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Relevant bibliographies by topics / Contractile Proteins

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Academic literature on the topic 'Contractile Proteins'

Author: Grafiati

Published: 4 June 2021

Last updated: 29 January 2023

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Journal articles on the topic "Contractile Proteins"

1

Ruff, R. L., and J. Weissman. "Iodoacetate-induced contracture in rat skeletal muscle: possible role of ADP." American Journal of Physiology-Cell Physiology 261, no. 5 (1991): C828—C836. http://dx.doi.org/10.1152/ajpcell.1991.261.5.c828.

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The effects of iodoacetic acid (IAA) and ischemic contraction were studied in rat extensor digitorum longus muscles. Ischemic contraction of IAA-treated muscles produced contracture. The onset of contracture was not associated with a change in sarcolemmal electrical properties or reduction in intracellular [ATP]; however, [creatine phosphate] was reduced by 75% and free [ADP] was increased by 665%. Continued stimulation of IAA-treated fibers resulted in depolarization, loss of membrane excitability, further depletion of creatine phosphate, and reduction in [ATP]. The effects seen in IAA-treate

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2

Gros, Francois, and Margaret Buckingham. "Polymorphism of contractile proteins." Biopolymers 26, S0 (1987): S177—S192. http://dx.doi.org/10.1002/bip.360260016.

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Syrový, I. "Isoforms of contractile proteins." Progress in Biophysics and Molecular Biology 49, no. 1 (1987): 1–27. http://dx.doi.org/10.1016/0079-6107(87)90007-1.

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Ordway, George A., P. Darrell Neufer, Eva R. Chin, and George N. DeMartino. "Chronic contractile activity upregulates the proteasome system in rabbit skeletal muscle." Journal of Applied Physiology 88, no. 3 (2000): 1134–41. http://dx.doi.org/10.1152/jappl.2000.88.3.1134.

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Remodeling of skeletal muscle in response to altered patterns of contractile activity is achieved, in part, by the regulated degradation of cellular proteins. The ubiquitin-proteasome system is a dominant pathway for protein degradation in eukaryotic cells. To test the role of this pathway in contraction-induced remodeling of skeletal muscle, we used a well-established model of continuous motor nerve stimulation to activate tibialis anterior (TA) muscles of New Zealand White rabbits for periods up to 28 days. Western blot analysis revealed marked and coordinated increases in protein levels of

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Yamaguchi, Osamu, Yoshinari Sakagami, Takayuki Suzuki, Masato Kobayashi, and Yasuo Shiraiwa. "CONTRACTILE PROTEINS IN THE KIDENY." Japanese Journal of Urology 79, no. 2 (1988): 326–31. http://dx.doi.org/10.5980/jpnjurol1928.79.2_326.

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Sakagami, Yoshinari. "CONTRACTILE PROTEINS IN THE KIDNEY." Japanese Journal of Urology 79, no. 2 (1988): 332–38. http://dx.doi.org/10.5980/jpnjurol1928.79.2_332.

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KIEHART, D. P., A. KETCHUM, P. YOUNG, et al. "Contractile Proteins in Drosophila Development." Annals of the New York Academy of Sciences 582, no. 1 Cytokinesis (1990): 233–51. http://dx.doi.org/10.1111/j.1749-6632.1990.tb21683.x.

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Mehta, D., M. F. Wu, and S. J. Gunst. "Role of contractile protein activation in the length-dependent modulation of tracheal smooth muscle force." American Journal of Physiology-Cell Physiology 270, no. 1 (1996): C243—C252. http://dx.doi.org/10.1152/ajpcell.1996.270.1.c243.

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The active isometric force developed by a muscle decreases at muscle lengths below an optimal length (Lo). However, when the length of an actively contracting muscle is abruptly decreased, a lower level of isometric force is reached during force redevelopment than when the contraction is initiated at the shorter length. This has been attributed to a deactivation of contractile proteins caused by shortening. In this study, intracellular Ca2+ and myosin light chain (MLC) phosphorylation were measured to assess the mechanisms for the modulation of isometric force caused by changing smooth muscle

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Dou, Ying, Per Arlock, and Anders Arner. "Blebbistatin specifically inhibits actin-myosin interaction in mouse cardiac muscle." American Journal of Physiology-Cell Physiology 293, no. 3 (2007): C1148—C1153. http://dx.doi.org/10.1152/ajpcell.00551.2006.

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Blebbistatin is a powerful inhibitor of actin-myosin interaction in isolated contractile proteins. To examine whether blebbistatin acts in a similar manner in the organized contractile system of striated muscle, the effects of blebbistatin on contraction of cardiac tissue from mouse were studied. The contraction of paced intact papillary muscle preparations and shortening of isolated cardiomyocytes were inhibited by blebbistatin with inhibitory constants in the micromolar range (1.3–2.8 μM). The inhibition constants are similar to those previously reported for isolated cardiac myosin subfragme

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Pavalko, F. M., L. P. Adam, M. F. Wu, T. L. Walker, and S. J. Gunst. "Phosphorylation of dense-plaque proteins talin and paxillin during tracheal smooth muscle contraction." American Journal of Physiology-Cell Physiology 268, no. 3 (1995): C563—C571. http://dx.doi.org/10.1152/ajpcell.1995.268.3.c563.

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Reorganization of cytoskeletal-membrane interactions during contractile stimulation may contribute to the regulation of airway smooth muscle contraction. We investigated the effect of contractile stimulation on the phosphorylation of the actin-membrane attachment proteins talin, vinculin, and paxillin. Stimulation of 32P-labeled canine tracheal smooth muscle strips with acetylcholine (ACh; 10(-3) M) resulted in a rapid 2.6-fold increase in phosphorylation of serine and/or threonine residues, compared with resting levels of 0.22 mol PO4(3-)/mol talin. After stimulation with ACh, phosphorylation

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Dissertations / Theses on the topic "Contractile Proteins"

1

Bayliss, Christopher Richard. "Dysfunction of contractile proteins in hypertrophic cardiomyopathy." Thesis, Imperial College London, 2012. http://hdl.handle.net/10044/1/9293.

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The contractility of human heart samples from patients diagnosed with hypertrophic cardiomyopathy were studied using a quantitative in vitro motility assay. The aim of this work was to investigate the molecular phenotype of thin filament proteins in the HCM heart. Three biopsy samples with thin filament mutations were studied alongside samples acquired from a subset of HCM patients classified with hypertrophic obstructive cardiomyopathy. The primary effect of thin filament mutations was investigated by reconstituting Factin with ACTC E99K into thin filament with donor troponin. The E99K actin

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Du, Fei. "A RabGAP protein and BEACH Family proteins regulate contractile vacuole formation and activity and chemotaxis in Dictyostelium." Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2007. http://wwwlib.umi.com/cr/ucsd/fullcit?p3274747.

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Thesis (Ph. D.)--University of California, San Diego, 2007.<br>Title from first page of PDF file (viewed Oct. 5, 2007). Available via ProQuest Digital Dissertations. Vita. Includes bibliographical references (p. 87-99).

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Stromme, Adrianna. "The characterization of the cytoskeleton and associated proteins in the formation of wound-induced contractile arrays /." Thesis, McGill University, 2008. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=116078.

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The cytoskeleton is an intrinsic aspect of all cells, and is essential for many cellular events including cell motility, endocytosis, cell division and wound healing. Remodeling of the cytoskeleton in response to these cellular activities leads to significant alterations in the morphology of the cell. One such alteration is the formation of an actomyosin contractile array required for cytokinesis, wound healing and embryonic development.<br>Cellular structure and shape depends upon tensional prestress brought about by the organization of cytoskeletal components. Using the Xenopus laevis oocyte

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Bexis, Sotiria. "The relationship between vascular structure, contractile proteins, vascular reactivity and blood pressure in animal models of hypertension /." Title page, contents and abstract only, 1997. http://web4.library.adelaide.edu.au/theses/09PH/09phb572.pdf.

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Llinares, Elisa. "Function, regulation and intracellular trafficking of the vacuolaryeast pq-loop (Ypq) proteins." Doctoral thesis, Universite Libre de Bruxelles, 2012. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/209704.

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The cytoplasm of eukaryotic cells contains several membrane-delimited compartments of specific molecular compositions and functions. Among those, the vacuole of fungal cells is often described as an organelle equivalent to the lysosomes of animal cells and the vacuoles of plant cells. These compartments indeed share two similar features: they contain a wide variety of hydrolases and are the most acidic compartments of the cell, which accounts for their key role in the intracellular degradation of macromolecules. In humans, dysfunctions of the lysosomes often give rise to lysosomal related dise

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Crampin, Helen. "The identification of a SpitzenkoÌˆrper in 'C. albicans' and the partial characterisation of the contractile ring proteins". Thesis, University of Sheffield, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.425604.

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Willott, Ruth Heather. "Functional analyses of cardiomyopathic contractile proteins : mutations in troponin that cause familial hypertrophic cardiomyopathy and familial dilated cardiomyopathy." Thesis, University of Oxford, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.400293.

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Bengreed, Amal H. I. "Characterisation of P2Y receptor-mediated contractile signalling and its regulation by G protein coupled receptor kinases and arrestin proteins in a rat bladder smooth muscle." Thesis, University of Leicester, 2018. http://hdl.handle.net/2381/42795.

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ATP released from parasympathetic nerves can mediated bladder contraction, can activate purinergic P2Y/Gq/11-coupled G protein-coupled receptors (GPCR) expressed on detrusor (bladder) smooth muscle cells (DSMC). P2Y/Gq/11 signalling activates phospholipase C (PLC) and increases intracellular calcium concentrations to induce contraction. DSMC contractile GPCR activity is tightly regulated to prevent inappropriate contraction/incontinence. Additionally, GPCRs activity is regulated by G protein-coupled receptor kinase (GRK) and arrestin proteins, it is likely that they play a similar role in DSMC

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Johnsen, Lisa 1987. "Lipid droplet regulation by the differentially spliced proteins Osw5L and Osw5S." Doctoral thesis, Universitat Pompeu Fabra, 2016. http://hdl.handle.net/10803/565566.

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Epithelial tissues undergo extensive remodeling during embryonic development. Recent studies have revealed that, in a number of developmental processes, epithelial remodeling is associated with pulsations of individual cell surface areas and cortical actomyosin flows. During Drosophila dorsal closure, the amnioserosa (AS), a contractile tissue covering the dorsal region of the embryo, shows contractile pulsations and regular actomyosin flows during the reduction of its apical surface area. The biophysical mechanism driving these shape pulsations as well as the role of contractile actomyosin w

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Sjuve, Rolf. "Function of contractile and cytoskeletal proteins in smooth muscle effects of hypertrophy and age and of desmin removal in a transgenic animal /." Lund : Dept. of Physiology and Neuroscience, Lund University, 1998. http://books.google.com/books?id=ccFqAAAAMAAJ.

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Books on the topic "Contractile Proteins"

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1952-, Johnson Robert G., Kranias Evangelia G, and New York Academy of Sciences., eds. Cardiac sarcoplasmic reticulum function and regulation of contractility. New York Academy of Sciences, 1998.

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2

R, Barton Paul J., ed. Molecular biology of cardiac development and growth. R.G. Landes Co., 1995.

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Sabry, Mohamed Abdalla. Development transitions of the contractile regulatory proteins, troponin I and troponin T, in striated muscles: An immunochemical study. University of Birmingham, 1992.

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4

Carrington, Charlotte A. The pressor response to isometric exercise in man: Its relationship to age, muscle contractile characteristics and contractile protein profile. University of Birmingham, 1993.

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McHale, James M. Contracting with the federal government: Awards, protests, and disputes. M. Bender, 1985.

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1933-, Pette Dirk, and Symposium "The Dynamic State of Muscle Fibers" (1989 : University of Konstanz), eds. The Dynamic state of muscle fibers: Proceedings of the international symposium, October 1-6, 1989, Konstanz, Federal Republic of Germany. De Gruyter, 1990.

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7

Nature's versatile engine: Insect flight muscle inside and out. Landes Bioscience/Eurekah.com, 2006.

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8

1934-, Ozawa Eijirō, Masaki Tomoh, and Nabeshima Yoichi, eds. Frontiers in muscle research: Myogenesis, muscle contraction, and muscle dystrophy : proceedings of the Uehara Memorial Foundation Symposium on Frontiers in Muscle Research, Tokyo, 15-19 July 1990. Excerpta Medica, 1991.

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9

B, Vallee Richard, ed. Structural and contractile proteins. Academic Press, 1986.

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10

W, Cunningham Leon, ed. Structural and contractile proteins. Academic Press, 1987.

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Book chapters on the topic "Contractile Proteins"

1

Bagshaw, Clive R. "Contractile proteins." In Muscle Contraction. Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-015-6839-5_4.

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Schliwa, Manfred. "Cytoplasmic Contractile Proteins." In The Cytoskeleton. Springer Vienna, 1986. http://dx.doi.org/10.1007/978-3-7091-7667-2_2.

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Walsh, Michael P. "Contractile Proteins of Smooth Muscle." In Physiology and Pathophysiology of the Heart. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4613-0873-7_42.

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Winegrad, S., G. McClellan, L. E. R. Lin, and S. Weindling. "Modulation properties of myocardial contractile proteins." In Developments in Cardiovascular Medicine. Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-009-3313-2_15.

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Fox, Joan E. B. "The Organization of Platelet Contractile Proteins." In Platelet Membrane Glycoproteins. Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-4880-1_13.

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Izumi, Tohru, Haruo Hanawa, Makihiko Saeki, and Makoto Kodama. "Cardiac Contractile Proteins and Autoimmune Myocarditis." In Cellular Function and Metabolism. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-3078-7_10.

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Ebashi, S. "Contractile and Regulatory Proteins in Cardiovascular System." In Developments in Cardiovascular Medicine. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-2053-1_18.

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Roberts, Robert, Jeffrey Towbin, Thomas Parker, and Roger D. Bies. "Molecular Biology of Contractile and Cytoskeletal Proteins." In A Primer of Molecular Biology. Springer US, 1992. http://dx.doi.org/10.1007/978-1-4684-6680-5_5.

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Goldfine, S. M., I. Peng, and D. A. Fischman. "The Insertion and Release of Contractile Proteins." In The Dynamic State of Muscle Fibers, edited by Dirk Pette. De Gruyter, 1990. http://dx.doi.org/10.1515/9783110884784-011.

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Kaneko, Masanori, and Yuji Matsumoto. "Changes in Contractile Proteins under Oxidative Stress." In Developments in Cardiovascular Medicine. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4613-1235-2_10.

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Conference papers on the topic "Contractile Proteins"

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Saban, Melissa, Narayanan Venkatesan, Michelle L. D'Antoni, Stephanie Pasternyk, and Mara S. Ludwig. "Effect Of Decorin On Airway Smooth Muscle Cell Contractile Proteins." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a2074.

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Spangenberg, P., W. Lobche, and U. Till. "ALTERATIONS OP THE ACTIN STATUS OP PLATELETS APPECTS THE FUNCTIONAL BEHAVIOUR OP THE CELL." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644874.

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Many platelet responses to agonists involve contractile proteins. Particularly, the status of the most abundant protein actin is changed in motile and contractile events. We have studied the effects of SH-reagents on platelets and found significant effects of a SH-oxidizing agent on the actin organization which are neutralized following the addition of a disulfide-reducing compound. SH-oxidation of intact platelets was achieved by incubation with diamide (azodicarboxylic acid-bis-dimethylamide). The P-actin of those cells is increased and the filaments became centralized indicating a disturban

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Ramirez, Angelica Maria, Begoña Calvo Calzada, and Jorge Grasa. "The Effect of the Fascia on the Stress Distribution in Skeletal Muscle." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19696.

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The human and vertebrate interaction with the environment is done primarily through the movement. This is possible due the skeletal muscle: anatomical structure able to contract voluntarily. The skeletal muscles are made up of contractile proteins which slide one over another allowing the muscle shortening and the body force generation. This protein structure of actin and myosin maintains its organization through the connective tissue that surrounds it (endomysium, perimysium and epimysium), creating arrays of myofibrils, fibre bundles, fascicles until conform the whole muscle. All this connec

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Southern, B. D., H. Li, J. F. Crish, L. M. Grove, R. G. Scheraga, and M. A. Olman. "S100a4 Mediates Myofibroblast Differentiation and Experimental Pulmonary Fibrosis Through Subcellular Redistribution of Contractile Cytoskeleton Proteins." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a6391.

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Scott, Devon, Robin Shandas, and Wei Tan. "Effect of Vessel Stiffening and High Pulsatility Flow on Contractile Function and Proliferation of Small Arterial Cells." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19597.

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Recent studies have identified arterial stiffening as a predictor of some vascular diseases such as pulmonary hypertension, which is characterized by dysfunction of small arteries. Stiffening is shown to cause changes in blood flow, extending high pulsatile flow into small arteries that normally experience steady flow conditions (Chui 2004). However, few studies have investigated the mechanisms underlying the effects of arterial stiffening on vascular remodeling. We hypothesized that arterial stiffness effects dysfunction of downstream vascular endothelium and smooth muscle through changes in

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Schen, Aaron, and Lisa X. Xu. "Preliminary Study of Vascular Endothelial Ca2+ Response to Elevated Temperature." In ASME 2000 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2000. http://dx.doi.org/10.1115/imece2000-2487.

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Abstract The thermal regulation of tissue is controlled by the sympathetically mediated redistribution of cardiac output and change in local flow resistance of arterioles. The diameter change of the vessel from its resting level is governed by the state of the contractile proteins in vascular smooth muscle which can be influenced by the concentration of free cytosolic calcium ([Ca2+]i) in the vascular endothelial cells (Falcone, 1995).

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Sewell-Loftin, M. K., and W. David Merryman. "The Role of SRC in Strain- and Ligand- Dependent Phenotypic Modulation of Mouse Embryonic Fibroblasts." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53604.

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Connective tissue fibrosis represents a significant portion of mortality and morbidity in our society. These diseases include many illnesses such as heart valve disease, atherosclerosis, macular degeneration, and cirrhosis, meaning that millions of lives are affected by these conditions each year. Fibrotic tissues form when quiescent fibroblasts activate becoming myofibroblasts, the phenotype of active tissue construction and fibrosis. During this process, the cells produce smooth muscle α-actin (αSMA), a contractile element considered to be the hallmark of cellular activation [1]. Following t

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Southern, B. D., H. Li, J. F. Crish, et al. "Endogenous Fibroblast S100a4 Mediates Myofibroblast Differentiation and Pulmonary Fibrosis Through Matrix-Stiffness Dependent Redistribution of Contractile Proteins." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a4416.

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Shikhaleva, E., T. Sulman, A. Dokuchaev, Larisa V. Nikitina, and Leonid B. Katsnelson. "Mathematical modeling of the role of cooperativity between contractile and regulatory proteins in the mechano-calcium feedbacks in myocardium." In 2015 Computing in Cardiology Conference (CinC). IEEE, 2015. http://dx.doi.org/10.1109/cic.2015.7408651.

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Vernerey, Franck J. "Biophysical Model of the Coupled Mechanisms of Cell Adhesion, Contraction and Spreading." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80309.

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Recent research has shown that cell spreading is highly dependent on the contractility of its cytoskeleton and the mechanical properties of its surrounding environment. This extended abstract introduces a mathematical formulation of cell spreading and contraction that couples the processes of stress fiber formation, protrusion growth through actin polymerization at the cell edge and dynamics of cross-membrane protein (integrins) enabling cell-substrate attachment. The evolving cell’s cytoskeleton is modeled as a mixture of fluid, proteins and filaments that can exchange mass and generate contr

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Reports on the topic "Contractile Proteins"

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Rafaeli, Ada, and Russell Jurenka. Molecular Characterization of PBAN G-protein Coupled Receptors in Moth Pest Species: Design of Antagonists. United States Department of Agriculture, 2012. http://dx.doi.org/10.32747/2012.7593390.bard.

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The proposed research was directed at determining the activation/binding domains and gene regulation of the PBAN-R’s thereby providing information for the design and screening of potential PBAN-R-blockers and to indicate possible ways of preventing the process from proceeding to its completion. Our specific aims included: (1) The identification of the PBAN-R binding domain by a combination of: (a) in silico modeling studies for identifying specific amino-acid side chains that are likely to be involved in binding PBAN with the receptor and; (b) bioassays to verify the modeling studies using mut

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