Relevant bibliographies by topics / Congenital sensorineural hearing loss

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Congenital sensorineural hearing loss'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Congenital sensorineural hearing loss"

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Kadom, Nadja, and Raymond W. Sze. "Radiological Reasoning: Congenital Sensorineural Hearing Loss." American Journal of Roentgenology 194, no. 3_supplement (March 2010): WS1—WS4. http://dx.doi.org/10.2214/ajr.07.7120.

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Palacios, Enrique, Galdino Valvassori, and Michael D'Antonio. "Congenital Sensorineural Hearing Loss: Mondini's Deformity." Ear, Nose & Throat Journal 80, no. 4 (April 2001): 198. http://dx.doi.org/10.1177/014556130108000405.

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Masi, Roberto. "Sensorineural hearing loss and congenital diaphragmatic hernia." Audiological Medicine 8, no. 1 (January 2010): 24–27. http://dx.doi.org/10.3109/16513861003647035.

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Rathore, M. H. "Sensorineural Hearing Loss After Congenital CMV Infection." AAP Grand Rounds 20, no. 1 (July 1, 2008): 5–6. http://dx.doi.org/10.1542/gr.20-1-5-a.

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Sharifian, Shahriar Mohammad Re, and Rahim Vakilimo. "R048: Sensorineural Hearing Loss in Congenital Hypothyroidism." Otolaryngology–Head and Neck Surgery 135, no. 2_suppl (August 2006): P120. http://dx.doi.org/10.1016/j.otohns.2006.06.800.

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Johansson, Marlin, Filip Asp, and Erik Berninger. "Children With Congenital Unilateral Sensorineural Hearing Loss." Ear and Hearing 41, no. 1 (2020): 55–66. http://dx.doi.org/10.1097/aud.0000000000000730.

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Wiwanitkit, Viroj, and Beuy Joob. "Sensorineural Hearing Loss and Congenital Cytomegalovirus Infection." Journal of Pediatric Genetics 07, no. 01 (December 13, 2017): 045. http://dx.doi.org/10.1055/s-0037-1612599.

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ARAO, Harumi, Reiko BEPPU, and Keiko MURAHASHI. "Sensorineural Hearing Loss in Congenital Cytomegalovirus Infection." Practica Oto-Rhino-Laryngologica 90, no. 4 (1997): 391–98. http://dx.doi.org/10.5631/jibirin.90.391.

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Akisada, Takeshi, Yozo Orita, Tsuyoshi Yoshihiro, Akimitsu Kawai, Yasuhiko Higashikawa, Kanae Okumoto, Takuji Takemoto, Kotaro Take, and Toshimi Hidaka. "Congenital Cytomegalovirus Infection and Sensorineural Hearing Loss." Practica oto-rhino-laryngologica. Suppl. 1998, Supplement96 (1998): 36–43. http://dx.doi.org/10.5631/jibirinsuppl1986.1998.supplement96_36.

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Umrigar, Ayesha, Amanda Musso, Danielle Mercer, Annette Hurley, Cassondra Glausier, Mona Bakeer, Michael Marble, Chindo Hicks, and Fern Tsien. "Delayed diagnosis of a patient with Usher syndrome 1C in a Louisiana Acadian family highlights the necessity of timely genetic testing for the diagnosis and management of congenital hearing loss." SAGE Open Medical Case Reports 5 (January 1, 2017): 2050313X1774590. http://dx.doi.org/10.1177/2050313x17745904.

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Advances in sequencing technologies and increased understanding of the contribution of genetics to congenital sensorineural hearing loss have led to vastly improved outcomes for patients and their families. Next-generation sequencing and diagnostic panels have become increasingly reliable and less expensive for clinical use. Despite these developments, the diagnosis of genetic sensorineural hearing loss still presents challenges for healthcare providers. Inherited sensorineural hearing loss has high levels of genetic heterogeneity and variable expressivity. Additionally, syndromic hearing loss (hearing loss and additional clinical abnormalities) should be distinguished from non-syndromic (hearing loss is the only clinical symptom). Although the diagnosis of genetic sensorineural hearing loss can be challenging, the patient’s family history and ethnicity may provide critical information, as certain genetic mutations are more common in specific ethnic populations. The early identification of the cause of deafness can benefit patients and their families by estimating recurrence risks for future family planning and offering the proper interventions to improve their quality of life. Collaboration between pediatricians, audiologists, otolaryngologists, geneticists, and other specialists are essential in the diagnosis and management of patients with hearing disorders. An early diagnosis is vital for proper management and care, as some clinical manifestations of syndromic sensorineural hearing loss are not apparent at birth and have a delayed age of onset. We present a case of Usher syndrome (congenital deafness and childhood-onset blindness) illustrating the challenges encountered in the diagnosis and management of children presenting with congenital genetic sensorineural hearing loss, along with helpful resources for clinicians and families.
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Dissertations / Theses on the topic "Congenital sensorineural hearing loss"

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Strike, Brian J. "The Clinical Utility of Comprehensive Genetic Testing for Individuals with Congenital Sensorineural Hearing Loss." University of Cincinnati / OhioLINK, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1212164935.

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Cahill, Lisa D. "Cortical responses to speech stimuli in hearing impaired infants measured by fMRI and auditory evoked potentials." University of Cincinnati / OhioLINK, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1273174165.

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Geldenhuys, Wilhelmien. "The relationship between motor proficiency, bilateral vestibular hypofunction and dynamic visual acuity in children with congenital or early acquired sensorineural hearing loss." Master's thesis, University of Cape Town, 2010. http://hdl.handle.net/11427/10505.

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Includes bibliographical references (leaves 105-115).
The functional integrity of the vestibular system in children is not often tested. Due to the close relationship between the cochlea and the peripheral vestibular system, the function of the vestibular system may be impaired in children with sensorineural hearing loss.The aims of this study were to determine the prevalence of impairments of motor performance, vestibular function and dynamic visual acuity, and the nature and extent of interaction between these in children between the ages of four and fourteen years with congenital and early acquired sensorineural hearing loss. Motor performance was evaluated by means of the Movement Assessment Battery for Children-2, dynamic visual acuity was determined by means of the Dynamic Visual Acuity Test, and vestibular function with the Southern California Postrotary Nystagmus Test.
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Silva, Adriana Carnevale da. "Infecção congênita por CMV: potenciais marcadores preditivos de alterações tardias em crianças assintomáticas." Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/17/17144/tde-23032018-104745/.

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Os objetivos do estudo consistiram em verificar o impacto da infecção congênita por CMV ao nascer e avaliar potenciais preditores prognósticos de anormalidades tardias em uma coorte de criança s com esta infecção. Métodos: Por meio de uma triagem neonatal, 66 de 11.957 crianças foram identificadas como portadoras de infecção congênita por CMV. Todas as crianças infectadas sintomáticas e assintomáticas foram avaliadas ao nascimento por meio de exame físico detalhado, avaliação auditiva, fundoscopia ocular, neurossonografia transfontanelar (NTF) realizada por neurorradiologista infantil e quando alterada, foram submetidas à ressonância magnética de encéfalo (RMc). Análise de regressão logística foi realizada para verificar a associação de potenciais fatores de risco para ocorrência de surdez neurossensorial relacionada ao CMV e/ou presença de achados anormais em NTF e/ou RMc. A análise da Curva ROC foi utilizada para avaliar a associação da carga do DNA do CMV expresso em log10, com a presença de sintomas ao nascer, achados anormais em NTF e/ou RMc e ocorrência de surdez neurosensorial. Resultados: Em 66 crianças infectadas, os sinais clínicos sugestivos de infecção congênita ao nascer foram observados em 8/66 (12,12%; IC95%: 5,74-23,03), sendo considerados sintomáticos. As 58 crianças restantes foram considerados assintomáticos. A surdez neurossensorial foi observada em 8/66 (12,12%; IC95%: 5,74-23,03%) das crianças. Destas, 4/58 (6,9%; IC95%: 2,23- 17,54%) e 4/8 (50%; IC95%: 17,44-82,55%) crianças eram assintomáticas e sintomáticas, respectivamente. Achados de NTF e/ou RMc sugestivas de infecção congênita foram observadas em 7 de 8 crianças sintomáticas (87,5%; IC95%: 46,67-99,34%). Dentre as 58 assintomáticas, 53 (92,4%) completaram todas as avaliações e destas 29/52 apresentaram alterações sugestivas de infecção congênita em NTF e/ou RMc (55,7%; IC95%: 41,41 - 69,27%). O achado mais comum foi a vasculopatia lenticuloestriada acompanhado ou não de cistos subpendimários (13/29: 43.3%). Outros achados anormais foram os cistos subpendimários como achado isolado (11/29: 37.9%), calcificação única ou periventricular com ou sem áreas de gliose (4/29: 13,8%) e ventriculomegalia (4/29: 13,8%). Análise de regressão logística mostrou que apenas a presença de sintomas clínicos ao nascer foi preditivo para a ocorrência de surdez neurossensorial. Achados de neuroimagens (NTF e/ou RMc) foram observados em 3 de 4 crianças assintomáticas (75%) enquanto 26 (54,2%) de 48 crianças sem surdez tinham avaliações normais de NTF e/ou RMc (p=0,42; RR:2,38: IC95%: 0,26-21,39). Nenhum dos outros fatores de risco foram independentemente associados com surdez. A presença de plaquetopenia e/ou níveis altos de gama glutamil transferase (?GT) foi associado com a presença de achados anormais em NTF e/ou RMc uma análise univariada. O poder discriminatório pela determinação do ponto de corte do valor da carga do DNA do CMV foi avaliada pela área abaixo da curva ROC (AUC) e não houve associação entre a carga viral e a ocorrência de surdez e/ou achados anormais de NTF e/ou RMc. Conclusões: Embora a triagem neonatal da infecção congênita por CMV permita identificar a maioria das crianças infectadas que são clinicamente assintomáticas ao nascer, uma proporção significante destas crianças poderá ser beneficiada por uma avaliação do sistema nervoso central através de NTF, uma vez que, achados anormais são muito frequentes. Embora não tenha sido possível determinar fatores independentemente preditivos de ocorrência de surdez neurossensorial, os achados de NTF podem ser potenciais fatores preditivos de anormalidades tardias em crianças assintomáticas.
The objectives of this study were to verify the impact of congenital cytomegalovirus (CMV) infection at birth and to evaluate potential prognostic predictors of late abnormalities in a cohort of children with this infection. Methods: By means of a CMV neonatal screening, 66 of 11.957 infants were identified as congenitally infected. Infants with and without clinical abnormalities detectable at birth underwent physical examination, cranial ultrasound performed by a paediatric radiologist and/or cranial magnetic resonance imaging, ocular fundoscopy, and hearing evaluation using evoked otoacoustic emissions and auditory brainstem response. Logistic regression analysis was carried out to verify the association between the risk factors for occurrence of hearing loss related to CMV and/ or abnormal cranial ultrasound findings. ROC curve was plotted using the log10 value of CMV DNA load to evaluate the association between viral load and clinical symptoms at birth, abnormal cranial ultrasound findings and hearing loss. Results: Of all 66 infected children, the clinical signs suggestive of congenital infection at birth were observed in 8/66 (12.12%; IC95%: 5.74- 23.03) symptomatic infants. Sensorineural hearing loss was observed in 8/66 (12,12%; IC95%: 5.74-23.03%) children. Of these, 4/58 (6,9%; IC95%: 2.23-17.54%) and 4/8 (50%; IC95%: 17,44-82.55%) children were asymptomatic and symptomatic, respectively. Cranial ultrasound findings suggestive of congenital infection were observed in 7 of the 8 symptomatic children (87.5%; IC95%: 46.67-99.34%). Among the 58 asymptomatic infants, 53 underwent complete evaluation and 29/52 had abnormal cranial ultrasound results (55,7%; IC95%: 41.41 - 69,27%). The most prevalent findings was lenticulostriate vasculopathy with subependymal pseudocysts present in 13 of the /29 (43.3%) infants with cranial ultrasound. Other abnormal findings were isolated subependymal pseudocysts (11/29: 37.9%); single or periventricular calcifications and/or gliosis (4/29: 13.8%); and ventriculomegaly (4/29: 13,8%). Logistic regression analysis showed that only the presence of clinical findings predicted the occurrence of hearing loss. Cranial ultrasound findings were observed in 3 of asymptomatic infants (75.0%) while 26 (54.2%) of 48 infants with no hearing loss had abnormal imaging features (p=0,42; RR:2,38: IC95%: 0,26-21,39). None of the other factors risk were independely associated with development of hearing loss. The presence of thrombocytopenia and/or high level of gamma-glutamyltranspeptidase (?GT) was associated with cranial ultrasound findings on univariated analysis. No discrimination power was achieved using the area under the ROC curve to verify the association between CMV DNA load in the urine of the infected children and the developing of hearing loss, presence of cranial ultrasound findings and clinical signs at birth. Conclusions: Although a neonatal screening of cCMV will identify the majority of infected infants who are clinically asymptomatic, a significant proportion of them could benefit from a central nervous system image evaluation, since abnormal findings are frequent. Althoug it was not possible to determine risk factors that are independently associated to development of sensorineural hearing loss, cranial ultrasound findings could be a potential prognostic markers of adverse outcomes of congenital CMV in asymptomatic infants.
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Seeman, Scott E. "Informational Masking and Sensorineural Hearing Loss." The Ohio State University, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=osu1250704738.

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Nakashima, T., T. Yoshida, S. Nakata, M. Teranishi, I. M. Ishida, S. Naganawa, and M. Sugiura. "Vestibular aqueduct in sudden sensorineural hearing loss." Cambridge University Press, 2008. http://hdl.handle.net/2237/14312.

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Pizarro, Luzia Maria Pozzobom Ventura. "Maturação cortical e habilidades auditivas em usuários experientes de Vibrant Soundbridge: estudo eletrofisiológico e comportamental." Universidade de São Paulo, 2018. http://www.teses.usp.br/teses/disponiveis/5/5143/tde-27092018-084134/.

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Introdução: A atresia congênita de orelha constitui uma deformidade presente ao nascimento, de prevalência unilateral, decorrente da alteração no desenvolvimento das estruturas das orelhas externa e média. Geralmente, provoca perda auditiva condutiva, e pode ser acompanhada por componente sensorioneural. Dentre as formas de tratamento disponíveis, encontra-se o implante de orelha média Vibrant Soundbridge (VSB), que tem se mostrado eficaz no tratamento deste tipo de alteração. A literatura mostra melhora nos limiares tonais e nos resultados dos testes de percepção auditiva da fala, realizados com o uso do processador de fala após a cirurgia. Considerando que os indivíduos com este tipo de malformação podem passar por um período de privação sensorial auditiva anterior à reabilitação, torna-se interessante avaliar o estágio maturacional das estruturas auditivas corticais e o processamento das informações auditivas em nível central, bem como, verificar o benefício da indicação do VSB unilateral em situação de escuta difícil. Não foram encontrados estudos que abordam este aspecto e o emprego dos potenciais evocados auditivos corticais (PEAC) e do P300 em usuários de VSB. Objetivo: Analisar o impacto da perda auditiva condutiva e mista nos PEAC e P300 em usuários de VSB unilateral, com atresia de orelha bilateral, e verificar as habilidades auditivas, em situação de escuta difícil, considerando a indicação do VSB unilateral. Casuística e método: Vinte indivíduos, divididos em dois grupos, pareados em idade, sexo e grau de escolaridade. G1: dez indivíduos com perda auditiva condutiva ou mista bilateral, usuários de VSB unilateral, atendidos na Instituição de realização da pesquisa. Todos fizeram uso de aparelhos auditivos convencionais antes do VSB. G2: Dez indivíduos normo-ouvintes. Realização de audiometria em campo livre com o uso do VSB (apenas o G1), avaliação das habilidades auditivas pelo Hearing in Noise Test, pesquisa dos componentes P1, N1, P2, N2 e P300, em campo calibrado. Resultados: A média dos limiares tonais nas frequências de 500 a 3000 Hz, de 20 a 36 dB NA, mostrou que o VSB possibilitou o acesso aos sons da fala. Não foi observada diferença estatisticamente significante entre os valores de latência dos PEAC e P300 entre os grupos. Foi observada diferença estatisticamente significante entre o limiar de reconhecimento de sentenças e a relação sinal/ruído entre os grupos, sendo os melhores resultados apresentados pelo G2. Conclusão: Indivíduos com atresia de orelha e perda auditiva condutiva ou mista bilateral, quando adequadamente reabilitados, podem atingir a maturação das vias auditivas centrais e o processamento da informação auditiva em nível cortical. As habilidades de reconhecimento auditivo, sem e com ruído competitivo, mostraram-se defasadas quanto à normalidade, apontando para a indicação do VSB bilateral
Introduction: Congenital aural atresia is a congenital deformity. It is unilaterally prevalent due to alterations in the development of the external and middle ear structures. Congenital aural atresia causes conductive hearing loss and can be accompanied by sensorineural component. Among the available forms of treatment is the middle ear implant, Vibrant Soundbridge (VSB), which has been shown to be effective in treating this type of alteration. The literature shows improvement in tonal thresholds and in the results of tests of auditory perception of speech that were performed using the speech processor after surgery. Individuals with this type of malformation often experience a period of auditory sensory deprivation prior to rehabilitation. Hence, it is important to evaluate the maturation stage of the cortical auditory structures, the processing of auditory information at the central level, and to verify the benefit of unilateral VSB in difficult listening situations. There are no previous data on this aspect and with the use of cortical auditory evoked potentials (CAEP) and event-related potential (P300) in users of VSB. Aim: To analyze the impact of conductive and mixed hearing loss on CAEP and P300 in unilateral VSB users with bilateral ear atresia. To verify the auditory abilities in a difficult listening situation considering the indication for unilateral VSB. Materials and methods: Twenty individuals were divided into two groups matched for age, sex, and educational level. G1 comprised ten individuals with bilateral conductive or mixed hearing loss and users of unilateral VSB, who visited the research institution. All subjects used conventional hearing aids prior to VSB. G2 comprised ten normal hearing individuals. Audiometry in the free field was performed with the use of VSB (G1 only) and evaluation of hearing skills by the Hearing in Noise Test was conducted; components P1, N1, P2, N2, and P300 in a calibrated field were recorded. Results: Evaluation of the mean tonal thresholds in the frequencies between 500 and 3000 Hz, from 20 to 36 dB HL, demonstrated that VSB allowed access to speech sounds. There was no statistically significant difference in the CAEP and P300 latency values between the two groups. A statistically significant difference was observed in the sentence recognition threshold and the signal-to-noise ratio between the groups, with best results presented by G2. Conclusion: Individuals with congenital aural atresia and bilateral conductive or mixed hearing loss may reach maturation of the central auditory pathway and achieve adequate processing of auditory information at the cortical level, when rehabilitated. The auditory recognition skills, with and without competitive noise, were shown to be out of phase with normality, indicating the need for a bilateral VSB
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Graf, Isaac John 1972. "Simulation of the effects of sensorineural hearing loss." Thesis, Massachusetts Institute of Technology, 1997. http://hdl.handle.net/1721.1/42761.

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Thesis (M.S.)--Massachusetts Institute of Technology, Dept. of Electrical Engineering and Computer Science, 1997.
Includes bibliographical references (leaves 103-112).
by Isaac John Graf.
M.S.
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Cullen, J. R. "Sudden hearing loss : an animal model." Thesis, Queen's University Belfast, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.326426.

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Johnson, Earl E. "Amplification Options for Severe-to-Profound Sensorineural Hearing Loss." Digital Commons @ East Tennessee State University, 2014. https://dc.etsu.edu/etsu-works/1974.

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Books on the topic "Congenital sensorineural hearing loss"

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International Wullstein Symposium (1989 Würzburg, Germany). Sensorineural hearing loss and equilibrium disturbances. Stuttgart ; New York: Georg Thieme Verlag ; New York : Thieme Medical Publishers, 1990.

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Reger, Conference (1984 Iowa City Iowa). Sensorineural hearing loss: Mechanisms, diagnosis, treatment. Iowa City: University of Iowa, 1986.

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Reger Conference (1984 Iowa City, Iowa). Sensorineural hearing loss: Mechanisms, diagnosis, treatment. Iowa City: University of Iowa, 1986.

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White, Karl. Implementing universal newborn hearing screening programs: Early identification of hearing loss. [Rockville, MD]: Division of Services for Children with Special Health Needs, Maternal and Child Health Bureau, U.S. Dept. of Health & Human Services, Public Health Service, Health Resources & Services Administration, 1999.

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Hearing in aging. San Diego, Calif: Singular Pub. Group, 1995.

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Annemarie, Sommer, ed. Handbook of congenital and early onset hearing loss. New York: Igaku-Shoin, 1991.

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1935-, Honjō Iwao, and Takahashi H. 1952-, eds. Cochlear implant and related sciences update: 1st Asia Pacific Symposium on Cochlear Implant and Related Sciences, Kyoto, April 3-5, 1996. Basel: Karger, 1997.

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Razi, Mohammad Shakil. Cause of bilateral sensorineural hearing loss in school children in Karachi, Pakistan. Manchester: University of Manchester, 1994.

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Diagnosis and treatment of hearing impairment in children. 2nd ed. San Diego, Calif: Singular Pub. Group, 1998.

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Diagnosis and treatment of hearing impairment in children: A clinical manual. San Diego, Calif: College-Hill Press, 1985.

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Book chapters on the topic "Congenital sensorineural hearing loss"

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Ng, Matthew, and Drew M. Horlbeck. "Sensorineural Hearing Loss-Congenital-Genetics." In Encyclopedia of Otolaryngology, Head and Neck Surgery, 2399–407. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-23499-6_543.

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Park, Albert H. "Congenital Cytomegalovirus and Sensorineural Hearing Loss." In Encyclopedia of Otolaryngology, Head and Neck Surgery, 579–82. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-23499-6_813.

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Mikulec, Anthony A. "Congenital Hearing Loss (Sensorineural and Conductive)." In Pediatric Otolaryngology for the Clinician, 75–79. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-127-1_10.

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Bursle, C., A. Narendra, R. Chuk, J. Cardinal, R. Justo, B. Lewis, and D. Coman. "COXPD9 an Evolving Multisystem Disease; Congenital Lactic Acidosis, Sensorineural Hearing Loss, Hypertrophic Cardiomyopathy, Cirrhosis and Interstitial Nephritis." In JIMD Reports, 105–9. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/8904_2016_13.

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Miller, Sean M., and Anita Jeyakumar. "Sensorineural Hearing Loss." In Encyclopedia of Otolaryngology, Head and Neck Surgery, 2368–83. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-23499-6_840.

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Hempel, John Martin, and Karin Schorn. "Autoimmune Sensorineural Hearing Loss." In Hearing Impairment, 250–55. Tokyo: Springer Japan, 2004. http://dx.doi.org/10.1007/978-4-431-68397-1_48.

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Kanzaki, Jin, and Sho Kanzaki. "Curable Sensorineural Hearing Loss." In Hearing Impairment, 282–87. Tokyo: Springer Japan, 2004. http://dx.doi.org/10.1007/978-4-431-68397-1_55.

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Kessel, Aharon, and Elias Toubi. "Autoimmune Sensorineural Hearing Loss." In Diagnostic Criteria in Autoimmune Diseases, 449–53. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-60327-285-8_83.

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Larem, Aisha, Zaid Altamimi, and Adham Aljariri. "Sensorineural Hearing Loss (SNHL)." In Textbook of Clinical Otolaryngology, 111–19. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-54088-3_10.

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Zarandy, Masoud Motasaddi, and John Rutka. "Sudden Sensorineural Hearing Loss." In Diseases of the Inner Ear, 35–39. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-05058-9_4.

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Conference papers on the topic "Congenital sensorineural hearing loss"

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Holland Brown, T., FE Walston, and KEM McDevitt. "G465 A regional quality improvement project for congenital cytomegalovirus associated sensorineural hearing loss." In Royal College of Paediatrics and Child Health, Abstracts of the Annual Conference, 13–15 March 2018, SEC, Glasgow, Children First – Ethics, Morality and Advocacy in Childhood, The Journal of the Royal College of Paediatrics and Child Health. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2018. http://dx.doi.org/10.1136/archdischild-2018-rcpch.453.

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Li, Qiang, and Zhen Wang. "Altered functional connectivity in infants with congenital bilateral severe sensorineural hearing loss: A resting-state functional MRI study under sedation." In 2016 9th International Congress on Image and Signal Processing, BioMedical Engineering and Informatics (CISP-BMEI). IEEE, 2016. http://dx.doi.org/10.1109/cisp-bmei.2016.7852787.

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Li, Qiang, and Zhen Wang. "The relationship of development of cochlear nerve and inner ear morphology in children with congenital sensorineural hearing loss using high resolution MRI." In 2015 8th International Congress on Image and Signal Processing (CISP). IEEE, 2015. http://dx.doi.org/10.1109/cisp.2015.7407930.

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Khan, Z., R. Brown, V. Banks, and T. Holland Brown. "G266(P) A regional quality improvement project for congenital cytomegalovirus (cCMV) associated sensorineural hearing loss leading to improved prioritisation of finite resources during the covid-19 pandemic." In Royal College of Paediatrics and Child Health, Abstracts of the RCPCH Conference–Online, 25 September 2020–13 November 2020. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2020. http://dx.doi.org/10.1136/archdischild-2020-rcpch.230.

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Arpitha Nagesh, K., P. Kavya, B. K. Kavyashree, K. S. Kruthishree, T. P. Surekha, and D. L. Girijamba. "Digital Hearing Aid for Sensorineural Hearing Loss : (Ski-Slope Hearing Loss)." In 2017 International Conference on Current Trends in Computer, Electrical, Electronics and Communication (CTCEEC). IEEE, 2017. http://dx.doi.org/10.1109/ctceec.2017.8455016.

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Andreeva, Irina. "SPATIAL HEARING IN PATIENTS WITH SENSORINEURAL HEARING LOSS." In XVI International interdisciplinary congress "Neuroscience for Medicine and Psychology". LLC MAKS Press, 2020. http://dx.doi.org/10.29003/m916.sudak.ns2020-16/66.

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Kimyon, U., S. N. Esatoglu, E. Kara, A. Atas, E. E. Gunay, E. Karaman, E. D. Gozen, V. Hamuryudan, H. Yazici, and E. Seyahi. "SAT0546 Sensorineural hearing loss in takayasu’s arteritis." In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.2962.

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Storz, CS, O. Gross, J. Böckhaus, D. Beutner, and N. Strenzke. "Characterization of sensorineural hearing loss in children with Alport syndrome." In Abstract- und Posterband – 91. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Welche Qualität macht den Unterschied. © Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1711322.

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Shukurov, D. "Effectiveness of Intratympanic Steroid Treatment for Sudden Sensorineural Hearing Loss." In Abstract- und Posterband – 89. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Forschung heute – Zukunft morgen. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1640613.

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Heinz, Michael, and Kenneth S. Henry. "Modeling disrupted tonotopicity of temporal coding following sensorineural hearing loss." In ICA 2013 Montreal. ASA, 2013. http://dx.doi.org/10.1121/1.4800522.

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