Academic literature on the topic 'Complexe immun circulant'

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Journal articles on the topic "Complexe immun circulant"

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Nicaise-Roland, Pascale. "Complexes immuns circulants." EMC - Biologie Médicale 1, no. 1 (2006): 1–2. http://dx.doi.org/10.1016/s2211-9698(06)76223-9.

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Yang, Hui, Jing Guan, Pei Ma, et al. "Beneficial Effects of Qingzixiaoban Granule on Henoch–Schönlein Purpura Nephritis Mice through Inhibiting Immune Complex Deposition and Th2 Immunodeviation." Evidence-Based Complementary and Alternative Medicine 2019 (October 16, 2019): 1–14. http://dx.doi.org/10.1155/2019/3050248.

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Background. Henoch–Schönlein purpura nephritis (HSPN) is the principal cause of morbidity and mortality in Henoch–Schönlein purpura (HSP). However, there is no absolute consensus for the best management of severe HSPN till now. Qingzixiaoban Granule (QZXB GR), a traditional Chinese medicine formula, has been applied to treat HSP in clinical in China. However, the therapeutic effects and potential mechanism of QZXB GR on HSPN is still unknown. Methods. A Gliadin plus Indian Ink-induced HSPN mice model was established. Renal histopathologic changes and the subcutaneous hemorrhage on left legs we
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Mihailov, CI, A. Lamour, B. Bendaoud, et al. "Signification diagnostique au cours des connectivities de l'isotype des complexes immuns circulants." La Revue de Médecine Interne 13, no. 7 (1992): S326. http://dx.doi.org/10.1016/s0248-8663(05)80913-0.

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Ghiran, Ionita, Aleksandra M. Glodek, Gregory Weaver, Lloyd B. Klickstein, and Anne Nicholson-Weller. "Ligation of erythrocyte CR1 induces its clustering in complex with scaffolding protein FAP-1." Blood 112, no. 8 (2008): 3465–73. http://dx.doi.org/10.1182/blood-2008-04-151845.

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Abstract The primary identified function of complement receptor 1 (CR1/CD35) on primate erythrocytes is to bind complement-tagged inflammatory particles including microbes and immune complexes. When erythrocytes circulate through liver and spleen, sinusoidal phagocytes remove CR1-adherent particles and erythrocytes return to the circulation. This process of immune adherence clearance is important for host defense and prevention of autoimmunity. CR1 was previously described as clustered in the human erythrocyte membrane, which was thought to be necessary for binding complement-opsonized particl
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Candolfi, E., B. Molet, and T. Kien. "L'antigénémie et les complexes immuns circulants spécifiques dans l'échinococcose hydatique humaine. A propos de 16 cas." Médecine et Maladies Infectieuses 16, no. 5 (1986): 369–73. http://dx.doi.org/10.1016/s0399-077x(86)80038-5.

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Bentley-Hibbert, Stuart I., Xin Quan, Thomas Newman, Kris Huygen, and Henry P. Godfrey. "Pathophysiology of Antigen 85 in Patients with Active Tuberculosis: Antigen 85 Circulates as Complexes with Fibronectin and Immunoglobulin G." Infection and Immunity 67, no. 2 (1999): 581–88. http://dx.doi.org/10.1128/iai.67.2.581-588.1999.

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ABSTRACT Antigen 85 (Ag85) complex proteins are major secretory products ofMycobacterium tuberculosis and induce strong cellular and humoral immune responses in infected experimental animals and human beings. We have previously shown that nanogram doses of these 30- to 32-kDa fibronectin-binding proteins inhibit local expression of delayed hypersensitivity by a T-cell fibronectin-dependent mechanism. Circulating levels of Ag85 might be expected to be elevated in patients with active tuberculosis and possibly to play a role in systemic anergy in these patients. To test this hypothesis, Ag85 was
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Halat, Monika, Magdalena Klimek-Chodacka, Jagoda Orleanska, Malgorzata Baranska, and Rafal Baranski. "Electronic Circular Dichroism of the Cas9 Protein and gRNA:Cas9 Ribonucleoprotein Complex." International Journal of Molecular Sciences 22, no. 6 (2021): 2937. http://dx.doi.org/10.3390/ijms22062937.

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The Streptococcus pyogenes Cas9 protein (SpCas9), a component of CRISPR-based immune system in microbes, has become commonly utilized for genome editing. This nuclease forms a ribonucleoprotein (RNP) complex with guide RNA (gRNA) which induces Cas9 structural changes and triggers its cleavage activity. Here, electronic circular dichroism (ECD) spectroscopy was used to confirm the RNP formation and to determine its individual components. The ECD spectra had characteristic features differentiating Cas9 and gRNA, the former showed a negative/positive profile with maxima located at 221, 209 and 19
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Tsai, Chang-Youh, Chieh-Yu Shen, Chih-Wei Liu, et al. "Aberrant Non-Coding RNA Expression in Patients with Systemic Lupus Erythematosus: Consequences for Immune Dysfunctions and Tissue Damage." Biomolecules 10, no. 12 (2020): 1641. http://dx.doi.org/10.3390/biom10121641.

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Systemic lupus erythematosus (SLE) is a complex systemic autoimmune disease with heterogeneous clinical manifestations. A diverse innate and adaptive immune dysregulation is involved in the immunopathogenesis of SLE. The dysregulation of immune-related cells may derive from the intricate interactions among genetic, epigenetic, environmental, and immunological factors. Of these contributing factors, non-coding RNAs (ncRNAs), including microRNAs (miRNAs, miRs), and long non-coding RNAs (lncRNAs) play critical roles in the post-transcriptional mRNA expression of cytokines, chemokines, and growth
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Nourmohammad, Armita, Jakub Otwinowski, Marta Łuksza, Thierry Mora, and Aleksandra M. Walczak. "Fierce Selection and Interference in B-Cell Repertoire Response to Chronic HIV-1." Molecular Biology and Evolution 36, no. 10 (2019): 2184–94. http://dx.doi.org/10.1093/molbev/msz143.

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Abstract During chronic infection, HIV-1 engages in a rapid coevolutionary arms race with the host’s adaptive immune system. While it is clear that HIV exerts strong selection on the adaptive immune system, the characteristics of the somatic evolution that shape the immune response are still unknown. Traditional population genetics methods fail to distinguish chronic immune response from healthy repertoire evolution. Here, we infer the evolutionary modes of B-cell repertoires and identify complex dynamics with a constant production of better B-cell receptor (BCR) mutants that compete, maintain
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Serebryanaya, N. B., P. P. Yakutseni, and N. N. Klimko. "Platelets in invasive aspergillosis: role in pathogenesis and immune defense." Journal Infectology 11, no. 2 (2019): 26–34. http://dx.doi.org/10.22625/2072-6732-2019-11-2-26-34.

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Invasive aspergillosis (IA) is a serious disease, with mortality rate up to 80%. A. fumigatus is an angiovasive pathogen, fragments of its hyphae can detach and circulate in the bloodstream. Platelets are activated by surface structures, metabolites and soluble fungal complexes, resulting in adhesion to conidia and fungal hyphae. The melanin and hydrophobin contained in the conidia, as well as the galactosaminogalactan contained in the hyphae and the glyphotoxin secreted by the hyphae, suppress phagocytic cells, but activate the platelets. Activated platelets show direct antifungal activity by
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Dissertations / Theses on the topic "Complexe immun circulant"

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STAMM, DIDIER. "Pneumopathies interstitielles du syndrome tardif de la rubeole congenitale : evolution du taux des complexes immuns circulants : a propos de 2 observations." Lyon 1, 1988. http://www.theses.fr/1988LYO1M166.

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Conference papers on the topic "Complexe immun circulant"

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Nachman, R. L., R. L. Silverstein, and A. S. Asch. "THROMBOSPONDIN: CELL BIOLOGY OF AN ADHESIVE GLYCOPROTEIN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644653.

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Thrombospondin (TSP), a multifunctional 450 KD glycoprotein is a secretory product of thrombin stimulated platelets. It is a major component of the platelets alpha granule constituting approximately 3% of total platelet protein. Thrombospondin does not circulate in appreciable concentrations ∽0 100 ng/ml); however, the tissue distribution is broad. In addition to its expression on the membrane of activated platelets, the protein is synthesized by fibroblasts endothelial cells, glial cell smooth muscle cells alveolar pneumocytes mononuclear phagocytes and various tumor cells. TSP is a major con
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