Relevant bibliographies by topics / Connexin 26

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Academic literature on the topic 'Connexin 26'

Author: Grafiati
Published: 4 June 2021
Last updated: 26 July 2025

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Journal articles on the topic "Connexin 26"

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Birkenhäger, Ralf, Nicola Prera, Antje Aschendorff, Roland Laszig, and Susan Arndt. "A Novel Homozygous Mutation in the EC1/EC2 Interaction Domain of the Gap Junction Complex Connexon 26 Leads to Profound Hearing Impairment." BioMed Research International 2014 (2014): 1–7. http://dx.doi.org/10.1155/2014/307976.

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To date, about 165 genetic loci or genes have been identified which are associated with nonsyndromal hearing impairment. In about half the cases, genetic defects in theGJB2gene (connexin 26) are the most common cause of inner-ear deafness. The genesGJB2andGJB6are localized on chromosome 13q11-12 in tandem orientation. Connexins belong to the group of “gap junction” proteins, which form connexons, each consisting of six connexin molecules. These are responsible for the exchange of ions and smaller molecules between neighboring cells. Mutational analysis in genesGJB2andGJB6was brought by direct
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Sosinsky, Gina E. "Diversity in gap junction structures." Proceedings, annual meeting, Electron Microscopy Society of America 50, no. 1 (1992): 442–43. http://dx.doi.org/10.1017/s0424820100122617.

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Gap junctions are the specialized regions between two adjoining cells responsible for regulated communication. The morphological unit of the gap junction is composed of 12 copies of the connexin molecule. Six connexins form a hexamer in each cell membrane called a connexon and two connexons pair across the two cell membranes of coupled cells to form gated channels. Although other proteins are found in enriched gap junction preparations, it is generally accepted that the gap junction structures are formed from a family of connexin proteins. The connexins are named according to their DNA deduced
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Zong, Yan-Jun, Xiao-Zhou Liu, Lei Tu, and Yu Sun. "Cytomembrane Trafficking Pathways of Connexin 26, 30, and 43." International Journal of Molecular Sciences 24, no. 12 (2023): 10349. http://dx.doi.org/10.3390/ijms241210349.

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The connexin gene family is the most prevalent gene that contributes to hearing loss. Connexins 26 and 30, encoded by GJB2 and GJB6, respectively, are the most abundantly expressed connexins in the inner ear. Connexin 43, which is encoded by GJA1, appears to be widely expressed in various organs, including the heart, skin, the brain, and the inner ear. The mutations that arise in GJB2, GJB6, and GJA1 can all result in comprehensive or non-comprehensive genetic deafness in newborns. As it is predicted that connexins include at least 20 isoforms in humans, the biosynthesis, structural compositio
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TÖRÖK, Katalin, Kathryn STAUFFER, and W. Howard EVANS. "Connexin 32 of gap junctions contains two cytoplasmic calmodulin-binding domains." Biochemical Journal 326, no. 2 (1997): 479–83. http://dx.doi.org/10.1042/bj3260479.

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A fluorescent calmodulin derivative, 2-chloro-[4-(ϵ-amino-Lys75)]-[6-(4-diethylaminophenyl)-1,3,5-triazin-4-yl]-calmodulin (TA-calmodulin) [Török and Trentham (1994) Biochemistry 33, 12807–12820], and equilibrium fluorescence methods were used to identify calmodulin-binding domains of connexin subunits of gap junctions. Synthetic peptides corresponding to six extramembrane regions of connexin 32, a major component of rat liver gap junctions, and peptides derived from connexin 43 and 26, were tested. Two cytoplasmically oriented peptides that correspond to an N-terminal 21-amino-acid sequence a
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Garcia-Vega, Laura, Erin M. O’Shaughnessy, Ahmad Albuloushi, and Patricia E. Martin. "Connexins and the Epithelial Tissue Barrier: A Focus on Connexin 26." Biology 10, no. 1 (2021): 59. http://dx.doi.org/10.3390/biology10010059.

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Epithelial tissue responds rapidly to environmental triggers and is constantly renewed. This tissue is also highly accessible for therapeutic targeting. This review highlights the role of connexin mediated communication in avascular epithelial tissue. These proteins form communication conduits with the extracellular space (hemichannels) and between neighboring cells (gap junctions). Regulated exchange of small metabolites less than 1kDa aide the co-ordination of cellular activities and in spatial communication compartments segregating tissue networks. Dysregulation of connexin expression and f
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Torres, L. N., J. M. Matera, C. H. Vasconcellos, J. L. Avanzo, F. J. Hernandez-Blazquez, and M. L. Z. Dagli. "Expression of Connexins 26 and 43 in Canine Hyperplastic and Neoplastic Mammary Glands." Veterinary Pathology 42, no. 5 (2005): 633–41. http://dx.doi.org/10.1354/vp.42-5-633.

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Gap junctions are the only communicating junctions found in animal tissues and are composed of proteins known as connexins. Alterations in connexin expression have been associated with oncogenesis; reported studies in rodent and human mammary glands, which normally express connexins 26 and 43, confirm these alterations in malignancies. Mammary neoplasms represent the second most frequent neoplasm in dogs, and since there are no reports on the study of connexins in canine mammary glands, the present study investigated the expression of connexins 26 and 43 in normal, hyperplastic, and neoplastic
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AHMAD, Shoeb, Juan A. DIEZ, Christopher H. GEORGE, and W. Howard EVANS. "Synthesis and assembly of connexins in vitro into homomeric and heteromeric functional gap junction hemichannels." Biochemical Journal 339, no. 2 (1999): 247–53. http://dx.doi.org/10.1042/bj3390247.

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The biogenesis of connexins and their assembly into functional gap junction hemichannels (connexons) was studied with the use of a cell-free transcription/translation system. Velocity sedimentation on sucrose gradients showed that a small proportion of connexin (Cx) 26 and Cx32 that were co-translationally translocated into microsomes were oligomers of Cx26 and Cx32. Chemical cross-linking studies showed that these corresponded to hexameric connexons. Reconstitution of connexons synthesized in vitro into liposomes induced permeability properties consistent with the view that open gap junction
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Luu, Savannah, Cynthia Bell, Sarah Schneider, and Thu Annelise Nguyen. "Connexin 26 and Connexin 43 in Canine Mammary Carcinoma." Veterinary Sciences 6, no. 4 (2019): 101. http://dx.doi.org/10.3390/vetsci6040101.

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Incidence of canine mammary carcinoma is two times higher than the rate of human breast cancer. Mammary tumors are the most common type of cancer in intact female dogs and account for about half of all neoplasms in these dogs. Well-established models of breast cancer have shown that neoplastic cells often have a loss of intercellular communication, particularly gap junction proteins. Thus, the objective of this study is to explore the aspect of gap junction intercellular communication in canine mammary carcinoma, non-cancerous (CMEC) and cancerous (CMT12, CMT27, and CF41.Mg) cells, and patient
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Hosoya, Makoto, Masato Fujioka, Ayako Y. Murayama, Kaoru Ogawa, Hideyuki Okano, and Hiroyuki Ozawa. "Dynamic Spatiotemporal Expression Changes in Connexins of the Developing Primate’s Cochlea." Genes 12, no. 7 (2021): 1082. http://dx.doi.org/10.3390/genes12071082.

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Connexins are gap junction components that are essential for acquiring normal hearing ability. Up to 50% of congenital, autosomal-recessive, non-syndromic deafness can be attributed to variants in GJB2, the gene that encodes connexin 26. Gene therapies modifying the expression of connexins are a feasible treatment option for some patients with genetic hearing losses. However, the expression patterns of these proteins in the human fetus are not fully understood due to ethical concerns. Recently, the common marmoset was used as a primate animal model for the human fetus. In this study, we examin
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Lee, Y. C., C. E. Yellowley, Z. Li, H. J. Donahue, and D. E. Rannels. "Expression of functional gap junctions in cultured pulmonary alveolar epithelial cells." American Journal of Physiology-Lung Cellular and Molecular Physiology 272, no. 6 (1997): L1105—L1114. http://dx.doi.org/10.1152/ajplung.1997.272.6.l1105.

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Recent observations suggest that cell-cell interactions may modulate the response of the alveolar epithelium to injury. Expression and function of gap junctions were thus evaluated in isolated alveolar type II cells. Freshly isolated (day 0) type II cells expressed mRNAs for gap junctional connexins 26, 32, and 43. Whereas connexin 26 mRNA declined approximately 40% in cultured cells, connexin 32 message decreased rapidly and was not detectable on day 1. In contrast, connexin 43 expression increased 10-fold by day 3 compared with day 0. Western blot confirmed a 30-fold elevation in connexin 43
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Dissertations / Theses on the topic "Connexin 26"

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Meigh, Louise. "On the modulation of connexin 26 by CO2." Thesis, University of Warwick, 2014. http://wrap.warwick.ac.uk/61992/.

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The mechanism through which changes in PCO2 in the blood are detected is much disputed. Although many believe the stimulus for CO2 detection to be the associated increase in H+, increasing evidence supports a role for direct CO2 detection. In a recent development, Huckstepp et al demonstrated that connexin 26 hemichannels open in response to elevated CO2 in the absence of a pH change. This model however remained incomplete, with the mechanism of CO2 interaction with the channel being unknown. In this work I have employed site directed mutagenesis and dye loading studies to identify the CO2 bin
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Balandin, Taras [Verfasser]. "Expression and purification of human connexin 26 / Taras Balandin." Düsseldorf : Universitäts- und Landesbibliothek der Heinrich-Heine-Universität Düsseldorf, 2016. http://d-nb.info/111776124X/34.

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Petersen, Lars. "Molekulargenetische Diagnostik von Connexin 26 bei genetisch bedingter, nicht-syndromaler, sensorineuraler Schwerhörigkeit." [S.l.] : [s.n.], 2001. http://archiv.ub.uni-marburg.de/diss/z2001/0302/.

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Sinnathuray, A. R. "Connexin 26 (GJB2) gene-related sensorineural deafness and outcomes after cochlear implantation." Thesis, Queen's University Belfast, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.446129.

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Whitehead, Caragh (Caragh Bryony). "Molecular analysis of GJB2 (connexin 26) and GJB6 (connexin 30) gene mutations in non-syndromic hereditary deafness in South Africa." Thesis, Stellenbosch : Stellenbosch University, 2004. http://hdl.handle.net/10019.1/50028.

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Thesis (MSc)--University of Stellenbosch, 2004.<br>ENGLISH ABSTRACT: The most common inherited sensory disorder that affects I in 1 000 children is severe hearing loss. In developed countries, about a third of cases have a genetic origin, 80% of which are autosomal recessive forms (DFNB). Before 1993 few genes causing hearing loss had been identified, but since then a large number of genes related to this problem have been identified. Studies indicate that the DFNBI locus, located at position 13q11-12, contributes to 20% of all childhood deafness and may have a carrier rate as high as 2
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Donnelly, Steven. "Molecular mechanisms underlying mutations in Connexin 26 associated with genetically inherited skins disorders." Thesis, Glasgow Caledonian University, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.688293.

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Iftoda, O. M. "Gender aspects of connexin 26 (GJB2) gene polymorphism in children with hearing loss." Thesis, БДМУ, 2022. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/19712.

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Simpson, Charlotte Louise. "Molecular mechanisms regulating the epithelial barrier : key roles for Cx26 and ADAM17 during bacterial infection." Thesis, Queen Mary, University of London, 2015. http://qmro.qmul.ac.uk/xmlui/handle/123456789/9059.

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This study investigated how gastrointestinal and skin bacterial infections were affected by differential expression of connexin (Cx) 26 and a disintegrin and metalloprotease (ADAM) 17 in vitro. Cx26 is a component of gap junctions, which facilitate the transfer of small molecules between two cells. Recessive mutations in Cx26 cause non syndromic hearing loss (NSHL), and in certain populations, specific mutations account for the majority of Cx26 related NSHL. Their common occurrence suggests that they may provide a heterozygous, protective advantage to carriers. In this study adherence by the a
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Brockmeyer, Phillipp [Verfasser], Bernhard [Akademischer Betreuer] Hemmerlein, and Ralph [Akademischer Betreuer] Rödel. "Connexine als potenzielle Biomarker für den Progress oraler Plattenepithelkarzinome: Analyse der Expressionsmuster von Connexin 26, 43 und 45 und ihres Einflusses auf das Überleben / Phillipp Brockmeyer. Gutachter: Bernhard Hemmerlein ; Ralph Rödel. Betreuer: Bernhard Hemmerlein." Göttingen : Niedersächsische Staats- und Universitätsbibliothek Göttingen, 2014. http://d-nb.info/1053555377/34.

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Al-Hazza, Aseel [Verfasser], Wafaa [Gutachter] Shehata-Dieler, Kathleen [Gutachter] Wermke, and Thomas [Gutachter] Haaf. "Zu Grad, Konfiguration und Verlauf der Schallempfindungsschwerhörigkeit bei Kindern mit einer Connexin-26-Mutation / Aseel Al-Hazza ; Gutachter: Wafaa Shehata-Dieler, Kathleen Wermke, Thomas Haaf." Würzburg : Universität Würzburg, 2017. http://d-nb.info/1138196231/34.

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Books on the topic "Connexin 26"

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Schmidt, David. Bioanalytische Struktur-Funktionsmessungen an Ionenkanälen: Entwicklung optischer Analysemethoden an punktmutierten KcsA und Connexin 26. Springer Spektrum, 2017.

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Minutes of Several Conversations Between the Methodist Preachers in the Connexion Established by the Late Rev. John Wesley, A. M.: At Their Ninety-Fourth Annual Conference, Begun in Leeds, on Wednesday July 26 1837. Creative Media Partners, LLC, 2021.

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ATLAS '99: Arabic translation and localized symposium : Symposium sur la traduction et la localisation en Arabe : Proceedings Actes : Les Berges du Lac, Tunis : le 26 et 27 Mai 1999 : Symposium organise en connexion avec IDOL : Le project IDOL (INCO 961263). Imp. JMS, 1999.

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Book chapters on the topic "Connexin 26"

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Desmond, Adrian. "26. Provisions for the Afterlife." In Reign of the Beast. Open Book Publishers, 2024. http://dx.doi.org/10.11647/obp.0393.26.

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Intimations of mortality—Saull turned 70 in 1853 and had already been dangerously ill—should have made the search for a posthumous home for the museum paramount. Holyoake was reminding him of the late John Barber Beaumont and Charles Jenkins, whose bequests had not resulted in institutions, as they had intended. Saull still spent his days at the Mechanics’ Hall of Science, and we have first-hand accounts of life here, and its hosting of Chartists from Bonner’s Fields after their skirmishes with the police. Nor had co-operation lost its attraction: Saull could be found at the Co-Operative Leagu
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Mesnil, Marc, V. Krutovskikh, C. Piccoli, et al. "Growth inhibition by expression of connexin 26 in HeLa cells." In Intercellular Communication through Gap Junctions. Elsevier, 1995. http://dx.doi.org/10.1016/b978-0-444-81929-1.50031-9.

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Kamibayashi, Yoshihito, Masahito Oyamada, Yumiko Oyamada, Seishu Abe, Makoto Takahashi, and Michio Mori. "Expression and localization of connexin 26 and 43 in mouse epidermis in vivo." In Intercellular Communication through Gap Junctions. Elsevier, 1995. http://dx.doi.org/10.1016/b978-0-444-81929-1.50072-1.

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"Connexins: indicators for hormonal and blastocyst-mediated endometrial differentiation." In The Endometrium. CRC Press, 2008. http://dx.doi.org/10.3109/9780203091500-26.

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Bargiello, Thaddeus A., Seunghoon Oh, Yi Ri, Priscilla E. Purnick, and Vytas K. Verselis. "Chapter 14: Molecular Determinants of Voltage Gating of Gap Junctions Formed by Connexin32 and 26." In Gap Junctions - Molecular Basis of Cell Communication in Health and Disease. Elsevier, 1999. http://dx.doi.org/10.1016/s0070-2161(08)61018-7.

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"No. 927. Agreement between the Government of the Union of South Africa and the Government of the United Kingdom of Great Britain and Northern Ireland in connexion with the establishment of civil air services between the Union and the United Kingdom. Signed at Pretoria, on 26 October 1945." In United Nations Treaty Series. UN, 1999. http://dx.doi.org/10.18356/95f1ffa8-en-fr.

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Conference papers on the topic "Connexin 26"

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Steffens, M., H. Wang, T. Lenarz, A. Warnecke, K. Wissel, and C. Zeilinger. "Temperatursensitivität von Connexin 26." In Abstract- und Posterband – 90. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Digitalisierung in der HNO-Heilkunde. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1686283.

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Steffens, M., H. Wang, T. Lenarz, A. Warnecke, K. Wissel, and C. Zeilinger. "Temperature sensitivity of connexin 26." In Abstract- und Posterband – 90. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Digitalisierung in der HNO-Heilkunde. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1686512.

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Bijnsdorp, Irene V., Laurence Rozendaal, R. Jeroen A. van Moorselaar, and Albert A. Geldof. "Abstract LB-14: Low connexin-26 predicts the development of metastasis after radical prostatectomy." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-lb-14.

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Lima, Joalisson Batista De, Guilherme James Carvalho Costa, Jamilly Lima Santos, Delmo Soares Freire, Rodolfo Rodrigo Santos Feitosa, and ERBS CINTRA DE SOUZA GOMES. "IMPLANTAÇÃO DE QUINTAIS PRODUTIVOS DE BATATA-DOCE BIOFORTIFICADA ‘CIP-BRS-Nuti' COMO ESTRATÉGIA DE SUPERAÇÃO DA POBREZA EXTREMA NO ESTADO DE PERNAMBUCO." In Anais do Congresso Norte Nordeste de Pesquisa e Inovação (CONNEPI). Even3, 2023. http://dx.doi.org/10.29327/1202042.1-26.

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Reports on the topic "Connexin 26"

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Hrynick, Tabitha, Godefroid Muzalia, and Myfanwy James. Considérations clés : Communication des risques et engagement communautaire pour la vaccination contre la mpox dans l’est de la République démocratique du Congo. Institute of Development Studies, 2024. http://dx.doi.org/10.19088/sshap.2024.032.

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Cette note stratégique présente des considérations sociales et politiques pour la conception et la mise en œuvre de stratégies de communication des risques et d’engagement communautaire (CREC) liées à la vaccination contre la mpox en République démocratique du Congo (RDC). Une flambée épidémique de mpox (clade I) à l’échelle nationale a été déclarée fin 2022 et touche désormais 23 de ses 26 provinces. En particulier, la flambée épidémique est caractérisée par une transmission interhumaine généralisée, contrairement aux précédentes, qui impliquaient principalement un contact animal-humain. Tand
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