Relevant bibliographies by topics / CK-1δ

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  1. Journal articles
  2. Dissertations / Theses

Academic literature on the topic 'CK-1δ'

Author: Grafiati
Published: 1 April 2023
Last updated: 31 July 2025

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Journal articles on the topic "CK-1δ"

1

Morales-Garcia, José A., Irene G. Salado, Marina Sanz-San Cristobal та ін. "Biological and Pharmacological Characterization of Benzothiazole-Based CK-1δ Inhibitors in Models of Parkinson’s Disease". ACS Omega 2, № 8 (2017): 5215–20. http://dx.doi.org/10.1021/acsomega.7b00869.

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Francucci, Beatrice, Simone Angeloni, Diego Dal Ben та ін. "Dual Anta-Inhibitors Targeting Protein Kinase CK1δ and A2A Adenosine Receptor Useful in Neurodegenerative Disorders". Molecules 28, № 12 (2023): 4762. http://dx.doi.org/10.3390/molecules28124762.

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Currently, the number of patients with neurodegenerative pathologies is estimated at over one million, with consequences also on the economic level. Several factors contribute to their development, including overexpression of A2A adenosine receptors (A2AAR) in microglial cells and up-regulation and post-translational alterations of some casein kinases (CK), among them, CK-1δ. The aim of the work was to study the activity of A2AAR and CK1δ in neurodegeneration using in-house synthesized A2A/CK1δ dual anta-inhibitors and to evaluate their intestinal absorption. Experiments were performed on N13
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3

Redenti, Sara, Irene Marcovich, Teresa De Vita та ін. "A Triazolotriazine‐Based Dual GSK‐3β/CK‐1δ Ligand as a Potential Neuroprotective Agent Presenting Two Different Mechanisms of Enzymatic Inhibition". ChemMedChem 14, № 3 (2019): 310–14. http://dx.doi.org/10.1002/cmdc.201800778.

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Sharma, Vinita, Jurnal Reang, Vivek Yadav, Prabodh Chander Sharma, Jaseela Majeed та Kalicharan Sharma. "Discovery of 6-amino pyridine clubbed heterocycles as potent dual GSK-3β/CK-1δ inhibitors for the treatment of Alzheimer's disease". Bioorganic Chemistry 159 (червень 2025): 108409. https://doi.org/10.1016/j.bioorg.2025.108409.

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Rodríguez-Periñán, Guiomar, Ana de la Encarnación, Fermín Moreno, et al. "Progranulin Deficiency Induces Mitochondrial Dysfunction in Frontotemporal Lobar Degeneration with TDP-43 Inclusions." Antioxidants 12, no. 3 (2023): 581. http://dx.doi.org/10.3390/antiox12030581.

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Loss-of-function (LOF) mutations in GRN gene, which encodes progranulin (PGRN), cause frontotemporal lobar degeneration with TDP-43 inclusions (FTLD-TDP). FTLD-TDP is one of the most common forms of early onset dementia, but its pathogenesis is not fully understood. Mitochondrial dysfunction has been associated with several neurodegenerative diseases such as Alzheimer’s disease (AD), Parkinson’s disease (PD) and amyotrophic lateral sclerosis (ALS). Here, we have investigated whether mitochondrial alterations could also contribute to the pathogenesis of PGRN deficiency-associated FTLD-TDP. Our
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Gürbüz, Perihan, Alim Hüseyin Dokumacı, Miyase Gözde Gündüz та ін. "In vitro biological activity of Salvia fruticosa Mill. infusion against amyloid β-peptide-induced toxicity and inhibition of GSK-3β, CK-1δ, and BACE-1 enzymes relevant to Alzheimer's disease". Saudi Pharmaceutical Journal 29, № 3 (2021): 236–43. http://dx.doi.org/10.1016/j.jsps.2021.01.007.

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Hondius, David C., Frank Koopmans, Conny Leistner, et al. "The proteome of granulovacuolar degeneration and neurofibrillary tangles in Alzheimer’s disease." Acta Neuropathologica 141, no. 3 (2021): 341–58. http://dx.doi.org/10.1007/s00401-020-02261-4.

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AbstractGranulovacuolar degeneration (GVD) is a common feature in Alzheimer’s disease (AD). The occurrence of GVD is closely associated with that of neurofibrillary tangles (NFTs) and GVD is even considered to be a pre-NFT stage in the disease process of AD. Currently, the composition of GVD bodies, the mechanisms associated with GVD and how GVD exactly relates to NFTs is not well understood. By combining immunohistochemistry (IHC) and laser microdissection (LMD) we isolated neurons with GVD and those bearing tangles separately from human post-mortem AD hippocampus (n = 12) using their typical
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Joshi, Kopal, Sukriti Goyal, Sonam Grover та ін. "Novel group-based QSAR and combinatorial design of CK-1δ inhibitors as neuroprotective agents". BMC Bioinformatics 17, S19 (2016). http://dx.doi.org/10.1186/s12859-016-1379-9.

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Martínez-González, Loreto, Carmen Rodríguez-Cueto, Diego Cabezudo та ін. "Motor neuron preservation and decrease of in vivo TDP-43 phosphorylation by protein CK-1δ kinase inhibitor treatment". Scientific Reports 10, № 1 (2020). http://dx.doi.org/10.1038/s41598-020-61265-y.

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Sharma, Vinita, Prabodh Chander Sharma, Jurnal Reang та ін. "Impact of GSK-3β and CK-1δ on Wnt signaling pathway in alzheimer disease: A dual target approach". Bioorganic Chemistry, квітень 2024, 107378. http://dx.doi.org/10.1016/j.bioorg.2024.107378.

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Dissertations / Theses on the topic "CK-1δ"

1

REDENTI, SARA. "DESIGN, SYNTHESIS AND CHARACTERIZATION OF GSK-3β AND CK-1δ INHIBITORS". Doctoral thesis, Università degli Studi di Trieste, 2017. http://hdl.handle.net/11368/2908172.

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Glycogen Synthase Kinase 3-β (GSK-3β) and Casein Kinase 1-δ (CK-1δ) are both highly conserved serine/threonine protein kinases that seem to be involved in neuroinflammation and in the development and progression of several disorders of the central nervous system (CNS), such as Alzheimer’s disease (AD) and Parkinson’s disease (PD). In particular, both are related to the hyperphosphorylation of tau-protein, one of the major hallmark of AD and other taupathies. So, the inhibition of GSK-3β and or CK-1δ could represent a strategy for the treatment of these unmet diseases. The main purpose of this
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