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Journal articles on the topic 'Chronic gastritis'

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1

Sipponen, Pentti, and Heidi-Ingrid Maaroos. "Chronic gastritis." Scandinavian Journal of Gastroenterology 50, no. 6 (2015): 657–67. http://dx.doi.org/10.3109/00365521.2015.1019918.

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2

TAKEMOTO, Tadavoshi. "Chronic gastritis." Japanese Journal of Medicine 24, no. 3 (1985): 276–82. http://dx.doi.org/10.2169/internalmedicine1962.24.276.

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TAKEMOTO, Tadayoshi. "Chronic gastritis." Nihon Naika Gakkai Zasshi 74, no. 7 (1985): 867–79. http://dx.doi.org/10.2169/naika.74.867.

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4

Ali, Hussain Alawadh Suzan Essam Jiffri Thamer Saad Alghalbi M. Afnan Essam Jiffri Aisha Jamal Alali Mohammed Samy Tayb Hussain Jassim Alzaid Saeed Mohammed Alqahtani Faisal Abdullah Alamri Sultan Majed Almalki Zainab Malik alnasser Yara Hasan Nouri Obaydo Ali Hussain alawadh Ali Hadi Yahya madkhali Arwa Saad Alzahrani. "MANAGEMENT OF CHRONIC GASTRITIS." INDO AMERICAN JOURNAL OF PHARMACEUTICAL SCIENCES o6, no. 02 (2019): 4026–31. https://doi.org/10.5281/zenodo.2566103.

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<strong><em>Introduction: </em></strong><em>Chronic gastritis is one of the common serious pandemic infections in developing countries with severe sequelae such as peptic ulcer or gastric ulcer, though the prevalence of chronic gastritis has markedly declined in developed population. More than half of the people are suffering from chronic gastritis globally at present. Helicobacter pylori infection in childhood is a major contributing factor of chronic gastritis, which microbial origin is a critical factor for understanding epidemiology and course of the disease.&nbsp; An aggressive inflammati
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5

PALMER, EDDY D. "Chronic Hypertrophic Gastritis." Acta Medica Scandinavica 147, no. 1 (2009): 43–49. http://dx.doi.org/10.1111/j.0954-6820.1954.tb12213.x.

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6

Farinati, F., R. Cardin, F. Valiante, G. Delia Libera, M. Plebani, and F. Di Mario. "Chronic atrophic gastritis." European Journal of Cancer Prevention 2, Supplement (1993): 24. http://dx.doi.org/10.1097/00008469-199301001-00057.

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7

Haot, J., B. Weynand, and A. Jouret-Mourin. "Chronic lymphocytic gastritis." Gut 31, no. 7 (1990): 840. http://dx.doi.org/10.1136/gut.31.7.840.

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8

Simonova, V. G., and E. I. Belova. "Children’s chronic gastritis." Международный студенческий научный вестник (International Student Scientific Herald), no. 6 2022 (2022): 3. http://dx.doi.org/10.17513/msnv.21007.

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9

Uppal, Rajiv. "Chronic Alcoholic Gastritis." Archives of Internal Medicine 151, no. 4 (1991): 760. http://dx.doi.org/10.1001/archinte.1991.00400040102023.

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10

Bakulin, Igor G., Anastasiia G. Sushilova, and Alexander V. Zharkov. "Chronic atrophic gastritis: focus on diagnosis." HERALD of North-Western State Medical University named after I.I. Mechnikov 15, no. 3 (2023): 13–24. http://dx.doi.org/10.17816/mechnikov623654.

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Currently, chronic atrophic gastritis is a risk factor for the development of gastric cancer. Data on the prevalence of chronic atrophic gastritis in Russia and the world are limited. Among the adult population of St. Petersburg aged 21 to 81 years, the prevalence of histologically verified chronic atrophic gastritis is 5.3%. Moreover, the chances of developing chronic atrophic gastritis in people over the age of 60 years compared to the group of 45–59 years are 2.5 times higher (95% confidence interval 1.5–4.1).&#x0D; Morphological verification of the Operative Link for Gastritis Assessment d
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11

Skvortsov, V. V., L. V. Zaklyakova, B. N. Levitan, M. Yu Bolgova, I. K. Zaklyakov, and E. A. Golieva. "Modern approaches to pharmacotherapy of chronic gastritis." Meditsinskiy sovet = Medical Council, no. 15 (October 19, 2021): 40–47. http://dx.doi.org/10.21518/2079-701x-2021-15-40-47.

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The definition of gastritis is based on the histological features of the gastric mucosa. This is not the erythema observed during gastroscopy, and there are no specific clinical manifestations or symptoms that determine it. The modern classification of gastritis is based on time (acute and chronic), histological features, anatomical distribution and the main pathological mechanisms. Acute gastritis will develop into chronic if left untreated. Helicobacter pylori (H. pylori) is the most common cause of gastritis worldwide. However, from 60 to 70% H. pylori-negative subjects with functional dysp
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12

Gabueva, N. V., V. S. Kalinchuk, D. R. Radchenko, S. A. Donchenko, A. A. Panesh, and S. A. Popov. "Chronic gastritis in pediatrics." CARDIOMETRY, no. 29 (November 1, 2023): 54–61. http://dx.doi.org/10.18137/cardiometry.2023.29.5461.

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Chronic gastritis is one of the most common diseases of the gastrointestinal tract. High rates of gastritis in children are associated with poor nutrition, physical inactivity, drug abuse, bad habits, a number of concomitant diseases, immune disorders, allergization of the body, and stress. Although a significant proportion in the structure of chronic gastritis is occupied by gastritis associated with Helicobacter pylori infection, many clinical forms are diagnosed that are not associated with the presence of a microbial pathogen in the gastric mucosa. The goal is to highlight the problem of c
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13

Tsimmerman, Yakov S., and Yu A. Zakharova. "Topical problems of chronic gastritis." Clinical Medicine (Russian Journal) 95, no. 1 (2017): 8–14. http://dx.doi.org/10.18821/0023-2149-2017-95-1-8-14.

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The authors propose the definition of chronic gastritis with a brief overview of the history of investigation of this diseases and its prevalence with special reference to (1) the bacterial profile and its role in the development of infectious and inflammatory process in gastric mucosa, (2) the modern state of and new trends in the classification of chronic gastritis including their advantages and disadvantages, (3) the relationship between chronic gastritis and functional gastroduodenal dyspepsia syndrome. It is concluded that mucous microflora plays an important factor in the development ofp
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14

Yuan, Mingyang. "Research status of risk factors of chronic atrophic gastritis." Theoretical and Natural Science 16, no. 1 (2023): 171–77. http://dx.doi.org/10.54254/2753-8818/16/20240566.

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Gastritis, also known as inflammation of the gastric mucosa, is a prevalent disorder of the digestive system that can be attributed to a range of reasons. Based on the level of illness severity, gastritis can be categorized into two distinct types: acute gastritis and chronic gastritis. Based on the observed pathological alterations, acute gastritis can be categorized into several subtypes, namely simple gastritis, erosive and hemorrhagic gastritis, corrosive gastritis, and suppurative gastritis. Based on the observed pathological alterations, chronic gastritis can be categorized into three di
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15

Siregar, Gontar Alamsyah, and Lydia Imelda Laksmi. "Risk Factors of Chronic Atrophic Gastritis." Indonesian Journal of Gastroenterology, Hepatology, and Digestive Endoscopy 15, no. 3 (2014): 152–56. http://dx.doi.org/10.24871/1532014152-156.

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Background: Chronic atrophic gastritis is a well-established precursor of gastric cancer. The development of atrophic chronic gastritis is multifactorial, involving the environment as well as host responses to the Helicobacter pylori (H. pylori) infection. The aim of this study is to determine prevalence and risk factors of chronic atrophic gastritis.Method: The study was a cross sectional study on gastritis patients admitted to endoscopy units at Adam Malik General Hospital and Permata Bunda Hospital, Medan, from May-October 2014. A simple random sampling was performed to obtain 50 patients.
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16

Wang, Ting, Yuyin Feng, Hesong Wang, et al. "The Mechanisms of Sijunzi Decoction in the Treatment of Chronic Gastritis Revealed by Network Pharmacology." Evidence-Based Complementary and Alternative Medicine 2020 (October 23, 2020): 1–10. http://dx.doi.org/10.1155/2020/8850259.

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Chronic gastritis is characterized by inflammation in the gastric mucosa with a vicious circle in inflammatory cells and inflammatory mediators. Stomach adenocarcinoma would occur in the metaplastic gastric mucosa of chronic gastritis. Sijunzi decoction is a famous classical formula for the treatment of chronic gastritis. Although previous studies revealed some functions of Sijunzi decoction in treating chronic gastritis, the underlying mechanisms have not been illustrated clearly. In this study, we used network pharmacology to investigate the mechanism of Sijunzi decoction in treating chronic
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17

K.C., Shiva Raj, A. Lakhey, K. Koirala, and GL Amatya. "Prevalence of Helicobacter pylori among patients with dyspepsia and correlation between endoscopic and histological diagnosis." Journal of Pathology of Nepal 6, no. 11 (2016): 942–46. http://dx.doi.org/10.3126/jpn.v6i11.15678.

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Background: Dyspepsia is a prevalent complaint in general practice and gastrointestinal clinics. Helicobacter pylori have major causal relationship with gastro duodenal disease. The following study seeks to identify the prevalence of H. pylori based on histology and to correlate endoscopic findings with histopathology.Materials and Methods: This was a cross-sectional observational study conducted in GRP Polyclinic and Om Hospital and research centre from April 2015-September 2015. The upper gastrointestinal endoscopic findings were recorded and were correlated with histopathological findings.
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18

Romshoo, Gh Jeelani, G. M. Malik, M. Youssuf Bhat, Ab Rashid rather, Javaid Ahmad Basu, and Khursheed Ahmad Qureshi. "Helicobacter Pylori Associated Antral Gastritis in Peptic Ulcer Disease Patients and Normal Healthy Population of Kashmir, India." Diagnostic and Therapeutic Endoscopy 4, no. 3 (1998): 135–39. http://dx.doi.org/10.1155/dte.4.135.

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Aim: To study the association of Helicobacter pylori infection with chronic antral gastritis in peptic ulcer disease patients and healthy population of Kashmir.Methods: 50 peptic ulcer patients (duodenal ulcer = 46, gastric ulcer = 2 and combined duodenal and gastric ulcer = 2) and 30 asymptomatic healthy volunteers were included in this study. Peptic ulcer was diagnosed on endoscopic examination. 4–6 punch biopsies were taken from gastric antrum in all the individuals and in case of gastric ulcer an additional biopsy was taken from the edge of the ulcer to exclude its malignant nature. Helico
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19

Zhang, Yanzhao, and Yongpan Xu. "Exploration of the Pathogenesis of Hp-associated Chronic Atrophic Gastritis in Association with the Theory of Latent Damp-heat." Journal of Contemporary Medical Practice 6, no. 7 (2024): 101–5. http://dx.doi.org/10.53469/jcmp.2024.06(07).21.

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The incidence of Helicobacter pylori infection in China is relatively high, and Helicobacter pylori can mediate the development of normal gastric mucosa to chronic atrophic gastritis, while chronic atrophic gastritis can develop into precancerous changes such as intestinal metaplasia over a long period of time, ultimately leading to gastric cancer. The nature of Helicobacter pylori is similar to that of the latent damp-heat of Traditional Chinese Medicine (TCM). Therefore, this paper discusses the occurrence and development mechanism of chronic atrophic gastritis associated with Helicobacter p
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20

Alturkustani, Murad A. "Gastric Histopathology of Chronic Gastritis in Obese Patients Undergoing Laparoscopic Sleeve Gastrectomy: A Local Experience." Journal of King Abdulaziz University - Medical Sciences 24, no. 2 (2017): 9–15. http://dx.doi.org/10.4197/med.24-2.2.

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A retrospective review was conducted using the database at King Abdulaziz University Hospital, to determine the most common cause of chronic gastritis among obese individuals. One hundred and thirty-one specimens were examined, obtained from patients who had laparoscopic sleeve gastrectomy and diagnosed with chronic gastritis. Helicobacter pylori was detected in 15 cases only. The most common diagnosis was “Chronic inactive gastritis without Helicobacter pylori infection” in 57 (43.50%) cases, then “Chronic gastritis with lymphoid follicular hyperplasia” in 54 (41.20%) cases, and lastly “Chron
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21

Rahmawati, Rahmawati. "FAKTOR – FAKTOR YANG BERHUBUNGAN DENGAN TIMBULNYA GEJALA GASTRITIS PADA SISWA SEKOLAH MENENGAH ATAS KOTA JAMBI." Jurnal Ilmiah Dikdaya 8, no. 2 (2018): 334. http://dx.doi.org/10.33087/dikdaya.v8i2.115.

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Gastritis is an inflammation of the gastric mucosa that can be acute, chronic, diffuse or localized, the stomach is a digestive device that functions mechanically in storing food and drinks, breaking food into small particles, mixing with gastric fluid and gradually entering the intestine . The clinical picture of erosive acute gastritis varies from mild, asymptomatic, to very severe. Factors for gastritis are foods contaminated with bacteria, caffeine-containing drinks, alcohol, drugs such as non-steroidal anti-inflammatory drugs aspirin, acidic foods and drinks, spicy foods, and irregular di
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22

Pavlovich, I. M., G. A. Al’per, A. V. Gordienko, D. I. Proskunov та V. V. Yakovlev. "Сhronic gastritis: impaired motor-evacuation function of the stomach". Bulletin of the Russian Military Medical Academy 22, № 4 (2020): 37–42. http://dx.doi.org/10.17816/brmma62802.

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The effect of morphological changes in the gastric mucosa on the motor-evacuation function of the stomach was evaluated in 90 patients with chronic atrophic gastritis and 93 patients with chronic non-atrophic gastritis aged 18 to 82 years. Motor function disturbances were discovered of 90% of patients with chronic atrophic gastritis and 80,6% with chronic non-atrophic gastritis. It was established that the pyloric insufficiency compared with its spasm was significantly more (p0,01) often in chronic atrophic gastritis. Insufficiency of the lower esophageal sphincter in patients with atrophic ga
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23

Elanchezhian, Gayathri, DR Thanka J, and S. Mary Lilly. "H Pylori Associated Chronic Gastritis and Carcinogenic Effect." International Journal of Science and Research (IJSR) 11, no. 2 (2022): 406–9. http://dx.doi.org/10.21275/sr22128171311.

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24

Sousa, Bruno, and Nelson Tavares. "Chronic gastritis (Clinical case)." Journal Biomedical and Biopharmaceutical Research 15, no. 2 (2018): 167–69. http://dx.doi.org/10.19277/bbr.15.2.183.

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25

Riestra Guiance, Irene T., Ernesto Robalino Gonzaga, Richard Henriquez, and Vishwas N. Vanar. "S3588 Chronic Lymphocytic Gastritis." American Journal of Gastroenterology 115, no. 1 (2020): S1852—S1853. http://dx.doi.org/10.14309/01.ajg.0000716400.37559.97.

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26

Varbanova, Mariya, Katrin Frauenschläger, and Peter Malfertheiner. "Chronic gastritis – An update." Best Practice & Research Clinical Gastroenterology 28, no. 6 (2014): 1031–42. http://dx.doi.org/10.1016/j.bpg.2014.10.005.

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27

Darenskaya, Marina A., Olga V. Smirnova, Edward V. Kasparov, et al. "Evaluation of the lipid peroxidation reactions activity in middle-aged men with chronic gastritis on the background of H. pylori infection." Annals of the Russian academy of medical sciences 74, no. 3 (2019): 149–56. http://dx.doi.org/10.15690/vramn1087.

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Background: There is not enough information about the course of the processes of lipid peroxidation-antioxidant defense in middle-aged men who are at risk of developing gastric cancer on the Correa cascade.&#x0D; Aims: To analyze of the processes of lipid peroxidation activity and antioxidant defense in men with chronic gastritis and chronic atrophic gastritis in combination with and without the presence of Helicobacter pylori.&#x0D; Materials and methods: 173 middle-aged men with an established diagnosis of chronic gastritis were examined, which were divided into 4 groups: group 1 ― patients
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Nardone, G., S. Staibano, A. Rocco, et al. "Effect of Helicobacter pyloriinfection and its eradication on cell proliferation, DNA status, and oncogene expression in patients with chronic gastritis." Gut 44, no. 6 (1999): 789–99. http://dx.doi.org/10.1136/gut.44.6.789.

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BACKGROUNDHelicobacter pylori, the main cause of chronic gastritis, is a class I gastric carcinogen. Chronic gastritis progresses to cancer through atrophy, metaplasia, and dysplasia. Precancerous phenotypic expression is generally associated with acquired genomic instability.AIMTo evaluate the effect of H pylori infection and its eradication on gastric histology, cell proliferation, DNA status, and oncogene expression.METHODS/SUBJECTSMorphometric and immunohistochemical techniques were used to examine gastric mucosal biopsy specimens from eight controls, 10 patients withH pylori negative chro
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Ivashkin, V. T., I. V. Maev, T. L. Lapina, et al. "H. pylori-Associated Gastritis, Gastritis after H. pylori Eradication and H. pylori-Negative Gastritis: Algorithm of Diagnosis and Treatment (Literature Review and Resolution of the Expert Panel of the Russian Gastroenterological Association)." Russian Journal of Gastroenterology, Hepatology, Coloproctology 34, no. 3 (2024): 7–23. http://dx.doi.org/10.22416/1382-4376-2024-34-3-7-23.

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Aim: to provide the basic points of the Expert Panel of the Russian Gastroenterological Association with discussion of measures to improve diagnostics, treatment and management of chronic gastritis patients.Key points. In the Russian Federation in 2021 more than 30 million patients diagnosed with “Gastritis and duodenitis” were recorded (K29 in accordance with International Classification of Diseases-10). H. pylori incidence rate in the Russian population has reduced and presently is about 40 %. In chronic gastritis pattern H. pylori-associated gastritis has still dominated though gastritis pe
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Tsimmerman, Y. S., and Yu A. Zakharova. "Problematic issues of chronic gastritis studies." Herald of Pancreatic Club 44, no. 3 (2019): 54–61. http://dx.doi.org/10.33149/vkp.2019.03.06.

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The article defines chronic gastritis as a polyetiological and polypathogenetic stomach disease with a chronic, slowly progressing course, which is based on a specific inflammatory process with lymphoplasmocytic infiltration of its mucosa and neutrophilic component indicating its activity, and with development of disregenerative, dystrophic changes, leading to its secretory insufficiency, manifested hypo- and achlorhydria and gastric achilia.&#x0D; The history of studying chronic gastritis from the beginning of the 19th century till present days is briefly described.&#x0D; It is proposed to di
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31

El-Serag, H. B., A. Sonnenberg, M. M. Jamal, J. M. Inadomi, L. A. Crooks, and R. M. Feddersen. "Corpus gastritis is protective against reflux oesophagitis." Gut 45, no. 2 (1999): 181–85. http://dx.doi.org/10.1136/gut.45.2.181.

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BACKGROUNDGastric acid is important in the pathogenesis of reflux oesophagitis. Acid production by the gastric corpus is reduced in corpus gastritis.AIMSTo determine whether corpus gastritis protects against reflux oesophagitis.METHODSPatients presenting for elective oesophagogastroduodenoscopy were studied. Two biopsy specimens were taken from the antrum, corpus, and cardia and stained with haematoxylin/eosin and Diff-Quick II stains. The presence and severity of gastritis were graded according to a modified updated Sydney classification.RESULTSOf 302 patients, 154 had endoscopic signs of ref
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32

Novikova, V. P., А. I. Khavkin, N. V. Baryshnikova, et al. "Draft of clinical guidelines “Gastritis and duodenitis in children” (excerpt)." Voprosy praktičeskoj pediatrii 19, no. 5 (2024): 114–28. https://doi.org/10.20953/1817-7646-2024-5-114-128.

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The authors present modern data on the etiopathogenesis of chronic gastritis and duodenitis, classification and clinical characteristics of various variants of this nosology. In accordance with the developed clinical recommendations, information on the methods of treatment of chronic gastritis and duodenitis, as well as their combination is given. A special emphasis in the presented fragment of clinical recommendations is made on the schemes of therapy of gastritis associated with Helicobacteriosis infection, prevention of complications and rehabilitation methods. Key words: chronic gastritis,
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Rivas-Ortiz, Claudia Ivette, Stephanie Euridice Morales-Guerrero, Sergio Ponce-de-León-Rosales, et al. "Overview of Gene Expression Analysis in Gastric Disease Infected with Helicobacter pylori: CLDN1 and MMP9 Could Be Biomarkers for Early Diagnosis of Gastric Cancer." Processes 10, no. 2 (2022): 196. http://dx.doi.org/10.3390/pr10020196.

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Chronic Helicobacter pylori infection produces several lesions in the human stomach, which can progress to chronic atrophic gastritis and gastric cancer. To date, there is very little information on gene expression in chronic atrophic gastritis and its relationship with progression to gastric cancer. In this study, we performed a gene expression analysis during chronic atrophic gastritis in order to identify possible biomarkers that allow an early diagnosis of gastric cancer. We studied biopsies from patients with chronic atrophic gastritis and gastric cancer. The biopsies were analyzed by a g
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34

Selma, İrem SÖZEN, Kovancı Hafize, Said Dalkılıç Muhammed, and SÖZEN Selim. "Nutrition and Helicobacter pylori infection in gastric disease." World Journal of Advanced Research and Reviews 18, no. 3 (2023): 021–30. https://doi.org/10.5281/zenodo.8429966.

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<em>H. pylori</em>&nbsp;is a highly prevalent bacterial infection among humans. It is considered the major etiological factor in the development of chronic gastritis, peptic ulcers, and stomach cancer. Nutritional therapy is important for the prevention of gastritis, as well as for reducing stomach irritation (irritation) in the treatment of gastritis,&nbsp;<em>and it&nbsp;</em>plays an important role by helping to eradicate or strengthen the stomach wall.&nbsp; Chronic gastritis should be diagnosed before it progresses and causes fatal diseases such as peptic ulcer or cancer, and it should be
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35

Minushkin, O. N., I. V. Zverkov, N. V. Lvova, Yu S. Skibina, and V. S. Inevatova. "Chronic gastrite: modern state of the problem." Terapevticheskii arkhiv 92, no. 8 (2020): 18–23. http://dx.doi.org/10.26442/00403660.2020.08.000693.

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Aim. Тo evaluate the modern view on the problem of chronic gastritis and the effectiveness of the drug ursodexic acid (UHC) Grinterol in the treatment of patients with chronic antral reflux-gastritis (biliar).&#x0D; Materials and methods. The work provides modern ideas about chronic gastritis, the issues of etiology and pathogenesis are considered. Contemporary classifications and the attitude of the authors of the work to them are presented. Clinical studies were conducted in 50 patients with chronic antral reflux-gastritis biliary (32 women and 18 men) between the ages of 20 and 80 years (av
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36

De Giacomo, C., R. Fiocca, L. Villani, et al. "Helicobacter pylori Infection and Chronic Gastritis." Journal of Pediatric Gastroenterology and Nutrition 11, no. 3 (1990): 310–16. http://dx.doi.org/10.1002/j.1536-4801.1990.tb10118.x.

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SummaryTwenty‐three children with Helicobacter (Campylobacter) pylori‐associated chronic gastritis are reported. Family history of peptic disease, previous digestive procedures, and nonspecific epigastric pain were the most frequently encountered clinical features. Antral nodularity at endoscopy and histologic evidence of follicular gastritis were characteristic morphological aspects. Rapid urease tests suggested the diagnosis in 90% of patients. Significant increases of serum IgG and IgA against Helicobacter pylori allowed the identification of infected children with 95% cumulative sensitivit
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37

Diah, Lestari, Nurhikmah, and Setiawan Lyana. "Analysis of the Relationship between Inflammatory Activity and Fecal Calprotectin Levels and Fecal Occult Blood as Biomarkers for Gastritis Sufferers." INTERNATIONAL JOURNAL OF PHARMACEUTICAL AND BIO-MEDICAL SCIENCE 04, no. 09 (2024): 730–35. https://doi.org/10.5281/zenodo.13710859.

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Gastritis is a disease in the form of inflammation of the stomach. Inflammatory activity in gastritis sufferers can be detected through biomarker examinations such as fecal calprotectin and fecal occult blood tests (FOB). This study was to analyze the relationship between inflammatory activity and fecal calprotectin levels and occult blood as a biomarker in chronic gastritis sufferers. The research method uses a cross-sectional design, Chi-Square statistical test data analysis, with a confidence level of 95% (&alpha; 0.05). The research used secondary data with a sample size of 64 people suffe
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Segarra, Chanlir, and Wojciech Blonski. "S3589 Lymphocytic Gastritis: A Rare Form of Chronic Gastritis." American Journal of Gastroenterology 115, no. 1 (2020): S1853. http://dx.doi.org/10.14309/01.ajg.0000716404.26235.85.

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39

Brown, Joanne, and Harold Lukse. "7. Russell body gastritis – a lesser known chronic gastritis." Pathology 47 (2015): S102—S103. http://dx.doi.org/10.1097/01.pat.0000461616.42266.57.

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40

Livzan, M. A., O. V. Gaus, and S. I. Mozgovoi. "Chronic atrophic gastritis: patient manage." Russian Medical Inquiry 5, no. 6 (2021): 427–32. http://dx.doi.org/10.32364/2587-6821-2021-5-6-427-432.

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Patient management in chronic atrophic gastritis (CAG) in real clinical practice is a difficult task for a clinician. It is mainly due to the lack of reliable clinical stigmas that allow suspecting the presence of gastric mucosal atrophy. The diagnosis of chronic atrophic gastritis is valid only after a morphological assessment of gastrobiopaths taken during an endoscopy. According to a contemporary view, regardless of the inflammatory process etiology, CAG can progress to stomach cancer. At the same time, the point of no-return (at which the risk of inflammatory changes progression in the gas
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Chikulaeva, Elena V., Valentina F. Melnikova, and Ruslan A. Nasyrov. "Lesion of the mucous membrane of the stomach, caused by chlamydia in children with chronic glomerulonephritis." Pediatrician (St. Petersburg) 8, no. 3 (2017): 51–56. http://dx.doi.org/10.17816/ped8351-56.

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We studied biopsies of gastric mucosa and the gastric antrum bodies obtained from 60 children with chronic gastritis and chronic gastritis in combination with chronic glomerulonephritis. Clinical data of patients (age, gender, duration of the underlying disease, conducted therapy, the results of laboratory and instrumental studies) were studied. It conducts research using histological and immunohistochemical methods. A morphological study of the sections found gastrobiopsies characteristic manifestations of chronic gastritis c signs of chlamydial lesions. The immunohistochemical method determi
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Tertychnyy, А. S., Т. L. Lapina, D. Р. Nagornaya, et al. "Spectrum of Chronic Gastritis Based on Morphological Examination of Gastric Biopsies." Russian Journal of Gastroenterology, Hepatology, Coloproctology 34, no. 2 (2024): 45–56. http://dx.doi.org/10.22416/1382-4376-2024-34-2-45-56.

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Aim: in the retrospective study to assess the frequency of various gastritis variants based on the results of morphological examination of gastric biopsies over a 5-year observation period.Methods. The study included 3162 individuals who underwent esophagogastroduodenoscopy with biopsy collection between 2017 and 2022. Pathological examination of biopsies was conducted using the updated Sydney system, determining the histological variant and, when possible, the etiology of gastritis. In some cases, chronic gastritis was assessed using the OLGA/OLGIM system.Results. The most frequently diagnose
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Tkach, S. M., N. V. Kharchenko, A. E. Dorofieiev, Yu Z. Hurkalo, and L. M. Ryzhiy. "Current concepts of chronic gastritis and gastropathies: new international consensus RE.GA.IN." GASTROENTEROLOGY 58, no. 4 (2024): 286–94. https://doi.org/10.22141/2308-2097.58.4.2024.641.

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Recent decades have been marked by several important milestones in the field of gastritis. New epidemiological reports were received, especially from countries with a high incidence of gastric cancer, and H.pylori infection was recognized as the most important cause of gastritis, due to which it was quickly included in the primary infectious oncogenic agents. Clinical strategies for the detection and eradication of H.pylori infection were developed, the previously unsuspected clinical and pathological significance of gastric microbiota was established, and extraordinary progress was made in th
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Kim, Na Young, Jin Lee, Ji Eun Na, Yong Eun Park, Jongha Park, and Tae Oh Kim. "Chronic Eosinophilic Gastritis Presenting as Gastric Outlet Obstruction in a Young Man: A Case Report." Korean Journal of Helicobacter and Upper Gastrointestinal Research 25, no. 2 (2025): 172–77. https://doi.org/10.7704/kjhugr.2025.0006.

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Eosinophilic gastritis, a relatively rare condition characterized by eosinophilic infiltration of the gastrointestinal wall, has a chronic course involving relapses and remissions. Herein, we describe the case of a 25-year-old man with chronic eosinophilic gastritis complicated by gastric outlet obstruction. The patient had a history of persistent gastrointestinal symptoms since adolescence and had developed severe dyspepsia and vomiting two months before presentation. Eosinophilic gastritis was confirmed using endoscopy and biopsy. The symptoms improved after steroid treatment, although the m
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Alakoski, Anna, Teea T. Salmi, Kaisa Hervonen, et al. "Chronic Gastritis in Dermatitis Herpetiformis: A Controlled Study." Clinical and Developmental Immunology 2012 (2012): 1–5. http://dx.doi.org/10.1155/2012/640630.

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Background and Objective. Previous small studies suggest that chronic atrophic gastritis is common in dermatitis herpetiformis (DH). We here examined the frequency and topography of chronic gastritis in 93 untreated DH subjects and in 186 controls with dyspepsia.Methods. Specimens were drawn from the gastric corpus and antrum and examined for atrophy, intestinal metaplasia, andHelicobacter pylori. Duodenal biopsies were taken.Results. Atrophic corpus gastritis was more frequent in DH than in controls (16.0% and 2.7%, resp.,P&lt;0.001); atrophy in the antrum was rare in both groups (3.2% and 1.
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Barbosa, A. J. A., D. M. M. Queiroz, A. M. M. F. Nogueira, et al. "Chronic gastritis and Helicobacter pylori in digestive form of Chagas' disease." Revista do Instituto de Medicina Tropical de São Paulo 35, no. 2 (1993): 117–21. http://dx.doi.org/10.1590/s0036-46651993000200002.

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Patients with the digestive form of Chagas'disease frequently present chronic gastritis. As the microorganism Helicobacter pylori is now accepted as the most common cause of human chronic gastritis, the present work was undertaken to verify a possible relationship between the presence of this bacterium and inflammatory changes of antral mucosa in chagasic patients. Seventeen chagasics, with megaesophagus and or megacolon were studied. Fragments from two different regions of antral mucosa were obtained by endoscopy, fixed in 4% neutral formaldehyde and embedded in paraffin. The sections were st
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Safira, Fathi Rahmah, and Sugiarto Sugiarto. "Histopathological Features of the Gastric Mucosa in Patients with Chronic Gastritis and Helicobacter pylori Infection at Pertamina Central Hospital Jakarta." Muhammadiyah Medical Journal 2, no. 2 (2021): 70. http://dx.doi.org/10.24853/mmj.2.2.70-74.

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Background: Chronic gastritis is a chronic inflammation of the gastric mucosa, accompanied by changes in mucosal histology with or without Helicobacter pylori infection. Changes in the gastric mucosa include gastric mucosal atrophy, intestinal metaplasia, and epithelial dysplasia. Purposes: This study aims to determine the microscopic appearance of the mucosa in chronic gastritis patients based on standard histopathological criteria, which include gland atrophy, intestinal metaplasia, dysplasia with or without Helicobacter pylori infection at Pertamina Central Hospital Jakarta period 2018 - 20
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Mazhar, Syeda Ayeman. "Management and Treatment of Gastritis (War’m-e-Meda) with Herbal Remedies: An Overview." Journal of Integrated Community Health 11, no. 1 (2022): 22–24. http://dx.doi.org/10.24321/2319.9113.202203.

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Stomach complaints are still a common health problem. Among them, gastritis affects 25-33% of the total population of India, of which most of them belong to the age group of 15-50 years. There are two types of gastritis - acute and chronic. Acute gastritis is a mucosal inflammatory process that may be asymptomatic. Chronic gastritis is one of the most common serious epidemic infections, e.g. peptic ulcer or gastric ulcer. In the Unani system of medicine, gastritis is also known by many names: Hurqat-e-Meda, Sozish-e-Meda, Warm-e-Meda, and Ilttehab-e-Meda. Gastritis is most commonly caused due
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Ma, Ting, and Yahui Huang. "Application of the Theory of "Treating the Future Disease" in Spleen and Stomach Damp-heat Type Chronic Atrophic Gastritis: an Investigation." Journal of Contemporary Medical Practice 6, no. 7 (2024): 132–36. http://dx.doi.org/10.53469/jcmp.2024.06(07).27.

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Chronic atrophic gastritis is a kind of digestive disease which is easy to recur and difficult to be cured. Damp-heat syndrome of spleen and stomach is one of the main syndromes of chronic atrophic gastritis, and the intractability of this disease determines the importance of its prevention and treatment. In this paper, we analyze the prevention and treatment of chronic atrophic gastritis of the damp-heat spleen and stomach type from three aspects of the theory of "treating the disease before it gets worse", and elaborate the etiology of chronic atrophic gastritis of the damp-heat spleen and s
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Gubanova, A. V., M. A. Livzan, T. S. Krolevets, S. I. Mozgovoi, A. V. Rubtsov, and M. A. Stepanchenko. "Autoimmune gastritis and stomach cancer: assessing the risks." Experimental and Clinical Gastroenterology, no. 3 (July 25, 2023): 112–19. http://dx.doi.org/10.31146/1682-8658-ecg-211-3-112-119.

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The purpose of this publication is to systematize available data on the risks of developing stomach cancer in patients with a chronic autoimmune gastritis with a demonstration of the clinical case of a patient with a chronic autoimmune gastritis and a neuroendocrine gastric tumor of the type 1. Discussion: the article discusses the risks of stomach cancer in patients with chronic autoimmune gastritis. A mechanism for the formation of a neuroendocrine gastric tumor of the type 1, associated with autoimmune gastritis, is given. A clinical example of a patient with a long history of dyspepsia, th
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