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Journal articles on the topic 'Chronic non-atrophic gastritis'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

Pavlovich, I. M., G. A. Al’per, A. V. Gordienko, D. I. Proskunov та V. V. Yakovlev. "Сhronic gastritis: impaired motor-evacuation function of the stomach". Bulletin of the Russian Military Medical Academy 22, № 4 (2020): 37–42. http://dx.doi.org/10.17816/brmma62802.

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The effect of morphological changes in the gastric mucosa on the motor-evacuation function of the stomach was evaluated in 90 patients with chronic atrophic gastritis and 93 patients with chronic non-atrophic gastritis aged 18 to 82 years. Motor function disturbances were discovered of 90% of patients with chronic atrophic gastritis and 80,6% with chronic non-atrophic gastritis. It was established that the pyloric insufficiency compared with its spasm was significantly more (p0,01) often in chronic atrophic gastritis. Insufficiency of the lower esophageal sphincter in patients with atrophic ga

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2

Yuan, Mingyang. "Research status of risk factors of chronic atrophic gastritis." Theoretical and Natural Science 16, no. 1 (2023): 171–77. http://dx.doi.org/10.54254/2753-8818/16/20240566.

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Gastritis, also known as inflammation of the gastric mucosa, is a prevalent disorder of the digestive system that can be attributed to a range of reasons. Based on the level of illness severity, gastritis can be categorized into two distinct types: acute gastritis and chronic gastritis. Based on the observed pathological alterations, acute gastritis can be categorized into several subtypes, namely simple gastritis, erosive and hemorrhagic gastritis, corrosive gastritis, and suppurative gastritis. Based on the observed pathological alterations, chronic gastritis can be categorized into three di

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3

Osipenko, M. F., E. A. Bikbulatova, Yu V. Makarova, and V. P. Drobysheva. "Eternal intrigue of gastritis." RMJ, no. 10 (2024): 32–36. https://doi.org/10.32364/2225-2282-2024-10-6.

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The article discusses the issues of classification, etiology, pathogenesis, diagnosis and treatment of gastritis, including chronic gastritis (CG). Currently, CG is defined as a long-term and usually non-self-limiting inflammation of the gastric mucosa (GM). The disease commonly lasts for life with the constant impact of an etiological factor. The most common etiological factor of CG is Helicobacter pylori. Gastritis caused by H. pylori can progress from non-atrophic to atrophic with further progression to gastric cancer. The authors also consider other forms of CG, including autoimmune and dr

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4

Zhang, Xia, Mao Zhang, Gang Wei, and Jia Wang. "Research on Predictive Auxiliary Diagnosis Method for Gastric Cancer Based on Non-Invasive Indicator Detection." Applied Sciences 14, no. 16 (2024): 6858. http://dx.doi.org/10.3390/app14166858.

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Chronic atrophic gastritis is a serious health issue beyond the stomach health problems that affect normal life. This study aimed to explore the influencing factors related to chronic atrophic gastritis (CAG) using non-invasive indicators and establish an optimal prediction model to aid in the clinical diagnosis of CAG. Electronic medical record data from 20,615 patients with CAG were analyzed, including routine blood tests, liver function tests, and coagulation tests. The logistic regression algorithm revealed that age, hematocrit, and platelet distribution width were significant influences s

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Lin, Mei, Wenmin YUAN, Xiaohui SUN, and Zhiqiang QIN. "Expression and Significance of Beclin-1 and Bcl-2 in Gastric Cancer." International Journal of Sciences Volume 5, no. 2015-12 (2016): 58–61. https://doi.org/10.5281/zenodo.3349067.

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Objective To investigate the role and correlation of Beclin-1 and Bcl-2 in the occurrence and progression of gastric cancer. Methods The expression of Beclin-1 and Bcl-2 in 78 cases of gastric cancer, 30 cases of atypical hyperplasia and 20 cases of chronic non-atrophic gastritis was examined by immunohistochemical staining. Results The positive rate of Beclin-1 in gastric cancer was significantly lower than those in atypical hyperplasia and chronic non-atrophic gastritis(χ2=9.298,7.437, P<0.05), while there was no significant difference between atypical hyperplasia and chronic non-atrophic

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Bulgar, K. I., I. M. Pavlovich, A. S. Parcernyak, and B. A. Chumak. "The effect of <i>Helicobacter pylori</i> on the morphological picture of the gastric mucosa in patients with chronic gastritis." Experimental and Clinical Gastroenterology, no. 12 (December 18, 2023): 19–26. http://dx.doi.org/10.31146/1682-8658-ecg-220-12-19-26.

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Helicobacter pylori (H. pylori) is one of the most common infections worldwide. According to modern ideas, it is the main cause of a number of diseases of the stomach and duodenum (peptic ulcer, chronic gastritis, MALT lymphoma) and the leading risk factor for stomach cancer. In the hospital therapy clinic of the Kirov Military Medical Academy, we observed 118 patients. The study groups included patients with various types of chronic gastritis (atrophic and non-atrophic). The patients were comparable in gender and age. Infection H. pylori was verified in 22 patients (68.8%) suffering from chro

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Dekhnich, Natalya N., Pavel Р. Kirsov, Inna A. Sysoeva, and Anastasiia S. Gaidaichuk. "Clinical case of autoimmune atrophic gastritis." Clinical review for general practice 5, no. 6 (2024): 6–11. http://dx.doi.org/10.47407/kr2024.5.6.00427.

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The article presents a clinical case of a 62-year-old female patient with a diagnosis of autoimmune atrophic gastritis, who has been troubled by symptoms of dyspepsia for 5 years. In October 2019, she was diagnosed non-atrophic gastritis associated with Helicobacter pylori, anti-Helicobacter pylori therapy was performed, but the symptoms of the disease continued to bother. This article presents key studies that identify autoimmune atrophic gastritis. The characteristic endoscopic signs of this disease and the current treatment tactics are described. Autoimmune gastritis is a chronic autoimmune

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8

Sablina, Anastasiya O., Oleg A. Sablin, Julia V. Andreeva, et al. "H. pylori eradication therapy: impact of gastric mucosa atrophy on transport of amoxicillin to H. pylori colonization area." HERALD of North-Western State Medical University named after I.I. Mechnikov 13, no. 2 (2021): 57–66. http://dx.doi.org/10.17816/mechnikov70280.

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AIM: The aim was to assess systemic transport of amoxicillin, the most common antibiotic in H. pylori eradication regimens to the gastric in atrophic gastritis. MATERIALS AND METHODS: Systemic transport of amoxicillin to the gastric lumen of rats was evaluated in washes from the gastric mucosa in the model of atrophic gastritis after intravenous drug infusion. Transport of amoxicillin from bloodstream to the gastric lumen was also assessed in patients with atrophic and non-atrophic gastritis in aspirated via nasogastric probe gastric juice after oral drug administration. Amoxicillin conc

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9

Avramenko, Anatoly A. "INFLUENCE OF PECULARITIES OF ANATOMICAL STRUCTURE (SHAPE) OF DUODENAL BULB ON THE MECHANISM OF FORMATION OF “KISSING” ULCERS OF PATIENTS WITH CHRONIC NON-ATROPHIC GASTRITIS." Wiadomości Lekarskie 73, no. 12 (2020): 2568–71. http://dx.doi.org/10.36740/wlek202012102.

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The aim: To determine of the factors influencing the formation of “kissing” duodenal bulb ulcer in patients with chronic non-atrophic gastritis. Materials and methods: The data of a comprehensive examination of 34 patients with chronic non-atrophic gastritis were analyzed, in which the examination revealed “kissing” ulcers of the duodenal bulb (primary group), and 37 patients with chronic non-atrophic gastritis, in which single ulcers were detected in the bulb (control group). Comprehensive examination included: step-by-step intragastric pH-metry, esophagogastroduodenoscopy, helicobacter infec

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10

Nasierowska-Guttmejer, A., and A. W. Szawlowski. "Immunohistochemical Detection of Carcinoembryonic Antigen (CEA) in Non-Cancerous and Cancerous Gastric Mucosa." International Journal of Biological Markers 4, no. 1 (1989): 8–12. http://dx.doi.org/10.1177/172460088900400102.

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Carcinoembryonic antigen (CEA) was stained by the PAP immunoperoxidase method in cancerous and non-cancerous gastric mucosa of 40 patients (25 non-cancerous dyspeptic patients and 15 patients with gastric carcinoma). The pattern of CEA localization was apical or membranous-cytoplasmic and immuno-reactivity was mild (+), moderate (++) or intensive (+++). No CEA immunoreactivity was detected in normal gastric mucosa whereas it was marked in gastric mucosa of non-cancerous dyspeptic patients with chronic atrophic gastritis and dysplasia (intense). In patients with superficial gastritis and epithe

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11

Zhumabaevna, Saule Akhmetova, Abdiraman Kaliev Rsalievich, Anarzhan Tulyaeva Balkashevna, et al. "Proliferation and Apoptosis in H. Pylori-associated Chronic Gastritis with Epithelial Metaplasia." West Kazakhstan Medical Journal 67, no. 1 (2025): 123–31. https://doi.org/10.18502/wkmj.v67i1.16911.

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Helicobacter pylori - causes pathologies such as gastritis, peptic ulcer disease, adenocarcinoma. Pseudopyloric mucosal metaplasia in the incisura region is significantly correlated with the development of precancerous changes - atrophy and intestinal metaplasia. Pseudopyloric metaplasia of the gastric mucosa develops as a result of persistence of H. pylori infection. Immunohistochemical and histological study of gastric mucosa biopsy specimens from patients with dyspeptic complaints was performed. Chronic non-atrophic gastritis was histologically detected in 21 patients. Of these, 17 (80.95%)

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Botezatu, Adriana, Nicolae Bodrug, and Viorel Istrate. "Precancerous gastric lesions: pathophysiology and symptomatology." Moldovan Medical Journal 63(5) (November 1, 2020): 62–67. https://doi.org/10.5281/zenodo.4018962.

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<strong>Background</strong>: Independent risk factors for chronic atrophic gastritis, gastric intestinal metaplasia and gastric cancer are: Helicobacter pylori infection (especially virulent CagA strains), genetic factors (advanced age, reflecting the duration of Helicobacter pylori infection, male gender, family history of gastric cancer in first-degree relatives), gastric ulcer, biliary reflux, acute or chronic gastric inflammation, smoking, alcohol consumption, prolonged use of proton pump inhibitors or non-steroidal anti-inflammatory drugs, diet low in fruits, vegetables and vitamin C, exc

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13

Farinati, F., R. Cardin, P. Degan, et al. "Oxidative DNA damage accumulation in gastric carcinogenesis." Gut 42, no. 3 (1998): 351–56. http://dx.doi.org/10.1136/gut.42.3.351.

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Background—Gastric carcinogenesis is a multifactorial, multistep process, in which chronic inflammation plays a major role.Aims—In order to ascertain whether free radical mediated oxidative DNA damage is involved in such a process, concentrations of 8-hydroxydeoxyguanosine (8OHdG), a mutagenic/carcinogenic adduct, and thiobarbituric acid reactive substances (TBARS), as an indirect measure of free radical mediated damage, were determined in biopsy specimens from patients undergoing endoscopy.Patients—Eighty eight patients were divided into histological subgroups as follows: 27 with chronic non-

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14

Sukhan, D., and S. Vernigorodskiy. "Prognostic aspects of immunohistochemical assessment of cellular renewal and transcription factor SOX2 with precancerous changes in the gastric mucosa depending on the genotype of Helicobacter pylori in patients with various types of chronic gastritis." Reports of Vinnytsia National Medical University 22, no. 4 (2018): 674–81. http://dx.doi.org/10.31393/reports-vnmedical-2018-22(4)-18.

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The precancerous potential of chronic gastritis (CG) associated with H. pylori is discussed in numerous writings, and today, CG is central to precancerous conditions of the stomach. A convincing theoretical basis for such an assessment of chronic gastritis is its characteristic feature — an interruption of cell renewal with the proliferation phase predominating over the differentiation phase. However, the determination of the degree of proliferative activity and impaired differentiation of the epithelium of the gastric mucosa (GM) remains not fully understood. The goal of our study was the imm

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15

Lee, Sun-Young. "Type A, Type B, and Non-atrophic Gastritis." Korean Journal of Helicobacter and Upper Gastrointestinal Research 23, no. 2 (2023): 108–17. http://dx.doi.org/10.7704/kjhugr.2023.0015.

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The gastric cancer risk varies based on the etiology and severity of gastritis, which depends on a history of Helicobacter pylori infection and the secretory capacity of the stomach. Type A gastritis is associated with reverse atrophy of the corpus and type B with progressive atrophy extending from the antrum to the corpus. Diffuse or spotty redness in the corpus together with high serum pepsinogen (PG) II levels and a low PG I/II ratio are observed in patients with H. pylori infection when secretory capacity of the stomach is intact. Diffuse-type gastric cancer may develop near the gastric fo

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16

Imam, M., and S. Raphael. "Histopathological pattern of Gastric biopsies: Experience in a tertiary institution in Northern Nigeria and review of literature." J Med & Bas Sci Res 3, no. 1 (2022): 47–52. https://doi.org/10.5281/zenodo.6342140.

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ABSTRACT Background: Disorders of the stomach are a frequent cause of clinical disease and a major reason for general and gastrointestinal outpatient clinics visits. We undertook this study to evaluate and document the pattern of gastric lesions in a tertiary institution in northern Nigeria. Methodology: The study included a total of 543 cases of gastric biopsies received at the Pathology department of Aminu Kano Teaching Hospital from January 2009 to December 2015.  Results: There were 346 (63.7%) males and 197 (36.3%) females with a male to female ratio of 1.8:1. The ages of the Patient

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Avramenko, Anatoly A. "INFLUENCE OF SYSTEMATIC TAKING BLOCKERS OF H2-HISTAMINE RECEPTORS ON THE DEGREE OF SEMINATION OF GASTRIC MUCOSA WITH HELICOBACTER PYLORI INFECTION OF PATIENT WITH CHRONIC NON – ATROPHIC GASTRITIS." Wiadomości Lekarskie 73, no. 11 (2020): 2503–6. http://dx.doi.org/10.36740/wlek202011130.

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The aim: To determine the effect of prolonged use of H2-histamine receptor blockers on the degree of contamination of the gastric mucosa with HP infection in patients with chronic non-atrophic gastritis. Materials and methods: 28 patients with chronic atrophic gastritis (the main group), who regularly took H2-histamine receptor blockers for 2 to 7 years, and 30 patients (control group), who never used them were comprehensively examined. Comprehensive examination included: step-by-step intragastric pH-metry, esophagogastroduodenoscopy, helicobacter infection test (НР) (helicobacter urease test

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18

Khlynova, R. I., O. M. Khromtsova, I. B. Khlinov, et al. "Prevalence of <i>Helicobacter pylori</i>-associated diseases in the Ural Federal District." Ural Medical Journal 22, no. 5 (2023): 14–22. http://dx.doi.org/10.52420/2071-5943-2023-22-5-14-22.

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Introduction Helicobacter pylori has the most pronounced oncogenic potential among known biological carcinogens of humans. Helicobacter positivity leads to the development of chronic gastritis and with prolonged persistence of infection in the gastric mucosa it is associated with the development of atrophic gastritis and adenocarcinoma of the stomach. According to international data, the prevalence of infection in Russia corresponds to the level of countries with low socio-economic status. The domestic literature contains data for a limited number of reggions, such as North-West Russia, Siberi

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Avramenko, Anatoly A. "FREQUENCY OF IMPLEMENTATION AND IMPACT OF APPENDECTOMY AT ACTIVATION OF THE PATHOLOGICAL PROCESS IN PATIENTS WITH CHRONIC NON-ATROPHIC GASTRITIS." Wiadomości Lekarskie 73, no. 7 (2020): 1489–91. http://dx.doi.org/10.36740/wlek202007133.

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The aim: Determine the frequency of surgery-appendectomy of patients with chronic non-atrophic gastritis and the impact of this transaction on the pathogenesis of chronic gastritis. Materials and methods: Data of disease history and life were analyses, as well as the results of a comprehensive survey of 245 patients with chronic non-atrophic gastritis. Comprehensive examination included: step-by-step рН-metry, esophagogastroduodenoscopy, helicobacter infection test (НР) (helicobacter urease test and microscopic examination of stained smears), histological investigations of the gastric stump mu

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20

Gorcheva, Zornica, Valeriya Racheva, and Mirela Vasileva. "SERUM PEPSINOGEN I AND PEPSINOGEN II AS NON-INVASIVE BIOMARKERS FOR THE DIAGNOSIS OF CHRONIC ATROPHIC GASTRITIS." Journal of IMAB - Annual Proceeding (Scientific Papers) 28, no. 2 (2022): 4356–60. http://dx.doi.org/10.5272/jimab.2022282.4356.

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Atrophic gastritis, intestinal metaplasia and dysplasia of the gastric mucosa are major risk factors for the development of an intestinal type of gastric cancer. The selection of patients with such lesions could help with early detection and improve better the prognosis. Esophagogastroscopy is the "gold standard" in detecting gastric cancer and precancerous lesions of the stomach. However, this examination is invasive, expensive, and cannot serve as a screening method. Measurement of serum levels of pessinogen I and pessinogen II is used in high-risk populations in Europe and Asia as a non-inv

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Chen, Ping, Yun Cui, Qing Yan Fu, You Yong Lu, Jing Yuan Fang, and Xiao Yu Chen. "Positive relationship betweenp42.3gene and inflammation in chronic non-atrophic gastritis." Journal of Digestive Diseases 16, no. 10 (2015): 568–74. http://dx.doi.org/10.1111/1751-2980.12282.

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Loor, Alexandra, and D. L. Dumitraşcu. "Helicobacter pylori Infection, Gastric Cancer and Gastropanel." Romanian Journal Of Internal Medicine 54, no. 3 (2016): 151–56. http://dx.doi.org/10.1515/rjim-2016-0025.

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Abstract Gastric cancer (GC) is one of the most widespread types of cancer worldwide. Helicobacter pylori infection has been clearly correlated with gastric carcinogenesis. At present and in the near future, the most important challenge is and will be the significant reduction of mortality due to GC. That goal can be achieved through the identification of higher-risk patients, such as those with atrophic gastritis, intestinal metaplasia and dysplasia. In this review we intend to discuss the importance of diagnosing H. pylori infection and chronic atrophic gastritis in preventing gastric cancer

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Belkovets, A. V., and N. V. Ozhiganova. "Are all the polyps in the stomach the same?" Experimental and Clinical Gastroenterology 1, no. 6 (2021): 134–39. http://dx.doi.org/10.31146/1682-8658-ecg-190-6-134-139.

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The article presents a clinical case of a 38-year-old patient with revealed polyps of the stomach body and iron deficiency anemia on the background of chronic atrophic gastritis. On the example of this observation, variants of the course were demonstrated, including endoscopic and histological manifestations of autoimmune (atrophic corpus) gastritis (AIG). In parallel, the issues of diagnosis and management of patients with the most common polyps in the stomach are discussed. The problem of timely diagnosis of AIG and the advantages of non-invasive methods for assessing the functional state of

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Yang, Ying-Yun, Ke-Min Li, Gui-Fang Xu, et al. "Clinical manifestation, lifestyle, and treatment patterns of chronic erosive gastritis: A multicenter real-world study in China." World Journal of Gastroenterology 30, no. 9 (2024): 1108–20. http://dx.doi.org/10.3748/wjg.v30.i9.1108.

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BACKGROUND Although chronic erosive gastritis (CEG) is common, its clinical characteristics have not been fully elucidated. The lack of consensus regarding its treatment has resulted in varied treatment regimens. AIM To explore the clinical characteristics, treatment patterns, and short-term outcomes in CEG patients in China. METHODS We recruited patients with chronic non-atrophic or mild-to-moderate atrophic gastritis with erosion based on endoscopy and pathology. Patients and treating physicians completed a questionnaire regarding history, endoscopic findings, and treatment plans as well as

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Zhao, Yashuo, Jianing Zhao, Hongyu Ma, et al. "High Hepcidin Levels Promote Abnormal Iron Metabolism and Ferroptosis in Chronic Atrophic Gastritis." Biomedicines 11, no. 9 (2023): 2338. http://dx.doi.org/10.3390/biomedicines11092338.

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Background: Chronic atrophic gastritis (CAG) is a chronic inflammatory disease and premalignant lesion of gastric cancer. As an antimicrobial peptide, hepcidin can maintain iron metabolic balance and is susceptible to inflammation. Objectives: The objective of this study was to clarify whether hepcidin is involved in abnormal iron metabolism and ferroptosis during CAG pathogenesis. Methods: Non-atrophic gastritis (NAG) and chronic atrophic gastritis (CAG) patient pathology slides were collected, and related protein expression was detected by immunohistochemical staining. The CAG rat model was

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Ivashkin, V. T., I. V. Maev, T. L. Lapina, et al. "Clinical Recommendations of Russian Gastroenterological Association and RENDO Endoscopic Society on Diagnosis and Treatment of Gastritis and Duodenitis." Russian Journal of Gastroenterology, Hepatology, Coloproctology 31, no. 4 (2021): 70–99. http://dx.doi.org/10.22416/1382-4376-2021-31-4-70-99.

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Aim. The clinical guidelines are intended to supplement specialty decision-making for improved aid quality in patients with gastritis and duodenitis though acknowledging the latest clinical evidence and principles of evidencebased medicine.Key points. Gastritis is an inflammatory disease of stomach mucosa, with a separate definition of acute and chronic gastritis. Chronic gastritis is a cohort of chronic diseases uniting a typical morphology of persistent inflammatory infiltration, impaired cellular renewal with emergent intestinal metaplasia, atrophy and epithelial dysplasia of gastric mucosa

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ZHANG, Rui-qing, Li LIU, Cheng ZHOU, Yong-feng YANG, Cong-wei ZHANG, and Liang WANG. "Research Progress of Treating Chronic Atrophic Gastritis by Non-Drug Therapy." World Journal of Integrated Traditional and Western Medicine 6, no. 12 (2020): 18–25. https://doi.org/10.70976/j.2096-0964.2020.12.18-25.

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Ryabokon, R. V., V. V. Tsukanov, V. A. Khorzhevskii, A. V. Vasyutin, and J. L. Tonkikh. "Epithelial cell proliferation index in patients with atrophic gastritis depending on the presence of complete or incomplete intestinal metaplasia in the gastric antrum." Meditsinskiy sovet = Medical Council, no. 8 (June 5, 2024): 28–34. http://dx.doi.org/10.21518/ms2024-199.

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Introduction. There is a debate about the significance of intestinal metaplasia (IM) subtypes for the development of gastric cancer. Therefore, determining the indicators of cellular renewal in individuals with complete and incomplete IM is certainly a topical issue.Aim. To study the proliferative activity of epithelial cells of the gastric antrum in patients with Helicobacter pylori-positive antral atrophic gastritis depending on the subtype of IM.Materials and methods. The study included 20 people with chronic antral non-atrophic gastritis (CNG; group A), 20 patients with chronic antral atro

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Vernigorodskiy, S. V., and D. S. Sukhan. "The role of the CagA gene in the occurrence of the inflammatory response of the gastric mucosa in patients with chronic Helicobacter pylori-associated gastritis." Reports of Morphology 24, no. 3 (2018): 11–18. http://dx.doi.org/10.31393/morphology-journal-2018-24(3)-02.

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Currently, Helicobacter pylori infection (H. pylori) is recognized as one of the most important risk factors for gastrocarcinogenesis. It is known that this infection does not directly cause neoplastic changes in the gastric mucosa, and this is due to a number of consecutive events due to the long persistence of the pathogen in the human body. The initial stage of this cascade, of course, is the inflammatory response, due to the body’s ability to adapt to extraneous infection, which is the inevitable result of the interaction of H. pylori with cells of the gastric epithelium. This direct damag

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Nawahir, Sultan, George Kurian, Thomas Alexander, and Susy Kurian. "Impact of alcohol on gastric mucosa in a population with high prevalence of Helicobacter pylori." International Journal of Advances in Medicine 8, no. 6 (2021): 764. http://dx.doi.org/10.18203/2349-3933.ijam20212096.

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Background: The purpose of the study was to see whether chronic alcohol abuse had any effect on the gastric mucosa in a population already affected by a high prevalence of Helicobacter pylori.Methods: 35 males with a history of chronic alcohol abuse were compared with 35 males who were abstinent or social drinkers. All subjects had complaints of dyspepsia. All subjects underwent endoscopy and targeted biopsies were taken from three specific sites in the stomach, namely body, antrum and incisura. Biopsies were studied to look for changes of atrophic gastritis and intestinal metaplasia. The pres

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张, 磊. "Research Progress of Chronic Non-Atrophic Gastritis Diagnostic Methods and Diagnostic Criteria." Advances in Clinical Medicine 04, no. 01 (2014): 5–9. http://dx.doi.org/10.12677/acm.2014.41002.

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Avramenko, A. A., and T. M. Yablonska. "Frequency detection of gastroesophageal reflux disease in patients with low back pain, which treated in rehabilitation center." Likarska sprava, no. 7-8 (December 12, 2016): 70–72. http://dx.doi.org/10.31640/ls-2016(7-8)11.

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The data analysis of the results of complex examination of 43 patients with osteochondrosis were presented. From in 30 (69.8 %) of patients who were identified gastroesophageal reflux disease, in 100% pathology of the thoracic spine, as well as the presence of chronic non-atrophic gastritis with a high degree of colonization of the gastric mucosa by Helicobacter pylori infection.

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Safira, Fathi Rahmah, and Sugiarto Sugiarto. "Histopathological Features of the Gastric Mucosa in Patients with Chronic Gastritis and Helicobacter pylori Infection at Pertamina Central Hospital Jakarta." Muhammadiyah Medical Journal 2, no. 2 (2021): 70. http://dx.doi.org/10.24853/mmj.2.2.70-74.

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Background: Chronic gastritis is a chronic inflammation of the gastric mucosa, accompanied by changes in mucosal histology with or without Helicobacter pylori infection. Changes in the gastric mucosa include gastric mucosal atrophy, intestinal metaplasia, and epithelial dysplasia. Purposes: This study aims to determine the microscopic appearance of the mucosa in chronic gastritis patients based on standard histopathological criteria, which include gland atrophy, intestinal metaplasia, dysplasia with or without Helicobacter pylori infection at Pertamina Central Hospital Jakarta period 2018 - 20

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Akhtar, D., M. A. Donaldson, N. H. Akhtar, H. Yang, and F. Donnellan. "A136 A RARE CAUSE OF AUTOIMMUNE ATROPHIC PANGASTRITIS COMPLICATED WITH GASTRIC OUTLET OBSTRUCTION." Journal of the Canadian Association of Gastroenterology 5, Supplement_1 (2022): 7–8. http://dx.doi.org/10.1093/jcag/gwab049.135.

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Abstract Background Chronic gastritis comes in two well recognized forms: environmental, which is most commonly antral or multifocal in distribution and is typically caused by Helicobacter Pylori (HP) infection, and autoimmune gastritis(AIG), which affects the corpus and fundus. Presented here is a case of autoimmune atrophic pangastritis(AIAP). Aims To increase awareness of a rare condition with limited data on available treatment modalities. Methods Case Report Results A 68-year old female with autoimmune hypothyroidism, presented with weight loss and elevated anti-TTG serology. Index esopha

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Turdieva, Sh T., and E. A. Shamansurova. "Features of the endoscopic picture in paediatric gastroduodenal diseases caused by Helicobacter pylori." Russian Journal of Infection and Immunity 10, no. 3 (2020): 543–50. http://dx.doi.org/10.15789/2220-7619-fot-1249.

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The aim of the study was to examine the features of the endoscopic picture in the upper digestive tract mucosa in paediatric chronic gastroduodenal pathology (CGDP) associated with Helicobacter pylori. Materials and methods. There were examined 286 patients, aged 6–15 years. Diagnostic criteria for chronic gastroduodenal pathology were anamnestic as well as instrumental and functional studies data: gastric fractional intubation, esophagogastroduodenoscopy (EPGDS) with endoscopic pH-metry without biopsy, and ultrasound examination of abdominal organs. H. pylori testing was carried out by two un

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Frandeș, Sergiu Ioan, Oana Chibulcutean, and Simona Bățagă. "A Link Between Platelet-to-Lymphocyte Ratio and Helicobacter pylori Infection." Journal of Interdisciplinary Medicine 6, no. 4 (2021): 182–84. http://dx.doi.org/10.2478/jim-2021-0040.

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Abstract Background: Gastritis is a progressive disease that evolves from a non-atrophic to an atrophic state and progresses through intestinal metaplasia, even leading to gastric cancer. Gastritis is defined by an inflammatory process of the mucosal lining of the stomach. Aim: We aimed to identify any association between Helicobacter pylori (H. pylori)-positive gastritis and simple inflammatory markers derived from a complete blood count such as the platelet-to-lymphocyte ratio (PLR). Material and Methods: We performed a retrospective case-control study in which we included patients who under

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Prikhodko, M. N., and Zh G. Simonova. "Helicobacter pylori genotypes in patients with stable angina combined with chronic gastritis." Experimental and Clinical Gastroenterology, no. 3 (May 22, 2021): 119–24. http://dx.doi.org/10.31146/1682-8658-ecg-187-3-119-124.

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Research objective. То study the genotypes of Helicobacter pylori and their antibiotic sensitivity in patients with stable angina in combination with chronic gastritis. Material and methods. 46 patients with stable angina with a combination of chronic H. pylori-associated gastritis were included in the open prospective clinical study. To diagnose H. pylori, serological method of detection of antibodies in blood serum was used, PCR - diagnosis of H. pylori genes. Antibiotic sensitivity of H. pylori strains was studied by serial dilution method. Results. In the patients (n = 46) stable angina in

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Karimov, M. M., G. N. Sobirova, and U. K. Abdullayeva. "Chronic gastritis and carcinogenesis issues." Herald of Pancreatic Club 45, no. 4 (2019): 65–70. http://dx.doi.org/10.33149/vkp.2019.04.09.

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Risk factors contributing to the transformation of chronic atrophic gastritis into gastric cancer are analyzed. Detection and monitoring of patients with precancerous conditions/lesions (precancerous changes), proper screening of H. pylori make early diagnosis of gastric cancer real. Features of precancerous conditions are given in order of increasing risk of developing gastric cancer. Adenomatous polyps of the stomach take the first place. Subsequent precancerous conditions include: cancer of the operated stomach, Menetria disease (hypertrophic gastropathy), B12-deficient anemia, and gastric

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Liu, Naihua, Yuancheng Huang, Fengbin Liu, and Hong Liu. "Serum exosomal miR-122-5p, GAS, and PGR in the non-invasive diagnosis of CAG." Open Medicine 16, no. 1 (2021): 1350–55. http://dx.doi.org/10.1515/med-2021-0342.

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Abstract Objective The aim of this study was to integrate the serum exosomal miRNA miR-122-5p with canonical serological biomarkers for the non-invasive screening of chronic atrophic gastritis (CAG) patients. Methods miR-122-5p and U6 were amplified by the quantitative reverse transcription polymerase chain reaction (RT-qPCR), gastrin (GAS), pepsinogen I (PG-I), and PG-II and were measured by ELISA. The area under the receiver operating characteristic (ROC) curves and their correlation were analyzed. Results In the present study, GAS level and PG-I/PG-II ratio (PGR) were increased in CAG group

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Avramenko, A. A. "Pathogenesis of chronic non-atrophic gastritis in the staff of Ministry of Home Affairs." Bukovinian Medical Herald 21, no. 1 (81) (2017): 3–6. http://dx.doi.org/10.24061/2413-0737.xxi.1.81.2017.1.

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Avramenko, A., A. Magdenko, and V. Dymo. "A case of worsening condition of armed forces of ukraine fighter after treatment for chronic non-atrophic gastritis excernsation." Journal of Education, Health and Sport 68 (September 24, 2024): 55490. http://dx.doi.org/10.12775/jehs.2024.68.55490.

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A pathogenetic explanation was analyzed and given for the case of deterioration in the condition of a Ukrainian Armed Forces fighter after treatment for an exacerbation of his chronic non-atrophic gastritis. It was found that the treatment was carried out without a complete preliminary examination of the patient and without taking into account the interaction of drugs that were used in the treatment regimen and their effect on Helicobacter pylori infection, which was the reason for the sharp deterioration in the fighter’s condition.

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Tran, Van Huy, Quang Trung Tran, Thi Huyen Thuong Nguyen, et al. "Non-cardia early gastric cancer in Central Vietnam: noticeable uncommon background mucosa and results of endoscopic submucosal dissection." Endoscopy International Open 10, no. 08 (2022): E1029—E1036. http://dx.doi.org/10.1055/a-1854-4587.

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Abstract Background and study aims Gastric cancer (GC) is one of the leading causes of malignancy-related death in Vietnam, with increasing incidence of non-cardia early gastric cancer (N-EGC). Data on accurate diagnosis of EGC and treatment by endoscopic submucosal dissection (ESD) in Vietnam are very sparse. The aim of this study was to describe the characteristics of N-EGC and evaluate the effectiveness and the safety of ESD in Central Vietnam. Patients and methods We prospectively enrolled patients with N-EGC detected by magnified chromoendoscopy from December 2013 to August, 2018 in Centr

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Zhang, Yin, Yue Liu, Rui Song, et al. "Validating traditional Chinese syndrome features in varied stages of chronic gastritis malignant transformation: study protocol for a cross-sectional study." BMJ Open 8, no. 8 (2018): e020939. http://dx.doi.org/10.1136/bmjopen-2017-020939.

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IntroductionThe transition from chronic non-atrophic gastritis (CNAG) to chronic atrophic gastritis (CAG) and gastric carcinoma (GC) is regarded as a representative disease model of gastric mucosa malignant transformation led by uncontrolled inflammation. Traditional Chinese medicine (TCM) syndrome-targeted therapies have been applied in treating chronic gastritis (CG) malignant transformation in China with satisfying efficacy. This study aims to validate TCM syndrome features in each stage of CG malignant transformation. The findings may shed light on the TCM hypothesis of CG malignant transf

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He, Xiao-Ming, and Xuan Huang. "Clinical efficacy of Weifuchun combined with quadruple therapy in treatment of chronic non-atrophic gastritis." World Chinese Journal of Digestology 25, no. 6 (2017): 521. http://dx.doi.org/10.11569/wcjd.v25.i6.521.

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Hu, Qichao, Jinhao Zeng, Xiaomei Zhang, et al. "Metabolomics Profiles Reveal the Efficacy of Wuzhuyu Decoction on Patients with Chronic Non-Atrophic Gastritis." Drug Design, Development and Therapy Volume 17 (November 2023): 3269–80. http://dx.doi.org/10.2147/dddt.s428783.

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Kakehasi, Adriana M., Cláudia M. C. Mendes, Luiz G. V. Coelho, Luiz P. Castro, and Alfredo J. A. Barbosa. "The presence of Helicobacter Pylori in postmenopausal women is not a factor to the decrease of bone mineral density." Arquivos de Gastroenterologia 44, no. 3 (2007): 266–70. http://dx.doi.org/10.1590/s0004-28032007000300016.

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BACKGROUND: Osteoporosis affects approximately 30% of postmenopausal women. Gastrectomy, pernicious anemia, and more recently Helicobacter pylori infection, have all been implicated in the pathogenesis of osteoporosis. A reduced parietal cell mass is a common feature in these conditions. AIM: To study a possible relationship between chronic gastritis, parietal cell density of the oxyntic mucosa and bone mineral density in postmenopausal women, as chronic gastritis, Helicobacter pylori infection and osteoporosis are frequently observed in the elderly. METHODS: Fifty postmenopausal women (61.7 ±

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Yue, Pan, Juan Zhong, Jiajun Huang, Zhaoxi Lan, and Sen Zhong. "The efficacy and safety of Xiangsha Liujunzi decoction in the treatment of chronic non-atrophic gastritis." Medicine 100, no. 4 (2021): e24504. http://dx.doi.org/10.1097/md.0000000000024504.

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Sorokman, T. V., P. M. Moldovan, L. Yu Khlunovska, and I. Ya Lozyuk. "Dynamics of inflammatory changes of the gastric mucosa in children with duodenal ulcer." CHILD`S HEALTH 16, no. 4 (2021): 285–88. http://dx.doi.org/10.22141/2224-0551.16.4.2021.236907.

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Background. The problem of gastrointestinal diseases in children remains quite relevant due to the high prevalence and presence of atypical symptoms and comorbidities pathogenetically associated with acid-dependent diseases. The purpose was to study the dynamics of inflammatory changes of the mucous membrane in children with duodenal ulcer (DU). Materials and methods. One hundred and six children aged 10 to 18 years with DU were examined. To verify the diagnosis, all children underwent endoscopy, pH-metry, morphological examination of gastrobiopsy samples, examination for Helicobacter pylori (

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M. Ghoneim, Enas, Eman H. Hassan, Hassan Zaghla, Doha Taie, and Samah M. Awad. "The impact of the nucleotide-binding oligomerization domain 1 (NOD 1) gene polymorphism on Helicobacter Pylori induced chronic gastritis in hepatic patients." Egyptian Journal of Medical Microbiology 30, no. 3 (2021): 9–19. http://dx.doi.org/10.51429/ejmm30302.

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Background: Chronic infection with Helicobacter pylori (H.pylori ) causes atrophic and even gastric metaplastic changes, and it has a well-known link to peptic ulceration. Nucleotide-binding oligomerization domain-containing protein 1 (NOD1) is a protein receptor that is presented by the NOD1 gene. It distinguishes H.pylori bacterial molecules and enhances an immune response Objectives: to describe the relation between the NOD1 gene (rs2075820) polymorphism and H.pylori infection in hepatic and non hepatic patients with chronic gastritis, study its impact on the degree of chronic gastritis in

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Zak, Maxim, and Lyudmila Pasiyeshvili. "CHRONIC GASTRITIS CLINICAL FEATURES AND STOMACH FUNCTIONAL STATE DURING NONSTEROIDAL ANTI-INFLAMMATORY DRUGS ADMINISTRATION IN PATIENTS WITH OSTEOARTHRITIS." EUREKA: Health Sciences 5 (September 30, 2016): 17–22. http://dx.doi.org/10.21303/2504-5679.2016.00178.

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Aim. Determination of chronic gastritis clinical features and stomach functional state during nonsteroidal anti-inflammatory drugs (NSAID) administration in patients with osteoarthritis (OA). Materials and methods. 122 patients with OA and verified chronic gastritis (CG) (50 males and 72 females) aged 42 to 64 years (mean age – 49.65±3.51) were observed. Depending on gastritis morphological form, patients were divided into 2 groups: 54 patients with OA in combination with non-atrophic gastritis (NAG) were included into the group I, 68 patients with OA in combination with atrophic gastritis (AG

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