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Relevant bibliographies by topics / Clinical endocrinology

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Academic literature on the topic 'Clinical endocrinology'

Author: Grafiati

Published: 4 June 2021

Last updated: 28 July 2024

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Journal articles on the topic "Clinical endocrinology"

1

Polychronakos, Constantin. "Genetic Testing in Clinical Endocrinology." HORMONES 2, no. 4 (October 15, 2003): 201–10. http://dx.doi.org/10.14310/horm.2002.11101.

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Mazzaferri, Ernest L. "Clinical endocrinology." Trends in Endocrinology & Metabolism 4, no. 4 (May 1993): 142–43. http://dx.doi.org/10.1016/1043-2760(93)90040-l.

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Khaledi, A. "Clinical endocrinology." Postgraduate Medical Journal 71, no. 831 (January 1, 1995): 61. http://dx.doi.org/10.1136/pgmj.71.831.61-b.

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Berlinger, F. G. "Clinical Endocrinology." JAMA: The Journal of the American Medical Association 279, no. 20 (May 27, 1998): 1663—a—1663. http://dx.doi.org/10.1001/jama.279.20.1663-a.

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Felicetta, James V. "Clinical Endocrinology." JAMA: The Journal of the American Medical Association 257, no. 17 (May 1, 1987): 2362. http://dx.doi.org/10.1001/jama.1987.03390170118042.

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Rovner, David R. "Reviews and Notes: Endocrinology: Clinical Endocrinology." Annals of Internal Medicine 120, no. 2 (January 15, 1994): 175. http://dx.doi.org/10.7326/0003-4819-120-2-199401150-00039.

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Byrne, Geoff. "Clinical paediatric endocrinology." Medical Journal of Australia 152, no. 4 (February 1990): 219. http://dx.doi.org/10.5694/j.1326-5377.1990.tb125162.x.

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Swift, P. G. F. "Clinical Paediatric Endocrinology." Archives of Disease in Childhood 64, no. 10 (October 1, 1989): 1520. http://dx.doi.org/10.1136/adc.64.10.1520.

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Dickey, MD, FACP, FACE, Richard A. "SAVING CLINICAL ENDOCRINOLOGY." Endocrine Practice 7, no. 6 (November 2001): 474–78. http://dx.doi.org/10.4158/ep.7.6.474.

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Rosenbloom, Arlan L. "Clinical pediatric endocrinology." Trends in Endocrinology & Metabolism 4, no. 5 (July 1993): 175–77. http://dx.doi.org/10.1016/1043-2760(93)90111-q.

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Dissertations / Theses on the topic "Clinical endocrinology"

1

El, Sanadi Caroline Elizabeth. "DEVELOPMENT AND VALIDATION OF CLINICAL PREDICTION TOOLS FOR AIDING IN SELECTION OF 2ND LINE THERAPIES ADDED TO METFORMIN IN TREATMENT OF TYPE 2 DIABETES." Case Western Reserve University School of Graduate Studies / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1607604907339227.

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Martins, Dinorah Fernandes Gioia. "Obesidade: Estudo das Representações Sociais de Endocrinologistas em Hospital Público." Universidade de São Paulo, 1998. http://www.teses.usp.br/teses/disponiveis/47/47133/tde-30112012-113617/.

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O presente trabalho teve como objetivo estudar a psicodinâmica das Representações Sociais (RS) de endocrinologistas de hospital público sobre obesidade, identificando-as e buscando detectar seu inconsciente relativo, ou seja, a lógica emocional segundo a qual se estruturam. Foram realizadas 10 (dez) entrevistas com médicos endocrinologistas de rede pública, (05 do sexo feminino e 05 do sexo masculino) com idade variável de 28 a 44 anos de idade. O tempo de especialização variou de dois a dezoito anos. As entrevistas foram semi-estruturadas, no sentido de haver uma pré-estrutura mínima, permitindo ao entrevistado espontaneidade e fluência de expressão. Usou-se técnicas encobertas, com perguntas gerais e abrangentes. Desejou-se que o tema - obesidade - surgisse espontaneamente. O tratamento dos dados foi de acordo com o referencial psicodinâmico, numa abordagem qualitativa. Conclui-se que o médico é o intérprete das ideologias socialmente circulantes a respeito da obesidade. Suas condutas são pautadas pelas características de personalidade, pelas informações científicas, e pela influência midiática
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Preda, Veronica Angela. "Clinical & Genetic Characteristics of Sellar Masses Including Pituitary Adenomas and Craniopharyngiomas." Thesis, The University of Sydney, 2016. http://hdl.handle.net/2123/15701.

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There are numerous tumours arising in the sellar region. This thesis focuses on two distinct entities, pituitary adenomas and craniopharyngiomas. The biology of both these tumours is incompletely understood. The studies performed in this thesis aim to identify specific molecular genetic lesions in pituitary adenomas and craniopharyngiomas. More broadly it explores tumorigenesis pathways in general to correlate these with the behaviour of pituitary adenomas and craniopharyngiomas. Important genetic mutations have been described in pituitary adenomas. These discoveries have resulted in interest focusing on the mechanism of tumorigenesis based on the adenoma subtype formation and the prediction of the clinical behaviour. This thesis focuses on a number of genetic mutations, which can have significant clinical consequence, including AIP gene mutations and MEN-1. We also have yet to fully understand, through exploring with the assistance of new techniques of genome-wide analysis, the genetic mutations responsible in distinct clinical cases, such as twins of consanginous parents, with pituitary adenomas and puerperal alactogenesis. Craniopharyngiomas are rare sellar masses, of two distinct subtypes, which are at times difficult to clinically and histopathologically delineate. Herein we characterise craniopharyngiomas more completely in order to understand and describe their distinct features of the papillary and adamantinomatous variants. In addition this thesis uses craniopharyngiomas as a platform to explore common tumour pathways to understand their formation but also to contribute to our knowledge across neoplasia subtypes. This thesis has already contributed to the medical and scientific literature with a number of new discoveries, broadly in three main areas. Firstly, in pituitary adenomas it has clinically characterised one of the largest single centre cohort of adult sporadic adenomas occurring at a young age of < 40 years and specifically looked at their AIP mutation rates. This has bench-to-bedside implications to suggest screening of young patients with sporadic pituitary adenomas, particularly of the growth hormone or prolactin secreting variants and the importance of long-term follow-up rather than discharge from clinic. In the specific clinical setting of suspected MEN-1 syndrome, this thesis presents a malignant peripheral nerve sheath tumour (MPNST) not previously described in association with this cancer syndrome. We use our experience to describe assessment and monitoring of this tumour. This case suggests that clinical investigation is warranted in all other MEN-1 cases with consideration for a possible association with MPNST. Secondly, in craniopharyngiomas this thesis carefully characterises how to immunohistochemically and genetically distinguish the adamantinomatous (aCP) and papillary (pCP) subtypes. This thesis characterises the other interacting partners of the adherens junction, which previously have not been published in the literature. No disruption of the β-catenin binding partners were found; suggesting that loss of junction integrity is not associated with characteristic β-catenin translocation, as had previously been suggested in other masses of the sellar region and specifically in pituitary adenomas. We describe distinct tumour cascades responsible for contributing to tumour growth, the Wnt pathway via β-catenin in aCPs and the MAPKinase pathway via BRAF in pCP. However, there appears to be some overlap in distinct case examples. This could be explained by our suggestion of an evolutionary continuum of these tumours, consistent with their ectodermal derivation, explaining the novel finding of CTNNB1 mutations in a sub-cohort of pCPs. This contribution to tumorigenesis and thesis findings will potentially enable targeted pharmacotherapies to β-catenin and BRAF, the latter used in treating other endocrine malignancies such as thyroid cancer. Thirdly, this thesis sets the foundation for considering the role of telomerase and transcriptional regulators of stem cell-ness, such as KLF-4 in sellar masses. It has contributed to elucidating craniopharyngioma tumorigenesis by describing novel mutations in the TERT promoter region as well as novel mutations in their binding partner sites, such as KLF-4, which appears to play an integral role in craniopharyngioma tumorigenesis.

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Claes, Anthony N. J. "ANTI-MÜLLERIAN HORMONE IN STALLIONS AND MARES: PHYSIOLOGICAL VARIATIONS, CLINICAL APPLICATIONS, AND MOLECULAR ASPECTS." UKnowledge, 2014. http://uknowledge.uky.edu/gluck_etds/18.

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Anti-Müllerian hormone (AMH) is a homodimeric glycoprotein that is best known for its role in regression of the Müllerian duct in the male fetus. Accumulating evidence indicates that AMH also has an important role during different physiological processes after birth. In contrast to other species, relatively little is known about AMH in the horse. In chapter one, developmental and seasonal changes in serum AMH concentrations in male horses were determined, and the use of AMH for determination of retained cryptorchid testes was established. In chapter two, the interrelationship between plasma AMH concentrations, antral follicle counts (AFC), and age in mares was evaluated. Molecular and hormonal changes in the equine follicle with regard to variations in antral follicle count and follicular development were examined in chapter three. In chapter four, the effect of AFC on age-related changes in follicular parameters in light-type horse mares was examined. Peripheral AMH concentrations were significantly higher in prepubertal colts than in postpubertal stallions and varied with season in mature stallions with higher concentrations during the physiological breeding season. Furthermore, serum AMH concentrations were significantly higher in cryptorchid stallions compared to intact stallions or geldings. Circulating AMH concentrations varied widely amongst mares of the same age while the repeatability of AMH was high within and between estrous cycles. More importantly, AMH concentrations were positively associated with AFC, and this relationship increased with mare age. In addition, variations in AMH concentrations or AFC were associated with molecular differences in granulosa cells of growing follicles, and the expression of AMH and genes co-expressed with AMH in the equine follicle as well as intrafollicular AMH concentrations decreased during follicular development. Finally, the inter-ovulatory interval and length of the follicular phase is increased in aged mares with low AFC. In conclusion, AMH is a useful biomarker for cryptorchidism in stallions and ovarian reserve in mares. Furthermore, follicular function was interrelated to AFC or AMH based upon molecular differences in growing follicles, while age-related changes in follicular parameters are linked to differences in AFC.

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Alimohammadi, Mohammad. "Molecular Targets in Autoimmune Polyendocrine Syndrome Type1 and Their Clinical Implications." Doctoral thesis, Uppsala universitet, Institutionen för medicinska vetenskaper, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-9549.

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Autoimmune diseases occur when the immune system attacks and destroys healthy body tissue. Autoimmunity is known to cause a wide range of disorders, and is suspected to be responsible for many more. Most autoimmune disorders are chronic and cause severe morbidity for the patients, and are also costly for society. A majority of these disorders are today considered as complex diseases with incompletely known etiology. Hence, model systems for studying the pathogenesis of autoimmunity are important to unravel its causes. Autoimmune Polyendocrine Syndrome Type 1 (APS-1), (OMIM 240300), is a rare autoimmune disorder. Patients with APS-1 progressively develop multiple organ-specific autoimmune lesions involving both endocrine and non endocrine tissues. Typical autoimmune disease components in APS-1 are hypoparathyroidism, Addison’s disease, vitiligo, alopecia and type 1 diabetes. The gene preventing APS-1 has been identified and designated Autoimmune Regulator (AIRE). It has been shown that mutations of AIRE cause loss of tolerance to self-structures, resulting in organ-specific autoimmunity. Although APS-1 is a rare syndrome occurring mainly in genetically isolated populations, the disease components of APS-1 are, in isolated forms, not unusual in the general population and affect many patients. Hence, APS-1 is an attractive model disease for studies of molecular mechanisms underlying organ-specific autoimmunity. This thesis concerns investigations in which two novel autoantigens are identified in APS-1 and used in serological diagnosis of the disease. NALP5, is identified as a parathyroid autoantigen - an important finding since autoimmune hypoparathyroidism is one of the cardinal symptoms of APS-1. Additionally, KCNRG is identified as a bronchial autoantigen in APS-1 patients with respiratory symptoms. Finally, studies that compare the immune response in APS-1 patients and the mouse model for APS-1 are presented.

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Mughal, Saima Amin. "The clinical and genetic characterisation of young-onset diabetes." Thesis, University of Oxford, 2014. http://ora.ox.ac.uk/objects/uuid:ba1ddd3f-a96c-4cc5-9298-24aeee4efda1.

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Maturity-onset diabetes of the young (MODY), due to hepatocyte nuclear factor 1 alpha mutations (HNF1A-MODY), is the most common form of monogenic diabetes presenting in young adults. An accurate genetic diagnosis of HNF1A-MODY has therapeutic implications for the patients and their family members. However, the majority of people with HNF1A-MODY are not referred for genetic testing and remain misdiagnosed as type 1 or type 2 diabetes. As part of measures to address this misdiagnosis, over the last few years there have been efforts to define clinical features and biomarkers that can be used to identify those at high risk of HNF1A-MODY. Secreted hepatic proteins regulated by HNF1A are attractive candidates for diagnostic biomarkers that would be specific for this form of diabetes. Apolipoprotein M (apoM), C-reactive protein (CRP) and plasma glycan profile have all been investigated as biomarkers to improve selection of suspected MODY cases for genetic testing. In my thesis, I have addressed questions about the variation in apoM between different forms of diabetes and assessed the performance of hsCRP and plasma glycan profile to identify HNF1A-MODY in previously uninvestigated individuals with young-onset diabetes and in a non-European population. Additionally because CRP and plasma glycans are both important components of an acute inflammatory response, I examined the effect of haploinsufficiency of HNF1A in a standardised model of inflammation. When investigating apoM, I showed that serum apoM levels are lower in HNF1A-MODY than controls, and have demonstrated for the first time that serum apoM provides good discrimination between HNF1A-MODY and type 1 diabetes. CRP and plasma glycan profile both performed well in identifying HNF1A-MODY cases in unselected young adults with diabetes. The results also suggested that both biomarkers have value for assessing the functional impact of novel HNF1A variants. I went on to examine the use of a low CRP for selecting those at risk of HNF1A-MODY in South Asian subjects with young-onset diabetes. This study suggests that the overall population prevalence of HNF1A-MODY is similar in South Asians to Europeans, but that MODY represents a lower proportion of those with diabetes (due to the higher prevalence of type 2 diabetes in South Asians). The specific selection strategy employed in this study was not successful in identifying subjects at high risk of HNF1A-MODY (only 3% of those sequenced had mutations), suggesting that additional clinical and biochemical features will be required in addition to CRP to distinguish South Asians at high risk of HNF1A-MODY. Lastly, using endotoxaemia as a standardised model of acute inflammation for the first time in HNF1A-MODY, I have shown that despite low baseline levels, subjects with HNF1A-MODY had peak stimulated CRP levels comparable to non-diabetic controls. An attenuated cytokine response was observed in HNF1A-MODY, which requires further investigation. This is also the first report of inflammation-associated changes in plasma and white cell membrane glycan profile in diabetes. This research work adds substantially to current understanding of performance of HNF1A-MODY biomarkers, a critical step before their clinical translation. The work presented also provides novel insights into the regulation of the acute inflammatory response in HNF1A-MODY.

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Ramsumeer, Soy. "A Plan for the Implementation and Evaluation of Diet Education in Type 2 Diabetes." ScholarWorks, 2016. https://scholarworks.waldenu.edu/dissertations/1920.

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Abstract Type 2 Diabetes Mellitus (T2DM) is the seventh leading cause of death in the United States with a projected increase of 552 million people worldwide who will be affected with this illness by 2030. The need to address this issue is vital to prevent complications and reduce healthcare spending. The DNP project is aimed at planning and designing a nutritional education program tailored toward specific ethnic groups in order to increase knowledge in making healthy food choices. This project is intended to educate Registered Nurses (RNs) on nutrition so that they can offer dietary knowledge to T2DM patients. Additional patients can be reached by educating the RNs rather than patients being limited to consultations with a Certified Diabetes Educator or Registered Dietician. This project focused on whether healthy nutrition tailored toward the individual's own ethnic foods helps to stabilize glycemic values for patients with Type 2 diabetes. A toolkit was utilized to aid with the RNs' learning on healthy nutrition and its impact on the management of blood glucose. It addressed areas such as food groups and calories, grocery shopping, preparation methods, and portion control. The framework for design utilized the basic concepts associated with the systems theory with an intended goal to prevent further complications and improve patients' glycemic value through consuming nutritious foods. The logic model will be used to evaluate the impact of healthy nutrition on blood glucose through pre- and post-program tests of the RNs' nutritional knowledge on healthy eating. The continuation of this program will promote positive social change by helping patients to achieve a healthier lifestyle and reduce healthcare expenditures.

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Marsh, Wendy K. "Clinical Course of Bipolar Disorder During the Menopausal Transition: Comparison with Reproductive Age and Post Menopausal Women: A Master's Thesis." eScholarship@UMMS, 2010. https://escholarship.umassmed.edu/gsbs_diss/517.

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Introduction: The late menopausal transition is a time of increased risk of depression in the general population. Nonetheless, mood course during the late menopausal transition in women with bipolar disorder in relatively unknown. Methods: Mood state data in 519 reproductive age women (5989 clinic visits), 116 late menopausal transition (perimenopausal) women (2046 visits), and 133 postmenopausal women (1,437 visits) with bipolar disorder who were receiving optimized naturalistic treatment in the multisite STEP-BD study over an average of 19.8±15.5 months were analyzed for proportion of clinic visits with syndromal depression, mood elevation and euthymia between the three groups. History of postpartum and perimenstrual mood exacerbation as well as hormone therapy use were evaluated as potential predictors of mood. Results: No significant difference in the proportion of clinic visits with syndromal depression was found between reproductive age (18.1%), perimenopausal (18.1%) and postmenopausal (19.3%) women. Reproductive age women had significantly greater proportion of visits with syndromal mood elevation (5.3%) compared to perimenopausal (4.1%, Z=2.1, p2(3, N = 9960) = 19.8, p Conclusions: While proportion of clinic visits with syndromal depression did not differ among the three reproductive groups, thirteen women who had recorded transition from perimenopause to postmenopause showed significantly greater depression than reproductive age, perimenopausal or postmenopausal women. Proportion of visits with euthymia or with syndromal mood elevation decreased from reproductive age to perimenopausal to postmenopausal women. Reported history of mood exacerbation during times of hormonal fluctuation, or current use of hormone therapy, was not significantly associated with depression during the perimenopause. Limitations include women excluded due to absence of menstrual data. Future studies should include hormonal assessments.

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Beer, Nicola L. "The role of genetic variation in glucokinase and glucokinase regulatory protein in diabetes and related traits." Thesis, University of Oxford, 2011. http://ora.ox.ac.uk/objects/uuid:87f8ea0d-9528-49fd-8f01-5f976cf9f210.

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The rising prevalence of type 2 diabetes (T2D) is a global problem, and suggests that we need better therapeutic strategies against this disease. The glycolytic enzyme glucokinase (GCK) catalyses the phosphorylation of glucose, and is a well-established T2D drug target. Rare GCK mutations cause monogenic beta-cell dysfunction, whilst common genetic variants within GCK are associated with fasting plasma glucose (FPG) levels and T2D risk. Since GCK is expressed in both the pancreas and liver, pharmacological GCK activation provides the promise of a two-pronged attack on hyperglycaemia. In vivo, GCK activity is modulated by the hepatic inhibitor glucokinase regulatory protein (GKRP, gene GCKR). GKRP negatively regulates GCK activity competitively with respect to glucose, and is controlled by fructose 6- and fructose 1-phosphate (F6P and F1P), which compete with each other for binding and enhance or diminish GCK inhibition respectively. GKRP also sequesters GCK in the nucleus and paradoxically stabilises the enzyme. As GCK and its regulatory protein are fundamental to glucose homeostasis, we aimed to investigate the role of genetic variation in both GCK and GCKR to further our understanding of these important T2D drug targets in a system that would be relevant to man. I demonstrated that two novel GCK mutations (T103S and V389L) identified in patients with hyperinsulinaemic hypoglycaemia were kinetically activating and through structural modelling identified a novel regulatory site for GCK activation by small molecular activators. Genome-wide association studies (GWAS) identified GCKR as a regulator of FPG and triglyceride levels, and showed a role for GKRP in T2D risk. Unlike most GWAS hits, this signal included a non-synonymous variant within GCKR (P446L), thus facilitating functional studies. P446L-GKRP was characterised kinetically and at the cellular sequestration-level. This variant showed diminished F6P-mediated modulation, which was proposed to reduce hepatic GCK inhibition, increase glycolytic flux (decreasing FPG), and feed metabolites into liver pathways (elevating triglycerides). As GCKR was not expressed at functional levels within human islets, this phenotype was thought to be driven by the liver. Preliminary analysis at the cellular level was inconclusive, with optimisation required to study human P446L-GKRP in this cellular system. Finally, I showed that mutations within GCKR are not a common cause of “GCK-Like” phenotypes in man, despite the regulatory protein directly modulating GCK activity. These data provide further insight as to the pathogenic consequences of perturbing GCK activity. This must be considered if this enzyme is to be the subject of therapeutic intervention in T2D.

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Osama, Mohammad. "Function of Vascular Endothelial Cells in Aging and Hypothermia: Clinical Implications." Ohio University / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1534939514503588.

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Books on the topic "Clinical endocrinology"

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Michael, Conn P., and Melmed Shlomo, eds. Endocrinology: Basic and clinical principles. Totowa, N.J: Humana Press, 1997.

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Labhart, Alexis. Clinical Endocrinology. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-70509-0.

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Brazilian Congress of Endocrinology and Metabolism (18th 1988 Rio de Janeiro, Brazil). Clinical endocrinology. Edited by Meirelles Ricardo M. R, Machado Álvaro, Póvoa Luiz César, and Brazilian Society of Endocrinology and Metabolism. Amsterdam: Excerpta Medica, 1988.

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Ashley, Grossman, ed. Clinical endocrinology. Oxford: Blackwell Scientific Publications, 1992.

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1938-, Kohler Peter O., and Jordan Richard M, eds. Clinical endocrinology. New York: Wiley, 1986.

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Ashley, Grossman, ed. Clinical endocrinology. 2nd ed. Osney Mead, Oxford: Blackwell Science, 1998.

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Brook, Charles G. D., Peter E. Clayton, Rosalind S. Brown, and Martin O. Savage, eds. Clinical Pediatric Endocrinology. Oxford, UK: Blackwell Publishing Ltd, 2005. http://dx.doi.org/10.1002/9780470987117.

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Kavoussi, Parviz K., Raymond A. Costabile, and Andrea Salonia, eds. Clinical Urologic Endocrinology. London: Springer London, 2013. http://dx.doi.org/10.1007/978-1-4471-4405-2.

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Igaz, Peter, ed. Practical Clinical Endocrinology. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-62011-0.

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Brown, Rosalind S., C. G. D. Brook, and Peter E. Clayton. Clinical pediatric endocrinology. 5th ed. Malden, Mass: Blackwell Pub., 2005.

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Book chapters on the topic "Clinical endocrinology"

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Johnston, Alison, and Jacqueline Lowdon. "Endocrinology." In Clinical Paediatric Dietetics, 195–221. Chichester, UK: John Wiley & Sons, Ltd, 2014. http://dx.doi.org/10.1002/9781118915349.ch10.

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Centello, Roberta, Antongiulio Faggiano, and Elisa Giannetta. "MEN2 in Clinical Practice." In Endocrinology, 413–20. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-319-89497-3_21.

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Centello, Roberta, Antongiulio Faggiano, and Elisa Giannetta. "MEN2 in Clinical Practice." In Endocrinology, 1–8. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-319-73082-0_21-1.

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Ranuccio, Nuti. "Bone Remodeling – Clinical Evaluation." In Endocrinology, 1–21. Cham: Springer Nature Switzerland, 2023. http://dx.doi.org/10.1007/978-3-030-19632-5_6-1.

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Hill-Briggs, Felicia, Stephanie L. Fitzpatrick, Kristina P. Schumann, and Sherita Hill Golden. "Endocrinology." In Handbook of Clinical Psychology in Medical Settings, 417–58. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-0-387-09817-3_17.

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Zapf, J., A. Jakob, and C. Meuli. "General Aspects of Endocrinology." In Clinical Endocrinology, 1–23. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-70509-0_1.

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Migliaccio, Silvia, and Andrea Lenzi. "Osteoporosis: Etiology and Clinical Evaluation." In Endocrinology, 1–8. Cham: Springer Nature Switzerland, 2023. http://dx.doi.org/10.1007/978-3-030-19632-5_9-1.

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Ceccato, Filippo, and Gianluca Occhi. "Carney Complex in Clinical Practice." In Endocrinology, 387–94. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-319-89497-3_18.

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Ceccato, Filippo, and Gianluca Occhi. "Carney Complex in Clinical Practice." In Endocrinology, 1–8. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-319-73082-0_18-1.

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Malik, Mohammed Faysal, Asiya Maula, Dominic Greenyer, and Maurice A. Smith. "Endocrinology." In The Ultimate Guide to Passing Clinical Medicine Finals, 99–124. London: CRC Press, 2021. http://dx.doi.org/10.1201/9781846198526-5.

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Conference papers on the topic "Clinical endocrinology"

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"20th ESE Postgraduate Training Course on Endocrinology Diabetes and Metabolism: Abstracts and clinical cases." In 20th ESE Postgraduate Training Course on Endocrinology Diabetes and Metabolism. Media Sphera, 2017. http://dx.doi.org/10.14341/probl2017631s1-40.

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Breuker, C., F. Clement, Y. Audurier, P. Renaudin, C. Boegner, A. Jalabert, M. Villiet, A. Castet-Nicolas, A. Avignon, and A. Sultan. "CP-190 Impact of the deployment of a clinical pharmacy team in endocrinology–nutrition unit." In 22nd EAHP Congress 22–24 March 2017 Cannes, France. British Medical Journal Publishing Group, 2017. http://dx.doi.org/10.1136/ejhpharm-2017-000640.188.

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Martinez-Hervas, Sergio, Dolores Ortiz Masia, José F. Martinez-Valls, Juan Francisco Merino Torres, Carlos Sanchez Juan, and José T. Real. "GUIDED CLINICAL CASE AS A METHODOLOGY FOR LEARNING: EXPERIENCE IN THE SUBJECT OF ENDOCRINOLOGY AND NUTRITION IN THE MEDICAL DEGREE." In 14th International Conference on Education and New Learning Technologies. IATED, 2022. http://dx.doi.org/10.21125/edulearn.2022.0323.

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McCallion, S., S. Smith, H. Kyle, J. McQuarrie, G. Wilkinson, and A. Kyriakou. "G431(P) Gender dysphoria – a description of the changes in prevalence, demographics and the clinical care provided by a paediatric endocrinology department." In Royal College of Paediatrics and Child Health, Abstracts of the RCPCH Conference–Online, 25 September 2020–13 November 2020. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2020. http://dx.doi.org/10.1136/archdischild-2020-rcpch.372.

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Chirita, Anca Livia, Mihaela Popescu, Veronica Calborean, Victor Gheorman, and Ion Udristoiu. "PSYCHIATRIC DISORDERS ASSOCIATED WITH ENDOCRINE DYSFUNCTIONS." In The European Conference of Psychiatry and Mental Health "Galatia". Archiv Euromedica, 2023. http://dx.doi.org/10.35630/2022/12/psy.ro.25.

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Introduction: Psychiatric disorders occurring during endocrine dysfunction and, conversely, endocrine dysfunctions associated with mental disorders were the emergence of a new discipline, psychoendocrinology. Psychiatric disorders correlated with endocrine diseases are defined as psychopathological manifestations of variable intensity and clinical symptomatology, determined by complex psycho-neuro-endocrinological interrelationships. Defining elements consist of the association between diagnosis of mental disorders and specific symptoms for endocrine dysfunction. Methods: We conducted a prospective one-year study (January 2018 - December 2018 on 112 patients hospitalized in the Clinic of Psychiatry who also had an endocrinological comorbidity. We investigated the frequency and severity of psychoendocrinological associations by studying a number of demographic and clinical items. Results: The results showed that the highest incidence belongs to thyroid disorder - 55.36%, followed by gonadal disorders - 24.11%, and, rarely, pituitary diseases and diabetes. Hyperthyroidism was associated most frequently with manic episodes, while unipolar depression prevailed in patients with hypothyroidism. In gonadal disorders, present in majority in female patients (secondary amenorrhea, menopause or erectile dysfunction in males), depression accompanied by anxiety, often severe in intensity, was the most frequent psychiatric diagnosis. Psychotic disorders were met in a smaller number of cases, especially in patients with long history of endocrine disorders and instability of biological constants. Conclusions: We may state that affective disorders are the most frequent nosologically category in patients with endocrine dysfunctions. It requires a better collaboration between specialists in endocrinology and psychiatry, to highlight the determinants which contribute to the development of psychopathological manifestations in endocrine diseases and to individualize the treatment depending on cases’ particularities.

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Martinez-Hervas, Sergio, Dolores Ortiz Masiá, Herminia Gonzalez-Navarro, F. Javier Ampudia-Blasco, J. Francisco Merino Torres, Carlos Sánchez Juan, and José T. Real. "USEFULNESS OF THE GUIDED CLINICAL CASE TO IMPROVE STUDENT MOTIVATION AND LEARNING IN THE SUBJECT OF ENDOCRINOLOGY AND NUTRITION IN THE MEDICAL DEGREE. TWO YEARS EXPERIENCE." In 15th International Conference on Education and New Learning Technologies. IATED, 2023. http://dx.doi.org/10.21125/edulearn.2023.1192.

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Souza, Isabela Silva, Beatriz Cassarotti, Lucas de Oliveira Pinto Bertoldi, Alana Strucker Barbosa, Eduardo Silveira Marques Branco, Isabela Badan Fernandes, Bruno Eji Nakano, et al. "Fahr syndrome associated with post-thyroidectomy hypoparathyroidism." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.567.

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Context: Fahr’s syndrome is a rare disorder characterized by bilateral and symmetrical abnormal calcifications in basal ganglia and cerebral cortex. Those calcified deposits are due to changes in calcium and phosphorus metabolisms that can be caused by endocrine disorders, mitochondrial myopathies, dermatological and infectious diseases. Clinical manifestations may include a variety of extrapyramidal, cerebelar and neuropsychiatric syndromes. Case report: This study describes a 75-year-old female patient that underwent total thyroidectomy in 1985 due to a multinodular goiter and presented postsurgical hypoparathyroidism. The patient missed follow-up apppoointments with Endocrinology and stopped treating her parathyroid condition. Some time later, she presented with change in behavior, drowsiness, paraesthesias, limb spasms and seizures. A CT scan of the brain was performed, showing multiple and extensive calcifications reaching the cerebellar hemispheres, basal ganglia, thalamus and white subcortical substance symmetrically. Laboratory examinations revealed hypocalcemia, hyperphosphatemia, and low parathyroid hormone (PTH) levels. Intravenous calcium gluconate was used to corret the Ca/P dysfunction. Additionally, appropriate antiepileptic drugs for seizures were used. She presented with progressive improvement of symptoms after treatment. Conclusions: This case report demonstrates the importance of post- thyroidectomy follow-up and early recognition of Fahr syndrome’s symptoms, which prevents the progression of neurological conditions.

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Yafi, Michael, Belinda Perzan, Michelle Rivera-Davila, Katherine Velez, and Robert Yetman. "200 Telemedicine as a method of providing physician coverage A pilot experience in a pediatric endocrinology clinic." In 10th Europaediatrics Congress, Zagreb, Croatia, 7–9 October 2021. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2021. http://dx.doi.org/10.1136/archdischild-2021-europaediatrics.200.

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Mahmoud, Lubna Abdalla, Khobieb Mohammed, Fadwa Mohammed Saad, and Mohamed Ahmed Abdullah. "408 Short stature: problem severity and etiological characteristics in patients presented to two pediatric endocrinology clinics in Khartoum, Sudan." In Royal College of Paediatrics and Child Health, Abstracts of the RCPCH Conference, Liverpool, 28–30 June 2022. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2022. http://dx.doi.org/10.1136/archdischild-2022-rcpch.436.

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Burks, Jessica, Usha Kollipara, Jacqueline Mutz, Jason Fish, Estefania Andrioaia Lopez, Jessica Abramowitz, Nancy Drobycki, and Sadia Ali. "18 Utilizing lean six sigma and quality improvement (QI) to increase diabetes self-management education referrals within the endocrinology clinic." In IHI Scientific Symposium. British Medical Journal Publishing Group, 2020. http://dx.doi.org/10.1136/bmjoq-2020-ihi.18.

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Reports on the topic "Clinical endocrinology"

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Surendra G, Dr Prasad, Dr Bhuyan Ashok K, Dr Baro Abhamon, Dr Saikia Uma K, and Dr Kumar Angad. CLINICAL AND METABOLIC CHARACTERISTICS OF PRIMARY HYPERPARATHYROIDISM IN DIFFERENT AGE GROUPS- A TERTIARY CENTRE EXPERIENCE. World Wide Journals, February 2023. http://dx.doi.org/10.36106/ijar/6005490.

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Background and Objectives- Symptomatic Primary Hyperparathyroidism (PHPT) is common in India in comparison to the western population. But there is very little data on the inuence of age on the presentation of PHPT. In the present study we aimed to analyse the clinical and metabolic prole among different age groups of symptomatic primary hyperparathyroidism. Methods: This retrospective analysis was done in PHPT patients who attended Department of Endocrinology, Gauhati Medical college and Hospital. Thirty-one PHPT subjects who presented to us over a period of last ve years were divided into three different age groups i.e, children and adolescents <18yrs, adults ≥18-50 years, and older group >50years. All major clinical, metabolic and imaging parameters were compared among these groups. Appropriate statistical methods were used to compare different variables. The age distribution ranged from 13 to Results: 72 years with mean age of 38.6±16.3years and with equal female to male ratio. Bony deformity (Rickets) as initial manifestation was seen in three adolescents and bone pain was common in adolescents(p=0.05). Prevalence of renal stones were higher in adult group(p=0.002), gastrointestinal manifestations were higher in older group (p=0.02). There was no signicant difference in fracture rate(P=0.17), brown tumours(P=0.56) and other symptoms among different age groups. Alkaline phosphatase(p=0.006) and iPTH(p=0.01) were signicantly higher in adolescent group. There was no signicant difference in serum calcium, phosphate, 25(OH)Vitamin-D3 and haemoglobin levels among different age groups. Age has substantial inuence on PHPT presentation. Bone Interpretation & Conclusion: pain and deformity was common in adolescents, while renal stones and gastrointestinal manifestations were common in middle aged and elderly group respectively

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Totten, Annette, Dana M. Womack, Marian S. McDonagh, Cynthia Davis-O’Reilly, Jessica C. Griffin, Ian Blazina, Sara Grusing, and Nancy Elder. Improving Rural Health Through Telehealth-Guided Provider-to-Provider Communication. Agency for Healthcare Research and Quality, December 2022. http://dx.doi.org/10.23970/ahrqepccer254.

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Objectives. To assess the use, effectiveness, and implementation of telehealth-supported provider-to-provider communication and collaboration for the provision of healthcare services to rural populations and to inform a scientific workshop convened by the National Institutes of Health Office of Disease Prevention on October 12–14, 2021. Data sources. We conducted a comprehensive literature search of Ovid MEDLINE®, CINAHL®, Embase®, and Cochrane CENTRAL. We searched for articles published from January 1, 2015, to October 12, 2021, to identify data on use of rural provider-to-provider telehealth (Key Question 1) and the same databases for articles published January 1, 2010, to October 12, 2021, for studies of effectiveness and implementation (Key Questions 2 and 3) and to identify methodological weaknesses in the research (Key Question 4). Additional sources were identified through reference lists, stakeholder suggestions, and responses to a Federal Register notice. Review methods. Our methods followed the Agency for Healthcare Research and Quality Methods Guide (available at https://effectivehealthcare.ahrq.gov/topics/cer-methods-guide/overview) and the PRISMA reporting guidelines. We used predefined criteria and dual review of abstracts and full-text articles to identify research results on (1) regional or national use, (2) effectiveness, (3) barriers and facilitators to implementation, and (4) methodological weakness in studies of provider-to-provider telehealth for rural populations. We assessed the risk of bias of the effectiveness studies using criteria specific to the different study designs and evaluated strength of evidence (SOE) for studies of similar telehealth interventions with similar outcomes. We categorized barriers and facilitators to implementation using the Consolidated Framework for Implementation Research (CFIR) and summarized methodological weaknesses of studies. Results. We included 166 studies reported in 179 publications. Studies on the degree of uptake of provider-to-provider telehealth were limited to specific clinical uses (pharmacy, psychiatry, emergency care, and stroke management) in seven studies using national or regional surveys and claims data. They reported variability across States and regions, but increasing uptake over time. Ninety-seven studies (20 trials and 77 observational studies) evaluated the effectiveness of provider-to-provider telehealth in rural settings, finding that there may be similar rates of transfers and lengths of stay with telehealth for inpatient consultations; similar mortality rates for remote intensive care unit care; similar clinical outcomes and transfer rates for neonates; improvements in medication adherence and treatment response in outpatient care for depression; improvements in some clinical monitoring measures for diabetes with endocrinology or pharmacy outpatient consultations; similar mortality or time to treatment when used to support emergency assessment and management of stroke, heart attack, or chest pain at rural hospitals; and similar rates of appropriate versus inappropriate transfers of critical care and trauma patients with specialist telehealth consultations for rural emergency departments (SOE: low). Studies of telehealth for education and mentoring of rural healthcare providers may result in intended changes in provider behavior and increases in provider knowledge, confidence, and self-efficacy (SOE: low). Patient outcomes were not frequently reported for telehealth provider education, but two studies reported improvement (SOE: low). Evidence for telehealth interventions for other clinical uses and outcomes was insufficient. We identified 67 program evaluations and qualitative studies that identified barriers and facilitators to rural provider-to-provider telehealth. Success was linked to well-functioning technology; sufficient resources, including time, staff, leadership, and equipment; and adequate payment or reimbursement. Some considerations may be unique to implementation of provider-to-provider telehealth in rural areas. These include the need for consultants to better understand the rural context; regional initiatives that pool resources among rural organizations that may not be able to support telehealth individually; and programs that can support care for infrequent as well as frequent clinical situations in rural practices. An assessment of methodological weaknesses found that studies were limited by less rigorous study designs, small sample sizes, and lack of analyses that address risks for bias. A key weakness was that studies did not assess or attempt to adjust for the risk that temporal changes may impact the results in studies that compared outcomes before and after telehealth implementation. Conclusions. While the evidence base is limited, what is available suggests that telehealth supporting provider-to-provider communications and collaboration may be beneficial. Telehealth studies report better patient outcomes in some clinical scenarios (e.g., outpatient care for depression or diabetes, education/mentoring) where telehealth interventions increase access to expertise and high-quality care. In other applications (e.g., inpatient care, emergency care), telehealth results in patient outcomes that are similar to usual care, which may be interpreted as a benefit when the purpose of telehealth is to make equivalent services available locally to rural residents. Most barriers to implementation are common to practice change efforts. Methodological weaknesses stem from weaker study designs, such as before-after studies, and small numbers of participants. The rapid increase in the use of telehealth in response to the Coronavirus disease 2019 (COVID-19) pandemic is likely to produce more data and offer opportunities for more rigorous studies.

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