Relevant bibliographies by topics / Coeliac (celiac) disease

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters

Academic literature on the topic 'Coeliac (celiac) disease'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Coeliac (celiac) disease"

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Ellis, HJ, and PJ Ciclitira. "In vivo gluten challenge in coeliac disease." Canadian Journal of Gastroenterology 15, no. 4 (2001): 243–47. http://dx.doi.org/10.1155/2001/127241.

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In vivo gluten challenge has been used since the early 1950s to study the role of cereal fractions in celiac disease. While early studies relied on crude indicators of celiac toxicity, the advent of jejunal biopsy and sophisticated immunohistochemical techniques has allowed accurate studies to be performed. Studies to determine the nature of the cereal component that is toxic to patients with celiac disease have concentrated on wheat because of its nutritional importance. A number of in vitro studies indicated the presence of one or more celiac-activating epitopes with theN-terminus of the A-gliadin molecule. In vivo challenge with three synthetic peptides subsequently indicated the toxicity of a peptide corresponding to amino acids 31 to 49 of A-gliadin. In vivo gluten challenge is the gold standard for the assessment of celiac toxicity; however, jejunal biopsy is a relatively invasive procedure, thus, other methods have been investigated. Direct infusion of the rectum with gluten has been shown to result in an increase in mucosal intraepithelial lymphocytes, occurring only in celiac patients. This method has been used to study the celiac toxicity of gliadin subfractions. The in vitro technique of small intestinal biopsy organ culture is also a useful tool and appears to give the same results as in vivo challenge. The importance of tiny amounts of gliadin in the diet, such as that which occurs in wheat starch, has been studied by in vivo challenge; this technique has clarified the position of oats in the gluten-free diet. Several studies suggest that this cereal may be included in the diet of most adult celiac patients. Studies of the transport of gliadin across the enterocyte following ingestion or challenge suggest that gliadin may be metabolized by a different pathway in celiac disease. This could result in an abnormal presentation to the immune system, triggering a pathogenic rather than a tolerogenic response.
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Kocsis, Dorottya, Nóra Béres, Gábor Veres, Dolóresz Szabó, Katalin Eszter Müller, András Arató, and Márk Juhász. "Genetic and epigenetic aspects of celiac disease." Orvosi Hetilap 155, no. 3 (January 2014): 83–88. http://dx.doi.org/10.1556/oh.2014.29795.

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Genetic backround of coeliac disease has been subjects to intensive research since decades. However, only results of HLA phenotyping have been taken over to routine clinical practice. Meanwhile, data on the role of epigenetical factors in the manifestation of diseases have been emerging. In coeliac disease, there are several questions both in the fields of genetics and epigenetics yet to be answered. In this review, a cross section of current knowledge on these issues is presented with special interest regarding the future clinical applications. Orv. Hetil., 2014, 155(3), 83–88.
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Hindryckx, Pieter, Barrett G. Levesque, Tom Holvoet, Serina Durand, Ceen-Ming Tang, Claire Parker, Reena Khanna, et al. "Disease activity indices in coeliac disease: systematic review and recommendations for clinical trials." Gut 67, no. 1 (October 31, 2016): 61–69. http://dx.doi.org/10.1136/gutjnl-2016-312762.

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ObjectiveAlthough several pharmacological agents have emerged as potential adjunctive therapies to a gluten-free diet for coeliac disease, there is currently no widely accepted measure of disease activity used in clinical trials. We conducted a systematic review of coeliac disease activity indices to evaluate their operating properties and potential as outcome measures in registration trials.DesignMEDLINE, EMBASE and the Cochrane central library were searched from 1966 to 2015 for eligible studies in adult and/or paediatric patients with coeliac disease that included coeliac disease activity markers in their outcome measures. The operating characteristics of histological indices, patient-reported outcomes (PROs) and endoscopic indices were evaluated for content and construct validity, reliability, responsiveness and feasibility using guidelines proposed by the US Food and Drug Administration (FDA).ResultsOf 19 123 citations, 286 studies were eligible, including 24 randomised-controlled trials. Three of five PROs identified met most key evaluative criteria but only the Celiac Disease Symptom Diary (CDSD) and the Celiac Disease Patient-Reported Outcome (CeD PRO) have been approved by the FDA. All histological and endoscopic scores identified lacked content validity. Quantitative morphometric histological analysis had better reliability and responsiveness compared with qualitative scales. Endoscopic indices were infrequently used, and only one index demonstrated responsiveness to effective therapy.ConclusionsCurrent best evidence suggests that the CDSD and the CeD PRO are appropriate for use in the definition of primary end points in coeliac disease registration trials. Morphometric histology should be included as a key secondary or co-primary end point. Further work is needed to optimise end point configuration to inform efficient drug development.
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Degterev, D. A., S. V. Bykova, and A. I. Parfenov. "Gluten related headaches." Medical alphabet, no. 1 (June 11, 2020): 36–39. http://dx.doi.org/10.33667/2078-5631-2020-1-36-39.

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Coeliac disease and non-celiac gluten sensitivity are disorders whose diagnosis can be delayed for many years from the first clinical manifestations. Typical debut of coeliac disease is an extraintestinal manifestation as gluten-related disorders. One of such impairments is headache. There are currently data about gluten-associated headache including prevalence, clinical manifestations, diagnostic features and treatment results in this paper.
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Tokodi, István. "Nem minden coeliakia, ami annak látszik. Coeliakia és gyulladásos bélbetegség együttes előfordulása." Orvosi Hetilap 156, no. 26 (June 2015): 1059–64. http://dx.doi.org/10.1556/650.2015.30181.

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The prevalence of inflammatory bowel disease is ten times more common in patients with celiac disease; however, studies investigating the reverse relation have contradictory findings. Many gene polymorphisms are known to be present in both diseases; furthermore, similarities observed in their pathophysiological mechanism, their family concomitance, results of the serologic analysis and their macroscopic and microscopic symptoms in the gastro-intestinal system suggest a relevant association between the two diseases. The author presents the history of four patients, of whom two had both Crohn’s and coeliac diseases. In the two other patients with inflammatory bowel disease the possible diagnosis of coeliac disease was suspected, but after additional examinations coeliac disease was excluded in one patient and seemed to be unlikely in the other patient. The author concludes that the differential diagnosis of the two diseases is not easy and if one of them is diagnosed, the possible presence of the other one should be taken into consideration. Orv. Hetil., 2015, 156(26), 1059–1064.
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Biecker, Erwin, Hans-Peter Fischer, and Michael Schepke. "Nodular Regenerative Hyperplasia and Portal Hypertension in a Patient with Coeliac Disease." Case Reports in Gastrointestinal Medicine 2011 (2011): 1–3. http://dx.doi.org/10.1155/2011/938580.

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Nodular regenerative hyperplasia (NRH) of the liver is often associated with rheumatologic or lymphoproliferative disorders and a cause of portal hypertension in some patients. We report the case of a 71-year-old patient with celiac disease and unexplained portal hypertension. Biopsy of the liver revealed NRH as the underlying cause. The patient did not suffer from an autoimmune, rheumatologic or lymphoproliferative disease. A thrombophilic disorder that might cause NRH was ruled out. Celiac disease is often associated with mild elevation of liver enzymes and steatosis of the liver, but the association with NRH was described in only a few patients. We discuss the possible relationship of celiac disease and NRH.
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Olteanu, Dan, Alexandru Diaconescu, Radu Voiosu, Andrei Voiosu, and Cristina Olariu. "PRESENT DATA IN THE DIAGNOSIS AND TREATMENT OF COELIAC DISEASE (2)." Romanian Medical Journal 64, no. 1 (March 31, 2017): 25–33. http://dx.doi.org/10.37897/rmj.2017.1.5.

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Coeliac disease incidence rised during the last 50 years and represents a concern by diagnostic problems and costs. The recent data regarding etiology, pathogeny, comparative diagnostic value of serology and small intestinal biopsy are summarised. The new data about refractory celiac disease to gluten free diet and therapeutic perspectives are also presented (glutenases, larazotide acetate, genetic alteration of cereals, tissulary transglutaminase inhibitors etc).
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Kamalova, Aelita A., Daria O. Timofeeva, and Almazia R. Shakirova. "Modern Aspects of Celiac Disease Diagnosis in Children." Current Pediatrics 19, no. 5 (December 24, 2020): 371–78. http://dx.doi.org/10.15690/vsp.v19i5.2217.

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Celiac disease is an immune-mediated systemic disorder caused by gluten in people with genetic predisposition. Celiac disease is characterized by wide range of clinical manifestations (both gastroenterological and extraintestinal), that can complicate the diagnosis. Thus, celiac disease often remains undiagnosed. ESPGHAN has published updated clinical guidelines with adjusted coeliac disease diagnosis algorithms in 2020. It is proposed to determine antibodies to tissue transglutaminase (TGA-IgA) and total IgA within normal content of gluten-containing products in the diet on the first stage of children screening. The diagnosis of celiac disease can be established without small intestine biopsy in case of increased levels of TGA-IgA ≥ 10 of upper limit of normal and presence of antibodies to endomysium (EMA-IgA) in secondary serum. In such cases, ESPGHAN does not recommend any additional genetic testing to confirm celiac disease as it does not increase the reliability of the diagnosis. Antigen tests on class G or A antibodies against native gliadin are not specific and are not recommended for use in the diagnosis of celiac disease.
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Gwiggner, M., and P. Patel. "An Unusual Case of Obscure Gastrointestinal Bleeding in a Patient with Coeliac Disease." Case Reports in Gastrointestinal Medicine 2011 (2011): 1–3. http://dx.doi.org/10.1155/2011/634684.

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This paper describes the journey of a patient with coeliac disease who presented with overt obscure gastrointestinal bleeding. Upper and lower gastrointestinal endoscopy did not reveal a source of bleeding, but an abdominal CT scan detected abnormal lymphadenopathy and a wireless capsule endoscopy diagnosed a jejunal tumour, which was surgically removed. Gastrointestinal bleeding is rare in celiac disease. Malignant tumours of the small intestine are generally uncommon, but celiac disease represents a significant risk factor. Wireless capsule endoscopy has been a useful tool to investigate patients with obscure gastrointestinal bleeding.
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Olteanu, Dan, Alexandru Diaconescu, Radu Voiosu, Andrei Voiosu, and Cristina Olariu. "Present data in the diagnosis and treatment of coeliac disease (Part I)." Romanian Medical Journal 63, no. 4 (December 31, 2016): 272–79. http://dx.doi.org/10.37897/rmj.2016.4.2.

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Coeliac disease incidence raised during the last 50 years and represents a concern by diagnostic problems and costs. The recent data regarding etiology, pathogeny, comparative diagnostic value of serology and small intestinal biopsy are summarised. New data about refractory celiac disease to gluten free diet and therapeutic perspectives are also presented (glutenases, larazotide acetate, genetic alteration of cereals, tissulary transglutaminase inhibitors etc).
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Dissertations / Theses on the topic "Coeliac (celiac) disease"

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Popat, Sanjaykumar Batukial. "A genetic study of coeliac disease." Thesis, Institute of Cancer Research (University Of London), 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.271488.

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Holmes, Geoffrey. "Morbidity and mortality in coeliac disease." Thesis, University of Derby, 2018. http://hdl.handle.net/10545/623486.

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Celiac disease is a small intestinal immune-mediated enteropathy precipitated by exposure to gluten, a protein complex in the cereals wheat, barley and rye, in genetically susceptible people. Once considered an uncommon disorder restricted to children of European descent, it is now known to be one of the most common chronic diseases encountered in the Western world, with a serological prevalence of 1% that can be diagnosed at any age. Since it is so common much co-morbidity comprising malignant and non-malignant conditions will occur in association. Malignant complications particularly lymphoma were first described over 50 years ago but the natural history and how commonly these occurred were unknown until relatively recently. Similarly, many non-malignant conditions were known to occur but initially the risks were unclear. It was not until the frequency of coeliac disease could be determined accurately in the community and population-based studies of morbidity and mortality in coeliac disease patients carried out in defined cohorts that these questions could be answered. My research into these aspects of coeliac disease began in 1971 and a body of 35 of my publications spanning the years 1974 to 2018 on the morbidity and mortality of the disorder are presented in this thesis. I have introduced my research findings at many international and national meetings and these data have been influential in shaping the research agenda of other workers. One of my papers (Publication 9) published in Gut in 1989, was the most cited of all papers which appeared in the journal for that year. To date it has been cited 1122 times. Information exists for 24 papers presented here and for these the total number of citations stands at 3,887. This excludes references to book chapters. Anecdotal evidence indicates frequent mentions in lectures and clinical practice.
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Midhagen, Gunnar. "Adult Coeliac Disease in Clinical Practice." Doctoral thesis, Linköping : Univ, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-7373.

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Hollén, Elisabet. "Coeliac disease in childhood : on the intestinal mucosa and the use of oats /." Linköping : Univ, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-7690.

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Kristjánsson, Guðjón. "Food antigen sensitivity in coeliac disease assessed by the mucosal patch technique /." Uppsala : Acta Universitatis Upsaliensis, 2005. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-6020.

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Sverker, Annette. "Dilemmas and consequences of chronic disease : lived experiences of coeliac disease and neuropathic pain /." Göteborg : Department of Public Health and Community Medicine, Social Medicine, Sahlgrenska Academy at Göteborg University, 2007. http://hdl.handle.net/2077/7362.

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Apell, Amandine, and Viktoria Burman. "Hur upplevs och hanteras familjens vardag av mödrar till barn med celiaki? : en kvalitativ intervjustudie." Thesis, Umeå universitet, Institutionen för kostvetenskap, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-128826.

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SAMMANFATTNING Bakgrund: Celiaki är en autoimmun sjukdom med en prevalens på 2/100 i Sverige. Enda behandlingen är att äta en kost fri från gluten. Gluten förekommer i spannmålen vete, råg, korn och att diagnostiseras med celiaki innebär ofta att en förändring av matvanor blir nödvändig. Att leva tillsammans med någon med celiaki har visat sig kunna vara problematiskt då familjelivet kan påverkas negativt och föräldrar till barn med celiaki upplever ofta oro och ängslan för sitt barn. Syfte: Syftet med studien var att undersöka hur mödrar till barn med celiaki upplever och hanterar familjens vardag i relation till barnets specialkost. Metod: Ett målinriktat urval användes och kvalitativa, semi-strukturerade intervjuer genomfördes med nio mödrar till barn med celiaki. Materialet spelades in och transkriberades ordagrant för att sedan analyseras med kvalitativ innehållsanalys. Resultat: Samtliga mödrar upplevde att vardagen hade anpassats efter barnet med celiaki och det fanns olika sätt att hantera den glutenfria kosten. Vissa valde att ha helt glutenfritt hemma för att undvika stress över rädslan att ge barnet fel mat. Andra valde att ha både gluteninnehållande och glutenfria livsmedel hemma p.g.a. ekonomiska skäl samt preferenser från övriga familjemedlemmar. Kunskapsnivån kring celiaki i samhället ansågs låg, vilket upplevdes försvåra vardagen för mödrar till ett barn med celiaki. Majoriteten av mödrarna uttryckte att bästa stödet fanns att tillgå via internet och sociala medier. Slutsats: Enligt mödrar till barn med celiaki innebar sjukdomen en del dilemman och vardagen upplevdes stundvis som orofylld, vilket kan påverka livskvaliteten. Internet och sociala medier sågs som ett bra stöd i vardagen. Ökad kunskap samt större förståelse från omgivningen skulle kunna förbättra familjernas vardag.
ABSTRACT                          Background: Celiac disease (CD) is an autoimmune disorder with a prevalence of 2/100 in Sweden. The only treatment is a diet free from gluten. Gluten occurs in grains of wheat, rye, barley and being diagnosed with CD often means that a change in eating habits is necessary. Living together with someone with CD has been shown to be problematic. Family life may be negatively affected and parents of children with CD experience worry and anxiety for their child. Objective: The aim of the study was to examine how mothers of children with CD experienced and handled everyday life in relation to their child's gluten-free diet (GFD). Method: A targeted selection was used and qualitative, semi-structured interviews were conducted with nine mothers of children with CD. The interviews were recorded and transcribed verbatim and then analyzed with qualitative content analysis. Results: All participants felt that everyday life had been adapted to the child with CD and there were different ways to deal with the GFD. Some chose to have the home as a gluten-free zone to avoid the stress of risking to give the child wrong food. Others chose to have both gluten-containing and gluten-free foods at home because of economic aspects and preferences from other family members. The level of knowledge about CD in the community was considered low, which was perceived to complicate everyday life for mothers of a child with CD. The majority of participants expressed that best support was found through Internet and social media. Conclusion: According to mothers of children with CD, the disease involved some dilemmas and daily life was at times perceived as worrisome, which can affect quality of life. Internet and social media were considered as good support in everyday life. Increased knowledge in society and greater understanding from the environment could improve families everyday life.
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Crocker, Helen. "Coeliac disease : health-related quality of life and patients' experiences of health care services." Thesis, University of Oxford, 2016. http://ora.ox.ac.uk/objects/uuid:7758bef9-d019-4b9f-b992-d7bf453ad427.

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Coeliac disease (CD) is a chronic gastrointestinal condition, the only treatment for which is a gluten-free diet (GFD). Following a GFD is restrictive, burdensome, and can impact health-related quality of life (HRQOL). People with CD can experience long delays to diagnosis and evidence suggests large variations in follow-up care, but the relationship between health care experiences and HRQOL is unknown. The main aim of this research was to develop a patient-reported outcome measure and patient experience questionnaire, and use these to investigate the relationship between adults' experiences of health care services and HRQOL in CD. The questionnaires, named the Coeliac Disease Assessment Questionnaire (CDAQ) and the Coeliac Disease Patient Experience Questionnaire (CD-PEQ), were developed following qualitative interviews with adults with CD, and refined with input from experts, and cognitive interviews. The CDAQ was also subject to a translatability assessment to assess its linguistic and cultural translatability, and a cross-sectional survey to assist with item reduction and scale generation. Members of Coeliac UK (n=267) completed the CDAQ and CD-PEQ, together with the SF-36v2 and demographic questions as part of a postal survey. Psychological health, vitality, general health, and dietary burden were found to have the greatest impact on HRQOL, with physical health and social isolation the least affected. HRQOL was found to have a strong correlation with patients' experiences of health care services. Aspects most strongly related were: the provision of information; communication with HCPs; difficulty obtaining prescriptions; and GPs' knowledge. This research has identified aspects of health care services that are strongly related to HRQOL in CD. Health care providers are recommended to focus service improvement efforts on these areas. A reliable and valid disease-specific patient-reported outcome measure and patient experience questionnaire have been developed as part of this study. The CDAQ is suitable for use in research studies, including clinical trials, to assess HRQOL in CD.
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Rosén, Anna. "Mass screening for celiac disease in 12-year-olds : Finding them and then what?" Doctoral thesis, Umeå universitet, Epidemiologi och global hälsa, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-58950.

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Background Mass screening for celiac disease (CD) as a public health intervention is controversial. Before implementation, a suitable screening strategy should be outlined, and the acceptability of the screening scrutinized. Also, the benefits of early detection and possible negative consequences should be explored and compared. The overall aim of this thesis was to evaluate different strategies for finding 12-year-olds with undiagnosed CD in the general population, and to explore the experiences of those receiving the diagnosis in a mass screening. Methods A school-based CD screening of 12-year-olds was conducted in five study sites across Sweden. Out of 10041 children who were invited, 7208 had a blood sample analyzed for CD-marker tissue transglutaminase of isotype IgA (tTG-IgA) and 7161 for total serum IgA (s-IgA). If the s-IgA value was low, tTG-IgG was also measured. Additional analysis of endomysial antibodies (EMA) was performed if borderline values of tTG were found. In total, 192 had elevated CD-markers, 184 underwent a small intestinal biopsy and 153 eventually had CD diagnosed. Before receiving knowledge about their CD status, children and their parents filled in questionnaires regarding symptoms and CD-associated conditions. Questionnaires were returned by 7054 children (98%) and 6294 parents (88%). Later, all adolescents who had been diagnosed with CD more than one year ago (n=145), and their parents, were invited to a mixed-method follow-up study in which they shared their experiences in questionnaires, written narratives and focus group discussions. In total, we have information on 117 (81%) of these adolescents, either from the adolescents themselves (n=101) and/or from their parent/s (n=125). Data were analyzed using a combination of descriptive and analytical quantitative and qualitative methodologies. Results We found that information on symptoms and CD-associated conditions were poor predictors for finding undiagnosed CD in the study population. Questionnaire-based case-finding by asking for CD-associated symptoms and conditions would have identified 52 cases (38% of all cases) at a cost of blood-sampling 2282 children (37% of the study population). The tTG-IgA test had an excellent diagnostic accuracy with the area under the receiver operating characteristic curve of 0.988. If using the recommended cut-off for tTG-IgA (>5 U/mL) 151 had fulfilled biopsy criteria and 134 CD cases had been identified. The strategy of lowering the cut-off to tTG-IgA>4 U/mL, and adding the EMA analysis in those with tTG-IgA between 2-4 U/mL, identified another 17 cases (a 12% increase) at the cost of performing 32 additional biopsies. Measuring total s-IgA in 7161 children discovered only two additional cases at the cost of performing 5 additional biopsies. The positive predictive value of our screening strategy was 80%.  Results from the follow-up study of the screening-detected CD cases illustrated that 54% reported health improvement after initiated treatment, but also that these health benefits had to be balanced against social sacrifices. We also found that although the screening-detected diagnosis was met with surprise and anxiety, the adolescents and their parents were grateful for being made aware of the diagnosis. A majority of parents (92%) welcomed a future screening, but both adolescents and parents suggested that it should be conducted earlier in life. Conclusion Obtaining information on symptoms and CD-associated conditions was not a useful step in finding undiagnosed CD cases in a general population. The serological marker tTG-IgA, however, had excellent diagnostic accuracy also when lowering the cut-off. The diagnosis had varying impact on adolescents’ quality of life, and their perceived change in health had to be balanced against the social sacrifices resulting from the diagnosis. Overall, CD mass screening seemed acceptable to most of those who were diagnosed and their parents.
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Walkinshaw, Rosemary. "User-centred design the investigation, design and evaluation of an information handbook for coeliac patients : this thesis is submitted to the Auckland University of Technology in partial fulfilment of the degree of Masters of Arts (Art and Design), 2008 /." Click here to access this resource online, 2008. http://hdl.handle.net/10292/446.

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Coeliac disease is an intestinal disorder, requiring patients to maintain a life-long gluten-free diet to ensure better health and reduce the risks of osteoporosis, intestinal lymphoma and other associated diseases. Patients must have access to clear information about the disease and about what foods are safe to eat, detect foods that contain gluten and make adjustments that need to be made to their diet. There is very little well designed information currently available for Coeliac patients. Eating out and shopping in supermarkets can become a nightmare. A User-centred design approach was used to generate information and insights on the Coeliac disease and patients' needs to conceptualise, design and evaluate an information handbook that is both functional and pleasurable to use. Questionnaires, interviews and focus groups were used to generate empirical data that guided a participative design process before the evaluation of the handbook. This project consists of a handbook as practical work that represents the main body of applied research. The practical work and the exegesis constitute 50% each of the thesis value.
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Books on the topic "Coeliac (celiac) disease"

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Griffiths, Helen. Coeliac disease: Nursing care and management. Hoboken, NJ: John Wiley & Sons, 2008.

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1934-, Auricchio S., and Visakorpi J. K, eds. Common food intolerances 1: Epidemiology of coeliac disease. Basel: Karger, 1992.

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Stott, Heather. Active again: Clearing my chronic fatigue and coeliac minefield. Brough: Tulip, 2002.

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International Symposium on Coeliac Disease (6th 1992 Trinity College, Dublin, Ireland). Gastrointestinal immunology and gluten-sensitive disease: Proceedings of the Sixth International Symposium on Coeliac Disease held at Trinity College, Dublin, in July 1992. Edited by Feighery Conleth and O'Farrelly Cliona. Dublin: Oak Tree Press, 1994.

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Rawcliffe, Peter. The gluten-free diet book: A guide to coeliac disease, dermatitis herpetiformis and gluten-free cookery. London: Martin Dunitz, 1985.

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Rawcliffe, Peter. The gluten-free diet book: A guide to coeliac disease, dermatitis, herpetiformis and gluten-free cookery. Cape Town: Timmins, 1985.

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Rawcliffe, Peter. The Gluten-free diet book: A guide to coeliac disease, dermatitis herpetiformis and gluten-free cookery. London: Optima, 1990.

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Marsh, M. N., and Michael N. Marsh. Coeliac Disease. Blackwell Science, 1992.

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Coping with Coeliac Disease. Ulverscroft Large Print, 2002.

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McConnell, R. B. Genetics of Coeliac Disease. Springer, 2014.

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Book chapters on the topic "Coeliac (celiac) disease"

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Gilissen, Luud J. W. J., Ingrid M. van der Meer, and Marinus J. M. Smulders. "Beyond Coeliac Disease Toxicity." In Frontiers in Celiac Disease, 139–47. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000128995.

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Auricchio, Renata, and Riccardo Troncone. "Diagnosis of Coeliac Disease." In Frontiers in Celiac Disease, 99–106. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000128664.

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Gianfrani, Carmen, Alessandra Camarca, Virginia Salvati, Giuseppe Mazzarella, Maria Grazia Roncarolo, and Riccardo Troncone. "Regulatory T Cells in the Coeliac Intestinal Mucosa." In Frontiers in Celiac Disease, 181–87. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000129063.

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Al-toma, A., W. H. M. Verbeek, and C. J. J. Mulder. "Update on the Management of Refractory Coeliac Disease." In Frontiers in Celiac Disease, 123–32. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000128672.

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Howdle, P. D. "CELIAC (COELIAC) DISEASE." In Encyclopedia of Food Sciences and Nutrition, 987–94. Elsevier, 2003. http://dx.doi.org/10.1016/b0-12-227055-x/00266-2.

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J. Ciclitira, Paul, and Alastair Forbes. "Management of Patients with Refractory Coeliac Disease." In Celiac Disease. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.96231.

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Abstract:
Coeliac disease (CD) is an immune-mediated disorder affecting the small intestine. The condition represents an intolerance to gluten. Removal of dietary gluten permits recovery, with a full recovery for the majority of affected subjects. A percentage of affected subjects who do not improve with a gluten-free diet are considered to have refractory coeliac disease (RCD). Refractory coeliac disease is subdivided into type 1, characterised by a polyclonal expansion of intraepithelial lymphocytes (IELs) that have a normal phenotype, and type 2 (RCD2) which exhibits IELs with a monoclonal phenotype. Subjects with RCD carry a high risk of complications, including ulcerative jejunitis and lymphoma affecting the small intestine, the latter termed enteropathy-associated T-cell lymphoma (EATL).
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"Celiac Disease (coeliac disease, sprue, 6p21.3)." In Encyclopedia of Genetics, Genomics, Proteomics and Informatics, 296–97. Dordrecht: Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6754-9_2609.

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Sziksz, Erna, Leonra Himer, Gabor Veres, Beta Szebeni, Andrs Arat, Tivadar Tulassay, and Adam Vannay. "Heat shock proteins (HSPs) in coeliac disease." In Celiac Disease - From Pathophysiology to Advanced Therapies. InTech, 2012. http://dx.doi.org/10.5772/47911.

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Hallert, Claes. "On treatment outcomes in coeliac disease diagnosed in adulthood." In Celiac Disease - From Pathophysiology to Advanced Therapies. InTech, 2012. http://dx.doi.org/10.5772/50268.

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Cornell and Teodor Stelmasiak. "Enzyme Therapy for Coeliac Disease: Is It Ready For Prime Time?" In Celiac Disease - From Pathophysiology to Advanced Therapies. InTech, 2012. http://dx.doi.org/10.5772/50247.

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