Log in

Relevant bibliographies by topics / Cholesterol control / Journal articles

To see the other types of publications on this topic, follow the link: Cholesterol control.

Journal articles on the topic 'Cholesterol control'

Author: Grafiati

Published: 4 June 2021

Last updated: 20 February 2023

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 50 journal articles for your research on the topic 'Cholesterol control.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse journal articles on a wide variety of disciplines and organise your bibliography correctly.

1

Kiberstis, P. A. "Idolizing Cholesterol Control." Science Signaling 2, no. 78 (July 7, 2009): ec232-ec232. http://dx.doi.org/10.1126/scisignal.278ec232.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Fletcher, Gerald F. "Fat and Cholesterol Control." Journal of Cardiopulmonary Rehabilitation 12, no. 3 (May 1992): 162–63. http://dx.doi.org/10.1097/00008483-199205000-00002.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Marecic, Maryfrances, and Robin Bagby. "Take Control of Cholesterol." Nutrition Today 23, no. 5 (September 1988): 47. http://dx.doi.org/10.1097/00017285-198809000-00011.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Belavic, Jennifer. "Drugs to control cholesterol." Nursing 42, no. 3 (March 2012): 68. http://dx.doi.org/10.1097/01.nurse.0000398753.52565.6a.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Haug, Anna, Arne T. Høstmark, Øystein Spydevold, and Einar Eilertsen. "Hypercholesterolaemia, hypotriacylglycerolaemia and increased lipoprotein lipase activity following orchidectomy in rats." Acta Endocrinologica 113, no. 1 (September 1986): 133–39. http://dx.doi.org/10.1530/acta.0.1130133.

Full text

Abstract:

Abstract. Plasma lipoproteins, faecal cholesterol excretion, and activities of lecithin: cholesterol acyltransferase (LCAT) hepatic lipase (HL), and lipoprotein lipase (LPL) were determined in castrated rats, in rats treated with testosterone propionate after castration, and in sham-operated controls. Compared to control rats, whole-plasma total cholesterol (TC) rose, and triacylglycerols (TG) fell in castrated rats, but were normalized by androgen substitution. VLDL components tended to be reduced, whereas HDL2 components rose following castration. In general, testosterone substitution normal

APA, Harvard, Vancouver, ISO, and other styles

6

Khan, B., H. G. Wilcox, and M. Heimberg. "Cholesterol is required for secretion of very-low-density lipoprotein by rat liver." Biochemical Journal 258, no. 3 (March 15, 1989): 807–16. http://dx.doi.org/10.1042/bj2580807.

Full text

Abstract:

To study potential effects of hepatic cholesterol concentration on secretion of very-low-density lipoprotein (VLDL) by the liver, male rats were fed on unsupplemented chow, chow with lovastatin (0.1%), or chow with lovastatin (0.1%) and cholesterol (0.1%) for 1 week. Livers were isolated from these animals and perfused in vitro, with a medium containing [2-14C]acetate, bovine serum albumin and glucose in Krebs-Henseleit buffer, and with an oleate-albumin complex. With lovastatin feeding, the hepatic concentrations of cholesteryl esters and triacylglycerols before perfusion were decreased, alth

APA, Harvard, Vancouver, ISO, and other styles

7

Anonymous. "Steps Overlooked in Cholesterol Control." Journal of Gerontological Nursing 20, no. 12 (December 1994): 47. http://dx.doi.org/10.3928/0098-9134-19941201-13.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Florez, Hermes, Kelly J. Hunt, and Willy Marcos Valencia. "Reducing Disparities in Cholesterol Control." JAMA 328, no. 8 (August 23, 2022): 714. http://dx.doi.org/10.1001/jama.2022.13284.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Björkhem, Ingemar. "Do oxysterols control cholesterol homeostasis?" Journal of Clinical Investigation 110, no. 6 (September 15, 2002): 725–30. http://dx.doi.org/10.1172/jci0216388.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Kiberstis, P. A. "MiR-33 in Cholesterol Control." Science Signaling 3, no. 127 (June 22, 2010): ec189-ec189. http://dx.doi.org/10.1126/scisignal.3127ec189.

Full text

APA, Harvard, Vancouver, ISO, and other styles

11

Collins, Sonya. "Statin therapy for cholesterol control." Pharmacy Today 19, no. 10 (October 2013): 36–37. http://dx.doi.org/10.1016/s1042-0991(15)31126-9.

Full text

APA, Harvard, Vancouver, ISO, and other styles

12

Gilutz, Harel, Julian Zelingher, Yaakov Henkin, Dan Y. Bonneh, Zvi Liss, Roni Peleg, Max Mayslus, Reuben Ilia, and Avi Porath. "Computerized community cholesterol control (4C)." Journal of the American College of Cardiology 39 (March 2002): 263. http://dx.doi.org/10.1016/s0735-1097(02)81178-0.

Full text

APA, Harvard, Vancouver, ISO, and other styles

13

Nugraheni, Kartika, and Siti Harnina Bintari. "Aktivitas antidislipidemia Tepung tempe dan susu kedelai pada profil lipid tikus diabetes yang diinduksi streptozotocin." Jurnal Gizi dan Dietetik Indonesia (Indonesian Journal of Nutrition and Dietetics) 4, no. 3 (May 22, 2017): 147. http://dx.doi.org/10.21927/ijnd.2016.4(3).147-153.

Full text

Abstract:

<p><strong>ABSTRACT</strong></p><p><strong>Background :</strong> dyslipidemia increases risk of cardiovascular disease on diabetes patients. Soybean contain many bioactive compounds which can help control lipid profile.</p><p><strong>Objectives :</strong> analyze the difference between fermented soybean (tempe flour) and unfermented soybean (soymilk) on lipid profile in diabetic rats.</p><p><strong>Methods : </strong>thirty male sprague dawley rats divided into 3 groups (1) diabetic control (2) tempe flou

APA, Harvard, Vancouver, ISO, and other styles

14

Slotte, J. P., and E. L. Bierman. "Depletion of plasma-membrane sphingomyelin rapidly alters the distribution of cholesterol between plasma membranes and intracellular cholesterol pools in cultured fibroblasts." Biochemical Journal 250, no. 3 (March 15, 1988): 653–58. http://dx.doi.org/10.1042/bj2500653.

Full text

Abstract:

This study examines the relationship between cellular sphingomyelin content and the distribution of unesterified cholesterol between the plasma-membrane pool and the putative intracellular regulatory pool. The sphingomyelin content of cultured human skin fibroblasts was reduced by treatment of intact cells with extracellularly added neutral sphingomyelinase, and subsequent changes in the activities of cholesterol-metabolizing enzymes were determined. Exposure of fibroblasts to 0.1 unit of sphingomyelinase/ml for 60 min led to the depletion of more than 90% of the cellular sphingomyelin, as det

APA, Harvard, Vancouver, ISO, and other styles

15

Hallikainen, Maarit, Henri Tuomilehto, Tarja Martikainen, Esko Vanninen, Juha Seppä, Jouko Kokkarinen, Jukka Randell, and Helena Gylling. "Cholesterol Metabolism and Weight Reduction in Subjects with Mild Obstructive Sleep Apnoea: A Randomised, Controlled Study." Cholesterol 2013 (May 16, 2013): 1–9. http://dx.doi.org/10.1155/2013/769457.

Full text

Abstract:

To evaluate whether parameters of obstructive sleep apnoea (OSA) associate with cholesterol metabolism before and after weight reduction, 42 middle-aged overweight subjects with mild OSA were randomised to intensive lifestyle intervention (N=23) or to control group (N=18) with routine lifestyle counselling only. Cholesterol metabolism was evaluated with serum noncholesterol sterol ratios to cholesterol, surrogate markers of cholesterol absorption (cholestanol and plant sterols) and synthesis (cholestenol, desmosterol, and lathosterol) at baseline and after 1-year intervention. At baseline, art

APA, Harvard, Vancouver, ISO, and other styles

16

Shahnaz, Begum, Satoshi Tada, Tatsushi Kajikawa, Toshihiko Ishida, and Koichi Kawanishi. "Automated Fluorimetric Determination of Cellular Cholesterol." Annals of Clinical Biochemistry: International Journal of Laboratory Medicine 35, no. 5 (September 1998): 665–70. http://dx.doi.org/10.1177/000456329803500511.

Full text

Abstract:

We developed a completely automated fluorimetric method for the determination of cellular cholesterol, consisting of enzymatic hydrolysis of cholesteryl ester to free cholesterol and enzymatic oxidation of free cholesterol in the presence of an indicator substrate to produce a fluorescent product. For control preparations of monocytes, the mean detection limit was 2.57 μmol/5 × 105 cells and the mean within-batch coefficients of variation were 9.30, 600 and 3.73% at mean cholesterol concentrations of 1.94, 9.05 and 12.49 μmol/5 × 105 cells, respectively. The results correlated well with those

APA, Harvard, Vancouver, ISO, and other styles

17

Bakkeren, H. F., F. Kuipers, R. J. Vonk, and T. J. C. Van Berkel. "Evidence for reverse cholesterol transport in vivo from liver endothelial cells to parenchymal cells and bile by high-density lipoprotein." Biochemical Journal 268, no. 3 (June 15, 1990): 685–91. http://dx.doi.org/10.1042/bj2680685.

Full text

Abstract:

Acetylated low-density lipoprotein (acetyl-LDL), biologically labelled in the cholesterol moiety of cholesteryl oleate, was injected into control and oestrogen-treated rats. The serum clearance, the distribution among the various lipoproteins, the hepatic localization and the biliary secretion of the [3H]cholesterol moiety were determined at various times after injection. In order to monitor the intrahepatic metabolism of the cholesterol esters of acetyl-LDL in vivo, the liver was subdivided into parenchymal, endothelial and Kupffer cells by a low-temperature cell-isolation procedure. In both

APA, Harvard, Vancouver, ISO, and other styles

18

Redgrave, T. G., C. L. Elsegood, J. C. L. Mamo, and M. J. Callow. "Effects of hypothyroidism on the metabolism of lipid emulsion models of triacylglycerol-rich lipoproteins in rats." Biochemical Journal 273, no. 2 (January 15, 1991): 375–81. http://dx.doi.org/10.1042/bj2730375.

Full text

Abstract:

Methimazole-treated hypothyroid rats were injected intravenously with triacylglycerol/cholesteryl oleate/cholesterol/phospholipid emulsions designed to model the composition of chylomicrons. Compared with controls, hypothyroidism decreased the clearance rates of emulsion cholesteryl oleate. Clearance of emulsion triolein was affected much less and could be accounted for by residual triolein in remnants, suggesting that triacylglycerol lipolysis by lipoprotein lipase was unaffected by hypothyroidism but that clearance of remnants from plasma was decreased. Assays in vitro showed increased activ

APA, Harvard, Vancouver, ISO, and other styles

19

HIGGINS, MALCOLM J. P., and DESPINA K. PAPACHRISTODOULOU. "Control of hepatic acyl-CoA: cholesterol acyltransferase in cholesterol-fed rats." Biochemical Society Transactions 17, no. 1 (February 1, 1989): 156. http://dx.doi.org/10.1042/bst0170156.

Full text

APA, Harvard, Vancouver, ISO, and other styles

20

Hussain, Syed Saad, Megan T. Harris, Alex J. B. Kreutzberger, Candice M. Inouye, Catherine A. Doyle, Anna M. Castle, Peter Arvan, and J. David Castle. "Control of insulin granule formation and function by the ABC transporters ABCG1 and ABCA1 and by oxysterol binding protein OSBP." Molecular Biology of the Cell 29, no. 10 (May 15, 2018): 1238–57. http://dx.doi.org/10.1091/mbc.e17-08-0519.

Full text

Abstract:

In pancreatic β-cells, insulin granule membranes are enriched in cholesterol and are both recycled and newly generated. Cholesterol’s role in supporting granule membrane formation and function is poorly understood. ATP binding cassette transporters ABCG1 and ABCA1 regulate intracellular cholesterol and are important for insulin secretion. RNAi interference–induced depletion in cultured pancreatic β-cells shows that ABCG1 is needed to stabilize newly made insulin granules against lysosomal degradation; ABCA1 is also involved but to a lesser extent. Both transporters are also required for optim

APA, Harvard, Vancouver, ISO, and other styles

21

Bouillet, Benjamin, Thomas Gautier, Damien Denimal, Maxime Samson, David Masson, Jean Paul Pais de Barros, Guillaume Maquart, et al. "Glucocorticoids impair HDL-mediated cholesterol efflux besides increased HDL cholesterol concentration: a proof of concept." European Journal of Endocrinology 183, no. 3 (September 2020): 297–306. http://dx.doi.org/10.1530/eje-20-0477.

Full text

Abstract:

Objective: Glucocorticoids (GC) are associated with increased cardiovascular morbidity despite increased HDL-C concentration. HDL-mediated cholesterol efflux, a major anti-atherogenic property of HDL particles, is negatively associated with CVD risk. We aimed to determine whether HDL-mediated cholesterol efflux was influenced by GC. Design: Prospective, observational study. Methods: Lipid parameters, HDL composition, HDL-mediated cholesterol efflux, cholesteryl ester transfer protein, phospholipid transfer protein and lecithin cholesterol acyl-transferase (LCAT) activities were determined in t

APA, Harvard, Vancouver, ISO, and other styles

22

Poli, Andrea, Franca Marangoni, Alberto Corsini, Enzo Manzato, Walter Marrocco, Daniela Martini, Gerardo Medea, and Francesco Visioli. "Phytosterols, Cholesterol Control, and Cardiovascular Disease." Nutrients 13, no. 8 (August 16, 2021): 2810. http://dx.doi.org/10.3390/nu13082810.

Full text

Abstract:

The use of phytosterols (or plant sterols) for the control of plasma cholesterol concentrations has recently gained traction because their efficacy is acknowledged by scientific authorities and leading guidelines. Phytosterols, marketed as supplements or functional foods, are formally classified as food in the European Union, are freely available for purchase, and are frequently used without any health professional advice; therefore, they are often self-prescribed, either inappropriately or in situations in which no significant advantage can be obtained. For this reason, a panel of experts wit

APA, Harvard, Vancouver, ISO, and other styles

23

Brown, Andrew J., and Joanne Hsieh. "Foiling IDOL to Help Control Cholesterol." Circulation Research 118, no. 3 (February 5, 2016): 371–73. http://dx.doi.org/10.1161/circresaha.116.308191.

Full text

APA, Harvard, Vancouver, ISO, and other styles

24

Topping, David. "Hydroxypropylmethylcellulose, Viscosity, and Plasma Cholesterol Control." Nutrition Reviews 52, no. 5 (April 27, 2009): 176–78. http://dx.doi.org/10.1111/j.1753-4887.1994.tb01416.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

25

Russell, David W. "Nuclear Orphan Receptors Control Cholesterol Catabolism." Cell 97, no. 5 (May 1999): 539–42. http://dx.doi.org/10.1016/s0092-8674(00)80763-1.

Full text

APA, Harvard, Vancouver, ISO, and other styles

26

Strzyz, Paulina. "Cholesterol feeds into cell growth control." Nature Reviews Molecular Cell Biology 18, no. 5 (April 5, 2017): 277. http://dx.doi.org/10.1038/nrm.2017.41.

Full text

APA, Harvard, Vancouver, ISO, and other styles

27

Franceschini, Guido, JoséP werba, and Laura Calabresi. "Drug control of reverse cholesterol transport." Pharmacology & Therapeutics 61, no. 3 (January 1994): 289–324. http://dx.doi.org/10.1016/0163-7258(94)90014-0.

Full text

APA, Harvard, Vancouver, ISO, and other styles

28

Thompson, P. D. "Putting Cholesterol Control on a Diet." JAMA: The Journal of the American Medical Association 262, no. 21 (December 1, 1989): 2998. http://dx.doi.org/10.1001/jama.1989.03430210036018.

Full text

APA, Harvard, Vancouver, ISO, and other styles

29

Grove, David D. "Putting Cholesterol Control on a Diet." JAMA: The Journal of the American Medical Association 262, no. 21 (December 1, 1989): 2998. http://dx.doi.org/10.1001/jama.1989.03430210036020.

Full text

APA, Harvard, Vancouver, ISO, and other styles

30

Klipsic, Devon, Danilo Landrock, Gregory G. Martin, Avery L. McIntosh, Kerstin K. Landrock, John T. Mackie, Friedhelm Schroeder, and Ann B. Kier. "Impact of SCP-2/SCP-x gene ablation and dietary cholesterol on hepatic lipid accumulation." American Journal of Physiology-Gastrointestinal and Liver Physiology 309, no. 5 (September 1, 2015): G387—G399. http://dx.doi.org/10.1152/ajpgi.00460.2014.

Full text

Abstract:

While a high-cholesterol diet induces hepatic steatosis, the role of intracellular sterol carrier protein-2/sterol carrier protein-x (SCP-2/SCP-x) proteins is unknown. We hypothesized that ablating SCP-2/SCP-x [double knockout (DKO)] would impact hepatic lipids (cholesterol and cholesteryl ester), especially in high-cholesterol-fed mice. DKO did not alter food consumption, and body weight (BW) gain decreased especially in females, concomitant with hepatic steatosis in females and less so in males. DKO-induced steatosis in control-fed wild-type (WT) mice was associated with 1) loss of SCP-2; 2)

APA, Harvard, Vancouver, ISO, and other styles

31

Chang, Chen-Kang, and Jean T. Snook. "The cholesterolaemic effects of dietary fats in cholesteryl ester transfer protein transgenic mice." British Journal of Nutrition 85, no. 6 (June 2001): 643–48. http://dx.doi.org/10.1079/bjn2001320.

Full text

Abstract:

In order to investigate the role of cholesteryl ester transfer protein (CETP) in the cholesterolaemic response to dietary fats, we analysed plasma lipid profiles of CETP-transgenic and control C57BL/6 mice fed standard chow (AIN-93G; AIN), a low-fat diet, and diets high in butter (saturated fatty acids; SFA), high-oleic acid safflower oil (monounsaturated fatty acids; MUFA), and safflower oil (polyunsaturated fatty acids; PUFA) for 5 weeks. Each group contained four or five mice. There were significant diet and diet×genotype effects on plasma total cholesterol (TC; P = 0·035 and P = 0·008 resp

APA, Harvard, Vancouver, ISO, and other styles

32

Neary, Richard H., Mark D. Kilby, Padma Kumpatula, Francis L. Game, Deepak Bhatnagar, Paul N. Durrington, and P. M. Shaughn O'Brien. "Fetal and Maternal Lipoprotein Metabolism in Human Pregnancy." Clinical Science 88, no. 3 (March 1, 1995): 311–18. http://dx.doi.org/10.1042/cs0880311.

Full text

Abstract:

1. Lipid, apolipoprotein concentration and composition were determined in maternal venous and umbilical arterial and venous blood at delivery by elective Caesarean section in 13 full-term pregnancies and in 25 healthy non-pregnant females. The indications of Caesarean section were a previous Caesarean section or breech presentation. None of the women was in labour and there were no other complications of pregnancy or fetal distress. 2. The objectives of the study were to establish whether the placenta has a role in feto-maternal cholesterol metabolism through either synthesis or transplacental

APA, Harvard, Vancouver, ISO, and other styles

33

NAPOLITANO, Mariarosaria, Kelly V. BATT, Michael AVELLA, Elena BRAVO, and Kathleen M. BOTHAM. "Lipid synthesis in macrophages derived from the human cell line THP-1: modulation of the effects of native and oxidized chylomicron-remnant-like particles by oestrogen." Clinical Science 101, no. 4 (September 14, 2001): 403–13. http://dx.doi.org/10.1042/cs1010403.

Full text

Abstract:

The effects of native and oxidized chylomicron remnants on the synthesis of cholesteryl ester and triacylglycerol in macrophages, and the way that this is influenced by exposure of the cells to oestrogen, was investigated using the human monocyte cell line THP-1 and chylomicron-remnant-like particles containing human apolipoprotein E (CRLPs). Synthesis of the lipids was measured by the incorporation of [3H]oleate into cholesteryl ester and triacylglycerol. CRLPs (5-40μg of cholesterol/ml) containing either trilinolein or triolein as the triacylglycerol component caused a dose-dependent decreas

APA, Harvard, Vancouver, ISO, and other styles

34

Vecka, Marek, Magdalena Dušejovská, Barbora Staňková, Ivan Rychlík, and Aleš Žák. "A Matched Case-Control Study of Noncholesterol Sterols and Fatty Acids in Chronic Hemodialysis Patients." Metabolites 11, no. 11 (November 12, 2021): 774. http://dx.doi.org/10.3390/metabo11110774.

Full text

Abstract:

Dyslipidemia is common among patients on hemodialysis, but its etiology is not fully understood. Although changes in cholesterol homeostasis and fatty acid metabolism play an important role during dialysis, the interaction of these metabolic pathways has yet to be studied in sufficient detail. In this study, we enrolled 26 patients on maintenance hemodialysis treatment (high-volume hemodiafiltration, HV HDF) without statin therapy (17 men/9 women) and an age/gender-matched group of 26 individuals without signs of nephropathy. The HV-HDF group exhibited more frequent signs of cardiovascular dis

APA, Harvard, Vancouver, ISO, and other styles

35

Lucić, A., V. Bradamante, M. Peraica, B. Radić, A.-M. Domijan, and R. Fuchs. "Changes in plasma lipids after a non-lethal dose of cycloheximide in rats." Human & Experimental Toxicology 22, no. 5 (May 2003): 245–48. http://dx.doi.org/10.1191/0960327103ht355oa.

Full text

Abstract:

This paper describes a study of the effect of a single intraperitoneal non-lethal dose of cycloheximide (CHM; 2.0 mg/kg body weight) on the concentration of plasma lipids and lipoproteins in male rats killed one, two, three, four and nine days after receiving the dose. The concentration of triglycerides, total cholesterol, high-density lipoproteins (HDL)-cholesterol and low-density lipoproteins (LDL)-cholesterol was measured in treated and control animals. The effect of CHM on the concentration of triglycerides, total cholesterol, HDL-cholesterol, and LDL-cholesterol was visible in rat plasma

APA, Harvard, Vancouver, ISO, and other styles

36

Feingold, K. R., and A. H. Moser. "Effect of lactation on cholesterol synthesis in rats." American Journal of Physiology-Gastrointestinal and Liver Physiology 249, no. 2 (August 1, 1985): G203—G208. http://dx.doi.org/10.1152/ajpgi.1985.249.2.g203.

Full text

Abstract:

Lactation induces a variety of morphological and functional changes in the gastrointestinal tract. In the present study we employed tritiated water as the substrate to demonstrate that in the intact rat lactation results in a twofold increase in cholesterol synthesis in the small intestine. Feeding a high-cholesterol diet did not markedly inhibit small intestinal cholesterol synthesis in either control or lactating animals, and the difference in cholesterol synthesis between the two groups persisted. In the large intestine, cholesterol synthesis is increased threefold in the lactating animals,

APA, Harvard, Vancouver, ISO, and other styles

37

Singh, Pravin Kumar. "Hyperlipidemia: Etiology and Possible Control Through Homoeopathic Remedies." International Journal of Advanced Ayurveda, Yoga, Unani, Siddha and Homeopathy 11, no. 1 (May 26, 2022): 696–700. http://dx.doi.org/10.23953/cloud.ijaayush.518.

Full text

Abstract:

Hyperlipidemia is a condition characterized by an elevation of any or all lipid profile and/or lipoproteins in the blood. Hyperlipidemia is the most important atherosclerotic risk factor. Review of population-based studies in India shows increasing mean total cholesterol levels. Recent studies have reported that high cholesterol is present in 25–30% of urban and 15–20% rural subjects. This prevalence is lower than high-income countries. The most common Hyperlipidemia in India are borderline high LDL cholesterol, low HDL cholesterol and high triglycerides. Studies have reported that over a 20-y

APA, Harvard, Vancouver, ISO, and other styles

38

Reboulleau, Anne, Véronique Robert, Benoît Vedie, Aline Doublet, Alain Grynberg, Jean-Louis Paul, and Natalie Fournier. "Involvement of cholesterol efflux pathway in the control of cardiomyocytes cholesterol homeostasis." Journal of Molecular and Cellular Cardiology 53, no. 2 (August 2012): 196–205. http://dx.doi.org/10.1016/j.yjmcc.2012.05.015.

Full text

APA, Harvard, Vancouver, ISO, and other styles

39

Terlingen, J. B., H. J. van Dreumel, A. van Heiningen, G. J. Boerma, and J. C. Koedam. "Improved preparation of cholesterol calibration and control sera." Clinical Chemistry 31, no. 7 (July 1, 1985): 1201–3. http://dx.doi.org/10.1093/clinchem/31.7.1201.

Full text

Abstract:

Abstract We prepared several serum batches with various cholesterol concentrations, to be used as calibrators and controls in a proficiency testing program of an organization in The Netherlands that is in charge of the standardization of cholesterol determinations for epidemiological purposes. The sera were of human origin, to avoid abnormal matrix effects. To decrease the cholesterol content in some samples, we adsorbed them onto colloidal silicic acid. To increase it, we added lipoproteins that had been precipitated from human serum with heparin and calcium ions. The precipitation method we

APA, Harvard, Vancouver, ISO, and other styles

40

Gudbrandsen, Oddrun A., Hege Wergedahl, Bjørn Liaset, Marit Espe, and Rolf K. Berge. "Dietary proteins with high isoflavone content or low methionine–glycine and lysine–arginine ratios are hypocholesterolaemic and lower the plasma homocysteine level in male Zucker fa/fa rats." British Journal of Nutrition 94, no. 3 (September 2005): 321–30. http://dx.doi.org/10.1079/bjn20051496.

Full text

Abstract:

It has previously been demonstrated that soya protein, which contains isoflavones and low methionine–glycine and lysine–arginine ratios, has a hypocholesterolaemic effect. In the present study, the hypocholesterolaemic effects of an isoflavone-enriched casein diet (HDI) and a single-cell protein-based diet (SCP) devoid of isoflavones but with low methionine–glycine and lysine–arginine ratios were investigated in obese Zucker rats after 6 weeks of feeding. The control diet contained casein, which has high ratios of methionine–glycine and lysine–arginine. HDI and SCP feeding reduced the concentr

APA, Harvard, Vancouver, ISO, and other styles

41

Rymaszewski, Z., R. L. Yunker, M. Ashraf, M. Park, and M. T. Subbiah. "Regulation of cholesterol metabolism in fetal rabbit aorta: role of amniotic fluid factors." American Journal of Physiology-Heart and Circulatory Physiology 255, no. 1 (July 1, 1988): H160—H168. http://dx.doi.org/10.1152/ajpheart.1988.255.1.h160.

Full text

Abstract:

This study shows that amniotic fluid enhances cholesterol esterification in arterial wall, as measured by in vitro assay of acyl-CoA:cholesterol acyltransferase (ACAT) activity and by incorporation of oleic acid to cholesteryl esters in cultured fetal aortas and smooth muscle cells. This property is mostly evident in the fraction of molecular weight greater than 100,000, and it is abolished by delipidation, indicating that stimulating factor is probably lipoprotein in nature. Despite an increased cholesterol esterification by the presence of amniotic fluid in medium of cultured fetal aortas, t

APA, Harvard, Vancouver, ISO, and other styles

42

Zhang, Hanrui, Jianting Shi, Melanie A. Hachet, Chenyi Xue, Robert C. Bauer, Hongfeng Jiang, Wenjun Li, et al. "CRISPR/Cas9-Mediated Gene Editing in Human iPSC-Derived Macrophage Reveals Lysosomal Acid Lipase Function in Human Macrophages—Brief Report." Arteriosclerosis, Thrombosis, and Vascular Biology 37, no. 11 (November 2017): 2156–60. http://dx.doi.org/10.1161/atvbaha.117.310023.

Full text

Abstract:

Objective— To gain mechanistic insights into the role of LIPA (lipase A), the gene encoding LAL (lysosomal acid lipase) protein, in human macrophages. Approach and Results— We used CRISPR (clustered regularly interspaced short palindromic repeats)/Cas9 (CRISPR–associated protein 9) technology to knock out LIPA in human induced pluripotent stem cells and then differentiate to macrophage (human-induced pluripotent stem cells–derived macrophage [IPSDM]) to explore the human macrophage LIPA loss-of-function phenotypes. LIPA was abundantly expressed in monocyte-derived macrophages and was markedly

APA, Harvard, Vancouver, ISO, and other styles

43

Talamillo, Ana, Leiore Ajuria, Marco Grillo, Orhi Barroso-Gomila, Ugo Mayor, and Rosa Barrio. "SUMOylation in the control of cholesterol homeostasis." Open Biology 10, no. 5 (May 2020): 200054. http://dx.doi.org/10.1098/rsob.200054.

Full text

Abstract:

SUMOylation—protein modification by the small ubiquitin-related modifier (SUMO)—affects several cellular processes by modulating the activity, stability, interactions or subcellular localization of a variety of substrates. SUMO modification is involved in most cellular processes required for the maintenance of metabolic homeostasis. Cholesterol is one of the main lipids required to preserve the correct cellular function, contributing to the composition of the plasma membrane and participating in transmembrane receptor signalling. Besides these functions, cholesterol is required for the synthes

APA, Harvard, Vancouver, ISO, and other styles

44

Safavi, Seyyed Morteza, Rahele Ziaei, and Mohammad Reza Maracy. "Association of Serum Ceruloplasmin Level with Obesity: Some Components of Metabolic Syndrome and High-Sensitive C-Reactive Protein in Iran." Journal of Obesity 2012 (2012): 1–5. http://dx.doi.org/10.1155/2012/951093.

Full text

Abstract:

Background. One of the mechanisms that has been suggested for obesity related metabolic disturbances is obesity-induced inflammation. Pro-inflammatory cytokines generated in adipose tissue can increase hepatic synthesis of inflammation-sensitive plasma proteins (ISPs) including ceruloplasmin (Cp). In this study we aimed to investigate the relation between serum Cp level and obesity.Methods. 61 persons with body mass index (BMI) ≥ 25 kg/m2(case group) and 61 persons with BMI < 25 kg/m2(control group) were included in this study with a case-control design. Serum Cp levels, triglyceride level,

APA, Harvard, Vancouver, ISO, and other styles

45

Garg, M. L., and J. R. Sabine. "Homoeostatic control of membrane cholesterol and fatty acid metabolism in the rat liver." Biochemical Journal 251, no. 1 (April 1, 1988): 11–16. http://dx.doi.org/10.1042/bj2510011.

Full text

Abstract:

Experiments were designed to assess the effect of cholesterol feeding, with or without high levels of either saturated (coconut oil) or unsaturated (sunflower-seed oil) fat on the fatty acid composition of hepatic microsomal membrane lipids, as well as on the activities of several membrane-bound enzymes of cholesterol synthesis and metabolism. Administration of 2% (w/w) cholesterol in the rat diet inhibited hydroxymethylglutaryl-CoA reductase activity, and this inhibition was much more pronounced when cholesterol was fed in combination with unsaturated rather than with saturated fat. Cholester

APA, Harvard, Vancouver, ISO, and other styles

46

Caudill, Samuel P., Gerald R. Cooper, S. Jay Smith, and Gary L. Myers. "Assessment of current National Cholesterol Education Program guidelines for total cholesterol, triglyceride, HDL-cholesterol, and LDL-cholesterol measurements." Clinical Chemistry 44, no. 8 (August 1, 1998): 1650–58. http://dx.doi.org/10.1093/clinchem/44.8.1650.

Full text

Abstract:

Abstract We examine the effect of systematic bias and random error, quality control, and intraperson biological variation on the National Cholesterol Education Program (NCEP) clinical classifications for reported lipid measurements. We consider misclassification to occur if a true lipid homeostatic set point is within a desirable range but the reported lipid value is in a high-risk range, or if a true lipid homeostatic set point is in a high-risk range but the reported lipid value is in a desirable range. To evaluate the overall adequacy of the NCEP guidelines to ensure correct patient classif

APA, Harvard, Vancouver, ISO, and other styles

47

Franzini, C., and P. Luraschi. "Commutability of control materials in cholesterol measurement." Scandinavian Journal of Clinical and Laboratory Investigation 53, no. 1 (February 1, 1993): 51–55. http://dx.doi.org/10.3109/00365519309092531.

Full text

APA, Harvard, Vancouver, ISO, and other styles

48

Franzini, C., and P. Luraschi. "Commutability of control materials in cholesterol measurement." Scandinavian Journal of Clinical and Laboratory Investigation 53, no. 1 (January 1993): 51–55. http://dx.doi.org/10.1080/00365519309092531.

Full text

APA, Harvard, Vancouver, ISO, and other styles

49

MINEGISHI, YOSHIHIKO. "Tissue-specificity of cholesterol biosynthesis control mechanism." Kagaku To Seibutsu 41, no. 6 (2003): 375–77. http://dx.doi.org/10.1271/kagakutoseibutsu1962.41.375.

Full text

APA, Harvard, Vancouver, ISO, and other styles

50

Passey, Sarah. "Cholesterol control might help treat Alzheimer's disease." Lancet Neurology 3, no. 12 (December 2004): 700. http://dx.doi.org/10.1016/s1474-4422(04)00923-8.

Full text

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!