Relevant bibliographies by topics / Chronic gastritis with hyperacidity

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Academic literature on the topic 'Chronic gastritis with hyperacidity'

Author: Grafiati
Published: 5 June 2025
Last updated: 16 July 2025

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Journal articles on the topic "Chronic gastritis with hyperacidity"

1

Golubkina, E. V., V. M. Sorokin, A. P. Umerova, and N. V. Kamneva. "Acid formation in gastroenterological patients with colonization of the stomach with virulent and non-virulent strains of helicobacter." Experimental and Clinical Gastroenterology, no. 9 (March 24, 2020): 32–37. http://dx.doi.org/10.31146/1682-8658-ecg-169-9-32-37.

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Objective. To prove that increased acid production in patients with gastric ulcer and duodenal ulcer is associated with the impact of the virulent strains of Helicobacter pylori but due not to the persistence of non-virulent strains.Materials and methods. Patients with active gastroduodenal ulcer and patients with active chronic pancreatitis accompanied by the gastritis were compared in the respect of the level of pH in the antrum and corpus gastricum, as well as Helicobacter pylori virulence according to the presence cagA gene, especially in combination with vacA allele s1 / m1 (if any of the strains were found in gastric biopsy specimen).Results. In patients with gastric ulcer the average values of pH were significantly lower, both in the antrum and corpus gastricum, than in patients with chronic pancreatitis accompanied by gastritis. Helicobacter pylori strains were found only in half of the patients, either in the gastric ulcer group or in the group of chronic pancreatitis accompanied by the gastritis. Significant difference was revealed after virulent genes identification: virulent strains prevailed in patients with gastric ulcer and in contrast to the prevalence of non-virulent strains in patients with chronic pancreatitis accompanied by the gastritis (Mann-Whitney test, p = 0.001). Since there is no available data that Helicobacter has an affinity for a highly acidic medium in comparison with moderately acidic medium, it is concluded that just primary colonization of the stomach with virulent strains results in hyperacidity (as the consequence of cytotoxicity) and that persistence of non-virulent strains hardly effects hyperacidity.
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Zherebak, Nadiia, and Olena Gnatko. "COURSE OF THREATENED ABORTION IN CHRONIC GASTRITIS OF DIFFERENT TYPES." Ukrainian Scientific Medical Youth Journal 115, no. 1 (2020): 53–62. http://dx.doi.org/10.32345/usmyj.1(115).2020.53-62.

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Abortion is one of the most important medical and social problems in the world. Functional and morphological sexual changes associated with the normal fetal growth and development during physiological pregnancy involve other maternal organs and systems to varying extent, and provide changes in metabolic and homeostasis processes in new conditions to preserve pregnancy. Gastrointestinal motility disorders can be a leading pathogenetic factor that contributes to the development of many common gastrointestinal disorders. During pregnancy, decreased intestinal motility may play a pathogenetic role in pregnancy as a factor leading to decreased intestinal motor and evacuation function and negative impact on pregnancy. The aim of the study was to determine the clinical course of threatened abortion in early gestation in women with different types of chronic gastritis to optimize the management of pregnancy. 67 pregnant women with threatened abortion at 8-12 weeks of gestation were examined. They were divided into 2 groups: Group 1 - 35(52.2%) women with chronic gastritis and Group 2 - 32(47.8%) patients without gastritis. At the beginning of pregnancy, all women with chronic gastritis had remission. A comparative assessment of the frequency and severity of manifestations of this complication at an early stage on the leading clinical signs: pain, bleeding, retrochorial hematoma in combination with major (vomiting, constipation) motor-evacuation function of the gastrointestinal tract in different types of gastritis was performed to determine the role of motor and evacuation function of the gastrointestinal tract in the development of the risk of abortion in pregnant women with gastritis. The type of gastritis was confirmed according to the level of basal secretion of serum gastrin 17 (G-17) with the stimulation test. The obtained results were processed by mathematical and statistical analysis methods with determination of average values ​​(M±m), Student's test and confidence index (statistically significant difference was considered as р˂0,05).
 Results. All women were of reproductive age, the average age of pregnant women in groups had no statistical difference. Analysis of the frequency of clinical manifestations of threatened abortion showed that 35(100%) had pain symptoms, 30(85.7%) had bloody discharge and 10(28.6%) had retrochorial hematoma in Group 1, and 32(100%), 12(37.5%), 5(15.6%), respectively, in Group 2. Assessment of the intensity of threatened abortion symptoms showed that moderate pain was most often observed in all types of gastritis. Mild symptoms were in normoacidic women of Group 1 (11.4%) and Group 2 (9.4%). 2.9% of hyperacidic pregnant women in Group 1 and 3.1% of hypoacidic women in Group 2 reported severe pain. Analysis of the bleeding nature and intensity showed that women in Group 1 with all types of gastritis had spotting: 11.4% of hyperacidic type, 20.0% of normoacidic type and 11.4% of hypoacidic type. Moderate bleeding was observed in different types of chronic gastritis in pregnant women of Group 1 and various acidic conditions in Group 2. Moderate bleeding was significantly more common (7.4-fold) in hyperacidic pregnant women of Group 1 compared with Group 2. Retrochorial hematomas were registered in Group 1 as 14.3% in hyperacidic patients, 5.7% in normoacidic patients and 8.6% in hypoacidic women. Retrochorial hematomas were 2.5-fold more common in hyperacidic gastritis compared with normoacidic and 1.7-fold more common in hypoacidic gastritis. Estimation of the frequency of combined clinical manifestations of functional gastrointestinal diseases in pregnant women with threatened abortion showed that the manifestations of threatened abortion only were observed in 65.6% without gastritis, and only in 5.7% with gastritis. Different variants of combined clinical manifestations of gastrointestinal functional disorders and threatened abortion significantly prevailed in women with chronic gastritis: threatened abortion and vomiting were 3-fold more common, threatened abortion and vomiting and constipation - 4.3-fold, threatened abortion and constipation - 1.6-fold. The combination of threatened abortion and vomiting with various acidity was most common in hyperacidic condition (2,7-fold more common in Group 1 than in Group 2). The variant of the combination of threatened abortion, vomiting and constipation in hyperacid state was 3.6 times more common in Group 1 than in Group 2. Threatened abortion with constipation was registered in 17.1% of women in Group 1 with hyperacidity, which was 2.7 times more often than in women of Group 2. The most common clinical manifestations of threatened abortion and functional gastrointestinal disorders were seen in pregnant women with chronic hyperacidic gastritis.
 Conclusion. Dysfunction of the motor and evacuation gastrointestinal activity is important in the clinical course of threatened abortion based on the type of chronic gastritis, mediated by the different acid-producing gastric function which indicates the need to optimize treatment of threatened abortion in pregnant women with different types of chronic gastritis.
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Babinets, L. S., and I. V. Makhnitska. "Role of basal-topographic pH-metry of stomach in management of patients with chronic recurrent pancreatitis in case of comorbidity." Modern Gastroenterology, no. 1 (March 26, 2025): 22–27. https://doi.org/10.30978/mg-2025-1-22.

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The comorbidity of chronic recurrent pancreatitis (CRP) and chronic gastritis (CG) and/or chronic duodenitis (CD) associated with Helicobacter pylori remains poorly understood. The progression of such comorbidity affects the clinical symptoms, is accompanied by morphological changes of the mucous membrane, a violation of the acid‑producing function of the stomach. In the prism of studying the influence of gastroduodenal disorders on CRP, it is relevant to perform basal‑topographic express‑pH‑metry, which makes it possible to determine the state of acidity in the stomach, as well as the possibility of providing the patient with the fastest and most appropriate differentiated treatment (treatment selection and control). Objective — to determine the state of basal‑topographic acidity of the stomach in patients with isolated CRP, as well as in its comorbidity with CG and/or CD associated with H. pylori, in order to optimize the tactics of management of such patients in clinical practice. Materials and methods. The study included 147 patients registered as out‑patients and under the supervision of a family doctor in the Center for Primary Health Care. The main group consisted of 117 patients with CRP in combination with CG and/or CD associated with H. pylori and 30 patients with isolated CRP. All patients underwent basal‑topographic pH‑metry along the course of the stomach. The results were evaluated using the proposed functional intervals (FI) of pH (FI pH) from 0 to 5 (in the direction of increasing gastric acidity) and unimodal analysis of One‑way ANOVA followed by post‑hoc Tukey HSD test. Results. The following ranking of the distribution of measurement points in patients with CRP in the intervals of basal‑topographic express pH‑metry was revealed, starting with the highest (р <0.05): 3 – 2 >4 – 5 – 1, which indicates the predominance of normacidity with a tendency to moderate hypoacidity, and in patients with CRP and CG and/or CD associated with H. pylori — 4 >5 >3 >2 – 1, (р <0.05), which indicates moderate hyperacidity with a tendency to pronounced hyperacidity. Conclusions. Moderate hyperacidity with a tendency to pronounced hyperacidity has been proven in patients with comorbidity of CRP and CG and/or CD associated with H. pylori, according to the revealed rating of the distribution of measurement points in the intervals of basal‑topographic express pH‑metry, which requires mandatory correction.
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Goldstein, Richard E., Stanley L. Marks, Philip H. Kass, and Larry D. Cowgill. "Gastrin concentrations in plasma of cats with chronic renal failure." Journal of the American Veterinary Medical Association 213, no. 6 (1998): 826–28. http://dx.doi.org/10.2460/javma.1998.213.06.826.

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Objective— To determine the prevalence of hypergastrinemia in cats with naturally developing chronic renal failure (CRF) and the correlation between gastrin concentration in plasma and severity of CRF. Design— Cohort study. Animals— 30 cats with naturally developing CRF and 12 clinically normal control cats. Procedure— Gastrin concentrations in plasma were determined by double-antibody radioimmunoassay of blood samples obtained from cats after food was withheld 8 hours. Concentrations were compared, using a nonparametric Kruskal-Wallis ANOVA. Results— 18 cats with CRF had high gastrin concentrations (median, 45 pg/ml; range, < 18 to > 1,333 pg/ml), compared with those for control cats (< 18 pg/ml). Prevalence of hypergastrinemia increased with severity of renal insufficiency. Three of 9 cats with mild CRF, 6 of 11 cats with moderate CRF, and 9 of 10 cats with severe CRF had high gastrin concentrations. Gastrin concentrations were significantly different between control cats and cats with CRF, regardless of disease severity. Clinical Implications— The potential role of high concentrations of gastrin on gastric hyperacidity, uremic gastritis, bleeding from the gastrointestinal tract, and associated clinical signs of hypergastrinemia (eg, anorexia and vomiting) may justify use of histamine2-receptor antagonists or proton pump inhibitors to suppress gastric acid secretion in cats with CRF that have these clinical signs. (J Am Vet Med Assoc 1998;213:826-828)
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Thakur, Balaji, Priyanka Kumari, and Bhanu Pratap Singh. "A CONCEPTUAL STUDY ON THE EFFECT OF NARIKELA KHANDA IN AMLAPITTA." International Ayurvedic Medical Journal 9, no. 9 (2021): 2073–77. http://dx.doi.org/10.46607/iamj2209092021.

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Nowadays due to unawareness about Prakriti (psychosomatic constitution) people are practising inappropriate diet and lifestyle which leads to disturbances in the digestive system. The core principles in Ayurveda give prime im- portance to Agni, Prakriti, Ahara (food) and Vihara (lifestyle) in maintaining health. In the present era, people are scheduled to one or the other works due to which they are following unwholesome food and habits which may lead to the manifestation of many diseases. One of the common disorders which troubles a person a lot is Amlapitta. It is composed of the word Amla and Pitta. Amlapitta is a very common disease caused by Vidagdha Pitta with features like Amlaudgara, Tiktaudgara, Hrud Kantha Daha etc. It is the GI disorder described in Ayurvedic texts that closely resembles Gastritis in modern science. In the chronic stage, it may lead to ulcerative colitis. In modern medical science, they give some anta-acids, H2 blockers, Proton pump inhibitors, some steroids which have high prevalence costs and side effects. Today there is a need for a potent herbal formulation that can cure hyperacidity and ulcers. The present study is being undertaken to approach the disease Amlapitta through the formulation of Narikela Khanda. Keywords: Ayurveda, Amlapitta, Narikela Khanda.
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Kerzyk, Oksana P., and Mykola M. Rozhko. "STATUS OF ORAL MUCOSA MICROBIOTOPE IN PATIENTS WITH GASTROESOPHAGEAL REFLUX DISEASE." Актуальні проблеми сучасної медицини: Вісник Української медичної стоматологічної академії 22, no. 1 (2022): 85–88. http://dx.doi.org/10.31718/2077-1096.22.1.85.

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In recent years, the issues of "extraesophageal manifestations" of gastroesophageal reflux disease has been receiving more attention from researchers and clinicians that can be explained by the complexity in diagnosis and treatment of the disease, and the necessity in collaboration between medical professional of different specialties as extra-esophageal manifestations can come to the fore imitating diseases of other organs. In the oral cavity there is a favourable environment for the development of beneficial, pathogenic and opportunistic microorganisms. The aim of this study is to investigate the state of oral mucosa microbiotope in patients with gastroesophageal reflux disease. The study included 90 individuals aged from 25 to 55 years, the two test groups involved 30 people in each who had clinically and laboratory confirmed diagnosis of gastroaesophageal reflux disease with hyperacidity and hypoacidity; control group consisted of 30 healthy individuals. The study has demonstrated that the number of microorganisms in the oral cavity differs significantly in sick and healthy people. The most significant changes have been found on the tongue (presence of a large number of opportunistic microorganisms and pathogens) and observed in patients with gastroesophageal reflux disease, especially with high gastric juice acidity. Changes in the micro-biotope on the tongue can be used for confirming diagnosis of gastrointestinal disorders (chronic gastritis, peptic ulcer disease, duodenitis), and gastroesophageal reflux disease in particular.
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Bakulina, Natalia V., Sergey V. Tikhonov, Sergey V. Okovityi, et al. "Pharmacokinetics and pharmacodynamics of rebamipide. New possibilities of therapy: A review." Terapevticheskii arkhiv 94, no. 12 (2023): 1431–37. http://dx.doi.org/10.26442/00403660.2022.12.202000.

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The MedLine database contains 570 publications, including 71 randomized clinical trials and 6 meta-analyses on the rebamipide molecule in 2022. Indications for the use of rebamipide are gastric ulcer, chronic gastritis with hyperacidityin the acute stage, erosive gastritis, prevention of damage to the gastrointestinal mucosa while taking non-steroidal anti-inflammatory drugs, eradication of Helicobacter pylori. Currently trials are studying the efficacy and safety of the drug in gouty and rheumatoid arthritis, osteoarthritis, Sjgren's syndrome, bronchial asthma, vitiligo, atherosclerosis, diseases of the kidneys and liver; using in traumatology to accelerate bone regeneration; in ophthalmology to improve the regeneration of corneal epithelium; in oncology to reduce inflammatory changes in the oral mucosa after chemoradiotherapy. The review article is about the main pharmacokinetic and pharmacodynamic characteristics of rebamipide. A detailed understanding of pharmacodynamics and pharmacokinetics allows for individual selection of therapy based on the characteristics of the patient's body gender, age, comorbidities; choose the optimal route of administration and dosing regimen; predict adverse effects and drug interactions; be determined with new clinical indications.
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Barnich, I. I., and Y. G. Romanova. "INDICES OF �UBT (UREA BREATH TESTS)� IN PATIENTS WITH CHRONIC GENERALIZED PERIODONTITIS OF MODERATE SEVERITY AGAINST HYPERACIDIC GASTRITIS." Bulletin of Problems Biology and Medicine 1, no. 2 (2018): 356. http://dx.doi.org/10.29254/2077-4214-2018-2-144-356-359.

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Lonigro, N., E. Martello, F. Perondi, et al. "Assessing the Efficacy of Natural Pet Products in Protecting Gastric Cells and Reducing Cytotoxicity under Hyperacidity Conditions: An In Vitro Study." Journal of Pharmacy and Nutrition Sciences 14 (August 8, 2024): 8–15. http://dx.doi.org/10.29169/1927-5951.2024.14.02.

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Gastritis in pets necessitates effective acid suppression for successful treatment. However, the synergistic potential of antacid salts within natural feed products remains underexplored. In this in vitro study, we aimed to compare six supplements comprising natural ingredients for their ability to safeguard gastric cells and mitigate cytotoxicity under hyperacidity conditions. While Product 1 showed ineffectiveness in cell protection, Products 2, 3, 4, and 5 exhibited varying degrees of reversal of hyperacidity-induced cytotoxicity. Notably, Product 6 demonstrated superior efficacy in shielding gastric cells from acidic pH-induced cytotoxicity, displaying a dose-dependent response. These findings highlight the potential of natural supplements, particularly Product 6, as promising candidates for mitigating gastritis-related conditions in pets. Further research, including in vivo studies, is warranted to validate these observations and explore their clinical applicability.
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Divya Singh, Nakhat Mal, Vinod Kumar, Rajendra Prasad Sharma, and Purva Pareek. "Scientific explanation of mode of action of Kamadudha Rasa in Amlapitta with special reference to Acid Peptic Disorders: A Review." Journal of Ayurveda and Integrated Medical Sciences 8, no. 4 (2023): 99–105. http://dx.doi.org/10.21760/jaims.8.4.17.

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Amlapitta, also known as hyperacidity, is an illness that has been around for a while and is still very prevalent today. It is a very troublesome illness that, if left untreated, can result in a number of severe issues. Amlapitta symptoms and signs closely resemble gastritis or hyperacidity. The body's Pitta Dosha becoming more active is the primary contributor to hyperacidity. The Ayurvedic humour of Pitta represents heat or flames. There are a variety of factors that can aggravate Pitta, but the major ones are eating foods that are unsuitable for the body type or foods that shouldn't be consumed together (such as milk and fish or milk and salt), as well as drinking overly sour or spicy foods and liquids. In Ayurveda, hyperacidity is referred to as Amlapitta. Thus, a situation known as hyperacidity is one in which the body becomes more hot and sour. Pitta imbalances the digestive fire, resulting in poor food digestion and the creation of Ama. An essential medication used in Amlapitta is Kamadudha Rasa. We are attempting to provide a scientifically sound explanation of the drug's mechanism of action in this review paper.
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Dissertations / Theses on the topic "Chronic gastritis with hyperacidity"

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Redéen, Stefan. "Chronic Gastritis : Diagnosis, natural history and consequences." Doctoral thesis, Linköpings universitet, Kirurgi, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-56575.

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Background & alms: The main cause of chronic gastritis is Helicobacter pylori (H. pylori). Clinical manifestations of chronic gastritis are ulcer disease, gastric cancer and mucosa-associated lymphoma tissue (MALT) lymphoma in the stomach. It is uncertain whether gastritis can be diagnosed macroscopically at endoscopy. H. pylori infection may be diagnosed by several different methods, the accuracy of which needs to be explored. Some individuals with H. pylori related chronic gastritis will develop atrophy of the gastric mucosa. This condition is the main risk factor for cancer development and may also be associated with vitamin B12 deficiency leading to hyperhomocysteinaemia. The natural history of chronic gastritis in terms of development of atrophy and ulcer disease in the adult general population is largely unknown. Material & methods: A sample of 50 I volunteers from the general population in the municipality of Linköping was examined with esophago-gastro-duodenoscopy (EGD) with biopsy. Blood samples were collected in the fasting state and the subjects answered a questionnaire about lifestyle factors, medications and disease history. In-hospital diagnoses and causes of death during follow-up of the population were extracted from local and national patient files. Re-examination was done in 314 subjects after a median follow-up interval of 8.4 years. Five diagnostic tests (serology UBT, RUT, culture and microscopic examination) for H. pylori infection were used at re-examination. Results: The best values of sensitivity and specificity were for visible vessels in relation to microscopic presence of severe atrophy in the gastric corpus mucosa (80% and 87%, respectively). There was a positive relation of S-homocysteine to male gender, age, S-cystatin C (renal function), methylenetetrahydrofolate reductase 677TT genotype and atrophic gastritis. Logistic regression analysis showed an association of S-homocysteine higher than 14.5 Ilmol/L to cardiovascular diseases (OR 2.05), but not to dementia overall. The incidence ofulcer was 0.45 per 100 person years and was associated with weekly NSAID use, weekly alcohol consumption (OR 19.4) and smoking (OR 31.0), but not with H. pylori status. Among subjects with chronic gastritis, the incidence of atrophy of the corpus mucosa was 1.4 per 100 person years. Considering diagnostic test for H. pylori infection the accuracy was 0.86 for serology, 0.94 for UBT, 0.94 for RUT, 0.93 for culture, and 0.93 for histological examination. There was a strong correlation between the results of UBT and the histological scores of H. pylori colonisation as well as between the results of UBT and scores of RUT. Conclusions: The occurrence of chronic gastritis or H. pylori infection is not evaluable macroscopically at gastroscopy, except for the absence of rugae or visible vessels in the gastric corpus mucosa. Serum Hcy concentrations are dependent on gender, age, the levels of vitamin B12 and folate, renal function, the occurrence of atrophic gastritis and the MTHFR 677 TT genotype. Elevated S-Hcy is a risk factor for cardiovascular disease. The incidence of atrophy of the corpus mucosa is 1.4 per 100 person years for chronic gastritis overall. Chronic gastritis with or without H. pylori infection is a variable process in which milder degrees of atrophy of the corpus mucosa may appear or disappear. In contrast, moderate-to-severe atrophy of the corpus mucosa rarely regresses. Age and the degree of chronic inflammation in the gastric corpus mucosa are major risk factors for the development of atrophy. The incidence of ulcer was 0.45 per 100 person years. There are only minor differences in accuracy between the three invasive tests for H. pylori infection. The UBT is recommended for situations where endoscopy is not required. RUT may be recommended as the first non-invasive method of choice in the diagnosis of H. pylori infection.
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許偉武 and Wai-mo Hui. "The pathogenesis and treatment of chronic antral gastritis." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1990. http://hub.hku.hk/bib/B31981380.

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Adonai, Lynette. "The role of glycoconjugates in the colonisation of the gastric tract by Helicobacter pylori." Thesis, University of Westminster, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.252056.

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Sitas, Frederick. "The seroepidemiology of Helicobacter pylori and its relationship to chronic atrophic gastritis." Thesis, University of Oxford, 1990. http://ora.ox.ac.uk/objects/uuid:518be074-87cc-49fc-9228-596c709cb938.

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There were three main aims to this thesis: 1. to test whether infection with Helicobacter pylori was related specifically to chronic atrophic gastritis (CAG), a precursor of gastric cancer; and to measure the specificity and sensitivity of Helicobacter pylori antibody markers for detecting H.pylori infection of the gastric mucosa; and CAG; 2. to measure the distribution of H.pylori in relation to sociodemographic, dietary and lifestyle characteristics in non-clinical populations from Caerphilly (Wales), Italy, China, and Kenya; 3. to examine the relationship between H.pylori and gastric cancer. H.pylori was strongly associated with CAG and H.pylori antibody level was a sensitive and specific marker of both infection (84.8%, 92.7%) and CAG (71.4%, 90.9%). In Caerphilly and Italy, the prevalence of H.pylori increased steeply to 40-50% in those aged 45-54 years and levelled off in older age groups. In China, over 50% of those aged 35-44 were infected. In Kenya, almost all people studied between 18-30 years were infected with H.pylori. In Caerphilly, H.pylori infection was significantly higher in poorer socioeconomic groups, whilst in Italy, socioeconomic differences with regard to infection were in the same direction but not as pronounced. In Caerphilly, H.pylori infection was related to the number of the subject's siblings but not to the number of children or adults sharing the house of the subject. In Caerphilly, those infected with H.pylori consumed less vitamin C and more alcohol than those not infected. Those infected in Italy consumed more vitamin C but similar amounts of alcohol than those not infected. In China, smoking was inversely related to H.pylori infection. In China, a geographic correlation of +0.34 (2p = 0.02) was found between the prevalence of H.pylori infection (measured in 46 counties) and gastric cancer mortality. There was also a positive (but not significant) correlation with peptic ulcer mortality. No other type of cancer showed a significant association with H.pylori.
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Jaskiewicz, Kazimierz. "Chronic gastritis, helicobacter pylori and micronutrient studies in patients at risk for gastric carcinoma." Thesis, University of Cape Town, 1991. http://hdl.handle.net/11427/26354.

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Peterson, Richard A. II. "Investigations into the role of proinflammatory cytokines in the pathogenesis of gastric epithelial proliferation in chronic helicobacter pylori gastritis." The Ohio State University, 2003. http://rave.ohiolink.edu/etdc/view?acc_num=osu1055881993.

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Peterson, Richard Allan. "Investigations into the role of proinflammatory cytokines in the pathogenesis of gastric epithelial proliferation in chronic helicobacter pylori gastritis." Columbus, Ohio : Ohio State University, 2003. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1055881993.

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Thesis (Ph. D.)--Ohio State University, 2003.<br>Title from first page of PDF file. Document formatted into pages; contains xxv, 330 p.; also includes graphics (some col.). Includes abstract and vita. Advisor: Kathryn A. Eaton, Dept. of Veterinary Bioscience. Includes bibliographical references (p. 297-330).
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O'Gara, Elizabeth Ann. "Investigations into the activity of plant preparations against Helicobacter pylori, causal agent of chronic gastritis and gastric and duodenal ulcer." Thesis, University of Wolverhampton, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.343256.

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Zhang, Qibo. "Toxinogenicity of Helicobacter pylori in the pathogenesis of chronic gastritis and peptic ulcer and the nature of the host's immune responses." Thesis, University of Glasgow, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.263002.

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Teixeira, Rute Saraiva Canejo dos Santos Rodrigues. "Helicobacter spp. e gastrite crónica em canídeos e felídeos." Bachelor's thesis, Universidade Técnica de Lisboa. Faculdade de Medicina Veterinária, 2010. http://hdl.handle.net/10400.5/2798.

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Dissertação de Mestrado Integrado em Medicina Veterinária<br>Em clínica de animais de companhia, uma das doenças mais frequentemente encontradas é a gastrite idiopática, uma situação frustrante tanto para o clínico como para os proprietários. No Homem, a bactéria Helicobacter pylori tem recebido muita atenção pela sua capacidade oncogénica e existe a possibilidade de algumas espécies, nomeadamente “H. heilmannii”, H. salomonis, H. felis, H. bizzozeronii, e H. bilis, terem algum poder patogénico em animais de companhia. A finalidade do estudo desenvolvido ao longo do estágio foi de estabelecer a prevalência de várias espécies de Helicobacter numa população Portuguesa típica de animais de companhia, e de procurar eventuais relações entre estado de colonização e patologia gástrica. Num total de 103 animais (56 canídeos, 47 felídeos) foram determinadas, com recurso a técnicas de PCR, prevalências de 37,5%/48,9% para H. spp., 28,6%/34% para H. bizzozeronii e 17,9%/48,9% para “H. heilmannii” em cães e gatos respectivamente. H. pylori foi identificado num único gato (2,1%); H. salomonis e H. felis foram identificados exclusivamente em cães, com prevalências de 10,7% e 7,1%, respectivamente. H. bilis não foi identificado em qualquer animal. Em 19 das amostras fecais testadas (14 de origem canina e cinco de origem felina) 50% dos cães e 60% dos gatos acusaram a presença de H. spp, sendo apenas possível identificar, a nível de espécie, H. felis e H. bizzozeronii em amostras de dois cães. Foi identificada uma relação entre sintomatologia gástrica e colonização por “H. heilmannii” nos gatos, enquanto nos cães se determinou uma relação entre colonização por H. bizzozeronii e a presença de alterações inflamatórias na mucosa gástrica. A utilização de esfregaços da mucosa gástrica permitiu avaliar mais adequadamente o estado de colonização dos animais do que a observação de espiroquetas em cortes histológicos. Este estudo demonstra a presença de Helicobacter spp. numa população Portuguesa típica e sugere uma potencial relação entre Helicobacter spp. e gastrite crónica em animais de companhia, especialmente em gatos. A informação sobre presença de espécies de Helicobacter e respectivas prevalências poderá contribuir para uma decisão mais fundamentada sobre a prescrição de antibioterapia específica para Helicobacter em pacientes apresentados à consulta com sinais gastrointestinais e um diagnóstico de gastrite idiopática.<br>ABSTRACT - Helicobacter spp. and chronic gastritis in dogs and cats - Chronic idiopathic gastritis, one of the most frequently observed diseases in companion animal practice, can be a source of frustration for both the clinician and the owner. In humans, Helicobacter pylori has been thoroughly investigated due to its’ oncogenic capabilities. The possibility exists that some species of Helicobacter, namely H. salomonis, “H. heilmannii”, H. felis, H. bizzozeronii and H. bilis may play a role in the pathogenesis of chronic gastritis in companion animals. The current study, completed as part of the final year trainee program, was designed to investigate the prevalence of Helicobacter in a typical companion animal population, while searching for any eventual relationship between colonization and gastric disease. Prevalence was determined through the use of PCR in 103 animals (56 canines, 47 felines) as 37.5%/48.9% H. spp, 28.6%/34% H. bizzozeronii and 17.9%/48.9% “H. heilmannii” in dogs and cats, respectively. H. pylori was identified in a single feline sample (2.1%) while H. salomonis and H. felis were oberved exclusively in canine samples with a prevalence of 10.7% and 7.1% respectively. H. bilis could not be demonstrated in any of the samples analysed. Fecal samples from 19 patients were similarly tested (14 from canines, 5 from felines), 50% of dogs and 60% of cats tested positive for H. spp. Species specific PCR, identified the presence of H. felis and H. bizzozeronii in two dogs. In canines, a relationship between colonization with H. bizzozeronii and inflammatory alterations in the gastric mucosa was shown, while in cats colonization with “H. heilmannii” could be related to the presence of clinical signs of gastritis. Brush cytology was found to be more effective than histology in demonstrating the presence of Helicobacter in the gastric mucosa of both canines and felines. This study showed the presence of Helicobacter in a sample population of companion animals and the data gathered strongly suggests a relationship, especially in cats, between colonization and disease. The results obtained may contribute to an informed decision in prescribing antibiotic therapy for Helicobacter-associated gastritis in patients that present idiopathic chronic gastritis.
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Books on the topic "Chronic gastritis with hyperacidity"

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Piracha, Kashif. Chronic Atrophic Gastritis. Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-09872-7.

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R, Holt Peter, and Russell Robert 1941-, eds. Chronic gastritis and hypochlorhydria in the elderly. CRC Press, 1993.

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O'Gara, Elizabeth Ann. Investigations into the activity of plant preparations against Helicobacter pylori, causal agent of chronic gastritis and gastric and duodenal ulcer. University of Wolverhampton, 2001.

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Falk Symposium (81st 1994 Berlin, Germany). Malignancy and chronic inflammation in the gastrointestinal tract: New concepts : proceedings of the 81st Falk Symposium, held in Berlin, Germany, November 3-5, 1994. Kluwer Academic Publishers, 1995.

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Nutrition During Chronic Gastritis. Books on Demand GmbH, 2018.

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Yue, Guo Haiying; Wang. Typical TCM Therapy for Chronic Gastritis. Shanghai University of Traditional Chinese Medicine Press, 2003.

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Mario, F. Di, and Di Mario. Advances in Gastroenterology: 4. Chronic Atrophic Gastritis (Advances in Gastroenterology). Gordon & Breach Science Publishers, 1991.

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Gastroesophageal Anti-Reflux Diet: Low Fat, Low Acid, Weight Management. UMass Memorial Medical Center, 2019.

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Book chapters on the topic "Chronic gastritis with hyperacidity"

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Cheli, Rodolfo, Alessandro Perasso, and Attilio Giacosa. "Chronic Gastritis and Chronic Duodenitis." In Gastritis. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71845-8_15.

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Köster, Liza S. "Chronic Gastritis." In Chronic Disease Management for Small Animals. John Wiley & Sons, Inc., 2017. http://dx.doi.org/10.1002/9781119201076.ch16.

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van der Post, Chella R. S., and Fátima Carneiro. "Chronic Gastritis." In Encyclopedia of Pathology. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-40560-5_1610.

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Cheli, Rodolfo, Alessandro Perasso, and Attilio Giacosa. "Chronic Gastritis and Polyps." In Gastritis. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71845-8_17.

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Cheli, Rodolfo, Alessandro Perasso, and Attilio Giacosa. "Chronic Gastritis and Gastric Ulcer." In Gastritis. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71845-8_13.

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Cheli, Rodolfo, Alessandro Perasso, and Attilio Giacosa. "Chronic Gastritis and Duodenal Ulcer." In Gastritis. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71845-8_14.

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Cheli, Rodolfo, Alessandro Perasso, and Attilio Giacosa. "Chronic Atrophic Gastritis and Cancer." In Gastritis. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71845-8_16.

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Cheli, Rodolfo, Alessandro Perasso, and Attilio Giacosa. "Chronic Gastritis and Endocrine Tumors." In Gastritis. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71845-8_18.

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Kalantar, J., G. D. Eslick, and N. J. Talley. "Chronic Gastritis and Nonulcer Dyspepsia." In Gastroduodenal Disease and Helicobacter pylori. Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-642-60013-5_3.

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Vaiphei, Kim. "Chronic Gastritis: Infectious, Non-infectious (Autoimmune, Chemical, Granulomatous), Chronic Superficial and Diffuse Antral Gastritis." In Interpretation of Endoscopic Biopsy - Gastritis, Gastropathies and Beyond. Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-6026-9_8.

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Conference papers on the topic "Chronic gastritis with hyperacidity"

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Liu, Yige, Kun Leng, Haichuan Ren, Xudong Liu, Shaoli Wang, and Nan Luo. "Tongue Manifestation Feature Classification and Detection for Chronic Gastritis Based on the YOLO Framework." In 2024 International Annual Conference on Complex Systems and Intelligent Science (CSIS-IAC). IEEE, 2024. https://doi.org/10.1109/csis-iac63491.2024.10919443.

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Lu, Shengda, Jiaxin Wu, Ruiyu Li, Xiaoran Liu, Kaijin Chen, and Diangui Li. "The medication pattern of Li Diangui in the treatment of chronic atrophic gastritis and gastric precancerous lesions." In 2024 14th International Conference on Information Technology in Medicine and Education (ITME). IEEE, 2024. https://doi.org/10.1109/itme63426.2024.00043.

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Guo, Hongtao, and Guang Zheng. "Mechanisms of Atractylodis Macrocephalae Rhizoma against Chronic Gastritis." In 2019 IEEE International Conference on Bioinformatics and Biomedicine (BIBM). IEEE, 2019. http://dx.doi.org/10.1109/bibm47256.2019.8983170.

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Yan, Jian-Jun, Tao Zhong, Guo-Ping Liu, et al. "Prosvms based diagnostic model of chronic gastritis in TCM." In 2014 11th IEEE International Conference on Control & Automation (ICCA). IEEE, 2014. http://dx.doi.org/10.1109/icca.2014.6871076.

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Novikov, Yakov Semyonovich, and Vyacheslav Georgievich Vonogel. "APOPTOSIS: HEALTH AND DISEASE. CHRONIC GASTRITIS AND GASTRODUODENAL ULCERS." In Наука и социум. Автономная некоммерческая организация дополнительного профессионального образования "Сибирский институт практической психологии, педагогики и социальной работы", 2022. http://dx.doi.org/10.38163/978-5-6046740-7-9_2022_140.

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Novikov, Yakov Semenovich, and Vyacheslav Georgievich Vonogel. "APOPTOSIS: HEALTH AND DISEASE. CHRONIC GASTRITIS AND GASTRODUODENAL ULCERS." In Наука и социум. Коррекционно-развивающая среда и инклюзивная практика помощи детям с ОВЗ. Автономная некоммерческая организация дополнительного профессионального образования "Сибирский институт практической психологии, педагогики и социальной работы", 2021. http://dx.doi.org/10.38163/978-5-6045317-3-0_2021_157.

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Chen, Tong-sheng, Shao-zi Li, and Chang-le Zhou. "Knowledge discovery of chronic gastritis diagnosis by logistic discriminant analysis." In 2008 IEEE International Symposium on IT in Medicine and Education (ITME). IEEE, 2008. http://dx.doi.org/10.1109/itme.2008.4743968.

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Chen, Tong-Sheng, Shao-Zi Li, and Chang-Le Zhou. "Knowledge discovery of chronic gastritis diagnosis by inferences and predictions." In Education (ITIME). IEEE, 2009. http://dx.doi.org/10.1109/itime.2009.5236246.

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Liu, Qian, Jingbin Niu, Weixi Mao, Yixin Zheng, Guoping Liu, and Jin Zhao. "Chronic Gastritis Syndrome Diagnosis and Symptom Selection with Ensemble Learning." In 2021 IEEE International Conference on Bioinformatics and Biomedicine (BIBM). IEEE, 2021. http://dx.doi.org/10.1109/bibm52615.2021.9669852.

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Wang, Huazhen, Chengde Lin, Fan Yang, and Xueqin Hu. "Hedged Predictions for Traditional Chinese Chronic Gastritis Diagnosis with Confidence Machine." In 2008 International Conference on Computer Science and Information Technology (ICCSIT). IEEE, 2008. http://dx.doi.org/10.1109/iccsit.2008.144.

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Reports on the topic "Chronic gastritis with hyperacidity"

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Yingjun, Nie, Wang Chengyulin, and Wu Qiaofeng. Moxibustion for the treatment of chronic gastritis: A systematic review protocol. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2020. http://dx.doi.org/10.37766/inplasy2020.11.0080.

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Guo, MEngland, zhenghua Xiao, bingqing chen, qianren tan, and jun song cui. Meta-analysis of the efficacy and safety of the hepatosplenomyosis method in the treatment of chronic atrophic gastritis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.6.0015.

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Chen, Qian, and Xia Ding. Meta-analysis of modified Sijunzi decoction combined with conventional western medicine in the treatment of chronic atrophic gastritis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2024. http://dx.doi.org/10.37766/inplasy2024.11.0062.

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Yan, Huixin, Yun An, Jiangna Zhao, Tao Zhang, and Juntao Yan. The efficacy and safety of acupoint injection for chronic atrophic gastritis: A protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.9.0105.

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Ying, Dashi, zhitao Feng, Jingzhou Zhang, et al. Efficacy and Safety of acupoint catgut embedding for chronic atrophic gastritis A protocol of systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.4.0040.

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Ma, Yujing, Jianping Liu, Xiaomeng Lang, Xin Kang, Xioameng Liu, and Hong Jiao. The efficacy and safety of Chinese patent medicines in the treatment of chronic atrophic gastritis: A bayesian network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2020. http://dx.doi.org/10.37766/inplasy2020.12.0063.

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Xi, Xiaomei, and Xingmin Wei. Meta-analysis of the treatment of chronic gastritis infected by Helicobacter pylori by clearing heat and removing dampness combined with quadruple therapy and the mechanism of common drugs. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.2.0007.

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Leelahavanichkul, Asada. Helicobacter pylori infection increased anti-dsDNA and enhanced lupus severity in symptomatic FcγRIIb-deficient lupus mice. Chulalongkorn University, 2019. https://doi.org/10.58837/chula.res.2019.10.

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Abstract:
The defect on Fc gamma receptor IIb (FcyRIIb), the only inhibitory FcyR, has been identified as one of the genetic factors increasing susceptibility to lupus. The prevalence of Helicobacter pylori (HP) and FcyRIIb dysfunction-polymorphisms are high among Asians, and their co-existence is possible. Unfortunately, the influence of HP against lupus progression in patients with lupus is still controversial. In this study, the interactions between these conditions were tested with HP infection in 24-week-old Fc yRIIb−/− mice (symptomatic lupus). HP induced failure to thrive, increased stomach bacterial burdens and stomach injury (histology and cytokines) in both wild type and FcyRIIb−/− mice. While the severity of HP infection, as determined by these parameters, was not different between both strains, antibodies production (anti-HP, anti-dsDNA and serum gammaglobulin) were higher in FcyRIIb−/− mice compared to wild type. Accordingly, HP infection also accelerated the severity of lupus as determined by proteinuria, serum creatinine, serum cytokines, renal histology, and renal immune complex deposition. Although HP increased serum cytokines in both wild type and FcyRIIb−/− mice, the levels were higher in FcyRIIb−/− mice. As such, HP also increased spleen weight and induced several splenic immune cells responsible for antibody productions (activated B cell, plasma cell and follicular helper T cell) in FcyRIIb−/− mice, but not in wild type. These data describe the different systemic responses against localized HP infection from diverse host genetic background. In conclusion, the mutual interactions between HP and lupus manifestations of FcyRIIb−/−mice were demonstrated in this study. With the prominent immune responses from the loss of inhibitory signaling in FcyRIIb−/− mice, HP infection in these mice induced intense chronic inflammation, increased antibody production, and enhanced lupus severity. Thus, the increased systemic inflammatory responses due to localized HP inducing gastritis in some patients with lupus may enhance lupus progression. More studies are needed.
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