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Books on the topic 'Coma/therapy'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

Elizabeth, Sandel M., and Ellis, David W., Ph. D., eds. The Coma-emerging patient. Hanley & Belfus, Inc., 1990.

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2

Silva, Eduardo Missel, Eduardo Silva, and Gabriel Cruz. Comas and syncope: Causes, prevention, and treatment. Nova Science Publishers, 2011.

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3

Society for Treatment of Coma. (6 1997 Tokyo). The society for treatment of coma: Proceedings of the 6th Annual Meeting of the Society for Treatment of Coma, Tokyo 1997. Neuron Publishing, 1998.

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4

D, Schiff Nicholas, Laureys Steven, and Association for Research in Nervous and Mental Disease. Meeting, eds. Disorders of consciousness. Wiley-Blackwell, 2009.

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5

National Institute of Diabetes and Digestive and Kidney Diseases (U.S.), ed. Insuficiencia renal: Coma bien para sentirse bien durante su tratamiento de hemodiaĺisis. U.S. Dept. of Health and Human Services, National Institutes of Health, National Institute of Diabetes and Digestive and Kidney Diseases, 2002.

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6

Association, Reader's Digest, ed. Coma bien, siéntase bien: 500 deliciosas recetas elaboradas con alimentos saludables. Reader's Digest, 2002.

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7

Webb, Robyn. Coma y combata la hipertensión: Recetas sencillas y deliciosas para una vida larga y saludable : Incluye el plan DASH-Plus para que ¡reduzca 10% más su presión arterial sin medicamentos! Reader's Digest, 2006.

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8

Andrews, V. C. Mi dulce Audrina. 3rd ed. Plaza & Janés, 1993.

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9

Andrews, V. C. Öç yuvası. 2nd ed. Altın Kitaplar, 1996.

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10

Andrews, V. C. My sweet Audrina. Harper Collins, 1991.

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11

Andrews, V. C. Das Netz im Dunkel. Goldmann, 1986.

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12

Andrews, V. C. My Sweet Audrina. Pocket Books, 1989.

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13

Andrews, V. C. My Sweet Audrina. Pocket Books, 1999.

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14

Andrews, V. C. My sweet Audrina. Center Point Pub., 2001.

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15

Andrews, V. C. My Sweet Audrina. Fontana, 1990.

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16

Andrews, V. C. My Sweet Audrina. France Loisirs, 1988.

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17

Sacks, Oliver W. Przebudzenia. Wydawnictwo Zysk i S-ka, 2011.

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18

Sacks, Oliver W. Awakenings: Das Buch zum Film = Zeit des Erwachens. Rowohlt, 1995.

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19

Sacks, Oliver W. Awakenings. Vintage Books, 2002.

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20

Sacks, Oliver W. Awakenings. HarperPerennial, 1990.

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21

Sacks, Oliver W. Awakenings. Dutton, 1985.

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22

Sacks, Oliver W. Awakenings. Summit Books, 1987.

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23

Sacks, Oliver W. Renādo no asa. Shōbunsha, 1993.

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24

Sacks, Oliver W. Awakenings. Duckworth, 1996.

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25

(Editor), Eric Victorino, Deanna V. Lopez (Editor), and Photography: Beau Roulette (Illustrator), eds. Coma Therapy. 2nd ed. Orchard City Books And Noise, 2006.

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26

Sandel, M., ed. Coma-emerging Patient. Hanley & Belfus, 1990.

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27

Coma arousal: The family as a team. Doubleday, 1985.

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28

Hamel, Ronald P. Artificial Nutrition and Hydration and the Permanently Unconscious Patient: The Catholic Debate. Georgetown University Press, 2007.

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29

Hundert, Joshua S., Rashmi Verma, Ritika Suri, Anika T. Singh, and Ajay Singh. Neurological Manifestations of Renal Disease. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0191.

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In the United Syates, chronic kidney disease (CKD) affects approximately 5% to 10% of the general population. It is estimated that about 20 million Americans have some degree of CKD. Central nervous system (CNS) abnormalities are common in patients with CKD, especially in those individuals with end stage renal disease (ESRD) who require renal replacement therapy, such as dialysis or transplant. Neurological symptoms in patients with CKD may range from mild altered sensorium and cognitive dysfunction to tremors and coma. By the time patients require renal replacement therapy, some patients may

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30

Tacchi, Mary Jane, and Jan Scott. 5. The evolution of treatments. Oxford University Press, 2017. http://dx.doi.org/10.1093/actrade/9780199558650.003.0005.

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For many centuries, the only intervention for melancholia involved admission into an asylum, initially to keep individuals away from society and then, from the 18th century, to provide therapeutic care. ‘The evolution of treatments’ discusses the crude treatments that were first introduced for inpatients such as sedation (barbiturates and insulin coma therapy) and physical treatments (electroconvulsive therapy and psychosurgery). Next, it discusses the development of the medications that are used today for inpatients and outpatients, such as antidepressants and the mood stabilizer lithium. Fin

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31

Green, Sue. Personal experience of lifelong illness. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199644957.003.0047.

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The author describes his experience of mental illness since the age of 13. He was admitted to the local psychiatric hospital, where his treatment included time in a padded cell, insulin coma therapy, and a leucotomy. He describes his long admission, and other aspects of his life – his marriage and employment, as well as the experience of stigma related to his illness. He reflects on what helps him to cope and how he has managed his life despite all the tribulations.

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32

The song remains the same. G.P. Putnam's Sons, 2012.

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33

The song remains the same. Berkley Books, 2013.

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34

Annane, Djillali, and B. Jérôme Aboab. Management of carbon monoxide poisoning. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0328.

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CO poisoning is the commonest cause of toxic death. Carbon monoxide is colourless, odourless, and tasteless, and is produced under various conditions. When people inhale CO, the gas diffuses rapidly to the body and replaces oxygen at the level of haemoglobin, myoglobin, and other oxygen carriers. Subsequently, CO causes oxygen deprivation of all body tissues. CO also induces oxidative stress and systemic inflammation. After CO poisoning a broad variety of symptoms may occur. Survivors of CO poisoning often present with persistent neurological sequels or develop delayed neurological symptoms. T

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35

Nuwer, Marc R., Ronald G. Emerson, and Cecil D. Hahn. Principles and Techniques for Long-Term EEG Recording (Epilepsy Monitoring Unit, Intensive Care Unit, Ambulatory). Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0031.

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Long-term monitoring is a set of methods for recording electroencephalographic (EEG) signals over a period of 24 hours or longer. Patient video recording is often synchronized to the EEG. Interpretation aids help physicians to identify events, which include automated spike and seizure detection and various trending displays of frequency EEG content. These techniques are used in epilepsy monitoring units for presurgical evaluations and differential diagnosis of seizures versus nonepileptic events. They are used in intensive care units to identify nonconvulsive seizures, to measure the effective

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36

Debaveye, Yves, Dieter Mesotten, and Greet Van den Berghe. Hyperglycaemia, diabetes, and other endocrine emergencies. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0069.

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Although endocrine pathology is usually treated in outpatient clinic, intensive care may be required when endocrinopathies are associated with other medical illnesses or reach a state of decompensation. Although endocrine emergencies are quite rare, they are potentially life-threatening, if not recognized promptly and managed effectively. Therefore, every clinician should always be attentive to a possible diagnosis of these complex disorders. The three major diabetic emergencies comprise diabetic ketoacidosis, hyperglycaemic hyperosmolar state, and prolonged hypoglycaemia. Hyperglycaemic crise

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37

Sebastio, Gianfranco, Manuel Schiff, and Hélène Ogier de Baulny. Lysinuric Protein Intolerance and Hartnup Disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0025.

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Lysinuric protein intolerance (LPI) is an inherited aminoaciduria caused by defective cationic amino acid transport at the basolateral membrane of epithelial cells in intestine and kidney. LPI is caused by mutations in the SLC7A7 gene, which encodes the y+LAT-1 protein, the catalytic light chain subunit of a complex belonging to the heterodimeric amino acid transporter family. Symptoms usually begin after weaning with refusal of feeding, vomiting, and consequent failure to thrive. Hepatosplenomegaly, hematological anomalies, and neurological involvement including hyperammonemic coma will progr

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38

Martinez, Tyler. Encephalitis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0007.

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Encephalitis is an inflammation of the brain parenchyma, typically due to a viral infection. Pure encephalitis will lack the signs and symptoms of meningeal irritation (eg, stiff neck and photophobia). New-onset seizures, cognitive deficits, new psychiatric symptoms, lethargy/coma, cranial nerve abnormalities, or movement disorders should alert the clinician to possible encephalitis. It is important to question the patient about foreign travel, immunocompromised state, and potential exposures. Empiric treatment for presumed viral encephalitis is with the antiviral acyclovir. Empiric broad-spec

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39

Mills, Kerry R., ed. Oxford Textbook of Clinical Neurophysiology. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199688395.001.0001.

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The Oxford Textbook of Clinical Neurophysiology provides a comprehensive account from world experts of the modern practice of the specialty. It deals with the full range of techniques giving the underpinning basic science and clinical use. The importance of clinical skills, as well as technical expertise are emphasized. Section I reviews the physiology of nerve, muscle, and cortex, and the digital techniques used to study them. Section II discusses the techniques for nerve conduction, electromyography (EMG), electroencephalography (EEG), magnetoencephalography, evoked potentials, and transcran

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40

Cooper, Mark S. Hormone therapies in critical illness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0049.

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A range of hormonal manipulations have been proposed as adjunctive therapy during critical care. These therapies might be used to treat a pre-existing or acquired hormonal disorder. Additionally, hormonal manipulation has been suggested to alter the long-term outcome of critical illness, even in patients without structural abnormalities of endocrine glands. Currently, the effectiveness of these anabolic therapies has not been established and they might be harmful in some patient groups. Recently, it has been recognized that many critically-ill patients have low levels of vitamin D and this is

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41

My Sweet Audrina. Simon & Schuster, 2011.

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42

Andrews, V. C. My Sweet Audrina. Pocket, 1987.

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43

Andrews, V. C. My Sweet Audrina. Pocket Books, 1990.

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44

Andrews, V. C. Mi Dulce Audrina. Plaza & Janes Editories Sa, 1986.

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45

Andrews, V. C. My Sweet Audrina. Pocket Books, 2015.

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46

Mi Dulce Audrina. Plaza & Janes Editores, S.A., 1986.

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47

Andrews, V. C. My Sweet Audrina: The Audrina Series, book 1. Simon & Schuster Audio, 2019.

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48

Andrews, V. C. My Sweet Audrina. Pocket, 1990.

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49

Andrews, V. C. My Sweet Audrina. Simon & Schuster, Limited, 2011.

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50

Andrews, V. C. My Sweet Audrina. Pocket, 1985.

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