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Dissertations / Theses on the topic 'Colon (Anatomy) Cancer Genetic aspects'

Author: Grafiati

Published: 4 June 2021

Last updated: 21 February 2023

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1

Butler, Lisa Maree. "Molecular analysis of the human Fas gene in colorectal cancer /." Title page, contents and summary only, 1998. http://web4.library.adelaide.edu.au/theses/09PH/09phb9858.pdf.

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2

陳安安 and On-on Annie Chan. "Methylation in colorectal cancer." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2002. http://hub.hku.hk/bib/B25256312.

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3

Warusavitarne, Janindra. "Analysis of the factors that influence the biological behaviour and response to chemotherapy in sporadic colorectal carcinomas with microsatellite instability." Thesis, The University of Sydney, 2005. https://hdl.handle.net/2123/27920.

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High frequency microsatellite instability (MSI—H) is an alternate pathway of colorectal carcinogenesis and is present in approximately 15% of all sporadic colorectal cancers. These tumours are predominantly located in the right colon, occur mainly in the elderly and have a better prognosis than microsatellite stable (MSS) CRCs. In addition they are usually of mucinous type histology, tend to be locally invasive, metastasise less and are of larger size than MSS CRCs. The mismatch repair (MMR) genes hMLHl is most commonly mutated or methylated in sporadic colorectal cancers with MSI-H.

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4

Li, Carmen, and 李嘉敏. "Characterisation of methylator phenotype of colorectal cancer in young patients." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hdl.handle.net/10722/207182.

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The majority of sporadic colorectal cancer (CRC) cases affects individuals over the age of 50, but about 10% of cases occur in young adults under 50 in Hong Kong. Apart from germline mutation of the DNA mismatch repair genes that predisposes to early-onset CRC with a high-level of microsatellite instability (MSI-H), it is unknown if the mechanisms that give rise to CRC in other young adults differ from those in older individuals. In an effort to understand the genetic and epigenetic basis of early and late-onset CRC outside the Lynch Syndrome setting, we performed a detailed characterization o

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5

Mui, Kin-cheong, and 梅堅祥. "Inactivation of DNA match repair proteins in premalignant lesions in Lynch syndrome." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B44659635.

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6

Tai, Bik-wah Diana, and 戴碧華. "Haplotype analysis of the family with Lynch syndrome." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B45153772.

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7

Wright, C. M. "The prognostic significance of microsatellite instability in sporadic stage C colorectal cancer." Thesis, The University of Sydney, 2008. https://hdl.handle.net/2123/28955.

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Identification and understanding of the molecular events involved in colorectal cancer (CRC) pathogenesis should lead to better comprehension of the disease process, hopefully leading to better prognostic stratification, and as a result more targeted treatment regimens and improved patient outcomes. A sub group of sporadic CRC exhibit microsatellite instability (MSI). MSI is seen when the fidelity of DNA replication is impaired. Cancers may be categorized as MSI-high (MSI-H), MSI-low (MSI-L), or microsatellite stable (MSS), according to the degree of MSI exhibited. This research proj

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8

Siu, Ho-yee Vivian. "The role of monitoring style in managing psychological distress associated with genetic colorectal cancer testing." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2004. http://hub.hku.hk/bib/B29760161.

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9

陳俊良 and Tsun-leung Chan. "Genomic instability and DNA mismatch repair gene mutations in colorectal cancer." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1999. http://hub.hku.hk/bib/B31238221.

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10

Chan, Ling-fung, and 陳凌鋒. "Expression of the DNA mismatch repair protein MLH1 in serrated polyps of the colon: an immunohistochemical study." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B45010584.

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11

Yuen, Siu-tsan Thomas, and 袁兆燦. "Molecular genetics of colorectal cancer and its relevance to epidemiology in Chinese population." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2003. http://hub.hku.hk/bib/B26627449.

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12

Wong, Chi-wai, and 黃志偉. "High resolution mapping of loss of heterozygosity and chromosomal aberrations using oligonucleotide single nucleotide polymorphismgenotyping arrays in colorectal adenoma to carcinoma progression." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2006. http://hub.hku.hk/bib/B3871923X.

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13

Li, Hung-sing, and 李鴻陞. "Identification of polycomb group protein CBX8 as a novel tumor suppressor in human colorectal cancer." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2014. http://hdl.handle.net/10722/197534.

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Polycomb group (PcG) proteins governs the regulation of diverse cellular functions, such as cell fate decision, cell cycle progression, maintenance of embryonic stem cell pluripotency, and DNA damage repair. Although aberrant expression of PcG proteins has been frequently reported in different cancer types, CBX8 is one of the least studied PcG family members in cancer. Recently, a study showed that forced expression of CBX8 in normal human and mouse fibroblasts demonstrated that cells could bypass senescence via INK4a-ARF repression; while another report demonstrated that CBX8 was involved in

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14

Guo, Dongli, and 郭冬麗. "Expression of Wnt signaling targets and their clinico-pathological significance in colorectal neoplasm: a tissuemicroarray study." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2006. http://hub.hku.hk/bib/B38610541.

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15

Choi, Siu-chung, and 蔡兆聰. "Genome-wide association study on colorectal cancer in the Hong Kong Chinese population." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hdl.handle.net/10722/193016.

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Colorectal cancer (CRC) is the second most common cancer in Hong Kong. While high-penetrance germline mutations account for up to 6% of cases, much of the variation in genetic risk may be attributable to multiple low-penetrance variants. Previous genome wide association studies (GWAS) have identified a number of CRC susceptibility alleles in Caucasian populations. Our GWAS investigated the association between genetic variants with CRC risk in the Han Chinese population in Hong Kong. In Stage I, genomic DNA samples from 455 female Chinese CRC subjects were genotyped using the Illumina 610 Quad

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16

張毅 and Ngai Cheung. "Expression of vascular endothelial growth factor and its receptors in tumours." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1998. http://hub.hku.hk/bib/B31220587.

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17

Li, Sze-wing Vivian, and 李思穎. "Profiling of gene expression changes in human colon crypt maturation and study of their dysregulation in tumourigenesis." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B40203384.

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18

He, Hua, and 何華. "Anti-tumor mechanisms of cyclooxygenase inhibitors and a c-Jun-N-terminal kinase inhibitor in gastrointestinal cancers." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2004. http://hub.hku.hk/bib/B30075245.

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19

Succi, Maysa [UNESP]. "Alteração da expressão da Anexina-A1 e Galectina-1 na progressão do câncer colorretal esporádico." Universidade Estadual Paulista (UNESP), 2013. http://hdl.handle.net/11449/92519.

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Made available in DSpace on 2014-06-11T19:26:05Z (GMT). No. of bitstreams: 0 Previous issue date: 2013-02-26Bitstream added on 2014-06-13T18:53:59Z : No. of bitstreams: 1 succi_m_me_sjrp_parcial.pdf: 268320 bytes, checksum: 47bca06cf986a823daf8462cd47bd92b (MD5) Bitstreams deleted on 2015-03-16T11:30:15Z: succi_m_me_sjrp_parcial.pdf,Bitstream added on 2015-03-16T11:31:00Z : No. of bitstreams: 1 000714071.pdf: 1014657 bytes, checksum: b46cd9f9aefc594fb472e9cf8b775085 (MD5)<br>Alterações nos níveis de expressão de moduladores da resposta inflamatória, como a Anexina-A1 (AnxA1/ANXA1) e a Galec

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20

Sibani, Sahar. "Genetic and nutritional folate deficiency : implications for homocystinuria and intestinal neoplasia." Thesis, McGill University, 2000. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=31539.

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Folate deficiency, a prevalent vitamin deficiency in America, can stem from environmental and/or genetic causes. The most common inborn error of folate metabolism is deficiency of methylenetetrahydrofolate reductase (MTHFR), which catalyzes the reduction of 5,10-methylenetetrahydrofolate to 5-methyltetrahydrofolate. Severe MTHFR deficiency results in hyperhomocysteinemia and homocystinuria; patients present with developmental delay, and various neurological and vascular disorders. This thesis describes three mutations identified in the MTHFR locus in patients with severe deficiency: 1025T&rarr

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21

Proença, Marcela Alcântara [UNESP]. "Associação de polimorfismos nos genes TLR2 e TLR4 com risco de câncer colorretal esporádico e influência na expressão gênica." Universidade Estadual Paulista (UNESP), 2013. http://hdl.handle.net/11449/92527.

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Made available in DSpace on 2014-06-11T19:26:05Z (GMT). No. of bitstreams: 0 Previous issue date: 2013-02-25Bitstream added on 2014-06-13T19:53:59Z : No. of bitstreams: 1 proenca_ma_me_sjrp.pdf: 521619 bytes, checksum: c975ed11c57b1194496e34885464bcca (MD5)<br>O câncer colorretal (CCR) é um dos modelos da associação inflamação-câncer. Assim, polimorfismos em genes que desempenham um papel importante na suscetibilidade a doenças inflamatórias, como os receptores Toll-like (TLR2 e TLR4), podem ser alvos interessantes para estudos de possíveis marcadores moleculares para CCR. Objetivos: Avaliar

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22

Park, Jin Young. "Diet, lifestyle factors and colorectal cancer risk : with focus on methodological issues." Thesis, University of Cambridge, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.609120.

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23

Khoo, Kong Kheong. "The influence of metabolic phenotypes upon the development of colorectal neoplasia /." Title page, table of contents and conclusions only, 1995. http://web4.library.adelaide.edu.au/theses/09MD/09mdk45.pdf.

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24

Ye, Yini. "Involvement of 5-lipoxygenase in the promotion of colonic tumorigenesis by cigarette smoke." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2004. http://hub.hku.hk/bib/B29939008.

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25

Bulcao, Candice. "Polyunsaturated fatty acid metabolism and effects on colon cancer cell biology in vitro." Thesis, Rhodes University, 2013. http://hdl.handle.net/10962/d1016128.

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Colon cancer is a leading cause of cancer related deaths worldwide. Lifestyle factors such as diet and exercise have been implicated as important agents in colon cancer development and progression. Epidemiological, in vivo and in vitro studies have found that n-3 polyunsaturated fatty acids (PUFAs) reduce colon carcinoma. The role of n-6 PUFAs remains a controversial topic, with studies indicating both promoting and preventing capabilities published. In order to better understand the effects of PUFAs on colon carcinoma, it is important to have an understanding of how they will be broken down i

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26

Liu, Shuk Ming. "Single nucleotide polymorphism in human microsomal glutathione s-transferase gene and colorectal cancer /." View Abstract or Full-Text, 2003. http://library.ust.hk/cgi/db/thesis.pl?BIOL%202003%20LIU.

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Thesis (M. Phil.)--Hong Kong University of Science and Technology, 2003.<br>Includes bibliographical references (leaves 95-105). Also available in electronic version. Access restricted to campus users.

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27

Mizaku, Alda. "Biomolecular feature selection of colorectal cancer microarray data using GA-SVM hybrid and noise perturbation to address overfitting." Diss., Online access via UMI:, 2009.

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Thesis (M.S.)--State University of New York at Binghamton, Thomas J. Watson School of Engineering and Applied Science, Department of Bioengineering, Biomedical Engineering, 2009.<br>Includes bibliographical references.

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28

Tabatabaei, Seyed Mehdi. "The relationship between dietary factors, meat consumption, heterocyclic amines, Benzo[a]pyrene, meat-derived mutagenic activity and colorectal cancer in Western Australia." University of Western Australia. School of Population Health, 2009. http://theses.library.uwa.edu.au/adt-WU2010.0059.

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The role of meat consumption in the development of cancer, including colorectal cancer (CRC), has been subject of much investigation in recent years. The observation of geographical variation in CRC incidence and increased CRC risks in populations consuming high levels of meat prompted researchers to hypothesise a link between meat and CRC. An area of particular interest in CRC pathogenesis is the meat-derived compounds such are heterocyclic amines (HCAs), polycyclic aromatic hydrocarbons (PAHs), and meatderived mutagenic activity. Australia is among the countries with high incidence of CRC an

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29

"Molecular mechanisms of autophagy mediated by silencing of EEF2K in colon cancer cells." 2014. http://repository.lib.cuhk.edu.hk/en/item/cuhk-1290641.

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Eukaryotic translation elongation factor-2 (EEF2) is regulated through phosphorylation by a specific kinase known as eukaryotic elongation factor-2 kinase (EEF2K), leading to translational down regulation. Currently, it has been reported that EEF2K could promote the autophagic survival in breast and glioblastoma cell lines. However, the precise function of EEF2K in cancer as well as the related mechanism is still poorly understood. Colorectal cancer is the third common malignant disease worldwide and more than half of the patients with colorectal cancer require chemotherapy after surgery. Howe

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30

"miR-133a inhibits colorectal cancer cell growth by direct targeting of ring finger and FYVE-like domain containing E3 ubiquitin protein ligase." 2012. http://library.cuhk.edu.hk/record=b5549551.

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運用miRNA微陣列手段，我們篩選到一批在結直腸腫瘤內高表達和低表達的miRNA。其中miR-133a是在腫瘤中最顯著降低的miRNA之一，但其在腫瘤的發生發展過程中的作用尚未可知，因此我們選擇miR-133a最為研究目標，論證其的生物學功能，分子機理，及其在結直腸癌中的靶分子。<br>我們首先在較大規模樣本中驗證miR-133a的低表達。定量PCR結果顯示，對比正常的癌旁組織，miR-133a在94例結直腸癌組織中顯著低表達（p < 0.001）。我們進一步分析miR-133a在9種常用的腫瘤細胞系內的表達情況。對比正常的直腸組織，miR-133a在9種常用的結直腸細胞系內的表達均明顯下降。在腫瘤發生發展過程中，癌細胞趨向於降低具有抑癌功能的基因的表達。因此我們推測miR-133a是一個潛在的腫瘤抑制miRNA。<br>為了論證我們的假設，我們首先選取miR-133a低表達的結直腸癌細胞系HCT116和LoVo最為研究模型，將miR-133a在這兩種細胞系內過表達。升高的miR-133a可以抑制腫瘤細胞生長（p < 0.01和p < 0.05），抑制腫瘤克隆集落形成（p < 0.01）。我們進一步發現過表達miR-133a可以抑制裸鼠體內腫瘤的生長（p < 0.05）。細胞週期分析顯示miR-133a抑制細胞生長的能力表現為誘導腫瘤細胞週期阻滯於G0/G1期。細胞週期特異性CD

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31

"Novel recurrent point mutation and gene fusion identified by new generation sequencing in colorectal cancer." 2013. http://library.cuhk.edu.hk/record=b5884462.

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He, Jun.<br>Thesis (Ph.D.)--Chinese University of Hong Kong, 2013.<br>Includes bibliographical references (leaves 136-156).<br>Electronic reproduction. Hong Kong : Chinese University of Hong Kong, [2012] System requirements: Adobe Acrobat Reader. Available via World Wide Web.<br>Abstract also in Chinese.

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32

"A novel amplification gene SLC12A5 promotes cell proliferation and tumor metastasis in colorectal cancer." 2014. http://repository.lib.cuhk.edu.hk/en/item/cuhk-1291470.

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Background & Aims: By whole genome sequencing, we identified for the first time that solute carrier family 12 member 5 (SLC12A5) gene located on chromosome 20q13.12 was amplified in colorectal cancer (CRC). We aimed to determine the amplification status of SLC12A5 and its clinical implication in CRC, and characterize the functional mechanisms of SLC12A5 in colorectal carcinogenesis.<br>Materials and Methods: Protein expression level of SLC12A5 was evaluated by immunohistochemistry. SLC12A5 amplification was verified by fluorescence in situ hybridization (FISH). The correlations between SLC12A5

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33

Otway, Robyn Tracy. "Germline mutations causing hereditary nonpolyposis colorectal cancer." Phd thesis, 2001. http://hdl.handle.net/1885/146220.

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34

"Characterization of long non-coding RNA H19 in epithelial to mesenchymal transition: 長非編碼RNA H19在上皮間充質轉化中的功能探究". 2014. http://repository.lib.cuhk.edu.hk/en/item/cuhk-1290666.

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Colorectal cancer (CRC), with an estimated 1.2 million new cases annually, is the third leading cause of cancer incidence and death worldwide. Generally, the majority of CRC patients are diagnosed at the advanced stages with poor prognosis and unfavorable response to multiple therapeutic drugs. In spite of increasing knowledge of the molecular mechanism for the tumorigenesis in CRC patients, the translation from basic science into clinical therapy has been limited for quite a long time. In order to develop novel treatment strategies against CRC, intensive and extensive attempts have been made

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35

"Promoter hypermethylation of tumor related genes in the progression of colorectal neoplasia." 2005. http://library.cuhk.edu.hk/record=b5892373.

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Bai Hsing Chen.<br>Thesis (M.Phil.)--Chinese University of Hong Kong, 2005.<br>Includes bibliographical references (leaves 89-94).<br>Abstracts in English and Chinese.<br>Acknowledgments --- p.ii<br>Publication --- p.iii<br>List of Abbreviations --- p.iv<br>List of Tables --- p.v<br>List of Figures --- p.vi<br>Abstract --- p.vii<br>摘要 --- p.x<br>Table of Contents --- p.xii<br>Chapter Chapter 1 --- INTRODUCTION<br>Chapter 1.1 --- Molecular Biology in Cancer Development --- p.2<br>Chapter 1.1.1 --- Cell Cycle and Cancer --- p.2<br>Chapter 1.1.2 --- Oncogenes and Tumor Suppressor Genes

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36

Smith-Roe, Stephanie L. "DNA mismatch repair-dependent responses to the food-borne carcinogen 2-Amino-1-methyl-6-phenylimidazo[4,5-b]pyridine (PhIP) in the mouse." Thesis, 2006. http://hdl.handle.net/1957/29017.

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37

Mundade, Rasika S. "Validation-based insertional mutagenesis (VBIM) technology identifies adenomatous polypossis coli (APC) like protein (ALP) as a novel negative regulator of NF-κB". Thesis, 2015. http://hdl.handle.net/1805/6591.

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Colorectal cancer (CRC) is the third leading cause of cancer related deaths in the United States. The nuclear factor κB (NF-κB) is an important family of transcription factors whose aberrant activation has been found in many types of cancer, including CRC. Therefore, understanding the regulation of NF-κB is of ultimate importance for cancer therapy. Using a novel validation-based insertional mutagenesis (VBIM) strategy, our lab has identified the novel adenomatous polyposis coli (APC) like protein (ALP) gene as a negative regulator of NF-κB. Preliminary studies from our lab demonstrated

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38

Berney, Christophe R. "Studies on genetic markers and in particular nm23 in sporadic colorectal cancer : predictors of liver metastasis /." 1999. http://www.library.unsw.edu.au/~thesis/adt-NUN/public/adt-NUN20010508.162326/index.html.

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39

Khoo, Kong Kheong. "The influence of metabolic phenotypes upon the development of colorectal neoplasia / by Kong Kheong Khoo." Thesis, 1995. http://hdl.handle.net/2440/38340.

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Bibliography: leaves 150-166.<br>ix, 166, xxii leaves<br>To assess the role of acetylator status and glutathione S-transferase[mu] (GST[mu]) null phenotype on the risk for development of colorectal neoplasms in humans and to determine whether this was influenced by the dietary intake of meat.<br>Thesis (M.D.) -- University of Adelaide, Dept. of Medicine, 1995?

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Park, Eunyoung 1976. "Retinol inhibits the growth and invasion of all-trans-retinoic acid resistant colon cancer in vitro and in vivo." Thesis, 2007. http://hdl.handle.net/2152/3793.

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Colorectal cancer is the third most common cancer and cause of death due to cancer in the United States. Death due to colorectal cancer is generally caused by hepatic metastasis rather than the primary tumor itself. The five-year survival rate is only 10% for patients whose colorectal cancer metastasized, which indicates the need for more effective therapies to treat colon cancer. The diet contains (1) preformed vitamin A as retinyl esters in animal-derived food sources and (2) provitamin A carotenoids in plant-derived food sources. Once absorbed, retinol is re-esterified and transported t

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Sinicrope, Pamela S. Vernon Sally W. "Development and preliminary validation of the cancer family impact scale for colorectal cancer /." 2007. http://proquest.umi.com.www5.sph.uth.tmc.edu:2048/pqdweb?did=1445048521&sid=2&Fmt=2&clientId=92&RQT=309&VName=PQD.

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Gathirua-Mwangi, Wambui Grace. "Obesity and obesity-related markers associated with breast and colorectal cancer occurence and mortality." Diss., 2016. http://hdl.handle.net/1805/11004.

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Indiana University-Purdue University Indianapolis (IUPUI)<br>Purpose: Obesity is a growing public health problem and the second most preventable cause of death in the US. Obesity has been linked as a risk factor for several cancers. However, there are limited studies that have examined the roles of metabolic syndrome (MetS) and C-reactive protein (CRP), as well as change in body composition from early adulthood to late adulthood on the risk of cancer. The overall objective of this dissertation was to determine the association of obesity and obesity-related markers with breast and c

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Winger, Joseph G. "Diet and exercise intervention adherence and health-related outcomes among older long-term breast, prostate, and colorectal cancer survivors." Thesis, 2013. http://hdl.handle.net/1805/5068.

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Indiana University-Purdue University Indianapolis (IUPUI)<br>Given the numerous benefits of a healthy diet and exercise for cancer survivors, there has been an increase in the number of lifestyle intervention trials for this population in recent years. However, the extent to which adherence to a diet and exercise intervention predicts health-related outcomes among cancer survivors is currently unknown. To address this question, data from the Reach out to ENhancE Wellness in Older Cancer Survivors (RENEW) diet and exercise intervention trial were analyzed. RENEW was a yearlong telephone and mai

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