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Relevant bibliographies by topics / Chronic inflammatory / Dissertations / Theses

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Dissertations / Theses on the topic 'Chronic inflammatory'

Author: Grafiati

Published: 5 June 2025

Last updated: 2 August 2025

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1

Cauli, Alberto. "The inflammatory milieu in chronic arthritis." Thesis, King's College London (University of London), 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.322233.

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2

Cexus, Olivier. "Immunological mechanisms controlling chronic inflammatory diseases." Thesis, University of Southampton, 2009. https://eprints.soton.ac.uk/67634/.

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Autoimmune diseases (AID) are chronic inflammatory diseases (CID) mediated by selfreactive T and B cells and are generally the results of the breakdown of T cell tolerance to self-antigen and failure of peripheral regulatory mechanisms. In this thesis I studied different mechanisms controlling the development of CIDs. I investigated the initial events involved in the activation of self-reactive CD4+ T cells which mediate the destruction of the thyroid in a mouse model of spontaneous thyroiditis. TAZ10 transgenic mice express a human T cell receptor (TCR) specific for a cryptic epitope of thyro

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3

Hood, Vivienne Claire. "Neurogenic influences on chronic inflammatory joint disease." Thesis, Queen Mary, University of London, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.252151.

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4

Roberts, Helen Michelle. "Neutrophil function in chronic inflammatory disease states." Thesis, University of Birmingham, 2016. http://etheses.bham.ac.uk//id/eprint/7018/.

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Inflammation is a central component of the immune response. In its acute form it aids the transition from disease to health via the activation of numerous immune cells, enabling them to reach the site of infection/injury and orchestrate themselves to combat pathogens, facilitating resolution and repair to restore the host to health. However, chronic inflammation is deleterious to the host and differs from the “classical” acute inflammatory process in that the inflammation is not necessarily so readily obvious and is not self-limiting; rather, the immune system is in a constant state of low-gra

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5

Yan, Wei Xing. "Target antigen in chronic inflammatory demyelinating polyradiculoneuropathy." Thesis, The University of Sydney, 2001. https://hdl.handle.net/2123/27718.

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The primary purposes of this study were to investigate humoral factors in chronic inflammatory demyelinating polyradiculoneuropathy (CIDP), to determine pathogenicity of the factors found, and to detect possible target antigens in CIDP. Sera from 22 CIDP patients of whom 12 responded to plasma exchange therapy and 10 needed other therapy, were tested by means of enzyme-linked immunosorbent assay, immunofluorescence and Western blot techniques. Anti-whole myelin, anti-myelin glycoproteins (e.g. P0, P2, PMP22 and myelin associated glycoprotein), anti-myelin glycolipids (e.g. GMl, GM2, Gl

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6

Thornton, Catherine Clare. "Mechanisms of vascular protection in chronic inflammatory disease." Thesis, Imperial College London, 2014. http://hdl.handle.net/10044/1/26595.

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Premature cardiovascular disease (CVD) complicates chronic systemic inflammatory disease, and is initiated by endothelial dysfunction. Despite an ever-improving array of medications to treat these diseases, how to prevent CVD is not known. Studying the actions of endogenous mediators of endothelial cytoprotection and of disease-modifying drugs might establish how best to protect patients with systemic inflammatory diseases from atherosclerosis, if specific beneficial effects on vascular endothelium could be demonstrated. High flow laminar shear stress (LSS) and vascular endothelial growth fact

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7

Raunio, T. (Taina). "Gene polymorphism and systemic inflammatory response in chronic periodontitis." Doctoral thesis, University of Oulu, 2009. http://urn.fi/urn:isbn:9789514292361.

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Abstract In this study, associations between periodontitis expression, serum levels of inflammatory markers and genetic factors were investigated. The periodontal status of 56 subjects with chronic periodontitis, 28 control subjects and 80 subjects with type I diabetes mellitus (DM) was examined. In addition, a reference group (n=178) with genetic but not with periodontal health data was included. The single nucleotide polymorphisms of CD14 -260, IL-6 -174, TNF-α -308, IL-10 -1082, IL-1A -889, IL-1B +3954, and TLR4 +896 were determined using PCR with RFLP or allele-specific primers, and compar

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8

Saha, Shironjit K. "Inflammatory profiles in chronic obstructive pulmonary disease and asthma." Thesis, University of Leicester, 2010. http://hdl.handle.net/2381/8206.

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Historically, asthma and chronic obstructive pulmonary disease (COPD) represent polar ends of the spectrum of airways disease defined in part by distinctive profiles of airway inflammation; in practice, overlap can exist between asthma and COPD. This thesis examined the pattern of inflammatory cell infiltration and cytokine expression within the bronchus in COPD and asthma with further study of moderate-severe asthma. In addition using sputum, cytokine expression was further assessed in COPD and asthma and its relation to severity. Based on previous studies, this thesis examined the expression

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9

Arsenescu, Violeta. "ROLE OF ARYL HYDROCARBON RECEPTOR IN CHRONIC INFLAMMATORY DISEASES." UKnowledge, 2009. http://uknowledge.uky.edu/gradschool_diss/772.

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Aryl Hydrocarbon Receptor (AhR) is a ligand-actviated receptor known as the dioxin receptor. Environmental pollutants called dioxin-like toxicants are found in food, cigarette smoke, automobile exhaust and air. Therefore, they could chronically amplify the pathology of numerous chronic inflammatory diseases. AhR is a well known target of these environmental chemicals that disrupt endocrine signaling. By the year 2020, the number of people older than 60 years is expected to top 1 billion. The burden of treating chronic disease is significant both in dollars spent and in lost productivity. The n

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10

Bethune, Jörn [Verfasser]. "Network-Assisted Analysis of Chronic Inflammatory Diseases / Jörn Bethune." Kiel : Universitätsbibliothek Kiel, 2016. http://d-nb.info/1118499875/34.

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11

German, Alexander James. "Immunological studies of chronic enteropathies in dogs." Thesis, University of Bristol, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.313080.

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12

Wallwork, Benjamin, and n/a. "The Anti-Inflammatory Effect of Macrolide Antibiotics in Chronic Rhinosinusitis." Griffith University. School of Biomolecular and Biomedical Science, 2006. http://www4.gu.edu.au:8080/adt-root/public/adt-QGU20070201.160023.

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Chronic rhinosinusitis is a common disorder of chronic inflammation of the upper respiratory tract. It is associated with significant symptoms and impairment of the quality of life of sufferers. Despite recent advances in the medical and surgical management of chronic rhinosinusitis, there remains a population of patients who fail to obtain relief from their symptoms. Chronic inflammation of the mucosa of the nasal cavity and paranasal sinuses is one of the hallmarks of chronic rhinosinusitis. This inflammation is demonstrated by an increased number of chronic inflammatory cells, elevated leve

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13

Press, Rayomand. "Immunopathogenesis of Guillain-Barré syndrome and chronic inflammatory demyelinating polyradiculoneuropathy /." Stockholm, 2002. http://diss.kib.ki.se/2002/91-7349-378-3/.

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14

Tarlow, Joanna Karen. "Interleukin-1 receptor antagonist gene polymorphism in chronic inflammatory diseases." Thesis, University of Sheffield, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.296761.

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15

Labrousse, Delphine. "Characterisation of myeloid-derived suppressor cells in chronic inflammatory diseases." Thesis, University of Southampton, 2010. https://eprints.soton.ac.uk/385140/.

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16

Wallwork, Benjamin. "The Anti-Inflammatory Effect of Macrolide Antibiotics in Chronic Rhinosinusitis." Thesis, Griffith University, 2006. http://hdl.handle.net/10072/367299.

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Chronic rhinosinusitis is a common disorder of chronic inflammation of the upper respiratory tract. It is associated with significant symptoms and impairment of the quality of life of sufferers. Despite recent advances in the medical and surgical management of chronic rhinosinusitis, there remains a population of patients who fail to obtain relief from their symptoms. Chronic inflammation of the mucosa of the nasal cavity and paranasal sinuses is one of the hallmarks of chronic rhinosinusitis. This inflammation is demonstrated by an increased number of chronic inflammatory cells, elevated leve

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17

RAVANI, Annalisa. "Pharmacological characterization of adenosine receptors in chronic inflammatory rheumatic diseases." Doctoral thesis, Università degli studi di Ferrara, 2018. http://hdl.handle.net/11392/2478762.

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L’artrite reumatoide, la spondilite anchilosante e l’artrite psoriasica sono malattie infiammatorie croniche, progressive e invalidanti che colpiscono le articolazioni provocando dolore e disabilità. I recettori dell’adenosina giocano un ruolo fondamentale nel meccanismo infiammatorio, in particolare l’attivazione dei sottotipi recettoriali A2A e A3 è spesso associata ad una riduzione dello stato infiammatorio. Il primo obiettivo di questo studio è stato quello di indagare il coinvolgimento dei recettori adenosinici nei pazienti affetti da artrite reumatoide all’esordio della patologia (non an

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18

Murphy, David Matthew. "Studies on chronic gastrointestinal disease in the horse." Thesis, University of Glasgow, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.360253.

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19

Gardiner, Philip Victor. "An evaluation of bronchoalveolar lavage methodology and its role in the investigation of the pulmonary complications of primary SjoÌˆegren's Syndrome". Thesis, Queen's University Belfast, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.333775.

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20

Allen, David Andrew. "The role of inflammatory cytokines in resistance to erythropoietin in chronic renal failure." Thesis, Queen Mary, University of London, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.394275.

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21

Kotecha, Sailesh. "The role of cytokines in chronic lung disease of prematurity." Thesis, Imperial College London, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.244032.

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22

Dastmalchi, Maryam. "Studies of immunopathogenic mechanisms and treatment of chronic, inflammatory myopathies, myositis /." Stockholm : Karolinska institutet, 2007. http://diss.kib.ki.se/2007/978-91-7357-323-8/.

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23

Allen, Derek Creswell. "The pathology of malignant mucosal changes in chronic inflammatory bowel disease." Thesis, Queen's University Belfast, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.356854.

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24

Burman, Angela Claire. "The role of leukocyte-stromal intractions in chronic inflammatory joint disease." Thesis, University of Birmingham, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.412099.

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25

Isebor, Peter. "Positive psychological interventions in chronic conditions : gratitude and inflammatory bowel disease." Thesis, University of Sheffield, 2018. http://etheses.whiterose.ac.uk/21308/.

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26

Böck, Janne [Verfasser]. "The role of ATG16L1 in chronic inflammatory bowel disease / Janne Böck." Kiel : Universitätsbibliothek Kiel, 2012. http://d-nb.info/1053683316/34.

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27

Bhuiya, Sumita. "The impact of inflammatory genetics on oocyte viability and embryo development in women with chronic pelvic inflammatory disease." Thesis, University of Leeds, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581454.

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Infection by Chlamydia trachomatis is the leading cause of tubal disease and accounts for a significant proportion of the physical, psychosocial and economic burden of infertility. As it is frequently asymptomatic and opportunities for early treatment are missed, repeated/chronic infection leads to permanent/irreversible inflammation- induced tubal damage, which may extend as far as the ovary. However, not all infected women develop tubal pathology, possibly due to inherent host-specific and environment-induced differences in immune response. This study investigated the impact of putative cyto

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28

Williams, David Leonard. "Canine chronic superficial keratitis : histochemical characterisation and clinical management." Thesis, Royal Veterinary College (University of London), 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.307438.

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29

El-Hashim, Ahmed Zakaria. "Characterisation of acute and chronic airway hyperresponsiveness in an allergic rabbit model." Thesis, King's College London (University of London), 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.286197.

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30

Nesheim, Nils [Verfasser]. "Inflammatory Gene Regulation in Chronic Prostatitis / Chronic Pelvic Pain Syndrome (CP/CPPS) and Prostate Cancer / Nils Martin Nesheim." Gießen : Universitätsbibliothek, 2020. http://d-nb.info/1216142742/34.

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31

Nesheim, Nils Martin [Verfasser]. "Inflammatory Gene Regulation in Chronic Prostatitis / Chronic Pelvic Pain Syndrome (CP/CPPS) and Prostate Cancer / Nils Martin Nesheim." Gießen : Universitätsbibliothek, 2020. http://d-nb.info/1216142742/34.

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32

Myers, Andrea. "The recurrence of different patterns of psoriatic arthritis within sibships." Thesis, University of Newcastle upon Tyne, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.289199.

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33

Mitchison, Harriet Caroline. "Primary biliary cirrhosis : studies in prognosis, early diagnosis, bone disease and treatment." Thesis, University of Newcastle Upon Tyne, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.241281.

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34

Bevan, Damon. "Cytokine involvement in ultraviolet (UV) B induced chronic and acute inflammation in porcine skin." Thesis, Royal Veterinary College (University of London), 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.300963.

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35

Angsana, Julianty. "The role of syndecan-1 in the resolution of chronic inflammatory responses." Diss., Georgia Institute of Technology, 2013. http://hdl.handle.net/1853/52947.

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Inflammation is an integral part of the body defense mechanism that occurs in vascularized tissue in response to harmful stimuli that is perceived as being a threat to tissue homeostasis. It is a complex physiological host response that is designed to neutralize and eliminate harmful agents, initiate tissue healing, and orchestrate a return to tissue homeostasis. While inflammation is designed to be an acute event that resolves following the elimination of harmful stimuli and tissue healing, there are instances where inflammation fails to resolve and instead evolves into chronic inflammation

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36

Leung, Bernard P. "Role of interleukin-15 and nitric oxide expression in chronic inflammatory disease." Thesis, University of Glasgow, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.298919.

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37

Hale, Elizabeth Dorothy Pamela. "Children of parent's with chronic inflammatory musculoskeletal diseases : experiences, needs and resources." Thesis, University of Leicester, 2017. http://hdl.handle.net/2381/40394.

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Overview: Whilst each section of the thesis is required to stand alone, it is presented in the following order so that the thesis has a logical flow. Literature review: Presents an overview of the rheumatic diseases and their impact upon parenting and families. We note the provision of patient education for parents, but not for their children. The comprehensive but selective narrative review focuses upon how adults and children conceptualise and understand illness, and explores the concept of normalisation as a potential family management strategy. Service Evaluation: We sought to establish wh

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38

Thompson, Melissa. "Anti-Inflammatory Properties of Xylitol in a Model of Chronic Sinus Disease." Thesis, Griffith University, 2013. http://hdl.handle.net/10072/367231.

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Background. Chronic rhinosinusitis is a highly prevalent condition estimated to affect 9% of the adult population in Australia. It is responsible for a significant burden of disease at a population level and imparts considerable morbidity and reduced quality of life to affected individuals. Recent American data has suggested an $8.6 billion annual direct healthcare cost associated with chronic rhinosinusitis. Few medical options for treating chronic rhinosinusitis exist and there is a paucity of evidence in support of a best medical treatment. Currently available treatments confer a modest ben

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39

Gaspardo, Alba <1991&gt. "Canine chronic inflammatory enteropathies: diagnostic markers, endoscopic tecniques and novel therapeutic approaches." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2021. http://amsdottorato.unibo.it/9696/1/Gaspardo_Alba_-_Tesi_dottorato_XXXIII_ciclo.pdf.

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Canine chronic inflammatory enteropathies (CIE) are a group of diseases characterized by chronic gastrointestinal (GI) symptoms. Once other causes for GI symptoms have been ruled out, subdivision of CIEs occurs through therapeutic response which involves a unique trial. The therapeutic steps include a dietary change, subsequently the introduction of antibiotic therapy (this step has recently been discouraged), the execution of an endoscopy of the digestive tract and the introduction of an immunosuppressive therapy. Understanding the pathogenesis and the type of CIE in is fundamental for a corr

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40

De, Pellegrin Annamaria. "Role of ANCA in necrotizing vasculitis and in chronic inflammatory intestinal disease." Doctoral thesis, Università degli studi di Padova, 2008. http://hdl.handle.net/11577/3426734.

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Background: Wegener's granulomatosis (WG), Microscopic Poliangitis and Churg-Strauss Syndrome, the necrotizing vasculitis, are a group of inflammatory diseases of the wall small-medium calibre blood vessels. This group of diseases are hystologically characterized by the presence of necrotic tissue with a consequent inflammatory infiltration and the formation of granulomas. GW is a disease which has periods of remission and recurrence treated by therapeutic cycles associated with several side-effects. It is caracterized by the presence in serum of anti-neutrophil cytoplasm antibodies (ANCA).

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41

Andreson, Helena. "Diversity of Helicobacter pylori genotypes in Estonian patients with chronic inflammatory gastric diseases /." Online version, 2006. http://dspace.utlib.ee/dspace/bitstream/10062/780/5/andresonhelena.pdf.

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42

Rogers, Andrew Vincent. "Inflammatory cell distribution in human bronchial mucosa : influence of chronic and acute disease." Thesis, Imperial College London, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.440463.

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43

Rodgers, Lewis Craig. "Metabolic control for inflammatory cascades in monocytes in response to chronic disease stimuli." Thesis, University of Glasgow, 2018. http://theses.gla.ac.uk/30725/.

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Tissue microenvironments within chronic inflammatory disease sites, such as the synovial compartment within rheumatoid arthritis (RA) patients contains a plethora of factors to drive immune responses. However, one specific characteristic that is commonly found at these sites is the presence of tissue hypoxia. Therefore, both resident and infiltrating immune cells need to adapt to these microenvironments in order to survive and function to promote chronic inflammation. Adaptation to hypoxia requires a level of metabolic reprogramming for this purpose. Therefore, this thesis aimed to examine wha

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44

Holloway, Rebecca Anne. "The role of elastase as an inflammatory stimulus in chronic obstructive pulmonary disease." Thesis, University of Southampton, 2010. https://eprints.soton.ac.uk/162673/.

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Chronic obstructive pulmonary disease (COPD) is a debilitating disease that as of yet has no cure and therapy is limited to symptomatic relief. A major risk factor for the development of COPD is smoking, although the disease does have some component of genetic predisposition. In excess of £500 million funding per year is required to accommodate the needs of COPD patients and approximately 27, 000 deaths per year in the UK can be attributed to COPD. COPD is comprised of three conditions-chronic bronchitis, bronchiolitis and emphysema and these will be present in the COPD patient to varying degr

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45

Schaefer, Charles, and Charles Schaefer. "Peripheral Inflammatory Pain and P-Glycoprotein in a Model of Chronic Opioid Exposure." Thesis, The University of Arizona, 2017. http://hdl.handle.net/10150/624091.

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The rates of opioid prescription and use have continued to increase over the last few decades. In turn, a greater number of patients suffer from opioid tolerance. Treatment of acute pain is a clinical challenge for these patients. Acute pain can arise from common occurrences like surgical pain and pain resulting from the injury. P-glycoprotein (p-gp) is a transporter at the blood-brain barrier (BBB) associated with a decrease in the analgesic efficacy of morphine. Peripheral inflammatory pain (PIP) is a pain state known to cause a change in p-gp trafficking at the BBB. P-gp traffics from the n

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46

Ricciardelli, I. "Mechanisms of immune activation and regulation in chronic inflammatory diseases of the gut." Thesis, University College London (University of London), 2008. http://discovery.ucl.ac.uk/1446058/.

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The gastrointestinal (GI) tract is the largest surface area of the body exposed to the external environment and it is subject to constant challenge from micro-organisms and food-derived antigens. Stringent regulation of the immune system of the gut is vital, as breakdown in tolerance to commensal bacteria in Inflammatory Bowel Diseases (IBD, such as Crohn's disease and Ulcerative Colitis (UC)) or to dietary antigens such as gluten in Celiac disease (CD) can lead to inflammation. Hence induction and maintenance of mucosal tolerance is of paramount importance to avoid inappropriate immune respon

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47

Stupar, Dario. "Longitudinal assessment of inflammatory factors in samples from patients with chronic leg ulcers." Thesis, Queensland University of Technology, 2016. https://eprints.qut.edu.au/96580/1/Dario_Stupar_Thesis.pdf.

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Chronic leg ulcers are a major health problem that affect 1-3% of adults over 65 and are a financial burden on healthcare systems worldwide. In this study biochemical assays were performed on sequential samples collected from both healing and non-healing ulcers. The results demonstrated that inflammatory resolution was necessary for ulcers to heal. Interestingly, in healing ulcers, proteins associated with dampening inflammation did not change over time whilst those keeping the wound in an inflammatory state decreased, suggesting an alternative mechanism for resolution of inflammation.

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48

White, C. Michael, Vinay Pasupuleti, Yuani M. Roman, Yangzhou Li, and Adrian V. Hernandez. "Oral turmeric/curcumin effects on inflammatory markers in chronic inflammatory diseases: A systematic review and meta-analysis of randomized controlled trials." Academic Press, 2019. http://hdl.handle.net/10757/652467.

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Turmeric extract or active component curcumin may have anti-inflammatory effects in people with chronic inflammatory diseases. The effect of turmeric or curcumin on a wide range of inflammatory markers has not been evaluated in a systematic review. We performed a systematic review of randomized controlled trials (RCTs)evaluating the effects of oral turmeric or curcumin on inflammatory markers (CRP, hsCRP, IL-1, IL-6, TNF)in patients with a wide range of chronic inflammatory diseases. Pubmed, EMBASE, Scopus, the Web of Science, and the Cochrane library were evaluated until June 2018. Random eff

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49

Ryu, Jae Kyu. "Microglial-mediated inflammatory responses and perturbed vasculature in an animal model of inflamed Alzheimer's disease brain." Thesis, University of British Columbia, 2008. http://hdl.handle.net/2429/2641.

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Chronic inflammation in response to Aß peptide deposits is a pathological hallmark of Alzheimer's disease (AD). The inflammatory environment includes populations of reactive and proliferating microglia and astrocytes and perturbed vasculature. However, the association between activated glial cells and cerebrovascular dysfunction remain largely unknown. This study has used Aß1-42 intrahippocampal injection as an animal model of inflamed AD brain to characterize mechanisms of glial-vasculature responses as a basis for chronic inflammation. Preliminary findings suggested Aß1-42-injected brain dem

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50

Momani, Aiman A. "Assessment of the impact of the West Virginia Medicaid's prior authorization policy for NSAIDs on chronic patients economic and humanistic outcomes /." Morgantown, W. Va. : [West Virginia University Libraries], 1999. http://etd.wvu.edu/templates/showETD.cfm?recnum=847.

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Thesis (Ph. D.)--West Virginia University, 1999.<br>Title from document title page. Document formatted into pages; contains xii, 150 p. : ill. Includes abstract. Includes bibliographical references (p. 124-128).

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