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Books on the topic 'Control of larvae'

Author: Grafiati

Published: 4 June 2021

Last updated: 17 February 2022

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1

Swink, William D. Comparison of mark retention and survival of sea lamprey larvae marked by pigment injection and tail clipping. Ann Arbor, Mich: Great Lakes Fishery Commission, 1995.

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2

Raquel, Paul F. Striped bass egg and larval monitoring near the proposed Montezuma Slough control structure, 1987. [California]: Interagency Ecological Study Program for the Sacramento-San Joaquin Estuary, 1988.

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3

West Nile virus targeted mosquito larval control program: 2005 grant program guidelines. Edmonton, Alta: Alberta Municipal Affairs, 2005.

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4

McKenney, Charles L. Influence of an insect growth regulator on larval development of a marine crustacean. Gulf Breeze, FL: U.S. Environmental Protection Agency, Environmental Research Laboratory, 1988.

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5

Gong shui xi tong hong chong fang zhi ji shu. Beijing: Zhongguo jian zhu gong ye chu ban she, 2008.

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6

West, Richard J. Ground spray applications of Virtuss, a nuclear polyhedrosis virus, against white-marked tussock moth larvae at Bottom Brook, Newfoundland in 1986. St. John's: Newfoundland Forestry Centre, 1987.

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7

Crease, Gary John. Studies of the biological efficacy of the pyrethroid insecticide cypermethrin applied to plant surfaces for the control of larvae of Spodoptera littoralis Boisd. Portsmouth: Portsmouth Polytechnic, School of Biological Sciences, 1987.

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8

Scholefield, Ronald J. A comparison of two methods for the simultaneous determination of TFM and Bayer 73 concentrations. Ann Arbor, Mich: Great Lakes Fishery Commission, 1987.

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9

Copeland, Lori. Out of control. South Yarmouth, Ma: Curley Pub., 1991.

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10

Connealy, Mary. Out of control. Waterville, Me: Thorndike Press, 2011.

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11

Scholefield, Ronald J. Toxicity of 2', 5-dichloro-4'-nitrosalicylanilide (Bayer 73) to three genera of larval lampreys. Ann Arbor, Mich: Great Lakes Fishery Commission, 1992.

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12

Taking control of arthritis. New York, N.Y: HarperCollins, 1990.

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13

Taking control of arthritis. Thorndike, Me: Thorndike Press, 1991.

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14

Boys in control. New York: Delacorte Press, 2003.

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15

Little, Jean. Boys in control. Waterville, Me: Thorndike Press, 2004.

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16

Baldacci, David. Total control. New York: Warner Vision Books, 1997.

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17

Baldacci, David. Total control. London: BCA, 1997.

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18

Baldacci, David. Total control. Thorndike, Me: Thorndike Press, 1997.

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19

Baldacci, David. Total control. New York: Warner Vision Books, 1997.

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20

Baldacci, David. Total control. London: Pocket Books, 2000.

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21

Baldacci, David. Total control. New York, NY: Warner Books, 1997.

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22

Joshi, S. M. Control of large flexible space structures. Berlin: Springer-Verlag, 1989.

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23

Damage control: [a novel]. Waterville, Me: Thorndike Press, 2011.

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24

Billerbeck, Kristin. She's out of control. Waterville, Me: Christian Large Print, 2009.

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25

Russell, Richard Allen. Space structure (dynamics and control) theme development. Hampton, Va: National Aeronautics and Space Administration, Langley Research Center, 1988.

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26

1947-, Hassan Mohamed F., and Darwish Mohamed G. 1948-, eds. Large-scale control systems: Theories and techniques. New York: M. Dekker, 1985.

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27

Efficient modeling and control of large-scale systems. New York: Springer, 2010.

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28

Veit, H. Untersuchungen zu den Befallsbedingungen, zum Befallsablauf und zur Abwehr des Befalls von Laub- und Nadelholz durch den Sägehörnigen Werftkäfer (Hylecoetus dermestoides Latr. (Coleoptera, Lymexylidae)). Freiburg: Forstliche Versuchs- und Forschungsanstalt Baden-Württemberg, Abteilung Waldschutz, 2005.

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29

She's out of control: A novel. Nashville, Tenn: WestBow Press, 2004.

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30

Kung, Hsiao-Feng. Dynamics and control of a spatial truss actuator. Blacksburg, Va: Virginia Polytechnic Institute and State University, 1988.

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31

Hamer, Harold A. Effects of model errors on control of large flexible space antenna with comparisons of decoupled and linear quadratic regulator control procedures. Hampton, Va: Langley Research Center, 1986.

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32

Killer in control: A Key West mystery. Waterville, Me: Thorndike Press, 2011.

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33

Liu, Yongqing. Stability, stabilization and control of large scale systems. Beijing, China: Science Press, 1989.

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34

Junkins, John L. Introduction to dynamics and control of flexible structures. Washington, D.C: American Institute of Aeronautics and Astronautics, 1993.

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35

Junkins, John L. Introduction to dynamics and control of flexible structures. Washington, D.C: American Institute of Aeronautics and Astronautics, 1993.

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36

Bayard, David S. Adaptive control for flexible space structures with measurement noise. [Washington, D. C.]: American Institute for Aeronautics and Astronautics, 1988.

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37

Johnson, C. D. A multiple pointing-mount control strategy for space platforms: Final report. [Washington, DC: National Aeronautics and Space Administration, 1992.

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38

Johnson, C. D. A multiple pointing-mount control strategy for space platforms: Final report. [Washington, DC: National Aeronautics and Space Administration, 1992.

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39

Cameron, Ian. Operating the Daisy facility to perform control experiments. [Downsview, Ont.]: University of Toronto, Graduate Dept. of Aerospace Science and Engineering, 1993.

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40

Little, Jean. Joey Pigza loses control. Thorndike, ME: Thorndike Press, 2001.

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41

Joey Pigza loses control. New York: Farrar, Straus and Giroux, 2000.

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42

Little, Jean. Joey Pigza loses control. New York: HarperTrophy, 2002.

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43

McLaren, David Stuart. The use of observers in the control of large, flexible spacecraft. [Downsview, Ont.]: Dept. of Aerospace Science and Engineering, 1985.

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44

Decentralized control and filtering in interconnected dynamical systems. Boca Raton: CRC Press, 2010.

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45

Potency of Bacillus thuringiensis strains and formulations against gypsy moth and spruce budworm larvae, 1980-86. Broomall, Pa: U.S. Dept. of Agriculture, Forest Service, Northeastern Forest Experiment Station, 1989.

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46

Stray, James Everett. Purification and characterization of mosquito-larvicidal toxins from Bacillus sphaericus. 1989.

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47

Savage, Thomas J. Thinning and urea fertilization effects on emerging grand fir (Abies grandis) foliage and growth of western spruce budworm (Choristoneura occidentalis) larvae. 1987.

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48

Nolan, T. J., T. B. Nutman, and G. A. Schad. Strongyloidosis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0064.

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Abstract:

Strongyloidosis is an intestinal parasitism caused by the threadworm, Strongyloides stercoralis. The parasite, occurring in dogs, primates and man, is found throughout the moist tropics, as well as in temperate areas where poor sanitation or other factors facilitate the occurrence of faecally transmitted organisms. In some parts of the world, notably Africa and New Guinea, human infections caused by S. fülleborni have been reported. In Africa, the latter is primarily a parasite of primates, but in New Guinea, no animal host is known. S. stercoralis is unique among zoonotic nematodes, in that larvae passing in the faeces can give rise to a free-living generation of worms which, in turn, give rise to infective larvae. This life history alternative (i.e. heterogonic development) acts as an amplification mechanism, increasing the population of infective larvae in the external environment. The infective larvae are active skin penetrators; infection per os , while possible, is probably of limited importance. Because the parasitic female’s eggs hatch internally, a potential for autoinfection exists when precociously developing larvae attain infectivity while still in the host. This is another virtually unique feature of S. stercoralis infections in both its human and animal hosts. Autoinfection can occasionally escape control by the host, with massive re-penetration and larval migration. This can cause pulmonary or cerebro-spinal strongyloidosis as well as fulminant intestinal parasitism. Control of canine strongyloidosis has been achieved in kennels by strategic use of anthelmintics. Given the lack of epidemiological information community-based programs to control human strongyloidosis have not been attempted. The growing importance of human strongyloidosis depends upon the unique ability of S. stercoralis to replicate within its host and to behave as a potentially fatal opportunistic pathogen in immunocompromised hosts, particularly in those receiving corticosteroids.

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49

Lloyd, Sheelagh, and Eric R. Morgan. Toxocarosis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0071.

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Toxocara canis and the syndromes of visceral and ocular larva migrans (VLM, OLM), covert toxocarosis, and neurological toxocarosis are described. Other potential agents, particularly Toxocara cati and Baylisascaris procyonis , are described. The transmission dynamics of toxocarosis to humans have never been fully elucidated, but the potential roles of pet and stray dogs, foxes, cats, and the influence of their population densities, and age demographies, are discussed in relation to contamination of the environment with eggs. Routes of infection with eggs by geophagia, poor hygiene outdoors and with dogs, and fly-borne contamination of food, and meat-borne ingestion of larvae are described. The development of prolonged in vitro culture and analyses of T. canis larval excretions/secretions (TES) and surface antigens helped explain the importance of the rapid production and shedding of TES in the prolonged course of infection and pathogenesis of disease. TES also have greatly improved serodiagnosis. However, we still have insufficient understanding of differences in the aetiology of the larvae or differences in immune responses among individuals to account for development of VLM, covert toxocarosis, or OLM in different individuals. Our understanding of the immunopathological response of the host to TES has emphasized the need for anti-inflammatory therapy in treatment; unfortunately, less information is available on the true efficacy of the anthelmintics available. The complexity of the T. canis life cycle in dogs is described and therapeutic regimens to prevent excretion of eggs by pet dogs are given. This, plus adequate control or exclusion of stray or wild canids from a property could prevent most cases of VLM. Control of infection from free-ranging stray dogs, cats and foxes, will be difficult and more data are needed to clarify the importance of these and of fly-borne and meat-borne transfer of infection to humans for control.

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50

Pozio, Edoardo. Trichinellosis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0068.

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Trichinellosis is caused by nematodes of the genus Trichinella. These zoonotic parasites show a cosmopolitan distribution in all the continents, but Antarctica. They circulate in nature by synanthropic-domestic and sylvatic cycles. Today, eight species and four genotypes are recognized, all of which infect mammals, including humans, one species also infects birds, and two other species infect also reptiles.Parasites of the genus Trichinella are unusual among the other nematodes in that the worm undergoes a complete developmental cycle, from larva to adult to larva, in the body of a single host, which has a profound influence on the epidemiology of trichinellosis. When the cycle is complete, the muscles of the infected animal contain a reservoir of larvae, capable of long-term survival. Humans and other hosts become infected by ingesting muscle tissuescontaining viable larvae.The symptoms associated with trichinellosis vary with the severity of infection, i.e. the number of viable larvae ingested, and the time after infection. The capacity of the worm population to undergo massive multiplication in the body is a major determinant. Progression of disease follows the biological development of the parasite. Symptoms are associated first with the gastrointestinal tract, as the worms invade and establish in the small intestine, become more general as the body responds immunologically, and finally focus on the muscles as the larvae penetrate the muscle cells and develop there. Although Trichinella worms cause pathological changes directly by mechanical damage, most of the clinical features of trichinellosis are immunopathological in origin and can be related to the capacity of the parasite to induce allergic responses.The main source of human infection is raw or under-cooked meat products from pig, wild boar, bear, walrus, and horses, but meat products from other animals have been implicated. In humans, the diagnosis of infection is made by immunological tests or by direct examination of muscle biopsies using microscopy or by recovery of larvae after artificial digestion. Treatment requires both the use of anthelmintic drugs to kill the parasite itself and symptomatic treatment to minimize inflammatory responses.Both pre-slaughter prevention and post-slaughter control can be used to prevent Trichinella infections in animals. The first involves pig management control as well as continuous surveillance programmes. Meat inspection is a successful post-slaughter strategy. However, a continuous consumer education is of great importance in countries where meat inspection is not mandatory.

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