Academic literature on the topic 'Clock control'

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Journal articles on the topic "Clock control"

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Clark, Amelia M., and Brian J. Altman. "Circadian control of macrophages in the tumor microenvironment." Journal of Immunology 208, no. 1_Supplement (May 1, 2022): 165.06. http://dx.doi.org/10.4049/jimmunol.208.supp.165.06.

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Abstract Introduction All leukocytes tested to date have functional circadian clocks, and nearly every arm of the immune response is subject to circadian regulation. Circadian clocks instruct the time-of-day-dependent, rhythmic expression of genes in a tissue- and cell-specific manner. In macrophages (mΦs), the circadian clock regulates several factors that are critical to executing effective immune responses. Tumor-associated mΦs are major contributors to immune suppression in the tumor microenvironment (TME). Evidence suggests that metabolically stressful factors in the TME such as acidic pH and nutrient limitation promote mΦ-mediated immune suppression, and recent data point to dysregulation of the circadian clock downstream of metabolic stress. Methods We study the effect of TME-associated metabolic stress on the circadian clock of mΦs in vitro by culturing bone marrow-derived mΦs in conditions mimicking acidic pH and nutrient limitations that have been observed in the TME. To study the impact of mΦ-intrinsic circadian rhythms on tumorigenesis in vivo, we use mice genetically engineered to have a myeloid cell-specific disruption of the circadian clock via deletion of the key clock protein BMAL1. Results Oscillation of core clock proteins is altered in mΦs subjected to TME-associated metabolic stress. Additionally, we observe increased tumor growth in mice co-injected with mΦs whose circadian clocks were disrupted compared to mice co-injected with mΦs whose circadian clocks were functional. Conclusion Our data suggests that stressful conditions associated with the TME can alter the mΦ circadian clock, and that a functional circadian clock in mΦs can suppress tumor growth in a syngeneic murine tumor model of pancreatic cancer. This research has been supported by the following fellowships and grants: 2021-Current: Wilmot Predoctoral Cancer Research Fellowship, Wilmot Cancer Institute, University of Rochester Medical Center, Rochester, NY 2020-2021: NIH T32 Training Grant in Cellular, Biochemical & Molecular Sciences, University of Rochester Medical Center, Rochester, NY
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Xiao, Yangbo, Ye Yuan, Mariana Jimenez, Neeraj Soni, and Swathi Yadlapalli. "Clock proteins regulate spatiotemporal organization of clock genes to control circadian rhythms." Proceedings of the National Academy of Sciences 118, no. 28 (July 7, 2021): e2019756118. http://dx.doi.org/10.1073/pnas.2019756118.

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Circadian clocks regulate ∼24-h oscillations in gene expression, behavior, and physiology. While the genetic and molecular mechanisms of circadian rhythms are well characterized, what remains poorly understood are the intracellular dynamics of circadian clock components and how they affect circadian rhythms. Here, we elucidate how spatiotemporal organization and dynamics of core clock proteins and genes affect circadian rhythms in Drosophila clock neurons. Using high-resolution imaging and DNA-fluorescence in situ hybridization techniques, we demonstrate that Drosophila clock proteins (PERIOD and CLOCK) are organized into a few discrete foci at the nuclear envelope during the circadian repression phase and play an important role in the subnuclear localization of core clock genes to control circadian rhythms. Specifically, we show that core clock genes, period and timeless, are positioned close to the nuclear periphery by the PERIOD protein specifically during the repression phase, suggesting that subnuclear localization of core clock genes might play a key role in their rhythmic gene expression. Finally, we show that loss of Lamin B receptor, a nuclear envelope protein, leads to disruption of PER foci and per gene peripheral localization and results in circadian rhythm defects. These results demonstrate that clock proteins play a hitherto unexpected role in the subnuclear reorganization of core clock genes to control circadian rhythms, revealing how clocks function at the subcellular level. Our results further suggest that clock protein foci might regulate dynamic clustering and spatial reorganization of clock-regulated genes over the repression phase to control circadian rhythms in behavior and physiology.
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Weitzman, Jonathan B. "Clock control." Genome Biology 3 (2002): spotlight—20021115–01. http://dx.doi.org/10.1186/gb-spotlight-20021115-01.

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Alvarez, J. D., and Amita Sehgal. "Finer clock control." Nature 419, no. 6909 (October 2002): 798–99. http://dx.doi.org/10.1038/419798a.

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Reed, Ruth. "Body clock control." New Scientist 191, no. 2570 (September 2006): 20. http://dx.doi.org/10.1016/s0262-4079(06)60541-1.

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Guan, Dongyin, Ying Xiong, Trang Minh Trinh, Yang Xiao, Wenxiang Hu, Chunjie Jiang, Pieterjan Dierickx, Cholsoon Jang, Joshua D. Rabinowitz, and Mitchell A. Lazar. "The hepatocyte clock and feeding control chronophysiology of multiple liver cell types." Science 369, no. 6509 (July 30, 2020): 1388–94. http://dx.doi.org/10.1126/science.aba8984.

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Most cells of the body contain molecular clocks, but the requirement of peripheral clocks for rhythmicity and their effects on physiology are not well understood. We show that deletion of core clock components REV-ERBα and REV-ERBβ in adult mouse hepatocytes disrupts diurnal rhythms of a subset of liver genes and alters the diurnal rhythm of de novo lipogenesis. Liver function is also influenced by nonhepatocytic cells, and the loss of hepatocyte REV-ERBs remodels the rhythmic transcriptomes and metabolomes of multiple cell types within the liver. Finally, alteration of food availability demonstrates the hierarchy of the cell-intrinsic hepatocyte clock mechanism and the feeding environment. Together, these studies reveal previously unsuspected roles of the hepatocyte clock in the physiological coordination of nutritional signals and cell-cell communication controlling rhythmic metabolism.
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Shakhmantsir, Iryna, and Amita Sehgal. "Splicing the Clock to Maintain and Entrain Circadian Rhythms." Journal of Biological Rhythms 34, no. 6 (August 7, 2019): 584–95. http://dx.doi.org/10.1177/0748730419868136.

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Circadian clocks drive daily rhythms of physiology and behavior in multiple organisms and synchronize these rhythms to environmental cycles of light and temperature. The basic mechanism of the clock consists of a transcription-translation feedback loop, in which key clock proteins negatively regulate their own transcription. Although much of the focus with respect to clock mechanisms has been on the regulation of transcription and on the stability and activity of clock proteins, it is clear that other regulatory processes also have to be involved to explain aspects of clock function. Here, we review the role of alternative splicing in circadian clocks. Starting with a discussion of the Drosophila clock and then extending to other major circadian model systems, we describe how the control of alternative splicing enables organisms to maintain their circadian clocks as well as to respond to environmental inputs, in particular to temperature changes.
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Costello, Hannah M., and Michelle L. Gumz. "Circadian Rhythm, Clock Genes, and Hypertension: Recent Advances in Hypertension." Hypertension 78, no. 5 (November 2021): 1185–96. http://dx.doi.org/10.1161/hypertensionaha.121.14519.

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Accumulating evidence suggests that the molecular circadian clock is crucial in blood pressure (BP) control. Circadian rhythms are controlled by the central clock, which resides in the suprachiasmatic nucleus of the hypothalamus and peripheral clocks throughout the body. Both light and food cues entrain these clocks but whether these cues are important for the circadian rhythm of BP is a growing area of interest. The peripheral clocks in the smooth muscle, perivascular adipose tissue, liver, adrenal gland, and kidney have been recently implicated in the regulation of BP rhythm. Dysregulation of the circadian rhythm of BP is associated with adverse cardiorenal outcomes and increased risk of cardiovascular mortality. In this review, we summarize the most recent advances in peripheral clocks as BP regulators, highlight the adverse outcomes of disrupted circadian BP rhythm in hypertension, and provide insight into potential future work in areas exploring the circadian clock in BP control and chronotherapy. A better understanding of peripheral clock function in regulating the circadian rhythm of BP will help pave the way for targeted therapeutics in the treatment of circadian BP dysregulation and hypertension.
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Yang, Guang You, Zhi Jian Ye, Shuang Qing Zhang, and Wan Xu. "Research and Implementations of the IEEE 1588 Precision Time Protocol Based on ARM-Linux." Advanced Materials Research 156-157 (October 2010): 1492–96. http://dx.doi.org/10.4028/www.scientific.net/amr.156-157.1492.

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The clock synchronization is the key technology in distributed control system. This paper investigates the method to adjust computer clock frequency and time in embedded control system based on Ethernet. This paper also analyses the basic working principle of the IEEE 1588 Precision Time Protocol. In particular, it outlines the working principle of the free PTPd that is the software only implementations of the IEEE 1588 Precision Time Protocol. In the ARM-Linux environment, it presents a clock synchronization method to achieve high precise clock synchronization in distributed control system using PTPd. The results indicate that it is able to synchronize distributed clocks with the accuracy less than 500 microseconds using PTPd without the support of specialized hardware.
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Li, Shujing, and Luoying Zhang. "Circadian Control of Global Transcription." BioMed Research International 2015 (2015): 1–8. http://dx.doi.org/10.1155/2015/187809.

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Circadian rhythms exist in most if not all organisms on the Earth and manifest in various aspects of physiology and behavior. These rhythmic processes are believed to be driven by endogenous molecular clocks that regulate rhythmic expression of clock-controlled genes (CCGs). CCGs consist of a significant portion of the genome and are involved in diverse biological pathways. The transcription of CCGs is tuned by rhythmic actions of transcription factors and circadian alterations in chromatin. Here, we review the circadian control of CCG transcription in five model organisms that are widely used, including cyanobacterium, fungus, plant, fruit fly, and mouse. Comparing the similarity and differences in the five organisms could help us better understand the function of the circadian clock, as well as its output mechanisms adapted to meet the demands of diverse environmental conditions.
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Dissertations / Theses on the topic "Clock control"

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Park, Myoung Jin. "An optimistic concurrency control mechanism based on clock synchronization." CSUSB ScholarWorks, 1996. https://scholarworks.lib.csusb.edu/etd-project/982.

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Ohlsson, Henrik. "Mathematical Analysis of a Biological Clock Model." Thesis, Linköping University, Department of Electrical Engineering, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-6750.

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Have you thought of why you get tired or why you get hungry? Something in your body keeps track of time. It is almost like you have a clock that tells you all those things.

And indeed, in the suparachiasmatic region of our hypothalamus reside cells which each act like an oscillator, and together form a coherent circadian rhythm to help our body keep track of time. In fact, such circadian clocks are not limited to mammals but can be found in many organisms including single-cell, reptiles and birds. The study of such rhythms constitutes a field of biology, chronobiology, and forms the background for my research and this thesis.

Pioneers of chronobiology, Pittendrigh and Aschoff, studied biological clocks from an input-output view, across a range of organisms by observing and analyzing their overt activity in response to stimulus such as light. Their study was made without recourse to knowledge of the biological underpinnings of the circadian pacemaker. The advent of the new biology has now made it possible to "break open the box" and identify biological feedback systems comprised of gene transcription and protein translation as the core mechanism of a biological clock.

My research has focused on a simple transcription-translation clock model which nevertheless possesses many of the features of a circadian pacemaker including its entrainability by light. This model consists of two nonlinear coupled and delayed differential equations. Light pulses can reset the phase of this clock, whereas constant light of different intensity can speed it up or slow it down. This latter property is a signature property of circadian clocks and is referred to in chronobiology as "Aschoff's rule". The discussion in this thesis focus on develop a connection and also a understanding of how constant light effect this clock model.

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Greene, Andrew Vanderford. "Organization of the circadian clock and control of rhythmicity in fungi." Diss., Texas A&M University, 2005. http://hdl.handle.net/1969.1/4161.

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Circadian rhythms in biological processes occur in a wide range of organisms and are generated by endogenous oscillators. In Neurospora crassa, the FRQ-oscillator (comprised of FRQ, WC-1 and WC-2) is essential for rhythms in asexual sporulation and gene expression. How this oscillator signals to the cell to control rhythmicity is unknown. Furthermore, under certain growth conditions, rhythms are observed in FRQ-null strains, indicating the presence of one or more FRQ-less oscillators (FLOs). Interestingly, while circadian rhythms are observed in the related Aspergillus spp., they lack the frq gene, leading to the hypothesis that a FLO is responsible for rhythms in Aspergillus. Thus, Aspergillus provides a useful organism to investigate the components of the FLO. To investigate how an oscillator controls circadian output, we characterized the role of N. crassa NRC-2. The nrc-2 gene is under control of the clock and encodes a putative serine-threonine protein kinase. In a NRC-2-null strain cultured in low glucose conditions, FRQ-oscillator-dependent outputs are arrhythmic, but are rhythmic in high glucose. Our data suggests a model whereby NRC-2 relays metabolic information to the FRQ-oscillator to control rhythmic output. To understand the role of FLO(s) in the N. crassa circadian system, we examined regulation of the ccg-16 gene. We show that ccg-16 transcript rhythmicity is FRQ-independent, but WC-1-dependent. Furthermore, in contrast to current models for the FRQ-oscillator, we observed that rhythms in WC-1 protein accumulation persist in the absence of FRQ. These data support a new model involving two oscillators that are coupled through the WC-1 protein and that regulate different outputs. One approach to identify components of the FLO involved characterizing circadian rhythms in Aspergillus spp, which lacks FRQ. We find that A. flavus and A. nidulans, display circadian rhythms in sporulation and gene expression, respectively. Together, these findings provide a foundation for the identification of FLO components in both Aspergillus and N. crassa, that will ultimately lead to an understanding of how a multi-oscillator system can generate and coordinate circadian rhythmicity.
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Lewis, Zachary Austin. "Control of rhythmic output from the circadian clock in Neurospora crassa." Texas A&M University, 2004. http://hdl.handle.net/1969.1/1376.

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Circadian rhythms are visible as daily oscillations in biochemical, physiological, or behavioral processes. These rhythms are produced by an endogenous clock that maintains synchrony with the external environment through responses to external stimuli such as light or temperature. The clock, in turn, coordinates internal processes in a time-dependent fashion. Genetic and molecular analysis of the filamentous fungus Neurospora crassa has demonstrated that the products of the frequency (frq) and white-collar (wc-1 and wc-2) genes interact to form an interlocked feedback loop that lies at the heart of the clock in this fungus. This feedback loop, termed the FRQ/WC oscillator, produces a ~24h oscillation in frq mRNA, FRQ protein, and WC-1 protein. In turn, the FRQ/WC oscillator regulates rhythmic behavior and gene expression. The goal of this dissertation is to understand how rhythmic outputs are regulated by the FRQ/WC oscillator in Neurospora. To this end, we have taken a microarray approach to first determine the extent of clock-controlled gene expression in Neurospora. Here, we show that circadian regulation of gene expression is widespread; 145 genes, representing 20% of the genes we analyzed, are clock-controlled. We show that clockregulation is complex; clock-controlled genes peak at all phases of the circadian cycle. Furthermore, we demonstrate the clock regulates diverse biological processes, such as intermediary metabolism, translation, sexual development and asexual development. WC-1 is required for all light- and clock-regulated gene expression in Neurospora. We have shown that overexpression of WC-1 is sufficient to activate clock-controlled gene expression, but is not sufficient to induce all light-regulated genes in Neurospora. This result indicates that cycling of WC-1 is sufficient to regulate rhythmic expression of a subset of clockcontrolled genes. Conversely, a post-translational mechanism underlies WC-1 mediated light signal transduction in Neurospora. Finally, we have demonstrated the Neurospora circadian system is comprised of mutually coupled oscillators that interact to regulate output gene expression in the fungus.
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Ruiter, Marieke. "Biological clock control of daily glucose metabolism hormonal and autonomic pathways /." [S.l. : Amsterdam : s.n.] ; Universiteit van Amsterdam [Host], 2005. http://dare.uva.nl/document/77997.

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Trané, Camilla. "Robustness Analysis of Intracellular Oscillators with Application to the Circadian Clock." Licentiate thesis, KTH, Automatic Control, 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-4815.

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Periodic oscillations underlie many intracellular functions, such as circadian time keeping, cell cycle control and locomotor pattern generation in nerve cells. These intracellular oscillations are generated in intricate biochemical reaction networks involving genes, proteins and other biochemical components. In most cases, robust oscillations are of pivotal importance for the organism, i.e., the oscillations must be maintained in the presence of internal and external perturbations.

Model based analysis of robustness in intracellular oscillators has attracted considerable attention in recent years. The analysis has almost exclusively been based on either complete removal of network components, e.g., single genes, or perturbation of model parameters. In this thesis, a control theoretic approach to analyze structural robustness of intracellular oscillators is proposed. The method is based on adding dynamic perturbations to the network interactions. Determination of the smallest perturbation translating the underlying steady-state into a Hopf bifurcation point is used to quantify the robustness. The method can be used to determine critical substructures within the overall network and to identify specific network fragilities. Also, an approach to nonlinear model reduction based on the robustness analysis is proposed.

The proposed robustness analysis method is applied to elucidate mechanisms underlying robust oscillations in circadian clocks. Circadian clocks, molecular oscillators generating 24 hour rhythms in many organisms, are known to display a striking robustness towards internal and external perturbations. The underlying networks involve a large number of genes that are transcribed into mRNA which produce proteins subsequently regulating the activity of other genes, together forming an intricate network with a large number of embedded feedback loops. An often recurring hypothesis is that the interlocked feedback loop structure of circadian clocks serves the purpose of robustness. From analysis of several recently published models of circadian clocks, it is found in this thesis that the robustness of circadian clocks primarily results from a high gain in a single gene regulatory feedback loop generating the oscillations. This gain can be elevated by additional feedback loops, involving either gene regulation or post-translational feedback, but a similar robustness can be achieved by simply increasing the amplification within the master feedback loop.

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Kawazoe, Ryo. "Control of chloroplast gene expression by a circadian clock in Chlamydomonas reinhardtii /." Digital version:, 2000. http://wwwlib.umi.com/cr/utexas/fullcit?p9992832.

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Grundy, Jack. "Control of environmental stress responses by the circadian clock and abscisic acid." Thesis, University of Warwick, 2016. http://wrap.warwick.ac.uk/90280/.

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Plants are exposed to a variety of abiotic stresses, including salinity and drought. These environmental stresses cause major losses in crop yield. High salinity stress alone impairs crop production on at least 20% of irrigated land worldwide. Thus, the development of stress-tolerant crops is of major importance for food security. Many physiological responses to ensure acclimation to adverse environmental conditions require the synthesis and perception of the plant hormone abscisic acid (ABA). Recent studies have shown that the function of the circadian clock is altered under some abiotic stress conditions such as drought, and osmotic stress. The first part of this thesis investigates the role of the stress response hormone abscisic acid in changing the function of the clock under osmotic stress. It was found that multiple core clock genes are responsive to ABA application, with sharp transient induction of morning associated genes in particular. In comparison, osmotic stress caused a damping of the amplitude of gene expression. It was then shown that the disruptive effect of osmotic stress on circadian leaf movement rhythms required the biosynthesis of ABA. This is important as it demonstrates that ABA is a key factor in mediating osmotic stress responses to the clock. The second half of this thesis then focuses on how altered function of the clock might impact plant performance under drought or osmotic stress. It was found that the morning associated LATE ELONGATED HYPOCOTYL (LHY) transcription factor, which functions as a key component of the circadian clock, regulates many of the components of the ABA signalling pathway. Evidence was provided that, while overexpression of LHY results in reduced ABA levels, ABA responsive gene expression is significantly increased upon ABA treatment. Finally, through phenotypic analysis it was determined that increased LHY expression leads to increased performance in drought and osmotic stress conditions. This is important as it suggests that manipulation of circadian clock function may be useful as a novel approach in the future engineering of stress tolerant crop lines.
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Zhang, Zhuming. "Clock Control of Circadian Changes of Ommatidial Structure in the Cockroach, Leucophaea Maderae (L.)." TopSCHOLAR®, 1993. https://digitalcommons.wku.edu/theses/3025.

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All eukaryotic organisms display rhythms which persist under constant environmental conditions with periods of approximately, but very seldom exactly, 24 hours. Such rhythms are "circadian" and are driven by an internal "biological clock." Circadian rhythms of locomotor activity, retinal sensitivity to light and ommatidial morphology have been characterized in the cockroach, Leucophaea maderae (L.). It is not known whether the same clock(s) controls both circadian rhythms of electroretinogram (ERG) amplitude and morphological changes of the compound eye. In order to determine whether the location of the clock that controls morphological changes is in the same location as the one that controls ERG, brain lesions were made proximal or distal to the region of the putative clock regulating the expression of a circadian rhythm in eye sensitivity to light in anesthetized cockroaches. These and sham operated control animals were held for approximately two weeks under LD 12:12 at 25±2°C conditions in environmental chambers. After this time period, conditions of continuous darkness were established in order to allow rhythms to free run. Eye tissue was removed on subjective midday two and subjective midnight two, fixed, embedded, sectioned and the sections were examined and photographed using a Zeiss transmission electron microscope. Observations were made to establish the presence or absence of a circadian rhythm of submicrovillar cysternae area (SMC), as well as rhabdom area and screening pigment granules organization (SPG). The results indicate that the clock, located in the lobular neuropil region of the optic lobe that controls the rhythm of morphological changes in the eye, is in the same proximity as that which controls the changes in ERG amplitude.
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Alhumaydhan, Norah. "The Role of the Circardian Clock in the Control of Plant Immunity in Arabidopsis Thaliana." Thesis, Université d'Ottawa / University of Ottawa, 2015. http://hdl.handle.net/10393/31915.

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The circadian clock regulates a wide range of biological processes, allowing plants to be prepared for predictable daily diurnal changes in environmental cues such as light and temperature. Recent studies have suggested that the circadian clock may also control plant immunity. The exact nature of the interaction between the circadian clock and plant pathogens remains unknown. Our focus in this study is on the elucidation of the role of the biological clock in plant immunity against the necrotrophic pathogen to Botrytis cinerea. In order to do this we tested the level of susceptibility to B. cinerea in Arabidopsis thaliana wild type and transgenic plants: toc1, cca1/lhy, cca1/toc1, lhy/toc1, cca1/lhy/toc1, GLK1 OE, GLK2 OE, glk1, glk2, and glk1/glk2. We demonstrated that the time of infection plays a role in susceptibility to B. cinerea. Specifically, we found that plants are more susceptible to infection in the subjective morning. We also found that genetic mutations in core clock components or in GLK genes leads to changes in susceptibility to B. cinerea. Our data suggests that clock genes are not solely responsible for plant immune responses to B. cinerea but rather the ways in which the biological clock system regulates outcome pathways. Furthermore, when we entrain the biological clock by changing the photoperiod (day length) in normal earth conditions LD 24h and SD 24h, we observed that short day plants had higher susceptibility to B. cinerea than long day plants. In addition, when we entrain the biological clock in different photoperiods, the LD 30h photoperiod plants displayed similar responses as those in the SD 24h photoperiod. The data indicates that day length is not responsible for the control of plant immunity; it is the ability of light to entrain the biological clock that is important. Together, the data strongly support the conclusion that the circadian clock plays a role in plant defense regulation.
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Books on the topic "Clock control"

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Vogel, Nancy. Furloughs in round-the-clock operations: Savings are illusory. Sacramento, CA: Senate Publications & Flags, 2009.

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Coleman, Richard M. The 24-hour business: Maximizing productivity through round-the-clock operations. New York: AMACOM, 1995.

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Freely, Maureen. Pandora's clock: Understanding our fertility : the choices we face over contraception, pregnancy, genetic screening, abortion and infertility. London: Heinemann, 1993.

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Freely, Maureen. Pandora's clock: Understanding our fertility - the choices we face over family planning, pregnancy, abortion, genetic screening and infertility. London: Cedar, 1994.

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Lee, Kang B. 2004 Conference on IEEE-1588, standard for a precision clock synchronization protocol for networked measurement and control systems. [Gaithersburg, Md.]: U.S. Dept. of Commerce, Technology Administration, National Institute of Standards and Technology, 2004.

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Stromboli Conference on Aging and Cancer (3rd 1993). The aging clock: The pineal gland and other pacemakers in the progression of aging and carcinogenesis : Third Stromboli Conference on Aging and Cancer. New York, N.Y: New York Academy of Sciences, 1994.

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Walter, Pierpaoli, Regelson William, and Fabris N, eds. The aging clock: The pineal gland and other pacemakers in the progression of aging and carcinogenesis : Third Stromboli Conference on Aging and Cancer. New York, N.Y: New York Academy of Sciences, 1994.

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The Mac made easy. Berkeley, Calif: Osborne McGraw-Hill, 1992.

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Dittus, Hansjorg, Claus Lammerzahl, and Slava G. Turyshev, eds. Lasers, Clocks and Drag-Free Control. Berlin, Heidelberg: Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-34377-6.

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Leon, Kreitzman, ed. Rhythms of life: The biological clocks that control the daily lives of every living thing. London: Profile Books, 2004.

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Book chapters on the topic "Clock control"

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Weik, Martin H. "indirect clock control." In Computer Science and Communications Dictionary, 768. Boston, MA: Springer US, 2000. http://dx.doi.org/10.1007/1-4020-0613-6_8864.

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Weik, Martin H. "direct clock control." In Computer Science and Communications Dictionary, 419. Boston, MA: Springer US, 2000. http://dx.doi.org/10.1007/1-4020-0613-6_5124.

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Thiriet, Marc. "Circadian Clock." In Control of Cell Fate in the Circulatory and Ventilatory Systems, 329–56. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4614-0329-6_5.

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Ruiter, Marieke, Ruud M. Buijs, and Andries Kalsbeek. "Biological Clock Control of Glucose Metabolism." In Neuroendocrine Correlates of Sleep/Wakefulness, 87–117. Boston, MA: Springer US, 2006. http://dx.doi.org/10.1007/0-387-23692-9_5.

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Deng, Wei, Peter Yun, Yi Zhang, Jiehua Chen, and Sihong Gu. "Embedded Control System for Atomic Clock." In Advances in Intelligent and Soft Computing, 61–67. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-27708-5_9.

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Bernadsky, Mikhail, and Rajeev Alur. "Symbolic Analysis for GSMP Models with One Stateful Clock." In Hybrid Systems: Computation and Control, 90–103. Berlin, Heidelberg: Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-71493-4_10.

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Wang, Yang. "A Novel Clock Synchronization Method Design in Wireless Sensor Network System." In Future Control and Automation, 213–20. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-31006-5_26.

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Xu, Chongming, Xuejun Wu, and Erwin Brüning. "Unified Formula for Comparison of Clock Rates and Its Applications." In Lasers, Clocks and Drag-Free Control, 181–92. Berlin, Heidelberg: Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-34377-6_7.

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Ishizaki, Hironori, Akira Mizoguchi, and Mariko Fujishita. "Circadian-Clock Control of Hormone Secretion inSamia Cynthia ricini." In Ciba Foundation Symposium 104 - Photoperiodic Regulation of Insect and Molluscan Hormones, 136–49. Chichester, UK: John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470720851.ch9.

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Zenner, Erik. "On the Efficiency of the Clock Control Guessing Attack." In Lecture Notes in Computer Science, 200–212. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/3-540-36552-4_14.

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Conference papers on the topic "Clock control"

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Bueno, Átila Madureira, Angelo Marcelo Tusset, Diego Paolo Ferruzzo Correa, José Roberto Castilho Piqueira, and José Manoel Balthazar. "Comparing LQG/LTR and the SDRE Techniques for Hybrid Fully-Connected PLL Network Control." In ASME 2013 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/detc2013-12649.

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Synchronization plays an important role in telecommunication systems and integrated circuits. The Master-Slave is a commonly used strategy for clock signal distribution. However, due to the wireless networks development and the higher operation frequency of integrated circuits, the Mutually-Connected clock distribution strategies are becoming important, and the Fully-Connected strategy appears as a convenient engineering solution. The main drawback of the Fully-Connected architecture is the definition of control algorithms that assure the stability of the network sinchronization. In hybrid synchronization techniques groups of nodes synchronized by the Fully-Connected architecture are synchronized with network master clocks by using the Master-Slave tecnique. In this arrangement, if a route of clock signal distribution becomes inoperative, the group of Fully-Connected nodes retain for some time the original phase and frequency received from the network. The Fully-Connected architecture complexity imposes difficulties to satisfy both stability and performance requirements in the control system design. For that reason the multi-variable LQG/LTR and the SDRE control techniques are applied in order to fulfill both stability and performance requirements. The performance of both techniques are compared, and the results seems to confirm the improvement in the transient response and in the precision of the clock distribution process.
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Xiong, Nan, and Minrui Fei. "Simulation of gossip averaging based clock synchronization protocol for wireless sensor networks." In 2016 UKACC 11th International Conference on Control (CONTROL). IEEE, 2016. http://dx.doi.org/10.1109/control.2016.7737623.

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Hollos, Adam Erik, and Tamas Kovacshazy. "Improved reference clock connection interface for prototype IEEE 1588 master clocks." In 2018 19th International Carpathian Control Conference (ICCC). IEEE, 2018. http://dx.doi.org/10.1109/carpathiancc.2018.8399658.

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Frueholz, R. P., and J. C. Camparo. "A Rubidium Clock Model." In 39th Annual Symposium on Frequency Control. IEEE, 1985. http://dx.doi.org/10.1109/freq.1985.200815.

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Wei, Liang, Eliyahu Danny, Ilchenko Vladimir, Savchenkov Anatoliy, Matsko Andrey, and Maleki Lute. "All-Optical Micro-Clock." In 2014 IEEE International Frequency Control Symposium (FCS). IEEE, 2014. http://dx.doi.org/10.1109/fcs.2014.6859854.

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Ravi, S., Suyash Trehan, Mohit Jain, and Harish M. Kittur. "High Performance Clock Path elements for Clock Skew reduction." In 2019 2nd International Conference on Intelligent Computing, Instrumentation and Control Technologies (ICICICT). IEEE, 2019. http://dx.doi.org/10.1109/icicict46008.2019.8993375.

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Peca, Marek, Vojtech Michalek, and Michael Vacek. "Clock composition by wiener filtering illustrated on two atomic clocks." In 2013 Joint European Frequency and Time Forum & International Frequency Control Symposium (EFTF/IFC). IEEE, 2013. http://dx.doi.org/10.1109/eftf-ifc.2013.6702293.

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Plantard, C., P. M. Mbaye, and F. Vernotte. "Composite clock including a Cs clock, a H-maser clock and a VCO." In 2009 Joint Meeting of the European Frequency and Time Forum (EFTF) and the IEEE International Frequency Control Symposium (FCS). IEEE, 2009. http://dx.doi.org/10.1109/freq.2009.5168167.

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Kovacshazy, Tamas. "Synchronization performance evaluation of reference clock connection methods for IEEE 1588 master clocks." In 2015 16th International Carpathian Control Conference (ICCC). IEEE, 2015. http://dx.doi.org/10.1109/carpathiancc.2015.7145082.

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Wagner, John, Cecil Huey, and Katie Knaub. "Clock Mechanism Fundamentals for Education: Modeling and Analysis." In ASME 2008 Dynamic Systems and Control Conference. ASMEDC, 2008. http://dx.doi.org/10.1115/dscc2008-2100.

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Time keeping devices have been designed, fabricated, and widely deployed throughout history to regulate daily functions including commerce and transportation. In addition, horology offers a catalog of mankind’s innovation and demonstrates important scientific and engineering concepts. The investigation and analysis of clock systems from a mechatronics perspective illustrates the evolution of gear systems, feedback control, and transformation of energy for time measurement. In this paper, the operational behavior of an eight day mechanical clock has been studied through mathematical models, numerical simulation, and computer animation. The classroom exploration of time keeping mechanisms offers practical applications of physical principles.
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Reports on the topic "Clock control"

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Weinberg, Robert A. Control of the Mammary Cell Cycle Clock By Estrogen and Progesterone. Fort Belvoir, VA: Defense Technical Information Center, August 1997. http://dx.doi.org/10.21236/ada338694.

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Lee, K. B. Workshop on IEEE-1588, standard for a precision clock synchronization protocol for networked measurement and control systems. Gaithersburg, MD: National Institute of Standards and Technology, 2003. http://dx.doi.org/10.6028/nist.ir.7070.

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Green, Pamela J. Regulated mRNA Decay in Arabidopsis: A global analysis of differential control by hormones and the circadian clock. Office of Scientific and Technical Information (OSTI), March 2010. http://dx.doi.org/10.2172/973679.

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Lee, Kang B., and John C. Eidson. 2004 conference on IEEE 1588, standard for a precision clock synchronization protocol for networked measurement and control systems. Gaithersburg, MD: National Institute of Standards and Technology, 2004. http://dx.doi.org/10.6028/nist.ir.7192.

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Lee, Kang B., john C. Eidson, Hans Weibel, and Dirk Mohl. Proceeding of the 2005 conference on IEEE 1588 standard for a precision clock synchronization protocol for networked measurement and control systems. Gaithersburg, MD: National Institute of Standards and Technology, 2005. http://dx.doi.org/10.6028/nist.ir.7302.

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Geib, Kent Martin, Gregory Merwin Peake, Joel Robert Wendt, Darwin Keith Serkland, and Gordon Arthur Keeler. VCSEL polarization control for chip-scale atomic clocks. Office of Scientific and Technical Information (OSTI), January 2007. http://dx.doi.org/10.2172/902214.

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