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1

Thiriet, Marc. Intracellular Signaling Mediators in the Circulatory and Ventilatory Systems. Springer New York, 2013.

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2

Thiriet, Marc. Tissue Functioning and Remodeling in the Circulatory and Ventilatory Systems. Springer New York, 2013.

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3

Smolander, Juhani. Circulatory and thermal adjustments to dynamic exercise in different combinations of ambient temperature, air humidity, and clothing. Dept. of Physiology, University of Kuopio, 1987.

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4

Noordergraaf, Abraham. Circulatory System Dynamics. Elsevier Science & Technology Books, 2012.

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5

Thiriet, Marc. Control of Cell Fate in the Circulatory and Ventilatory Systems. Springer, 2011.

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6

Thiriet, Marc. Control of Cell Fate in the Circulatory and Ventilatory Systems. Springer, 2014.

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7

Thiriet, Marc. Intracellular Signaling Mediators in the Circulatory and Ventilatory Systems. Springer, 2016.

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8

Thiriet, Marc. Intracellular Signaling Mediators in the Circulatory and Ventilatory Systems. Springer, 2012.

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9

Thiriet, Marc. Tissue Functioning and Remodeling in the Circulatory and Ventilatory Systems. Springer, 2013.

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10

Thiriet, Marc. Tissue Functioning and Remodeling in the Circulatory and Ventilatory Systems. Springer, 2016.

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11

Thiriet, Marc. Tissue Functioning and Remodeling in the Circulatory and Ventilatory Systems. Springer, 2013.

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12

Thiriet, Marc. Signaling at the Cell Surface in the Circulatory and Ventilatory Systems. Springer, 2011.

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13

Thiriet, Marc. Signaling at the Cell Surface in the Circulatory and Ventilatory Systems. Springer, 2011.

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14

Thiriet, Marc. Signaling at the Cell Surface in the Circulatory and Ventilatory Systems. Springer, 2016.

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15

Rowland, Thomas W. Cardiovascular function. Edited by Neil Armstrong and Willem van Mechelen. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198757672.003.0011.

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The circulatory response to increased metabolic demands of endurance exercise is best explained by a model in which volume of circulatory flow is governed by alterations in peripheral vascular resistance. These dynamics of the cardiovascular response to an acute bout of progressive endurance exercise are similar in children and adults, and, when adjusted for body size, true cardiovascular fitness (ability to generate cardiac output) is no different in healthy, untrained pre- and postpubertal individuals. As in adults, the capacity to eject stroke volume at maximal exercise differentiates level
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16

Ebm, Claudia, and Andrew Rhodes. Post-operative fluid and circulatory management in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0363.

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Fluid and circulatory management is an integral part of the peri-operative care of critically-ill patients. Precisely estimating the volumetric needs of post-operative patients remains difficult. While the majority of patients tolerate intra-operative fluid loss easily, patients with reduced physiological reserve present more of a challenge. Targeting specific physiological goals and optimizing haemodynamics with fluids and inotropes, means outcomes of these patients can be improved. This approach is often referred as goal-directed therapy (GDT). ‘Individualized goal-directed therapy’ can vary
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17

Tuxen, David V. Pathophysiology and causes of airflow limitation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0110.

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Exacerbations of asthma or chronic obstructive pulmonary disease (COPD) can be life-threatening emergencies, and require careful management to minimize the risks of morbidity and mortality. Prompt, full bronchodilator therapy, careful observation and appropriate mechanical ventilation technique is required. Dynamic hyperinflation of the lungs occurs in all patients, and must be careful assessed and regulated. Excessive dynamic hyperinflation can result in respiratory tamponade, hypotension, circulatory failure, pneumothoraces and, in severe cases, cardiac arrest. Intravenous or continuous nebu
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18

Vranckx, Pascal, Wilfried Mullens, and Johan Vijgen. Non-pharmacological therapy of acute heart failure: when drugs alone are not enough. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0053.

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Acute heart failure syndrome has been defined as new-onset or a recurrence of worsening signs and symptoms of heart failure, necessitating urgent or emergency management. The management of acute heart failure syndrome is challenging, given the heterogeneity of the patient population, in terms of the clinical presentation, pathophysiology, prognosis, and therapeutic options. The management of acute heart failure syndrome is a dynamic process, requiring ongoing simultaneous diagnosis (monitoring) and treatment. Pharmacological agents remain the mainstay of therapy for acute heart failure syndrom
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