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1

Sahakian, Barbara J., Annette B. Bruhl, Jennifer Cook, et al. "The impact of neuroscience on society: cognitive enhancement in neuropsychiatric disorders and in healthy people." Philosophical Transactions of the Royal Society B: Biological Sciences 370, no. 1677 (2015): 20140214. http://dx.doi.org/10.1098/rstb.2014.0214.

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In addition to causing distress and disability to the individual, neuropsychiatric disorders are also extremely expensive to society and governments. These disorders are both common and debilitating and impact on cognition, functionality and wellbeing. Cognitive enhancing drugs, such as cholinesterase inhibitors and methylphenidate, are used to treat cognitive dysfunction in Alzheimer's disease and attention deficit hyperactivity disorder, respectively. Other cognitive enhancers include specific computerized cognitive training and devices. An example of a novel form of cognitive enhancement us
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de Sousa, Paulo, William Sellwood, Martin Griffiths, and Richard P. Bentall. "Disorganisation, thought disorder and socio-cognitive functioning in schizophrenia spectrum disorders." British Journal of Psychiatry 214, no. 2 (2018): 103–12. http://dx.doi.org/10.1192/bjp.2018.160.

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BackgroundPoor social cognition is prevalent in schizophrenia spectrum disorders. Some authors argue that these effects are symptom-specific and that socio-cognitive difficulties (e.g. theory of mind) are strongly associated with thought disorder and symptoms of disorganisation.AimsThe current review tests the strength of this association.MethodWe meta-analysed studies published between 1980 and 2016 that tested the association between social cognition and these symptoms in schizophrenia spectrum disorders.ResultsOur search (PsycINFO, MEDLINE and Web of Science) identified 123 studies (N = 910
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Holmes, Monique C., Caroline L. Donovan, and Lara J. Farrell. "A Disorder-Specific, Cognitively Focused Group Treatment for Childhood Generalized Anxiety Disorder: Development and Case Illustration of the No Worries! Program." Journal of Cognitive Psychotherapy 29, no. 4 (2015): 275–301. http://dx.doi.org/10.1891/0889-8391.29.4.275.

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Generalized anxiety disorder (GAD) is a highly prevalent, chronic, and costly mental disorder in children, and there is a comparative lack of research on specific treatments for GAD, relative to other anxiety disorders. Furthermore, treatment programs for child GAD, unlike those for adults, are almost uniformly transdiagnostic in nature and do not specifically target the cognitive variables (e.g., intolerance of uncertainty, negative beliefs about worry, cognitive avoidance, and negative problem orientation) demonstrated to be correlated with the disorder. However, helping children to understa
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Oliver, Lindsay, Iska Moxon-Emre, Aristotle Voineskos, and Stephanie Ameis. "M49. BEHAVIOURAL SOCIAL COGNITION IN SCHIZOPHRENIA SPECTRUM DISORDERS IN COMPARISON TO AUTISM SPECTRUM DISORDER: A SYSTEMATIC REVIEW AND META-ANALYSIS." Schizophrenia Bulletin 46, Supplement_1 (2020): S152—S153. http://dx.doi.org/10.1093/schbul/sbaa030.361.

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Abstract Background Schizophrenia spectrum disorders (SSDs) and autism spectrum disorder (ASD) both feature social cognitive deficits, which are highly debilitating. These include lower-level processes (e.g. emotion recognition), thought to be subserved by a frontoparietal mirroring network, and higher-level mentalizing processes (e.g. theory of mind), involving cortical midline and lateral temporal brain regions. Across both disorders, impairments in social cognition persist over time, drive disability, and predict functional outcome. Overlapping symptoms in SSDs and ASD have long been recogn
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Veale, David, and Anna Stout. "Cognitive behaviour therapy meets psychopharmacotherapy." Cognitive Behaviour Therapist 3, no. 4 (2010): 117–31. http://dx.doi.org/10.1017/s1754470x10000127.

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AbstractThis article provides an overview of the role of psychopharmacotherapy in common emotional disorders for cognitive behaviour therapists. We consider some of the philosophical difference between CBT and medication, when medication might interfere with CBT, when it may enhance outcome and when it might be safely discontinued. We highlight how to differentiate side-effects and symptoms of discontinuation of antidepressants from that of the underlying disorder. The scope of this article is confined to common emotional disorders and does not discuss the interaction of CBT with medication in
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Sachdev, P. S., D. M. Lipnicki, J. D. Crawford, and H. Brodaty. "The Vascular Behavioral and Cognitive Disorders criteria for vascular cognitive disorders: a validation study." European Journal of Neurology 26, no. 9 (2019): 1161–67. http://dx.doi.org/10.1111/ene.13960.

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Savinkov, M. A., O. Yu Ustinova, A. E. Nosov, Yu A. Ivashova, and V. G. Kostarev. "Risks related to cognitive disorders development in workers with different work experience employed at an oil extracting facility." Health Risk Analysis, no. 2 (June 2021): 83–93. http://dx.doi.org/10.21668/health.risk/2021.2.08.eng.

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Our research goal was to examine cognitive functions parameters in dynamics among workers employed at an oil extracting facility depending on their work experience under exposure to adverse occupational factors. We estimated cognitive functions in 292 oil and gas extraction operators who were exposed to adverse occupational factors (aromatic hydrocarbons, hydrogen sulphide, occupational noise, labor hardness, and adverse microclimate). The reference group consisted of 65 administrative workers employed at the same enterprise. All the examined people were males aged 20–65; they were divided int
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Lense, Miriam D., Eniko Ladányi, Tal-Chen Rabinowitch, Laurel Trainor, and Reyna Gordon. "Rhythm and timing as vulnerabilities in neurodevelopmental disorders." Philosophical Transactions of the Royal Society B: Biological Sciences 376, no. 1835 (2021): 20200327. http://dx.doi.org/10.1098/rstb.2020.0327.

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Millions of children are impacted by neurodevelopmental disorders (NDDs), which unfold early in life, have varying genetic etiologies and can involve a variety of specific or generalized impairments in social, cognitive and motor functioning requiring potentially lifelong specialized supports. While specific disorders vary in their domain of primary deficit (e.g. autism spectrum disorder (social), attention-deficit/hyperactivity disorder (attention), developmental coordination disorder (motor) and developmental language disorder (language)), comorbidities between NDDs are common. Intriguingly,
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Aardema, Frederick, and Kieron O’Connor. "Seeing White Bears That Are Not There: Inference Processes in Obsessions." Journal of Cognitive Psychotherapy 17, no. 1 (2003): 23–37. http://dx.doi.org/10.1891/jcop.17.1.23.58270.

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Meta-cognition refers to the notion of thoughts about one’s own thoughts and has been defined as knowledge and cognition about cognitive phenomena (Flavell, 1979). In recent years, meta-cognitive models have provided accounts of the maintenance of anxiety disorders (e.g., Wells, 2000). Meta-cognitive models would argue that the thoughts about the appearance and utility of otherwise normal thoughts generate anxiety. In this article we apply a meta-cognitive approach to understanding obsessions but, rather than thoughts about thoughts, we suggest that the ruminations in obsessive-compulsive diso
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10

Hong, Jeong-Min. "Perioperative brain health: strategies to prevent perioperative neurocognitive disorders." Journal of the Korean Medical Association 63, no. 9 (2020): 540–47. http://dx.doi.org/10.5124/jkma.2020.63.9.540.

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Cognitive changes in patients after anesthesia and surgery have been recognized for over 100 years. Research on postoperative cognitive changes accelerated in the 1980s and the term postoperative cognitive dysfunction emerged, which was used until recently. Postoperative cognitive dysfunction has been used in research to describe an objectively measurable decline in cognitive function using neuropsychological tests. This dysfunction had significant heterogeneity in the type, number of tests, timing of tests, and the criteria for change. Therefore, a recent article recommended a new nomenclatur
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Iliadou, Paraskevi, Ioannis Paliokas, Stelios Zygouris, et al. "A Comparison of Traditional and Serious Game-Based Digital Markers of Cognition in Older Adults with Mild Cognitive Impairment and Healthy Controls." Journal of Alzheimer's Disease 79, no. 4 (2021): 1747–59. http://dx.doi.org/10.3233/jad-201300.

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Background: Electroencephalography (EEG) has been used to assess brain activity while users are playing an immersive serious game. Objective: To assess differences in brain activation as measured with a non-intrusive wearable EEG device, differences in game performance and correlations between EEG power, game performance and global cognition, between cognitively impaired and non-impaired older adults, during the administration of a novel self-administered serious game-based test, the Virtual Supermarket Test (VST). Methods: 43 older adults with subjective cognitive decline (SCD) and 33 older a
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Ioannidis, Panagiotis, and Dimitris Karacostas. "How Reversible are ‘˜Reversible Dementias’?" European Neurological Review 6, no. 4 (2011): 230. http://dx.doi.org/10.17925/enr.2011.06.04.230.

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Reversible dementias comprise different groups of disorders of variable aetiologies, such as structural brain lesions or metabolic, infectious, toxic, autoimmune, paraneoplastic and psychiatric disorders. When patients present with cognitive symptoms, especially in the younger age groups, the first thought of the attending neurologist should be to try to identify an underlying treatable cause. The incidence of degenerative dementia rises with older age and its symptoms progressively become more evident and typical; in such cases, a differential diagnosis is limited and the chance of uncovering
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Santos, Telma, Joaquim Pinheiro, and Pedro Barros. "Cognitive Impairment in Multiple Sclerosis." European Neurological Review 10, no. 2 (2015): 157. http://dx.doi.org/10.17925/enr.2015.10.02.157.

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Multiple sclerosis (MS) is a chronic inflammatory demyelinating disorder of the central nervous system. Although MS has been known since the 19th century, cognitive impairment (CI) was only recognised as an important feature of MS over the past 30 years. The reason is that, until recently, MS was perceived exclusively as a white matter disease, and cognition was thought to depend solely on the cortex. This article aims to review the prevalence, risk factors, profile and diagnosis of CI in MS. Imaging correlates and treatment will also be briefly discussed.
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Walterfang, Mark, and Dennis Velakoulis. "Niemann-Pick Disease Type C in Adulthood - A Psychiatric and Neurological Disorder." European Neurological Review 5, no. 1 (2010): 83. http://dx.doi.org/10.17925/enr.2010.05.01.83.

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Niemann-Pick disease type C (NPC) is a rare neurovisceral lipid storage disorder resulting from autosomal recessively inherited loss-of-function mutations in eitherNpc1orNpc2. This disrupts intracellular lipid transport, leading to the accumulation of lipid products in the late endosomes and lysosomes. Affecting both children and adults, it exhibits a less rapid disease course in older patients, where it is characterised by slow cognitive decline, neuropsychiatric illness, ataxia and dystonia. As NPC is heterogeneous in presentation, it is often misdiagnosed as other movement or psychiatric di
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Lackner, Jeffrey M. "No Brain, No Gain: The Role of Cognitive Processes in Irritable Bowel Syndrome." Journal of Cognitive Psychotherapy 19, no. 2 (2005): 125–36. http://dx.doi.org/10.1891/jcop.19.2.125.66788.

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This article seeks to summarize research supporting the influence of psychological factors in general and cognitive factors in shaping the onset, expression, and outcome of irritable bowel syndrome (IBS). To this end, 6 lines of research are reviewed, with a focus on studies showing (a) the lack of correspondence between IBS symptoms and measured gut motility; (b) IBS patients as a group tend to label visceral sensations negatively and show a lower tolerance for visceral sensations; (c) neuroimaging studies showing abnormalities in central pain processing mechanisms in response to rectal stimu
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Delgadillo, Mia, Megan Frank, Aidan Boese, Tilman Schulte, and J. Kaci Fairchild. "Psychiatric Disorders and Mild Cognitive Impairment in Older Veterans With Subjective Memory Complaints." Innovation in Aging 4, Supplement_1 (2020): 295. http://dx.doi.org/10.1093/geroni/igaa057.944.

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Abstract Psychiatric disorders pose a unique risk for Alzheimer’s disease (AD). Prior research indicates psychiatric disorders in MCI increase AD vulnerability. Less research has been done to understand how psychiatric disorders may affect the development of MCI. Understanding these potentially modifiable risk factors is important as they may represent a potential target of intervention for secondary prevention of AD. The present study examines the relationship between psychiatric disorders and amnestic MCI (aMCI) in a sample of Veterans with subjective memory complaints. The sample included 1
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Bonuck, Karen, and Roy Grant. "Sleep Problems and Early Developmental Delay: Implications for Early Intervention Programs." Intellectual and Developmental Disabilities 50, no. 1 (2012): 41–52. http://dx.doi.org/10.1352/1934-9556-50.1.41.

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Abstract Sleep disorders negatively impact behavior, cognition, and growth—the same areas targeted by early intervention. Conversely, developmental delays and disabilities may themselves precipitate sleep disorders. Young children with developmental delays experience sleep disorders at a higher rate than do typically developing children; the most common types are difficulties initiating or maintaining sleep and sleep disordered breathing. To date, attention has been focused on sleep problems in children with specific conditions (e.g., autism, genetic syndromes, prematurity, and seizure disorde
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18

Disner, Seth G., Mark D. Kramer, Nathaniel W. Nelson, et al. "Predictors of Postdeployment Functioning in Combat-Exposed U.S. Military Veterans." Clinical Psychological Science 5, no. 4 (2017): 650–63. http://dx.doi.org/10.1177/2167702617703436.

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Posttraumatic stress disorder (PTSD) and sequelae of mild traumatic brain injury (mTBI) are presumed to contribute to reintegration difficulties in combat-exposed veterans. Yet their relative impacts on postdeployment functioning are not well understood. The current study used structural equation modeling (SEM) to clarify the extent to which symptoms of internalizing disorders (e.g., depression, anxiety), mTBI symptoms, and cognitive performance are associated with functional impairment in 295 combat-exposed veterans. SEM results showed that internalizing symptoms most significantly predicted
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19

Sagar, H. J. "Specificity of Cognitive Impairment in Neurological Disease: A Methodological Critique of Parkinson’s Disease." Behavioural Neurology 4, no. 2 (1991): 89–102. http://dx.doi.org/10.1155/1991/625647.

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Multiple cognitive deficits have been recognized in many neurological disorders but the specificity of the findings and the relationship to the underlying neuropathology remain obscure. Definitions of dementia have been proposed based on symptom profiles of the cognitive disorder and qualitative differences have been claimed between dementias of different aetiology. Some conditions have been claimed to show patterns of cognitive deficit that are distinguished from dementia and related to specific neuropathology or psychological processes, e.g. frontal lobe deficits in Parkinson's disease. Some
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20

Levinson, Harold N. "The Cerebellar-Vestibular Predisposition to Anxiety Disorders." Perceptual and Motor Skills 68, no. 1 (1989): 323–38. http://dx.doi.org/10.2466/pms.1989.68.1.323.

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To test for a cerebellar-vestibular (CV) predisposition to anxiety disorder, 402 consecutively referred subjects with varying anxiety symptoms were separated into eight DSM-III—R diagnostic categories and evaluated for CV dysfunction, using neurological and electronystagmographic (ENG) examinations. Of the total sample, 94% evidenced CV-dysfunction on the basis of two or more abnormal neurological or ENG parameters per subject. All DSM-III—R diagnostic anxiety-disorder categories contained a high percentage of abnormal neurological and ENG parameters, regardless of the size of the subsample. M
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Torrent, Carla, Anabel Martínez-Arán, Claire Daban, et al. "Cognitive impairment in bipolar II disorder." British Journal of Psychiatry 189, no. 3 (2006): 254–59. http://dx.doi.org/10.1192/bjp.bp.105.017269.

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BackgroundPersistent impairments in neurocognitive function have been described in bipolar disorder.AimsTo compare the cognitive performance of patients with bipolar II disorder with that of patients with bipolar I disorder and a healthy control group.MethodThe study included 71 euthymic patients with bipolar disorder (38 bipolar I, 33 bipolar II), who were compared on clinical and neuropsychological variables (e.g. executive function, attention, verbal and visual memory) and contrasted with 35 healthy controls on cognitive performance.ResultsCompared with controls, both bipolar groups showed
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Bijsterbosch, Janine, Stephen Smith, Sophie Forster, Oliver P. John, and Sonia J. Bishop. "Resting State Correlates of Subdimensions of Anxious Affect." Journal of Cognitive Neuroscience 26, no. 4 (2014): 914–26. http://dx.doi.org/10.1162/jocn_a_00512.

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Resting state fMRI may help identify markers of risk for affective disorder. Given the comorbidity of anxiety and depressive disorders and the heterogeneity of these disorders as defined by DSM, an important challenge is to identify alterations in resting state brain connectivity uniquely associated with distinct profiles of negative affect. The current study aimed to address this by identifying differences in brain connectivity specifically linked to cognitive and physiological profiles of anxiety, controlling for depressed affect. We adopted a two-stage multivariate approach. Hierarchical cl
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Wu, Mark, Paul Rosenberg, Adam Spira, and Alexandra Wennberg. "Sleep Disturbance, Cognitive Decline, and Dementia: A Review." Seminars in Neurology 37, no. 04 (2017): 395–406. http://dx.doi.org/10.1055/s-0037-1604351.

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AbstractApproximately half of older people report sleep disturbances, which are associated with various health conditions, including neurodegenerative disease and dementia. Indeed, 60 to 70% of people with cognitive impairment or dementia have sleep disturbances, which are linked to poorer disease prognosis. Sleep disturbances in people with dementia have long been recognized and studied; however, in the past 10 years, researchers have begun to study disturbed sleep, including sleep fragmentation, abnormal sleep duration, and sleep disorders, as risk factors for dementia. In this review the au
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Robinson, Susan, and Max Birchwood. "The Relationship Between Catastrophic Cognitions and the Components of Panic Disorder." Journal of Cognitive Psychotherapy 5, no. 3 (1991): 175–86. http://dx.doi.org/10.1891/0889-8391.5.3.175.

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Cognitive models of panic postulate that panic attacks arise from the catastrophic misinterpretation of somatic symptoms. Hitherto, research has concentrated on the link between cognitions and the somatic sensations experienced during panic attacks; little attention has been directed towards the relationship between cognitions and other critical components of Panic Disorder (e.g., avoidance behavior). Fifty-eight patients presenting with Panic Disorder with Agoraphobia (DSM-III-R) rated their degree of belief in nine “core” catastrophic cognitions and completed self-report measures of the crit
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Riskind, John H., and Neil A. Rector. "Beyond Belief: Incremental Prediction of Obsessive-Compulsive Disorder by Looming Vulnerability Illusions." Journal of Cognitive Psychotherapy 21, no. 3 (2007): 243–56. http://dx.doi.org/10.1891/088983907781494564.

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Cognitive models argue that obsessions and compulsions arise from distorted beliefs and exaggerated interpretations of intrusive thoughts. While these models have led to important advances, recent research has suggested the need to go beyond the factors the models identify. One new factor to consider may involve looming vulnerability, the production of dynamic mental scenarios of danger outcomes (e.g., contamination, harming, losing wanted possessions) as rushing through time and space and escalating in odds of harm for the self. Looming vulnerability is a different form of cognition that diff
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Seel, Ronald T., Stephen Macciocchi, Jeffrey S. Kreutzer, Darryl Kaelin, and Douglas I. Katz. "Diagnosing Major Depression Following Moderate to Severe Traumatic Brain Injury – Evidence-based Recommendations for Clinicians." European Neurological Review 6, no. 1 (2011): 24. http://dx.doi.org/10.17925/enr.2011.06.01.24.

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While major depression (MD) is the most common psychiatric disorder following traumatic brain injury (TBI), diagnosing MD can be challenging due to cognitive, emotional and somatic symptoms that overlap with TBI and other psychiatric disorders. Current evidence suggests that the Diagnostic and Statistical Manual of Mental Disorders (DSM-IV) works well in the TBI population. The presence of ‘organic’ TBI sequelae that overlap with DSM-IV MD criteria do not appear to lead to false-positive MD diagnoses. Rumination, self-criticism and guilt may best differentiate depressed from non-depressed pers
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Donohoe, G., I. J. Deary, D. C. Glahn, A. K. Malhotra, and K. E. Burdick. "Neurocognitive phenomics: examining the genetic basis of cognitive abilities." Psychological Medicine 43, no. 10 (2012): 2027–36. http://dx.doi.org/10.1017/s0033291712002656.

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Cognitive deficits are core to the disability associated with many psychiatric disorders. Both variation in cognition and psychiatric risk show substantial heritability, with overlapping genetic variants contributing to both. Unsurprisingly, therefore, these fields have been mutually beneficial: just as cognitive studies of psychiatric risk variants may identify genes involved in cognition, so too can genome-wide studies based on cognitive phenotypes lead to genes relevant to psychiatric aetiology. The purpose of this review is to consider the main issues involved in the phenotypic characteriz
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Millichap, J. Gordon. "EEG Theta Activity, Cognitive Disorders and ADHD." Pediatric Neurology Briefs 19, no. 4 (2005): 30. http://dx.doi.org/10.15844/pedneurbriefs-19-4-6.

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Zaheed, Afsara B., Neika Sharifian, A. Zarina Kraal, Ketlyne Sol, Alyssia Hence, and Laura B. Zahodne. "Unique Effects of Perceived Neighborhood Physical Disorder and Social Cohesion on Episodic Memory and Semantic Fluency." Archives of Clinical Neuropsychology 34, no. 8 (2019): 1346–55. http://dx.doi.org/10.1093/arclin/acy098.

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Abstract Objective Objective measures of neighborhood quality are associated with physical and mental health outcomes for older adults, but the relationship between perceived neighborhood quality and cognitive health has not been fully explored. Furthermore, positive and negative neighborhood characteristics may influence cognition through different mechanisms. The present study aimed to determine whether perceptions of neighborhood quality predict cognitive functioning in two domains, above and beyond individual-level risk factors, in a nationally representative sample of older adults. Method
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Arioli, Maria, Chiara Crespi, and Nicola Canessa. "Social Cognition through the Lens of Cognitive and Clinical Neuroscience." BioMed Research International 2018 (September 13, 2018): 1–18. http://dx.doi.org/10.1155/2018/4283427.

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Social cognition refers to a set of processes, ranging from perception to decision-making, underlying the ability to decode others’ intentions and behaviors to plan actions fitting with social and moral, besides individual and economic considerations. Its centrality in everyday life reflects the neural complexity of social processing and the ubiquity of social cognitive deficits in different pathological conditions. Social cognitive processes can be clustered in three domains associated with (a) perceptual processing of social information such as faces and emotional expressions (social percept
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Vishnu Priya K. and Kavitha A. "Investigations on the Brain Connectivity Parameters for Co-Morbidities of Autism Using EEG." International Journal of Software Science and Computational Intelligence 10, no. 2 (2018): 50–65. http://dx.doi.org/10.4018/ijssci.2018040104.

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This article describes how the Autism Spectrum Disorder (ASD) is a collection of heterogeneous disorders with prevalent cognitive and behavioral abnormalities. ASD is generally considered a life-long disability occurring as a stand-alone disorder but it occurs with possible co-morbid conditions. Electroencephalography (EEG) studies have been identified as one of the most widely used tool for assessing the cognitive functions with strong evidences of stable pattern of EEG associated with ASD. With the understanding of the co-morbidities and the pathophysiology, it needs an appropriate signal pr
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Guerra, Nancy G., Paul Boxer, and Tia E. Kim. "A Cognitive-Ecological Approach to Serving Students with Emotional and Behavioral Disorders: Application to Aggressive Behavior." Behavioral Disorders 30, no. 3 (2005): 277–88. http://dx.doi.org/10.1177/019874290503000303.

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In this article we present a cognitive-ecological model for understanding and preventing emotional and behavioral difficulties and propose directions for school-based intervention programs, particularly with aggressive children. In the cognitive-ecological framework, intervention efforts should target certain cognitive skills (e.g., skills that encourage attention to multiple cues in a setting) and knowledge structures (e.g., normative beliefs about appropriate responses to conflict) across multiple contexts that change over time (e.g., classroom, peer, school, family). We also emphasize the i
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Alves, Guido, and Jan Petter Larsen. "Population-based Studies on the Clinical Progression of Motor and Non-motor Features in Parkinson’s Disease." European Neurological Review 4, no. 1 (2009): 36. http://dx.doi.org/10.17925/enr.2009.04.01.36.

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Although Parkinson’s disease (PD) is a movement disorder in which tremor, rigidity and bradykinesia constitute the cardinal signs of the disease, it is increasingly recognised to be associated with a wide range of motor and non-motor features. Population-based studies demonstrate that the motor course in PD is generally slowly progressive, with average annual progression rates of ≤3%. However, there is remarkable inter-individual variation in the clinical course of PD, with advanced age and predominant postural instability and gait difficulties being major risk factors for more rapid motor and
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Khalil, M. A., A. A. Saleh, N. M. El-Fayoumy, and S. M. Gohar. "Social and nonsocial cognitive functions in patients with schizophrenia: A comparative neuropsychological and neurophysiological study." European Psychiatry 41, S1 (2017): s817. http://dx.doi.org/10.1016/j.eurpsy.2017.01.1589.

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BackgroundPatients with schizophrenia suffer from cognitive deficits in seven domains in addition to social cognition. P300 latency and amplitude have been linked in these patients to the basic cognitive deficits.ObjectivesComparing patients suffering from schizophrenia with matched healthy subjects as regards auditory event related potential tests as measured by P300.Subjects and methodsFifty-two subjects were divided into 2 groups: group (A): 27 patients with schizophrenia according to the diagnostic and statistical manual of mental disorders-text revised (DSM-IV TR). Those with current subs
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Young, A. "Cognitive Impairment in Bipolar: Neurodevelopmental or Neuroprogressive?" European Psychiatry 41, S1 (2017): S13. http://dx.doi.org/10.1016/j.eurpsy.2017.01.092.

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BackgroundBipolar Disorders (BD) are common and complex diseases. Recent findings have provided evidence that impairments in cognition are evident in the various sub-groups of Bipolar Disorder and persist after resolution of acute episodes.MethodAn opinion paper based on a narrative review of the field.ResultsQuantifiable cognitive deficits are clearly found in Bipolar 1 and Bipolar 2 Disorders. These persist after recovery from acute episodes. The aetiopathogenesis of these phenomena is likely to be multifactorial. It seems clear that these cognitive impairments are not in general neurodevelo
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Simeon, J. G., H. B. Ferguson, and J. Van Wyck Fleet. "Bupropion Effects in Attention Deficit and Conduct Disorders." Canadian Journal of Psychiatry 31, no. 6 (1986): 581–85. http://dx.doi.org/10.1177/070674378603100617.

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Children with Attention Deficit and/or Conduct Disorders were treated with bupropion, a new antidepressant, to determine its clinical, cognitive, and EEG effects. Seventeen male patients (age range 7 to 13.4 years; mean 10.4) participated in an open clinical trial consisting of a baseline placebo period (4 weeks), bupropion therapy (8 weeks), and post-drug placebo (2 weeks). Evaluations included clinical assessments, parents, teachers, and self-ratings; cognitive tels and blood level measurements of bupropion. Fifteen patients received a daily maximum of 150 mg, one received 100 mg and one 50
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McIntyre, Roger. "Major depressive disorder and cognitive impairment." Journal of Psychiatry & Neuroscience 39, no. 5 (2014): E36—E37. http://dx.doi.org/10.1503/jpn.140050.

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Marchand, André, and Michel Wapler. "L'effet des troubles de la personnalité sur la réponse au traitement béhavioural-cognitif du trouble panique avec agoraphobie." Canadian Journal of Psychiatry 38, no. 3 (1993): 163–66. http://dx.doi.org/10.1177/070674379303800302.

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This study measures the effect of personality disorders on the efficacy of treatment of agoraphobia. Forty-one patients suffering from panic disorder with agoraphobia are evaluated for the presence of a personality disorder (according to DSM-III-R criteria) before the onset of treatment. The treatment consists in 14 sessions of behavioural cognitive therapy. Various measures of agoraphobic avoidance are obtained before (pre-test) and after the end of treatment (post-test, three month follow-up). Effect of treatment reaches statistical and clinical significance for all patients. Grouping of pat
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Bailey, Robin, and Adrian Wells. "Does Metacognition Make a Unique Contribution to Health Anxiety When Controlling for Neuroticism, Illness Cognition, and Somatosensory Amplification?" Journal of Cognitive Psychotherapy 27, no. 4 (2013): 327–37. http://dx.doi.org/10.1891/0889-8391.27.4.327.

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Somatosensory amplification (e.g., Barsky, 1992), illness cognition (e.g., Salkovskis & Warwick, 1986), and neuroticism (e.g., Noyes et al., 2003) have all been linked to health anxiety. The first two factors are disorder specific; however, neuroticism is a general vulnerability connected to a range of disorders. In the metacognitive model (Wells, 2009), beliefs about thinking have been implicated in the development of psychopathologies, but little is known about the contribution of individual differences in metacognition to health anxiety, specifically. A cross-sectional design was employ
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Dom, G. "Pharmacological Management of Impulsivity and Compulsivity." European Psychiatry 41, S1 (2017): S22. http://dx.doi.org/10.1016/j.eurpsy.2017.01.121.

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Increasingly patients present themselves to psychiatrists and other care providers with a specific request for treatment of one or more behavioral addictions. From a pathogenic point of view impulsivity and compulsivity are important drivers of these behavioral disorders, and as such may represent a target of pharmacological and broader neurobiological, e.g. Neuro-stimulation, treatment. Although currently treatment as usual has a focus on psychosocial and cognitive behavioral interventions, interest is growing toward the pharmacological interventions. In the presentation a state of the art wi
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Wall, Kathryn, Jessica Stark, Alexa Schillaci, et al. "The Enhanced Interactive Physical and Cognitive Exercise System (iPACESTM v2.0): Pilot Clinical Trial of an In-Home iPad-Based Neuro-Exergame for Mild Cognitive Impairment (MCI)." Journal of Clinical Medicine 7, no. 9 (2018): 249. http://dx.doi.org/10.3390/jcm7090249.

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Given increasing longevity worldwide, older adults and caregivers are seeking ways to curb cognitive decline especially for those with mild cognitive impairment (MCI, now mild neurocognitive disorder, mNCD, Diagnostic and Statistical Manual of Mental Disorders, 5th ed. (DSM-V). This quasi-experimental, within-subjects pilot clinical trial was designed to replicate and extend the study of cognitive benefits for MCI by improving upon our prior interactive Physical and Cognitive Exercise Study (iPACESTM v1.0) by increasing the usability of the neuro-exergame and exploring possible underlying neur
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Alexandrova, E. A., E. V. Parshina, I. V. Borodacheva, et al. "Possibilities of daytime anxolytics in the correction of residual neurological manifestations of COVID-19." Meditsinskiy sovet = Medical Council, no. 12 (September 19, 2021): 50–60. http://dx.doi.org/10.21518/2079-701x-2021-12-50-60.

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Introduction. In addition to acute manifestations, coronavirus infection is characterized by long-lasting symptoms: asthenia, somatic vegetative manifestations, sleep disorders and psychoemotional background, the question of therapeutic correction of which is especially relevant.The aim of the study was to study the mental, somatoform and cognitive aspects of anxiety disorders after coronavirus infection during treatment with tofizopam (Grandaxin®) at 150 mg / day.Materials and methods. The study involved patients who had a new coronavirus infection, who 4 weeks after the end of treatment for
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De Cola, Maria Cristina, Giuseppe Triglia, Martina Camera, et al. "Effect of neurological screening on early dementia detection in southern Italy." Journal of International Medical Research 48, no. 10 (2020): 030006052094976. http://dx.doi.org/10.1177/0300060520949763.

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Objective Population screening can facilitate early diagnosis of dementia and improve disease management. This study examined the effects of a screening campaign for neurodegenerative disorders on the early diagnosis of dementia using 2-year follow-up data. Methods A 5-day screening campaign was conducted that comprised neurological, neuropsychological and other specialist examinations. Identification of alterations during the neurological examination was followed-up by further diagnostic examinations to confirm the neurological impairment. Results Neurological alterations were observed in 39%
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Bordet, R., and D. Deplanque. "Brain–liver axis: a new pathway for cognitive disorders related to hepatic fibrosis." European Journal of Neurology 27, no. 11 (2020): 2111–12. http://dx.doi.org/10.1111/ene.14454.

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Brown, Jerrod, and Diane Harr. "Perceptions of Fetal Alcohol Spectrum Disorder (FASD) at a Mental Health Outpatient Treatment Provider in Minnesota." International Journal of Environmental Research and Public Health 16, no. 1 (2018): 16. http://dx.doi.org/10.3390/ijerph16010016.

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Resulting from prenatal exposure to alcohol, Fetal Alcohol Spectrum Disorder (FASD) is characterized by deficits in adaptive and cognitive functioning. This disorder is typically accompanied by co-occurring disorders and conditions (e.g., mood, anxiety, psychosis, and substance use disorders). This complicated presentation of diverse symptoms makes the process of screening, assessing, and diagnosing FASD very difficult, limiting the likelihood that clients receive the treatment and services that they need. Although mental health care providers have an opportunity to intervene on behalf of clie
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Maggio, Maria Grazia, Antonino Naro, Gianluca La Rosa, et al. "Virtual Reality Based Cognitive Rehabilitation in Minimally Conscious State: A Case Report with EEG Findings and Systematic Literature Review." Brain Sciences 10, no. 7 (2020): 414. http://dx.doi.org/10.3390/brainsci10070414.

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Chronic disorders of consciousness cause a total or partial and fluctuating unawareness of the surrounding environment. Virtual reality (VR) can be useful as a diagnostic and/or a neurorehabilitation tool, and its effects can be monitored by means of both clinical and electroencephalography (EEG) data recording of brain activity. We reported on the case of a 17-year-old patient with a disorder of consciousness (DoC) who was provided with VR training to improve her cognitive-behavioral outcomes, which were assessed using clinical scales (the Coma Recovery Scale-Revised, the Disability Rating Sc
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Mammarella, Nicola. "Towards the Affective Cognition Approach to Human Performance in Space." Aerospace Medicine and Human Performance 91, no. 6 (2020): 532–34. http://dx.doi.org/10.3357/amhp.5568.2020.

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INTRODUCTION: In recent decades, there has been investigation into the effects of microgravity and microgravity-like environments on cognition and emotion separately. Here we highlight the need of focusing on emotion-cognition interactions as a framework for explaining cognitive performance in space. In particular, by referring to the affective cognition hypothesis, the significant interplay between emotional variables and cognitive processing in space is briefly analyzed. Altogether, this approach shows an interesting pattern of data pointing to a dynamic relation that may be sensitive to mic
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Lorenzen, Bonnie, and Laura L. Murray. "Benefits of Physical Fitness Training in Healthy Aging and Neurogenic Patient Populations." Perspectives on Neurophysiology and Neurogenic Speech and Language Disorders 18, no. 3 (2008): 99–106. http://dx.doi.org/10.1044/nnsld18.3.99.

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Abstract Purpose: In recent years, research has identified a positive connection between physical fitness and exercise, and cognitive performance in healthy aging (e.g., Colcombe & Kramer, 2003) as well as a number of patient populations (e.g., Mostert & Kesselring, 2002). To increase awareness of the benefits of exercise on cognitive and communicative health, this paper reviews the literature pertaining to the cognitive effects of exercise in healthy individuals, as well as preliminary findings regarding the role of exercise in disordered populations including those with stroke, demen
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Gyllenberg, David, Bianca Arrhenius, Auli Suominen, and Andre Sourander. "S133. DIAGNOSED SPEECH, SCHOLASTIC AND MOTOR DISORDERS AS PREDICTORS FOR NON-AFFECTIVE PSYCHOSES." Schizophrenia Bulletin 46, Supplement_1 (2020): S86. http://dx.doi.org/10.1093/schbul/sbaa031.199.

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Abstract Background Premorbid cognitive impairments are associated with schizophrenia, but little is known about the risk of developing psychoses among children with diagnosed speech, scholastic and motor disorders. Our aim was to study if children diagnosed with these are at increased risk of non-affective psychoses in adolescence and early adulthood. Methods We identified all children born 1996–2001 that were diagnosed with a speech disorder (ICD-10 code F80), scholastic disorder (F81), motor disorder (F82) or mixed developmental disorder (F83) before age 15 in outpatient and inpatient speci
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Tsai, Sheng-Tzung, Horng-Jyh Harn, Shinn-Zong Lin, Guo-Fang Tseng, and Shin-Yuan Chen. "Deep Brain Stimulation for Amelioration of Cognitive Impairment in Neurological Disorders: Neurogenesis and Circuit Reanimation." Cell Transplantation 28, no. 7 (2018): 813–18. http://dx.doi.org/10.1177/0963689718804144.

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Acute (e.g., traumatic brain injury or stroke) and chronic (e.g., dementia or Parkinson’s disease dementia) neurological disorders that involve cognitive impairment and dysfunctional neural circuits always lead to a dreadful and costly experience for patients and their families. The application of deep brain stimulation for the treatment of neuropsychiatric disorders has shown great potential to modulate pathological neural circuits and trigger endogenous neurogenesis. We summarize several important clinical and translational studies that utilize deep brain stimulation to improve cognition bas
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