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Dissertations / Theses on the topic 'Cochlea – Physiology'

Author: Grafiati

Published: 4 June 2021

Last updated: 19 February 2022

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1

Jäger, Wanje. "Physiological aspects of cochlear excitation and neurotransmitter release /." Stockholm, 1998. http://diss.kib.ki.se/1998/91-628-3294-8/.

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2

Cheng, Jun. "Signal processing approaches on otoacoustic emissions /." Stockholm, 2000. http://diss.kib.ki.se/2000/91-628-4058-4.

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3

Ku, Emery Mayon. "Modelling the human cochlea." Thesis, University of Southampton, 2008. https://eprints.soton.ac.uk/64535/.

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One of the salient features of the human cochlea is the incredible dynamic range it possesses—the loudest bearable sound is 10,000,000 times greater than the softest detectable sound; this is in part due to an active process. More than twelve thousand hairlike cells known as outer hair cells are believed to expand and contract in time to amplify cochlear motions. However, the cochlea’s response is more than just the sum of its parts: the local properties of outer hair cells can have unexpected consequences for the global behaviour of the system. One such consequence is the existence of otoacou

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4

Jaggers, Robert Maxwell. "Is Polyvinylidene diflouride (PVDF) film biocompatible in the Murine Cochlea?" Wright State University / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=wright1440944212.

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5

Lennan, George William Thomas. "Mechanoelectrical transduction by hair cells of the neonatal mouse in tissue culture." Thesis, University of Sussex, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.296617.

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6

McMahon, Catherine. "The mechanisms underlying normal spike activity of the primary afferent synapse in the cochlea and its dysfunction : an investigation of the possible mechanisms of peripheral tinnitus and auditory neuropathy." University of Western Australia. School of Biomedical and Chemical Sciences, 2004. http://theses.library.uwa.edu.au/adt-WU2003.0034.

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[Truncated abstract] One of the problems in researching tinnitus is that it has often been assumed that the physiological mechanisms underlying the tinnitus percept cannot be objectively measured. Nonetheless, it is generally accepted that the percept results from altered spontaneous neural activity at some site along the auditory pathway, although it is still debated whether it is produced by: synchronisation of activity of adjacent neurones; a change in the temporal pattern of activity of individual neurones; or an increase in the spontaneous firing rate per se. Similarly, it is possible tha

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7

O'Beirne, Greg A. "Mathematical modelling and electrophysiological monitoring of the regulation of cochlear amplification." University of Western Australia. School of Biomedical and Chemical Sciences, 2005. http://theses.library.uwa.edu.au/adt-WU2006.0115.

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[Truncated abstract] The cochlea presumably possesses a number of regulatory mechanisms to maintain cochlear sensitivity in the face of disturbances to its function. Evidence for such mechanisms can be found in the time-course of the recovery of CAP thresholds during experimental manipulations, and in observations of slow oscillations in cochlear micromechanics following exposure to low-frequency tones (the “bounce phenomenon”) and other perturbations. To increase our understanding of these oscillatory processes within the cochlea, and OHCs in particular, investigations into cochlear regulatio

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8

Zagaeski, Mark. "Information processing in the mammalian auditory periphery." Thesis, Boston University, 1991. https://hdl.handle.net/2144/37176.

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Thesis (Ph.D.)--Boston University<br>PLEASE NOTE: Boston University Libraries did not receive an Authorization To Manage form for this thesis or dissertation. It is therefore not openly accessible, though it may be available by request. If you are the author or principal advisor of this work and would like to request open access for it, please contact us at open-help@bu.edu. Thank you.<br>Inner hair cells (IHC) are the primary sensory cells of the mammalian cochlea. They transduce sound energy into a changing receptor potential which stimulates electrical activity in the Type I spiral ganglion

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9

Jing, Zhizi Verfasser], Nicola [Akademischer Betreuer] Strenzke, Tobias [Akademischer Betreuer] [Moser, Fred [Akademischer Betreuer] Wolf, and Martin [Akademischer Betreuer] Göpfert. "Sound Encoding in the Mouse Cochlea: Molecular Physiology and Optogenetic Stimulation / Zhizi Jing. Gutachter: Tobias Moser ; Fred Wolf ; Martin Göpfert. Betreuer: Nicola Strenzke." Göttingen : Niedersächsische Staats- und Universitätsbibliothek Göttingen, 2014. http://d-nb.info/1050288599/34.

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10

Markessis, Emily. "Development of an objective procedure allowing frequency selectivity measurements using the masking function of auditory steady state evoked potentials." Doctoral thesis, Universite Libre de Bruxelles, 2010. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/209990.

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<u><b>Introduction</u></b><p><p align = "justify">Les surdités cochléaires induisent, outre une audibilité réduite, une série de distorsions de la représentation neurale des sons. Deux des mécanismes à la base de ces distorsions sont d’une part une atteinte de la sélectivité fréquentielle et d’autre part des zones neuro-épithéliales non fonctionnelles. Tant le premier que le second mécanisme apparaissent dans une proportion variable et non prédictible d’un sujet à un autre. Deux tests permettent le diagnostic de ces atteintes spécifiques: la Courbe d’Accord (Tuning Curve: TC) et le Threshold E

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11

Smurzynski, Jacek. "Recent Advances in Cochlear Physiology." Digital Commons @ East Tennessee State University, 2012. https://dc.etsu.edu/etsu-works/2161.

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12

Tobin, Mélanie. "Gradients in the mechanical properties of auditory hair cells." Thesis, Sorbonne Paris Cité, 2016. http://www.theses.fr/2016USPCC183/document.

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Notre capacité à communiquer et à apprécier la musique repose sur une discrimination de fréquences couvrant une large gamme de fréquences sonores. Cette propriété résulte de cellules mécanosensorielles « ciliées », qui sont réglées pour répondre de façon maximale à une fréquence caractéristique qui varie monotoniquement le long de l’axe de l’organe auditif, la cochlée. Les mécanismes cellulaires et moléculaires qui définissent la fréquence d’une cellule ciliée et régulent sa valeur pour différentes cellules afin de couvrir la gamme auditive demeurent néanmoins inconnus. Notre hypothèse de trav

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13

O'Beirne, G. A. "Mathematical modelling and electrophysiological monitoring of the regulation of cochlear amplification." Thesis, University of Canterbury. Communication Disorders, 2005. http://theses.library.uwa.edu.au/adt-WU2006.0115/.

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The cochlea presumably possesses a number of regulatory mechanisms to maintain cochlear sensitivity in the face of disturbances to its function. Evidence for such mechanisms can be found in the time-course of the recovery of CAP thresholds during experimental manipulations, and in observations of slow oscillations in cochlear micromechanics following exposure to low-frequency tones (the “bounce phenomenon”) and other perturbations. To increase our understanding of the these oscillatory processes within the cochlea, and OHCs in particular, investigations into cochlear regulation were carried ou

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14

Lacher-Fougère, Stéphane. "Etude psychophysique du codage temporel des sons en cas de pathologie cochléaire." Bordeaux 2, 1999. http://www.theses.fr/1999BOR23044.

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15

Huang, Zhi Wu. "Spectre moyen d'activité électrophysiologique cochléaire : un indice physiologique possible d'acouphène. Etudes expérimentales chez le cobaye." Bordeaux 2, 1997. http://www.theses.fr/1997BOR28487.

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16

Schweinzger, Ivy A. "Examining the Physiologic Phenotype of Cochlear Synaptopathy Using Narrowband Chirp-Evoked Compound Action Potentials." University of Cincinnati / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1573811742950316.

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17

Wang, Tzu-Lun Verfasser], Tobias [Akademischer Betreuer] [Moser, Martin [Akademischer Betreuer] Göpfert, and Fred [Akademischer Betreuer] Wolf. "Synaptic physiology of the cochlear sound encoding / Tzu-Lun Wang. Gutachter: Martin Göpfert ; Fred Wolf. Betreuer: Tobias Moser." Göttingen : Niedersächsische Staats- und Universitätsbibliothek Göttingen, 2013. http://d-nb.info/1044871296/34.

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18

Bonfils, Pierre. "Physiologie des systèmes efférents médians modulation des mécanismes actifs de la cochlée /." Grenoble 2 : ANRT, 1988. http://catalogue.bnf.fr/ark:/12148/cb37612053n.

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19

Blanchet, Christophe. "Caractérisation de la réponse cholinergique des cellules ciliées externes cochléaires." Bordeaux 2, 1997. http://www.theses.fr/1997BOR28490.

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20

Kutscher, Andrew. "Role of synaptic inhibition in shaping response properties in the intermediate nucleus of the lateral lemniscus /." Thesis, Connect to this title online; UW restricted, 2007. http://hdl.handle.net/1773/9046.

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21

Velez-Ortega, Alejandra C. "TRPA1 CHANNELS IN COCHLEAR SUPPORTING CELLS REGULATE HEARING SENSITIVITY AFTER NOISE EXPOSURE." UKnowledge, 2014. http://uknowledge.uky.edu/physiology_etds/20.

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TRPA1 channels are sensors for noxious stimuli in a subset of nociceptive neurons. TRPA1 channels are also expressed in cells of the mammalian inner ear, but their function in this tissue remains unknown given that Trpa1–/– mice exhibit normal hearing, balance and sensory mechanotransduction. Here we show that non-sensory (supporting) cells of the hearing organ in the cochlea detect tissue damage via the activation of TRPA1 channels and subsequently modulate cochlear amplification through active cellshape changes. We found that cochlear supporting cells of wild type but not Trpa1–/– mice gener

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22

Becker, Sebastian [Verfasser], Herbert [Akademischer Betreuer] Hudde, and Karlheinz [Akademischer Betreuer] Ochs. "A physiology-based circuit model of the human peripheral ear revealing the mechanisms of non-linear active cochlear gain and otoacoustic emissions / Sebastian Becker. Gutachter: Herbert Hudde ; Karlheinz Ochs." Bochum : Ruhr-Universität Bochum, 2016. http://d-nb.info/1095884980/34.

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23

Seluakumaran, Kumar. "Descending control of responses in the auditory midbrain." University of Western Australia. School of Biomedical, Biomolecular and Chemical Sciences, 2007. http://theses.library.uwa.edu.au/adt-WU2007.0152.

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[Truncated abstract] The mammalian inner ear is innervated by the efferent olivocochlear system which is divided into medial and lateral systems. In anaesthetised animals, medial olivocochlear (MOC) axons can be electrically stimulated at the floor of the IVth ventricle. MOC stimulation suppresses the spontaneous activity and sound-evoked responses of primary afferents by its actions on outer hair cells. Effects of MOC stimulation have been also reported on responses of neurons in the cochlear nucleus, the first central auditory center receiving cochlear input. However, very little is known ab

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24

Bartolami, Sylvain. "Caractérisation du système cholinergique muscarinique dans la cochlée de rat." Montpellier 2, 1993. http://www.theses.fr/1993MON20035.

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Le systeme muscarinique de la cochlee de rat a ete caracterise. Il est active via des recepteurs m3 couples a la synthese des inositols phosphates (ip). Le systeme de transduction est sensible aux molecules ototoxiques. Au cours du developpement, il participerait a la mise en place de l'innervation efferente mediane. Chez l'adulte, nous avons demontre que certains sites m3 sont des autorecepteurs presynaptiques qui reguleraient la liberation d'acetylcholine, en position postsynaptique, ils participeraient a la modulation des contractions lentes des cellules ciliees externes

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25

Magnan, Pascal. "Etude de la propagation et de la dissipation du signal acoustique au niveau de l'oreille interne du cobaye." Montpellier 2, 1995. http://www.theses.fr/1995MON20051.

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Mieux comprendre les phenomenes hydromecaniques cochleaires lors d'une stimulation sonore, tel est l'objectif de cette these. La propagation et la dissipation du signal acoustique dans l'oreille interne representent les etapes clefs de l'initiation du traitement de l'information sonore par l'organe neurosensoriel. Par la mesure directe de la pression acoustique de part et d'autre de la membrane basilaire, on peut evaluer les forces qui s'exercent sur la cloison cochleaire, dans les differents tours de la spire. La pression resultante peut etre comparee au potentiel microphonique differentiel q

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26

Le, Frious Françoise. "Etude et réalisation d'une instrumentation pour l'analyse des signaux d'oto-émission acoustique." Toulouse 3, 1992. http://www.theses.fr/1992TOU30151.

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L'oto-emission acoustique est un signal acoustique complexe, qui est enregistre dans le conduit auditif apres stimulation sonore de l'oreille. Ce signal est genere par des phenomenes actifs d'origine cochleaire. L'interpretation de l'oto-emission doit permettre l'evaluation de l'etat physiologique de la cochlee et precisement des cellules ciliees externes. Nous nous sommes interesses a la partie metrologique de l'oto-emission. Il s'agit, concretement, d'etudier et de realiser tout une chaine d'acquisition et de traitement de l'oto-emission. La premiere partie de l'etude est la mise au point d'

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27

Safieddine, Saaid. "Etude des innervations cochléaires par immunofluorescence et par hybridation in situ : détection des récepteurs et coexistence des neurotransmetteurs/neuromodulateurs." Montpellier 2, 1993. http://www.theses.fr/1993MON20047.

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1)coexpression des recepteurs des acides amines excitateurs par les neurones auditifs primaires i. L'expression des arnm de la sous-unite glur4 etait limitee aux seules cellules gliales. Les recepteurs nmdar-1, glur2, glur3 et mglur1, sont exprimes par les neurones auditifs primairesi innervant les cellules ciliees internes. En accord avec l'implication predominante des recepteurs ampa dans la neurotransmission cochleaire, le marquage obtenu par les sous-unites glur2 et glur3 etait plus intense que pour les recepteurs nmda et mglur1. 2)coexistence de neurotransmetteurs/neuromodulateurs dans le

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28

Jing, Zhizi. "Sound Encoding in the Mouse Cochlea: Molecular Physiology and Optogenetic Stimulation." Doctoral thesis, 2013. http://hdl.handle.net/11858/00-1735-0000-0022-5E93-3.

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29

Morton-Jones, Rachel. "Characterisation of ryanodine receptor expression in the rat cochlea." 2007. http://hdl.handle.net/2292/3186.

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Increases in intracellular Ca2+ play a central role in cochlear function. The ryanodine receptor (RyR) intracellular Ca2+ release channel, a ubiquitous element of Ca2+ signalling, has been implicated in the regulation of sound transduction and auditory neurotransmission. Despite this, the molecular basis underlying RyR-mediated Ca2+ signalling in the cochlea has been limited. This thesis investigates the molecular and functional characterisation of RyR expression in the cochlea. RT-PCR analysis showed expression of RyR1, RyR2 and RyR3 isoform mRNA transcripts in the rat cochlea and also in the

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30

Butola, Tanvi. "Molecular physiology of signal transmission along the auditory pathway." Doctoral thesis, 2017. http://hdl.handle.net/11858/00-1735-0000-0023-3FAB-7.

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31

Krinner, Stefanie. "Molecular physiology of synaptic sound encoding at the first auditory synapse." Doctoral thesis, 2017. http://hdl.handle.net/11858/00-1735-0000-002E-E396-A.

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32

"An active model for otoacoustic emissions and its application to time-frequency signal processing." 2001. http://library.cuhk.edu.hk/record=b6073343.

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Yao Jun.<br>Thesis (Ph.D.)--Chinese University of Hong Kong, 2001.<br>Includes bibliographical references.<br>Electronic reproduction. Hong Kong : Chinese University of Hong Kong, [2012] System requirements: Adobe Acrobat Reader. Available via World Wide Web.<br>Mode of access: World Wide Web.<br>Abstracts in English and Chinese.

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33

Wang, Tzu-Lun. "Synaptic physiology of the cochlear sound encoding." Doctoral thesis, 2013. http://hdl.handle.net/11858/00-1735-0000-0001-BAB4-8.

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34

Zhang, Su. "Physiology of morphologically identified neurons in the dorsal cochlear nucleus of mice." 1991. http://catalog.hathitrust.org/api/volumes/oclc/26025057.html.

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35

Meyer, Alexander. "Struktur und Funktion der afferenten Synapse innerer Haarzellen der Cochlea." Doctoral thesis, 2010. http://hdl.handle.net/11858/00-1735-0000-0006-AF82-B.

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