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Journal articles on the topic 'Colorectal neoplasia'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Taylor, Irving. "Colorectal neoplasia." Current Opinion in Gastroenterology 7, no. 1 (1991): 25–29. http://dx.doi.org/10.1097/00001574-199102000-00006.

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2

Grega, Tomáš, Gabriela Vojtěchová, Michal Voška, et al. "Predictors of advanced colorectal neoplasia in colorectal cancer screening – interim results of multicentric prospective study." Gastroenterologie a hepatologie 74, no. 5 (2020): 386–92. http://dx.doi.org/10.14735/amgh2020386.

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ntroduction: The incidence of advanced colorectal neoplasia in the screening population shows great diversity with a prevalence of 3–12 %. Due to the uneven distribution in the population, potential risk factors that would allow the stratification of individuals according to the degree of risk of colorectal neoplasia are searched. Aim: To determine the risk factors associated with the occurrence of advanced colorectal neoplasia in the screening population. Methods: Asymptomatic individuals aged 45–75 years who underwent preventive colonoscopy in 2012–2016 in a multicenter prospective study mon

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3

CAMPOS, Fábio Guilherme, Magaly Gemio TEIXEIRA, Arceu SCANAVINI, Maristela Gomes de ALMEIDA, Sergio Carlos NAHAS, and Ivan CECCONELLO. "INTESTINAL AND EXTRAINTESTINAL NEOPLASIA IN PATIENTS WITH INFLAMMATORY BOWEL DISEASE IN A TERTIARY CARE HOSPITAL." Arquivos de Gastroenterologia 50, no. 2 (2013): 123–29. http://dx.doi.org/10.1590/s0004-28032013000200021.

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Context The development of neoplasia is an important concern associated with inflammatory bowel disease (IBD), especially colorectal cancer (CRC). Objectives Our aim was to determine the incidence of intestinal and extraintestinal neoplasias among patients with inflammatory bowel disease. Methods There were retrieved information from 1607 patients regarding demographics, disease duration and extent, temporal relationship between IBD diagnosis and neoplasia, clinical outcomes and risk factors for neoplasia. Results Crohn's disease (CD) was more frequent among women (P = 0.0018). The incidence o

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4

Völkerer, Andreas, Sarah Wernly, Georg Semmler, et al. "Association between Diverticulosis and Colorectal Neoplasia: Analysis from a Large Austrian Database." Journal of Clinical Medicine 13, no. 20 (2024): 6078. http://dx.doi.org/10.3390/jcm13206078.

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Background: Colorectal neoplasia and diverticulosis are common findings on colonoscopies. While adenomas are precursors to colorectal cancer, diverticulosis is usually asymptomatic but can lead to diverticulitis. Despite their prevalence and coexistence, the relationship between these conditions remains unclear. This study investigates whether diverticulosis is associated with adenomas, considering shared risk factors and potential inflammation-driven mechanisms. Methods: We examined 6154 asymptomatic individuals undergoing colorectal cancer screening in Austria. Diverticulosis and colorectal

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5

Korotkevich, A. G., and N. M. Zhilina. "Gender features of localization of epithelial neoplasms of the colon according to the results of retroanalysis of colonoscopies of Novokuznetsk residents." Experimental and Clinical Gastroenterology, no. 5 (May 18, 2024): 26–31. http://dx.doi.org/10.31146/1682-8658-ecg-225-5-26-31.

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Purpose of the study. The article It is devoted to the analysis of the influence of the patient’s sex on the frequency and localization of epithelial neoplasms of the colon. Materials and methods. In a continuous cross-sectional retrospective study we studied the results of 3086 colonoscopies for 2019-2020. Results. A cohort of. 980 patients with neoplasia. Analysis of localization and number of detected neoplasms depending on age and gender revealed a significant increase in the number of tumors after 40 years of life. The work confirmed the connection male sex with the frequency of colorecta

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6

Van Lierop, L., M. te Groen, L. Derikx, et al. "P199 Clonal patterns between pouch neoplasia and prior colorectal neoplasia in Inflammatory Bowel Disease patients: an exploratory cohort study." Journal of Crohn's and Colitis 17, Supplement_1 (2023): i351—i352. http://dx.doi.org/10.1093/ecco-jcc/jjac190.0329.

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Abstract Background Inflammatory Bowel Disease (IBD) patients with an ileo-anal pouch anastomosis (IPAA) bear an increased risk of pouch neoplasia, with prior colorectal neoplasia as the strongest predictor. It is unknown if pouch neoplasia develops independently or is derived from prior colorectal neoplasia. Therefore, we aimed to elucidate the pathogenesis of pouch neoplasia by assessment of clonality between prior colorectal neoplasia and pouch neoplasia in IPAA patients with IBD. Methods In this explorative study we used the Dutch Nationwide Pathology Databank to identify IBD patients with

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7

Cohen, A. M. "Colorectal neoplasia: surgery." Current Opinion in Gastroenterology 6, no. 1 (1990): 38–40. http://dx.doi.org/10.1097/00001574-199002000-00008.

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8

Ho Kim, W., J. Hoon Suh, T. Il Kim, et al. "Colorectal flat neoplasia." Digestive and Liver Disease 35, no. 3 (2003): 165–71. http://dx.doi.org/10.1016/s1590-8658(03)00024-0.

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9

Snover, Dale C., and Kenneth P. Batts. "Serrated Colorectal Neoplasia." Surgical Pathology Clinics 3, no. 2 (2010): 207–40. http://dx.doi.org/10.1016/j.path.2010.05.001.

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10

IJspeert, Joep Evert Godfried, Jan Paul Medema, and Evelien Dekker. "Colorectal Neoplasia Pathways." Gastrointestinal Endoscopy Clinics of North America 25, no. 2 (2015): 169–82. http://dx.doi.org/10.1016/j.giec.2014.11.004.

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11

Kim, Min Cheol, Kyeong Ok Kim, and Min Kyu Kang. "Prevalence and associated risk of advanced colorectal neoplasia in adults with sarcopenia." Korean Journal of Internal Medicine 37, no. 2 (2022): 294–303. http://dx.doi.org/10.3904/kjim.2020.569.

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Background/Aims: Although several studies have shown that sarcopenia is associated with poor outcomes in colorectal cancer patients, the impact of sarcopenia on the development of colorectal neoplasia remains unclear. We aimed to evaluate the prevalence and association of colorectal neoplasia, especially advanced colorectal neoplasia, in adults with sarcopenia.Methods: We retrospectively analyzed the data for 10,676 adults who underwent firsttime colonoscopy and bioelectrical impedance analysis (BIA) on the same day in a health screening program at a single center. Sarcopenia was diagnosed usi

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12

Van Lierop, L., M. te Groen, L. Derikx, et al. "A207 CLONAL PATTERNS BETWEEN POUCH NEOPLASIA AND PRIOR COLORECTAL NEOPLASIA IN INFLAMMATORY BOWEL DISEASE PATIENTS: AN EXPLORATORY COHORT STUDY." Journal of the Canadian Association of Gastroenterology 6, Supplement_1 (2023): 46. http://dx.doi.org/10.1093/jcag/gwac036.207.

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Abstract Background Inflammatory bowel disease (IBD) patients with an ileo-anal pouch anastomosis (IPAA) bear an increased risk of pouch neoplasia, with prior colorectal neoplasia as the strongest predictor. It is unknown if pouch neoplasia develops independently or is derived from prior colorectal neoplasia. Purpose We aimed to assess potential clonality between prior colorectal neoplasia and pouch neoplasia in IPAA patients with IBD. Method In this explorative study we used the Dutch Nationwide Pathology Databank to identify IBD patients with both pouch neoplasia and colorectal neoplasia pri

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13

Brenner, D. E., Y. Su, D. P. Normolle, et al. "Detection of colorectal neoplasia associated K-Ras mutations in human urine." Journal of Clinical Oncology 24, no. 18_suppl (2006): 1005. http://dx.doi.org/10.1200/jco.2006.24.18_suppl.1005.

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1005 Background: Since colorectal neoplasia-associated genes have been detected in human blood, we hypothesized that small DNA fragments containing genetic mutations associated with colorectal neoplasias are filtered and excreted in the urine. If so, genes associated with colorectal cancer will be detected in the urine. K-ras mutations are commonly associated with colorectal neoplasia and do not occur in the urinary tract. Methods: K-ras mutation detection: 200 microl of urine was extracted with guanidine thiocyanate and purifed using a Wizard DNA isolation kit. Codon 12 K-ras mutation detecti

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14

Inadomi, John M. "Screening for Colorectal Neoplasia." New England Journal of Medicine 376, no. 2 (2017): 149–56. http://dx.doi.org/10.1056/nejmcp1512286.

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15

Wakeman, Chris, Jacqueline Keenan, Jimmy Eteuati, Paul Hollington, Tim Eglinton, and Frank Frizelle. "Chemoprevention of colorectal neoplasia." ANZ Journal of Surgery 87, no. 12 (2015): E228—E232. http://dx.doi.org/10.1111/ans.13392.

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16

Fajardo, Alyssa, and Bruce Robb. "Chemoprevention for Colorectal Neoplasia." Clinics in Colon and Rectal Surgery 21, no. 04 (2008): 304–12. http://dx.doi.org/10.1055/s-0028-1089947.

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17

Holick, Michael F., and Peter R. Holt. "Chemoprevention of Colorectal Neoplasia." Gastroenterology 135, no. 4 (2008): 1427–28. http://dx.doi.org/10.1053/j.gastro.2008.06.092.

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18

JENKINS, P. J., G. M. BESSER, and P. D. FAIRCLOUGH. "Colorectal neoplasia in acromegaly." Gut 44, no. 5 (1999): 585–87. http://dx.doi.org/10.1136/gut.44.5.585.

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19

Jenkins, Paul J., and Peter D. Fairclough. "Colorectal neoplasia in acromegaly*." Clinical Endocrinology 55, no. 6 (2001): 727–29. http://dx.doi.org/10.1046/j.1365-2265.2001.01418.x.

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20

Carey, Frank. "Pathology of colorectal neoplasia." Surgery (Oxford) 35, no. 3 (2017): 126–31. http://dx.doi.org/10.1016/j.mpsur.2016.12.006.

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21

Langenfeld, Sean J. "Advances in Colorectal Neoplasia." Surgical Clinics of North America 97, no. 3 (2017): i. http://dx.doi.org/10.1016/s0039-6109(17)30054-3.

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22

Anderson, Joseph, and Zvi Alpern. "Smoking and Colorectal Neoplasia." American Journal of Gastroenterology 103 (September 2008): S200—S201. http://dx.doi.org/10.14309/00000434-200809001-00519.

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23

Dolejs, Scott, Benjamin Gayed, and Alyssa Fajardo. "Prevention of Colorectal Neoplasia." Clinics in Colon and Rectal Surgery 29, no. 04 (2016): 353–62. http://dx.doi.org/10.1055/s-0036-1584086.

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AbstractColorectal cancer (CRC) is one of the leading causes of cancer-related morbidity and mortality worldwide. There are well-established screening protocols involving fecal testing, radiographic, and endoscopic evaluations that have led to decreased incidence and mortality of CRC in the United States. In addition to screening for CRC, there is interest in preventing colorectal neoplasia by targeting the signaling pathways that have been identified in the pathway of dysplasia progressing to carcinoma. This review will detail the efficacy of multiple potential preventative strategies includi

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24

Anderson, Joseph C., Zvi A. Alpern, Brendan J. Wiggins, Patricia M. Hubbard-Ells, and Carol M. Martin. "Smoking and Colorectal Neoplasia." American Journal of Gastroenterology 101 (September 2006): S211—S212. http://dx.doi.org/10.14309/00000434-200609001-00498.

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25

Kryachko, Andrei A., Vladimir M. Durleshter, Konstantin D. Chuguzov, and Anastasiya A. Kryachko. "Benign neoplasms of the colon: problems and solutions." Experimental and Clinical Gastroenterology, no. 11 (January 23, 2023): 227–33. http://dx.doi.org/10.31146/1682-8658-ecg-207-11-227-233.

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Benign neoplasms of the colon, rectum, anus and anal canal are true neoplasia of the colon mucosa and are associated with a high risk of colorectal cancer (CC). The literature review analyzes epidemiology, risk factors, and modern methods of diagnosis and treatment, and describes priority minimally invasive interventions for benign colorectal tumors.

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26

Dang, Luan Minh, Nhan Quang Le, Huy Minh Le, et al. "Colorectal neoplasia is not uncommon in siblings aged under 50 years of patients with early-onset colorectal advanced adenomas: A cross-sectional study." Medicine 104, no. 27 (2025): e43222. https://doi.org/10.1097/md.0000000000043222.

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Research on the prevalence of adenomas and colorectal cancer (CRC) in individuals aged 50 years or younger with first-degree relatives diagnosed with early-onset colorectal advanced adenoma (E-CAA), defined as the onset of colorectal advanced adenoma (CAA) at or before 50 years of age, is scarce. We examined the prevalence of colorectal neoplasia among siblings aged 50 years or younger of E-CAA patients. A cross-sectional study was conducted at the University Medical Center in Ho Chi Minh City, Vietnam. Study participants included 96 siblings aged 50 years or younger from 58 E-CAA patients, wh

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27

Kim, Kwang Woo, Hyoun Woo Kang, Hosun Yoo, et al. "Association between severe hepatic steatosis examined by Fibroscan and the risk of high-risk colorectal neoplasia." PLOS ONE 17, no. 12 (2022): e0279242. http://dx.doi.org/10.1371/journal.pone.0279242.

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The prevalence of colorectal neoplasm in patients with non-alcoholic fatty liver disease has increased twice as high as that in the general population. FibroScan is a new modality for evaluating hepatic steatosis. This study aimed to investigate the relationship between the risk of high-risk colorectal neoplasia and hepatic steatosis examined using FibroScan. This was a cross sectional study of prospectively enrolled subjects who were scheduled to undergo index colonoscopy as a health screening between March 2018 and February 2019. The severity of steatosis was graded as normal, mild, moderate

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28

Saade, Rayan, Zhiyan Fu, and Hwajeong Lee. "Endoscopically Unresectable Paneth Cell (PC)–Containing Adenomas and the Risk of Developing Colorectal Neoplasia." American Journal of Clinical Pathology 152, Supplement_1 (2019): S73. http://dx.doi.org/10.1093/ajcp/aqz113.089.

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Abstract Objectives Whether the presence of Paneth cells (PCs) in colorectal adenomas indicates an increased risk of colorectal neoplasia or not is controversial. We examined the clinicopathologic features of PC-containing adenomas (PCAs) that were surgically removed, focusing on the risk of developing subsequent colorectal neoplasia on follow-up. Methods A retrospective cohort of 154 patients with endoscopically unresectable colorectal adenomas who underwent surgical removal was retrieved. Archived pathology slides were evaluated for the presence of PC, villous features, and high-grade dyspla

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29

Acevedo, Alejandro, Yaritza Diaz, Cynthia M. Perez, Maria Garau, John Baron, and Marcia Cruz-Correa. "Diabetes Mellitus and Colorectal Neoplasia." Journal of Cancer Therapy 03, no. 06 (2012): 859–65. http://dx.doi.org/10.4236/jct.2012.326110.

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30

Morris, D. L. "Colorectal neoplasia: epidemiology and aetiology." Current Opinion in Gastroenterology 6, no. 1 (1990): 24–32. http://dx.doi.org/10.1097/00001574-199002000-00006.

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31

Ahlquist, D. A. "Colorectal neoplasia: diagnosis and screening." Current Opinion in Gastroenterology 6, no. 1 (1990): 33–37. http://dx.doi.org/10.1097/00001574-199002000-00007.

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32

Gibson, Joanna A., and Robert D. Odze. "Pathology of premalignant colorectal neoplasia." Digestive Endoscopy 28, no. 3 (2016): 312–23. http://dx.doi.org/10.1111/den.12633.

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33

Jass, Jeremy R., Vicki L. J. Whitehall, Joanne Young, and Barbara A. Leggett. "Emerging concepts in colorectal neoplasia." Gastroenterology 123, no. 3 (2002): 862–76. http://dx.doi.org/10.1053/gast.2002.35392.

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34

Ahlquist, David A. "Molecular Detection of Colorectal Neoplasia." Gastroenterology 138, no. 6 (2010): 2127–39. http://dx.doi.org/10.1053/j.gastro.2010.01.055.

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35

Young, Graeme P., and Richard K. Le Leu. "Resistant Starch and Colorectal Neoplasia." Journal of AOAC INTERNATIONAL 87, no. 3 (2004): 775–86. http://dx.doi.org/10.1093/jaoac/87.3.775.

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Abstract There are several approaches to examining the relationship between resistant starch (RS) and development of colorectal cancer (CRC). These include examination of epidemiological relationships, objective testing of effects of RS given to humans on biological events of relevance to CRC, and studies in animal models where protection and mechanisms of protection can be directly tested. Nine epidemiological studies have examined the relationship between starch and CRC and/or adenomas. Most show a significant protective effect. However, epidemiological tools for measuring consumption of RS

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36

HARDCASTLE, J. D. "Screening for colorectal neoplasia: Introduction." Journal of Gastroenterology and Hepatology 6, no. 6 (1991): 537. http://dx.doi.org/10.1111/j.1440-1746.1991.tb00903.x.

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37

JOHN, D. J. B. ST. "Screening tests for colorectal neoplasia." Journal of Gastroenterology and Hepatology 6, no. 6 (1991): 538–44. http://dx.doi.org/10.1111/j.1440-1746.1991.tb00904.x.

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38

FETTMAN, MARTIN J., ROSS N. BUTLER, ANTHONY J. McMICHAEL, and IAN C. ROBERTS-THOMSON. "Metabolic phenotypes and colorectal neoplasia." Journal of Gastroenterology and Hepatology 6, no. 1 (1991): 81–89. http://dx.doi.org/10.1111/j.1440-1746.1991.tb01151.x.

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39

Jacobs, Elizabeth T., Patricia A. Thompson, and Mar??a Elena Mart??nez. "Diet, Gender, and Colorectal Neoplasia." Journal of Clinical Gastroenterology 41, no. 8 (2007): 731–46. http://dx.doi.org/10.1097/mcg.0b013e3180338e56.

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40

Pai, Ajit, Slawomir Marecik, John Park, and Leela Prasad. "Robotic Colorectal Surgery for Neoplasia." Surgical Clinics of North America 97, no. 3 (2017): 561–72. http://dx.doi.org/10.1016/j.suc.2017.01.006.

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41

Sengupta, Shomik, Joe J. Tjandra, and Peter R. Gibson. "Dietary fiber and colorectal neoplasia." Diseases of the Colon & Rectum 44, no. 7 (2001): 1016–33. http://dx.doi.org/10.1007/bf02235491.

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42

Mueller, Rudolph, and Andrew Davis. "Flexible sigmoidoscopy for colorectal neoplasia." American Journal of Medicine 90, no. 4 (1991): 536–37. http://dx.doi.org/10.1016/0002-9343(91)80104-t.

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43

Mueller, Rudolph, and Andrew Davis. "Flexible sigmoidoscopy for colorectal neoplasia." American Journal of Medicine 90, no. 1 (1991): 536–37. http://dx.doi.org/10.1016/0002-9343(91)90624-7.

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44

Lang, Leslie H. "“Virtual colonoscopy” for colorectal neoplasia." Gastroenterology 126, no. 2 (2004): 390. http://dx.doi.org/10.1053/j.gastro.2003.12.043.

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45

Scott, N., and P. Quirke. "Molecular biology of colorectal neoplasia." Gut 34, no. 3 (1993): 289–92. http://dx.doi.org/10.1136/gut.34.3.289.

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46

Spigelman, A. D. "Endoscopic surveillance for colorectal neoplasia." British Journal of Surgery 81, no. 11 (1994): 1664–65. http://dx.doi.org/10.1002/bjs.1800811135.

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47

Schraibman, I. G. "Endoscopic surveillance for colorectal neoplasia." British Journal of Surgery 82, no. 4 (1995): 568. http://dx.doi.org/10.1002/bjs.1800820447.

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48

Chang, George J. "Laparoscopic treatment of colorectal neoplasia." Current Treatment Options in Gastroenterology 9, no. 3 (2006): 256–64. http://dx.doi.org/10.1007/s11938-006-0044-1.

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49

SARAGIOTTO, Laiz, Vânia Aparecida LEANDRO-MERHI, José Luis Braga de AQUINO, and José Alexandre MENDONÇA. "GASTROINTESTINAL CHANGES DURING NUTRITIONAL FOLLOW-UP OF CANCER PATIENTS UNDERGOING OUTPATIENT CHEMOTHERAPY." Arquivos de Gastroenterologia 57, no. 4 (2020): 354–60. http://dx.doi.org/10.1590/s0004-2803.202000000-68.

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ABSTRACT BACKGROUND: Cancer patients may have gastrointestinal changes that influence nutritional status. OBJECTIVE: To investigate the occurrence of gastrointestinal changes resulting from outpatient chemotherapy treatment in cancer patients. METHODS: In a retrospective longitudinal study, the nutritional status and chemotherapy gastrointestinal changes (nausea, vomit, diarrhea, constipation, mucositis, dysphagia, xerostomia, inappetence, dysgeusia and heartburn) in cancer patients (n=187) were investigated in an outpatient follow-up. For the study of the parameters over time, the generalized

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50

Thomas, Melissa L., Susanne K. Pedersen, Aidan McEvoy, et al. "Plasma Testing of Gene Expression Biomarkers for Colorectal Neoplasia Discovered in Neoplastic Colorectal Tissue." Gastroenterology 140, no. 5 (2011): S—416. http://dx.doi.org/10.1016/s0016-5085(11)61709-5.

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