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Books on the topic 'CNS damage'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

Christian, Alzheimer, ed. Molecular and cellular biology of neuroprotection in the CNS. Kluwer Academic / Plenum Publishers, 2003.

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2

Dupont, Anne-Sylvie. Le dommage écologique: Le rôle de la responsabilité civile en cas d'atteinte au milieu naturel. Schulthess, 2005.

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3

1923-, Cairns John, ed. Rehabilitating damaged ecosystems. 2nd ed. Lewis Publishers, 1995.

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4

Denny, Cleve, and Barbara Blakistone. Guidelines for evaluation and disposition of damaged food containers--cans and glass: For use by canners, distributors, retailers, and those assessing food container damage. 4th ed. National Food Processors Association, 1999.

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5

Eboma, Mosilo. Enquête jurisprudentielle pour une indemnisation judiciaire équitable: Cas des accidents de circulation. Presses universitaires du Congo, 2003.

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6

Letang, Joseph Brunet. En appel: Charles Legault dit Delorier, appellant [sic] and [sic] Joseph Brunet dit Letang, intimé : cas de l'intimé. s.n., 1986.

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7

1827-1896, Fabre Edouard C., and Fabrique de Notre-Dame (Montréal, Québec), eds. À Sa Grandeur Mgr. Edouard Chs. Fabre, évêque de Montréal, etc. etc. s.n., 1985.

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8

author, Mendouga Fidélie, and Nkou Abina Pierre author, eds. Sociologie critique des indemnisations au Cameroun: Cas de Lom Pangar, Mekin et Kribi. L'Harmattan, 2021.

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9

Seed, Philip. Developing holistic education: A case study of Raddery School for Emotionally Damaged Children. Falmer Press, 1992.

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10

Workshop on the Indemnification of Nuclear Damage in the Event of a Nuclear Accident (2001 Paris, France). Indemnification of damage in the event of a nuclear accident: Workshop proceedings, Paris, France, 26-28 November 2001 = Indemnisation des dommages en cas d'accident nucléaire : compte rendu d'un atelier, Paris, France, 26-28 novembre 2001. Nuclear Energy Agency, Organisation for Economic Co-operation and Development, 2003.

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11

Naritomi, H., and D. W. Krieger, eds. Clinical Recovery from CNS Damage. S. Karger AG, 2013. http://dx.doi.org/10.1159/isbn.978-3-318-02309-1.

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12

Moftah, Marie, and Emmanuel Moyse, eds. CNS Recovery after Structural and/or Physiological/Psychological Damage. Frontiers Media SA, 2016. http://dx.doi.org/10.3389/978-2-88945-040-4.

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13

Molecular and cellular biology of neuroprotection in the CNS. Kluwer Academic / Plenum Publishers, 2003.

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14

Alzheimer, Christian. Molecular and Cellular Biology of Neuroprotection in the CNS. Springer, 2003.

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15

Peterson, Patti L., and John W. Phillis. Novel Therapies for CNS Injuries: Rationales and Results. CRC, 1995.

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16

Alzheimer, Christian. Molecular and Cellular Biology of Neuroprotection in the CNS. Springer, 2012.

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17

Alzheimer, Christian. Molecular and Cellular Biology of Neuroprotection in the CNS. Springer London, Limited, 2012.

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18

Petzold, Axel. Tissue Biomarkers and Neuroprotection. Edited by David L. Reich, Stephan Mayer, and Suzan Uysal. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190280253.003.0004.

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This chapter reviews the evidence for using biomarkers to measure damage to the central nervous system (CNS) in neurocritical care and perioperative medicine. A conceptual framework is provided to guide the optimal timing of blood, cerebrospinal fluid, and structural imaging biomarker assessment in relation to the onset of injury. A selection of well-validated, cell type–specific biomarkers of CNS tissue damage are reviewed, including their composition, biokinetics, and specificity for neurons, axons, astrocytes, and microglia. Each of these biomarkers will be reviewed in the pertinent clinica

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19

Filippi, Massimo, and Maria A. Rocca. Multiple Sclerosis: White Matter versus Gray Matter Involvement (The Cause of Disability in MS). Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0083.

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The classic view of multiple sclerosis (MS) as a chronic, inflammatory-demyelinating condition affecting solely the white matter (WM) of the central nervous system (CNS) has been challenged by the demonstration, from pathologic and magnetic resonance imaging (MRI) studies, of an extensive and diffuse involvement of the gray matter (GM). This observation has driven the application of modern MR technology and methods of analysis to quantify the extent and distribution of damage to the different compartments of the CNS, with the ultimate goal of improving our understanding of the factors associat

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20

Costello, Kathleen, and Scott D. Newsome. Symptoms of Multiple Sclerosis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0085.

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Symptoms of MS can be categorized as primary, secondary, or tertiary. Primary symptoms are those that are a direct result of CNS damage, such as fatigue, bladder dysfunction, visual disturbances, incoordination or imbalance, etc. Secondary symptoms are complications arising from these symptoms such as falls due to imbalance, spasticity or weakness, or UTI due to bladder retention. Tertiary symptoms are the social and vocational consequences of primary and secondary symptoms and can include divorce, job loss, financial hardship, and social isolation. Careful evaluation of these symptoms along w

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21

Columb, Malachy O. Local anaesthetic agents. Edited by Michel M. R. F. Struys. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0017.

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Local anaesthetic agents cause a pharmacologically induced reversible neuropathy characterized by axonal conduction blockade. They act by blocking the sodium ionophore and exhibit membrane stabilizing activity by inhibiting initiation and propagation of action potentials. They are weak bases consisting of three components: a lipophilic aromatic ring, a link, and a hydrophilic amine. The chemical link classifies them as esters or amides. Local anaesthetics diffuse through the axolemma as unionized free-base and block the ionophore in the quaternary ammonium ionized form. The speed of onset of b

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22

Presidential commissions and national security: The politics of damage control. Lynne Rienner Publishers, 2006.

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23

Le Couvent des oiseaux: Ces jeunes filles de bonne famille. Perrin, 1991.

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24

Levtchenko, Elena N., and Mirian C. Janssen. Cystinosis. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0339_update_001.

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Cystinosis is a rare autosomal recessive disease caused by mutations in the lysosomal cystine transporter cystinosin encoded by the CTNS gene (17p.13.2). Cystinosis is characterized by lysosomal cystine accumulation throughout the body with renal Fanconi syndrome being the most common presenting symptom of a multisystem disorder. It must be distinguished from cystinuria in which formation of cystine stones is the core problem. When left untreated, kidney dysfunction gradually progresses towards end-stage renal failure during the first 10 years of life. The advent of renal replacement therapy a

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25

Ronin, Marguerite, and Cosima Möller, eds. Instandhaltung und Renovierung von Straßen und Wasserleitungen von der Zeit der römischen Republik bis zur Spätantike. Nomos Verlagsgesellschaft mbH & Co. KG, 2019. http://dx.doi.org/10.5771/9783748900269.

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Roads, bridges, aqueducts and canals are amongst the physical infrastructures that allowed Roman dominance over the Empire, while meeting economic, social and strategic needs. Due to their structural role in the management and control of a territory, they must be examined in view of the “longue durée”, which necessarily raises the issue of their regular maintenance and occasional restoration. By studying the interactions between different political and administrative authorities, but also the involvement of private individuals, be they users or riverside occupants, the papers gathered in this

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26

Flood Risk Management. Taylor & Francis Group, 2019.

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27

Penning-Rowsell, Edmund C., and Matilda Becker. Flood Risk Management: Global Case Studies of Governance, Policy and Communities. Taylor & Francis Group, 2019.

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28

Penning-Rowsell, Edmund C., and Matilda Becker. Flood Risk Management: Global Case Studies of Governance, Policy and Communities. Taylor & Francis Group, 2019.

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29

Penning-Rowsell, Edmund C., and Matilda Becker. Flood Risk Management: Global Case Studies of Governance, Policy and Communities. Taylor & Francis Group, 2019.

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30

Penning-Rowsell, Edmund C., and Matilda Becker. Flood Risk Management: Global Case Studies of Governance, Policy and Communities. Taylor & Francis Group, 2019.

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31

Wilson, Barbara A., Jill Winegardner, and Fiona Ashworth. Life after Brain Injury: Survivors' Stories. Taylor & Francis Group, 2013.

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32

Wilson, Barbara A., Jill Winegardner, and Fiona Ashworth. Life after Brain Injury: Survivors' Stories. Taylor & Francis Group, 2013.

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33

Wilson, Barbara A., Jill Winegardner, and Fiona Ashworth. Life after Brain Injury: Survivors' Stories. Taylor & Francis Group, 2013.

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34

Wilson, Barbara A., Jill Winegardner, and Fiona Ashworth. Life after Brain Injury: Survivors' Stories. Taylor & Francis Group, 2013.

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35

Wilson, Barbara A., Jill Winegardner, and Fiona Ashworth. Life after Brain Injury: Survivors' Stories. Taylor & Francis Group, 2013.

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36

Life after Brain Injury: Survivors' Stories. Taylor & Francis Group, 2013.

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37

Seed, Philip. Developing Holistic Education. Taylor & Francis Group, 2018.

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38

Seed, Philip. Developing Holistic Education. Taylor & Francis Group, 2020.

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39

Seed, Philip. Developing Holistic Education. Taylor & Francis Group, 1992.

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40

Shaibani, Aziz. Pseudoneurologic Syndromes. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190661304.003.0022.

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The term functional has almost replaced psychogenic in the neuromuscular literature for two reasons. It implies a disturbance of function, not structural damage; therefore, it defies laboratory testing such as MRIS, electromyography (EMG), and nerve conduction study (NCS). It is convenient to draw a parallel to the patients between migraine and brain tumors, as both cause headache, but brain MRI is negative in the former without minimizing the suffering of the patient. It is a “software” and not a “hardware” problem. It avoids irritating the patient by misunderstanding the word psychogenic whi

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