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Journal articles on the topic 'Chronic venous insufficiency'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Lukka, Harivadan, K. Sudhakar Rao, and Matta Sridevi. "Antenatal Chronic Venous Insufficiency." International Journal of Science and Research (IJSR) 10, no. 12 (2021): 70–72. https://doi.org/10.21275/sr211130180059.

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2

Panda, Dwijesh Kumar. "Chronic Venous Insufficiency Resemble Lymphedema Leg." Asian Pacific Journal of Health Sciences 6, no. 3 (2019): 52–55. http://dx.doi.org/10.21276/apjhs.2019.6.3.10.

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3

Kestler, Bri. "Chronic Venous Insufficiency." Physician Assistant Clinics 6, no. 2 (2021): 319–30. http://dx.doi.org/10.1016/j.cpha.2020.11.005.

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4

ÇINAR ÖZDEMİR, Özlem, Emre ALTINDAĞ, Fatma AVCI, and M. Fatih UYSAL. "Chronic Venous Insufficiency." Turkiye Klinikleri Journal of Health Sciences 1, no. 2 (2016): 125–38. http://dx.doi.org/10.5336/healthsci.2015-45121.

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5

Wilson, Paul. "Chronic venous insufficiency." Morecambe Bay Medical Journal 3, no. 3 (1998): 85–89. http://dx.doi.org/10.48037/mbmj.v3i3.549.

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6

Stadnicki, Antoni, Martin Rusnák, and Anna Stadnicka. "CHRONIC VENOUS INSUFFICIENCY." Scientific Journal of Polonia University 32, no. 1 (2019): 131–36. http://dx.doi.org/10.23856/3217.

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Chronic venous insufficiency (CVI) is a common, but underdiagnozed clinical disorder associated with a variety of signs and symptoms. The presence of leg edema in association with varicose veins, and venous leg ulcer in later disease stages defines the disease. The pathogenesis of chronic venous disease is based on venous reflux, obstruction, or a combination thereof. Prior postthrombotic syndrome is one of risk factor for CVI which may explain observed prevalence of thrombophilia in CVI. Color flow duplex ultrasound is the gold standard for nearly all diagnostic issues related to chronic veno

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7

Miller, Wayne L. "Chronic venous insufficiency." Current Opinion in CARDIOLOGY 10, no. 5 (1995): 543. http://dx.doi.org/10.1097/00001573-199509000-00016.

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8

Kwun, Woo-Hyung. "Chronic Venous Insufficiency." Yeungnam University Journal of Medicine 24, no. 2 Suppl (2007): S234–244. http://dx.doi.org/10.12701/yujm.2007.24.2s.s234.

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9

Reich-Schupke, S. "Chronic venous insufficiency." Phlebologie 46, no. 01 (2017): 34–36. http://dx.doi.org/10.12687/phleb2348-1-2017.

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10

Eberhardt, Robert T., and Joseph D. Raffetto. "Chronic Venous Insufficiency." Circulation 111, no. 18 (2005): 2398–409. http://dx.doi.org/10.1161/01.cir.0000164199.72440.08.

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11

Eberhardt, Robert T., and Joseph D. Raffetto. "Chronic Venous Insufficiency." Circulation 130, no. 4 (2014): 333–46. http://dx.doi.org/10.1161/circulationaha.113.006898.

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12

Earnshaw, Jonothan J. "Chronic venous insufficiency." Journal of Vascular Surgery 34, no. 5 (2001): 953. http://dx.doi.org/10.1067/mva.2001.117614.

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13

Capeheart, Jeanine Kunkel. "Chronic Venous Insufficiency." Journal of Wound, Ostomy and Continence Nursing 23, no. 4 (1996): 227–34. http://dx.doi.org/10.1097/00152192-199607000-00018.

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14

O’Donnell, Thomas F., C. Scott McEnroe, and Paula Heggerick. "Chronic Venous Insufficiency." Surgical Clinics of North America 70, no. 1 (1990): 159–80. http://dx.doi.org/10.1016/s0039-6109(16)45042-5.

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15

White, J. V. "Chronic Venous Insufficiency." Perspectives in Vascular Surgery and Endovascular Therapy 17, no. 4 (2005): 319–27. http://dx.doi.org/10.1177/153100350501700406.

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16

Vernick, Sanford H., and Frank D. Shaw. "Chronic venous insufficiency." Archives of Physical Medicine and Rehabilitation 75, no. 9 (1994): 1023–24. http://dx.doi.org/10.1016/0003-9993(94)90688-2.

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17

Gujja, Karthik, Jose Wiley, and Prakash Krishnan. "Chronic Venous Insufficiency." Interventional Cardiology Clinics 3, no. 4 (2014): 593–605. http://dx.doi.org/10.1016/j.iccl.2014.07.001.

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18

Donaldson, Magruder C. "Chronic venous insufficiency." Current Treatment Options in Cardiovascular Medicine 2, no. 3 (2000): 265–72. http://dx.doi.org/10.1007/s11936-000-0021-2.

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19

Schainfeld, Robert M. "Chronic venous insufficiency." Current Treatment Options in Cardiovascular Medicine 5, no. 2 (2003): 109–19. http://dx.doi.org/10.1007/s11936-003-0019-7.

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20

Kim, Esther S. H., Claudiu Diaconu, Leasa Baus, et al. "Chronic Cerebrospinal Venous Insufficiency." Journal of Ultrasound in Medicine 34, no. 6 (2015): 1097–106. http://dx.doi.org/10.7863/ultra.34.6.1097.

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21

Rasman, Alessandro. "Chronic Cerebrospinal Venous Insufficiency." Vascular Specialist International 31, no. 3 (2015): 106–7. http://dx.doi.org/10.5758/vsi.2015.31.3.106.

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22

Dua, Anahita, and Jennifer A. Heller. "Advanced Chronic Venous Insufficiency." Vascular and Endovascular Surgery 51, no. 1 (2016): 12–16. http://dx.doi.org/10.1177/1538574416682175.

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Introduction: Intervention for advanced chronic venous insufficiency is considered an appropriate standard of care. However, outcomes vary among patients who present in advanced clinical stages of disease. The main objectives of this study were to determine whether racial disparity exists at initial presentation and response to intervention. Methods: A retrospective database was created to include all radiofrequency ablation procedures performed by a single surgeon from January 14, 2009, through May 25, 2011. Demographics, clinical traits, race, procedure, and outcomes were analyzed. Stepwise

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23

Sternberg, Zohara. "Chronic Cerebrospinal Venous Insufficiency." Journal of Endovascular Therapy 22, no. 4 (2015): 647–49. http://dx.doi.org/10.1177/1526602815592204.

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24

Shami, S., S. Sarin, and J. Scurr. "Chronic venous insufficiency disease." International Journal of Angiology 6, no. 01 (2011): 30–48. http://dx.doi.org/10.1007/bf01616230.

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25

Antel, Jack, Alan Thompson, and William Carroll. "Chronic cerebrospinal venous insufficiency." Multiple Sclerosis Journal 16, no. 7 (2010): 770. http://dx.doi.org/10.1177/1352458510374342.

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26

Barreto, Andrew D., Staley A. Brod, Thanh‐Tung Bui, et al. "Chronic cerebrospinal venous insufficiency." Annals of Neurology 73, no. 6 (2013): 721–28. http://dx.doi.org/10.1002/ana.23839.

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27

Taheri, Syde A., Julie Cullen, and Thomas Wormer. "Venous Reconstruction in Chronic Venous Insufficiency." Vascular Surgery 23, no. 6 (1989): 470–74. http://dx.doi.org/10.1177/153857448902300609.

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28

Alguire, Patrick C., and Barbara M. Mathes. "Chronic Venous Insufficiency and Venous Ulceration." Journal of General Internal Medicine 12, no. 6 (1997): 374–83. http://dx.doi.org/10.1046/j.1525-1497.1997.00063.x.

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29

Alguire, Patrick C., and Barbara M. Mathes. "Chronic Venous Insufficiency and Venous Ulceration." Journal of General Internal Medicine 12, no. 6 (1997): 374–83. http://dx.doi.org/10.1007/s11606-006-5087-4.

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30

Barajas Colón, José Ángel, Juan José Granados-Romero, Baltazar Barrera-Mera, et al. "Chronic venous insufficiency: a review." International Journal of Research in Medical Sciences 9, no. 6 (2021): 1808. http://dx.doi.org/10.18203/2320-6012.ijrms20211928.

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Chronic venous insufficiency (CVI) comprises a complete spectrum of morphological and functional abnormalities of the venous system1 including any long-term functional and morphological alteration. CVI accounts for several abnormalities of the venous system. It is a highly prevalent disease that causes serious economic consequences, a decrease in the quality of life and can lead to serious complications. An exhaustive review was performed with the available literature, using the PubMed, ScienceDirect, Scopus and Cochrane databases from 2004 to 2021. The search criteria were formulated to ident

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31

Mendoza Rojas, Hubert J. "Association between insufficient venous systems and clinical manifestations in chronic venous disease of the lower limbs." Revista de la Facultad de Medicina Humana 24, no. 1 (2024): 09–13. http://dx.doi.org/10.25176/rfmh.v24i1.6011.

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Introduction: The clinical, etiological, anatomical, and pathophysiological classification (CEAP) standardizes the manifestations of lower limb venous disease. Objective: To investigate the association between insufficient venous systems and the clinical classification of CEAP. Methods: A quantitative, cross-sectional, analytical, correlational study was conducted. Sampling was non-probabilistic and convenience-based. The sample size was 136 lower limbs from 71 patients. Chi-square, Monte Carlo and Odd ratio (OR) statistical tests were used with 95% confidence intervals through bivariate logis

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32

Steins, Anke, Hans-Martin Häfher, Martin Hahn, and Michael Jünger. "Microcirculation in Chronic Venous Insufficiency." Phlebology: The Journal of Venous Disease 17, no. 3-4 (2002): 115–20. http://dx.doi.org/10.1177/026835550201700307.

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Objective: To study the microcirculation of the skin of the leg in patients with chronic venous disease of the lower limb, and to assess the effect of compression treatment. Patients and Methods: Patients were recruited from the vascular clinic and investigated by direct capillary pressure, transcutaneous oxygen tension, intravital video capillaroscopy and fluorescence video microscopy. The microcirculation was observed over the healing period in patients with venous leg ulcers. The effects of compression therapy on microcirculatory changes were studied in patients with Widmer stage I and II c

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33

Valencia, Isabel C., Anna Falabella, Robert S. Kirsner, and William H. Eaglstein. "Chronic venous insufficiency and venous leg ulceration." Journal of the American Academy of Dermatology 44, no. 3 (2001): 401–24. http://dx.doi.org/10.1067/mjd.2001.111633.

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34

Shcheglov, E. A. "Chronic Venous Insufficiency and Gonarthrosis." N.N. Priorov Journal of Traumatology and Orthopedics 19, no. 2 (2012): 31–34. http://dx.doi.org/10.17816/vto20120231-34.

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The rate of previously not diagnosed chronic insufficiency in patients with knee osteoarthrosis was determined. The effect of treatment measures directed to elimination of venous insufficiency was evaluated. It was shown that in 48.8% of patients chronic venous insufficiency was not diagnosed by orthopaedic surgeons. Observance of therapeutic regimen, use of elastic compression and phlebotropic drugs contributed to the elimination of gonarthrosis symptoms and improved the quality of life (by KOOS scale).

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35

Mościcka, Paulina, Justyna Cwajda-Białasik, Maria Teresa Szewczyk, and Arkadiusz Jawień. "Chronic venous insufficiency – clinical manifestation." Leczenie ran 16, no. 3-4 (2019): 84–91. http://dx.doi.org/10.5114/lr.2019.94621.

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36

Nuzum, Donald S., Tsion T. Gebru, and Samir A. Kouzi. "Pycnogenol for chronic venous insufficiency." American Journal of Health-System Pharmacy 68, no. 17 (2011): 1589–601. http://dx.doi.org/10.2146/ajhp100676.

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37

Fowkes, F. G. R. "Epidemiology of Chronic Venous Insufficiency." Phlebology: The Journal of Venous Disease 11, no. 1 (1996): 2–5. http://dx.doi.org/10.1177/026835559601100102.

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Objective: To determine the prevalence of chronic venous insufficiency in the general population and its association with varicose veins. Data sources: MEDLINE search 1980–94 plus scanning of reference lists in articles obtained. Study selection: Studies on venous disease in subjects not attending health services. Data synthesis: A formal systematic review of metaanalysis was not carried out because of the heterogeneity of the few available studies. Skin changes were found to occur in over 3% of adults, more so in women than men. The prevalence was higher in subjects with varicose veins and de

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38

Bauersachs, J., I. Fleming, and R. Busse. "Pathophysiology of Chronic Venous Insufficiency." Phlebology: The Journal of Venous Disease 11, no. 1 (1996): 16–22. http://dx.doi.org/10.1177/026835559601100105.

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Objective: To review the physiological mechanisms determining venous return to the heart and the pathophysiological events culminating in chronic venous insufficiency (CVI), focusing primarily on the role of alterations in nitric oxide (NO) production by the vascular endothelium. Background: Congenital valve incompetence, thrombotic damage or venous outflow obstruction result in the development of chronic venous hypertension which frequently leads to ulceration. One major aetiological factor of trophic changes in the skin of patients with CVI is the phenomenon of leucocyte trapping. Hypothesis

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39

De Backer, G., and G. De Backer. "Epidemiology of Chronic Venous Insufficiency." Angiology 48, no. 7 (1997): 569–76. http://dx.doi.org/10.1177/000331979704800703.

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40

Abascal, Kathy, and Eric Yarnell. "Botanicals for Chronic Venous Insufficiency." Alternative and Complementary Therapies 13, no. 6 (2007): 304–11. http://dx.doi.org/10.1089/act.2007.13609.

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41

Kistner, Robert L. "Diagnosis of chronic venous insufficiency." Journal of Vascular Surgery 3, no. 1 (1986): 185–88. http://dx.doi.org/10.1067/mva.1986.avs0030185.

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42

Gschwandtner, Michael E., and Herbert Ehringer. "Microcirculation in chronic venous insufficiency." Vascular Medicine 6, no. 3 (2001): 169–79. http://dx.doi.org/10.1177/1358836x0100600308.

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43

Rathbun, Suman. "Book review: Chronic Venous Insufficiency." Vascular Medicine 16, no. 6 (2011): 443. http://dx.doi.org/10.1177/1358863x11426616.

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44

BRUNK, DOUG. "Obesity Worsens Chronic Venous Insufficiency." Clinical Endocrinology News 6, no. 6 (2011): 21. http://dx.doi.org/10.1016/s1558-0164(11)70269-3.

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45

Hach, W., V. Hach-Wunderle, and F. Präve. "Staging of chronic venous insufficiency." Gefässchirurgie 5, no. 4 (2000): 0255–61. http://dx.doi.org/10.1007/s007720000103.

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46

Steins, Anke, Hans-Martin Häfner, Martin Hahn, and Michael Jünger. "Microcirculation in chronic venous insufficiency." Phlebology 17, no. 3-4 (2002): 115–20. http://dx.doi.org/10.1007/bf02638603.

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47

Rathbun, Suman W., and Angelia C. Kirkpatrick. "Treatment of chronic venous insufficiency." Current Treatment Options in Cardiovascular Medicine 9, no. 2 (2007): 115–26. http://dx.doi.org/10.1007/s11936-007-0005-6.

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48

Kistner, Robert L. "Diagnosis of chronic venous insufficiency." Journal of Vascular Surgery 3, no. 1 (1986): 185–88. http://dx.doi.org/10.1016/0741-5214(86)90098-4.

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49

Mosmiller, Lindsey T., Kelsey N. Steele, Carl D. Shrader, and Ashley B. Petrone. "Evaluation of inflammatory cell biomarkers in chronic venous insufficiency." Phlebology: The Journal of Venous Disease 32, no. 9 (2017): 634–40. http://dx.doi.org/10.1177/0268355517701806.

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Objective Inflammation has been implicated as a factor that may contribute to chronic venous insufficiency. The purpose of this study is to compare readily available inflammatory cell biomarkers, with an emphasis on neutrophil count, lymphocyte count, and neutrophil lymphocyte ratio, in patients with chronic venous insufficiency. We hypothesized that circulating leukocyte counts would be higher in the peripheral blood of patients with severe compared to mild chronic venous insufficiency. Methods We performed a retrospective medical record review of patients discharged from Ruby Memorial Hospit

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50

OU, Zhuravyev, Kurbaniyazov ZB, and Sayinaev FK. "CHRONIC VENOUS INSUFFICIENCY AND TROPHIC ULCERS OF THE LOWER EXTREMITIES." American Journal of Medical Sciences and Pharmaceutical Research 5, no. 12 (2023): 55–62. http://dx.doi.org/10.37547/tajmspr/volume05issue12-10.

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Chronic venous hypertension (CVH) triggers a whole cascade of pathological reactions, the ultimate result of which is gross changes in the trophism of soft tissues of the lower extremities. The main hemodynamic factor leading to hypertension in the superficial venous system and, subsequently, to trophic changes in the lower limb, is blood reflux from deep veins, and it is associated with valve insufficiency at the mouth of the great and small saphenous veins (vertical reflux), as well as incompetency of perforators (horizontal reflux). Moreover, 90% of the latter are localized in the calf regi

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