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Journal articles on the topic 'Common variable immunodeficiency (CVID)'

Author: Grafiati

Published: 4 June 2021

Last updated: 18 February 2022

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1

Suter-Meyer, Nigg, Kolyvanos Naumann, Käser, and Vetter. "Common Variable Immunodeficiency (CVID) – variables humorales Immundefektsyndrom." Praxis 96, no. 1 (2007): 3–11. http://dx.doi.org/10.1024/1661-8157.96.1.3.

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Vukas, Emina, Aida Dizdarević, Senka Mesihović - Dinarević, and Adisa Čengić. "Common variable immunodeficiency – case report." Journal of Health Sciences 3, no. 2 (2013): 170–72. http://dx.doi.org/10.17532/jhsci.2013.83.

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Common variable immunodeficiency (CVID) or acquired hypogammaglobulinemia is the type of primary immunodeficiency. Deregulation of the immune system, leading to hypogammaglobulinemia, defective activation and proliferation of T cells and dendritic cells, and malfunction of the cytokines are observed in CVID. The clinical picture of CVID varies, any organ or system can be affected, therefore the diagnosis is often difficult and delayed and sometimes is not always possible. This article describes a twelve years old boy with all the clinical signs of immunodeficiency, as confi rmed by laboratory.

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Capistrano, Giovany Gomes, Gdayllon Cavalcante Meneses, Fernanda Macedo de Oliveira Neves, Renata de Almeida Leitão, Alice Maria Costa Martins, and Alexandre Braga Libório. "Renal Evaluation in Common Variable Immunodeficiency." Journal of Immunology Research 2018 (2018): 1–6. http://dx.doi.org/10.1155/2018/5841031.

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Introduction. Common variable immunodeficiency (CVID) comprises a heterogeneous group of disorders characterized by impaired antibody production. Kidney involvement in CVID is described in isolated and sporadic case reports. The objective of this study was to study the renal function pattern in CVID patients through glomerular and tubular function tests. Methods. Study of 12 patients with CVID diagnosis and 12 healthy control individuals. Glomerular filtration rate (GFR), fractional excretion of sodium (FENa+) and potassium (FEK+), urinary concentration, and acidification capacity were measure

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4

Caza, Tiffany N., Samar I. Hassen, and Christopher P. Larsen. "Renal Manifestations of Common Variable Immunodeficiency." Kidney360 1, no. 6 (2020): 491–500. http://dx.doi.org/10.34067/kid.0000432020.

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BackgroundCommon variable immunodeficiency (CVID) is one of the most common primary immunodeficiency syndromes, affecting one in 25,000–50,000 people. Renal insufficiency occurs in approximately 2% of patients with CVID. To date, there are no case series of renal biopsies from patients with CVID, making it difficult to determine whether individual cases of renal disease in CVID represent sporadic events or are related to the underlying pathophysiology. We performed a retrospective analysis of renal biopsy specimens in our database from patients with a clinical history of CVID (n=22 patients, 2

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Tanir Basaranoglu, Sevgen, Sukru Cekic, Emine Kirhan, Melahat Dirican, and Sara S. Kilic. "Oxidative stress in common variable immunodeficiency." European Journal of Inflammation 19 (January 2021): 205873922110024. http://dx.doi.org/10.1177/20587392211002411.

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Common variable immunodeficiency (CVID) is a heterogenous group of immunologic disorders of unknown etiology. Alterations of the normal cellular balance due to an increase in reactive oxygen species and/or decrease in antioxidant defense may lead to increased oxidative stress. We aimed to evaluate the levels of oxidative stress biomarkers in patients with CVID who had different presentations. We investigated the serum catalase (CAT), erythrocyte superoxide dismutase (SOD), erythrocyte reduced glutathione as antioxidants and serum malondialdehyde levels as lipid peroxidation marker in patients

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Akkinepally, Sudha, Christian Mendez, and Vijayarama Poreddy. "Giardiasis in Common Variable Immunodeficiency (CVID)." American Journal of Gastroenterology 105 (October 2010): S247—S248. http://dx.doi.org/10.14309/00000434-201010001-00685.

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7

Coraglia, Ana, Nora Galassi, Diego S. Fernández Romero, et al. "Common Variable Immunodeficiency and Circulating TFH." Journal of Immunology Research 2016 (2016): 1–10. http://dx.doi.org/10.1155/2016/4951587.

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CD4+ T follicular helper cells (TFH) were assessed in adult patients with common variable immune deficiency (CVID) classified according to the presence of granulomatous disease (GD), autoimmunity (AI), or both GD and AI (Group I) or the absence of AI and GD (Group II).TFHlymphocytes were characterized by expression of CXCR5 and PD-1.TFHwere higher (in both absolute number and percentage) in Group I than in Group II CVID patients and normal controls (N). Within CXCR5+CD4+ T cells, the percentage of PD-1 (+) was higher and that of CCR7 (+) was lower in Group I than in Group II and N. The percent

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8

Ellwood, Amanda, and Caroline Hamm. "Common Variable Immunodeficiency Presenting with Hodgkin’s Disease." Blood 106, no. 11 (2005): 4642. http://dx.doi.org/10.1182/blood.v106.11.4642.4642.

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Abstract We report a case of a 32 year old man with classic Common Variable Immunodeficiency Disease (CVID) and Hodgkin’s disease. Repeated immunoglobulin studies as a child showed hypogammaglobulinemia and decreased mitogen responses, with the suggestion of CVID. He presented to the oncology clinic with nearly three decades of recurrent reactive lymphadenopathy and multiple lymph node biopsies throughout his lifetime, recurrent pulmonary infections, and multiple autoimmune manifestations including diabetes mellitus onset at age 4, autoimmune hemolytic anemia and autoimmune thrombocytopenia. H

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9

Pecoraro, Heidi L., M. Julia B. Felippe, Andrew D. Miller, et al. "Neuroborreliosis in a horse with common variable immunodeficiency." Journal of Veterinary Diagnostic Investigation 31, no. 2 (2019): 241–45. http://dx.doi.org/10.1177/1040638718824146.

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Common variable immunodeficiency (CVID) is a rare condition in adult horses characterized by hypogammaglobulinemia and increased susceptibility to parasitic and bacterial infections, including recurrent respiratory diseases, septicemia, and meningitis. Lyme disease is often included as a differential diagnosis in CVID horses with signs of meningitis; however, the Borrelia burgdorferi organism has not been demonstrated previously within central nervous system tissues of CVID horses with neurologic disease, to our knowledge. We report herein a case of neuroborreliosis in a CVID horse, confirmed

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10

Montazeri, Maryam, Mohammad Reza Ebadpour, Farideh Kouchak, and Naser Esmaeili. "Common Variable Immunodeficiency (CVID): A Case Report." Internal Medicine and Medical Investigation Journal 2, no. 1 (2017): 26. http://dx.doi.org/10.24200/imminv.v2i1.44.

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Common variable immune deficiency disease is the most prevalent acquired immune deficiency in human being after selective immunoglobulin A deficiency. It causes reduction of immunoglobulin levels and specific antibodies production and enhancement of recurrent and chronic infections risk, especially respiratory infections. CVID patients faces increased risk of granulomatous disease, autoimmune and phenomenon and malignancy. The disease involves males and females equally. Some studies showed that early diagnosis of CVID disease and regular treatment of patients with IVIG may have an efficient ro

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Fernandes, Karin Sá, Michella Bezerra Lima, Cíntia de Paula Martins, et al. "Salivary Immunoglobulins in Individuals with Common Variable Immunodeficiency." Brazilian Dental Journal 27, no. 6 (2016): 641–45. http://dx.doi.org/10.1590/0103-6440201601096.

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Abstract Oral manifestations of common variable immunodeficiency (CVID) are rare, have rarely been studied and have given controversial results. There are few data about IgA, IgG, and IgM antibody salivary levels in the literature, and there are few papers about the clinical impact of antibody deficiencies and CVID on the oral health of such patients. The aim of this study was to measure serum and salivary IgA, IgG, and IgM levels in CVID participants and controls, and to associate immunoglobulin levels with caries and periodontal disease. This was a case-control study involving 51 CVID indivi

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12

Cunningham-Rundles, Charlotte. "Common variable immune deficiency: case studies." Blood 134, no. 21 (2019): 1787–95. http://dx.doi.org/10.1182/blood.2019002062.

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In this review, the authors describe 3 patients with common variable immunodeficiency (CVID), noting the disease manifestations most relevant to the practicing hematologist, especially autoimmune cytopenias, benign lymphoproliferation, granulomatous disease, and lymphomas such as common noninfectious complications of CVID.

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Bonilla, Francisco A. "Personalized therapy for common variable immunodeficiency." Allergy and Asthma Proceedings 41, no. 1 (2020): 19–25. http://dx.doi.org/10.2500/aap.2020.41.190012.

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Background: Common variable immunodeficiency (CVID) represents a clinical descriptive diagnosis that was defined in the 1970s. Despite the vast increase in knowledge with regard to immune function and genetics, the pathophysiology of this disorder remains poorly understood in the majority of patients (75%); however, recent advances have led to a much clearer understanding of this heterogeneous group of disorders in the remaining 25%. These advances, along with developments in immune modulatory and reconstitution therapies, now permit sophisticated and specific targeting of therapies for indivi

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Speth, F. "Common Variable Immunodeficiency (CVID) und therapierefraktäre Polyarthritis." Arthritis und Rheuma 32, no. 05 (2012): 330–33. http://dx.doi.org/10.1055/s-0037-1618142.

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15

Spickett, G. P. "Current perspectives on common variable immunodeficiency (CVID)." Clinical & Experimental Allergy 31, no. 4 (2001): 536–42. http://dx.doi.org/10.1046/j.1365-2222.2001.01117.x.

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16

Mathew, S. "Thrombocytopenia associated with common variable immunodeficiency (CVID)." Journal of Allergy and Clinical Immunology 115, no. 2 (2005): S159. http://dx.doi.org/10.1016/j.jaci.2004.12.648.

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17

Nasa, Mukesh, Smruti Ranjan Mishra, Lipika Lipi, and Randhir Sud. "Common variable immunodeficiency syndrome with chronic diarrhoea." BMJ Case Reports 12, no. 3 (2019): e228240. http://dx.doi.org/10.1136/bcr-2018-228240.

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Common variable immunodeficiency syndrome (CVID) is a heterogeneous disorder characterised by diminished levels of IgG, IgA and/or IgM, and recurrent bacterial infections. Sinopulmonary infections are most commonly reported followed by gastrointestinal (GI) infections. GI tract represents the largest immune organ with abundance of lymphoid cells, its involvement can manifest variably ranging from asymptomatic involvement to florid symptoms and signs. Diffuse nodular lymphoid hyperplasia (DNLH) of the GI tract is characterised by numerous small polypoid nodules of variable size in the small int

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18

Huq, Madiha, Neha Sanan, Phuong Daniels, and Robert Hostoffer. "Posner-Schlossman Syndrome in Common Variable Immunodeficiency." Case Reports in Ophthalmological Medicine 2020 (October 15, 2020): 1–3. http://dx.doi.org/10.1155/2020/8843586.

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Introduction. Posner-Schlossman syndrome (PSS) is a rare glaucomatocyclitic crisis with clinical features including recurrent episodes of unilateral elevated intraocular pressure. Autoimmune and infectious causes have been proposed as potential etiologies of PSS. We report the first case of PSS in the setting of common variable immunodeficiency (CVID). Case Report. A sixty-two-year-old Caucasian female with a medical history of CVID and ulcerative colitis presented to the emergency room with complaints of acute right-sided vision changes. She reported image distortion, blurriness, and loss of

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Shabashova, N. V., L. V. Filippova, A. E. Uchevatkina, and E. V. Frolova. "Common variable immunodeficiency in adults." Terapevticheskii arkhiv 88, no. 11 (2016): 94–98. http://dx.doi.org/10.17116/terarkh2016881194-98.

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The paper analyzes 7 cases of common variable immune deficiency (CVID), a primary immunodeficiency disease. All the cases were detected in outpatients over the age of 40 years. The diagnosis was based on their history data and general clinical findings with due regard for the results of previously conducted functional studies, expert opinions, and the results of immunological studies including the quantitative and functional indices of T and B cells, phagocytes and the levels of immunoglobulins. The analysis showed that the early signs of impaired immunity in all the patients were seen by phys

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20

Ricciardi, Luisa, Fabiana Furci, Antonio Ieni, and Antonio Macrì. "Castleman Disease in a Patient with Common Variable Immunodeficiency." Case Reports in Immunology 2019 (February 14, 2019): 1–5. http://dx.doi.org/10.1155/2019/5476383.

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Common variable immunodeficiency (CVID) is a primary immunodeficiency due to a disorder of the adaptive immune system which causes hypogammaglobulinemia and therefore an increased susceptibility to infection; noninfectious, inflammatory conditions including systemic autoimmunity and lymphoproliferative complications are also commonly associated with CVID. Castleman disease (CD) is a systemic disease clinically characterized by diffuse lymphadenopathy, splenomegaly, anemia, and systemic inflammatory symptoms. This makes CD a great mimicker of more common benign and malignant masses in the neck,

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21

Silva, Gisele Barbosa e., Karla Pereira Fernandes, and Gesmar Rodrigues Silva Segundo. "Common variable immunodeficiency and isosporiasis: first report case." Revista da Sociedade Brasileira de Medicina Tropical 45, no. 6 (2012): 768–69. http://dx.doi.org/10.1590/s0037-86822012000600023.

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We report a severe case of diarrhea in a 62-year-old female HIV-negative patient from whom Giardia lamblia and Isospora belli were isolated. Because unusual and opportunistic infections should be considered as criteria for further analysis of immunological status, laboratory investigations led to a diagnosis of common variable immunodeficiency (CVID). This is the first reported case of isosporiasis in a patient with CVID and illustrates the importance of being aware of a possible link, particularly in relation to primary immunodeficiency.

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22

Holm, Are Martin, Pål Aukrust, Jan Kristian Damås, Fredrik Müller, Bente Halvorsen, and Stig S. Frøland. "Abnormal interleukin-7 function in common variable immunodeficiency." Blood 105, no. 7 (2005): 2887–90. http://dx.doi.org/10.1182/blood-2004-06-2423.

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AbstractCommon variable immunodeficiency (CVID) is characterized by low levels of circulating immunoglobulins, leading to frequent infections, particularly of the respiratory tract. Frequently, T-cell abnormalities are observed. Interleukin-7 (IL-7) is involved in the homeostasis of lymphocytes, and may be elevated in lymphopenia. Mutations of genes related to IL-7 may lead to severe immunodeficiency disorders. We report elevated plasma levels of circulating IL-7 in a subgroup of CVID. These patients have increased numbers of circulating CD8+ T cells with decreased apoptosis and a predominance

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23

Khan, Mohammad Zahirul Islam, Kamrul Laila, Mohammed Mahbubul Islam, Mohammad Imnul Islam, and Shahana Akhter Rahman. "Common variable immunodeficiency disorder associated with bronchiectasis: a case report." International Journal of Contemporary Pediatrics 8, no. 3 (2021): 565. http://dx.doi.org/10.18203/2349-3291.ijcp20210665.

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Common variable immunodeficiency disorder (CVID) is the commonest type of primary immunodeficiency disorders (PIDs) characterized by hypogammaglobulinemia, defective specific antibody production and increased susceptibility of recurrent infections. Autoimmunity, neoplasm and lymphoproliferative disorders are usually associated with CVID. In most cases, the cause is unknown, but multiple gene mutations (10%) may be associated with CVID. Here, we report an eight years old girl with CVID presented with recurrent infections, growth failure, generalized lymphadenopathy and hepatosplenomegaly. Chest

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Barmettler, Sara, Jocelyn Farmer, Alexandra Brady, Jolan E. Walter, and Mark Cobbold. "Gastrointestinal (GI) Manifestations in Common Variable Immunodeficiency (CVID)." Journal of Allergy and Clinical Immunology 141, no. 2 (2018): AB82. http://dx.doi.org/10.1016/j.jaci.2017.12.264.

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Shribman, Samuel E., Jessica Katanga, Nadeem Ali, et al. "Encephalomyelitis with Retinopathy in Common Variable Immunodeficiency (CVID)." Neuro-Ophthalmology 44, no. 1 (2018): 38–40. http://dx.doi.org/10.1080/01658107.2018.1542008.

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26

Patuzzo, Giuseppe, Alessandro Barbieri, Elisa Tinazzi, et al. "Autoimmunity and infection in common variable immunodeficiency (CVID)." Autoimmunity Reviews 15, no. 9 (2016): 877–82. http://dx.doi.org/10.1016/j.autrev.2016.07.011.

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Grossman, William J., Patrick O. Bonnet, and Yan Xiong. "Increased Prevalence of Neoplasms In Patients Diagnosed with Common Variable Immunodeficiency." Blood 116, no. 21 (2010): 1531. http://dx.doi.org/10.1182/blood.v116.21.1531.1531.

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Abstract Abstract 1531 Introduction: Common variable immunodeficiency (CVID) is a heterogenous disorder characterized by hypogammaglobulinemia, poor specific antibody responses, and increased numbers of infections. In addition, patients with CVID have been reported to carry an increased risk of developing other co-morbidities, including autoimmunity and certain types of neoplasms. Data establishing the overall risk and prevalence of these co-morbidities in patients with CVID has been limited due to the small sizes of patient cohorts examined to-date, and the lack of comprehensive primary immun

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Kokron, Cristina M., Paolo R. Errante, Myrthes T. Barros, et al. "Clinical and laboratory aspects of common variable immunodeficiency." Anais da Academia Brasileira de Ciências 76, no. 4 (2004): 707–26. http://dx.doi.org/10.1590/s0001-37652004000400007.

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Common variable immunodeficiency (CVID) is an immunological disorder characterized by defective antibody production, recurrent infections, most notably of the respiratory tract, autoimmune phenomena and cancer. Some CVID patients may also present disturbances of the cellular immune response such as a decrease in the number and proportion of different lymphocyte populations, diminished lymphoproliferative response to mitogens and antigens, altered production of cytokines, and deficient expression of cell-surface molecules. Most Brazilian CVID patients included in this study show a decrease in T

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Litzman, Jiří, Zita Chovancová, Petr Bejdák, Marek Litzman, Zdeněk Hel, and Marcela Vlková. "Common variable immunodeficiency patients display elevated plasma levels of granulocyte activation markers elastase and myeloperoxidase." International Journal of Immunopathology and Pharmacology 33 (January 2019): 205873841984338. http://dx.doi.org/10.1177/2058738419843381.

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Common variable immunodeficiency disorders (CVIDs) represent a group of primary immunodeficiency diseases characterized by hypogammaglobulinemia and dysfunctional immune response to invading pathogens. Previous studies have indicated that CVID is associated with microbial translocation and systemic myeloid cell activation. The goal of this study was to determine whether patients with CVID display elevated systemic levels of markers of granulocyte activation and whether the levels are further influenced by intravenous immunoglobulin (IVIg) infusions. The plasma levels of granulocyte activation

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Aytekin, Gökhan, Fatih Çölkesen, Eray Yıldız, Mitat Arıcıgil, and Şevket Arslan. "Hearing Assessment in Adult Patients with Common Variable Immunodeficiency." Asthma Allergy Immunology 19, no. 1 (2021): 38–45. http://dx.doi.org/10.21911/aai.600.

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ABSTRACT Objective: Common Variable Immunodeficiency (CVID) is a primary antibody disorder characterized by impaired B cell differentiation. Patients commonly present with acute and chronic sinusitis as well as otitis media, which may lead to hearing loss. Materials and Methods: Thirty-three CVID patients (20 male /13 female) with a mean age of 35 years (range 19-65 years) and 33 healthy individuals as a control group were included. Results: Among CVID patients, 17 (51.5%) had conductive hearing loss (CHL) , being unilateral in 4 (12.1%) and bilateral in 13 (39.4%). Unilateral and bilateral se

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López-Aldabe, K., L. Hidalgo, A. Antolí, G. Rocamora, X. Corbella, and X. Solanich. "OP0004 AUTOIMMUNE AND INFLAMMATORY MANIFESTATIONS IN COMMON VARIABLE IMMUNODEFICIENCY DISORDERS." Annals of the Rheumatic Diseases 80, Suppl 1 (2021): 1.2–2. http://dx.doi.org/10.1136/annrheumdis-2021-eular.2696.

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Background:Common variable immunodeficiency (CVID) disorders are the second most frequent immunodeficiency worldwide and autoimmune diseases (AD) are present in 20% of such patients, cytopenia being the most frequent manifestation [1]. Defects in central and peripheral tolerance, activation/proliferation of B cells, and hypogammaglobulinemia are key features of the disease, along with a reduction in CD4+T cells, abnormalities in Treg and defective secretion of regulatory cytokines, that could perpetuate autoimmune - autoinflammatory phenomena.Objectives:To describe immune and inflammatory diso

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Li, Ran, Yali Zheng, Yuqian Li, et al. "Common Variable Immunodeficiency with Genetic Defects Identified by Whole Exome Sequencing." BioMed Research International 2018 (September 30, 2018): 1–7. http://dx.doi.org/10.1155/2018/3724630.

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Common variable immunodeficiency (CVID) belongs to the primary immunodeficiency disorders (PIDs), presenting a profound heterogeneity in phenotype and genotype, with monogenic or complex causes. Recurrent respiratory infections are the most common clinical manifestations. CVID patients can also develop various autoimmune and lymphoproliferative complications. Genetic testing such as whole exome sequencing (WES) can be utilized to investigate likely genetic defects, helping for better clinical management. We described the clinical phenotypes of three sporadic cases of CVID, who developed recurr

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Cunningham-Rundles, Charlotte. "The many faces of common variable immunodeficiency." Hematology 2012, no. 1 (2012): 301–5. http://dx.doi.org/10.1182/asheducation.v2012.1.301.3798316.

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Abstract Common variable immunodeficiency (CVID) is a rare immune deficiency characterized by low levels of serum IgG, IgA, and/or IgM, with a loss of Ab production. The diagnosis is most commonly made in adults between the ages of 20 and 40 years, but both children and much older adults can be found to have this immune defect. The range of clinical manifestations is broad, including acute and chronic infections, inflammatory and autoimmune diseases, and an increased incidence of cancer and lymphoma. For all of these reasons, the disease phenotype is both heterogeneous and complex. In the past

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Sobko, E. A., I. V. Demko, I. A. Soloveva, et al. "Common variable immunodeficiency disorder: a clinical case." Medical Immunology (Russia) 23, no. 1 (2021): 185–90. http://dx.doi.org/10.15789/1563-0625-cvi-2089.

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Primary immunodeficiency is a rare congenital pathology associated with failure of immune system, manifested by disturbances of its functions. These defects lead to increased susceptibility of patients to various infectious agents, as well as the development of autoimmune, malignant and other diseases. Primary immunodeficiency is classified as a rare disease, which was previously associated with a poor prognosis with a high risk of mortality in childhood. To date, the emergence of highly effective treatment methods has changed the course and prognosis of these diseases. Clinicians of various s

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Mantzaris, Gerassimos J., Despina Chrysovergi, Kalliopi Petraki, et al. "H. pylori infection in common variable immunodeficiency disease (CVID)." Gastroenterology 118, no. 4 (2000): A1270. http://dx.doi.org/10.1016/s0016-5085(00)80927-0.

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Gause, Angela, Daniela Christine Inderrieden, Rudolf Laas, Andreas-Christoph Arlt, and Wolfgang Ludwig Gross. "Common Variable Immunodeficiency (CVID) and Inclusion Body Myositis (IBM)." Immunobiology 202, no. 2 (2000): 199–203. http://dx.doi.org/10.1016/s0171-2985(00)80067-4.

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Hammarström, L., I. Vorechovsky, and D. Webster. "Selective IgA deficiency (SIgAD) and common variable immunodeficiency (CVID)." Clinical & Experimental Immunology 120, no. 2 (2000): 225–31. http://dx.doi.org/10.1046/j.1365-2249.2000.01131.x.

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Sharma, T., and M. Riedl. "P243 Gastrointestinal manifestations of common variable immunodeficiency disorder (CVID)." Annals of Allergy, Asthma & Immunology 117, no. 5 (2016): S94. http://dx.doi.org/10.1016/j.anai.2016.09.255.

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Jacob, C. M. A., A. P. M. Castro, A. C. Pastorino, et al. "Common variable immunodeficiency (CVID) and cryptosporidiosis of difficult control." Journal of Allergy and Clinical Immunology 115, no. 2 (2005): S154. http://dx.doi.org/10.1016/j.jaci.2004.12.628.

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Goldacker, S., R. Draeger, K. Warnatz, et al. "Active vaccination in patients with common variable immunodeficiency (CVID)." Clinical Immunology 124, no. 3 (2007): 294–303. http://dx.doi.org/10.1016/j.clim.2007.04.011.

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Jeong, D., S. Skoda-Smith, A. Shimamura, and H. Ochs. "Dyskeratosis Congenita (DC) Presenting as Common Variable Immunodeficiency (CVID)." Journal of Allergy and Clinical Immunology 125, no. 2 (2010): AB14. http://dx.doi.org/10.1016/j.jaci.2009.12.086.

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Long, Christopher P., Hiroshi Suzuki, and Kenneth Vitale. "Peripheral Neuropathy Due to Common Variable Immunodeficiency: Case Report and Narrative Review." Gerontology and Geriatric Medicine 5 (January 2019): 233372141985064. http://dx.doi.org/10.1177/2333721419850644.

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A 63-year-old woman with common variable immunodeficiency (CVID) presented with 1 year of insidious onset lower extremity pain and weakness. She underwent a circuitous workup, failed to improve despite treatment for various presumed diagnoses. She presented to a University physical medicine and rehabilitation clinic with continued symptoms. Electrophysiologic testing was recommended revealing a lower extremity motor greater than sensory axonal neuropathy. While CVID has known central nervous system complications, to our knowledge, this represents the second known reported case of peripheral ne

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Karaulov, Alexander V., Irina V. Sidorenko, and Anna S. Kapustina. "Major approaches in early diagnostics of common variable immunodeficiency in adults in Moscow." F1000Research 1 (November 9, 2012): 46. http://dx.doi.org/10.12688/f1000research.1-46.v1.

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Common variable immunodeficiency (CVID) is a primary immunological disease characterized predominantly by hypogammaglobulinemia. The main clinical manifestations are severe recurrent infections that often lead to structural damage of affected organs. The early start of adequate intravenous immunoglobulin therapy has significantly improved the prognosis of this serious disorder. Patients with CVID are also predisposed to autoimmune and lymphoproliferative complications. This article deals with the features of this primary immunodeficiency in adults. Clinical manifestations, immunological featur

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Khan, Rishad, Mohamad Habbal, Michael A. Scaffidi, et al. "Gastrointestinal Disease in Patients with Common Variable Immunodeficiency: A Retrospective Observational Study." Journal of the Canadian Association of Gastroenterology 3, no. 4 (2019): 162–68. http://dx.doi.org/10.1093/jcag/gwz004.

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Abstract Background Gastrointestinal (GI) symptoms are common among patients with common variable immunodeficiency disorder (CVID) yet remain poorly understood. Aims The aim of this study was to characterize the demographic, clinical, endoscopic and histologic features of patients with CVID and GI symptoms. Methods We conducted a retrospective observational study of all patients with CVID at a large Canadian tertiary care centre between January 2000 and May 2018. Results We included 95 patients with CVID. The mean age of patients at the time of CVID diagnosis was 38.2(±16.0). Fifty-three (56%)

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Baloh, Carolyn H., Michele R. Henson, Anupama Reddy, Rebecca H. Buckley, and Patricia L. Lugar. "A study of cancer incidence in the most common immunodeficiency, common variable immunodeficiency (CVID)." Journal of Allergy and Clinical Immunology 143, no. 2 (2019): AB79. http://dx.doi.org/10.1016/j.jaci.2018.12.245.

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Wehr, Claudia, Teemu Kivioja, Christian Schmitt, et al. "The EUROclass trial: defining subgroups in common variable immunodeficiency." Blood 111, no. 1 (2008): 77–85. http://dx.doi.org/10.1182/blood-2007-06-091744.

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The heterogeneity of common variable immunodeficiency (CVID) calls for a classification addressing pathogenic mechanisms as well as clinical relevance. This European multicenter trial was initiated to develop a consensus of 2 existing classification schemes based on flowcytometric B-cell phenotyping and the clinical course. The clinical evaluation of 303 patients with the established diagnosis of CVID demonstrated a significant coincidence of granulomatous disease, autoimmune cytopenia, and splenomegaly. Phenotyping of B-cell subpopulations confirmed a severe reduction of switched memory B cel

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Alkan, Gülsüm, Sevgi Keles, and İsmail Reisli. "Evaluation of Clinical and Immunological Characteristics of Children with Common Variable Immunodeficiency." International Journal of Pediatrics 2018 (2018): 1–8. http://dx.doi.org/10.1155/2018/3527480.

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Background. Common variable immunodeficiency (CVID) is a primary immunodeficiency disorder (PID) that typically presents with hypogammaglobulinemia and impaired antibody production. Objectives. This study aimed to promote the awareness of CVID, whose clinical spectrum is quite broad. Methods. The demographic, clinical, and laboratory characteristics of 12 children (seven males and five females) with CVID were analyzed retrospectively. The patients were diagnosed using the diagnostic criteria of the European Society for Primary Immunodeficiencies. Results. The median disease onset age was 7.2±4

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Wheat, William H., Carlyne D. Cool, Yoshikazu Morimoto, et al. "Possible role of human herpesvirus 8 in the lymphoproliferative disorders in common variable immunodeficiency." Journal of Experimental Medicine 202, no. 4 (2005): 479–84. http://dx.doi.org/10.1084/jem.20050381.

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Patients who have common variable immunodeficiency (CVID) and granulomatous/lymphocytic interstitial lung disease (GLILD) are at high risk for early mortality and B cell lymphomas. Infection with human herpes virus type 8 (HHV8), a B cell lymphotrophic virus, is linked to lymphoproliferative disorders in people who have secondary immunodeficiencies. Therefore, we determined the prevalence of HHV8 infection in CVID patients with GLILD. Genomic DNA isolated from peripheral blood mononuclear cells was screened by nested- and real time-quantitative PCR (QRT-PCR) for the presence of HHV8 genome. It

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Gullo, Irene, Catarina Costa, Susana L. Silva, et al. "The Dysfunctional Immune System in Common Variable Immunodeficiency Increases the Susceptibility to Gastric Cancer." Cells 9, no. 6 (2020): 1498. http://dx.doi.org/10.3390/cells9061498.

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Gastric carcinoma (GC) represents the most common cause of death in patients with common variable immunodeficiency (CVID). However, a limited number of cases have been characterised so far. In this study, we analysed the clinical features, bacterial/viral infections, detailed morphology and immune microenvironment of nine CVID patients with GC. The study of the immune microenvironment included automated digital counts of CD20+, CD4+, CD8+, FOXP3+, GATA3+ and CD138+ immune cells, as well as the evaluation of PD-L1 expression. Twenty-one GCs from non-CVID patients were used as a control group. G

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Amer, R., G. Bamonte, and J. V. Forrester. "Resolution of Juvenile Idiopathic Arthritis-Associated Uveitis after Development of Common Variable Immunodeficiency." European Journal of Ophthalmology 17, no. 4 (2007): 666–68. http://dx.doi.org/10.1177/112067210701700429.

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Purpose To describe the occurrence of common variable immunodeficiency (CVID) in a patient with juvenile idiopathic arthritis (JIA) and JIA-associated uveitis. Methods/Results Case report. A 29-year-old woman was followed-up since the age of 10 years because of right eye JIA-associated recurrent anterior uveitis. She was treated with steroids and immunosuppressants with good control of uveitis and arthritis. At the age of 17 years, she did not experience any further relapse of uveitis or arthritis and both diseases were considered to be in remission. Concomitantly, she started to have recurren

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