Relevant bibliographies by topics / Cleft

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Cleft'

Author: Grafiati
Published: 4 June 2021
Last updated: 9 March 2023

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Journal articles on the topic "Cleft"

1

Weber, Elizabeth G. "The Bondei object pronoun in clefts and pseudo-clefts.pdf." Studies in African Linguistics 19, no. 2 (August 1, 1988): 233–57. http://dx.doi.org/10.32473/sal.v19i2.107464.

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Verbs in Bondei, a Bantu language spoken in East Africa, have crossreferencing pronouns which agree in noun class with the subject and object of the clause. This paper will examine the distribution of the syntactic category object pronoun in four grammatical constructions: (a) basic affirmative declarative clauses; (b) relative clauses; (c) clefts; and (d) pseudo-clefts. In declarative and relative clauses, the presence of the object pronoun does not require a definite interpretation of the object noun; the absence of the object pronoun does not preclude a definite interpretation of the object noun. In both cleft and pseudo-cleft constructions, however, the object pronoun obligatorily functions to grammatically mark clef ted and pseudo-clef ted objects as definite. In the cleft constructions, the definiteness of the clef ted NP forces a contrastive interpretation. Thus, a judgement concerning the function of this grammatical construction with regard to contrastive function will be made on the basis of the distribution of the syntactic category object pronoun. possible to make the same judgment. Contrastive function is unambiguously signaled by the relative morphology on the verb of the pseudo-cleft. In this constuction, the object pronoun serves only to force a definite interpretation of the NP. In the pseudo-cleft constructions, it is not possible to make the same judgment. Contrastive function is unambiguously signaled by the relative morphology on the verb of the pseudo-cleft. In this constuction, the object pronoun serves only to force a definite interpretation of the NP.
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Romanec, Cristian, Mariana Pacurar, Mioara Decusara, Monica Mihaela Scutariu, Delia Hinganu, Marius Valeriu Hinganu, and Corina Ciupilan. "Labio-palatine Cleft, Morphological Substrate." Revista de Chimie 69, no. 4 (May 15, 2018): 1002–5. http://dx.doi.org/10.37358/rc.18.4.6246.

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Labio-palatin clefts are the most common type of craniofacial malformation encountered in practice and among the most common congenital abnormalities; it represent morphological and functional defects in newborns caused by genetically changes or environmental factors; those of the cervico-facial region have an increased incidence due to local embryological complexity and multifactorial etiology. The anomaly, with its variants, results from an inherent defect between the internal and external nasal buds, both from the frontal and upper maxillary bud. Labio-palatine clefts are characterized by a tissue deficiency as well as an abnormal disposition of the present tissues. It is believed that the labial clefts are consecutive to the lack of apoptosis of the ectoderm which envelops the internal maxillary and nasal buds, thus constituting a barrier to the fusion process of buds. Depending on the location and the extentension of the cleft, the labial cleft occurs, or the more frequent, the labial-alveolar cleft. The study group consisted of 22 patients with labio-palatine clefts, of which 16 boys (72.72%) and 6 girls (27.27%), who presented in the clinic for treatment. When the causative factor acts earlier, the clfet develops posteriorly, since mesodermization begins from the posterior to the anterior. The more disturbing factor acts, the greater the cleft. The increase in the incidence of labial-palatine cleft is believed to be mainly due to the increase in the number of women of older gestational age in women; women over 35 years of age have a double risk of having children with labio-palatine clefts, the risk being three times higher for mothers over the 39 years.
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Carroll, K., and P. A. Mossey. "Anatomical Variations in Clefts of the Lip with or without Cleft Palate." Plastic Surgery International 2012 (November 29, 2012): 1–6. http://dx.doi.org/10.1155/2012/542078.

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Objective. Few orofacial cleft (OFC) studies have examined the severity of clefts of the lip or palate. This study examined associations between the severity of cleft of the lip with cleft type, laterality, and sex in four regional British Isles cleft registers whilst also looking for regional variations. Design. Retrospective analysis of cleft classification in the data contained in these four cleft registers. Sample. Three thousand and twelve patients from cleft registers based in Scotland, East England, Merseyside, and Belfast were sourced from the period 2002–2010. Submucous clefts and syndromic clefts were included whilst stillbirths, abortuses, and atypical orofacial clefts were excluded. Results. A cleft of the lip in CLP patients is more likely to be complete in males. A cleft of the lip in isolated CL patients is more likely to be complete in females. Variation in the proportion of cleft types was evident between Scotland and East England. Conclusions. Association between severity of cleft of the lip and sex was found in this study with females having a significantly greater proportion of more severe clefts of the lip (CL) and CLP males being more severe . This finding supports a fundamental difference between cleft aetiology between CL and CLP.
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Corcoran, Mirjami, Saujanya Karki, Leena Ylikontiola, Riitta Lithovius, George K. Sándor, and Virpi Harila. "Maxillary Arch Dimensions in 6-Year-Old Cleft Children in Northern Finland: A Cross-Sectional Study." International Journal of Environmental Research and Public Health 18, no. 14 (July 12, 2021): 7432. http://dx.doi.org/10.3390/ijerph18147432.

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The aim was to cross-sectionally examine the maxillary arch dimensions in 6-year-old children with cleft lip and/or palate and to compare them with the initial cleft sizes among patients with cleft palate. The study included 89 patients with clefts treated at the Oulu University Hospital. The subjects were divided into three groups: cleft palate, cleft lip, and cleft lip and palate. Study casts were scanned, and the maxillary arch dimensions were examined using a 3D program (3Shape Orthoanalyzer, Copenhagen, Denmark). The statistical methods Student’s t-test and one-way ANOVA were used to compare the means (SD) between the groups. Spearman’s correlation coefficient was used to determine the correlation between cleft severity and maxillary dimensions. A significant difference was found between different initial cleft sizes in terms of distance between the second deciduous molar and the first incisor on the right side. The intermolar width showed a negative correlation with the initial cleft size. The dimensions were shorter for clefts affecting the palate and largest for clefts affecting only the lip. Larger clefts resulted in a shorter maxilla on the right side. Many dimensions became shorter when the initial cleft was larger. Clefts of the palate resulted in smaller maxillas.
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Russell, Kathleen A., Victoria M. Allen, Mary E. MacDonald, Kirsten Smith, and Linda Dodds. "A Population-Based Evaluation of Antenatal Diagnosis of Orofacial Clefts." Cleft Palate-Craniofacial Journal 45, no. 2 (March 2008): 148–53. http://dx.doi.org/10.1597/06-202.1.

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Objectives: To evaluate the changes in prevalence and antenatal detection of cleft lip with or without cleft palate and isolated cleft palate and to describe the association between anomalies and rates of antenatal diagnosis in Nova Scotia from 1992 to 2002. Design: This population-based cohort study employed the Nova Scotia Atlee Perinatal Database, the Fetal Anomaly Database, and IWK Cleft Palate Database in Halifax, Nova Scotia, Canada. Outcome Measures: Cleft type, mode of diagnosis, and associated abnormalities of orofacial clefts for liveborn infants, stillbirths, and second trimester terminations of pregnancy between 1992 and 2002 were determined. Results: There were 225 fetuses identified as having orofacial clefts. The overall prevalence of clefts was 2.1 in 1000 live births, and this prevalence did not change with time. The overall antenatal detection of cleft lip with or without cleft palate was 23%; however, there was improvement in detection of cleft lip with or without cleft palate from the years 1992 to 1996 (14%) to the years 1997 to 2002 (30%, p = .02). No isolated cleft palates were detected antenatally. Associated structural anomalies were seen in 34.2% of cases with orofacial clefts, and chromosomal abnormalities were associated with 9.8%. Conclusions: The prevalence of orofacial clefts in Nova Scotia has not changed from 1992 to 2002. The proportion of antenatally diagnosed cleft lip with or without cleft palate in Nova Scotia is consistent with rates reported in the literature and has increased from 1992 to 2002.
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Forrester, Mathias B., and Ruth D. Merz. "Structural Birth Defects Associated with Oral Clefts in Hawaii, 1986 to 2001." Cleft Palate-Craniofacial Journal 43, no. 3 (May 2006): 356–62. http://dx.doi.org/10.1597/04-190.1.

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Objective To identify structural birth defects that occur in association with oral clefts. Methods Data were obtained from a birth defects registry and included all infants and fetuses with cleft palate without cleft lip or cleft lip with or without cleft palate delivered from 1986 to 2001. For 47 specific structural birth defects, rates among oral cleft cases were compared with the rates among all infants and fetuses with major birth defects, excluding those with oral clefts. Results Among cleft palate only cases, the rates were significantly higher than expected for encephalocele, microcephaly, and syndactyly. Among cases of cleft lip with or without cleft palate, the rates were significantly higher than expected for anophthalmia/microphthalmia, single ventricle, reduction deformity of upper limbs, and reduction deformity of lower limbs. When cases of cleft palate only and cleft lip with or without cleft palate were compared as to the rates for particular birth defects, the rates of the defects were either higher or lower than expected in both oral cleft categories for 38 (81%) of the defects. Conclusions Certain birth defects were more frequently associated with oral clefts than might be expected. For the majority of defects, their patterns of association were similar between cleft palate without cleft lip and cleft lip with or without cleft palate.
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Stahl, Franka, Rosemarie Grabowski, and Katrin Wigger. "Epidemiology of Hoffmeister's “Genetically Determined Predisposition to Disturbed Development of the Dentition” in Patients with Cleft Lip and Palate." Cleft Palate-Craniofacial Journal 43, no. 4 (July 2006): 457–65. http://dx.doi.org/10.1597/04-156.1.

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Objective Type and prevalence rates of the symptoms of Hoffmeister's “genetically determined predisposition to disturbed development of the dentition” were studied in patients with clefts. Patients Data of 263 patients with nonsyndromic clefts of lip (alveolus and palate) or isolated cleft palates were examined in a retrospective study. Setting The clefts were classified as cleft lip or cleft lip and alveolus, cleft palate, unilateral cleft lip and palate, and bilateral cleft lip and palate. All patients were scrutinized for 28 individual symptoms. Prevalences of the individual symptoms were statistically evaluated regarding cleft type and gender by using the chi-square test and were also compared with findings in patients without clefts. Results In 97.7% of the patients with clefts, at least one symptom was found. Microdontia of individual teeth, hypodontia, and hyperodontia were the symptoms most frequently recorded. Comparison of the different cleft types revealed differences regarding the prevalences of supernumerary lateral incisors (p = .051), infraposition of deciduous molars (p < .001), and atypical tooth bud position (p = .030). Comparison of the prevalences of 10 symptoms recorded in the patients with clefts with the prevalences recorded in patients without clefts showed nine symptoms were found much more frequently in the population with clefts. Conclusion These findings support the hypothesis that clefting is part of a complex malformation associated with other dental anomalies resulting from disturbed development of the dentition. Patients with clefts are also likely to present other deficiencies of dental development and tooth eruption in both dentitions, even in regions not affected by the cleft.
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Letra, A., R. Menezes, J. M. Granjeiro, and A. R. Vieira. "Defining Subphenotypes for Oral Clefts Based on Dental Development." Journal of Dental Research 86, no. 10 (October 2007): 986–91. http://dx.doi.org/10.1177/154405910708601013.

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Individuals with clefts present considerably more dental anomalies than do individuals without clefts. We used dental development to subphenotype clefts with the goal of identifying cleft subgroups that could have specific genetic contributions. We examined 1000 individuals, 500 with clefts and 500 without. We used several clinical features, such as cleft completeness or incompleteness, laterality, and the presence of dental anomalies to assess each individual’s cleft status. We performed chi-square and Fisher’s exact tests to compare the frequencies of observed anomalies between individuals with and individuals without clefts, and among individuals with different cleft subphenotypes. Agenesis of the lateral incisor on the non-cleft side was the most remarkable observation, and may suggest that such cases could be considered incomplete forms of bilateral clefts of the lip.
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Inoue, Hiroki, Shuji Kayano, Yoko Aoki, Shigeo Kure, Atsushi Yamada, Akira Hata, Yoichi Matsubara, and Yoichi Suzuki. "Association of the GABRB3 Gene with Nonsyndromic Oral Clefts." Cleft Palate-Craniofacial Journal 45, no. 3 (May 2008): 261–66. http://dx.doi.org/10.1597/06-142.

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Objective: Nonsyndromic oral clefts are common craniofacial anomalies classified into two subgroups: cleft lip with or without cleft palate and isolated cleft palate. Nonsyndromic oral clefts are multifactorial diseases, with both genetic and environmental factors involved in their pathogenesis. The inhibitory neurotransmitter, γ-aminobutyric acid plays a role in normal embryonic, and particularly facial, development and γ-aminobutyric acid receptor type A β-3 subunit (GABRB3) knockout mice have been shown to have cleft palate. The GABRB3 gene is therefore a strong candidate gene for nonsyndromic oral clefts. We investigated here whether genetic variations of the GABRB3 gene affect the risk for nonsyndromic oral clefts. Method: In this case-control study, a total of 178 Japanese patients with cleft lip with or without cleft palate and 374 unrelated controls were recruited and were genotyped for six single nucleotide polymorphisms and a dinucleotide repeat marker of the GABRB3 gene. Results: None of the single nucleotide polymorphisms showed complete linkage disequilibrium with other single nucleotide polymorphisms. In a case-control association study with the six-locus haplotype of the gene, TGTGCT haplotype frequency in patients with cleft lip with or without cleft palate was significantly higher than in the controls (corrected p value = .029). None of the alleles of the dinucleotide repeat marker showed significant association with cleft lip with or without cleft palate. Conclusions: Our data suggest that the GABRB3 gene is involved in the pathogenesis of cleft lip with or without cleft palate in the Japanese population.
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Al-Qattan, M. M. "Central and ulnar cleft hands: a review of concurrent deformities in a series of 47 patients and their pathogenesis." Journal of Hand Surgery (European Volume) 39, no. 5 (July 3, 2013): 510–19. http://dx.doi.org/10.1177/1753193413496945.

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Two main types of cleft hands have been described. The ulnar cleft hand deformity is very rare and is characterized by two constant features: a deep cleft radial to the little finger and hypoplasia of the ulnar digits. The pathogenesis of ulnar clefts is unknown. The second type is the central cleft hand deformity, which is characterized by a soft tissue/bone defect in the hand centrally. Patients with central clefts also have several concurrent deformities in the remaining digits. This paper reviews the clinical features of three cases with ulnar cleft hands and 44 cases of central cleft hands, with special emphasis on concurrent deformities. The author’s hypothesis of pathogenesis for both types of clefts and their concurrent deformities is then offered.
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Dissertations / Theses on the topic "Cleft"

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Kemp, Robin. "Cleft." ScholarWorks@UNO, 2002. http://www.uno.edu/theses/available/etd-11252002-141204/.

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Thesis (M.F.A.)--University of New Orleans, 2002.
Title from electronic submission form. Vita. "A thesis ... in partial fulfillment of the requirements for the degree of Master of Fine Arts in Drama and Comunications in the Creative Writing Workshop"--Thesis t.p. Includes bibliographical references.
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Reiser, Erika. "Cleft Size and Maxillary Arch Dimensions in Unilateral Cleft Lip and Palate and Cleft Palate." Doctoral thesis, Uppsala universitet, Käkkirurgi, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-160178.

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The wide variation in infant maxillary morphology and cleft size of children with unilateral cleft lip and palate (UCLP) and isolated cleft palate (CP) raise concerns about their possible influences on treatment outcome. The studies in this thesis aimed to investigate the relation between cleft size in infancy and crossbite at 5 years of age (Paper I); the impact of primary surgery on cleft size and maxillary arch dimensions from infancy to 5 years of age (Paper II); associations between cleft size, maxillary arch dimensions and facial growth in both UCLP and CP children (Paper III); and, to evaluate the relation between infant cleft size and nasal airway size and function in adults treated for UCLP (Paper IV). In homogenously treated groups of children with UCLP and CP, dental casts were used to measure cleft size and maxillary arch dimensions from infancy up to 5 years of age, and for crossbite recording at 5 years. Serial lateral cephalometric radiographs taken between 5 and 19 years of age in the same groups were used to study facial growth. Nasal airway size and function were evaluated by acoustic rhinometry, rhinomanometry, peak nasal inspiratory flow and odour test in a group of adults treated for UCLP. The main findings were: crossbite was a frequent malocclusion at 5 years of age in children with UCLP and large cleft widths at the level of the cuspid points in infancy were associated with less anterior and posterior crossbite in this group (Paper I). Cleft widths decreased after lip closure and/or soft palate closure in both UCLP and CP children. Initially, UCLP children had wider maxillary arch dimensions, but after hard palate closure, the transverse growth was reduced, and at 5 years, they had smaller maxillary arch widths than CP children had (Paper II). Maxillary arch depths and cleft widths in infancy were correlated with maxillary protrusion and sagittal jaw relationships in both UCLP and CP children (Paper III), but cleft width in infancy was not correlated with nasal airway size and function in adults treated for UCLP (Paper IV).
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Kutbi, Hebah Alawi. "THE ROLE OF OBESITY, DIABETES, AND HYPERTENSION IN CLEFT LIP AND CLEFT PALATE BIRTH DEFECTS." DigitalCommons@USU, 2014. https://digitalcommons.usu.edu/etd/3081.

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Orofacial clefts (OFCs) are among the most common structural birth defects and a public health problem. Several studies suggest that maternal obesity pre-existing diabetes mellitus (DM), and the underlying metabolic abnormalities, may be involved in the pathogenesis of cleft lip (CL) and cleft palate (CP) birth defects. Although hypertension and gestational diabetes mellitus (GDM) have been associated in a few studies with congenital birth defects, studies examining the risk associated with OFCs are limited. The overall objective of this dissertation was to examine the association between maternal obesity, DM, GDM, and hypertension and the risk of OFCs in case-control studies. Analyses of data from an international consortium revealed that maternal obesity (pre-pregnancy BMI >30), compared to normal weight (18.525), was associated with an increased risk of cleft palate with or without cleft lip (CP/L) (adjusted odds ratio (aOR) =1.13 [95% confidence intervals (CI) 1.01-1.25]). We also found a marginal association between maternal underweight and CP/L (1.0 [reference]; aOR=1.14 [0.97-1.34]. CL only was not associated with maternal bodyweight. Interestingly, among college-graduates, there was no increased risk of CP, but mothers with less than a completed college education had an increased risk of CP for underweight and obesity. Investigation of the Utah OFC data provided evidence that maternal GDM is significantly associated with isolated (aOR=2.63 [1.30-5.34]) and non-isolated clefts (aOR=2.66 [1.02-6.97]). Maternal hypertension is significantly associated with non-isolated clefts (aOR=6.56 [2.18-19.77]). We found a further elevated risk of OFCs among GDM mothers and those with hypertension who were also obese. The analyses of data from an international consortium revealed significant associations between maternal diabetes and the risk of OFCs. The estimated relative risk of DM for isolated OFCs was 1.33 [1.14-1.54] and was slightly higher for multiple OFCs (aOR=1.86 [1.44-2.40]). Diabetic mothers with abnormal body-mass-index had an increased risk for having inborn with OFCs. Throughout the dissertation, we demonstrated the extent in which maternal obesity, pre-existing DM, GDM, and maternal hypertension may increase the risk of OFC birth defects. The results highlight the need for pre-conceptional program planning for the prevention of OFCs with screening for abnormal glucose tolerance and hypertension.
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Crout, Richard Morrow. "Timing of alveolar cleft bone grafting in maxillary alveolar cleft defects." Morgantown, W. Va. : [West Virginia University Libraries], 2000. http://etd.wvu.edu/templates/showETD.cfm?recnum=1446.

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Delin, Judith Lesley. "Cleft constructions in discourse." Thesis, University of Edinburgh, 1990. http://hdl.handle.net/1842/6599.

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This thesis presents an analysis of the structure and function of cleft constructions in discourse. Drawing on a corpus of naturally-occurring spoken and written data, we present a multi-layered explanation of how it-clefts, wh-clefts, and reverse wh-clefts are different from non-clefts, and from one another. After a review of previous research on clefts in discourse, we explore the aspects of syntax, semantics, and pragmatics relevant to the structure and function of all three types of cleft. The discussion falls into three main parts: An analysis of the three cleft types, within the framework of Generalized Phrase Structure Grammar (cf. Gazdar et al. [1985]), in which particular attention is paid to the variety of constituents that can appear in particular positions in each type. The output of the grammar rules is compared to the examples that occur in the corpus of data. A treatment of cleft presupposition in terms of an analogy (suggested by van der Sandt [1988]) between presupposition and the treatment of pronominal anaphora in Kamp's [1981] Discourse Representation Theory An examination of the range of accentual patterns, presuppositional relations, and information structures typically appearing in clefts of all three kinds. We show that marked distinctions exist between the three cleft types in terms of all these factors, and suggest ways in which this helps to differentiate the range of discourse contexts in which clefts in general, and each cleft type in particular, are appropriate. At the end of the thesis we point to an analogy between the formal model for clefts presented and a psychological model of sentence processing We also suggest how the conclusions regarding both the structure and function of clefts as a class of construction and the distinction between the three types of cleft could be synthesised in a decision procedure for syntactic choice. Finally, we suggest some related areas for further research.
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Shaw, Diana Frances. "Genetic analysis of non-syndromic and syndromic cleft lip with or without cleft palate." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/nq24564.pdf.

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Ho, Kwan-wai Annie. "Variability of cleft palate speech." Click to view the E-thesis via HKUTO, 2001. http://sunzi.lib.hku.hk/hkuto/record/B36207883.

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Thesis (B.Sc)--University of Hong Kong, 2001.
"A dissertation submitted in partial fulfilment of the requirements for the Bachelor of Science (Speech and Hearing Sciences), The University of Hong Kong, May 4, 2001." Also available in print.
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Kizu, Mika. "Unbounded dependencies in cleft constructions." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp03/NQ64589.pdf.

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Hui, Edward. "Soft tissue changes following maxillary osteotomies in cleft lip and palate and non-cleft patients." Click to view the E-thesis via HKUTO, 1992. http://sunzi.lib.hku.hk/HKUTO/record/B38628338.

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許嘉榮 and Edward Hui. "Soft tissue changes following maxillary osteotomies in cleft lip and palate and non-cleft patients." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1992. http://hub.hku.hk/bib/B38628338.

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Books on the topic "Cleft"

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Cleft and pseudo-cleft constructions in English. London: Routledge, 1991.

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Lessing, Doris May. The Cleft. Glasgow: HarperCollins, 2008.

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Hartmann, Katharina, and Tonjes Veenstra, eds. Cleft Structures. Amsterdam: John Benjamins Publishing Company, 2013. http://dx.doi.org/10.1075/la.208.

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Cleft palate. Austin, Tex: PRO-ED, 1986.

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Lessing, Doris. The Cleft. New York: HarperCollins, 2007.

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Cleft structures. Amsterdam: John Benjamins Publishing Company, 2013.

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Foundation, Cleft Palate. Cleft lip and cleft palate: The first four years. 3rd ed. Chapel Hill, NC (104 South Estes Dr., Chapel Hill 27514): The Foundation, 1998.

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McKinstry, Robert E. Cleft palate dentistry. Arlington, VA: ABI Professional Publications., 1998.

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Albery, E. H. Cleft palate sourcebook. Bicester: Winslow, 1994.

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E, Losee Joseph, and Kirschner Richard E, eds. Comprehensive cleft care. New York: McGraw-Hill Medical, 2008.

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Book chapters on the topic "Cleft"

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Dhillon, Ramindar S., and James W. Fairley. "Cleft lip and cleft palate." In Multiple-choice Questions in Otolaryngology, 88. London: Palgrave Macmillan UK, 1989. http://dx.doi.org/10.1007/978-1-349-10805-3_125.

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Choudhury, Subhasis Roy. "Cleft Lip and Cleft Palate." In Pediatric Surgery, 67–71. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-6304-6_11.

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Chen, Harold. "Cleft Lip and/or Cleft Palate." In Atlas of Genetic Diagnosis and Counseling, 1–10. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4614-6430-3_43-2.

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Chen, Harold. "Cleft Lip and/or Cleft Palate." In Atlas of Genetic Diagnosis and Counseling, 475–84. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-2401-1_43.

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Parikh, Dakshesh. "Sternal Cleft." In Tips and Tricks in Thoracic Surgery, 241–48. London: Springer London, 2018. http://dx.doi.org/10.1007/978-1-4471-7355-7_18.

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Swan, Marc C., and David M. Fisher. "Cleft lip." In Plastic and reconstructive surgery, 204–18. Chichester, UK: John Wiley & Sons, Ltd, 2015. http://dx.doi.org/10.1002/9781118655412.ch17.

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Giddens, Cheryl L. "Cleft Palate." In Encyclopedia of Child Behavior and Development, 371–73. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-0-387-79061-9_568.

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Dobratz, Eric J. "Cleft Lip." In Encyclopedia of Otolaryngology, Head and Neck Surgery, 464–72. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-23499-6_332.

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Watters, Karen, Lynne Ferrari, and Reza Rahbar. "Laryngeal Cleft." In Advances in Oto-Rhino-Laryngology, 95–100. Basel: S. KARGER AG, 2012. http://dx.doi.org/10.1159/000334452.

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Balakrishnan, Karthik, and Kari A. Krein. "Laryngeal Cleft." In Multidisciplinary Management of Pediatric Voice and Swallowing Disorders, 269–83. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-26191-7_27.

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Conference papers on the topic "Cleft"

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Zhang, Yungeng, Yuru Pei, Si Chen, Yuke Guo, Gengyu Ma, Tianmin Xu, and Hongbin Zha. "Volumetric Registration-Based Cleft Volume Estimation of Alveolar Cleft Grafting Procedures." In 2020 IEEE 17th International Symposium on Biomedical Imaging (ISBI). IEEE, 2020. http://dx.doi.org/10.1109/isbi45749.2020.9098407.

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Jutinico, Andres Leonardo, and Flavio Prieto. "Visual Lip Segmentation in Patients with Cleft Lip and/or Cleft Palate." In 2011 IEEE Electronics, Robotics and Automotive Mechanics Conference (CERMA). IEEE, 2011. http://dx.doi.org/10.1109/cerma.2011.84.

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Schwan, F., V. Artinger, and HG Gassner. "Cleft rhinoplasty – advanced endonasal strategies." In Abstract- und Posterband – 89. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Forschung heute – Zukunft morgen. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1640801.

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Fu, Bingmei M., and Bin Chen. "A Model for the Structural Mechanisms in the Regulation of Microvessel Permeability by cAMP." In ASME 2001 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2001. http://dx.doi.org/10.1115/imece2001/htd-24416.

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Abstract To investigate the microstructural mechanisms of decreasing microvascular permeability induced by the enhancement of intraendothelial adenosine 3’,5’-cyclic monophosphate (cAMP) levels, we extended the previous analytical model developed by Fu et al. (J. Biomech. Eng., 1994) for the interendothelial cleft to include multiple junction strands in the cleft and an interface between the surface glycocalyx layer and the cleft entrance. Based on the electron microscopic observation by Adamson et al. (Am. J. Physiol., 1998), that elevation of intracellular cAMP levels would increase number of tight junction strands, numerical method was applied to test the case in which there are two junction strands in the cleft and there are large discontinuous breaks and a small continuous slit in each strand. Results from this two-junction-strand and two-pore model can successfully account for the experimental data for the decreased permeability to water (Adamson et al. Am. J. Physiol., 1998), small and intermediate-sized solutes (Fu et al., Am. J. Physiol., 1998) by cAMP.
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Benjamin, Israel, Rochelle Johns, Giovanni Sisti, Jana Yancey, Andrej Bogojevic, Ronald Bainbridge, and Kecia Gaither. "Prenatal Diagnosis of Cleft Lip + Palate." In 13th Philadelphia Prenatal Virtual Conference—Selected Abstracts. Thieme Medical Publishers, Inc., 2021. http://dx.doi.org/10.1055/s-0041-1735768.

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Benjamin, Israel, Rochelle Johns, Giovanni Sisti, Jana Yancey, Andrej Bogojevic, Ronald Bainbridge, and Kecia Gaither. "Prenatal Diagnosis of Cleft Lip + Palate." In 13th Philadelphia Prenatal Virtual Conference—Selected Abstracts. Thieme Medical Publishers, Inc., 2021. http://dx.doi.org/10.1055/s-0041-1735768.

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Chen, Shuang, Amir Atapour-Abarghouei, Jane Kerby, Edmond S. L. Ho, David C. G. Sainsbury, Sophie Butterworth, and Hubert P. H. Shum. "A Feasibility Study on Image Inpainting for Non-cleft Lip Generation from Patients with Cleft Lip." In 2022 IEEE-EMBS International Conference on Biomedical and Health Informatics (BHI). IEEE, 2022. http://dx.doi.org/10.1109/bhi56158.2022.9926917.

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Kurpukdee, Nattapong, Kwanchiva Thangthai, Vataya Chunwijitra, Patcharika Chootrakool, and Sawit Kasuriya. "NASAM 2.0: Cleft-Palate Speech Assessment Application." In 2022 25th Conference of the Oriental COCOSDA International Committee for the Co-ordination and Standardisation of Speech Databases and Assessment Techniques (O-COCOSDA). IEEE, 2022. http://dx.doi.org/10.1109/o-cocosda202257103.2022.9997825.

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Warrick, Amanda E., J. Douglas Swarts, and Samir N. Ghadiali. "Fluid Structure Interactions in the Eustachain Tube Under Normal and Pathological Conditions." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-175328.

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Cleft Palate is a craniofacial syndrome in which the two plates that form the hard palate are not completely joined. As a result, the soft tissue anatomy of the Eustachian Tube (ET) is altered. The ET is a collapsible tube which connects the middle ear (ME) with the nasopharynx (NP). The ET must be periodically opened to equalize ME and NP pressures and drain ME fluids. In healthy adults, ET openings occur during swallowing, where muscle contraction deforms the surrounding soft tissue. However, changes in tissue anatomy may lead to ET dysfunction (i.e. closure during swallowing) and the development of ME disorders such as Otitis Media (OM)[1]. These disorders are especially problematic in infants with cleft palate as they hinder speech, hearing and psychosocial development. Although surgical procedures can be used to repair a cleft palate, these procedures do not typically account the possible development of ET dysfunction and/or OM.
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C.M., Vikram, Nagaraj Adiga, and S. R. Mahadeva Prasanna. "Spectral Enhancement of Cleft Lip and Palate Speech." In Interspeech 2016. ISCA, 2016. http://dx.doi.org/10.21437/interspeech.2016-842.

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Reports on the topic "Cleft"

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Philp, Jennifer, Ariel Vovakes, Hope Lancaster, Jennifer Frey, and Nancy Scherer. Changes in Articulation and Phonological Patterns During Early Intervention in Children with Cleft Palate With or Without Cleft Lip. Journal of Young Investigators, July 2018. http://dx.doi.org/10.22186/jyi.35.1.20-28.

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Ahila, S. C. Technique of managing a cleft palate in a newborn. Science Repository, July 2019. http://dx.doi.org/10.31487/j.dobcr.2019.02.04.

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Lindaas, Kirsten. Oregon Survey of Initial Nursing Care for Infants with Cleft Lip ± Palate. Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.7187.

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Egeland, A., J. A. Holtet, and P. E. Sandholt. The Electrodynamics of the Dayside Cleft Region Based on Ground Observations at Svalbard. Fort Belvoir, VA: Defense Technical Information Center, January 1992. http://dx.doi.org/10.21236/ada251239.

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Qian, Ao, Xin Zhang, Gang Huo, Jiaojiao Yu, and Xiaoshu Wang. Risk factors of recurrence in Rathke cleft cyst: a systematic review and meta analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2021. http://dx.doi.org/10.37766/inplasy2021.10.0070.

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Egeland, A., J. A. Holtet, P. E. Sandholt, and B. Jacobsen. The Electrodynamics of the Dayside Cleft Region Based on a Net of Ground Observations in the Svalbard Area. Fort Belvoir, VA: Defense Technical Information Center, January 1991. http://dx.doi.org/10.21236/ada251235.

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Hellier, Paul C., Jerald S. Bradshaw, and Reed M. Izatt. Synthesis of Chiral Pyridine-Based Macrobicyclic Clefts. Fort Belvoir, VA: Defense Technical Information Center, May 1996. http://dx.doi.org/10.21236/ada309257.

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McCall, JeannieKay. Effects of Orofacial Clefts on Early Career Maturity. Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.7280.

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Mayer, Michael H., and Jeffrey Hollinger. The Use of Bone Repair Materials for Maxillary Alveolar Clefts. Fort Belvoir, VA: Defense Technical Information Center, September 1992. http://dx.doi.org/10.21236/ada259273.

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Wisniewski, Michael, Samir Droby, John Norelli, Dov Prusky, and Vera Hershkovitz. Genetic and transcriptomic analysis of postharvest decay resistance in Malus sieversii and the identification of pathogenicity effectors in Penicillium expansum. United States Department of Agriculture, January 2012. http://dx.doi.org/10.32747/2012.7597928.bard.

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Use of Lqh2 mutants (produced at TAU) and rNav1.2a mutants (produced at the US side) for identifying receptor site-3: Based on the fact that binding of scorpion alpha-toxins is voltage-dependent, which suggests toxin binding at the mobile voltage-sensing region, we analyzed which of the toxin bioactive domains (Core-domain or NC-domain) interacts with the DIV Gating-module of rNav1.2a. This analysis was based on the assumption that the dissociation of toxin mutants upon depolarization would vary from that of the unmodified toxin should the substitutions affect a site of interaction with the channel Gating-module. Using a series of toxin mutants (mutations at both domains) and two channel mutants that were shown to reduce the sensitivity to scorpion alpha-toxins, and by comparison of depolarization-driven dissociation of Lqh2 derivatives off their binding site at rNav1.2a mutant channels we found that the toxin Core-domain interacts with the Gating-module of DIV. Details of the experiments and results appear in Guret al (2011). Mapping receptor site 3 at Nav1.2a by extensive channel mutagenesis (Seattle): Since previous studies with photoaffinity labeling and antibody mapping implicated domains I and IV in scorpion alpha-toxin binding, Nav1.2 channel mutants containing substitutions at these extracellular regions were expressed and tested for receptor function by whole-cell voltage clamp. Of a large number of channel mutants, T1560A, F1610A, and E1613A in domain IV had ~5.9-, ~10.7-, and ~3.9-fold lower affinities for the scorpion toxin Lqh2, respectively, and mutant E1613R had 73-fold lower affinity. Toxin dissociation was accelerated by depolarization for both wild-type and mutants, and the rates of dissociation were also increased by mutations T1560A, F1610A and E1613A. In contrast, association rates for these three mutant channels at negative membrane potentials were not significantly changed and were not voltage-dependent. These results indicated that Thr1560 in the S1-S2 loop, Phe1610 in the S3 segment, and Glu1613 in the S3-S4 loop in domain IV participate in toxin binding. T393A in the SS2-S6 loop in domain I also showed a ~3.4-fold lower affinity for Lqh2, indicating that this extracellular loop may form a secondary component of the toxin binding site. Analysis with the Rosetta-Membrane algorithm revealed a three-dimensional model of Lqh2 binding to the voltage sensor in a resting state. In this model, amino acid residues in an extracellular cleft formed by the S1-S2 and S3-S4 loops in domain IV that are important for toxin binding interact with amino acid residues on two faces of the wedge-shaped Lqh2 molecule that are important for toxin action. The conserved gating charges in the S4 transmembrane segment are in an inward position and likely form ion pairs with negatively charged amino acid residues in the S2 and S3 segments (Wang et al 2011; Gurevitz 2012; Gurevitzet al 2013).
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