Relevant bibliographies by topics / Chronic Proliferelative Periodontal Inflammatory Lesion

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Academic literature on the topic 'Chronic Proliferelative Periodontal Inflammatory Lesion'

Author: Grafiati
Published: 3 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Chronic Proliferelative Periodontal Inflammatory Lesion"

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J Ford, Pauline, Erica Gemmell, and Gregory J Seymour. "The immunopathology of periodontal disease: links with atherosclerosis?" Microbiology Australia 26, no. 3 (2005): 127. http://dx.doi.org/10.1071/ma05127.

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Chronic inflammatory periodontal disease occurs as a result of an inflammatory response in the periodontium which is elicited by bacteria present in dental plaque. The specific cytokines and chemokines produced by this initial response cause a T cell/macrophage dominated inflammatory infiltrate to develop in the connective tissues. If this cell-mediated immune response does not control the bacterial challenge, progression to a B cell/plasma cell lesion occurs (reviewed in Gemmell et al. 20021). A component of the specific T cell and antibody response elicited by these bacteria however, has bee
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Smolyar, N. I., and M. M. Shylo. "Prevalence and structure of inflammatory periodontal diseases in tuberculosis in school-age children." Tuberculosis, Lung Diseases, HIV Infection, no. 3 (September 27, 2022): 39–44. http://dx.doi.org/10.30978/tb-2022-3-39.

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Objective — to study the prevalence and structure of inflammatory periodontal diseases in respiratory tuberculosis patients and infected with mycobacterium tuberculosis (MTB) school-aged children.
 Materials and methods. The examination was carried out in 168 (100 %) patients with respiratory tuberculosis and MBT-infected children and adolescents of school age (from 6 to 17 years) with inflammatory diseases of periodontal tissues. Among the examined schoolchildren, 102 (60.72 %) were school-aged children with respiratory tuberculosis (the first group). The second group included 66 (39.28
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Myhal, O. O., R. Z. Ogonovskyi, Z. M. Honta, K. A. Sichkoriz, K. A. Moroz, and O. Y. Kordiyak. "INDEX ASSESSMENT OF PERIODONTAL TISSUES AND DETERMINATION OF TREATMENT NEEDS IN PATIENTS WITH CHRONIC RHEUMATIC HEART DISEASE." Актуальні проблеми сучасної медицини: Вісник Української медичної стоматологічної академії 24, no. 2 (2024): 80–86. http://dx.doi.org/10.31718/2077-1096.24.2.80.

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This study addresses the pressing scientific and practical issue in modern dentistry: enhancing dental care effectiveness for patients with chronic rheumatic heart disease. The purpose of this research is to comprehensively assess the condition of tooth-supporting tissues and quantify inflammatory phenomena in periodontal tissues among patients with chronic rheumatic heart disease, as well as to determine the necessary extent of periodontal care based on periodontal indices. Participants and methods. The periodontal tissues of 719 patients with chronic rheumatic heart disease from the Rheumato
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Gu, Ying, Hsi-Ming Lee, Nicole Napolitano, et al. "4-Methoxycarbonyl Curcumin: A Unique Inhibitor of Both Inflammatory Mediators and Periodontal Inflammation." Mediators of Inflammation 2013 (2013): 1–10. http://dx.doi.org/10.1155/2013/329740.

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Chronic inflammatory diseases such as periodontitis have been associated with increased risk for various medical conditions including diabetes and cardiovascular disease. Endotoxin (lipopolysaccharide, LPS), derived from gram-negative periodonto-pathogens, can induce the local accumulation of mononuclear cells in the inflammatory lesion, increasing proinflammatory cytokines and matrix metalloproteinases (MMPs). This ultimately results in the destruction of periodontal connective tissues including alveolar bone. Curcumin is the principal dyestuff in the popular Indian spice turmeric and has sig
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Cortela, D. C. B., A. L. de Souza Junior, M. C. L. Virmond, and E. Ignotti. "Inflammatory Mediators of Leprosy Reactional Episodes and Dental Infections: A Systematic Review." Mediators of Inflammation 2015 (2015): 1–15. http://dx.doi.org/10.1155/2015/548540.

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Reactional episodes in leprosy are a result of complex interactions between the immune system,Mycobacterium leprae, and predisposing factors, including dental infections. To determine the main inflammatory mediators in the immunopathological process of dental infections and leprosy reactions, we conducted a systematic review of primary literature published between 1996 and 2013. A three-stage literature search was performed (Stage I, “leprosy reactions” and “inflammatory mediators”; Stage II, “dental infections” and “inflammatory mediators”; and Stage III, “leprosy reactions,” “dental infectio
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Barbudo-Selmi, Glenda Ramalho, Marileda Bonafim Carvalho, André Luis Selmi, and Silvio Emílio Cuevas Martins. "Periodontal disease characterization in dogs with normal renal function or chronic renal failure." Ciência Rural 34, no. 1 (2004): 113–18. http://dx.doi.org/10.1590/s0103-84782004000100017.

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The purpose of this study was to evaluate periodontal disease (PD) in dogs with chronic renal failure (CRF) and to compare it to PD in dogs with normal renal function (NRF). Twelve dogs with CRF and 24 dogs with NRF, all presenting dental pocket formation, were compared. In all dogs, serum creatinine, blood urea nitrogen, urine specific gravity and total red and white blood cells were determined. A complete oral examination was also performed including evaluation of bacterial plaque, gingivitis, gingival recession, pocket, calculus, dental mobility, dental loss, and ulcers. These data were use
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7

Moroz, P. I., A. K. Iordanishvili, V. A. Prohodnaya, S. Yu Maxyukov, A. V. Safronenko, and E. Sh Gulyaeva. "Features of clinical course and treatment principles of endodontic-periodontal lesions." Kazan medical journal 99, no. 3 (2018): 362–68. http://dx.doi.org/10.17816/kmj2018-362.

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Aim. To study the characteristic differences in the clinical course of chronic generalized periodontitis in the presence of periapical infection focus and the principles of combined treatment of endodontium and periodontium. Methods. A clinical and X-ray examination of 1525 patients of different age groups was performed. As a result, a clinical group of 68 patients with chronic generalized moderate and severe periodontitis was formed, who were divided into two groups depending on the presence or absence of periapical destruction. In group 1, 32 patients with the presence of chronic generalized
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8

Boșca, Adina Bianca, Elena Dinte, Carmen Mihaela Mihu, et al. "Local Drug Delivery Systems as Novel Approach for Controlling NETosis in Periodontitis." Pharmaceutics 16, no. 9 (2024): 1175. http://dx.doi.org/10.3390/pharmaceutics16091175.

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Periodontitis is a chronic inflammation caused by periodontopathogenic bacteria in the dental biofilm, and also involves the inflammatory-immune response of the host. Polymorphonuclear neutrophils (PMNs) play essential roles in bacterial clearance by multiple mechanisms, including the formation of neutrophil extracellular traps (NETs) that retain and destroy pathogens. During PD progression, the interaction between PMNs, NETs, and bacteria leads to an exaggerated immune response and a prolonged inflammatory state. As a lesion matures, PMNs accumulate in the periodontal tissues and die via NETo
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Kopetsky, Igor S., Lyudmila V. Pobozhieva, Alyona I. Kopetskaya, and Juliya V. Shevelyuk. "Problems of dental preservation and complex therapy of chronic generalized periodontitis." Medical Journal of the Russian Federation 27, no. 1 (2021): 71–78. http://dx.doi.org/10.17816/0869-2106-2021-27-1-71-78.

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Epidemiological studies have reported the prevalence of inflammatory periodontal diseases of 85%90% in individuals aged 30 years. Predicting the possibility of preserving teeth is an important integral part of treatment planning for periodontitis. Traditionally, the prognosis is based on the likelihood of tooth loss. The article presents a review of the literature characterizing the factors that affect the prognosis of the service life of teeth in case of periodontitis. For the literature review, a literature search was performed on the MedLine, EMBASE, CyberLeninka, and eLibrary databases. Th
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10

Eldzharov, Artem, Dzerassa Kabaloeva, Dmitry Nemeryuk, et al. "Evaluation of Microcirculation, Cytokine Profile, and Local Antioxidant Protection Indices in Periodontal Health, and Stage II, Stage III Periodontitis." Journal of Clinical Medicine 10, no. 6 (2021): 1262. http://dx.doi.org/10.3390/jcm10061262.

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Periodontitis, initiated by the subgingival biofilm and modified by the individual’s inflammatory/immune response, has been associated with vascular dysfunction. To analyze microcirculation indices in periodontal tissues and determine the activity of the enzymatic component of antioxidant defense and humoral immunity factors, a single-blind non-invasive clinical trial was realized. Forty subjects, aged from 30 to 65 years, with moderate to severe chronic periodontitis (chronic generalized periodontitis, CGP) vs. 40 subjects as periodontally healthy were recruited. Information such as capillary
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Book chapters on the topic "Chronic Proliferelative Periodontal Inflammatory Lesion"

1

Jebisha, Dr L., Dr R. Sathish Muthukumar, Dr C. Sreeja, and Dr Merlin Jayaraj. "ROLE OF IMMUNE CELLS IN PERIODONTAL DISEASE." In Emerging Trends in Oral Health Sciences and Dentistry. Technoarete Publishers, 2022. http://dx.doi.org/10.36647/etohsd/2022.01.b1.ch016.

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Periodontitis is intricate & multi-factorial lesion. It is characterized by a group of inflammatory conditions in the periodontium which is caused by bacteria. Chronic inflammation is precise by B and T cells by the initiation in cytokine secretion, then osteoclastogenesis is modulated. The objective of the article briefly reviews the recent research and the consequence in B cell & T lymphocytes subsets, and the expression of cytokine seen in the morbific of the periodontitis. Gingival homeostasis is maintained by T reg (T regulatory), CD8+ T, and tissue-resident γδ, T cells plays a vi
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Heasman, P. A., and P. J. Waterhouse. "Periodontal diseases in children." In Paediatric Dentistry. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198789277.003.0020.

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Periodontal diseases comprise a group of infections that affect the supporting structures of the teeth: marginal and attached gingiva, periodontal ligament, cementum, and alveolar bone. Acute gingival diseases—primarily herpetic gingivostomatitis and necrotizing gingivitis—are ulcerative conditions that result from specific viral and bacterial infection. Chronic gingivitis, however, is a non-specific inflammatory lesion of the marginal gingiva which reflects the bacterial challenge to the host when dental plaque accumulates in the gingival crevice. The development of chronic gingivitis is enha
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