Relevant bibliographies by topics / Compressive neuropathy

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  2. Dissertations / Theses
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  5. Conference papers

Academic literature on the topic 'Compressive neuropathy'

Author: Grafiati
Published: 4 June 2021
Last updated: 16 February 2022

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Journal articles on the topic "Compressive neuropathy"

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Meyer, Dale R. "Compressive Optic Neuropathy." Ophthalmology 114, no. 1 (January 2007): 199. http://dx.doi.org/10.1016/j.ophtha.2006.06.025.

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Chen, Szu-Han, Chia-Ching Wu, Sheng-Che Lin, Wan-Ling Tseng, Tzu-Chieh Huang, Anjali Yadav, Fu-I. Lu, Ya-Hsin Liu, Shau-Ping Lin, and Yuan-Yu Hsueh. "Investigation of Neuropathology after Nerve Release in Chronic Constriction Injury of Rat Sciatic Nerve." International Journal of Molecular Sciences 22, no. 9 (April 29, 2021): 4746. http://dx.doi.org/10.3390/ijms22094746.

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Peripheral compressive neuropathy causes significant neuropathic pain, muscle weakness and prolong neuroinflammation. Surgical decompression remains the gold standard of treatment but the outcome is suboptimal with a high recurrence rate. From mechanical compression to chemical propagation of the local inflammatory signals, little is known about the distinct neuropathologic patterns and the genetic signatures after nerve decompression. In this study, controllable mechanical constriction forces over rat sciatic nerve induces irreversible sensorimotor dysfunction with sustained local neuroinflammation, even 4 weeks after nerve release. Significant gene upregulations are found in the dorsal root ganglia, regarding inflammatory, proapoptotic and neuropathic pain signals. Genetic profiling of neuroinflammation at the local injured nerve reveals persistent upregulation of multiple genes involving oxysterol metabolism, neuronal apoptosis, and proliferation after nerve release. Further validation of the independent roles of each signal pathway will contribute to molecular therapies for compressive neuropathy in the future.
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Ziaei, Mohammed M., and Hadi Ziaei. "Compressive Optic Neuropathy Caused by Orbital Non-Hodgkin's Lymphoma." Case Reports in Ophthalmological Medicine 2012 (2012): 1–3. http://dx.doi.org/10.1155/2012/894062.

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Purpose. To present a unique case of Non-Hodgkin’s-Lymphoma- (NHL) associated compressive optic neuropathy.Method. An 89-year-old male presenting with acute unilateral visual loss and headache.Results. Patient was initially diagnosed with occult giant cell arteritis; however after visual acuity deteriorated despite normal inflammatory markers, an urgent MRI scan revealed an extensive paranasal sinus mass compressing the optic nerve.Conclusion. Paranasal sinus malignancies occasionally present to the ophthalmologist with signs of optic nerve compression and must be included in the differential diagnosis of acute visual loss.
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Barrett, Stephen L., A. Lee Dellon, John Fleischli, John S. Gould, and Charles Wang. "Metabolic and Compressive Neuropathy." Foot & Ankle Specialist 3, no. 3 (May 27, 2010): 132–39. http://dx.doi.org/10.1177/1938640010368028.

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Santosa, Katherine B., Kevin C. Chung, and Jennifer F. Waljee. "Complications of Compressive Neuropathy." Hand Clinics 31, no. 2 (May 2015): 139–49. http://dx.doi.org/10.1016/j.hcl.2015.01.012.

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Russell, Stephen M., Joshua Marcus, and David Levine. "PATHOGENESIS OF RAPIDLY REVERSIBLE COMPRESSIVE NEUROPATHY." Neurosurgery 65, suppl_4 (October 1, 2009): A174—A180. http://dx.doi.org/10.1227/01.neu.0000335641.17914.4c.

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Abstract OBJECTIVE Using the sequential inflation of 2 sphygmomanometers, Lewis et al. (Heart 16:1–32, 1931) concluded that compressive neuropathy was secondary to ischemia of the compressed nerve segment. Despite subsequent animal studies demonstrating that compressive lesions are more likely the result of mechanical nerve deformation, disagreement remains as to the etiology of rapidly reversible compressive neuropathy. Our hypothesis is that, during the classic sphygmomanometer experiments, the areas of nerve compression at the cuff margins overlapped, so that a region of transient nerve deformation persisted during the second cuff inflation. If true, the original results by Lewis et al. would be consistent with a mechanical pathogenesis. METHODS In our study, 6 patients underwent sequential upper extremity dual-sphygmomanometer inflation with serial assessment by grip-dynamometer and 2-point discrimination. The order of cuff inflation, as well as the distance between cuffs, was varied. Mean grip force and 2-point discrimination values were statistically compared between conditions. RESULTS Patients with overlapping cuffs maintained their neurological deficits, whereas those with separated cuffs experienced an improvement in both grip force (P = 0.02) and 2-point discrimination (P < 0.001) when cuff inflation was switched. CONCLUSION Rapidly reversible compressive neuropathy seems to be secondary to mechanical nerve deformation at the margins of the compressive force rather than the result of ischemia of the compressed nerve segment. Overlap of the mechanically deformed nerve segments likely explains why neurological deficits persisted despite sequential cuff inflation in the classic experiments by Lewis et al.
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Lally, Erin, Ann P. Murchison, Mark L. Moster, and Jurij R. Bilyk. "Compressive Optic Neuropathy From Neurosarcoidosis." Ophthalmic Plastic and Reconstructive Surgery 31, no. 3 (2015): e79. http://dx.doi.org/10.1097/iop.0000000000000342.

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CROWLEY, B., C. R. GSCHWIND, and C. STOREY. "Selective Motor Neuropathy of the Median Nerve Caused by a Ganglion in the Carpal Tunnel." Journal of Hand Surgery 23, no. 5 (October 1998): 611–12. http://dx.doi.org/10.1016/s0266-7681(98)80013-2.

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Carpal tunnel syndrome is the commonest peripheral compressive neuropathy. Typically, sensory symptoms predominate at presentation with motor dysfunction seen in more chronic cases. Isolated motor compression is rare. We present a case of selective median nerve motor neuropathy caused by a carpal tunnel ganglion.
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Wu, Wencan, Michelle T. Sun, Paul S. Cannon, Shi Jianbo, and Dinesh Selva. "Recovery of Visual Function in a Patient with an Onodi Cell Mucocele Compressive Optic Neuropathy Who Had a 5-Week Interval between Onset and Surgical Intervention: A Case Report." Journal of Ophthalmology 2010 (2010): 1–3. http://dx.doi.org/10.1155/2010/483056.

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Purpose. To report on a patient with compressive optic neuropathy secondary to an Onodi cell mucocele, who fully recovered visual function following surgery.Method. Case report.Results. A 28-year-old male was admitted with a right visual acuity of 20/100 following treatment for an initial diagnosis of optic neuritis. Subsequent examination suggested compressive optic neuropathy, and neuroimaging confirmed the presence of an Onodi mucocele compressing the optic nerve. The patient underwent a right endonasal sphenoethmoidectomy with decompression 5 weeks after the initial onset of symptoms. Three weeks following surgery, the visual acuity was 20/20, and there was complete resolution of the visual field defect, which has remained stable at 1 year.Conclusion. Onodi cell mucocele should be included in the differential diagnosis of a young patient with compressive optic neuropathy. Surgical decompression should be considered even when symptoms have been present for over a month.
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Nodera, Hiroyuki, and Ryuji Kaji. "Ulnar neuropathy at the elbow: not simply a compressive neuropathy?" Clinical Neurophysiology 122, no. 1 (January 2011): 1–2. http://dx.doi.org/10.1016/j.clinph.2010.06.001.

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Dissertations / Theses on the topic "Compressive neuropathy"

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BERNADINO, Silvya Nery. "Avaliação dos Limiares Dolorosos por Algometria de Pressão na Síndrome do Túnel do Carpo." Universidade Federal de Pernambuco, 2015. https://repositorio.ufpe.br/handle/123456789/16465.

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INTRODUÇÃO: A avaliação dos limiares dolorosos em pacientes com neuropatias compressivas é de grande utilidade quando se tenta explicar a presença de sintomas generalizados. Sabe-se que no processo de sensibilização periférica a liberação de prostaglandina e bradicinina altera receptores específicos TRPV1 levando a redução do limiar de disparo da fibra nervosa. Essa repetição leva a uma ampliação no campo receptivo cerebral com consequente sensibilização central. A síndrome do túnel do carpo (STC) é a mononeuropatia mais frequentemente diagnosticada, porém os estudos de limiares dolorosos são escassos e não avaliam segundo o grau de comprometimento do nervo. MÉTODOS: Foram avaliadas 160 mulheres divididas em Grupo A) Controle (n=40) e grupo B) pacientes com quadro clínico de síndrome do túnel do carpo (n=120) subdivididas de acordo com o grau de comprometimento neurofisiológico do nervo mediano no punho seguindo a classificação de Pádua em Grupo I (n=20): pacientes com sintomas sugestivos, porém sem confirmação da neuropatia do nervo mediano no punho (NNMP); Grupo II (n=20): NNMP discreta; Grupo III (n=20): NNMP leve; Grupo IV (n= 20): NNMP moderada; Grupo V (n=20): NNMP acentuada; Grupo VI (n=20): NNMP extrema. Realizaram-se algometria de pressão, estudo de condução nervosa, sensibilidade discriminatória entre dois pontos e foi aplicado o questionário de gravidade de sintomas de Boston. A algometria foi realizada em território inervado pelo nervo mediano após a passagem através do túnel do carpo, na região inervada pelo nervo cutâneo palmar, em território inervado pelo nervo ulnar e em áreas proximais ao túnel do carpo. Os valores algométricos em um mesmo grupo seguiram padrão de normalidade (Kolmogorov-Smirnov p <0.05). As comparações entre os grupos foram não-paramétricos (Shapiro- Wilk p= 0.1955) e, portanto, o teste de Kruskal-Wallis foi utilizado e o poshoc de Dunn quando houve diferença significativa. RESULTADOS: Houve diferença extremamente significativa entre o grupo controle e os grupos com NNMP discreta, NNMP leve, NNMP moderada e NNMP acentuada (p<0,0001). Porém, não houve diferença significativa em alguns pontos tanto na presença dos sintomas sem NNMP como na NNMP extrema. Portanto, os limiares reduzem à medida que a patologia se inicia e progride até certo ponto. Quando já não são mais obtidos potenciais sensitivos ou motores nos estudos de condução nervosa, os limiares dolorosos retornam aos valores próximos da normalidade. Sugere-se esse resultado à provável destruição de fibras finas, na qual já pode haver hipoestesia em substituição da hiperalgesia. Quanto à sensibilidade discriminatória entre dois pontos foi observado que quanto mais acentuada a NNMP maiores os valores da sensibilidade discriminatória, havendo diferença significativa entre o controle e os grupos III, IV, V e VI (p<0,0001). Quanto à escala de gravidade de sintomas, as queixas relacionadas à dor e dormência ou formigamento foram mais evidentes nos grupos I, III, IV e V. Enquanto a incapacidade de realizar atividades cotidianas predominou nos grupos III, IV e V. CONCLUSÃO: O limiar para sensação dolorosa é menor em pacientes com síndrome do túnel do carpo, tanto em território inervado pelo nervo mediano como em outras áreas.
INTRODUCTION: Pain threshold evaluation in compressive neuropathy is very useful for explain generalized symptoms. About peripheral sensitization process in the release of bradykinin and prostaglandin alters specific TRPV1 receptors leading to reduction of nerve fiber firing threshold. This repetition leads to a brain receptive field expansion with consequent central sensitization. Carpal tunnel syndrome (CTS) is the most frequently diagnosed mononeuropathy, but painful thresholds studies are scarce and not evaluate according to nerve impairment degree. METHODS: We have evaluated 160 female divided into Group A) Control (n = 40), B) patients with symptoms suggestive of CTS (n = 120) subdivided according to the neurophysiologic impairment of median nerve at the wrist according to Padua classification into Group I (n = 20): patients with symptoms suggestive, but without confirmated wrist median nerve neuropathy (WMNN); Group II (n = 20): Discrete MNNW; Group III (n = 20): Light MNNW; Group IV (n = 20): Moderate MNNW; Group V (n = 20): Severe MNNW; Group VI (n = 20): Extreme MNNW. METHODS: Pressure algometry was held in 320 hands, as well as nerve conduction study and discriminatory sensitivity between two points. The Boston symptom severity questionnaire were applied to the patients. The algometry was held on areas innervated by the median nerve after crossing through the carpal tunnel, palmar cutaneous territory, ulnar cutaneous territory and proximal areas to the carpal tunnel. RESULTS: The algometry values within the same group were normal (Kolmogorov-Smirnov p <0.05). Comparisons between groups were non-parametric (Shapiro-Wilk p = 0.1955) and thus, the Kruskal-Wallis test was used and when there the significant difference pairwise comparisons were performed whit the Dunn test (poshoc). RESULTS: The comparison of algometry data showed extremely significant differences between control group and groups with discrete MNNW, light MNNW, moderate MNNW and severe MNNW (p <0.0001). However, there was no significant difference at some points both in the presence of symptoms without MNNW as in MNNW extreme. Therefore, pain thresholds showed direct relation to MNNW severity to a certain point. When sensory or motor potential were no longer obtained, painful thresholds returned close to normal values. We suggest this result could be due to small fibers destruction, when hyperalgesia would be replaced by hypoesthesia. As for discriminatory sensitivity between two points has been observed higher values were obtained as most affected by MNNW with a significant difference between control group and III, IV, V and VI groups (p <0.0001). As for the scale of severity of symptoms, the complaints with pain and numbness or tingling were more evident in I, III, IV and V groups. About the inability to perform daily activities predominated in III, IV and V groups. CONCLUSION: Pain threshold is lower in patients with carpal tunnel syndrome, either the median nerve innervated area or another areas.
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Barbosa, Valéria Ribeiro Nogueira. "SÍNDROME DO TÚNEL DO CARPO: Dor e Exame Neurológico." Universidade Estadual da Paraíba, 2003. http://tede.bc.uepb.edu.br/tede/jspui/handle/tede/2118.

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Carpal Tunnel Syndrome (CTS) represents the most common entrapment neuropathy, better defined and more studied in the human being. The diagnosis is commonly presumed in patients with painful syndrome in the upper limbs, whose symptoms aggravate at night. The gold-standard for the diagnosis is the occurring of alterations in the sensitive and muscled conduction of the median nerve. Despite the CTS being well clinically characterized, when it is typical, a lot of painful factors in the upper limbs are not caused by CTS, and these patients neuralgic exam may vary from normal to serious alterations. This paper has as objectives: 1. To evaluate the profile of the painful symptoms that may presumably occur in patients with idiopatic CTS or without CTS; 2. to evaluate the profile of the neuralgic exam in patients with idiopatic CTS. Between April and December of 2002, 35 patients with idiopatic CTS (34 women and one man) with diagnosis confirmed by the clinical and electrophysiological exam were examined. They were paired according to their age and sex with 35 citzens of the general population (34 women and one man, aged between 34 and 72, average 51, +/- 9,7 years old). The frequency of paintful syndromes distribution in the two groups studied was analysed by the square test. The age average in both groups of patients was compared by the t-Student test. There was not statistical difference concerning the ages. In the group with CTS the complaints of pain were prevalent in the neck (45,7%), Phalen`s test (68,5%), and fist compression (74,3%) are common, being the last two ones prevalent ( α = 0,05). The severity of the CTS was evaluated by the eletrophysiological exam. Most of the CTS cases are of light degree and occur bilaterally. Just seven patients have unilateral CTS. Concluding: 1- One must cogitate the CTS diagnosis in every case of pain of obscure origin in the lower limbs, being the location either proximal or distal; 2- Just one of patients with CTS had, clearly, signs of cervical radicular injury. One cannot establish etiological relation between these two conditions. What is told about the existence of double-crush syndrome as a nosological entity; 3- In the neurogical exam, the alteration in the sensibility to pain was the most observed sign. The discriminatitive sensibility test seems not to have value to support the CTS diagnosis; 4- the Phalen and the carpal compression tests are more useful to the CTS diagnosis than the Tinel sign, for they are more prevalent.
A Síndrome do Túnel do Carpo (STC) representa a neuropatia compressiva mais comum, melhor definida e mais estudada no ser humano. O diagnóstico é comumente presumido em pacientes com síndrome dolorosa nos membros superiores, cujos sintomas se agravam à noite. O padrão-ouro para o diagnóstico é a ocorrência de alterações na condução sensitiva e motora do nervo mediano. Apesar da STC ser bem caracterizada clinicamente, quando é típica, muitos quadros dolorosos nos membros superiores não são causados por STC, e o exame neurológico desses pacientes pode variar de normal a alterações graves. Este trabalho tem como objetivos: 1- avaliar o perfil dos sintomas dolorosos que presumivelmente possam ocorrer em pacientes com STC idiopático e sem STC; 2- avaliar o perfil do exame neurológico em pacientes com STC idiopático. Entre abril e dezembro de 2002, 35 pacientes com STC idiopático (34 mulheres e um homem, idades entre 34 e 72 anos, média 51, + 9,8 anos) com diagnóstico confirmado pelo exame clínico e eletrofisiológico foram examinados. Eles foram pareados por idade e sexo com 35 sujeitos da população geral (34 mulheres e um homem, idades entre 34 e 72 anos, média 51, + 9,7 anos). A freqüência de distribuição de síndromes dolorosas nos dois grupos estudados foi analisada pelo teste do qui-quadrado. A média de idade dos dois grupos de pacientes foi comparada pelo teste t de Student. Não houve diferença estatística quanto às idades. No grupo com STC as queixas de dor são prevalentes no pescoço (42,8%), membros superiores (36,8%) e mãos (82,8%). Nos sujeitos sem STC a localização do quadro doloroso predomina na cabeça (11,4%), região axial do corpo (37,1%) e membros inferiores (22,8%), (α = 0,05). Entre os sujeitos com STC, 85,7% apresentam queixas de parestesias nos membros superiores e 74,2% destes apenas nas mãos. Os chamados testes provocativos: sinal de Tinel (45,7%), teste de Phalen (68,5%) e compressão do punho (74,3%) são comuns, sendo os dois últimos prevalentes (α = 0,05). A gravidade da STC foi avaliada pelo exame eletrofisiológico. A maioria dos casos de STC são de grau leve e ocorrem bilateralmente. Apenas sete pacientes têm STC unilateral. Em conclusão: 1 - Deve-se cogitar o diagnóstico de STC em todos os casos de dor de origem obscura nos membros superiores, seja de localização proximal ou distal; 2 - apenas uma das pacientes com STC teve, claramente, sinais de lesão radicular cervical. Não se pôde estabelecer relação etiológica entre as duas condições. O que fala contra a existência de síndrome da dupla compressão como uma entidade nosológica; 3 no exame neurológico, a alteração da sensibilidade à dor foi o sinal mais observado. O teste da sensibilidade discriminativa parece não ter valor para suportar o diagnóstico de STC; 4 os testes de Phalen e da compressão carpal são mais úteis para o diagnóstico de STC do que o sinal de Tinel, pois são mais prevalentes.
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Lyons, Danielle N. "ATTENUATING TRIGEMINAL NEUROPATHIC PAIN BY REPURPOSING PIOGLITAZONE AND D-CYCLOSERINE IN THE NOVEL TRIGEMINAL INFLAMMATORY COMPRESSION MOUSE MODEL." UKnowledge, 2014. http://uknowledge.uky.edu/physiology_etds/19.

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Approximately 22% of the United States population suffers from a chronic orofacial pain condition. One such condition is known as trigeminal neuropathic pain frequently reported as continuous aching and burning pain, often accompanied by intermittent electrical shock-like sensations. Dental procedures or trauma are known causes of peripheral trigeminal nerve injury and inflammation. Patients who have this type of facial pain also suffer from emotional distress. For these reasons, trigeminal neuropathic pain needs to be studied in more detail to improve the understanding of the etiology and maintenance of this condition, as well as to develop effective treatment strategies. The first experiment was focused on characterizing the behavioral aspects of the Trigeminal Inflammatory Compression (TIC) mouse model. The findings determined that the TIC injury model induced mechanical and cold hypersensitivity that persist at least 21 weeks. This orofacial, neuropathic pain condition was accompanied by anxiety- and depressive-like behaviors at week 8 post injury. The TIC injury mouse model’s chronicity and development of psychosocial impairments demonstrated its usefulness as a facial pain model. The second experiment used the mouse TIC injury model to test the ability of pioglitazone (PIO), a PPARγ agonist used clinically for treatment of diabetes, on alleviating trigeminal pain. A single low dose of PIO had no effect, but a higher dose attenuated facial pain. The third experiment determined that combining ineffective low doses of PIO and D-cycloserine (DCS) produced a potentiated anti-allodynic response of these drugs and attenuated the anxiety associated with the TIC injury. Ex vivo studies revealed that cortical mitochondrial dysfunction occurred after the TIC injury but could be reversed by the combination of DCS/PIO which improves mitochondrial function. Overall, the present studies determined that the novel mouse TIC injury model is a clinically relevant facial neuropathic pain model. The results suggest that PPARγ and brain mitochondria may represent new molecular targets for the treatment of trigeminal neuropathic pain. These studies support the future “repurposing” of PIO and DCS as well as the combination of the two drugs for this new use in patients with trigeminal neuropathic pain.
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Kouyoumdjian, João Aris. "Análise de fatores pessoais de risco para ocorrência e gravidade de síndrome do túnel do carpo." Faculdade de Medicina de São José do Rio Preto, 2000. http://bdtd.famerp.br/handle/tede/17.

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Between September/98 and May/99, 210 symptomatic carpal tunnel syndrome (CTS) patients were studied. All had bilateral nerve conduction studies and none had been surgically treated. Peripheral neuropathy was excluded. Three groups were defined according to the severity of nerve conduction changes: mild = median distal sensory latency, wrist-index finger, 14 cm (P2), 3.7 to 4.4 ms or sensory median/ulnar difference ³ 0.50 ms or median palm latency ³ 2.3 ms; moderate = P2 ³ 4.5 ms; severe = unrecordable sensory nerve action potential at P2. All latencies were measured to the negative peak. Only the right hand was considered for this study (200 hands), regardless of the electrophysiological findings in the left hand. Another group of 320 subjects without any CTS symptomatology or any known systemic disorders was used as control. The mean age was 50.3 ± 10.8 years for the study group (87.6% female) and 47.3 ± 14.8 years old for the controls (89.1% female). Body mass index (BMI) was 28.4 ± 5.0 for the CTS group and 25.4 ± 4.7 for controls (p < 0.001). Right wrist index (WI) was 0.706 ± 0.041 for the CTS group and 0.689 ± 0.037 for controls (p < 0.001). Logistic regression analysis for risk of having CTS showed an adjusted odds ratio of 1.11 (95% CI 1.07-1.16) per unit increase for BMI and 1.11 (95% CI 1.05-1.16) per 0.01 increase for WI. An ordinal polychotomous logistic regression analysis of the relationship between these factors and the electrophysiological severity of CTS showed proportional odds ratios (mild to severe) of 1.20 (95% CI 1.00-1.30) for 5 years increase in age and 1.10 (95% CI, 1.00-1.20) for 0.01 increase in WI. Higher BMI did not increase the risk of severe CTS. The conclusions were: 1. The variables WI and BMI were higher in CTS cases (p < 0.001). 2. More severe nerve conduction abnormalities were associated with higher age and WI but not with higher BMI.
Foram estudados 210 pacientes com diagnóstico clínico e eletrofisiológico de síndrome do túnel do carpo (STC) entre setembro/1998 e maio/1999. Os casos foram divididos em 3 grupos com progressiva gravidade eletrofisiológica: leve = diferença de latência mediano/ulnar (14 cm, IV dedo) ³ 0,5 ms ou latência palmar mediano (8 cm) ³ 2,3 ms ou latência distal mediano (14 cm) punho-II dedo (P2) entre 3,7 e 4,4 ms; moderado = P2 > 4,4 ms; grave = ausência de potencial de ação sensitivo em P2; todas latências medidas no pico. Todos casos tiveram estudo bilateral, sendo excluídos casos com cirurgia prévia ou evidência de neuropatia periférica. Foram considerados apenas os casos de STC direito independente dos achados do lado oposto (200 mãos). Foi constituído grupo controle (GC) de 320 pessoas sem qualquer sintomatologia compatível com STC ou doenças sistêmicas conhecidas, pareando para sexo e idade. A idade média foi de 50,3 ± 10,8 para STC e 47,3 ± 14,8 anos no GC; sexo feminino 87,6% (STC) e 89,1% (GC); IMC 28,4 ± 5,0 (STC) / 25,4 ± 4,7 (GC) com p < 0,001; IP direito 0,706 ± 0,041 (STC) / 0,689 ± 0,037 (GC) com p < 0,001. Houve incremento do IP e idade com a maior gravidade eletrofisiológica do STC, o mesmo não ocorrendo com IMC. A análise de regressão logística multivariada para avaliar risco de apresentar STC revelou odds ratio ajustado de 1,11 (IC 95% 1,07- 1,16) para IMC (incremento de 1 unidade) e de 1,11 (IC 95% 1,05- 1,16) para IP (incremento de 0,01 unidade). A análise de regressão logística ordinal politômica para avaliar fator de risco de gravidade de condução nervosa em pessoas com STC revelou odds ratio proporcional (casos leve a grave) de 1,20 (IC 95% 1,00-1,30) para incrementos de 5 anos na idade e 1,10 (IC 95% 1,00-1,20) para incrementos de 0,01 no IP. O IMC não determinou risco para desenvolvimento de STC de maior gravidade. Os resultados permitem concluir: 1. As variáveis IP e IMC estão mais elevadas (p < 0,001) no STC; 2. O aumento da gravidade eletrofisiológica do STC está associado ao incremento de idade e IP, porém não ao do IMC.
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Trachter, Robert. "Sensorimotor testing for the early identification of individuals at risk of developing carpal tunnel syndrome." Thesis, 2009. http://hdl.handle.net/1974/5153.

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Carpal tunnel syndrome (CTS) is one of the most common injuries responsible for lost time claims to the Workplace Safety and Insurance Board (WSIB). The main purpose of this study was to determine whether measurable sensorimotor changes exist in asymptomatic individuals who are at risk for CTS such that sensory impairment and/or functional tests may be used in the early detection and intervention to reduce the impact of CTS on individuals, industry and the health care system. Participants were recruited into three strata: (1) individuals diagnosed with mild CTS, (2) asymptomatic individuals who were deemed to be at risk of developing CTS due to exposure to etiological risk factors and (3) asymptomatic individuals who were deemed to be at minimal risk of developing CTS based on non-exposure to risk factors. The main outcome measures included two-point discrimination ability, pressure acuity, vibration sense, Purdue Pegboard Test performance and tracking error and tracking variance on a manual tracking task performed at two different speeds. Seven individuals with CTS, fourteen individuals at risk of developing CTS and nine control individuals with minimal risk participated. The CTS group was significantly different from the at-risk and control groups on the main and work sections of the DASH questionnaire, and the symptom severity scale and functional status scale of the Boston Carpal Tunnel Questionnaire. The only outcome measure that showed a significant difference between the at-risk and the minimal risk group was the assembly task of the Purdue Pegboard Test (p = 0.044), however other measures including median nerve conduction latencies, and manual tracking abilities showed promise that with further recruitment, a significant difference may be seen. The sensory impairment tests did not demonstrate degradation in sensory function in individuals at risk of developing CTS, however analysis of sensory nerve conduction latencies and some aspects of fine motor skills testing did show some promise in their ability to detect individuals at risk of developing CTS. A future prospective study that follows individuals at risk of developing CTS may determine that it is possible to implement a screening tool for the early identification and treatment of CTS.
Thesis (Master, Rehabilitation Science) -- Queen's University, 2009-09-15 12:15:45.208
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Books on the topic "Compressive neuropathy"

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Donaghy, Michael. Focal peripheral neuropathy. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0487.

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Some causes of focal peripheral nerve damage are self-evident, such as involvement at sites of trauma, tissue necrosis, infiltration by tumour, or damage by radiotherapy. Focal compressive and entrapment neuropathies are particularly valuable to identify in civilian practice, since recovery may follow relief of the compression. Leprosy is a common global cause of focal neuropathy, which involves prominent loss of pain sensation with secondary acromutilation, and requires early antibiotic treatment. Mononeuritis multiplex due to vasculitis requires prompt diagnosis and immunosuppressive treatment to limit the severity and extent of peripheral nerve damage. Various other medical conditions, both inherited and acquired, can present with focal neuropathy rather than polyneuropathy, the most common of which are diabetes mellitus and hereditary liability to pressure palsies. A purely motor focal presentation should raise the question of multifocal motor neuropathy with conduction block, which usually responds well to high-dose intravenous immunoglobulin infusions.
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Wilson, Thomas J., and Robert J. Spinner. Peroneal Neuropathy—Fibular Tunnel Syndrome. Edited by Meghan E. Lark, Nasa Fujihara, and Kevin C. Chung. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190617127.003.0009.

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Common peroneal neuropathy is the most common compressive neuropathy of the lower extremity, often presenting with acute, progressive foot drop. The most common site of compression of the peroneal nerve is at the point that it courses around the neck of the fibula beneath the fascia of the peroneus longus. This chapter aims to help the surgeon evaluate for peroneal neuropathy using a combination of clinical history, physical examination, electrodiagnostics, and imaging. The chapter also discusses surgical and nonsurgical management of peroneal neuropathy, including a detailed discussion of the operative technique for decompression of the peroneal nerve at the fibular tunnel.
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Katirji, Bashar. Case 8. Edited by Bashar Katirji. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190603434.003.0012.

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Peroneal (fibular) neuropathy is the most common entrapment/compressive mononeuropathy in the lower extremity, often presenting with foot drop and numbness. The majority of the lesions are across the fibular neck, but more proximal and distal lesions exist. This case presents the clinical and electrodiagnostic findings in peroneal neuropathy and discusses in detail the differential diagnoses of foot drop. It highlights the importance of distinguishing peroneal nerve lesions from L5 radiculopathy, lumbar plexopathy, and sciatic neuropathy. Causes of acute and subacute peroneal mononeuropathies are emphasized.
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Shaibani, Aziz. Numbness. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190661304.003.0023.

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Sensory symptoms are the most common symptoms in neuromuscular clinics, yet it is hard to capture them via video unless they have a very specific pattern and they are associated with objective loss of sensation. Distal sensory loss is a common neuropathic finding. It follows gloves and stocks distribution. Sensory neuropathies may present with ataxia which results in falls, or severe pain. Neuropathic pain with normal ankle reflexes and sural responses suggest small fiber neuropathy. Multifocal sensory loss is usually vascular. It can also be infectious (leprosy). Migratory neuritis is a poorly understood condition. Intercostal pain and numbness is usually due to radiculopathy (diabetic, zoster, or compressive radiculopathy). Foots ulcers and unfelt mosquito bites are markers for sensory loss. Loss of corneal sensation may led to keratitis and blindness.
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Maldonado, Andrés A., and Robert J. Spinner. Suprascapular Neuropathy. Edited by Meghan E. Lark, Nasa Fujihara, and Kevin C. Chung. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190617127.003.0007.

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Suprascapular neuropathy results from a lesion affecting the suprascapular nerve and is typically due to compression or traction in association with other injuries. The differential diagnosis of suprascapular neuropathy includes rotator cuff pathology, cervical radiculopathy, and Parsonage-Turner syndrome. Suprascapular neuropathy leads to a spectrum of clinical symptoms, including pain and selected weakness in shoulder abduction and external rotation of the arm. Atrophy of the shoulder musculature affecting the spinati muscles (supraspinatus and infraspinatus) often becomes apparent after some months. Suprascapular neuropathy is a challenging diagnosis to make based on the history and physical examination alone, especially since shoulder pain is relatively common and multifactorial; in addition, suprascapular neuropathy can often coexist with other shoulder pathology. The indications for a surgical procedure are the failure of nonoperative management, with lack of clinical or electrical improvement in 6 to 9 months, and the presence of a space-occupying lesion causing compression of the suprascapular nerve. Direct nerve decompression by release of the suprascapular ligament is typically recommended in cases of symptomatic isolated suprascapular neuropathy.
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Payne, Russell A., and Kimberly S. Harbaugh. Median Neuropathy—Pronator Teres Syndrome and Anterior Interosseous Neuropathy. Edited by Meghan E. Lark, Nasa Fujihara, and Kevin C. Chung. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190617127.003.0003.

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Pronator teres syndrome results from median nerve compression or irritation at the elbow region. Patients typically note volar forearm pain and median sensory disturbance that includes the palm. Electrodiagnostic studies are helpful in excluding carpal tunnel syndrome and cervical radiculopathy, and findings may be normal in pronator syndrome. A lack of sensory findings and motor loss in flexion of the distal phalanx of the radial three digits suggests anterior interosseous nerve palsy, typically due to neuralgic amyotrophy. When conservative treatment fails, surgical release of all potential points of compression is successful in alleviating symptoms in the majority of patients with pronator syndrome.
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Varma, Abhay K., and Ron Ron Cheng. Ulnar Neuropathy—Guyon’s Canal Syndrome. Edited by Meghan E. Lark, Nasa Fujihara, and Kevin C. Chung. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190617127.003.0005.

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The chapter describes ulnar nerve compression at the wrist secondary to a mass lesion. The clinical picture can mimic pathology of the nerve roots or the brachial plexus or ulnar nerve entrapment at the elbow. Meticulous clinical examination and electrodiagnostic study help to differentiate ulnar nerve compression at the wrist from radiculopathy and to localize the site of compression. Imaging of the wrist is essential, as compression by an organic lesion is common. Conservative management is recommended when organic pathology has been ruled out and there is no motor involvement. Surgery is indicated when conservative treatment fails, there is motor weakness, or a mass lesion has been identified.
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Lyons, Daniel A., and David L. Brown. Tibial Neuropathy—Tarsal Tunnel Syndrome. Edited by Meghan E. Lark, Nasa Fujihara, and Kevin C. Chung. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190617127.003.0010.

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Tarsal tunnel syndrome (TTS) is caused by compression of the tibial nerve and its branches within the tarsal tunnel at the ankle. The diagnosis of TTS is often made clinically, but imaging and electrodiagnostic studies should be considered when the diagnosis cannot be ascertained from the clinical history and physical examination. Surgical decompression of the tarsal tunnels should be pursued only after conservative measures have failed or when a space-occupying lesion or point of tibial nerve compression has been identified. Surgical intervention requires complete release of the flexor retinaculum at the medial ankle, as well as release of the three distinct tunnels enveloping the medial and lateral plantar nerves and the calcaneal branch. Success rates for tibial nerve decompression vary widely in the literature, ranging from 44% to 96%.
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Thaisetthawatkul, Pariwat, and Eric Logigian. Entrapment Neuropathy and Pregnancy. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190667351.003.0027.

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Entrapment neuropathy is caused by compression, angulation, or stretch of a peripheral nerve as it passes through a fibro-osseous canal such as the carpal or the cubital tunnel (in the case of the median or the ulnar nerves). In addition to true entrapment neuropathies, individual nerves can be injured at vulnerable anatomical locations such as the fibular head (in case of the fibular nerve). Pregnancy causes a variety of physiological changes related to reproductive hormone secretion that can affect peripheral nerve. These include weight gain, salt and water retention, edema and hyperglycemia.1 Two entrapment neuropathies that occur commonly in pregnancy are carpal tunnel syndrome and meralgia paresthetica.2 In addition to these true entrapment neuropathies, this chapter addresses other common focal mononeuropathies: femoral, obturator, and fibular neuropathies that may occur as a consequence of obstetrical procedures or of fetal or maternal positioning during delivery or in the postpartum period.
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Cheng, Ron Ron, and Abhay K. Varma. Ulnar Neuropathy—Cubital Tunnel Syndrome. Edited by Meghan E. Lark, Nasa Fujihara, and Kevin C. Chung. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190617127.003.0004.

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The chapter presents the typical scenario of ulnar nerve entrapment at the elbow. The clinical picture can mimic pathology of nerve roots, of the brachial plexus, or of the ulnar nerve at different sites. Electrodiagnostic study helps to differentiate ulnar nerve entrapment from radiculopathy and to localize the site of compression, while imaging (ultrasound and MR imaging) are useful adjuncts to clinical examination. Conservative management is recommended for intermittent symptoms and absence of motor involvement. Surgical procedures include in situ, open, or endoscopic decompression and nerve transposition. Subluxation of the nerve over the medial epicondyle and recurrent or persistent neuropathy after in situ decompression are indications for transposition.
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Book chapters on the topic "Compressive neuropathy"

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Distefano, Alberto G. "Compressive Optic Neuropathy." In Controversies in Neuro-Ophthalmic Management, 97–106. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-74103-7_10.

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Treat, Christopher M., and Christopher C. Schmidt. "Suprascapular Neuropathy." In Compressive Neuropathies of the Upper Extremity, 259–76. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-37289-7_27.

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Monteiro, Mário Luiz Ribeiro. "OCT and Compressive Optic Neuropathy." In OCT and Imaging in Central Nervous System Diseases, 169–94. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-26269-3_10.

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Monteiro, Mário Luiz Ribeiro. "OCT and Compressive Optic Neuropathy." In OCT in Central Nervous System Diseases, 69–86. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-24085-5_4.

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Anagnostakos, Konstantinos, Nikolaos P. Zagoreos, and Nickolaos A. Darlis. "Pronator Teres Syndrome: Anterior Interosseous Nerve Compressive Neuropathy." In Compressive Neuropathies of the Upper Extremity, 209–15. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-37289-7_21.

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Ali, Mohammad Javed. "Endonasal Apical Decompression for Compressive Optic Neuropathy." In Surgery in Thyroid Eye Disease, 95–107. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-32-9220-8_8.

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Wang, An-Guor. "Thyroid Eye Disease with Compressive Optic Neuropathy." In Emergency Neuro-ophthalmology, 167–72. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-7668-8_28.

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Galvez, Michael G., and Jeffrey Yao. "Median Compressive Neuropathy Proximal to the Carpal Tunnel." In Carpal Tunnel Syndrome and Related Median Neuropathies, 271–77. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-57010-5_28.

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Rosa, N., and Giovanni Cennamo. "Compressive optic neuropathy: echographic study in retrobulbar optic neuritis." In Documenta Ophthalmologica Proceedings Series, 571–73. Dordrecht: Springer Netherlands, 1997. http://dx.doi.org/10.1007/978-94-011-5802-2_73.

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Juvan, V. "A case of bilateral chronic myositis with compressive optic neuropathy." In Documenta Ophthalmologica Proceedings Series, 127–31. Dordrecht: Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-011-1846-0_6.

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Conference papers on the topic "Compressive neuropathy"

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Tadepalli, Srinivas C., Ahmet Erdemir, and Peter R. Cavanagh. "A Comparison of the Performance of Hexahedral and Tetrahedral Elements in Finite Element Models of the Foot." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19427.

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Characterization of the contact pressure patterns under the foot provides significant insight into pathological conditions such as diabetic peripheral neuropathy [1]. The finite element method (FEM) is widely used in foot biomechanics for predictive simulations of plantar pressures in barefoot and shod conditions [2–6]. In the analysis of the foot, mesh generation accounts for most of the labor in model development, due to the complex structure of the foot including highly partitioned, embedded, and branching geometries. In FEM, hexahedral elements are preferred over tetrahedral elements because of their superior performance in terms of convergence and accuracy of the solution [7]. This becomes more apparent as the convergence behavior of the simulations are hindered by large deformation, material incompressibility, and contact with friction, mechanical features which are commonly seen in foot biomechanics. Unfortunately, unlike tetrahedral meshing which is highly automated [8], hexahedral mesh generation is a time consuming process requiring considerable operator intervention. Despite their reputed advantages, the relative performance of tetrahedral meshes in foot models has not been well established; to our knowledge, there has not been a comprehensive study comparing the performance of hexahedral and tetrahedral elements when material and geometric nonlinearity are included combined with material incompressibility and shear force loading conditions. Hence, the objective of the present study was to evaluate various types of meshes that can be used to model the interaction of a bone-soft tissue construct and rigid floor complex under compressive and shear loading in a heel-pad analog model.
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Toosi, Kevin K., and Michael L. Boninger. "Wrist Kinematics and Ultrasound Measures of the Median Nerve During Computer Keyboarding." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53296.

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Carpal tunnel syndrome (CTS) is a common, costly problem in the general population and particularly in manual workers [1–3], with as many as 3 million individuals experiencing its symptoms and signs, including pain, tingling, numbness, fatigue and weakness in the hands and fingers [4]. Treatment of CTS is estimated to cost over one billion dollars a year [5]. The most prevalent theory for the pathogenesis of CTS is compression of median nerve in the carpal tunnel [6]. Although this theory is widely accepted, the cause of the compression in the carpal tunnel is not fully understood. Epidemiological research has identified several occupational risk factors associated with the development of CTS in general industry including: force, repetition, awkward/static postures, localized mechanical compression, and vibration [7]. Several studies have found greater prevalence of carpal tunnel syndrome in workers with highly repetitive manual jobs [8]. Keyboarding is a highly repetitive daily task, and its association with musculoskeletal disorders of the upper extremity has been a public health concern since the 1980s [1]. However, there are controversial results regarding the association between computer keyboarding and CTS which indicate that we have an insufficient understanding of an association between keyboarding and upper limb neuropathy. Using ultrasonographic techniques, our laboratory was able to explore acute changes in the median nerve following a one-hour keyboarding task [9].
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PEREIRA, Juliana Fernandes, Luis Carlos PASCHOARELLI, and Fausto Orsi MEDOLA. "AVALIAÇÃO DO USO DE SMARTPHONES NA INCIDÊNCIA DA NEUROPATIA COMPRESSIVA: SÍNDROME DO TUNEL DO CARPO." In 1º Congresso Internacional de Ergonomia Aplicada. São Paulo: Editora Blucher, 2016. http://dx.doi.org/10.5151/engpro-conaerg2016-7016.

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Ohno, Tsuyoshi, Takashi Mine, Hiroki Yoshioka, Mikiko Kosaka, Kazuhiro Matsuda, Maiko de Kerckhove, and Charles de Kerckhove. "Abstract P5-15-13: Restoration by compression therapy of skin blood perfusion levels decreased during breast cancer chemotherapy, alleviating peripheral neuropathy." In Thirty-Seventh Annual CTRC-AACR San Antonio Breast Cancer Symposium; December 9-13, 2014; San Antonio, TX. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.sabcs14-p5-15-13.

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Kida, Yoko, Rikiya Kouketsu, Toshiyasu Sakurai, Yusuke Inui, Tasuku Matsuoka, and Kimihide Tada. "Compression therapy using surgical gloves and elastic stockings to prevent chemotherapy induced peripheral neuropathy (CIPN) in advanced non-small cell lung cancer." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.1746.

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Júnior, Francisco Paulo Dias, Lara Victoria Pinheiro, Nathalia Viviane Araújo Pinheiro, Wandeclebson Ferreira Júnior, and Francisco Anderson Costa Batista Junior. "DESENVOLVIMENTO DE COMPLICAÇÕES NEUROLÓGICAS DECORRENTES DE HANSENÍASE." In II Congresso Brasileiro de Saúde On-line. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/1461.

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Introdução: A Hanseníase é uma patologia infecciosa crônica e não fatal, causada pela bactéria álcool-ácido resistente Mycobacterium Leprae, causa mais comum da neuropatia periférica. As manifestações clínicas mais recorrentes dessa enfermidade restringem-se, basicamente, à pele, ao Sistema Nervoso Periférico e ao trato respiratório superior. Em decorrência do seu impacto inflamatório no nervo periférico (neurite), há uma importante contribuição na disfunção sensorial e motora. Objetivos: Investigar, mediante uma revisão bibliográfica, o surgimento de manifestações clínicas neurológicas em pacientes afetados pela Hanseníase, por lesão no tronco dos nervos periféricos. Material e Métodos: Por intervenção de pesquisas bibliográficas disponíveis na literatura, selecionou-se artigos publicados entre fevereiro de 2011 e maio de 2016, manipulando as bases de dados WEB OF SCIENCE, PUBMED e SCIELO, manuseando os descritores “Hanseníase”, “Neuropatia”, “Complicações neurológicas” e “Doença de Hansen”, em português e inglês, empregando critérios de exclusão, como período de publicação e incompatibilidade de conteúdo, além de parâmetros de inclusão, previamente definidos; selecionando os artigos a serem utilizados. Resultados: A doença de Hansen se desenvolve a partir da infecção pela bactéria M. Leprae, que tem como principal porta de entrada as Vias Aéreas Superiores (VAS), causando manifestações clínicas inflamatórias nos nervos periféricos (ocasionadas pela forma tuberculóide da Hanseníase). A neurite, processo inflamatório do nervo, pode ser crônica ou aguda (hipersensibilidade) e é desencadeada pela bactéria já mencionada. Essa inflamação é decorrente da invasão bacilar, formando edemas importantes, bem como abscesso neural, resultando em uma degeneração e morte nervosa. Ocorre, inicialmente, uma constrição neural, categorizada como intrínseca ou extrínseca, desenvolvendo três estágios, a saber: I (irritativo), com manifestações clínicas de dor, hiperestesia e parestesia; II (compressivo), apresentando, principalmente, parestesia e hipoestesia; e o III (deficitário), caracterizado por anestesia, atrofia e paralisia. Conclusão: As complicações neurológicas decorrentes da Hanseníase são resultantes do comprometimento dos nervos periféricos, que causam manifestações sensitivas e motoras. À luz dessas considerações, o histórico e estigma social podem ser combatidos por meio de diagnóstico precoce e tratamento adequado, podendo ser facilitados, dentre outras coisas, pela Educação Popular em Saúde.
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Kashiwaba, Masahiro, Takahiro Nakayama, Takafumi Sangai, Takashi Morimoto, Hiroyuki Yasojima, Yutaka Yamamoto, Shinji Ohno, and Norikazu Masuda. "Abstract PS9-52: A randomized phase II trial of interventions with frozen groves and compression stockings to prevent nab-paclitaxel induced chemotherapy-induced peripheral neuropathy (SPOT trial)." In Abstracts: 2020 San Antonio Breast Cancer Virtual Symposium; December 8-11, 2020; San Antonio, Texas. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.sabcs20-ps9-52.

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Tsuyuki, S., N. Senda, Y. Kanng, A. Yamaguchi, H. Yoshibayashi, Y. Kikawa, N. Katakami, et al. "Abstract PD4-08: Efficacy of compression therapy using surgical gloves for nanoparticle albumin-bound-paclitaxel-induced peripheral neuropathy: A phase II multicenter study by the Kamigata breast cancer study group." In Abstracts: 2016 San Antonio Breast Cancer Symposium; December 6-10, 2016; San Antonio, Texas. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.sabcs16-pd4-08.

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