KOHLHOFF, and PETRA M. APFALTER. "Chlamydophila (Chlamydia) pneumoniae." In Mandell, Douglas, and Bennett's Principles and Practice of Infectious Diseases. Elsevier, 2010. http://dx.doi.org/10.1016/b978-0-443-06839-3.00182-x.

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Conference papers on the topic "Chlamydophia pneumoniae"

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Noguchi, Shingo, Kazuhiro Yatera, Toshinori Kawanami, et al. "Chlamydophila pneumoniaewas rarely detected in bronchoalveolar lavage fluid in patients with bacterial pneumonia using molecular methods." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa564.

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Reports on the topic "Chlamydophia pneumoniae"

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McDonough, E. A., C. P. Barrozo, K. L. Russell, and D. Metzgar. A Multiplex PCR for Detection of Mycoplasma pneumoniae, Chlamydophila pneumoniae, Legionella pneumophila, and Bordetella pertussis in Clinical Specimens. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada432554.

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