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1

Ezio, Giacobini, ed. Cholinesterases and cholinesterase inhibitors. Martin Dunitz, 2000.

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2

Yau, Kenneth Kwok Chi. Assessing and predicting treatment responses to cholinesterase inhibitor pharmacotherapy in Alzheimer's disease. National Library of Canada, 2000.

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3

International Meeting on Cholinesterases (3rd 1990 La Grande Motte, France). Cholinesterases: Structure, function, mechanism, genetics, and cell biology. American Chemical Society, 1991.

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4

1935-, Zakut Haim, ed. Human cholinesterases and anticholinesterases. Academic Press, 1993.

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5

Moralev, Serge N. Comparative enzymology of cholinesterases. International University Line, 2007.

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6

(Australia), Materials Research Laboratories, ed. Inhibition of EEL acetylcholinesterase by nerve agents: A stopped-flow study. Dept. of Defence, Materials Research Laboratories, 1985.

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7

Ezio, Giacobini, and Becker Robert E, eds. Current research in Alzheimer therapy: Cholinesterase inhibitors. Taylor & Francis, 1988.

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8

Todd, G. Daniel. Draft toxicological profile for diazinon. U.S. Department of Health and Human Services, Public Health Service, Agency for Toxic Substances and Disease Registry, 2006.

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9

Roney, Nickolette. Draft toxicological profile for guthion. U.S. Department of Health and Human Services, Public Health Service, Agency for Toxic Substances and Disease Registry, 2006.

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10

National Institute for Clinical Excellence (Great Britain). Guidance on the use of donepezil, rivastigmine and galantamine for the treatment of Alzheimer's disease. National Institute for Clinical Excellence, 2001.

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11

E, Chambers Janice, and Levi Patricia E, eds. Organophosphates: Chemistry, fate, and effects. Academic Press, 1992.

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12

Bryan, Ballantyne, and Marrs Timothy C, eds. Clinical and experimental toxicology of organophosphates and carbamates. Butterworth Heinemann, 1992.

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13

E, Becker Robert, and Giacobini Ezio, eds. Alzheimer disease: From molecular biology to therapy. Birkhäuser, 1997.

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14

United States. Dept. of the Army., ed. Assay techniques for detection of exposure to sulfur mustard, cholinesterase inhibitors, Sarin, Soman, GF, and cyanide. Headquarters, Dept. of the Army, 1996.

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15

L, Reid John, ed. Hypertension-drug therapy of tomorrow-calcium antagonists/ACE inhibitors combination as first-line treatment?: An international debate. Raven Health Care Communications, 1992.

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16

Parker, James N., and Philip M. Parker. Donepezil: A medical dictionary, bibliography, and annotated research guide to Internet references. ICON Health Publications, 2004.

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17

United States. Agency for Toxic Substances and Disease Registry, ed. Toxicological profile for methyl parathion. Agency for Toxic Substances and Disease Registry, 2001.

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18

E, Becker Robert, and Giacobini Ezio, eds. Cholinergic basis for Alzheimer therapy. Birkhäuser, 1991.

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19

Takao, Kumazawa, Kruger Lawrence, and Mizumura Kazue, eds. The polymodal receptor: A gateway to pathological pain. Elsevier, 1996.

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20

(Editor), Jean Massoulie, Eric Barnard (Editor), Arnaud Chatonnet (Editor), Francis Bacou (Editor), Bhupendra P. Doctor (Editor), and Daniel M. Quinn (Editor), eds. Cholinesterases: Structure, Function, Mechanism, Genetics, and Cell Biology (Conference Proceedings Series). An American Chemical Society Publication, 1998.

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21

Giacobini, Ezio. Cholinesterases and Cholinesterase Inhibitors: Basic Preclinical and Clinical Aspects. Informa Healthcare, 2000.

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22

Kitay, Brandon M., and Rajesh R. Tampi. Memantine in Patients with Moderate to Severe Alzheimer’s Disease Already Receiving Donepezil. Edited by Ish P. Bhalla, Rajesh R. Tampi, Vinod H. Srihari, and Michael E. Hochman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190625085.003.0018.

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This chapter provides a summary of a landmark study on the pharmacological management of cognitive disorders. In patients with moderate to severe Alzheimer disease treated with a cholinesterase inhibitor (donepezil), is the addition of a N-methyl-D-aspartate receptor inhibitor (memantine) a safe and efficacious augmentation strategy? Starting with that question, it describes the basics of the study, including funding, study location, who was studied, how many patients, study design, study intervention, follow-up, endpoints, results, and criticism and limitations. The chapter briefly reviews ot
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23

Serge N. Moralev and Eugene V. Rozengart. Comparative enzymology of cholinesterases. International University Line, 2007.

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24

Cholinesterase Inhibitors in Alzheimer's Disease. Lippincott Williams & Wilkins, 1999.

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25

Hopkins, Philip M. Adverse drug reactions in anaesthesia. Edited by Michel M. R. F. Struys. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0022.

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Adverse drug reactions are implicated in more than 40% of anaesthesia-related deaths. Undoubtedly, many more patients experience morbidity from adverse drug reactions. Widely cited definitions of adverse drug reactions encompass common side-effects but this chapter focuses on those that are unexpected reactions to drugs administered by anaesthetists and that occur at normal drug doses. The chapter includes a comprehensive account of malignant hyperthermia, which remains a major contributor to anaesthesia related to deaths. Malignant hyperthermia is a pharmacogenetic condition triggered by pote
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26

Ramesh C. Gupta, PhD, DABT, FACT, FATS. Toxicology of Organophosphate & Carbamate Compounds. Academic Press, 2005.

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27

Toxicology of Organophosphate & Carbamate Compounds. Academic Press, 2005.

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28

Giacobini, Ezio. Cholinesterase Inhibitors: Their Role in the Therapeutic Management of Alzheimer's Disease Pocketbook (Medical Pocketbooks). MARTIN DUNITZ, 2001.

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29

Halwe, Sandro. Versuchsprotokoll - Wirkung Von Cholinesterasen und Deren Inhibition Durch Neostigmin. GRIN Verlag GmbH, 2011.

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30

Cholinesterase inhibitors for Alzheimer's disease: A systematic review of randomized controlled trials. Canadian Coordinating Office for Health Technology Assessment, 2005.

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31

(Editor), Janice E. Chambers, and Patricia E. Levi (Editor), eds. Organophosphates: Chemistry, Fate, and Effects. Academic Press, 1992.

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32

(Editor), Janice E. Chambers, and Patricia E. Levi (Editor), eds. Organophosphates: Chemistry, Fate, and Effects. Academic Press, 1992.

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33

Ballantyne, Bryan, and Timothy C. Marrs. Clinical and Experimental Toxicology of Organophosphates and Carbamates. Elsevier Science & Technology Books, 2017.

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34

Behl, Pearl. Differential long-term cognitive, functional, and behavioral effects of cholinesterase inhibitors in Alzheimer's disease. 2006.

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35

Word for Windows Made Simple (Computing Made Simple). Made Simple, 1994.

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36

Gill, Sudeep Singh. Patterns of use and adverse events associated with cholinesterase inhibitors in older adults with dementia. 2004.

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37

Becker, Robert E., and Ezio Giacobini. Cholinergic Basis for Alzheimer Therapy. Birkhauser, 2013.

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38

Becker, Robert E., and Ezio Giacobini. Cholinergic Basis for Alzheimer Therapy. Birkhauser Verlag, 2013.

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39

Becher, Paul G. Insecticidal and cholinesterase-inhibitory bioactivity of indole alkaloids from benthic freshwater cyanobacteria of the genera Fischerella and Nostoc. 2006.

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40

Tousi, Babak. Cognitive Enhancement in Non-Alzheimer’s Dementias. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190214401.003.0004.

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Cognitive enhancement in non-Alzheimer’s dementias has not been studied as extensively as that in Alzheimer’s dementia. This chapter reviews the research on cognitive enhancement for three types of dementia: vascular dementia, dementia with Lewy bodies, and frontal lobe dementia. The chapter reviews both pharmacological and nonpharmacological approaches for treatment of dementia. The focus is on randomized controlled trials for currently available medications, specifically cholinesterase inhibitors and memantine. Major advances in physical and cognitive rehabilitation during the past decade ha
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41

Bajgar, Jiri. Nerve Agents Poisoning and Its Treatment in Schematic Figures and Tables. Elsevier, 2012.

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42

Bajgar, Jiri. Nerve Agents Poisoning and Its Treatment in Schematic Figures and Tables. Elsevier, 2012.

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43

Nerve Agents Poisoning and Its Treatment in Schematic Figures and Tables. Elsevier Science & Technology Books, 2012.

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44

Carlock, Linda L. Blood and brain cholinesterase inhibition and associated behaviors from exposure to phosphorodithioate organophosphorus pesticides: Implications for monitoring avian wildlife. 1992.

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45

Cholinergic Basis for Alzheimer Therapy. Birkhäuser Boston, 2014.

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46

Cummings, Jeffrey L., and Kate Zhong. Clinical Trials and Drug Development in Neurodegenerative Diseases. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190233563.003.0018.

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This chapter describes the common therapeutic targets, approaches to clinical trial design, biomarkers, and therapeutic interventions across neurodegenerative disorders (NDDs). Each unique NDD-Alzheimer’s disease (AD), Parkinson’s disease (PD), amyotrophic lateral sclerosis (ALS), etc.-has a unique phenotype associated with the regional cell population most affected. Each disease, however, is associated with protein misfolding, oxidation, inflammation, apoptosis, and cell death. If vulnerable cell populations include transmitter source nuclei, transmitter deficits also emerge (e.g. cholinergic
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47

Kuwabara, Satoshi. Neuromuscular junction disorders. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199658602.003.0014.

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Ten seminal papers on disorders of the neuromuscular junction are described, covering historical aspects, recent advances in immunological, biological, and genetic researches, and future perspectives. Early descriptions of myasthenia gravis (MG) date back to the seventeenth century, and MG acquired its name in the nineteenth century. The first symptomatic treatment with cholinesterase inhibitors was reported in 1934, leading to the development of modern immunological therapies. Following the discovery of anti-MuSK (muscle-specific tyrosine kinase) antibody in 2001, MG is currently classified i
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48

(Editor), T. Kumazawa, L. Kruger (Editor), and K. Mizumura (Editor), eds. The Polymodal Receptor - A Gateway to Pathological Pain (Progress in Brain Research). Elsevier Science, 1996.

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