Relevant bibliographies by topics / Clinico-Pathological Correlation

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Clinico-Pathological Correlation'

Author: Grafiati
Published: 5 June 2025
Last updated: 25 June 2025

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Journal articles on the topic "Clinico-Pathological Correlation"

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Tonape, Dr Trupti. "Clinico-Pathological Correlation of Thyroid Swellings." Journal of Medical Science And clinical Research 04, no. 12 (2016): 14382–85. http://dx.doi.org/10.18535/jmscr/v4i12.18.

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Handa, Sanjeev, AmrinderJ Kanwar, BishanDass Radotra, RanjanaW Minz, and Suruchi Gupta. "Cutaneous vasculitides: Clinico-pathological correlation." Indian Journal of Dermatology, Venereology and Leprology 75, no. 4 (2009): 356. http://dx.doi.org/10.4103/0378-6323.53130.

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Md, Dr S. Vasuki, Dr K. Suresh Durai Md, and Athira Sai. "Clinico Pathological Correlation in Pleural Effusion." IOSR Journal of Dental and Medical Sciences 15, no. 07 (2016): 45–47. http://dx.doi.org/10.9790/0853-150734547.

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Ramole, Yashpal, and Vijay Tekam. "Clinico-pathological correlation of abdominal lymphadenopathy." International Surgery Journal 5, no. 11 (2018): 3578. http://dx.doi.org/10.18203/2349-2902.isj20184625.

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Background: This study “clinico-pathological correlation of abdominal lymphadenopathy" intended to know the various etiological causes behind the abdominal lymphadenopathy and different modes of presentation which can help in treatment and prognosis in our setup which helps in better management of these cases thus helping to improve prognosis.Methods: The" Clinico-Pathological Correlation of Abdominal Lymphadenopathy is a clinical study of 250 consecutive cases of abdominal Lymphadenopathy which were found during intraoperative laparotomy in Hamidia Hospital (Gandhi Medical College), Bhopal. This is observational study will be carried out in the Department of Surgery, Gandhi Medical College. Total 250 consecutive cases will be included in this study having per-operative findings of lymphadenopathy, where laparotomy will be done for various regions. Prior to surgery thorough history will be taken and meticulous physical examination will be performed. Necessary laboratory and imaging studies shall do to establish the diagnosis.Results: Tuberculosis is one of the common causes of Abdomen lymphadenopathy. Age incidence more in males and in second and third decade of life is more common. Abdominal pain, loss of weight and appetite with bowel disturbances are the common clinical manifestation. It is obvious from this study that tuberculosis is a problem in our country. Regarding the abdominal tuberculosis, the diagnostic problem persists for those patients where pulmonary tuberculosis is not obvious. Most tubercular patients present with perforation peritonitis.Conclusions: This study support fact that abdominal lymphadenopathy is important indicator of underlying cause of pathogenesis from which we reach the diagnosis. In present study in 10% cases only lymph nodes are positive for tuberculosis without tissue diagnosis, which can be treated to prevent the further complications.
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Jain, Dr Sharad, Dr Rashmi Nayak, Dr Sanjay Totade, and Dr Neelam Shukla. "Clinico - Pathological Correlation of Thyroid Swellings." International Journal of Medical Research and Review 2, no. 6 (2014): 553–60. http://dx.doi.org/10.17511/ijmrr.2014.i06.08.

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FAGAN, J. J., and N. S. TOLLEY. "Clinico-pathological correlation of T3glottic carcinomas." Clinical Otolaryngology 19, no. 6 (1994): 532–36. http://dx.doi.org/10.1111/j.1365-2273.1994.tb01284.x.

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Saracibar, N., E. Ortiz, A. Preciado, and V. Almeida. "ERYTHEMA MULTIFORME: A CLINICO-PATHOLOGICAL CORRELATION." American Journal of Dermatopathology 20, no. 6 (1998): 589. http://dx.doi.org/10.1097/00000372-199812000-00018.

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RAMAN, TS RAGHU, DALJIT SINGH, YP JALPOTA, and PK MENON. "CLINICO-PATHOLOGICAL CORRELATION IN NEONATAL AUTOPSIES." Medical Journal Armed Forces India 52, no. 1 (1996): 19–22. http://dx.doi.org/10.1016/s0377-1237(17)30828-6.

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Eswaradass, P., and B. Namasivayam. "Clinico-pathological correlation in peripheral neuropathy." Journal of the Neurological Sciences 405 (October 2019): 258–59. http://dx.doi.org/10.1016/j.jns.2019.10.1291.

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Reddy, Sagili VijayaBhaskar, Manoj Jain, and SushilKumar Gupta. "Dermopathy of Graves′ disease: Clinico-pathological correlation." Indian Journal of Endocrinology and Metabolism 16, no. 3 (2012): 460. http://dx.doi.org/10.4103/2230-8210.95714.

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Dissertations / Theses on the topic "Clinico-Pathological Correlation"

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Alomar, Thaer Saad. "In vivo confocal microscopy of the abnormal cornea : a clinical and clinico-pathological correlation." Thesis, University of Nottingham, 2012. http://eprints.nottingham.ac.uk/12586/.

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In vivo confocal microscopy (IVCM) offers a unique real time non-invasive imaging method to explore live tissues at cellular and subcellular levels of histological detail with magnifications very much comparable to conventional ‘ex vivo’ light microscopy. Therefore it has been widely used over the past two decades to investigate the ocular surface and cornea in health, disease and following surgical procedures. One of the main challenges in understanding IVCM is to get a proper interpretation of the images that present various figures and patterns of tissue structures all in black and white with variable degree of reflectivity, being whiter (or brighter) when they are more (or hyper) reflective. The lack of good correlation between IVCM and corresponding light microscopy for the same tissue samples has lead to speculative interpretations of IVCM images in the literature. In this work we tried to fill that gap through performing IVCM to patients with various corneal and ocular surface disorders just few days prior to obtaining the tissue samples (corneal graft, excisional biopsy or alcohol delamination) for histopathological examination to make sure that IVCM images were truly representative to the tissue details that might change if more time was left to elapse between IVCM and tissue sampling. In ocular surface disease group (chapter three) we contrasted IVCM criteria in conjunctival epithelial overgrowth onto the cornea in limbal stem cell deficiency and Pterygium-like disorders with those seen in Corneal/Conjunctival Intraepithelial neoplasia (CIN) to confirm reliable diagnostic criteria of CIN with IVCM. In corneal oedema (chapter four) IVCM viewing of (histologically confirmed) subepithelial fibroblasts without clinically visible corneal scarring has been reported for the first time in IVCM literature particularly in Fuchs endothelial dystrophy. Sub-basal corneal nerve reduction as well as stromal keratocytes and endothelial changes were clearly illustrated with IVCM. In keratoconus cases (chapter five) morphological epithelial changes related to regenerative atypia have been studied for the first time and compared to those seen in CIN through light microscopy and IVCM in addition to other epithelial changes associated with keratoconus. Novel IVCM criteria for Bowman’s zone breaks have been described and compared carefully with histological sections. In corneal dystrophies (chapter six) IVCM criteria in Thiel-Behnke’s corneal dystrophy (CDBII) as well as in macular, granular and lattice dystrophy correlated well with light microscopic findings with a novel IVCM pattern in one Macular dystrophy case. In Acanthamoeba keratitis (chapter seven) a new form of Acanthamoeba cysts has been described for the first time through IVCM. Moreover this study presented for the first time IVCM diagnostic criteria in corneal intraepithelial neoplasia, keratoconus, Acanthamoeba keratitis and some corneal dystrophies. A comprehensive IVCM illustration of various types and stages of corneal fibrosis has been achieved as well.
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Barday, Zibya. "Clinico-pathological correlation and outcome in patients with mesangioproliferative glomerulonephritis in Cape Town: A single centre study." Master's thesis, University of Cape Town, 2018. http://hdl.handle.net/11427/29802.

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Background Glomerulonephritis is a major cause of end-stage kidney disease (ESRD) in Africa. There is scanty data on the clinico-pathological characteristics and outcome of the mesangioproliferative glomerulonephritides in Africa, despite the non-IgA subtype being reported as a common cause of nephrotic syndrome. This study will assess the outcome of patients with biopsy proven mesangioproliferative glomerulonephritis (MesPGN) from a single centre in Cape Town, South Africa. Methods The study is designed as 10-year retrospective analysis of patients with biopsy proven MesPGN. The MesPGN patterns were divided into non-IgA MesPGN and IgA nephropathy (IgAN), depending on the predominant type of immune deposit. Univariate cox regression analysis was used to determine factors associated with ESRD. Results Data of 109 patients with renal biopsy-proven MesPGN were included for the period between 2005-2014. The mean age at biopsy was 33.8 ±14.9 years, 53.2% were males, and 39.4% were black Africans. Clinically, 58.7% presented with nephrotic syndrome. On histology 79.8% had non-IgA MesPGN, and 20.2% had IgAN. Compared to the non-IgA group, most patients with IgAN were not treated with immunosuppression (72.7% vs. 40.2%; p=0.006). At the last visit, 10.1% reached ESRD (40.9% vs. 2.3%; p<0.0001) and 30.2% achieved complete remission (9.1% vs. 35.7%; p=0.015) for IgAN and non-IgA MesPGN respectively. The 5-year renal survival for IgAN and non-IgA MesPGN respectively, were: 63.3% vs. 97.6%, log rank p=0.001. Overall, hypertension (p=0.019), not receiving immunosuppression (p=0.046) and having IgAN (p=0.007) were independent predictors of progression to ESRD. Conclusion There is a significantly higher ESRD-free survival of patients with biopsy proven non-IgA MesPGN than IgAN. Whether this is related to the limited use of immunosuppressive therapy in IgAN patients or represents a true nature of the disease still requires further research.
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Al-Qahtani, Khalid Hussain. "Detection of human papillomavirus in primary site of oraloropharyngeal cancer and in cervical lymph nodes : correlation with clinico-pathological parameters and prognostic significance." Thesis, McGill University, 2005. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=83960.

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Objectives. (1) To Determine the presence of HPV 6, 11, 16, 18, 31, 33, 35, 52b, 58 subtypes in resected oral/oropharyngeal SCCA cancer and associated lymph nodes. (2) To Determine if a relationship exists between koilocytosis, tumor grade, stage, or prognosis.<br>Methods. Retrospective analysis and pathology review of patients with SCCA of the oral cavity at McGill in the last 5 years was performed. Age at diagnosis, risk factors, tumor stage, grade, koilocytosis, treatment, outcome, and presence of HPV by PCR were analysed.<br>Results. 199 patients included were included in the analyses; 5 years mortality was 18.5%. 146 cases reviewed by pathology revealed 67% koilocytosis. One sample was positive for HPV subtype 35 as determined by PCR. Radiotherapy (p<0,5) and complications from radiotherapy (p<0.5) significantly affected survival.<br>Conclusions. Many oral SCCA's do not contain HPV 6, 11, 16, 18, 31, 33, 35, 52b, 58 subtypes. Given the high prevelence of koilocytosis, probe for other subtypes should be utilized. Mortality rates and survival are similar to those published in the literature. The presence of koilocytosis, it is not related grade, stage or prognosis. Only radiotherapy and its complications affect survival.
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Marulli, Giuseppe. "Detection of Squamos Cell Carcinoma Antigen (SCCA) in early and end stage Idiopathic Pulmonary Fibrosis (IPF): molecular substrates and clinico-pathological correlations." Doctoral thesis, Università degli studi di Padova, 2009. http://hdl.handle.net/11577/3426462.

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BACKGROUND Idiopathic pulmonary fibrosis (IPF), morphologically characterized by usual interstitial pneumonia (UIP), represents a progressive disease of unknown aetiology that continues to be associated with poor prognosis. The cardinal pathological features are the epithelial damage/activation, fibroblastic/myofibroblastic foci formation and extracellular matrix remodelling. The current paradigm suggests a pivotal role of the epithelium in the disease pathogenesis. Epithelial injury and subsequent deregulated repair results in profibrogenic cytokines (like TGF-?1) release with consequent abnormal mesenchymal cell activation and proliferation. Therefore, epithelial instability seems a crucial step in the development and progression of the disease, including neoplastic transformation. Few molecular tissue markers have been studied in IPF in order to clarify the pathogenetic mechanisms leading to the disease. Squamous cell carcinoma antigen (SCCA) is a serine protease inhibitor (serpin) physiologically found in the normal squamous epithelium and typically expressed by dysplastic and neoplastic epithelial cells of various origin, more often in squamous cell tumours. No information is actually available on its expression in IPF. MATERIAL AND METHODS In this study we analysed SCCA and TGF-b1 expression in surgical open-lung biopsies from 22 IPF patients with early-stage disease (GROUP A), in native lungs from 48 IPF patients with end-stage disease (GROUP B) who underwent to lung transplantation and in 20 control cases (GROUP C, 10 normal lungs from cadaveric donors, 10 lungs from patients with other interstitial diseases). In vitro study using A549 pneumocytes was also conducted to investigate the relationship between SCCA and TGFb1 expression. SCCA and TGFb1 epithelial expression were evaluated by immunohistochemistry and reverse transcription-polymerase chain reaction (RT-PCR). Time course analysis of TGF-b1 expression in A549 pneumocytes incubated with different SCCA concentrations was assessed by real time RT-PCR. The quantitative immunohistochemical assessment of SCCA and TGF-?1 was undertaken in each IPF sample by counting at least 500 cuboidal cells. A quantitative assessment of different pathological parameters (fibrosis, fibroblast foci and honeycombing changes) have been also considered in each samples. Clinical data including lung function and cardiovascular parameters were correlated to pathological features. In GROUP A lung function tests were re-evaluated at 8-12 months after biopsy. RESULTS Alveolar SCCA expression was present in IPF patients, but was not detected in alveolar cells in any of control cases. In GROUP A SCCA was positively correlated with the extension of fibroblastic foci (r=0.49, p=0.02), expression of TGF-b1 (r=0.78, p<0.0001) and with DLCO decline at follow-up (r=0.59, p=0.01). In vitro experiments showed that incubation of cultured cells with SCCA induced TGF-b1 expression, with a peak at 24 hours. In GROUP B SCCA and TGF-b1 values were high and positively correlated (r=0.45, p<0.001), while an inverse correlation was found between SCCA and DLCO (r=-0.43, p=0.005) and TGF-b1 and DLCO (r=-0.42, p=0.04). Interestingly, among metaplastic alveolar epithelial cells, a significant difference in SCCA expression was found between cuboidal, bronchialized and squamous cells, with increased expression for squamous cells that also presented a significant higher grade of dysplasia. CONCLUSION The over-expression of SCCA and TGF-?1 in the alveolar epithelium corroborates the hypothesis that disturbed epithelial alveolar regeneration and abnormal secretion of the cytokines are important steps in the pathogenesis of remodelling and fibrosis of IPF. SCCA could have a double role influencing the epithelial proliferation (autocrine action) and promoting fibroblast proliferation/fibrosis through increased TGF-?1 secretion (paracrine action). SCCA may be considered a potential marker of disease activity being strictly correlated with impairing lung function.<br>INTRODUZIONE La fibrosi polmonare idiopatica (FPI), caratterizzata morfologicamente dal correlato patologico di polmonite interstiziale usuale (UIP), rappresenta una malattia ad eziologia sconosciuta, ad andamento progressivo e prognosi infausta. Gli elementi anatomo-patologici cardine sono il danno/attivazione epiteliale, la formazione di foci fibroblastici/miofibroblastici ed il rimodellamento della matrice extracellulare. La teoria patogenetica più accreditata attribuisce un ruolo determinante alla disfunzione epiteliale alveolare. Il danno epiteliale ed i successivi meccanismi deregolati di riparo portano al rilascio di citochine pro-fibrogenetiche (come il TGF-b1) con conseguente attivazione e proliferazione di cellule mesenchimali. Di conseguenza, l’instabilità epiteliale sembra un elemento cruciale nello sviluppo e progressione della malattia, inclusa la trasformazione neoplastica. Pochi markers molecolari sono stati studiati e descritti fino ad ora nella FPI, per meglio chiarire i meccanismi patogenetici della malattia. Lo squamous cell carcinoma antigen (SCCA) è un inibitore delle serin proteasi (serpine) fisiologicamente presente nell’epitelio squamoso normale e specificamente espresso dalle cellule displastiche e neoplastiche epiteliali di varia origine, più spesso nei tumori a cellule squamose. Non sono ancora disponibili informazioni specifiche sulla sua espressione nella FPI. MATERIALI E METODI In questo studio abbiamo analizzato l’espressione di SCCA e TGF-b1 in tessuto polmonare ottenuto da biopsie chirurgiche di 22 pazienti affetti da FPI in stadio clinico iniziale (Gruppo A), in polmoni nativi di 48 pazienti con malattia end-stage e sottoposti a trapianto di polmone (Gruppo B) e in 20 polmoni controllo (Gruppo C, 10 polmoni normali ottenuti da donatori cadaveri, 10 polmoni di pazienti affetti da altre malattie interstiziali). Abbiamo inoltre condotto uno studio in vitro utilizzando pneumociti della linea A549, per investigare il rapporto tra la produzione di SCCA e l’espressione di TGF-b1. L’espressione di SCCA e TGF-b1 nelle cellule epiteliali è stata valutata con tecniche di immunoistochimica e reazione a catena della polimerasi-trascrizione inversa (RT-PCR). La produzione di TGF-b1 nei pneumoniti A549 incubati a diverse concentrazioni di SCCA è stata verificata con real time PCR. La valutazione quantitativa immunoistochimica di SCCA e TGF-b1 è stata eseguita in ciascun campione contando almeno 500 cellule cuboidali. Una valutazione quantitativa dei differenti parametri patologici (fibrosi, foci fibroblastici e honeycombing) è stata eseguita ugualmente in ciascun campione. I dati clinici, inclusi la funzione respiratoria e i parametri cardiovascolari sono stati correlati ai dati patologici. Nel Gruppo A i test di funzionalità polmonare sono stati ripetuti a 8-12 mesi dalla biopsia. RISULTATI L’espressione di SCCA nelle cellule epiteliali alveolari era presente nei pazienti con FPI, mentre era assente nei controlli. Nel Gruppo A l’SCCA era correlato positivamente con l’estensione dei foci fibroblastici (r=0.49, p=0.02), l’espressione di TGF-b1 (r=0.78, p<0.0001) e con il declino della DLCO al follow up (r=0.59, p=0.01). L’esperimento in vitro ha dimostrato che l’incubazione di pneumociti con SCCA induceva l’espressione di TGF-b1, con un picco a 24 ore. Nel Gruppo B i valori di SCCA e TGF-b1 erano elevati e correlati positivamente (r=0.45, p<0.001), mentre vi era una correlazione inversa tra SCCA e DLCO (r=-0.43, p=0.005) e TGF-b1 e DLCO (r=-0.42, p=0.04). Tra le cellule epiteliali alveolari metaplastiche, abbiamo riscontrato una diversa espressione di SCCA tra le cellule cuboidali, bronchializzate e squamose, con una crescente espressione per le squamose che inoltre presentavano un maggior grado di displasia. CONCLUSIONI La over-espressione di SCCA e TGF-b1 nell’epitelio alveolare corrobora l’ipotesi che una alterata rigenerazione epiteliale alveolare e una secrezione anormale di citochine sono elementi importanti nella patogenesi del rimodellamento e della fibrosi della FPI. L’SCCA potrebbe avere un duplice ruolo influenzando la proliferazione epiteliale (azione autocrina) e promuovendo la fibrosi/proliferazione dei fibroblasti attraverso lo stimolo alla maggior secrezione di TGF-b1. L’SCCA potrebbe essere considerato un marker potenziale di attività della malattia essendo strettamente correlato con la perdita di funzione polmonare.
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Ferreira, Marli de Fátima Pereira. "An updated review of Parkinson disease genetics and clinico-pathological correlations." Master's thesis, 2016. https://repositorio-aberto.up.pt/handle/10216/89500.

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Ferreira, Marli de Fátima Pereira. "An updated review of Parkinson disease genetics and clinico-pathological correlations." Dissertação, 2016. https://repositorio-aberto.up.pt/handle/10216/89500.

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Books on the topic "Clinico-Pathological Correlation"

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Sorger, Karin. Postinfectious glomerulonephritis: Subtypes, clinico-pathological correlations, and follow-up studies. Fischer, 1986.

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Kallarakkal, Thomas George. Clinico-Pathological Correlation of Oral Diseases. Springer International Publishing AG, 2023.

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Smith, Owen P., and Jonathan Bond. Clinico-Pathological Correlations in Paediatric Haematology. Cambridge University Press, 2008.

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Sullivan, Mark D. Escaping the Autonomy Versus Objectivity Trap by Repersonalizing the Clinical Problem. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780195386585.003.0004.

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Respect for patient autonomy has been sought as the antidote to the depersonalization that ails modern medicine. It serves as a challenge to the dominance of impersonal disease diagnosis in treatment choice. We now repersonalize treatment at a late stage through the informed consent process. If we are to find another way to repersonalize health care, we need to understand the historical roots of the patient autonomy versus objective disease dynamic in which we are trapped. The same disengaged self that sees ethics in terms of autonomy also sees disease as an observable tissue lesion within the body at autopsy. Clinico-pathological correlation offers a gold standard for clinical diagnosis and a completely objective access to disease. This ability to diagnose objective disease is the source of physician paternalism. It can be countered by incorporating the patient’s view of the clinical problem back into the diagnostic process.
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Book chapters on the topic "Clinico-Pathological Correlation"

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Polack, Frank M. "Corneal Graft Rejection: Clinico-Pathological Correlation." In Ciba Foundation Symposium 15 - Corneal Graft Failure. John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/9780470719985.ch8.

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Hawaibam, Monalisa, Deepak Singh Lourembam, and Sushma Khuraijam. "Immunohistochemical Profiling and Clinico-Pathological Correlation of Breast Cancer: A Study from Regional Cancer Center in Northeast India." In Healthcare Research and Related Technologies. Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-99-4056-1_5.

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Hall, R., S. McLachlan, M. McGregor, A. P. Weetman, and B. Rees Smith. "Thyrotropin Receptor Antibodies: Clinico-Pathological Correlations." In Ciba Foundation Symposium 90 - Receptors, Antibodies and Disease. John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470720721.ch9.

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Wolters, E. Ch, and H. Braak. "Parkinson’s disease: premotor clinico-pathological correlations." In Parkinson’s Disease and Related Disorders. Springer Vienna, 2006. http://dx.doi.org/10.1007/978-3-211-45295-0_47.

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Mackay, Ian R., and E. Eric Gershwin. "The autoantibodies of primary biliary cirrhosis: clinico pathological correlations." In Manual of Biological Markers of Disease. Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-011-1670-1_48.

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Jaffrey, Ashna, Megalai A. Sakthi, R. Mahadhi, Divyabharat Ketan Rastogi, and R. Sankar Narayanan. "A Clinico-Pathological Study Correlating Particular Dermatoses Associated with Interface Dermatitis." In Recent Developments in Microbiology, Biotechnology and Pharmaceutical Sciences. CRC Press, 2025. https://doi.org/10.1201/9781003618140-47.

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Al-Khyatt, Waleed, and Syed Yusuf Iftikhar. "Serum Sex Hormone Profiles in Potentially Resectable Esophageal Cancer." In Reproductive Hormones. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.95030.

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Esophageal cancer (EC) affects men far more commonly than women. Numerous epidemiological studies have suggested that the hormonal milieu may play a role in this gender bias. However, there is little known about circulating sex hormone levels in relation to the risk of EC development. In this chapter, the correlation between circulating sex hormone levels and mRNA expression of estrogen receptors (ER) in normal esophageal mucosal samples and EC biopsies from patients with potentially resectable EC is studied. Moreover, the association of serum sex hormones levels with and clinico-pathological features of EC is analysed.
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"How to Take a Vulval Biopsy and the Importance of Clinico-Pathological Correlation." In A Practical Guide to Vulval Disease. John Wiley & Sons, Ltd, 2017. http://dx.doi.org/10.1002/9781119146087.ch3.

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Agarwal, Neeru M., V. C. Popat, and Atul Shrivastav. "Significance of Clinico-Pathological Correlation of Various Ear Masses – A Study at a Tertiary Care Hospital of Western Gujarat." In Perspective of Recent Advances in Medical Research Vol. 2. B P International (a part of SCIENCEDOMAIN International), 2022. http://dx.doi.org/10.9734/bpi/pramr/v2/4171e.

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Caselli Richard J. and Reiman Eric M. "Characterizing the Preclinical Stages of Alzheimer's Disease and the Prospect of Presymptomatic Intervention." In Advances in Alzheimer’s Disease. IOS Press, 2013. https://doi.org/10.3233/978-1-61499-154-0-405.

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Studies of asymptomatic carriers of genes that are known to predispose to Alzheimer's disease (AD) have facilitated the characterization of preclinical AD. The most prevalent genetic risk factor is the &amp;epsiv;4 allele of apolipoprotein E (APOE). Neuropathological studies of young deceased &amp;epsiv;4 carriers have shown modest but abnormal amounts of neocortical amyloid and medial temporal neurofibrillary tangles that is also reflected in cerebrospinal fluid (CSF) biomarkers, amyloid-&amp;beta;, and phospho-tau in particular. MRI studies have shown progressive hippocampal and gray matter atrophy with the advent of mild cognitive impairment (MCI), and fluorodeoxyglucose PET scans show reduced cerebral metabolism in posterior cingulate and related AD regions evident even in 30 year olds. Cerebral amyloidosis disclosed by more recent amyloid ligand PET studies in asymptomatic 60 year olds increases in parallel with &amp;epsiv;4 gene dose. Longitudinal neuropsychological studies have revealed accelerated memory decline in &amp;epsiv;4 carriers beginning around age 55&amp;ndash;60 years whose severity again parallels &amp;epsiv;4 gene dose. The clinico-pathological correlation of declining memory and AD-like neuropathological change defines preclinical AD and has set the stage for the accelerated evaluation of presymptomatic AD treatments. In this article, we briefly consider some of the earliest detectable changes associated with the predisposition to AD, and some of the prevention trial strategies that have been proposed to help find treatments to reduce the risk, postpone the onset of, or completely prevent AD symptoms as soon as possible.
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Conference papers on the topic "Clinico-Pathological Correlation"

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Mukhopadhyay, Asima, Nicola Curtin, and Richard Edmondson. "Clinico-pathological correlation of homologous recombination status in epithelial ovarian cancer: Surgeon’s perspective." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685292.

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Background: TCGA data using expensive multi-modality diagnostic platforms have shown that 50% epithelial ovarian cancers (EOCs) are estimated to be homologous recombination (HR) deficient (HRD). We developed a functional assay for HR using gamma H2AX-Rad51 immunofluoresence.[1] Methods: Primary cultures were developed in 50 consecutive EOCs from ascetic fluid and HR assay was performed. Results: 50% patients were HRD based on the functional assay and show improved ex-vivo chemosensitivity to PARP inhibitor (PARPi) (PPV = 92%, NPV = 100%). HRD patients showed improved platinum sensitivity (53.8% vs 16.7%), survival (12 month OS - 41.7% vs. 11.5%) and optimal cytoreduction (80% vs. 62%) rates compared to HR competent (HRC) tumours which are less responsive and represent an unmet clinical need. Conclusions: Personalised surgical and chemotherapeutic strategies may be developed for HR stratified EOCs. Primary surgery may be the preferred approach in HRC due to poor chemoresponse; surgical expertise/environment should be optimised to ensure optimal surgical outcome. Intra-operative hyperthermic treatment and selective HR inhibitors may improve subsequent chemoresponse in HRC and are currently being investigated.
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Ray, Animesh, Naveet Wig, Deepali Jain, et al. "Post-mortem minimally invasive tissue sampling and clinico-pathological correlation in fatal COVID-19 infection." In ERS International Congress 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/13993003.congress-2021.oa3968.

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Varela, M., A. Moreno, C. Falo, et al. "Correlation between pN0(mol+) and standard clinico-pathological prognostic factors in SLN of breast cancer patients." In CTRC-AACR San Antonio Breast Cancer Symposium: 2008 Abstracts. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-1025.

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Agarwal, Neha, Jagannath Mishra, Arunava Roy, et al. "#995 Surgical management with clinico-pathological and radiological correlation of suspicious cardiophrenic lymph nodes in advanced ovarian cancer." In ESGO 2023 Congress. BMJ Publishing Group Ltd, 2023. http://dx.doi.org/10.1136/ijgc-2023-esgo.686.

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Dharmarajan, Adarsh, and M. Geetha. "Audit on early stage carcinoma cervix primarily treated with radical surgery: A tertiary cancer care centre experience." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685262.

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Introduction: Clinical staging is universally accepted for ca cervix. In early stage of carcinoma cervix both radiation and radical hysterectomy given equivalent local control rates as well as survival. Poor prognostic factors following surgery would necessitate-post-operative adjuvant radiation. Selecting the patients who is unlikely to require adjuvant treatment after surgery spares them the toxicity of multiple treatment modalities, which is worse than alone. Aim: To find out clinico-pathological correlation in early stage carcinoma cervix treated with the surgery. Materials and Methods: It is a retrospective audit of study. All carcinoma cervix cases primarily treated with surgery. Results: A total of 25 cases were treated in this study. The median age of patients observed with 48 years. The common symptoms and stage were vaginal discharge (i.e., 42.30%) and 1B1 (61.53%). Most of patients were treated with type III radical hysterectomy and their clinical staging was correlated with the final histo-pathological staging. A total of 11 (i.e., 42.30%) required adjuvant treatment among them 7 (63.633%), 1 (9.09%) and 3 (27.27%) patients were in 1B1 1B2 and 2A respectively. The chi-square test has been performed to compute the correlation between clinical and histo-pathological finding. It shows that significant amount of relation present between clinical and histo-pathological findings.
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Parvathareddy, Sandeep K., Abdul K. Siraj, Padmanaban Annaiyappanaidu, et al. "Abstract 3186: Differential expression of PD-L1 between primary and metastatic Middle-Eastern epithelial ovarian carcinoma and its clinico-pathological correlation." In Proceedings: AACR Annual Meeting 2021; April 10-15, 2021 and May 17-21, 2021; Philadelphia, PA. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.am2021-3186.

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Xiu, Joanne, Sandeep Reddy, and Wafik S. El-Deiry. "Abstract 4923: Expression of class III beta-tubulin (TUBB3) in 3580 colorectal cancers (CRCs) and correlation with clinico-pathological and molecular features." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-4923.

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Al-Janabi, Omar, Helge Taubert, Sven Wach, et al. "Abstract 4683: Correlation of transcript levels of members of the urokinase-type plasminogen activator system with clinico-pathological parameters in prostate cancer ." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-4683.

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Bu, Rong, Prashant Bavi, Azhar R. Hussain, et al. "Abstract 2980: Clinico-pathological correlation of UBE2C alterations in colorectal carcinoma and efficacy of UBE2C inhibition by proteosome inhibitor as a viable therapeutic target." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-2980.

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Qiu, Qingchao, Yinghao Su, Wei Lu, et al. "Abstract 2596: Correlation of TGF beta receptor II and pSmad2 expression with clinico-pathological factors in human breast cancer: An immunohistochemical observation from the Shanghai Breast Cancer Study." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-2596.

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