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Relevant bibliographies by topics / Cholera, microbiology

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Academic literature on the topic 'Cholera, microbiology'

Author: Grafiati

Published: 4 June 2021

Last updated: 27 January 2023

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Journal articles on the topic "Cholera, microbiology"

1

Kaper, J. B., J. G. Morris, and M. M. Levine. "Cholera." Clinical Microbiology Reviews 8, no. 1 (January 1995): 48–86. http://dx.doi.org/10.1128/cmr.8.1.48.

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Despite more than a century of study, cholera still presents challenges and surprises to us. Throughout most of the 20th century, cholera was caused by Vibrio cholerae of the O1 serogroup and the disease was largely confined to Asia and Africa. However, the last decade of the 20th century has witnessed two major developments in the history of this disease. In 1991, a massive outbreak of cholera started in South America, the one continent previously untouched by cholera in this century. In 1992, an apparently new pandemic caused by a previously unknown serogroup of V. cholerae (O139) began in India and Bangladesh. The O139 epidemic has been occurring in populations assumed to be largely immune to V. cholerae O1 and has rapidly spread to many countries including the United States. In this review, we discuss all aspects of cholera, including the clinical microbiology, epidemiology, pathogenesis, and clinical features of the disease. Special attention will be paid to the extraordinary advances that have been made in recent years in unravelling the molecular pathogenesis of this infection and in the development of new generations of vaccines to prevent it.

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Larionova, L. V., R. V. Pisanov, D. I. Simakova, A. N. Narkevich, and I. V. Arkhangel’skaya. "Polimeric Immunoglobulin Diagnosticum for Detection of Cholera Toxin and Assessing the Level of Its Production by Vibrios." Problems of Particularly Dangerous Infections, no. 4 (January 26, 2022): 84–89. http://dx.doi.org/10.21055/0370-1069-2021-4-84-89.

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A marker of the epidemic significance of Vibrio cholerae is their toxigenicity. Therefore, much attention is currently paid to the creation of diagnostic preparations for the detection of cholera toxin and assessment of the level of its production. The volumetric immunosuspension agglomeration reaction, carried out with the help of latex diagnosticums, is an analogue of the indirect hemagglutination reaction, an affordable and technically simple method, since it does not require special equipment and can be used when conducting research in the field. The aim of the study was to design a polymeric immunoglobulin diagnosticum for determining cholera toxin and the level of its production by vibrio strains. Materials and methods. Cholera toxin was obtained from the producer strain Vibrio cholerae Classical 569 B. Rabbit serum to the toxin was obtained according to the method selected by the authors. A polymeric diagnostic immunoglobulin antitoxic drug was obtained through sensitizing immunoglobulins from cholera antitoxic rabbit serum on the surface of polyacrolein microspheres with a size of (1±0.1) μm. Results and discussion. The analytical sensitivity of the developed diagnostic preparation with control cholera toxin is 100 ng/ml. It detects cholera toxin in toxigenic strains of Vibrio cholerae in a titer of 1:16 – 1:512, gives negative results with non-toxigenic strains of V. cholerae O1, V. Cholerae nonO1/nonO139, with samples of heterologous cultures, LPS preparations, liquid nutrient medium used for the cultivation of V. cholerae. Thus, a polymeric immunoglobulin diagnosticum has been constructed to detect and quantify the production of cholera toxin by vibrio strains, and its analytical sensitivity and specificity have been established.

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Kitaoka, Maya, Sarah T. Miyata, Daniel Unterweger, and Stefan Pukatzki. "Antibiotic resistance mechanisms of Vibrio cholerae." Journal of Medical Microbiology 60, no. 4 (April 1, 2011): 397–407. http://dx.doi.org/10.1099/jmm.0.023051-0.

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As the causative agent of cholera, the bacterium Vibrio cholerae represents an enormous public health burden, especially in developing countries around the world. Cholera is a self-limiting illness; however, antibiotics are commonly administered as part of the treatment regimen. Here we review the initial identification and subsequent evolution of antibiotic-resistant strains of V. cholerae. Antibiotic resistance mechanisms, including efflux pumps, spontaneous chromosomal mutation, conjugative plasmids, SXT elements and integrons, are also discussed. Numerous multidrug-resistant strains of V. cholerae have been isolated from both clinical and environmental settings, indicating that antibiotic use has to be restricted and alternative methods for treating cholera have to be implemented.

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Chattaway, Marie Anne, Abdul Kamara, Fay Rhodes, Konneh Kaffeta, Amara Jambai, Wondimagegnehu Alemu, Mohammed Sirajul Islam, et al. "Establishing an enteric bacteria reference laboratory in Sierra Leone." Journal of Infection in Developing Countries 8, no. 07 (June 9, 2014): 933–41. http://dx.doi.org/10.3855/jidc.5074.

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In 2012, Sierra Leone experienced its worst cholera outbreak in over 15 years affecting 12 of the country’s 13 districts. With limited diagnostic capability, particularly in bacterial culture, the cholera outbreak was initially confirmed by microbiological testing of clinical specimens outside of Sierra Leone. During 2012 – 2013, in direct response to the lack of diagnostic microbiology facilities, and to assist in investigating and monitoring the cholera outbreak, diagnostic and reference services were established in Sierra Leone at the Central Public Health Reference Laboratory focusing specifically on isolating and identifying Vibrio cholerae and other enteric bacterial pathogens. Sierra Leone is now capable of confirming cholera cases by reference laboratory testing.

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LaRocque, Regina C., Bryan Krastins, Jason B. Harris, Lauren M. Lebrun, Kenneth C. Parker, Michael Chase, Edward T. Ryan, Firdausi Qadri, David Sarracino, and Stephen B. Calderwood. "Proteomic Analysis of Vibrio cholerae in Human Stool." Infection and Immunity 76, no. 9 (June 30, 2008): 4145–51. http://dx.doi.org/10.1128/iai.00585-08.

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ABSTRACT An effective vaccine for Vibrio cholerae is not yet available for use in the developing world, where the burden of cholera disease is highest. Characterizing the proteins that are expressed by V. cholerae in the human host environment may provide insight into the pathogenesis of cholera and assist with the development of an improved vaccine. We analyzed the V. cholerae proteins present in the stools of 32 patients with clinical cholera. The V. cholerae outer membrane porin, OmpU, was identified in all of the human stool samples, and many V. cholerae proteins were repeatedly identified in separate patient samples. The majority of V. cholerae proteins identified in human stool are involved in protein synthesis and energy metabolism. A number of proteins involved in the pathogenesis of cholera, including the A and B subunits of cholera toxin and the toxin-coregulated pilus, were identified in human stool. In a subset of stool specimens, we also assessed which in vivo expressed V. cholerae proteins were recognized uniquely by convalescent-phase as opposed to acute-phase serum from cholera patients. We identified a number of these in vivo expressed proteins as immunogenic during human infection. To our knowledge, this is the first characterization of the proteome of a pathogenic bacteria recovered from a natural host.

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Nalin, D. R. "Cholera or Choleric?" Clinical Infectious Diseases 46, no. 1 (January 1, 2008): 150. http://dx.doi.org/10.1086/524088.

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Khan, Ashraful Islam, Md Mahbubur Rashid, Md Taufiqul Islam, Mokibul Hassan Afrad, M. Salimuzzaman, Sonia Tara Hegde, Md Mazharul I. Zion, et al. "Epidemiology of Cholera in Bangladesh: Findings From Nationwide Hospital-based Surveillance, 2014–2018." Clinical Infectious Diseases 71, no. 7 (December 31, 2019): 1635–42. http://dx.doi.org/10.1093/cid/ciz1075.

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Abstract Background Despite advances in prevention, detection, and treatment, cholera remains a major public health problem in Bangladesh and little is known about cholera outside of limited historical sentinel surveillance sites. In Bangladesh, a comprehensive national cholera control plan is essential, although national data are needed to better understand the magnitude and geographic distribution of cholera. Methods We conducted systematic hospital-based cholera surveillance among diarrhea patients in 22 sites throughout Bangladesh from 2014 to 2018. Stool specimens were collected and tested for Vibrio cholerae by microbiological culture. Participants’ socioeconomic status and clinical, sanitation, and food history were recorded. We used generalized estimating equations to identify the factors associated with cholera among diarrhea patients. Results Among 26 221 diarrhea patients enrolled, 6.2% (n = 1604) cases were V. cholerae O1. The proportion of diarrhea patients positive for cholera in children <5 years was 2.1% and in patients ≥5 years was 9.5%. The proportion of cholera in Dhaka and Chittagong Division was consistently high. We observed biannual seasonal peaks (pre- and postmonsoon) for cholera across the country, with higher cholera positivity during the postmonsoon in western regions and during the pre–monsoon season in eastern regions. Cholera risk increased with age, occupation, and recent history of diarrhea among household members. Conclusions Cholera occurs throughout a large part of Bangladesh. Cholera-prone areas should be prioritized to control the disease by implementation of targeted interventions. These findings can help strengthen the cholera-control program and serve as the basis for future studies for tracking the impact of cholera-control interventions in Bangladesh.

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Agustanty, Adelia, and Andre Budi. "POLA RESISTENCY OF VIBRIO CHOLERAE BACTERIA TO THE ANTIBIOTIC CIPROFLOXACIN AND TETRACYCLINE." Journal Health & Science : Gorontalo Journal Health and Science Community 5, no. 3 (April 8, 2022): 73–78. http://dx.doi.org/10.35971/gojhes.v5i3.13611.

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Abstrak Diare merupakan kegiatan defekasi (buang air besar) yang biasanya berbentuk 1/2 padat atau cenderung lebih cair yang berlangsung lebih dari tiga kali sehari atau dalam waktu yang singkat, vibrio cholera adalah salah satu penyebabnya, bakteri ini merupakan bakteri gram negatif yang berbentuk koma galibnya masa inkubasi bakteri ini adalah 12-72 jam. Bakteri vibrio cholerae menyulut penyakit bakteri. Jenis penelitian ini adalah penelitian laboratorium eksperimental dengan menggunakan arsip sampel bakteri vibrio cholerae dan cakram antibiotik ciprofloxacin. Tujuan penelitian untuk mengetahui pola resistensi antibiotik ciprofloxacin terhadap bakteri vibrio cholerae. Populasi yang digunakan adalah isolate murni bakteri Vibrio cholera dan sampel yang digunakan adalah sediaan cakram dari antibiotik Ciprofloxacin dan Tetracycline. Nilai rata-rata (mm) selama 24 jam ciprofloxcacin : 37.425 , tetracycline : 24,175 Nilai rata-rata (mm) selam 48 jam ciprofloxacin : 29,875 tetracycline : 22,95 Berdasarkan hasil data dan gambar penelitian dapat di simpulkan bahwa diameter zona hambat atau zona bening dari biakan bakteri vibrio cholera yang terdapat dalam cawan petri dengan media MHA serta cakram antibiotik ciprofloxacin dan tetracycline menunjukkan bahwa bakteri uji masih sensitive terhadap kedua antibiotik uji yang dimana nilai rata-rata nya adalah 29,875 dan 22,95 mm dimana menurut standart CLSI (Clinical Laboratory Standards Institute), diameter zona hambat bakteri ≥ 17 mm, kategori intermediet apabila diameter zona hambat bakteri 14-16 mm, dan kategori resisten apabila diameter zona hambat bakteri yaitu ≤ 13mm. Kesimpulan bahwa biakan bakteri vibrio choleramasih sensitive terhadap kedua antibiotic ciprofloxacin dan tetracycline. Kata Kunci : Ciprofloxacin; Cholera; Diare; Tetracycline; Vibrio Cholerae. Abstract Diarrhea is a defecation activity (defecation) which is usually in the form of 1/2 solid or tends to be more liquid (watery) which lasts more than three times a day or in a short time, Vibrio cholera is one of the causes, this bacterium is a gram-negative bacterium that causes diarrhea. In the form of a comma, the incubation period for this bacterium is 12-72 hours. Vibrio cholerae bacteria cause bacterial disease. This type of research is an experimental laboratory study using archived samples of Vibrio cholerae bacteria and ciprofloxacin antibiotic discs. This study aims to determine the pattern of resistance to ciprofloxacin antibiotics against Vibrio cholerae bacteria. The population that will be used is pure isolate of Vibrio cholera bacteria and the sample used is disc preparation of the antibiotics Ciprofloxacin and Tetracycline. Average value (mm) for 24 hours ciprofloxcacin: 37.425, tetracycline: 24.175 Average value (mm) for 48 hours ciprofloxacin : 29.875 tetracycline : 22.95 Based on the results of the data and research images it can be concluded that the diameter of the inhibition zone or clear zone of the Vibrio cholera bacteria culture contained in petri dishes with MHA media and ciprofloxacin and tetracycline antibiotic discs showed that the test bacteria were still sensitive to the two test antibiotics where the average value was 29.875 and 22.95 mm where according to the CLSI (Clinical Laboratory Standards Institute) standard, the diameter of the bacterial inhibition zone was 17 mm, the intermediate category if the diameter of the bacterial inhibition zone was 14-16 mm, and the category of intermediate was 14-16 mm. resistant if the diameter of the bacterial inhibition zone is 13 mm. The conclusion is that the vibrio cholera bacteria culture is still sensitive to both ciprofloxacin and tetracycline antibiotics. Keywords: Ciprofloxacin ; Cholerae; Diarrhea ; Tetracycline ; Vibrio Cholerae.

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Kumar, P., M. Jain, A. K. Goel, S. Bhadauria, S. K. Sharma, D. V. Kamboj, L. Singh, T. Ramamurthy, and G. B. Nair. "A large cholera outbreak due to a new cholera toxin variant of the Vibrio cholerae O1 El Tor biotype in Orissa, Eastern India." Journal of Medical Microbiology 58, no. 2 (February 1, 2009): 234–38. http://dx.doi.org/10.1099/jmm.0.002089-0.

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A total of 32 Vibrio cholerae isolates were collected during a recent large cholera outbreak in Eastern India. Biochemical and serological studies revealed that all of the isolates belonged to serogroup O1, biotype El Tor, serotype Ogawa. Two multiplex PCR assays confirmed the presence of various toxigenic and pathogenic genes – ace, ctxAB, hlyA, ompU, ompW, rfbO1, rtx, tcp, toxR and zot – in all of the isolates. Sequencing of the ctxB gene from the isolates revealed a novel mutation in the gene. Sequencing also confirmed the presence of altered cholera toxin B of the classical biotype in all of the El Tor isolates, suggesting infection of isolates by classical CTXΦ. The molecular diversity of V. cholerae isolates studied by enterobacterial repetitive intergenic consensus sequence PCR, BOX-PCR and randomly amplified polymorphic DNA analysis uniformly showed the clonal relationship among the outbreak V. cholerae O1 isolates. The results of this study suggest that cholera-causing V. cholerae strains are constantly evolving in epidemic areas, highlighting the potential of the emergence of more virulent strains.

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Saidi, Suleiman M., Nityananda Chowdhury, Sharda P. Awasthi, Masahiro Asakura, Atsushi Hinenoya, Yoshio Iijima, and Shinji Yamasaki. "Prevalence of Vibrio cholerae O1 El Tor variant in a cholera-endemic zone of Kenya." Journal of Medical Microbiology 63, no. 3 (March 1, 2014): 415–20. http://dx.doi.org/10.1099/jmm.0.068999-0.

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Since 2007, Kenya has experienced an increase in cholera outbreaks characterized by a high fatality rate. In this study, we characterized 81 Vibrio cholerae isolates from diarrhoeal stool samples in Nyanza, a cholera-endemic lake region of Kenya, for virulence properties, clonality and antibiotic susceptibility. Eighty of these isolates were V. cholerae O1 El Tor variants carrying the classical ctxB gene sequence, while one isolate was V. cholerae non-O1/O139. All of the El Tor variants were of clonal origin, as revealed by PFGE, and were susceptible to ampicillin, tetracycline, ciprofloxacin, fosfomycin, kanamycin and norfloxacin. However, the isolates showed resistance to sulfamethoxazole/trimethoprim and streptomycin, and intermediate resistance to nalidixic acid, chloramphenicol and imipenem. The non-O1/O139 isolate carried the cholix toxin II gene (chxA II) and was susceptible to all antimicrobials tested except ampicillin. We propose that an El Tor variant clone caused the Nyanza cholera outbreak of 2007–2008.

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Dissertations / Theses on the topic "Cholera, microbiology"

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Amediavor, Rita Laryea. "The Persisting Threats Of Cholera: A Cyclical Public Health Problem In Ghana." Wright State University / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=wright1598992794308852.

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Saul-McBeth, Jessica. "Characterization of SipA, A Protein Important for Stress Responses in Vibrio cholerae." University of Toledo Health Science Campus / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=mco1544540466901883.

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Jahan, Nasrin. "Structural studies of Vibrio cholerae quorum sensing proteins." Thesis, University of St Andrews, 2011. http://hdl.handle.net/10023/2565.

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The spread of cholera is always associated with contaminated food or water and this is the reason this disease has been endemic in developing countries for centuries due to their lack of proper sanitation facilities and poor or no infrastructure for sewage systems. Cholera can spread quickly and sporadically after any natural disaster that destroys the sewage system or safe drinking water supply of both developed and undeveloped countries. In Southeast Asia in December 2004 and in Pakistan and Haiti 2010, cholera outbreaks followed the natural disasters; with most of the cholera victims being children. Although it is known that the best way to prevent cholera outbreak is the development of the infrastructure, provision of a safe drinking water supply and proper sanitation, this is a very long-term process, and most of the developing countries cannot afford such improvements. These situations can be made worse by natural disasters. Therefore there is a pressing need for the development of a cholera vaccine and there have been numerous research projects working towards this end for several decades. A few of them have been successful to date but because of the severe side effects and narrow range of protection, more effective and wider range vaccine development is still ongoing. In this study, crystallographic and enzymatic studies have been carried out on several novel proteins involved in the control of the production of the factors required for quorum sensing. Quorum sensing is a process in which bacterial cells communicate among themselves by the synthesis, release and detection of small chemical compounds called autoinducers. In this work, structural analysis was carried out on proteins involved in the synthesis and detection of the major autoinducer of Vibrio cholerae, named CAI-1. The crystal structure of CqsA involved in CAI-1 synthesis has been successfully solved and its enzymatic properties have been characterized. The structure of one domain of the cytoplasmic region of the CAI-1 receptor CqsS was also elucidated, and other domains were expressed. The crystal structure of another enzyme (VCA0859, an aldo-keto reductase) thought to have been involved in the synthesis of CAI-1 was also determined. Another protein named VCA0939 was also studied, due to its importance in biofilm development, and its ability to control quorum-sensing in an alternative pathway in the mutated version of pathogenic strains of V. cholerae that were responsible for the seventh cholera pandemic. The aim of this project was to understand the three dimensional structure of some proteins that are involved in quorum sensing and control of the expression of virulence genes for the pathogenesis of V. cholerae. Understanding the three dimensional structure of the proteins and the mode of autoinducer binding to its specific receptor could be highly valuable in the development of a chemical compound that could lead to the discovery of a novel drug with the ability to target cross species specification.

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Harper, Marina. "Virulence determinants of Pasteurella multocida." Monash University, Dept. of Microbiology, 2003. http://arrow.monash.edu.au/hdl/1959.1/9341.

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Lo, Miranda. "Characterisation of in vivo expressed proteins of Pasteurella multocida." Monash University, Dept. of Microbiology, 2003. http://arrow.monash.edu.au/hdl/1959.1/9429.

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Villeneuve, Sylvain. "Caractérisation immunochimique des déterminants antigéniques du lipopolysaccharide de Vibrio cholerae O1 en vue de la préparation d'un vaccin anticholérique chimiquement défini." Paris 5, 1999. http://www.theses.fr/1999PA05N124.

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Resch, Craig. "Biochemistry and physiology of NhaP-type antiporters in Vibrio cholerae." Elsevier, 2010. http://hdl.handle.net/1993/31098.

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Antiporters that exchange alkali cations (Na+ or K+) for protons play an important role in the physiology of all known bacterial species. They are involved in regulating intracellular pH and maintaining cellular volume as well as the formation of a chemical Na+ gradient across the membrane, which is important to many bacteria as an energy source for processes such as accumulation of substrates, ATP synthesis, and flagellar rotation. Another important role of cation/proton antiporters is homeostasis of intracellular cation content. The situation of a thermodynamic equilibrium of Na+ or K+ on the membrane would result in toxic intracellular levels of these cations, so bacteria have cation/proton antiporters, which expel excess of alkali cations at the expense of the proton motive force. Of many antiporters described to date, the NhaP-type family is one of the most interesting groups. Its members collectively demonstrate a great diversity of their features. There are three antiporters of NhaP type encoded in the genome of the dangerous human pathogen, Vibrio cholerae. Phenotype analysis of engineered chromosomal VcnhaP1, VcnhaP2 and VcnhaP3 deletion mutants has proven the three NhaP paralogues to be essential for V. cholerae growth at low pH in the presence of high or low concentrations of K+. Genes encoding Vc-NhaP1-3 were cloned and antiporters expressed in their functional form in an antiporter-less strains of Escherichia coli. Although initially annotated as Na+/H+ antiporters, when assayed in everted membrane vesicles, all three isoforms of Vc-NhaP were shown to be K+/H+ antiporters with only limited ability to Na+/H+ antiport. None of three proteins was able to mediate Li+/H+ exchange. Overall, the three antiporters differed in their biochemical profiles, predicted topology, and their phenotypic manifestations.
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Hillary, Janet Barbara. "Investigation into the molecular mechanism of toxin secretion from Vibrio cholerae." Thesis, University of Bristol, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.263928.

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Schroeder, Irmagard Cherè. "Molecular and functional characterization of the melanin biosynthetic genes from Vibrio cholerae 569B." Doctoral thesis, University of Cape Town, 1998. http://hdl.handle.net/11427/16110.

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Bibliography: p. 155-177.
V. cholerae 569B is a bacterium infamous for its role as the causative agent of the diarrhoeal disease cholera. Although the bacterium occurs naturally in brackish waters and estuaries, cholera outbreaks are closely linked to specific environmental conditions. For example, most outbreaks occur during the summer months when the bacterium experiences an increase in water temperature, play a role in activating virulence in V. cholerae 569B, the exact mechanism remains to be elucidated. Previously it was observed that when V. cholerae is exposed to elevated temperature and salinity, the bacterium initiates the synthesis of a brown-black pigment known as melanin. The function of the pigment and the genes involved in its synthesis was unknown. We therefore set out to determine the function of pigmentation in V. cholerae 569B, since pigmentation could significantly enhance the survival of the bacterium during adverse conditions and therefore aid in the persistence of the organism in the environment.

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Pérez-Soto, Nicolás. "Taming Vibrio cholerae with cationic polymers : engineering bacterial physiology by interfering with communication and virulence." Thesis, University of Birmingham, 2018. http://etheses.bham.ac.uk//id/eprint/8857/.

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The Gram-negative Vibrio cholerae is native to aquatic environments and an important human pathogen causing cholera disease. The induction of virulence in this bacterium is subjected to a wide variety of stimuli including environmental cues and quorum sensing. In this study, non-bactericidal cationic polymers were designed to capture Vibrio cholerae into clusters resulting in physiological changes. Poly(N-(3-aminopropyl) methacrylamide) (P1), poly(N-[3-dimethylamino)propyl] methacrylamide) (P2) or poly(acryloyl hydrazide) imidazole (P3) were synthesised via free radical polymerisation displaying amine groups that cluster cells mediated by electrostatic interactions. This binding resulted in a forced transition from planktonic to a sessile lifestyle. The clustering is accompanied by reduced motility, increased biofilm synthesis and repression of virulence since the expression cholera toxin was down-regulated. This avirulent phenotype was defective to colonise intestinal epithelial cells and the zebrafish digestive tract. Since the cell density increases locally as a result of the clustering, a quorum-sensing-controlled phenotype was observed as the lux operon was actively expressed. Overall, the bacterial physiology was modulated without genetic modification preventing virulence as the pathogen adapt its lifestyle during clustered lifestyle. This thesis highlights the use of polymeric materials as a mean to control pathogens beyond the use antibiotics.

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Books on the topic "Cholera, microbiology"

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Kaye, Wachsmuth, Blake Paul A, and Olsvik Ørjan, eds. Vibrio cholerae and cholera: Molecular to global perspectives. Washington, D.C: ASM Press, 1994.

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K, Bhattacharya S., and SpringerLink (Online service), eds. Epidemiological and Molecular Aspects on Cholera. New York, NY: Springer Science+Business Media, LLC, 2011.

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Robert, Koch. Essays of Robert Koch. New York: Greenwood Press, 1987.

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Yellow fever, black goddess: The coevolution of people and plagues. Reading, MA: Addison-Wesley Pub., 1996.

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Characterization of the maltose regulon of Vibrio cholerae: Involvement of maltose in production of outer membrane proteins and secretion of virulence factors. Uppsala: Swedish University of Agricultural Sciences, Dept. of Molecular Genetics, Uppsala Genetic Center, 1993.

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Cholera Toxins. Springer, 2009.

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Wachsmuth, I. Kaye, Paul A. Blake, and Orjan Olsvik. Vibrio Cholerae and Cholera: Molecular to Global Perspectives. Wiley & Sons, Limited, John, 2014.

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Wachsmuth, I. Kaye, and Paul A. Blake. Vibrio Cholerae and Cholera: Molecular to Global Perspectives. ASM Press, 1994.

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Bhattacharya, S. K., and T. Ramamurthy. Epidemiological and Molecular Aspects on Cholera. Springer, 2012.

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(Editor), S. Kuwahara, and N. F. Pierce (Editor), eds. Advances in Research on Cholera and Related Diarrheas 2 (New Perspectives in Clinical Microbiology). Springer, 1985.

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Book chapters on the topic "Cholera, microbiology"

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Hurst, Christon J. "Briefly Summarizing Our Understanding of Vibrio cholerae and the Disease Cholera." In Advances in Environmental Microbiology, 173–84. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-16775-2_7.

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Holmgren, J., J. Clemens, D. A. Sack, J. Sanchez, and A. M. Svennerholm. "Oral Immunization Against Cholera." In Current Topics in Microbiology and Immunology, 197–204. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74529-4_21.

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Mekalanos, J. J. "Cholera Toxin: Genetic Analysis, Regulation, and Role in Pathogenesis." In Current Topics in Microbiology and Immunology, 97–118. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70586-1_6.

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Elson, C. O. "Cholera Toxin and its Subunits as Potential Oral Adjuvants." In Current Topics in Microbiology and Immunology, 29–33. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74529-4_3.

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Serventi, I. M., J. Moss, and M. Vaughan. "Enhancement of Cholera Toxin-Catalyzed ADP-Ribosylation by Guanine Nucleotide-Binding Proteins." In Current Topics in Microbiology and Immunology, 43–67. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76966-5_3.

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Karaolis, David K. R., and Edgar C. Boedeker. "Enteric Pathogens: Population Genetics and Pathogenesis of Escherichia coli and Vibrio cholerae Infections." In Gastrointestinal Microbiology, 622–57. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4757-0322-1_16.

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Ritchie, Jennifer M., and Matthew K. Waldor. "Vibrio cholerae Interactions with the Gastrointestinal Tract: Lessons from Animal Studies." In Current Topics in Microbiology and Immunology, 37–59. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-01846-6_2.

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Gustafsson, B., and T. Holme. "Identification and Serotyping of Vibrio cholerae 0:1 by Monoclonal Antibody Based Enzyme-Linked Immunosorbent Assays." In Rapid Methods and Automation in Microbiology and Immunology, 135–40. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-69943-6_18.

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Primrose, Sandy B. "A Surprising Pathogen: Vibrio cholerae." In Microbiology of Infectious Disease, 53–59. Oxford University Press, 2022. http://dx.doi.org/10.1093/oso/9780192863843.003.0007.

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Abstract:

The natural habitat of Vibrio cholerae is warm, nutrient-rich water, particularly estuarine water. Vibrio cholerae produces a chitinase that degrades the exoskeletons of insects and shellfish. The presence of chitin induces competence for genetic transformation that will permit horizontal gene transfer. Most strains of Vibrio cholerae do not cause disease but those that do acquired their virulence by horizontal gene transfer. Pathogenic strains mostly belong to the O1 serotype of which there are two biotypes: classical and El Tor. Disease-causing strains have at least two pathogenicity islands (VPI-1 and VPI-2) and carry the prophage CTXΦ‎. The prophage genome encodes cholera toxin. CTXΦ‎ resemble coliphage M13 and the cholera toxin is similar to that found in enterotoxigenic Escherichia coli. Therefore, pathogenic strains of Vibrio cholerae may have acquired some of their virulence characteristics from Escherichia coli by horizontal gene transfer. The first six major epidemics of cholera were caused by the classical biotype of Vibrio cholerae. The seventh epidemic was caused by the El Tor biotype. The original El Tor isolates were of low virulence compared with the 7PET strain and the genetic changes that led to the creation of the 7PET strain have been identified.

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Meehan, C. D., and H. Markel. "Cholera, Historical." In Encyclopedia of Microbiology, 24–29. Elsevier, 2009. http://dx.doi.org/10.1016/b978-012373944-5.00301-1.

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Conference papers on the topic "Cholera, microbiology"

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Anas, Abdulaziz, Kiran Krishna, Sreelakshmi PK, Syamkumar V, Jasmin C, Beena James, and Sobha kurien. "Multiple drug-resistant <em>Vibrio cholerae </em>responsible for cholera outbreak among migrant domestic workers in Kerala, South India." In 1st International Electronic Conference on Microbiology. Basel, Switzerland: MDPI, 2020. http://dx.doi.org/10.3390/ecm2020-07103.

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