Relevant bibliographies by topics / Cognition disorders – Endocrine aspects

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters

Academic literature on the topic 'Cognition disorders – Endocrine aspects'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Cognition disorders – Endocrine aspects"

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Ruiz-Gayo, Mariano, and Nuria D. Olmo. "Interaction Between Circadian Rhythms, Energy Metabolism, and Cognitive Function." Current Pharmaceutical Design 26, no. 20 (June 21, 2020): 2416–25. http://dx.doi.org/10.2174/1381612826666200310145006.

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The interaction between meal timing and light regulates circadian rhythms in mammals and not only determines the sleep-wake pattern but also the activity of the endocrine system. Related with that, the necessity to fulfill energy needs is a driving force that requires the participation of cognitive skills whose performance has been shown to undergo circadian variations. These facts have led to the concept that cognition and feeding behaviour can be analysed from a chronobiological perspective. In this context, research carried out during the last two decades has evidenced the link between feeding behaviour/nutritional habits and cognitive processes, and has highlighted the impact of circadian disorders on cognitive decline. All that has allowed hypothesizing a tight relationship between nutritional factors, chronobiology, and cognition. In this connection, experimental diets containing elevated amounts of fat and sugar (high-fat diets; HFDs) have been shown to alter in rodents the circadian distribution of meals, and to have a negative impact on cognition and motivational aspects of behaviour that disappear when animals are forced to adhere to a standard temporal eating pattern. In this review, we will present relevant studies focussing on the effect of HFDs on cognitive aspects of behaviour, paying particular attention to the influence that chronobiological alterations caused by these diets may have on hippocampaldependent cognition.
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Spinedi, Eduardo, and Daniel P. Cardinali. "Neuroendocrine-Metabolic Dysfunction and Sleep Disturbances in Neurodegenerative Disorders: Focus on Alzheimer’s Disease and Melatonin." Neuroendocrinology 108, no. 4 (October 28, 2018): 354–64. http://dx.doi.org/10.1159/000494889.

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Alzheimer’s disease (AD) is associated with altered eating behavior and metabolic disruption. Amyloid plaques and neurofilament tangles are observed in many hypothalamic nuclei from AD brains. Some of these areas (suprachiasmatic nuclei, lateral hypothalamic area) also play a role in the regulation of the sleep/wake cycle and may explain the comorbidity of eating and sleep disorders observed in AD patients. Inadequate sleep increases the neurodegenerative process, for example, the decrease of slow-wave sleep impairs clearance of β-amyloid peptide (Aβ) and tau protein from cerebral interstitial fluid. Cerebrospinal fluid (CSF) melatonin levels decrease even in preclinical stages (Braak-1 stage) when patients manifest no cognitive impairment, suggesting that reduction of melatonin in CSF may be an early marker (the cause for which is still unknown) of oncoming AD. Melatonin administration augments glymphatic clearance of Aβ and reduces generation and deposition of Aβ in transgenic animal models of AD. It may also set up a new equilibrium among hypothalamic feeding signals. While melatonin trials performed in the clinical phase of AD have failed to show or showed only modest positive effects on cognition, in the preclinical stage of dementia (minimal cognitive impairment) the effect of melatonin is demonstrable with significant improvement of sleep and quality of life. In this review, we discuss the main aspects of hypothalamic alterations in AD, the association between interrupted sleep and neurodegeneration, and the possible therapeutic effect of melatonin on these processes.
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Papageorgiou, Sokratis G., and Christos Koros. "Rapidly Progressive Dementia - Clinical Aspects and Management." European Neurological Review 6, no. 4 (2011): 238. http://dx.doi.org/10.17925/enr.2011.06.04.238.

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Although no precise definition for rapidly progressive dementia (RPD) exists, this term is generally used to refer to cases with significant and progressive cognitive impairment that occurs over weeks or months. RPD represents an unusual but severe condition that causes distress not only for patients and their relatives but also for the clinicians involved, as multiple investigations and decisions about management must be made urgently to avoid misdiagnosing a treatable condition and to preserve as much of the neural tissue as possible from definite damage. While Creutzfeldt-Jacob disease (CJD) has for a long period of time been regarded as the prototype of RPD, this infrequent but severe condition can be produced by an extensive variety of causes such as various endocrine, metabolic or toxic disorders, central nervous system (CNS) infections, primary or secondary CNS neoplasms, various CNS vasculitides and various autoimmune conditions in which autoantibodies against neural tissue are produced, whether in the presence of a neoplasm or not. However, even in the more common and usually slowly progressive dementias such as Alzheimer’s disease, frontotemporal lobar degeneration, dementia with Lewy bodies and other degenerative dementias, as well as vascular dementia, establishment and progression of the disease is occasionally surprisingly accelerated, leading to a clinical presentation of RPD. The few published case series of RPD have shown that the relative frequency of underlying diseases depends mainly on the clinical setting. Thus, CJD has been found to be the most prevalent cause in referral centres for spongiform encephalopathies, while secondary causes are more prevalent in general referral centres for dementia diagnosis. In clinical practice, for the cases presenting with RPD, the diagnostic procedure must be exhaustive, starting with a detailed clinical evaluation and proceeding to a complete laboratory work-up and sophisticated neuroimaging studies. There has been recent enormous progress in imaging, with sensitive new sequences of magnetic resonance imaging and immunology; as a result, a plethora of antibodies against the CNS can now be detected in cases of autoimmune dementias, which has dramatically changed the diagnostic approach and early management of cases of RPD. The same favourable effect in clinical practice comes from the accumulated knowledge of the complex clinical picture of various causes of RPD, associated specific neurological features (pyramidal signs, ataxia, myoclonus) and systematic features (weight loss, hyponatraemia, hepatic disorders) and their mode of progression.
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Sedaghat, Katayoun. "A review on insulin presence and function in brain." International Journal of Scientific Reports 6, no. 3 (February 25, 2020): 118. http://dx.doi.org/10.18203/issn.2454-2156.intjscirep20200649.

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<p class="abstract">Hormones have major role in maintaining the homeostasis of the body and mental functions. One of the crucial hormones that act to regulate the metabolism and growth is insulin. Insulin activity in the periphery has been the subject of study since long time ago, though, beginning to understand the central activity of insulin is rather recent and because of complexity of actions and interference with neurodegenerative diseases such as Alzheimer’s and Parkinson’s and mood disorders, like anxiety and depression, it is considered very crucial hormone in the maintenance of the mental health. This mini-review will discuss briefly the recent main aspects of insulin transfer to the brain, receptor and mediators, signaling pathways with particular attention to neural system and its role in cognitive and emotional processing in brain. Insulin is an endocrine hormone with receptor distribution in different parts of brain and has role in various neural functions such as; growth factor, modulating learning or memory, mood, neural growth and survival, also controlling neurotransmitters functions in different brain regions. Insulin regulates metabolic homeostasis in brain through complex routes, which inevitably makes it involved with some neurodegenerative or affective disorders, that raises its importance for more serious studies.</p>
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Mills, Edouard G. A., Kevin T. O'Byrne, and Alexander N. Comninos. "Kisspeptin as a Behavioral Hormone." Seminars in Reproductive Medicine 37, no. 02 (March 2019): 056–63. http://dx.doi.org/10.1055/s-0039-3400239.

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AbstractSuccessful reproduction is dependent not only on hormonal endocrine responses but also on suitable partner selection, copulatory acts, as well as associated emotional, behavioral, and cognitive processes many of which are supported by the limbic system. The reproductive hormone kisspeptin (encoded by the KISS1/kiss1 gene) is now recognized as the key orchestrator of the reproductive axis. In addition to the hypothalamus, prominent kisspeptin neuronal populations have been identified throughout limbic and paralimbic brain regions across an assortment of species. In this review, we detail the emerging roles of kisspeptin signaling in the broader aspects of behavioral, emotional, and cognitive control. Recent studies from zebrafish through humans have provided new molecular and neural insights into the complex role of kisspeptin in interpreting olfactory and auditory cues to govern sexual partner preference, in regulating copulatory behaviors and in influencing mood and emotions. Furthermore, emerging roles for kisspeptin in facilitating memory and learning are also discussed. To this end, these findings shed new light onto the importance of kisspeptin signaling, while informing the pharmacological development of kisspeptin as a potential therapeutic strategy for individuals suffering from associated reproductive, emotional, and cognitive disorders.
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Gerardi, Chiara, Eleonora Allocati, Carla Minoia, Attilio Guarini, and Rita Banzi. "Long-Term Follow-Up of Classical Hodgkin Lymphoma and Diffuse Large B-Cell Lymphoma Survivors: Aims and Methodological Approach for Fondazione Italiana Linfomi Systematic Reviews." Cancers 13, no. 12 (June 14, 2021): 2976. http://dx.doi.org/10.3390/cancers13122976.

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Advances in diagnosis and treatment of hematological malignancies has boosted attention on optimal follow-up care of survivors after cancer. To collect evidence that could inform the development of an optimal model for Italian hematology centers and the scientific community, Fondazione Italiana Linfomi (FIL) commissioned an analysis of the international follow-up approaches for long-term survivors after classical Hodgkin lymphoma (cHL) or diffuse large B-cell lymphoma (DLBCL). FIL set up multidisciplinary teams, representing all different skills relevant for cancer survivors. They conducted a series of systematic reviews focused on three main aspects: incidence of long-term toxicity; comparison of old or standard therapies and more recent ones; and evidence on specific follow-up approaches. The teams applied this framework to cardiological, endocrine-metabolic, neurological/cognitive, and psychological disorders, secondary cancers, fertility preservation, and lifestyles. Each team conducted comprehensive literature searches on PubMed, Embase and Cochrane Library databases up to 2020. Screening followed the PRISMA statement for reporting systematic reviews. The reviews report the results of this wide project covering the main areas of late toxicity and conditions in the long-term survival of cHL and DLBCL patients and their follow-up. From a clinical point of view, the series confirmed that the evidence on follow-up tended to focus on solid tumors with scant evidence on hematological malignancies.
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Penchev Georgiev, Iv. "Neurophysiological control of sleep with special emphasis on melatonin." Trakia Journal of Sciences 18, no. 4 (2020): 355–76. http://dx.doi.org/10.15547/tjs.2020.04.011.

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Sleep and wakefulness are two main types of human and animal behavior. On the average human beings spend about one-third of their lives asleep. The sleep-wake cycle is the most important circadian rhythms which alternates in a periodic manner lasting for about 24 hours. Sleep is determined as the natural periodic suspension of consciousness characterized by relative immobility and reduced responsiveness to external stimuli. The researchers have found and identified many special brain structures and systems controlling waking, rapid eye movement (REM) sleep and non-rapid eye (NREM) sleep and the transitions among these states. Currently, there is an enhanced interest of researchers toward sleep and its neurophysiological mechanisms of regulation because the number of people suffering from various sleep disturbance such as insomnia, delayed sleep onset, duration and propensity of sleep, worldwide dramatically increases. In addition to the next day drowsiness, nervousness, tiredness and decreased workability, it has been suggested that sleep is important also for the maintaining of mood, memory and cognitive function of the brain and is essential for the normal functioning of the endocrine and immune systems. More recently, new studies show a sustained link between sleep disorders and different serious health problems, including obesity, insulin resistance, type 2 diabetes mellitus, cardio-vascular diseases and depression. Therefore, the purpose of this review is to summarize and analyze the available data about the neurological control of wakefulness, non-rapid-eye-movement (NREM) sleep and rapid- eye-movement (REM) sleep creating a substantial basis for better understanding different sleep disorders. Special attention is paid on the pharmacological aspects and use of some new classes of sleep promoting agents – melatonin, melatonin receptor agonists and orexin receptor antagonists.
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Franks, Stephen. "Endocrine Aspects of Menstrual Disorders." Medicine 29, no. 11 (November 2001): 34–37. http://dx.doi.org/10.1383/medc.29.11.34.28432.

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Joffe, Russell T., Jennifer S. Brasch, and Glenda M. MacQueen. "Psychiatric aspects of endocrine disorders in women." Psychiatric Clinics of North America 26, no. 3 (September 2003): 683–91. http://dx.doi.org/10.1016/s0193-953x(03)00033-9.

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Wieck, Angelika. "10 Endocrine aspects of postnatal mental disorders." Baillière's Clinical Obstetrics and Gynaecology 3, no. 4 (December 1989): 857–77. http://dx.doi.org/10.1016/s0950-3552(89)80069-0.

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Dissertations / Theses on the topic "Cognition disorders – Endocrine aspects"

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Rodrigues, Mark. "Hormonal and non-hormonal factors associated with cognition in post-menopausal women." University of Western Australia. School of Psychiatry and Clinical Neurosciences, 2004. http://theses.library.uwa.edu.au/adt-WU2004.0075.

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[Truncated abstract.] Alzheimer’s disease (AD) is the most common form of dementia world-wide accounting for more than two thirds of all dementia cases. AD is characterised by the presence of extracellular amyloid plaques, neurofibrillary tangles and congophillic amyloid angiopathy in the brain tissue of affected individuals. Of these neuropathological features the extracellular amyloid plaques are the most characteristic containing a peptide termed amyloid- beta (Aβ); the major protein component of these structures. In addition a number of genetic risk factors for AD have been identified. Of these the ε4 allele of the apolipoprotein E (APOE) gene found on chromosome 19 is considered to be the main genetic risk factor attributing to about 40-60% of all AD cases in most populations. Although there is strong evidence that genetic risk factors play an important role in AD they do not actually trigger the disease process. Deficits in memory and learning are the most common clinical signs of AD in the initial stages of the disease. Neuropsychological tests such as the CAMCOG and California Verbal Learning Test (CVLT) are important diagnostic tools used for the assessment of cognition. The CAMCOG is an accurate and efficient measure of global cognitive ability, while the CVLT is more specific to areas of cognition influenced in the early stages of the disease such as verbal memory. Substantial evidence indicates that changes in sex hormones following menopause in women are important factors in AD. Specifically, the reduced levels of oestrogen in post-menopausal women have been linked to cognitive decline and an increased risk of dementia. In addition the elevated level of the gonadotropins, a characteristic of the post-menopausal period, have been implicated with the disease. Numerous nonhormonal factors such as age and education may also be associated with the development and progression of cognitive decline.
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Heaney, Anthony Patrick. "Aspects of pituitary and adrenal disease." Thesis, Queen's University Belfast, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.295358.

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Pettersson, Anna. "Motor function and cognition : aspects on gait and balance /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-431-7/.

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Rascovsky, Katya. "Neuropsychological aspects of frontotemporal dementia /." Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2005. http://wwwlib.umi.com/cr/ucsd/fullcit?p3167837.

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Lee, Ching-wai Rosa. "The effectiveness of cognition behavior group therapy for mood disorders outpatients in Hong Kong." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2002. http://hub.hku.hk/bib/B29758683.

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Lafferty, Patricia. "THE STABILITY OF FIELD DEPENDENCE AMONG ALCOHOLICS IN TREATMENT AND THE RELATIONSHIP BETWEEN EMBEDDED FIGURES TEST PERFORMANCE AND COGNITIVE IMPAIRMENT." Thesis, The University of Arizona, 1985. http://hdl.handle.net/10150/275265.

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Steuber, Lucas Carl. "Disordered Thought, Disordered Language: A corpus-based description of the speech of individuals undergoing treatment for schizophrenia." PDXScholar, 2011. https://pdxscholar.library.pdx.edu/open_access_etds/63.

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The characteristics of patient speech are used in clinical settings to make assumptions about the thought processes of people with psychotic disorders such as schizophrenia. However, there have not been any studies of the language of people with schizophrenia that present evidence drawn from a large group of speakers. This study employs a combination of quantitative and qualitative methods to determine whether 140 medicated individuals diagnosed with schizophrenia exhibit the linguistic abnormalities claimed in the literature. It also compares the speech of people with schizophrenia with that of people diagnosed with depression in order to assess whether there is a statistically significant difference in presence and/or frequency of abnormal speech between the two groups. Ultimately this study finds that all of the specific types of abnormal language behavior described in the literature do occur among a large group of individuals with schizophrenia. However, many such behaviors also occur among individuals with depression; there was a significant difference between the two groups for three of the twelve categories of language features assessed in this study, which were peculiar word choice, illogicality and distractibility. Further characteristics of the language of individuals with schizophrenia were also found, which could be a basis for improving clinical diagnostic materials.
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Cohen, Diane L. "Psychological correlates of eating disorders: Exploring the continuum perspective." Thesis, University of North Texas, 2002. https://digital.library.unt.edu/ark:/67531/metadc3260/.

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Psychological and behavioral characteristics of female undergraduates with varying levels of disordered eating, as measured by the Questionnaire for Eating Disorder Diagnoses (Q-EDD; Mintz, O'Halloran, Mulholland, & Schneider, 1997), were investigated. Results suggest that the Q-EDD is an appropriate instrument for measuring eating disorder symptomatology. Greater disordered eating was associated with more bulimic, dieting, and weight fluctuation symptoms, higher impression management and approval-seeking needs, more dichotomous thinking, self control, and rigid weight regulation, and increased concern with body shape and dissatisfaction with facial features. Eating-disordered and symptomatic women evidenced more severe eating disorder behaviors and psychological distress than asymptomatic women. Findings are congruent with a redefined discontinuity perspective of eating disorder symptomatology. Treatment implications and campus-wide preventions are suggested.
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陳嘉乾 and Ka-kin King Chan. "Alcohol and cognitive impairment in Chinese older adults: a cross sectional study." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B40733920.

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Niemela-Waller, Kirsi (Kirsi M. ). "Cognitive Dysfunction in Systemic Lupus Erythematosus." Thesis, University of North Texas, 1997. https://digital.library.unt.edu/ark:/67531/metadc278952/.

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The purpose of the study was to determine the point prevalence of cognitive dysfunction in patients with systemic lupus erythematosus (SLE) and to investigate its association with corticosteroids and depression. The severity of dysfunction and the pattern of cognitive changes were examined. This study hypothesized that cognitive dysfunction is common in SLE and many previous studies have underestimated its prevalence, partially due to using limited neuropsychological batteries and insensitive test instruments. It was further hypothesized that the pattern of cognitive changes in SLE patients will resemble that observed in subcortical dementias.
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Books on the topic "Cognition disorders – Endocrine aspects"

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Menopause and the mind: The complete guide to coping with memory loss, foggy thinking, verbal slips, and other cognitive effects of perimenopause and menopause. New York: Free Press, 1999.

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Marco, Cappa, ed. Endocrine involvement in developmental syndromes. Basel: Karger, 2009.

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Heidler, Maria-Dorothea. Kognitive Dysphasien: Differenzialdiagnostik aphasischer und nichtaphasischer zentraler Sprachstörungen sowie therapeutische Konsequenzen. Frankfurt am Main: Peter Lang, 2006.

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1932-, Jewelewicz Raphael, and Warren Michelle P, eds. The menstrual cycle: Physiology, reproductive disorders, and infertility. New York: Oxford University Press, 1993.

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1949-, Kaszniak Alfred W., and Tomoeda Cheryl K, eds. Communication and cognition in normal aging and dementia. Boston: Little, Brown, 1987.

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Bayles, Kathryn A. Communication and cognition in normal aging and dementia. Boston: College Hill Press, 1987.

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Bayles, Kathryn A. Communication and cognition in normal aging and dementia. Austin, Tex: Pro-Ed, 1991.

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1949-, Kaszniak Alfred W., and Tomoeda Cheryl K, eds. Communication and cognition in normal aging and dementia. London: Taylor & Francis, 1987.

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Anderson, Nicole D. Living with mild cognitive impairment: A guide to maximizing brain health and reducing risk of dementia. Oxford: Oxford University Press, 2012.

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J, Papadatos Constantine, and Bartsokas Chrēstos Spyrou, eds. Endocrine genetics and genetics of growth: Proceedings of the Fourth International Clinical Genetics Seminar held in Athens, Greece, May 22-25, 1985. New York: Liss, 1985.

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Book chapters on the topic "Cognition disorders – Endocrine aspects"

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Giuzio, E., M. Bria, F. Romeo, M. Misasi, and C. Brancati. "Imaging, Ultrastructural Aspects, and Trace Elements in the Thalassemic Condrodystrophy." In Endocrine Disorders in Thalassemia, 59–64. Milano: Springer Milan, 1995. http://dx.doi.org/10.1007/978-88-470-2183-9_9.

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Gimm, O. "Multiple Endocrine Neoplasia Type 2: Clinical Aspects." In Genetic Disorders of Endocrine Neoplasia, 103–30. Basel: KARGER, 2001. http://dx.doi.org/10.1159/000061050.

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Mulligan, L. M. "Multiple Endocrine Neoplasia Type 2: Molecular Aspects." In Genetic Disorders of Endocrine Neoplasia, 81–102. Basel: KARGER, 2001. http://dx.doi.org/10.1159/000061051.

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Marsh, D. J., and C. A. Stratakis. "Hamartoma and Lentiginosis Syndromes: Clinical and Molecular Aspects." In Genetic Disorders of Endocrine Neoplasia, 167–213. Basel: KARGER, 2001. http://dx.doi.org/10.1159/000061045.

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Gonciulea, Anda R., and Suzanne M. Jan de Beur. "Acute Medical Aspects Related to Phosphate Disorders." In Endocrine and Metabolic Medical Emergencies, 422–32. Oxford, UK: John Wiley & Sons, Ltd, 2018. http://dx.doi.org/10.1002/9781119374800.ch25.

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Chandrasekharappa, S. C., and B. Tean Teh. "Clinical and Molecular Aspects of Multiple Endocrine Neoplasia Type 1." In Genetic Disorders of Endocrine Neoplasia, 50–80. Basel: KARGER, 2001. http://dx.doi.org/10.1159/000061047.

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Levine, Barton S., and Arnold J. Felsenfeld. "Acute Medical Aspects Related to Phosphate Disorders." In Endocrine and Metabolic Medical Emergencies: A Clinician's Guide, 202–8. 2055 L Street, NW, Suite 600, Washington, DC 20036 www.endo-society.org: The Endocrine Society, 2014. http://dx.doi.org/10.1210/eme.9781936704811.ch20.

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Bredella, Miriam A., and Bruno C. Vande Berg. "Metabolic-Endocrine." In IDKD Springer Series, 169–82. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-71281-5_12.

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AbstractAll components of the musculoskeletal system can be involved by metabolic disorders as a result of endocrine diseases, genetic alterations, and environmental or nutritional aspects, with important worldwide variations in prevalence and severity. Early detection of these disorders is crucial because of the efficacy of preventive measures and availability of treatments. The current chapter will focus on the imaging appearance of metabolic disorders of bone marrow and of the mineralized skeleton. Marrow and bone disorders in athletes, the elderly, and individuals with eating disorders will be reviewed.
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Daskalakis, Nikolaos P., Marlon A. McGill, Amy Lehrner, and Rachel Yehuda. "Endocrine Aspects of PTSD: Hypothalamic-Pituitary-Adrenal (HPA) Axis and Beyond." In Comprehensive Guide to Post-Traumatic Stress Disorders, 245–60. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-08359-9_130.

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Linder, Jessica, Maria Miteva, and Paolo Romanelli. "Pigmentary Deposition Disorders." In Clinical and Pathological Aspects of Skin Diseases in Endocrine, Metabolic, Nutritional and Deposition Disease, 171–80. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-60761-181-3_22.

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