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Relevant bibliographies by topics / Clonal expansion

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Academic literature on the topic 'Clonal expansion'

Author: Grafiati

Published: 4 June 2021

Last updated: 4 March 2023

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Journal articles on the topic "Clonal expansion"

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Nowak, Martin A., Franziska Michor, and Yoh Iwasa. "Genetic instability and clonal expansion." Journal of Theoretical Biology 241, no. 1 (2006): 26–32. http://dx.doi.org/10.1016/j.jtbi.2005.11.012.

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Masuko, K., S. Kato, M. Hagihara, et al. "Stable clonal expansion of T cells induced by bone marrow transplantation." Blood 87, no. 2 (1996): 789–99. http://dx.doi.org/10.1182/blood.v87.2.789.bloodjournal872789.

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The immune mechanisms of T cells regeneration after bone marrow transplantation (BMT) and the factors maintaining allogeneic marrow graft in the host are still unknown. To pursue this issue, we analyzed T-cell clonality of peripheral blood lymphocytes (PBLs) in BMT recipients, using reverse transcription polymerase chain reaction with T-cell receptor (TCR) V beta gene segment-specific primers and single- strand conformation polymorphism. PBLs from patients and donors showed a heterogeneous T-cell population with oligoclonal accumulations of CD8+ T cells. When PBLs were cultured in HLA-matched

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Mele, Giuseppe, Marilena Greco, Maria Rosaria Coppi, et al. "Small-Sized Clone of T Cells in Multiple Myeloma Patient after Auto-SCT: T-LGL Leukemia Type or Clonal T-Cell Aberration?" Case Reports in Hematology 2013 (2013): 1–3. http://dx.doi.org/10.1155/2013/417353.

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Second cancers and particularly postransplant lymphoproliferative disorders (PTLDs) are extremely rare in patients undergoing autologous peripheral blood stem cell transplantation (auto-SCT). We report the case of clonally rearranged T-cell expansion which occurred after auto-SCT for Multiple Myeloma (MM). Does asymptomatic clonal T-cell large granular lymphocytic proliferation, in our experience, represent either a secondary cancer after auto-SCT or clonal T cell aberration or derive from expansion of coexisting undetected small-sized clone of T cells?

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Iwasa, Yoh, Martin A. Nowak, and Franziska Michor. "Evolution of Resistance During Clonal Expansion." Genetics 172, no. 4 (2006): 2557–66. http://dx.doi.org/10.1534/genetics.105.049791.

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Kakiuchi, Nobuyuki, and Seishi Ogawa. "Clonal expansion in non-cancer tissues." Nature Reviews Cancer 21, no. 4 (2021): 239–56. http://dx.doi.org/10.1038/s41568-021-00335-3.

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Resar, Linda M., and Robert A. Brodsky. "“Let”-ing go with clonal expansion?" Blood 117, no. 22 (2011): 5788–90. http://dx.doi.org/10.1182/blood-2011-04-346668.

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Joachims, Michelle, Kerry Leehan, Christina Lawrence, et al. "Expansions of salivary gland CD4+ T cells from Sjögren’s syndrome patients: single-cell repertoire analysis and correlation with clinical measures of disease (HUM3P.255)." Journal of Immunology 194, no. 1_Supplement (2015): 121.15. http://dx.doi.org/10.4049/jimmunol.194.supp.121.15.

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Abstract Sjögren’s syndrome is a systemic rheumatic disorder characterized by dry eyes, dry mouth and T cell infiltration of exocrine gland tissue. To determine the extent of salivary gland (SG) CD4+ memory T cell clonal expansion and whether these expansions are as frequent in peripheral blood (PB), a multiplex PCR method was used to amplify both the α and β T cell receptor (TCR) sequences from memory CD4+ T cells sorted from SG lip biopsy tissue and PB of 10 primary Sjögren’s syndrome patients. Over 3,000 TCR sequences were obtained from 50-115 (median 91) individual SG and 75-121 (median 10

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Gurung, S., D. P. G. Short, Z. K. Atallah, and K. V. Subbarao. "Clonal Expansion of Verticillium dahliae in Lettuce." Phytopathology® 104, no. 6 (2014): 641–49. http://dx.doi.org/10.1094/phyto-10-13-0282-r.

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Few studies in population biology have documented how structure and diversity of pathogens evolve over time at local scales. With the historical samples of Verticillium dahliae available from lettuce, we investigated the structure and diversity of this pathogen in time and space. Three hundred twenty-nine V. dahliae isolates from lettuce fields collected over 18 years were characterized with polymorphic microsatellite markers and polymerase chain reaction tests for race and mating type. Genetic variation within and among commercial lettuce fields in a single season was also investigated using

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Schmidt, Amber, Rami S. Komrokji, Jeffrey S. Painter, et al. "Positive Impact of T-Cell Clonal Expansion On Overall Survival in Patients with High-Risk Myelodysplastic Syndromes." Blood 114, no. 22 (2009): 1572. http://dx.doi.org/10.1182/blood.v114.22.1572.1572.

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Abstract Abstract 1572 Poster Board I-598 Background Suppression of clonal T-cells has been linked to immunosuppressive therapy response in patients with lower-risk MDS and with other forms of autoimmune bone marrow failure suggesting that T-cell clonal expansion is pathogenic to bone marrow hematopoiesis. It is possible, however, that clonal T-cells expanding in response to leukemia-associated antigens (LAA) may ultimately suppress tumor progression through immunosurveillance. Our group previously identified clonal T-cell expansion in 50% of MDS patients (n=52) compared to 5% in age matched c

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Zacharakis, Nikolaos, Emilia Oleszak, Nicholas Klaiber, Allen Myers та Chris Platsoucas. "Clonally expanded α-chain TCR transcripts are present in skin biopsies of patients with Systemic Sclerosis (135.33)". Journal of Immunology 184, № 1_Supplement (2010): 135.33. http://dx.doi.org/10.4049/jimmunol.184.supp.135.33.

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Abstract Systemic Sclerosis (SSc) is a chronic autoimmune disease characterized by excessive deposition of collagen in the skin and many internal organs, production of autoantibodies and microvascular alterations. Previous studies in our laboratory have demonstrated the presence of high proportions of identical β-chains TCR transcripts in skin biopsies from patients with SSc of recent onset, suggestive of clonal expansions of T cells. To identify the antigen(s) recognized by clonally expanded T cells in SSc biopsies, we examine the clonality of the α-chain TCR transcripts in patients with SSc.

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Dissertations / Theses on the topic "Clonal expansion"

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Ventayol-García, Tania. "Clonal expansion in the human upper gastrointestinal tract." Thesis, Queen Mary, University of London, 2013. http://qmro.qmul.ac.uk/xmlui/handle/123456789/8694.

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The high incidence of gastrointestinal cancers in the general population and the presence of premalignant dysplastic precursor lesions in the gastrointestinal tract make the gastrointestinal tract an ideal environment to study cancer clonality and clonal expansion. Background: Intestinal metaplastic (IM) glands in the human stomach are clonal, contain multiple stem cells and spread by fission. This mechanism of gland fission causes field cancerisation. We hypothesised that gastric adenocarcinoma (GA) progresses through a series of genetic events arising from a founder mutation. A process analo

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Campbell, Georgia Elizabeth. "Investigating the mechanism of clonal expansion of deleted mtDNA species." Thesis, University of Newcastle upon Tyne, 2014. http://hdl.handle.net/10443/2519.

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Mitochondrial DNA deletions are a primary cause of inherited and sporadic mitochondrial disease, whilst somatic mtDNA deletions contribute to the focal respiratory chain deficiency observed in post-mitotic cells associated with ageing and neurodegenerative disorders. As mtDNA deletions only cause cellular pathology at high levels of heteroplasmy, an mtDNA deletion formed within a cell must accumulate by a process known as clonal expansion to levels which result in biochemical dysfunction. The mechanism driving clonal expansion remains uncertain; this research aimed to investigate clonally expa

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Lecault, Véronique. "Microfluidic cell culture arrays for clonal expansion and characterization of mammalian cells." Thesis, University of British Columbia, 2012. http://hdl.handle.net/2429/43667.

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Jawad, Noor. "Exploring stem cell dynamics, clonal expansion and pseudopolyps in inflammatory bowel disease." Thesis, Queen Mary, University of London, 2015. http://qmro.qmul.ac.uk/xmlui/handle/123456789/9099.

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Inflammatory bowel disease (IBD) confers a high risk of development of colitis-associated colorectal cancer in patients with extensive colitis. Crypt fission is a mechanism of clonal expansion in the intestinal epithelium. Although fission is rare in the normal colon, many crypts in IBD patients are in the process of fission. Protumourigenic mutations can spread through the entire inflamed colon relatively quickly indicating that stem cell dynamics are altered in IBD. Some patients with IBD develop pseudopolyps as a result of mucosal ulceration and epithelial regeneration. The aim of this PhD

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Humphries, A. "Stem cell biology and clonal expansion in normal and adenomatous human intestinal crypts." Thesis, University College London (University of London), 2012. http://discovery.ucl.ac.uk/1348481/.

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Gastrointestinal cancer is thought to be primarily a disease of stem cells, whereby a tumorigenic stem cell clone can expand within an individual colonic crypt and then within the epithelium to form an adenoma - the pre-malignant lesion of the colon. However, data demonstrating stem cell populations and the dynamics of clonal expansion in human intestinal crypts is lacking. Naturally occurring, somatic clonal mutations in mitochondrial DNA were used to identify the progeny of a putative single stem cell lineage within crypts; this allowed the visualization of putative stem cell clones arising

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Alraies, Amr. "Characterisation of clonal dental pulp progenitors and the effects of in vitro expansion and hydrogen peroxide." Thesis, Cardiff University, 2013. http://orca.cf.ac.uk/53906/.

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Dental pulp progenitor cells (DPPCs) are among many stem cell sources potentially beneficial for tissue engineering. DPPCs offer advantages over other mesenchymal stem cell sources, due to their accessibility and multi-lineage differentiation. However, distinct DPPC clones exist within dental pulp, with contrasting proliferative/regenerative capabilities. This is a key consideration for the exploitation of DPPCs, in terms of the abilities of isolated clones to undergo sufficient in vitro proliferative expansion, while maintaining their regenerative potential. This Thesis supports the heterogen

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Carr, J. M. "The role of BCL6b and CD27 in the clonal expansion of CD8+T cells without effector differentiation." Thesis, University of Cambridge, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.597306.

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It has been proposed that a self-renewing, “stem-cell” like stage of CD8<sup>+</sup> T cell development exists in which a potential for proliferation and clonal expansion is maintained without effector differentiation. The possibility that the transcriptional repressor BCL6b, or costimulation via a receptor of the tumour necrosis factor superfamily of proteins, was responsible for allowing the expansion of CD8<sup>+</sup> T cells without the development of effector properties was assessed. This study demonstrates that BCL6b partially blocked the IL-2 induced increase in CD8<sup>+</sup> T cell

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Roufosse, Florence. "Clonal expansion of helper type 2 lymphocytes as a cause of the hypereosiophilic syndrome: clinical and immunological aspects." Doctoral thesis, Universite Libre de Bruxelles, 2001. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/211498.

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Ide, Jennifer C. "Effects of Macrophage-conditioned Medium on Preadipocyte Cyclin-dependent Kinase Regulation During Adipogenesis." Thesis, Université d'Ottawa / University of Ottawa, 2011. http://hdl.handle.net/10393/19752.

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Macrophage-conditioned medium (MacCM) inhibits the differentiation of rodent and human preadipocytes. Previous studies report that murine J774A.1-MacCM inhibits clonal expansion (early required phase of adipogenesis), including Rb phosphorylation. I hypothesized that MacCM induced alterations in cyclins and/or cyclin-dependent kinases (CDKs) were responsible for impairing Rb phosphorylation. My first objective was to assess the effect of J774A.1-MacCM on CDK4, CDK2, and their regulatory cyclins. Murine 3T3-L1 preadipocytes were differentiated with control medium or J774A.1-MacCM. Expression of

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Dumont, Alain. "Insights into the dynamics of T cell clonal expansion and the functional heterogeneity of memory CD4 T lymphocytes using superantigens." Thesis, McGill University, 2004. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=84235.

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Superantigens trigger the polyclonal activation of human T cells. We exploited this property to gain insight into the mechanisms governing CD4 T cell expansion and to study the functional heterogeneity of naive and memory CD4 T cell subsets. We show that the amount of TCR ligand affects the evolution of a T cell response in two ways: by shaping the diversity of the T cell population recruited in the proliferative pool and by affecting the progression of these precursors into cell cycle. These two processes characterize a hierarchy of recruitment of cells that strongly correlates with th

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Books on the topic "Clonal expansion"

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Huffman, David W. Regeneration of salal: Seedling establishment and the effects of overstory stand density on clonal morphology and expansion. 1992.

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Misbah, Siraj. Immunosuppressive therapy and therapeutic monoclonal antibodies. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0302.

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The term immunosuppressive therapy encompasses all forms of treatment that dampens function of the recipient’s immune system, with a view to controlling severe autoimmune, inflammatory, or allergic disease. The predominant targets of these agents are T-lymphocytes with multiple downstream effects, including containment of T-cell activation, inhibition of cytokine production, restriction of clonal expansion, and varying degrees of suppression of B-cell function. This chapter reviews the clinical use of monoclonal antibodies and other immunosuppressive agents, and their mechanisms of action.

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Rodríguez Henao, Eberto, Luis Carlos Grajales Guzmán, Julián Ossa Gutiérrez, and Dubert Yamil Cañar Serna. Producción clonal de material de siembra de guayabo mediante la técnica de injertación en púa termina. Corporación Colombiana de Investigación Agropecuaria (Agrosavia), 2022. http://dx.doi.org/10.21930/agrosavia.manual.7405262.

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La creciente demanda de material de propagación de guayabo con calidad genética, física, fisiológica y sanitaria en el país ha dinamizado la expansión de este cultivo y ha generado interés en su manejo técnico y procedimental, tanto para las variedades mejoradas como para el material regional seleccionado por el agricultor. Sin embargo, el productor de semilla de guayabo afronta diversos retos que han dificultado el éxito en la cadena de abastecimiento y en la logística de producción del material de siembra, lo que, a su vez, ha limitado el potencial de esta especie frutícola en calidad y dese

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Ronco, Pierre M. Kidney involvement in plasma cell dyscrasias. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0150.

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Monoclonal proliferations of the B-cell lineage are characterized by abnormal and uncontrolled expansion of a single clone of B cells at different maturation stages, with a variable degree of differentiation to immunoglobulin-secreting plasma cells. Therefore, they are usually associated with the production and secretion in blood of a monoclonal immunoglobulin and/or a fragment thereof which may become deposited in tissues. These deposits can take the form of casts (in myeloma cast nephropathy), crystals (in myeloma-associated Fanconi syndrome), fibrils (in light-chain and exceptional heavy-ch

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Book chapters on the topic "Clonal expansion"

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Tarapore, Danesh, Anders Lyhne Christensen, Pedro U. Lima, and Jorge Carneiro. "Clonal Expansion without Self-replicating Entities." In Lecture Notes in Computer Science. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-33757-4_15.

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Chen, Chao W., and Assad Moini. "Cancer Dose-Response Models Incorporating Clonal Expansion." In Scientific Issues in Quantitative Cancer Risk Assessment. Birkhäuser Boston, 1990. http://dx.doi.org/10.1007/978-1-4684-9218-7_9.

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Shlomchik, M. J., S. Litwin, and M. Weigert. "The Influence of Somatic Mutation on Clonal Expansion." In Progress in Immunology. Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-83755-5_55.

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Nishimura, Jun-ichi, Yuzuru Kanakura, Russell E. Ware, et al. "Serial Analysis of Clonal Expansion in PNH by Flow Cytometry." In Paroxysmal Nocturnal Hemoglobinuria and Related Disorders. Springer Japan, 2003. http://dx.doi.org/10.1007/978-4-431-67867-0_22.

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Inoue, Norimitsu, Tomohisa Izui, Maki Kuwayama, et al. "A Possible Intrinsic Mechanism for Clonal Expansion of PNH Abnormal Cells." In Paroxysmal Nocturnal Hemoglobinuria and Related Disorders. Springer Japan, 2003. http://dx.doi.org/10.1007/978-4-431-67867-0_10.

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Chen, Feng. "Quantification of Clonal Expansion of Hepatocytes in Normal and Injured Liver." In Methods in Molecular Biology. Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2557-6_15.

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De Rooij, Dirk G. "Morphometric Description of Spermatogonial Stem Cells and Expansion of Their Clonal Derivatives." In Male Germline Stem Cells: Developmental and Regenerative Potential. Humana Press, 2010. http://dx.doi.org/10.1007/978-1-61737-973-4_4.

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Sun, Yifei, Maoguo Gong, Lin Hao, and Licheng Jiao. "Clonal Selection Algorithm with Search Space Expansion Scheme for Global Function Optimization." In Lecture Notes in Computer Science. Springer Berlin Heidelberg, 2006. http://dx.doi.org/10.1007/11881070_111.

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Heydenrych, Mark, and Elizabeth Marie Ehlers. "PARA-Antibodies: An Immunological Model for Clonal Expansion Based on Bacteriophages and Plasmids." In Advances in Intelligent Systems and Computing. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-27400-3_16.

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Bottomly, K., M. Luqman, J. Murray, J. West, A. Woods, and S. Carding. "Clonal Expansion and Differentiation to Effector Function in Normal CD4 T Cell Subpopulations." In Progress in Immunology. Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-83755-5_80.

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Conference papers on the topic "Clonal expansion"

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Ishida, Yoshihiro, Nobuyuki Kakiuchi, Yoichi Fujii, et al. "Abstract 2675: Clonal expansion of skin keratinocyte." In Proceedings: AACR Annual Meeting 2021; April 10-15, 2021 and May 17-21, 2021; Philadelphia, PA. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.am2021-2675.

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OR-GUIL, MICHAL, FABIO LUCIANI, and JORGE CARNEIRO. "CLONAL EXPANSION OF CYTOTOXIC T CELL CLONES: THE ROLE OF THE IMMUNOPROTEASOME." In Proceedings of the International Symposium on Mathematical and Computational Biology. WORLD SCIENTIFIC, 2006. http://dx.doi.org/10.1142/9789812773685_0012.

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Strauss, Bryan E., Daniela Zanatta, and Rodrigo B. de Aguiar. "Abstract 2843: A quantitative sequencing based method for the monitoring of clonal expansion." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-2843.

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Tieniber, Andrew, Andrew Hanna, Benjamin Medina, et al. "Abstract P050: Oncogenic kinase therapy restricts CD8 T cell differentiation and clonal expansion." In Abstracts: AACR Virtual Special Conference: Tumor Immunology and Immunotherapy; October 5-6, 2021. American Association for Cancer Research, 2022. http://dx.doi.org/10.1158/2326-6074.tumimm21-p050.

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Saul, Louise, Kristina M. Ilieva, Heather J. Bax, et al. "Abstract A116: IgG antibody switching and clonal expansion in melanoma and normal skin microenvironments." In Abstracts: Second CRI-CIMT-EATI-AACR International Cancer Immunotherapy Conference: Translating Science into Survival; September 25-28, 2016; New York, NY. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/2326-6066.imm2016-a116.

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Korenjak, Michael, Hana Huskova, Maude Ardin, et al. "Abstract 5154: Modeling cancer driver-like events in barrier bypass-clonal expansion in vitro assays." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-5154.

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Worssam, Matt. "BS31 VSMC contribution to neointimal lesions arises from the clonal expansion of few primed cells." In British Cardiovascular Society Virtual Annual Conference, ‘Cardiology and the Environment’, 7–10 June 2021. BMJ Publishing Group Ltd and British Cardiovascular Society, 2021. http://dx.doi.org/10.1136/heartjnl-2021-bcs.229.

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Penter, Livius, Kerstin Dietze, Felix Aigner, Lars Bullinger, Thomas Blankenstein, and Leo Hansmann. "Abstract 4679: Rectal cancer-infiltrating T cells show clonal expansion associated with spatially restricted tolerogenic phenotypes." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-4679.

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Yan, Bowen, and Yi Qiu. "Abstract 3023: Clonal expansion of TP53 mutated cells is associated with chemoresistance in acute myeloid leukemia." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-3023.

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Snyder, M. E., T. Tabib, I. Popescu, et al. "Clonal Expansion and Persistence of Recipient-Derived Alloreactive T Cells in the Lungs of Transplant Recipients." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a1321.

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Reports on the topic "Clonal expansion"

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Cohen, Yuval, Christopher A. Cullis, and Uri Lavi. Molecular Analyses of Soma-clonal Variation in Date Palm and Banana for Early Identification and Control of Off-types Generation. United States Department of Agriculture, 2010. http://dx.doi.org/10.32747/2010.7592124.bard.

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Date palm (Phoenix dactylifera L.) is the major fruit tree grown in arid areas in the Middle East and North Africa. In the last century, dates were introduced to new regions including the USA. Date palms are traditionally propagated through offshoots. Expansion of modern date palm groves led to the development of Tissue Culture propagation methods that generate a large number of homogenous plants, have no seasonal effect on plant source and provide tools to fight the expansion of date pests and diseases. The disadvantage of this procedure is the occurrence of off-type trees which differ from t

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Barg, Rivka, Erich Grotewold, and Yechiam Salts. Regulation of Tomato Fruit Development by Interacting MYB Proteins. United States Department of Agriculture, 2012. http://dx.doi.org/10.32747/2012.7592647.bard.

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Background to the topic: Early tomato fruit development is executed via extensive cell divisions followed by cell expansion concomitantly with endoreduplication. The signals involved in activating the different modes of growth during fruit development are still inadequately understood. Addressing this developmental process, we identified SlFSM1 as a gene expressed specifically during the cell-division dependent stages of fruit development. SlFSM1 is the founder of a class of small plant specific proteins containing a divergent SANT/MYB domain (Barg et al 2005). Before initiating this project,

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Banai, Menachem, and Gary Splitter. Molecular Characterization and Function of Brucella Immunodominant Proteins. United States Department of Agriculture, 1993. http://dx.doi.org/10.32747/1993.7568100.bard.

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The BARD project was a continuation of a previous BARD funded research project. It was aimed at characterization of the 12kDa immunodominant protein and subsequently the cloning and expression of the gene in E. coli. Additional immunodominant proteins were sought among genomic B. abortus expression library clones using T-lymphocyte proliferation assay as a screening method. The 12kDa protein was identified as the L7/L12 ribosomal protein demonstrating in the first time the role a structural protein may play in the development of the host's immunity against the organism. The gene was cloned fro

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Friedman, Haya, Julia Vrebalov, James Giovannoni, and Edna Pesis. Unravelling the Mode of Action of Ripening-Specific MADS-box Genes for Development of Tools to Improve Banana Fruit Shelf-life and Quality. United States Department of Agriculture, 2010. http://dx.doi.org/10.32747/2010.7592116.bard.

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Fruit deterioration is a consequence of a genetically-determined fruit ripening and senescence programs, in which developmental factors lead to a climacteric rise of ethylene production in ethylene-sensitive fruits such as tomato and banana. Breeding of tomato with extended fruit shelf life involves the incorporation of a mutation in RIN, a MADS-box transcription factor participating in developmental control signalling of ripening. The RIN mode of action is not fully understood, and it may be predicted to interact with other MADS-box genes to execute its effects. The overall goal of this study

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