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Journal articles on the topic 'Coma/therapy'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

Anchikova, L. I., and L. N. Kurshakova. "Diabetic coma therapy." Kazan medical journal 66, no. 1 (1985): 63–68. http://dx.doi.org/10.17816/kazmj60573.

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2

Freeman, EA. "Coma arousal therapy." Clinical Rehabilitation 5, no. 3 (1991): 241–49. http://dx.doi.org/10.1177/026921559100500311.

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3

Gibson, Alan. "Insulin coma therapy." Psychiatric Bulletin 38, no. 4 (2014): 198. http://dx.doi.org/10.1192/pb.38.4.198.

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4

LNU, Mandeep, and Pravin Kumar. "Effectiveness of Early Intervention of Coma Arousal Therapy in Traumatic Head Injury Patients." International Journal of Head and Neck Surgery 3, no. 3 (2012): 137–42. http://dx.doi.org/10.5005/jp-journals-10001-1114.

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ABSTRACT Objective To find out efficacy and benefits of early intervention of coma arousal therapy on coma patients after sustaining traumatic head injury. Materials and methods Thirty comatose patients with traumatic head injury were systematic randomly selected. Both experimental group and control group were having 15 patients each. Patients in experimental group were given coma arousal therapy while those in control group did not receive any coma arousal therapy. Glasgow coma scale (GCS) and coma recovery scale (CRS) were assessed before and after 1 and 2 weeks protocol. Results The indepen

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5

Kots, Kots I. "Adrenal coma hormone therapy case." Kazan medical journal 43, no. 3 (2021): 52–53. http://dx.doi.org/10.17816/kazmj83776.

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When diagnosing various coma, one should bear in mind the possibility of coma due to acute adrenal insufficiency, which develops as a result of partial or complete destruction of the adrenal glands in infectious diseases, adrenal hemorrhages, adrenal vascular thrombosis or surgery on the adrenal glands.

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6

Jones, Kingsley. "Insulin coma therapy in schizophrenia." Journal of the Royal Society of Medicine 93, no. 3 (2000): 147–49. http://dx.doi.org/10.1177/014107680009300313.

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7

Crammer, J. L. "Insulin coma therapy for schizophrenia." Journal of the Royal Society of Medicine 93, no. 6 (2000): 332–33. http://dx.doi.org/10.1177/014107680009300617.

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8

Smythies, John. "Insulin coma therapy for schizophrenia." Journal of the Royal Society of Medicine 93, no. 8 (2000): 449–50. http://dx.doi.org/10.1177/014107680009300821.

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9

Steinhoff, Bernhard J., Klaus-Dieter Stoll, Stefan R. G. Stodieck, and Walter Paulus. "Hyponatremic coma under oxcarbazepine therapy." Epilepsy Research 11, no. 1 (1992): 67–70. http://dx.doi.org/10.1016/0920-1211(92)90023-m.

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10

Chung, Dong Suk, Hyun Sook Kim, Young Moon Han, et al. "Rebounded Response in Propofol Coma Therapy." Korean Journal of Anesthesiology 39, no. 3 (2000): 433. http://dx.doi.org/10.4097/kjae.2000.39.3.433.

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11

Waldman, Scott A., and Dan Park. "Myxedema coma associated with lithium therapy." American Journal of Medicine 87, no. 3 (1989): 355–56. http://dx.doi.org/10.1016/s0002-9343(89)80168-8.

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12

Bourne, Harold. "Insulin coma therapy: let's be factual." Psychiatric Bulletin 38, no. 6 (2014): 308. http://dx.doi.org/10.1192/pb.38.6.308.

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13

Riedel, R. R., P. Clarenbach, and K. P. Reetz. "Coma during azidothymidine therapy for AIDS." Journal of Neurology 236, no. 3 (1989): 185. http://dx.doi.org/10.1007/bf00314341.

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14

IWASA, Kozo, Keiko IRIE, Junji YOSHIOKA, et al. "Barbiturate Coma Therapy for Cerebral Vasospasm in Patients with Severe Subarachnoid Hemorrhage." Surgery for Cerebral Stroke 17, no. 4 (1989): 378–83. http://dx.doi.org/10.2335/scs1987.17.4_378.

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15

Johnson, David A., and Karen Roethig-Johnston. "Coma Stimulation: A Challenge to Occupational Therapy." British Journal of Occupational Therapy 51, no. 3 (1988): 88–90. http://dx.doi.org/10.1177/030802268805100306.

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Sensory stimulation of the severely head injured patient in coma is a controversial topic. There are sound theoretical and experimental bases which suggest the potential importance of stimulation in the acute stage after injury. In the continued absence of any efficacious treatment to improve outcome, the authors consider that it is time to try more diverse approaches to helping the severely head injured patient. Occupational therapists are in a unique position to pursue this goal.

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16

Goebel, H. H., G. Walther, and M. Meuth. "Fresh cell therapy followed by fatal coma." Journal of Neurology 233, no. 4 (1986): 242–47. http://dx.doi.org/10.1007/bf00314028.

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17

Kurbonova, Sabina Tavakalovna Rajabov Og'abek Baxtiyor o'g'li Rashidova Farangiz Musulmon qizi Haydarov Farhod To'ramurodovich Soatova Xolnisa Raxmat qizi. "INSULIN THERAPY." SOLUTION OF SOCIAL PROBLEMS IN MANAGEMENT AND ECONOMY 2, no. 2 (2023): 52–55. https://doi.org/10.5281/zenodo.7634180.

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18

Sinclair, J. R., D. A. K. Watters, and A. Bagshaw. "Non-Traumatic Coma in Zambia." Tropical Doctor 19, no. 1 (1989): 6–10. http://dx.doi.org/10.1177/004947558901900103.

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A series of 170 patients with non-traumatic coma seen over a 16-month period is reported. The Glasgow coma scale significantly correlated with outcome ( P<0.001). The diagnosis was also important in determining outcome. Hospital mortality was lowest in patients with cerebral malaria (22.7%), eclamptic coma (36.4%), and organophosphorous poisoning (30.4%). A diagnostic approach to non-traumatic coma is outlined and the management of the different causes is discussed. Most hospitals in tropical Africa should be able to diagnose up to 90% of cases with non-traumatic coma and simple therapy is

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19

Myles, GL, MD Malkoff, AG Perry, RD Bucholz, and CR Gomez. "Therapeutic Intervention Scoring System used in the care of patients in pentobarbital-induced coma to determine nurse-patient ratios." American Journal of Critical Care 5, no. 1 (1996): 74–79. http://dx.doi.org/10.4037/ajcc1996.5.1.74.

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BACKGROUND: Critical care patients generally require extensive interventions, thereby consuming a large percentage of healthcare resources. Induced pentobarbital coma for the management of increased intracranial pressure is one such intervention, required to maintain patient stability. Quantification of these interventions, as well as the amount of nursing work required, has not been addressed in the literature. OBJECTIVE: To use the Therapeutic Intervention Scoring System to analyze and quantify how interventions affect nurse-patient ratios in the management of patients in pentobarbital coma

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20

Cuturic, Miroslav, and Ruth K. Abramson. "Acute Hyperammonemic Coma with Chronic Valproic Acid Therapy." Annals of Pharmacotherapy 39, no. 12 (2005): 2119–23. http://dx.doi.org/10.1345/aph.1g167.

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21

Kaplan, Robert M. "A history of insulin coma therapy in Australia." Australasian Psychiatry 21, no. 6 (2013): 587–91. http://dx.doi.org/10.1177/1039856213500361.

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22

Agarwal, Vratika, Valay Parikh, Philip E. Otterbeck, and James Lafferty. "Myxedema Coma Induced by Short-term Amiodarone Therapy." American Journal of the Medical Sciences 347, no. 3 (2014): 258–59. http://dx.doi.org/10.1097/maj.0000000000000241.

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23

Jordan, Richard M. "Myxedema coma: Pathophysiology, Therapy, and Factors Affecting Prognosis." Medical Clinics of North America 79, no. 1 (1995): 185–94. http://dx.doi.org/10.1016/s0025-7125(16)30091-8.

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24

Mercadante, Sebastiano, and Fabio Fulfaro. "An Unusual Coma After Therapy For Bone Pain." Journal of Pain and Symptom Management 19, no. 5 (2000): 323–24. http://dx.doi.org/10.1016/s0885-3924(00)00133-0.

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25

Saumya, Gupta, Lalchandani Arati, Gupta Vishal, and Gupta Rajeev. "Prognostic Efficacy Assessment of ACT Therapy and Quinine in Severe Falciparum Malaria." PJSR 12, no. 2 (2019): 20–29. https://doi.org/10.5281/zenodo.8237953.

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In this study, the changes in prognostic indicators such as haemoglobin levels, serum lactate levels, liver function tests, blood sugar levels and glassgow coma scale in patients of severe falciparum malaria treated with Artesunate combination therapy and Quinine have been observed. One hundred patients of severe falciparum malaria were selected from medicine indoor wards. The patients were randomly assigned to artesunate based combination therapy or quinine based therapy. Prognostic parameters like haemoglobin, glasgow coma scale, serum lactates, liver function test and blood sugar levels wer

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26

Martins, Cláudio, Clara Rosa, and Ana Paula Oliveira. "Amiodarone-induced myxoedema coma as a rare differential diagnosis of consciousness disturbance." Case Reports in Internal Medicine 4, no. 1 (2017): 42. http://dx.doi.org/10.5430/crim.v4n1p42.

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Myxoedema coma is an uncommon and life-threatening endocrine emergency with a high mortality rate. This entity is the extreme expression of severe hypothyroidism and the cardinal features are decreased mental status and hypothermia. Diagnosis is challenging and a high index of suspicion is crucial because it allows an early recognition of this condition and prompt initiation of supportive measures along with adequate thyroid replacement therapy. Herein, we report an extremely rare case of myxoedema coma secondary to amiodarone in a 90-year-old female with no previous history of thyroid disease

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27

Roberts, Daniel L., David A. Galbreath, Bhavesh M. Patel, Timothy J. Ingall, Amer Khatib, and Daniel J. Johnson. "Hyperammonemic Coma in an Adult due to Ornithine Transcarbamylase Deficiency." Case Reports in Critical Care 2013 (2013): 1–4. http://dx.doi.org/10.1155/2013/493216.

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Objective. To report an unusual cause of coma in an adult.Design. Case report.Setting. University teaching hospital.Patient. A previously healthy 53-year-old man initially presented with altered mental status and progressed to coma. He was found to be substantially hyperammonemic and did not improve with lactulose therapy and continuous venovenous hemodialysis.Results. Biochemical testing revealed previously undiagnosed ornithine transcarbamylase deficiency, and the patient responded to arginine, sodium phenylacetate, and sodium benzoate.Conclusion. Even in adult patients with no known history

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28

Mahmood Sheikh, Sheikh Abdul Khaliq, Iqbal Azhar, and Shahlla Imam. "Comparative Risk Reduction of Diabetic-Coma in Allopathic and Combination Systems of Therapy – A Cross Sectional Study." RADS Journal of Pharmacy and Allied Health Sciences 1, no. 2 (2023): 34–38. https://doi.org/10.37962/jphs.v1i2.49.

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Objectives: Diabetes-mellitus (DM) may have uncontrolled hyperglycemia or hypoglycemia; which may lead to diabetic-coma. Diabetic-coma is a life-threatening condition. Limited availability of direct comparisons of allopathic and combination system of treatment for risk reduction of diabetic-coma is the main reason to conduct current cross-sectional survey. Methodology: Cross-sectional observational study was conducted at outpatient care-centers. 149 confirmed cases of Type-II Diabetes (T2D) were enrolled in the study after taken written informed consents. Precision analytical technique was use

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29

Blerina Asllanaj, Nazanin Sheikhan, George Trad, and Sean Darmal. "Amiodarone induced myxedema coma presenting with cardiogenic shock and junctional rhythm." World Journal of Advanced Research and Reviews 18, no. 2 (2023): 352–55. http://dx.doi.org/10.30574/wjarr.2023.18.2.0635.

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We describe the case of an elderly female, with no prior history of thyroid dysfunction, who presented with facial swelling and worsening fatigue. She was found to have myxedema coma, with thyroid-stimulating hormone (TSH) 98.90 uIU/mL and free thyroxine (fT4) 0.8 ng/dL, following two months of amiodarone therapy for paroxysmal atrial fibrillation. The patient developed cardiogenic shock and received liothyronine, levothyroxine, stress dose steroids, inotropic and vasopressor therapy while in the intensive care unit (ICU). She was weaned off vasopressors and switched to oral levothyroxine with

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30

Blerina, Asllanaj, Sheikhan Nazanin, Trad George, and Darmal Sean. "Amiodarone induced myxedema coma presenting with cardiogenic shock and junctional rhythm." World Journal of Advanced Research and Reviews 18, no. 2 (2023): 352–55. https://doi.org/10.5281/zenodo.8379923.

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We describe the case of an elderly female, with no prior history of thyroid dysfunction, who presented with facial swelling and worsening fatigue. She was found to have myxedema coma, with thyroid-stimulating hormone (TSH) 98.90 uIU/mL and free thyroxine (fT4) 0.8 ng/dL, following two months of amiodarone therapy for paroxysmal atrial fibrillation. The patient developed cardiogenic shock and received liothyronine, levothyroxine, stress dose steroids, inotropic and vasopressor therapy while in the intensive care unit (ICU). She was weaned off vasopressors and switched to oral levothyroxine with

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31

Kumar, Sanjiv, Nupur Agarwal, and Thankappan S. Sanal. "Effectiveness of coma arousal therapy on patients with disorders of consciousness – A systematic review and meta-analysis." Brain Circulation 10, no. 2 (2024): 119–33. http://dx.doi.org/10.4103/bc.bc_112_23.

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Abstract: BACKGROUND: Disorders of consciousness (DOC) incorporate stages of awareness and arousal. Through coma arousal therapy sensory deprivation experienced by patients with DOC can be mitigated. Nevertheless, consensus concerning its effectiveness on these patients is still fractional. PURPOSE: This review aims to investigate the effectiveness of coma arousal therapies on patients with DOC. METHODS: A meta-analysis was performed by searching electronic databases using search terms, the studies investigating the effect of coma arousal therapy in patients with DOC using the Coma Recovery Sc

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32

Shukla, Dhaval. "Description of coma and coma arousal therapy in Caraka Saṁhitā and its corollary in modern medicine". Neurology India 65, № 2 (2017): 250. http://dx.doi.org/10.4103/neuroindia.ni_1110_16.

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33

Bulashova, O. V., A. V. Jesters, A. K. Saetgaraev, et al. "A case of successful treatment of twelve-day hypoglycemic coma." Kazan medical journal 81, no. 3 (2022): 228. http://dx.doi.org/10.17816/kazmj96717.

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The development of hypoglycemic coma is a severe complication of insulin therapy for diabetes mellitus. According to the literature, being in a hypoglycemic coma for more than 12 hours is associated with a threat to life due to the development of decortication and leads to high mortality. Most often, hypoglycemic coma occurs as a result of the administration of insulin in combination with the intake of large amounts of alcohol against the background of insufficient food intake. The patient's condition in connection with the intake of alcohol is underestimated by others, which leads to late hos

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34

Parihar, Aisha. "Myxedema Coma: Improving Outcomes With Prompt Recognition and Therapy." Journal of the Endocrine Society 5, Supplement_1 (2021): A943. http://dx.doi.org/10.1210/jendso/bvab048.1927.

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Abstract Background: Myxedema coma, a misnomer for severe hypothyroidism, is a rare endocrine emergency with an incidence of 1.08 cases per million people per year and a high mortality rate ranging from 30-50%. A delay in diagnosis and treatment worsens the prognosis and increases morbidity and mortality. Delayed management often leads to decompensation, presenting as uncontrolled persistent hypothermia, severe electrolyte derangements, and a potential for ventilator requirement needing ICU care. We present a patient in hypothyroid crisis who was promptly managed in a non-ICU setting who demon

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35

Aldridge, D., D. Gustorff, and H.-J. Hannich. "Where am I? Music therapy applied to coma patients." Journal of the Royal Society of Medicine 83, no. 6 (1990): 345–46. http://dx.doi.org/10.1177/014107689008300602.

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36

Church, Chelsea O., and Erin C. Callen. "Myxedema Coma Associated with Combination Aripiprazole and Sertraline Therapy." Annals of Pharmacotherapy 43, no. 12 (2009): 2113–16. http://dx.doi.org/10.1345/aph.1m369.

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37

Perez, N., M. A. Quera-Salva, B. Claustrat, C. Ioos, F. Chéliout-Héraut, and J. M. Pinard. "260 Light therapy and post-traumatic coma in children." European Journal of Paediatric Neurology 3, no. 6 (1999): A153. http://dx.doi.org/10.1016/s1090-3798(99)91386-7.

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38

Kwon, Hyun Mook, Jin Wook Baek, Sang Pyung Lee, and Jae Ik Cho. "A Fatal Adverse Effect of Barbiturate Coma Therapy: Dyskalemia." Korean Journal of Neurotrauma 12, no. 2 (2016): 156. http://dx.doi.org/10.13004/kjnt.2016.12.2.156.

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39

O'Brien, D. R. "The effective agent in electroconvulsive therapy: Convulsion or coma?" Medical Hypotheses 28, no. 4 (1989): 277–80. http://dx.doi.org/10.1016/0306-9877(89)90084-4.

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40

Mandeep, Naveen Chitkara, Sandeep Goel, and Sudhir Sood. "Traumatic head injury: Early intervention by coma arousal therapy." Indian Journal of Neurotrauma 10, no. 1 (2013): 13–18. http://dx.doi.org/10.1016/j.ijnt.2013.05.004.

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41

Spiegal, David M. "Acute Renal Failure and Coma Secondary to Acyclovir Therapy." JAMA: The Journal of the American Medical Association 255, no. 14 (1986): 1882. http://dx.doi.org/10.1001/jama.1986.03370140080027.

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42

Aleksandrov, M. V., T. V. Aleksandrova, V. S. Chernyi1, and M. A. Lucyk. "Effect of reconstituted glutathione preparations on bioelectric brain activity in severe ethanol poisoning." Toxicological Review, no. 2 (May 15, 2020): 18–24. http://dx.doi.org/10.36946/0869-7922-2020-2-17-23.

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Fifty patients with acute severe ethanol poisoning (depression of consciousness at the time of admission was up to the level of coma) were examined. Based on the purpose of the study, patients were divided into two groups. In the first group, basic etiopathogenetic therapy was supplemented by the intravenous administration of a drug based on reduced glutathione: inosine glycyl-cysteinyl-glutamate disodium (IGCGD). In the second group, only basic therapy was performed. All patients underwent in the intensive care long-term continuous EEG monitoring, which was started at the end of basic resusci

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43

Ito, Ryo, Toshihide Hamabe, Masaya Ono, et al. "10051-CBMS-4 CURCUMIN ANALOG COMPOUND A INHIBITS THE GROWTH OF GLIOBLASTOMA BY INDUCING G2/M PHASE ARREST AND APOPTOSIS AT A LOWER CONCENTRATION THAN CURCUMIN." Neuro-Oncology Advances 5, Supplement_5 (2023): v7. http://dx.doi.org/10.1093/noajnl/vdad141.027.

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Abstract PURPOSE Glioblastoma (GBM) is the most common and aggressive primary brain tumor. Over 15,000 patients are diagnosed with GBM yearly, with a median survival expectancy of&lt;15 months and&lt;10% survival rate beyond five years, despite applying conventional multimodal treatment containing surgical resection, chemotherapy, and radiation therapy. The efficiency of conventional therapies is limited due to the lack of specificity and the accuracy of eradicating aggressive diffusing GBM cells. Developing new therapeutic agents against GBM is strongly recommended. The purpose of thi

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44

Mangla, Pragya, Khalid Hussain, Sian Ellard, Sarah E. Flanagan, and Vijayalakshmi Bhatia. "Diazoxide toxicity in a child with persistent hyperinsulinemic hypoglycemia of infancy: mixed hyperglycemic hyperosmolar coma and ketoacidosis." Journal of Pediatric Endocrinology and Metabolism 31, no. 8 (2018): 943–45. http://dx.doi.org/10.1515/jpem-2018-0112.

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Abstract Background: Persistent hyperinsulinemic hypoglycemia of infancy (PHHI), also known as congenital hyperinsulinism, has been known to go into spontaneous remission, with patients developing diabetes in later life. A temporary phase of hyperglycemia is, however, rarely reported. Case presentation: We describe a 16-month-old child, a known case of diazoxide responsive PHHI, presenting with mixed hyperglycemic hyperosmolar coma and ketoacidosis with rhabdomyolysis while on diazoxide treatment. The patient required temporary cessation of diazoxide and initiation of insulin infusion, followe

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45

Agulnik, Asya, Daniel P. Kelly, Rebecca Bruccoleri, et al. "Combination Clearance Therapy and Barbiturate Coma for Severe Carbamazepine Overdose." Pediatrics 139, no. 5 (2017): e20161560. http://dx.doi.org/10.1542/peds.2016-1560.

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46

Barrueto, Fermin, and Jason B. Hack. "Hyperammonemia and Coma without Hepatic Dysfunction Induced by Valproate Therapy." Academic Emergency Medicine 8, no. 10 (2001): 999–1001. http://dx.doi.org/10.1111/j.1553-2712.2001.tb01102.x.

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47

Sohn, Seil, and Chul-Kee Park. "Fatal Fluctuation of Serum Potassium Level during Barbiturate Coma Therapy." Journal of Korean Neurotraumatology Society 5, no. 2 (2009): 99. http://dx.doi.org/10.13004/jknts.2009.5.2.99.

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48

Patoliya, Foram, Bansi Khunt, Labhu Pithiya, et al. "A Novel Approach to Facilitate Arousal Following Infectious Encephalitis in Acute Setup – A Case Report." Asian Pacific Journal of Health Sciences 9, no. 4 (2022): 1–2. http://dx.doi.org/10.21276/apjhs.2022.9.4s1.01.

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Encephalitis is the inflammatory disease of brain parenchyma that manifests with a spectrum of disorders including altered consciousness, impaired cognition, associated neurological impairments, aphasia, behavioral abnormalities leading to an increase in economic burden as well as delayed prognosis. However, initiating early rehabilitation to promote arousal that is task based and has associated personal salience to the patient can pay way to early arousal and shorter length of stay in acute unit. We developed a subject specific integrated multisensory stimulation technique (SSIMS) to facilita

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49

Veselý, Pavol, Ľudovít Veselý, Veronika Combová, and Matěj Žukovič. "Specific Corneal Parameters and Visual Acuity Changes After Corneal Crosslinking Treatment for Progressive Keratoconus." Czech and Slovak Ophthalmology 77, no. 4 (2021): 184–89. http://dx.doi.org/10.31348/2021/21.

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Aim: To evaluate the effect of crosslinking (CXL) therapy on the change in the quality of visual acuity and the change in the topographic properties of the cornea – curvature, pachymetry, and change of astigmatism, coma abberation and CLMIaa (Cone Localisation and Magnitude Index). Methods: A retrospective analytical study included 29 eyes of 24 patients who had progressed in the last 12 months and were suitable candidates for CXL surgery. The monitored parameters were the steepest, flatest and mean anterior instantaneous curvature (AICS, AICF, AICM) and the steepest, flatest and mean posterio

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50

Savateeva-Lyubimova, T. N., A. G. Aleksandrov, S. B. Kazakova, and K. I. Stosman. "Comparative analysis of the effect of Reamberin and Methadoxyl on the course of acute severe ethanol poisoning." Ural Medical Journal 22, no. 6 (2023): 59–67. http://dx.doi.org/10.52420/2071-5943-2023-22-6-59-67.

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Introduction. One of the ways to improve the quality of intensive therapy in severe forms of acute alcohol poisoning is the inclusion in the complex of measures of means capable of reducing the manifestations of oxidative stress. The aim of the work was a comparative evaluation of the effectiveness of drugs Reamberin and Methadoxyl in acute severe ethanol poisoning on rats. Materials and methods. Acute severe alcohol poisoning was modeled in Wistar rats by 40 % ethanol solution at a dose 8,6 g/kg. The studied drugs were administered infusively in the volume of 20 ml/kg for 60 min at an average

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