Relevant bibliographies by topics / Cholesterol and inflammatory molecules

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Cholesterol and inflammatory molecules'

Author: Grafiati
Published: 7 June 2025
Last updated: 2 August 2025

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Journal articles on the topic "Cholesterol and inflammatory molecules"

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Huwait, Etimad, Nouf Al-Gharawi, Maryam A. Al-Ghamdi, et al. "Thymoquinone (TQ) Inhibits Inflammation and Migration of THP-1 Macrophages: Mechanistic Insights into the Prevention of Atherosclerosis Using In-Vitro and In-Silico Analysis." Current Issues in Molecular Biology 44, no. 4 (2022): 1740–53. http://dx.doi.org/10.3390/cimb44040120.

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Atherosclerosis is an inflammatory disease mediated by interferon (IFN-γ) in concert with cell adhesion molecules and chemokines. Thymoquinone (TQ), a flavonoid derived from Nigella sativa, is reported to have anti-inflammatory, antioxidant, and cardiovascular protective properties. We evaluated the effects of TQ on the key pathogenic stages of atherosclerosis, including cell viability, inflammatory gene expression, cell migration, and cholesterol efflux, on human THP-1 macrophages in-vitro. Moreover, in-silico analysis was performed to predict the molecular targets and signaling mechanisms. W
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Carvalho, JF, RMR Pereira, and Y. Shoenfeld. "Vaccination, atherosclerosis and systemic lupus erythematosus." Lupus 18, no. 13 (2009): 1209–12. http://dx.doi.org/10.1177/0961203309345725.

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Atherosclerosis is an inflammatory disease, leading to the formation of pro-inflammatory and pro-oxidative lipids that generate an immune response. Several antigens have been shown to activate the immune response and affect the development of atherogenesis. Systemic lupus erythematosus is an autoimmune and inflammatory disease strongly associated with premature development of atherosclerotic plaques. Modulation of the immune system could represent a useful approach to prevent and/or treat atherosclerosis. A vaccination-based approach might be a useful, effective tool in the modern arsenal of c
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Yvan-Charvet, Laurent, Fabrizia Bonacina, Rodolphe Renè Guinamard, and Giuseppe Danilo Norata. "Immunometabolic function of cholesterol in cardiovascular disease and beyond." Cardiovascular Research 115, no. 9 (2019): 1393–407. http://dx.doi.org/10.1093/cvr/cvz127.

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Abstract Inflammation represents the driving feature of many diseases, including atherosclerosis, cancer, autoimmunity and infections. It is now established that metabolic processes shape a proper immune response and within this context the alteration in cellular cholesterol homeostasis has emerged as a culprit of many metabolic abnormalities observed in chronic inflammatory diseases. Cholesterol accumulation supports the inflammatory response of myeloid cells (i.e. augmentation of toll-like receptor signalling, inflammasome activation, and production of monocytes and neutrophils) which is ben
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Kremkow, Jan, Meike Luck, Daniel Huster, Peter Müller, and Holger A. Scheidt. "Membrane Interaction of Ibuprofen with Cholesterol-Containing Lipid Membranes." Biomolecules 10, no. 10 (2020): 1384. http://dx.doi.org/10.3390/biom10101384.

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Deciphering the membrane interaction of drug molecules is important for improving drug delivery, cellular uptake, and the understanding of side effects of a given drug molecule. For the anti-inflammatory drug ibuprofen, several studies reported contradictory results regarding the impact of ibuprofen on cholesterol-containing lipid membranes. Here, we investigated membrane localization and orientation as well as the influence of ibuprofen on membrane properties in POPC/cholesterol bilayers using solid-state NMR spectroscopy and other biophysical assays. The presence of ibuprofen disturbs the mo
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DeLong, Jonathan Howard, Sofia Evelyn Velazquez, Leonardo Shiuchi Sun, Margaret J. Landreneau, and Lauren Hachmann Sansing. "Cholesterol, LXR activation, and myelin uptake in the macrophage response to diverse signals." Journal of Immunology 208, no. 1_Supplement (2022): 111.23. http://dx.doi.org/10.4049/jimmunol.208.supp.111.23.

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Abstract Introduction Monocyte-derived macrophages contribute to neurotoxic inflammation during hemorrhagic stroke. Cholesterol is an immunosuppressive molecule present at high levels in the brain, primarily as a component of myelin. Uptake of myelin and activation of the transcription factor LXR downstream of myelin and cholesterol uptake limits the macrophage response to LPS. However, the roles of these molecules in macrophage responses to the endogenous signals present during sterile inflammation are poorly understood. The present study seeks to determine what role myelin, cholesterol, and
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Orekhov, Alexander N., Alexander M. Markin, Vasily N. Sukhorukov, Victoria A. Khotina, and Ekaterina Ivanova. "Pro-inflammatory molecules induce cholesterol accumulation in macrophages: Role of inflammatory response in foam cell formation." Atherosclerosis 320 (March 2021): 129–30. http://dx.doi.org/10.1016/j.atherosclerosis.2021.01.007.

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Fernando, Lawrence, Jing Echesabal-Chen, Murphy Miller, et al. "Cholesterol Efflux Decreases TLR4-Target Gene Expression in Cultured Macrophages Exposed to T. brucei Ghosts." Microorganisms 12, no. 8 (2024): 1730. http://dx.doi.org/10.3390/microorganisms12081730.

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Trypanosoma brucei causes African trypanosomiasis in humans. Infection with T. brucei elicits a potent pro-inflammatory immune response within infected human hosts, and this response is thought to at least be partially due to Toll-like receptor (TLR) activation. In response to stimulation by lipopolysaccharide and other pathogen antigens, TLR4 translocates to lipid rafts, which induces the expression of pro-inflammatory genes. However, cholesterol efflux is acknowledged as anti-inflammatory due to promoting lipid raft disruption. In this study, we wanted to assess the impact of T. brucei “ghos
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Packard, René R. S., and Peter Libby. "Inflammation in Atherosclerosis: From Vascular Biology to Biomarker Discovery and Risk Prediction." Clinical Chemistry 54, no. 1 (2008): 24–38. http://dx.doi.org/10.1373/clinchem.2007.097360.

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Abstract Recent investigations of atherosclerosis have focused on inflammation, providing new insight into mechanisms of disease. Inflammatory cytokines involved in vascular inflammation stimulate the generation of endothelial adhesion molecules, proteases, and other mediators, which may enter the circulation in soluble form. These primary cytokines also induce production of the messenger cytokine interleukin-6, which stimulates the liver to increase production of acute-phase reactants such as C-reactive protein. In addition, platelets and adipose tissue can generate inflammatory mediators rel
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Miller, Yury I., Juliana M. Navia-Pelaez, Maripat Corr, and Tony L. Yaksh. "Lipid rafts in glial cells: role in neuroinflammation and pain processing." Journal of Lipid Research 61, no. 5 (2019): 655–66. http://dx.doi.org/10.1194/jlr.tr119000468.

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Activation of microglia and astrocytes secondary to inflammatory processes contributes to the development and perpetuation of pain with a neuropathic phenotype. This pain state presents as a chronic debilitating condition and affects a large population of patients with conditions like rheumatoid arthritis and diabetes, or after surgery, trauma, or chemotherapy. Here, we review the regulation of lipid rafts in glial cells and the role they play as a key component of neuroinflammatory sensitization of central pain signaling pathways. In this context, we introduce the concept of an inflammaraft (
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Biagioli, Michele, Silvia Marchianò, Adriana Carino, et al. "Bile Acids Activated Receptors in Inflammatory Bowel Disease." Cells 10, no. 6 (2021): 1281. http://dx.doi.org/10.3390/cells10061281.

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Once known exclusively for their role in nutrients absorption, bile acids have emerged as signaling molecules, generated from cholesterol breakdown, acting on several immune cells by activating a variety of receptors including the G protein-coupled bile acid receptor 1 (GPABR1 or TGR5), the Farnesoid-X-receptor (FXR) and, as recently discovered, the retinoid-related orphan receptors (ROR)γt. GPBAR1, FXR, and RORγt are highly expressed in cells of the innate and adaptive immune system (i.e., dendritic cells (DCs), macrophages, innate lymphoid 3 cells (ILC3s), and T helper 17 (Th17) lymphocytes)
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Dissertations / Theses on the topic "Cholesterol and inflammatory molecules"

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Lee, Dong-Young. "Modification of high-density lipoprotein 3 by secretory sphingomyelinase, its effects on cholesterol trafficking/ transport, and secretory sphingomyelinase as a protein biomarker for inflammatory disease." Thesis, McGill University, 2008. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=21990.

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Secretory sphingomyelinase (S-SMase), one of the products of acid sphingomyelinase (ASM) or sphingomyelin phosphodiesterase-1 (SMPD-1) gene, hydrolyses sphingomyelin (SM) present in plasma membrane and lipoprotein particles. A number of studies have indicated that S-SMase plays an important role in intracellular or paracrine ceramide second messenger signaling pathways and development of subendothelial atherosclerotic plaque. However, a well-defined function of this enzyme in reverse cholesterol pathway (RCT) in vivo is yet to be elucidated. In this dissertation, we investigated modification
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Kanamori, Yohei. "Transcriptional regulation of hepcidin by molecules mediating inflammatory responses." Kyoto University, 2018. http://hdl.handle.net/2433/232336.

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Kyoto University (京都大学)<br>0048<br>新制・課程博士<br>博士(農学)<br>甲第21135号<br>農博第2261号<br>新制||農||1057(附属図書館)<br>学位論文||H30||N5109(農学部図書室)<br>京都大学大学院農学研究科応用生物科学専攻<br>(主査)教授 松井 徹, 教授 久米 新一, 教授 廣岡 博之<br>学位規則第4条第1項該当
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Li, L. "The crosstalk between Toll-like receptor signalling and cholesterol homeostasis under inflammatory stress." Thesis, University College London (University of London), 2013. http://discovery.ucl.ac.uk/1406398/.

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It is proposed that dysregulated cholesterol homeostasis resulting from inflammation and the crosstalk that develops between these two factors are important mechanisms for the progression of both atherosclerosis and chronic kidney disease (CKD). This thesis emphasizes the identification of various pathways of inflammation, Toll-like receptors (TLRs) and dysregulated cholesterol homeostasis. These factors are inter-dependent and interact in initiating foam cell formation in the vascular wall and kidney. THP-1 and HK2 cells were used as in vitro cell models. The roles of myeloid differentiation
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Singh, Baljit. "The role of co-stimulatory molecules and inflammatory cytokines in a murine model of inflammatory bowel disease." Thesis, University of Oxford, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.275392.

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Crone, Elizabeth. "Spatial localisation of oxidative and inflammatory markers within advanced atherosclerotic plaques." Thesis, University of Canterbury. School of Biological Sciences, 2008. http://hdl.handle.net/10092/2259.

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Five atherosclerotic carotid and femoral plaques were sliced longitudinally. Each section was analysed for the concentrations of neopterin, α-tocopherol, TBARS, DOPA, dityrosine, protein carbonyl, protein and cholesterol. The spatial concentrations of the oxidative and inflammatory markers were diverse across and between the individual plaques suggested by the lack of consistent correlations and trends. The only correlation that occurred twice within the individual plaques was a positive correlation between α-tocopherol and cholesterol levels. In the combined plaque analysis which included dat
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Bloom, Stuart. "Adhesion molecules in the gastrointestinal tract : a search for cell surface molecules upregulated in inflammatory bowel disease." Thesis, University of Oxford, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.239338.

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Sakthivel, Priya. "Co-stimulatory molecules : genes to protein in autoimmune and inflammatory disorders /." Stockholm, 2007. http://diss.kib.ki.se/2007/978-91-7357-425-9/.

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Nygård, Mikael. "Effects of aging and inflammatory molecules on the suprachiasmatic circadian clock /." Stockholm, 2007. http://diss.kib.ki.se/2007/978-91-7357-394-8/.

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Smailbegovic, Amir. "The effects of heparin and related molecules on inflammatory cell function." Thesis, King's College London (University of London), 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.406674.

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Coste, Emmanuel. "Development of small molecules as anti-inflammatory and anti-resorptive drugs." Thesis, University of Edinburgh, 2011. http://hdl.handle.net/1842/9941.

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Rheumatoid arthritis is an auto-immune inflammatory disease that leads to stiff and swollen joints. Patients also have severe bone destruction of the affected joints and another common symptom of rheumatoid arthritis is a generalised bone loss that can lead to osteoporosis. Currently, there are many treatments for rheumatoid arthritis, which provide a recession of the inflammatory symptoms. However, none of these treatments are able to provide a complete protection against the rheumatoid arthritis-induced bone loss. Furthermore, the most effective available treatments such as glucocorticoids o
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Books on the topic "Cholesterol and inflammatory molecules"

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Sharma, Ramesh Kumar, Maria Anna Coniglio, and Pasqualina Laganà. Natural Inflammatory Molecules in Fruits and Vegetables. Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-88473-4.

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M, Harlan John, and Liu David Y. 1950-, eds. Adhesion: Its role in inflammatory disease. W.H. Freeman, 1992.

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G, Bazán Nicolás, Botting Jack H, and Vane John R, eds. New targets in inflammation: Inhibitors of COX-2 or adhesion molecules : proceedings of a conference held on April 15-16, 1996, in New Orleans, USA, supported by an educational grant from Boehringer Ingelheim. Kluwer Academic Publishers, 1996.

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Keith, Lierre. The vegetarian myth: Food, justice and sustainability. Flashpoint Press, 2009.

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Sharma, Ramesh Kumar, Pasqualina Laganà, and Maria Anna Coniglio. Natural Inflammatory Molecules in Fruits and Vegetables. Springer International Publishing AG, 2022.

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(Editor), John M. Harlan, and David Y. Liu (Editor), eds. Adhesion: Its Role in Inflammatory Disease (Breakthroughs in Molecular Biology Series). Oxford University Press, USA, 1991.

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(Editor), N. Bazan, Jack H. Botting (Editor), and Sir John R. Vane (Editor), eds. New Targets in Inflammation: Inhibitors of COX-2 or Adhesion Molecules. Springer, 1996.

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Newcom, Gregory. Anti-Inflammatory Low Cholesterol Fast Track Cookbook - $10 Discount Now This Month Only! Blurb, 2023.

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Pezzi, Vincenzo, Rosa Sirianni, and Michihisa Umetani, eds. Cholesterol and Oxysterols as Signal Molecules in Human Pathophysiology and Cancer: Implications for New Therapeutic Strategies. Frontiers Media SA, 2020. http://dx.doi.org/10.3389/978-2-88963-278-7.

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Bazan, N., Jack H. Botting, and Sir John R. Vane. New Targets in Inflammation: Inhibitors of COX-2 or Adhesion Molecules Proceedings of a Conference Held on April 15-16, 1996, in New Orleans, USA, Supported by an Educational Grant from Boehringer Ingelheim. Springer London, Limited, 2012.

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Book chapters on the topic "Cholesterol and inflammatory molecules"

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Cohen Tervaert, Jan Willem. "Cholesterol and Modifications of Cholesterol in Rheumatic Disorders." In Inflammatory Diseases of Blood Vessels. Wiley-Blackwell, 2012. http://dx.doi.org/10.1002/9781118355244.ch43.

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Gimpl, Gerald. "Cholesterol–Protein Interaction: Methods and Cholesterol Reporter Molecules." In Cholesterol Binding and Cholesterol Transport Proteins:. Springer Netherlands, 2010. http://dx.doi.org/10.1007/978-90-481-8622-8_1.

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Gewies, Andreas, Jürgen Ruland, Alexey Kotlyarov, et al. "Macrophage Inflammatory Protein-1 Alpha." In Encyclopedia of Signaling Molecules. Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_100734.

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Gewies, Andreas, Jürgen Ruland, Alexey Kotlyarov, et al. "Macrophage Inflammatory Protein-1 Beta." In Encyclopedia of Signaling Molecules. Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_100735.

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Nkooyeh, B., and T. R. Neyestani. "24. Cholesterol and vitamin D: how the ‘mother’ and ‘daughter’ molecules interact." In Handbook of cholesterol. Wageningen Academic Publishers, 2016. http://dx.doi.org/10.3920/978-90-8686-821-6_24.

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Sharanya, C. S., and Madathilkovilakath Haridas. "Anti-inflammatory Molecules: Enzyme Inhibitors." In Bioresources and Bioprocess in Biotechnology. Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-4284-3_9.

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Kok, Klaartje, Ana Ibarra, Krishna Shah, and James O. Lindsay. "Medical Management X: Small Molecules." In Inflammatory Bowel Disease Nursing Manual. Springer Nature Switzerland, 2025. https://doi.org/10.1007/978-3-031-87503-8_14.

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Vejlsgaard, Gunhild Lange, Nils Lauge Hansen, Elisabeth Ralfkiaer, and Robert Rothlein. "Role of Cellular Adhesion in Inflammatory Cutaneous Disorders." In Leukocyte Adhesion Molecules. Springer New York, 1990. http://dx.doi.org/10.1007/978-1-4612-3234-6_8.

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Nagaich, Upendra, and Vandana Chaudhary. "Nonsteroidal Anti-Inflammatory Drugs: Concepts and Innovations." In Biologically Active Small Molecules. Apple Academic Press, 2022. http://dx.doi.org/10.1201/9781003283119-11.

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Bhavya, B. C., and Madathilkovilakath Haridas. "Anti-inflammatory Molecules: Immune System Mediators." In Bioresources and Bioprocess in Biotechnology. Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-4284-3_10.

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Conference papers on the topic "Cholesterol and inflammatory molecules"

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Payares, Lily Margareth, Virginia Paredes Méndez, Margareth Dugarte, Juan Carlos Rincón Montenegro, and Lizeth Gutiérrez Púa. "Use of the Microalgae Chlorella Sp. to Biofunctionalized Magnesium Surfaces in Order to Decrease the Rate of Corrosion and Promote the Bone Remodeling Process." In CONFERENCE 2022. AMPP, 2022. https://doi.org/10.5006/c2022-18514.

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Extended Abstract Frequently, the human body is susceptible to injuries such as the fracture of one or more of its bones, so it is necessary to use implants or some fixation element to keep the fragments of the fracture together, allowing an orderly repair that facilitates healing or compensates the lack or loss of bone tissue. Temporary orthopedic implants have been used clinically to repair broken or fractured bones during the healing process. The most used are plates, screws, nails, wires, and intramedullary nails. It is usually necessary to perform operations to open the injury site, join
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Guha, Snigdha, та Kaustav Majumder. "Dietary γ-glutamyl Valine in Reducing Inflammation in Endothelial Cells and in a Mouse Model for Atherosclerosis". У 2022 AOCS Annual Meeting & Expo. American Oil Chemists' Society (AOCS), 2022. http://dx.doi.org/10.21748/mree8379.

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Cardiovascular diseases (CVDs) are the leading cause of mortality, and their prevalence is estimated at nearly 37% in the United States. Elevated blood glucose, elevated triglycerides, high blood pressure, and low levels of high-density lipoprotein, result in increased levels of inflammation and pathogenesis of various CVDs such as atherosclerosis and hypertension. Dietary γ-glutamyl valine (γ-EV), naturally found in dry beans, has exhibited beneficial biological activities to reduce chronic inflammation. The intervention of γ-EV was found to reduce the inflammatory responses in vascular endot
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Guha, Snigdha. "Efficacy of Great Northern Beans-derived γ-glutamyl Peptides in Reducing Vascular Inflammation". У 2022 AOCS Annual Meeting & Expo. American Oil Chemists' Society (AOCS), 2022. http://dx.doi.org/10.21748/pykq1684.

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The prevalence of cardiovascular diseases (CVDs) is estimated at nearly 37% in the United States. Elevated blood glucose, elevated triglycerides, high blood pressure, and low levels of high-density lipoprotein, result in increased levels of inflammation and pathogenesis of various CVDs such as atherosclerosis and hypertension. Dietary γ-glutamyl peptides (γ-GPs), naturally found in dry beans, have exhibited beneficial biological activities to reduce chronic inflammation. One such type of dry bean, Great Northern bean (GNB), is an important agricultural commodity for the US and for the state of
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Lammi, Carmen. "From the bench to the bedside: the history of lupin bioactive peptides as useful ingredient for the prevention of metabolic syndrome." In 2022 AOCS Annual Meeting & Expo. American Oil Chemists' Society (AOCS), 2022. http://dx.doi.org/10.21748/bwgm4089.

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Proteins derived from lupin seeds are gaining attention as a source of bioactive peptides. In fact, several pieces of evidence highlight the biological activities of lupin protein hydrolysates and peptides, including hypocholesterolemic, hypoglycemic, antimicrobial, anti-inflammatory, and immunomodulatory effects. The health-promoting activities of lupin peptides have been tested in different animal models and clinical trials. Briefly, diets containing protein from L. albus or L. angustifolius significantly reduce both total cholesterol and low-density lipoprotein (LDL) cholesterol (LDL-C) lev
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Petran, A., L. Losensky, A. Arbuzova, and J. Liebscher. "Cholesterol modified nucleosides as precursors for microtube self-assembly." In PROCESSES IN ISOTOPES AND MOLECULES (PIM 2013). AIP, 2013. http://dx.doi.org/10.1063/1.4833736.

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Griffiths, William. "Bile Acid Precursors: Intermediates in Cholesterol Removal or Signaling Molecules?" In Virtual 2021 AOCS Annual Meeting & Expo. American Oil Chemists’ Society (AOCS), 2021. http://dx.doi.org/10.21748/am21.32.

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Naveed, B., MD Weiden, A. Nolan, GS Kang, WN Rom, and LC Chen. "Costimulatory Molecules in the Inflammatory Response to PM2.5Exposure." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a3138.

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Villarreal-Calderon, R., J. Palacios-Moreno, S. Keefe, N. Osnaya, R. Torres-Jardon, and L. Calderon-Garciduenas. "Air Pollution, Heart Inflammatory Signaling Molecules and Chocolate." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a3149.

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Vale, Abel, Mariana Lucas, Daniela Ribeiro, and Eduarda Fernandes. "Research into New Molecules with Anti-Inflammatory Activity." In International Electronic Conference on Biomedicines. MDPI, 2023. http://dx.doi.org/10.3390/ecb2023-14095.

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Bachkova, I. K., I. V. Chernikov, A. V. Sen’kova, et al. "CHOLESTEROL-CONJUGATED SMALL INTERFERING RNA TARGETING IL6 ALLEVIATES ACUTE LUNG INJURY IN MICE." In XI МЕЖДУНАРОДНАЯ КОНФЕРЕНЦИЯ МОЛОДЫХ УЧЕНЫХ: БИОИНФОРМАТИКОВ, БИОТЕХНОЛОГОВ, БИОФИЗИКОВ, ВИРУСОЛОГОВ, МОЛЕКУЛЯРНЫХ БИОЛОГОВ И СПЕЦИАЛИСТОВ ФУНДАМЕНТАЛЬНОЙ МЕДИЦИНЫ. IPC NSU, 2024. https://doi.org/10.25205/978-5-4437-1691-6-285.

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Inflammatory diseases affect a substantial portion of the global population, and the current treatment is not universally effective. We demonstrate that locally administered cholesterol-conjugated siRNA targeting Il6 alleviates the symptoms of acute lung injury, which makes it a promising strategy for the treatment of inflammatory diseases with potential applications beyond the lungs.
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Reports on the topic "Cholesterol and inflammatory molecules"

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Alwagdani, Abdullah. Review Of mPGES-1 Inhibitors Based On The Benzoxazole And Its Isostere Scaffold For The Treatment Of Inflammatory Diseases. University of Tennessee Health Science Center, 2024. http://dx.doi.org/10.21007/com.lsp.2024.0021.

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The vital role of the prostanoid pathway in inflammation, pain, cancer, Alzheimer’s and many other diseases has attracted the drug discovery community to discover targets for therapeutic development. Although existing non-steroidal anti-inflammatory drugs (NSAIDs) inhibiting cyclooxygenases (COX) are widely used, the side effects of these NSAIDs limit the ling time medication. Microsomal prostaglandin E synthase-1 (mPGES-1) is an attractive target that is overexpressed during inflammations, and it could be a safe alternative to NSAIDs for treating inflammatory diseases.Since the discovery of m
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Mahanonda, Rangsini, Orawan Charatkulangkun, and Sathit Pichyangkul. The role of IL-17 in periodontitis. Chulalongkorn University, 2006. https://doi.org/10.58837/chula.res.2006.12.

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Background and objectives: IL-17 is a novel T cell-derived cytokine that promotes inflammatory responses. It is presence in inflamed gingival tissues and gingival crevicular fluid of periodontitis patients. In this study we investigated the effects of IL-17 alone or in combination with IFN-Y on the immune modulation of human gingival fibroblasts (HGFs) which would contribute to pathogenesis of periodontium. Methods and results: Various concentrations of IL-17, IFN-Y or the combination of these two cytokines were added to HGF cultures. The expression of ICAM-1, HLA-DR, and CD40 was assessed by
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You might also be interested in the extended bibliographies on the topic 'Cholesterol and inflammatory molecules' for particular source types:
Journal articles Dissertations / Theses Books
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