Relevant bibliographies by topics / Cholera Vibrio cholerae

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Academic literature on the topic 'Cholera Vibrio cholerae'

Author: Grafiati
Published: 4 June 2021
Last updated: 10 February 2022

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Journal articles on the topic "Cholera Vibrio cholerae"

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Daniszewski, Piotr. "Vibrio cholerae - As Biological Weapons." International Letters of Social and Humanistic Sciences 9 (September 2013): 65–73. http://dx.doi.org/10.18052/www.scipress.com/ilshs.9.65.

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Terrorism is defined as use of unlawful violence or threat of unlawful violence to indulge fear; intended to coerce or to intimidate governments or societies in the pursuit of goals that are generally political, social or religious. Bioterrorism is terrorism by intentional release or dissemination of biological agents, mainly bacteria or viruses. Use of biological weapons is attractive from the terrorists’ point of view because of low production costs, major range and easiness of transmission. The first mention of the use of primitive biological weapons date back to the 6th century. Use of plague-infested corpses as offensive means in the 14th century caused a spread of bubonic plague through the whole Europe. The biggest development of biological weapons took place in the interwar period and in the cold war era. Biological weapon trails and research were conducted by super powers such as USSR, UK, USA and Japan. At the beginning of the 20th century a new form of bioterrorism occurred, which put humanity in the face of a terrifying threat. Cholera is a deadly disease that has caused a worldwide phenomenon throughout history. Its imperative weapon, the Vibrio cholerae bacterium, has allowed cholera to seize control and wipe out a huge percentage of the human population. V. cholerae’s toxins are the primary causes of cholera’s lethal symptoms. The bacterium contains toxins that help it accomplish its job of invading the human system and defeating the body’s powerful immune system. With its sibling bacterium Escherichia coli, V. cholerae has become one of the most dominant pathogens in the known world. V. cholerae’s strategies in causing the infamous deadly diarrhea have been widely studied, from the irritation of the intestinal epithelium to the stimulation of capillary leakage, as well as the internal effects of the disease such as the Peyer’s patches on the intestinal walls. Overall, the Vibrio cholera bacterium has made cholera a tough disease to overcome, and because of its deadly virulence factors, cholera has become one of the most frightening diseases a human body could ever encounter. Vibrio cholerae is a Gram-negative, comma-shaped bacterium. Some strains of V. cholerae cause the disease cholera. V. cholerae is facultatively anaerobic and has a flagellum at one cell pole. V. cholerae was first isolated as the cause of cholera by Italian anatomist Filippo Pacini in 1854, but his discovery was not widely known until Robert Koch, working independently 30 years later, publicized the knowledge and the means of fighting the disease. V. cholerae pathogenicity genes code for proteins directly or indirectly involved in the virulence of the bacteria. During infection, V. cholerae secretes cholera toxin, a protein that causes profuse, watery diarrhea. Colonization of the small intestine also requires the toxin coregulated pilus (TCP), a thin, flexible, filamentous appendage on the surface of bacterial cells.
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Savelieva, I. V., S. N. Tikhonov, V. N. Saveliev, D. A. Kovalev, S. V. Pisarenko, E. S. Kotenev, B. V. Babenyshev, L. S. Zinich, N. N. Pidchenko, and A. N. Kulichenko. "RETROSPECTIVE ANALYSIS OF BIOLOGICAL AND MOLECULAR-GENETIC PROPERTIES OF STRAINS - CAUSATIVE AGENTS OF CHOLERA - ISOLATED IN UKRAINE IN 1994 - 2011." Journal of microbiology epidemiology immunobiology, no. 1 (February 28, 2017): 49–55. http://dx.doi.org/10.36233/0372-9311-2017-1-49-55.

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Aim. Retrospective analysis of biological and molecular-genetic properties of strains - causative agents of cholera - isolated in the period of epidemics in Ukraine in 1994 - 2011. Materials and methods. Phenotypic and molecular-genetic properties of 5 strains of cholera vibrios, biovar El Tor isolated from cholera patients and 4 strains from the environmental samples were studied using traditional bacteriological and genetic methods. Detection of DNA for toxigenicity genes and genes characteristic for El Tor and classic biovar were carried out by PCR method using reagent kits «AmpliSens- Vibrio cholerae FRT» and «.Vibrio cholerae ctxB-rstR-rstC genes, REF» (an experimental test system). Sequencing of genomes of 4 strains of causative agents of cholera was carried out in genetic analyzer Ion Torrent Personal Genome Machine. Results. Strains of cholera vibrios identified in Ukraine in 1994 and 2011 such as a typical toxigenic biovar El Tor (V cholerae 01, El Tor, Ogawa, Hly-, ctxA+, tcpA+) contain genes of the classic cholera vibrio in their genome and are genetically altered (hybrid) variants of cholera vibrio biovar El Tor producing enterotoxin CT1 and having increased virulence, that was clinically manifested in predominance of severe forms of cholera in Mariupol of Donetsk region in 2011. Genome sequences of the 4 studied strains were deposited into the international database DDBJ/EMBL/GenBank. Conclusion. The studied isolates were established to belong to a clade of strains associated with cholera outbreaks in Haiti and Asian continent, from where genetically altered strains of cholera vibrios biovar El Tor were introduced to Haiti in 2010, based on results of comparison of genomic sequences of the studied strains with genomes of V. cholera strains from the international database GenBank.
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Savelieva, I. V., A. N. Kulichenko, V. N. Saveliev, D. A. Kovalev, O. V. Vasilieva, A. M. Zhirov, E. I. Eremenko, et al. "MLVA-TYPING OF CLINICAL STAMPS OF GENETICALLY CHANGED VIBRIO CHOLERAE BIOTYPE EL TOR INSULATED IN RUSSIA AND UKRAINE IN THE PERIOD OF SEVENTH PANDEMIC CHOLERA." Journal of microbiology epidemiology immunobiology, no. 6 (December 28, 2018): 37–43. http://dx.doi.org/10.36233/0372-9311-2018-6-37-43.

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Aim. Conduct in a comparative aspect MLVA-typing of genetically altered cholera vibrio biovar El Tor, isolated from patients during the epidemic (1994) and outbreaks (1993, 1998) in Dagestan with isolates in Mariupol (Ukraine) in 1994-2011 in Moscow (2010, 2012), India (1964, 2006, 2007), Bangladesh 1991, 1994, 2001, 2004) and to establish Phylogenetic connections between strains of cholera vibrios isolated in different years in these territories, to ascertain the source of their drift. Materials and methods. MLVA-tyP-ing was carried out in PCR at 5 variable loci of 35 clinical strains of genetically modified Vibrio cholerae byotyPe El Tor. The obtained amPlicon was studied in the system of automatic caPillary electroPhoresis ExPerion («Bio Rad Laboratories», USA). For Phylogenetic analysis, along with MLVA-genotyPes, 35 strains of Vibrio cholerae from the Institute's collection used Published genotyPes of strains isolated in India, Bangladesh, Haiti. Results. The investigated strains of cholera vibrio are referred to 21 MLVA-tyPes, divided into 2 main clades and 1 seParate branch with clonal clusters and subclusters, each of which contains closely related strains of cholera vibrio genovariants having a different degree of Phylogenetic relationshiP - full or Partial identity of allelic Profiles of five variable loci. The sources of drift of genetically modified Vibrio cholerae byotyPe El Tor to Russia and Ukraine from disadvantaged cholera of India, Bangladesh, Azerbaijan and the countries of the Middle East have been established. Conclusion. The obtained data testify to the PolymorPhism of MLVA-tyPes of genetically altered strains of cholera vibrio of the biologist El Tor, evolved in different years and caused ePidemics or outbreaks of cholera in different territories during different time Periods of the course of the seventh cholera Pandemic, and also suggest the Polyclonal origin of the Vibrio cholerae biovar El Tor and the source of their drift to the territory of the Russian Federation and Ukraine.
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Vanden Broeck, Davy, Caroline Horvath, and Marc J. S. De Wolf. "Vibrio cholerae: Cholera toxin." International Journal of Biochemistry & Cell Biology 39, no. 10 (2007): 1771–75. http://dx.doi.org/10.1016/j.biocel.2007.07.005.

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Lomov, Yu M., N. R. Telesmanich, I. T. Andrusenko, E. A. Moskvitina, and O. A. Areshina. "PROPERTIES OF VIBRIO CHOLERAE STRAINS ISOLATED IN ASIA AND THEIR RELATIONSHIP TO THE STRAINS CIRCULATING IN OTHER CONTINENTS DURING THE SEVENTH CHOLERA PANDEMIC." Epidemiology and Infectious Diseases 17, no. 1 (February 15, 2012): 39–45. http://dx.doi.org/10.17816/eid40654.

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The review deals with the properties of Vibrio cholerae (classical, El Tor, 0139, non-01/non-0139 strains) circulating worldwide during the seventh cholera pandemic. Particular attention is given to the variability in the cholera pathogen: the replacement of classical Vibrio cholerae by the El Tor biotype and subsequently the emergence of serogroup Vibrio cholerae 0139 and genetically altered El Tor Vibrio cholerae; the causes giving rise to these changes and spread of Vibrio cholera in the countries of the Asian continent. A large genetic variability found in Asian strains suggests that there is a real possibility of the emergence of new clones with new properties, including those with an epidemic potential. The Vibrio cholerae strains, that periodically appear in Asia and have an epidemic potential and new properties, spread over all continents, by causing cholera infection. The cholera pathogen adapts to new existence conditions in some cases, by altering some properties and, by having been rooted in a certain area, causes mainly sporadic cases of the disease. These Vibrio cholerae strains, unlike the Asian strains (the pathogens of the seventh pandemic), may be virulent, by preserving the virulence genes in the genome; however, they are, in most cases, non-endemic and unable to spread widely.
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Maurice Bilung, Lesley, Mintra Prommani Etriam, Ahmad Syatir Tahar, Teng Sing Tung, and Kasing Apun. "Detection of Cholera Toxin-Producing Vibrio cholerae in Phytoplankton from Santubong and Samariang Estuaries." Borneo Journal of Resource Science and Technology 9, no. 1 (June 30, 2019): 36–43. http://dx.doi.org/10.33736/bjrst.1584.2019.

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Many cholera outbreaks worldwide were associated with cholera toxin-producing Vibrio cholerae. The bacteria are ubiquitous in aquatic environment, whilst phytoplankton is associated with adaptation of the Vibrio species. This study was conducted to detect cholera toxin-producing Vibrio cholerae, and to determine association of the selected water physicochemical parameters with the number of the bacteria. In this study, a total of ten phytoplankton samples were collected at Santubong and Samariang Estuaries in Kuching, Sarawak. Water physicochemical parameters (temperature, pH and salinity) were recorded. Vibrio bacteria were cultivated on thiosulfate citrate bile-salts sucrose selective agar and analysed for cholera toxin-producing Vibrio cholerae using polymerase chain reaction by targeting ctxA gene that encodes for virulence cholera enterotoxin subunit A. The result revealed that a range of 1.0 × 107 – 8.0 × 107 CFU/ml of yellow colonies growing on the thiosulfate citrate bile-salts sucrose agars. Inversely, no samples were positive with cholera toxin-producing Vibrio cholerae. The physicochemical parameters at Samariang Estuary were more associated with the number of bacteria in the samples compared to Santubong Estuary.
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Nasreen, Tania, Nora A. S. Hussain, Mohammad Tarequl Islam, Fabini D. Orata, Paul C. Kirchberger, Rebecca J. Case, Munirul Alam, Stephanie K. Yanow, and Yann F. Boucher. "Simultaneous Quantification of Vibrio metoecus and Vibrio cholerae with Its O1 Serogroup and Toxigenic Subpopulations in Environmental Reservoirs." Pathogens 9, no. 12 (December 16, 2020): 1053. http://dx.doi.org/10.3390/pathogens9121053.

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Vibrio metoecus is a recently described aquatic bacterium and opportunistic pathogen, closely related to and often coexisting with Vibrio cholerae. To study the relative abundance and population dynamics of both species in aquatic environments of cholera-endemic and cholera-free regions, we developed a multiplex qPCR assay allowing simultaneous quantification of total V. metoecus and V. cholerae (including toxigenic and O1 serogroup) cells. The presence of V. metoecus was restricted to samples from regions that are not endemic for cholera, where it was found at 20% of the abundance of V. cholerae. In this environment, non-toxigenic O1 serogroup V. cholerae represents almost one-fifth of the total V. cholerae population. In contrast, toxigenic O1 serogroup V. cholerae was also present in low abundance on the coast of cholera-endemic regions, but sustained in relatively high proportions throughout the year in inland waters. The majority of cells from both Vibrio species were recovered from particles rather than free-living, indicating a potential preference for attached versus planktonic lifestyles. This research further elucidates the population dynamics underpinning V. cholerae and its closest relative in cholera-endemic and non-endemic regions through culture-independent quantification from environmental samples.
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LaRocque, Regina C., Bryan Krastins, Jason B. Harris, Lauren M. Lebrun, Kenneth C. Parker, Michael Chase, Edward T. Ryan, Firdausi Qadri, David Sarracino, and Stephen B. Calderwood. "Proteomic Analysis of Vibrio cholerae in Human Stool." Infection and Immunity 76, no. 9 (June 30, 2008): 4145–51. http://dx.doi.org/10.1128/iai.00585-08.

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ABSTRACT An effective vaccine for Vibrio cholerae is not yet available for use in the developing world, where the burden of cholera disease is highest. Characterizing the proteins that are expressed by V. cholerae in the human host environment may provide insight into the pathogenesis of cholera and assist with the development of an improved vaccine. We analyzed the V. cholerae proteins present in the stools of 32 patients with clinical cholera. The V. cholerae outer membrane porin, OmpU, was identified in all of the human stool samples, and many V. cholerae proteins were repeatedly identified in separate patient samples. The majority of V. cholerae proteins identified in human stool are involved in protein synthesis and energy metabolism. A number of proteins involved in the pathogenesis of cholera, including the A and B subunits of cholera toxin and the toxin-coregulated pilus, were identified in human stool. In a subset of stool specimens, we also assessed which in vivo expressed V. cholerae proteins were recognized uniquely by convalescent-phase as opposed to acute-phase serum from cholera patients. We identified a number of these in vivo expressed proteins as immunogenic during human infection. To our knowledge, this is the first characterization of the proteome of a pathogenic bacteria recovered from a natural host.
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Mushayabasa, Steady, and Claver P. Bhunu. "Assessing the Impact of Increasing Antimicrobial Resistance of Vibrio cholerae on the Future Trends of Cholera Epidemic." ISRN Biomathematics 2012 (December 4, 2012): 1–10. http://dx.doi.org/10.5402/2012/127492.

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Cholera, an acute intestinal infection caused by the bacterium Vibrio cholerae, remains a major public health problem in many parts of Africa, Asia, and Latin America. A mathematical model is developed, to assess the impact of increasing antimicrobial resistance of Vibrio cholerae on the future trends of the cholera epidemic. Equilibrium states of the model are determined and their stabilities have been examined. The impacts of increasing antimicrobial resistance of Vibrio cholerae on the future trends of cholera epidemic have been investigated through the reproductive number. Numerical results are provided to support analytical findings.
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Kitaoka, Maya, Sarah T. Miyata, Daniel Unterweger, and Stefan Pukatzki. "Antibiotic resistance mechanisms of Vibrio cholerae." Journal of Medical Microbiology 60, no. 4 (April 1, 2011): 397–407. http://dx.doi.org/10.1099/jmm.0.023051-0.

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As the causative agent of cholera, the bacterium Vibrio cholerae represents an enormous public health burden, especially in developing countries around the world. Cholera is a self-limiting illness; however, antibiotics are commonly administered as part of the treatment regimen. Here we review the initial identification and subsequent evolution of antibiotic-resistant strains of V. cholerae. Antibiotic resistance mechanisms, including efflux pumps, spontaneous chromosomal mutation, conjugative plasmids, SXT elements and integrons, are also discussed. Numerous multidrug-resistant strains of V. cholerae have been isolated from both clinical and environmental settings, indicating that antibiotic use has to be restricted and alternative methods for treating cholera have to be implemented.
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Dissertations / Theses on the topic "Cholera Vibrio cholerae"

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Nygren, Erik. "A mouse model for direct evaluation of cholera vaccines /." Göteborg : Dept. of Microbiology and immunology, Institute of Biomedicine, The Sahlgrenska Academy at University of Gothenburg, 2009. http://hdl.handle.net/2077/19376.

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Moore, Sandra. "Dynamics of cholera epidemics in Haiti and Africa." Thesis, Aix-Marseille, 2016. http://www.theses.fr/2016AIXM5505/document.

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Le cholera est une maladie diarrhéique aiguë due à la consommation d’eau ou d’aliments contaminés par des souches toxigéniques de Vibrio cholerae. Selon le “paradigme du choléra”, la maladie est provoquée par une exposition à un réservoir environnemental de V. cholerae avec des épidémies directement modulées par des facteurs environnementaux. Cependant, comme divers arguments plaident contre ce dogme, nous avons voulu élucider les mécanismes de la dynamique des épidémies de cholera dans trois foyers situés en Haïti, en République Démocratique du Congo (RDC) et en Afrique de l’Ouest. Nous avons associé une analyse temporo-spatiale des épidémies à une étude génétique des isolats de V. cholerae. En Haïti, nous avons cherché à savoir si les épidémies actuelles étaient dues à des souches toxigéniques de V. cholerae O1 durablement implantées dans l’environnement aquatique. En Afrique de l’Ouest, notre étude a révélé qu’Accra, la capitale du Ghana, était le principal foyer de choléra pour l’ensemble des pays d’Afrique de l’Ouest situés à l’Ouest du Nigeria. Le réseau d’eau d’Accra a probablement joué un rôle dans la propagation rapide de V. cholerae vers la majorité des quartiers de la ville. Les épidémies de choléra ont diffusé vers les autres pays sous la forme de vagues épidémiques et plusieurs épidémies ont été liées à la migration de populations à risque comme certains pêcheurs. En conclusion, notre réflexion globale sur les épidémies de choléra dans ces trois foyers distincts nous donne une vision cohérente des mécanismes d’émergence et de diffusion du choléra
Cholera is an acute diarrheal disease caused by consumption of water or food contaminated with toxigenic Vibrio cholerae. According to the "cholera paradigm", the disease is contracted by exposure to environmental reservoirs of V. cholerae, with outbreaks driven directly by climatic factors. However, as recent findings argue against this dogma, we aimed to elucidate the dynamics of cholera outbreaks in three global foci: Haiti, Democratic Republic of the Congo (DRC) and West Africa. We combined spatiotemporal analysis of epidemics with genetic assessment of V. cholerae isolates. In Haiti, we assessed whether outbreak re-emergence during the rainy season was due to toxigenic V. cholerae O1 strains that have settled into the aquatic environment. Instead, we found that the re-emergence of outbreaks was likely due to persisting outbreaks during the dry season that were insufficiently controlled, rather than an environmental reservoir of V. cholerae O1. In West Africa, our study revealed that Accra, Ghana was the hotspot of cholera in the entire region of West Africa, west of Nigeria. The Accra water network likely played a role in rapid diffusion of V. cholerae throughout the city. Cholera outbreaks spread from Accra into other countries in a wave-like fashion. Distinct outbreaks were linked via migration of at-risk populations, such as certain fishermen. In conclusion, our global reflection of cholera epidemics in these three distinct foci provides a coherent vision of the mechanisms of cholera emergence and diffusion
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Le, Roux Wouter Jacobus. "Population dynamics of Vibrio cholerae in the Vaal Barrage." Pretoria : [s.n.], 2005. http://upetd.up.ac.za/thesis/available/etd-02162007-175110.

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Falklind, Jerkérus Susanna. "Vibrio cholerae O139 : identification, characterization and vaccine strategies /." Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-696-0/.

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Occhino, Deborah Ann. "Vibrio cholerae iron transport : characterization of two tonB systems and components of a heme transport system /." Digital version accessible at:, 1998. http://wwwlib.umi.com/cr/utexas/main.

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Zo, Young-Gun. "Phylogenomic and structural analyses of Vibrio cholerae populations and endemic cholera." College Park, Md. : University of Maryland, 2005. http://hdl.handle.net/1903/3090.

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Thesis (Ph. D.) -- University of Maryland, College Park, 2005.
Thesis research directed by: Marine-Estuarine-Environmental Sciences. Title from t.p. of PDF. Includes bibliographical references. Published by UMI Dissertation Services, Ann Arbor, Mich. Also available in paper.
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Mann, Maretta Clare, and n/a. "Sialylmimetics as Potential Inhibitors fo Vibrio Cholerae Sialidase." Griffith University. Institute for Glycomics, 2004. http://www4.gu.edu.au:8080/adt-root/public/adt-QGU20061006.083947.

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Cholera is an epidemic infectious diarrhoeal disease that for centuries has proven its frightening ability to cause rapid and widespread loss of human life. All symptoms associated with cholera are a result of rapid dehydration due to infection by pathogenic strains of the bacterium Vibrio cholerae. The damaging effects associated with cholera are mainly attributed to the toxin, which is secreted by the bacterium and infects cells lining the gastrointestinal tract. A sialidase, also secreted by the bacterium, is believed to facilitate toxin uptake by the gastrointestinal epithelium. V. cholerae sialidase is therefore a potential target for therapeutic intervention. A survey of the literature reveals that sialidases from different species share common features with respect to their structure, substrate specificity and catalytic mechanism. The unsaturated sialic acid, Neu5Ac2en, inhibits most exosialidases with a dissociation constant of inhibitor of -10-4 to-10-6 M and has frequently been used as a template in the design of more potent sialidase inhibitors. In the case of V. cholerae sialidase, there have been no inhibitors reported to date that are significantly more potent than Neu5Ac2en itself The present research aimed to develop a range of mimics of Neu5Ac2en, which contain various substituents to replace the C-6 glycerol side chain, as potential inhibitors of V cholerae sialidase. The x-ray crystal structure of V cholerae sialidase was used to explore potential interactions between active site residues and C-6 modified Neu5Ac2en mimetics of known inhibitory potency. Opportunities for interactions within the glycerol side chain pocket in the active site of V cholerae sialidase are discussed. A novel synthetic strategy was developed for the synthesis of a series of glucuronidebased Neu5Ac2en mimetics starting from readily available GIcNAc. This approach was employed for the preparation of Neu5Ac2en mimetics that contained an ether or thioether substituent as replacement of the glycerol side chain of Neu5Ac2en. Progress was also made towards the synthesis of a series of C-6 acylamino Neu5Ac2en mimetics. Analysis by 1H NMR spectroscopy showed that the acylamino derivatives adopted a half-chair conformation that was similar to the conformation of Neu5Ac2en but different to the conformation adopted by the ether and thioether derivatives prepared. The inhibitory activity of the C-6 ether and thioether Neu5Ac2en mimetics prepared was evaluated in vitro using an enzyme assay. It was found that most of the derivatives inhibited V. cholerae sialidase with a K1 of approximately 1O-4 M. The derivatives containing a hydrophobic side chain were found to be slightly more potent compared to derivatives with more hydrophilic side chains. A more detailed study of binding interactions between the C-6 thioether Neu5Ac2en mimetics and V cholerae sialdiase was carried out using STD 1H NMR spectroscopy and computational molecular modelling.
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Bougoudogo, Fiabou. "Contribution à l'étude de l'immunité protectrice contre le choléra : rôle des anticorps vibriocides reconnaissant le polysaccharide spécifique du lipopolysaccharide de "Vibrio cholerae" O:1." Paris 11, 1994. http://www.theses.fr/1994PA114831.

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Mitchell, Daniel David. "Cholera toxin inhibition and EpsF from its secretion system /." Thesis, Connect to this title online; UW restricted, 2006. http://hdl.handle.net/1773/9210.

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Jahan, Nasrin. "Structural studies of Vibrio cholerae quorum sensing proteins." Thesis, University of St Andrews, 2011. http://hdl.handle.net/10023/2565.

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The spread of cholera is always associated with contaminated food or water and this is the reason this disease has been endemic in developing countries for centuries due to their lack of proper sanitation facilities and poor or no infrastructure for sewage systems. Cholera can spread quickly and sporadically after any natural disaster that destroys the sewage system or safe drinking water supply of both developed and undeveloped countries. In Southeast Asia in December 2004 and in Pakistan and Haiti 2010, cholera outbreaks followed the natural disasters; with most of the cholera victims being children. Although it is known that the best way to prevent cholera outbreak is the development of the infrastructure, provision of a safe drinking water supply and proper sanitation, this is a very long-term process, and most of the developing countries cannot afford such improvements. These situations can be made worse by natural disasters. Therefore there is a pressing need for the development of a cholera vaccine and there have been numerous research projects working towards this end for several decades. A few of them have been successful to date but because of the severe side effects and narrow range of protection, more effective and wider range vaccine development is still ongoing. In this study, crystallographic and enzymatic studies have been carried out on several novel proteins involved in the control of the production of the factors required for quorum sensing. Quorum sensing is a process in which bacterial cells communicate among themselves by the synthesis, release and detection of small chemical compounds called autoinducers. In this work, structural analysis was carried out on proteins involved in the synthesis and detection of the major autoinducer of Vibrio cholerae, named CAI-1. The crystal structure of CqsA involved in CAI-1 synthesis has been successfully solved and its enzymatic properties have been characterized. The structure of one domain of the cytoplasmic region of the CAI-1 receptor CqsS was also elucidated, and other domains were expressed. The crystal structure of another enzyme (VCA0859, an aldo-keto reductase) thought to have been involved in the synthesis of CAI-1 was also determined. Another protein named VCA0939 was also studied, due to its importance in biofilm development, and its ability to control quorum-sensing in an alternative pathway in the mutated version of pathogenic strains of V. cholerae that were responsible for the seventh cholera pandemic. The aim of this project was to understand the three dimensional structure of some proteins that are involved in quorum sensing and control of the expression of virulence genes for the pathogenesis of V. cholerae. Understanding the three dimensional structure of the proteins and the mode of autoinducer binding to its specific receptor could be highly valuable in the development of a chemical compound that could lead to the discovery of a novel drug with the ability to target cross species specification.
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Books on the topic "Cholera Vibrio cholerae"

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Kaye Wachsmuth, I., Paul A. Blake, and Ørjan Olsvik, eds. Vibrio cholerae and Cholera. Washington, DC, USA: ASM Press, 1994. http://dx.doi.org/10.1128/9781555818364.

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Drasar, B. S., and B. D. Forrest, eds. Cholera and the Ecology of Vibrio cholerae. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-009-1515-2.

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Cholera: Current African perspectives. Hauppauge, N.Y: Nova Science Publishers, 2009.

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Sikora, Aleksandra E., ed. Vibrio Cholerae. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8685-9.

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Characterization of the maltose regulon of Vibrio cholerae: Involvement of maltose in production of outer membrane proteins and secretion of virulence factors. Uppsala: Swedish University of Agricultural Sciences, Dept. of Molecular Genetics, Uppsala Genetic Center, 1993.

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1935-, Takeda Yoshifumi, ed. Vibrio cholerae and cholera. Tokyo: KTK Scientific Publishers, 1988.

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Cholera and the Ecology of Vibrio cholerae. Springer, 2011.

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S, Drasar B., and Forrest B. D, eds. Cholera and the ecology of Vibrio cholerae. London: Chapman & Hall, 1996.

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Wachsmuth, I. Kaye, and Paul A. Blake. Vibrio Cholerae and Cholera: Molecular to Global Perspectives. ASM Press, 1994.

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Kaye, Wachsmuth, Blake Paul A, and Olsvik Ørjan, eds. Vibrio cholerae and cholera: Molecular to global perspectives. Washington, D.C: ASM Press, 1994.

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Book chapters on the topic "Cholera Vibrio cholerae"

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Colwell, Rita R., and Anwarul Huq. "Vibrios in the Environment: Viable but Nonculturable Vibrio cholerae." In Vibrio cholerae and Cholera, 117–33. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555818364.ch9.

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Vugia, Duc J. "Cholera Surveillance." In Vibrio cholerae and Cholera, 371–78. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555818364.ch24.

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Kaper, James B., Alessio Fasano, and Michele Trucksis. "Toxins of Vibrio cholerae." In Vibrio cholerae and Cholera, 143–76. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555818364.ch11.

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Glenn Morris, J. "Vibrio cholerae O139 Bengal." In Vibrio cholerae and Cholera, 95–102. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555818364.ch7.

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Wachsmuth, Kaye, Ørjan Olsvik, Gracia M. Evins, and Tanja Popovic. "Molecular Epidemiology of Cholera." In Vibrio cholerae and Cholera, 357–70. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555818364.ch23.

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Levine, Myron M., and Carol O. Tacket. "Recombinant Live Cholera Vaccines." In Vibrio cholerae and Cholera, 395–413. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555818364.ch26.

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Holmgren, J., J. Osek, and A. M. Svennerholm. "Protective Oral Cholera Vaccine Based on a Combination of Cholera Toxin B Subunit and Inactivated Cholera Vibrios." In Vibrio cholerae and Cholera, 415–24. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555818364.ch27.

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Kay, Bradford A., Cheryl A. Bopp, and Joy G. Wells. "Isolation and Identification of Vibrio cholerae O1 from Fecal Specimens." In Vibrio cholerae and Cholera, 1–25. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555818364.ch1.

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Barrett, Timothy J., and John C. Feeley. "Serologic Diagnosis of Vibrio cholerae O1 Infections." In Vibrio cholerae and Cholera, 135–41. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555818364.ch10.

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Ottemann, Karen M., and John J. Mekalanos. "Regulation of Cholera Toxin Expression." In Vibrio cholerae and Cholera, 177–85. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555818364.ch12.

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Conference papers on the topic "Cholera Vibrio cholerae"

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Anas, Abdulaziz, Kiran Krishna, Sreelakshmi PK, Syamkumar V, Jasmin C, Beena James, and Sobha kurien. "Multiple drug-resistant <em>Vibrio cholerae </em>responsible for cholera outbreak among migrant domestic workers in Kerala, South India." In 1st International Electronic Conference on Microbiology. Basel, Switzerland: MDPI, 2020. http://dx.doi.org/10.3390/ecm2020-07103.

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Konnov, Nikolai P., Vil B. Baiburin, Svetlana P. Zadnova, and Uryi P. Volkov. "Comparative microscopy study of Vibrio cholerae flagella." In BiOS '99 International Biomedical Optics Symposium, edited by Eiichi Tamiya and Shuming Nie. SPIE, 1999. http://dx.doi.org/10.1117/12.350626.

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Mourino-Perez, Rosa R., and Josue Alvarez-Borrego. "Color correlation for the recognition of Vibrio cholerae O1 in seawater." In ICO XVIII 18th Congress of the International Commission for Optics, edited by Alexander J. Glass, Joseph W. Goodman, Milton Chang, Arthur H. Guenther, and Toshimitsu Asakura. SPIE, 1999. http://dx.doi.org/10.1117/12.354900.

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Sun, Jun, Abdoul Nasser Ibrahim, Jianhua Gong, Liyang Yang, Yi Li, and Jieping Zhou. "Study on spread of vibrio cholera in rivers based on Cellular Automata Model." In IGARSS 2012 - 2012 IEEE International Geoscience and Remote Sensing Symposium. IEEE, 2012. http://dx.doi.org/10.1109/igarss.2012.6351379.

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Haselhorst, Thomas, Carolyn Trower, Jennifer Wilson, Ross Coppel, and Mark von Itzstein. "EPITOPE MAPPING BY SATURATION TRANSFER DIFFERENCE NMR OF SIALIC ACID MIMETICS WITH VIBRIO CHOLERAE SIALIDASE." In XXIst International Carbohydrate Symposium 2002. TheScientificWorld Ltd, 2002. http://dx.doi.org/10.1100/tsw.2002.514.

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Mandal, Rahul S., Atri Ta, and Santasabuj Das. "In silico designing and experimental validation of a potential small molecule inhibitor against vibrio cholerae AphB." In BCB '14: ACM-BCB '14. New York, NY, USA: ACM, 2014. http://dx.doi.org/10.1145/2649387.2660778.

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Mulyani, Yani, Ingrid Vitriyani Artauli, and Katarina Turnip. "Antibacterial Activity from Ethanol Extracts and Fractions of Family Asteraceae Leaf Against Bacillus cereus and Vibrio cholera." In 2nd Bakti Tunas Husada-Health Science International Conference (BTH-HSIC 2019). Paris, France: Atlantis Press, 2020. http://dx.doi.org/10.2991/ahsr.k.200523.073.

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Priyadarzini, T. R. K., J. Fermin Angelo Selvin, and K. Veluraja. "Molecular Dynamics Simulation Studies on Sialic Acid and Its Acetylated Derivatives and Their Interaction with Vibrio Cholerae Neuraminidase." In 2009 International Association of Computer Science and Information Technology - Spring Conference. IEEE, 2009. http://dx.doi.org/10.1109/iacsit-sc.2009.62.

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"Phenotypic characterization of marine phage cocktail from Batangas Philippines against Multi-Drug Resistant Pseudomonas aeruginosa, Methicillin Resistant Staphylococcus aureus, and Vibrio cholera." In Multi-Disciplinary Manila (Philippines) Conferences Jan. 23-24, 2017, Manila (Philippines). Universal Researchers (UAE), 2017. http://dx.doi.org/10.17758/uruae.ae0117608.

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Vodyanickaya, S. YU, N. V. Pavlovich, O. V. Sergienko, S. V. Volovikova, V. V. Batashev, O. V. Lyah, and N. G. Ivanova. "On the study of the resistance of vibrio cholerae strains in chemical decontamination of ballast water with a disinfectant from the polyguanidine group." In Scientific achievements of the third millennium. LJournal, 2019. http://dx.doi.org/10.18411/scienceconf-05-2019-12.

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Reports on the topic "Cholera Vibrio cholerae"

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Yeates, Elissa, Kayla Cotterman, and Angela Rhodes. Hydrologic impacts on human health : El Niño Southern Oscillation and cholera. Engineer Research and Development Center (U.S.), January 2020. http://dx.doi.org/10.21079/11681/39483.

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Abstract:
A non-stationary climate imposes considerable challenges regarding potential public health concerns. The El Niño Southern Oscillation (ENSO) cycle, which occurs every 2 to 7 years, correlates positively with occurrences of the waterborne disease cholera. The warm sea surface temperatures and extreme weather associated with ENSO create optimal conditions for breeding the Vibrio cholerae pathogen and for human exposure to the pathogenic waters. This work explored the impacts of ENSO on cholera occurrence rates over the past 50 years by examining annual rates of suspected cholera cases per country in relation to ENSO Index values. This study provides a relationship indicating when hydrologic conditions are optimal for cholera growth, and presents a statistical approach to answer three questions: Are cholera outbreaks more likely to occur in an El Niño year? What other factors impact cholera outbreaks? How will the future climate impact cholera incidence rates as it relates to conditions found in ENSO? Cholera outbreaks from the 1960s to the present are examined focusing on regions of Central and South America, and southern Asia. By examining the predictive relationship between climate variability and cholera, we can draw conclusions about future vulnerability to cholera and other waterborne pathogenic diseases.
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Buckley, Patricia E., James J. Valdes, and Kevin P. O'Connell. Construction and Analysis of a MutL Knockout Strain of Vibrio cholerae. Fort Belvoir, VA: Defense Technical Information Center, October 2007. http://dx.doi.org/10.21236/ada473545.

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Perez-Rosas, N., and T. C. Hazen. Survival and distribution of Vibrio cholerae in a tropical rain forest stream. Office of Scientific and Technical Information (OSTI), December 1988. http://dx.doi.org/10.2172/666266.

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