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Relevant bibliographies by topics / COPD

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Academic literature on the topic 'COPD'

Author: Grafiati

Published: 4 June 2021

Last updated: 13 June 2025

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Journal articles on the topic "COPD"

1

Mishima, Michiaki. "Update of COPD." Nihon Naika Gakkai Zasshi 101, no. 6 (2012): 1529–31. http://dx.doi.org/10.2169/naika.101.1529.

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Irusen, E. "COPD sine COPD." South African Respiratory Journal 22, no. 3 (2016): 76. http://dx.doi.org/10.7196/sarj.2016.v22i3.91.

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Shpagina, Lyubov A., Natal’ya V. Kamneva, Ilya Semenovich Shpagin, Olga S. Kotova, Ekaterina V. Anikina, and Dmitrij A. Gerasimenko. "Molecular Markers in Occupational Chronic Obstructive Pulmonary Disease Comorbid with Heart Failure." Annals of the Russian academy of medical sciences 75, no. 5 (2020): 541–51. http://dx.doi.org/10.15690/vramn1381.

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Background.Comorbid heart failure (HF) is common in chronic obstructive pulmonary disease (COPD). Comorbid condition features are studied well in COPD due to tobacco smoke. There is a lack of data about mechanisms, clinical and functional specificity of occupational COPD and HF comorbidity. As occupational COPD and HF share common symptoms and sometimes lung function disorders, there is an unmet need in new markers of HF in occupational COPD.Aims to establish molecular markers associated with occupational COPD with HF comorbidity.Methods.Subjects with occupational COPD were enrolled in a single-center prospective cohort observational study. Comparison group COPD due to tobacco smoke. Then groups were stratified according to HF so the following subgroups were compared: occupational COPD with HF (n=63), occupational COPD without HF (n=52), COPD due to tobacco smoke with HF (n=41), COPD due to tobacco smoke without HF (n=74). Control group healthy people (n=115). Groups were matched by demographics, duration of COPD and HF. CODP was diagnosed according to GOLD 20112020 criteria, HF according to Russian Federal clinical guidelines. Occupational etiological factors were silica dust, organic solvents, metal fumes. Clinical and functional characteristics of CODP and HF were obtained. Serum levels of pulmonary and activation-regulated chemokine (PARC/CCL-18), protein S100, troponin, N terminal pro brain natriuretic peptide (NT-pro-BNP), von Willebrand factor, C-reactive protein were measured by enzyme linked immunosorbent assay, fibrinogen were measured by Clauss method, lactate dehydrogenase, creatine phosphokinase, alanine aminotransferase, aspartate aminotransferase were measured by standard biochemical method. Data are presented as median and interquartile range. Linear regression were used to explore relationships.Results.The molecular specificity of occupational COPD comorbid with HF were the largest increase in serum concentration of PARC-CCL18, NT-pro-BNP, protein S100, troponin, von Willebrand factor and fibrinogen. This factors were associated with length of service. For PARC-CCL18 В=1.1; for NT-pro-BNP В=0.9; for protein S100 В=1.3; for troponin В=0.8, for von Willebrand factor В=1.5 and for fibrinogen В=1.1. Molecular factors also were related to phenotype characteristics of COPD and HF. In multiply regression model the best predictors of comorbidity of CODP and HF were PARC-CCL18 (В=1.1; р=0.002), NT-pro-BNP (В=1.5; р=0.001), protein S100 (В=1.2; р=0.002), troponin (В=0.9; р=0.003). The model was adjusted for gender, age, duration of CODP and HF, FEV1.Conclusions.Occupational CODP comorbid with heart failure is the distinct phenotype. The perspective molecular markers of this phenotype are serum levels of PARC-CCL18, NT-pro-BNP, protein S100, troponin.

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de Marco, Roberto, Simone Accordini, and Alessandro Marcon. "COPD-Lite, COPD-Hard, or COPD-for-What?" American Journal of Respiratory and Critical Care Medicine 184, no. 4 (2011): 486–87. http://dx.doi.org/10.1164/ajrccm.184.4.486a.

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Alqalaf, Sayed Mahmood. "Asthma & COPD." Pharmaceutics and Pharmacology Research 5, no. 8 (2022): 01–05. http://dx.doi.org/10.31579/2693-7247/092.

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Asthma & COPD are the 2 major respiratory diseases that are widespread globally, though big differences between them exist in regard to their pathophysiology and treatment. The main treatment modalities for the two diseases are the inhalation routes of administration. The use of the inhalational routes of administration for the drugs used for the management of asthma and COPD is justified by many advantages such as the low effective dose, faster onset of action and lower systemic side effects. However, some advantages for these routes also exist. The major disadvantage is the technique of use, which many patients do not master even with repeated sessions of teaching by the healthcare providers. Difficulty in applying and following the correct inhalers use was found to be a major reason for patients’ non-adherence and consequently failure of the treatment plan.

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Suzuki, Tomoko, Masaru Yanai, Mutsuo Yamaya, et al. "Erythromycin and Common Cold in COPD." Chest 120, no. 3 (2001): 730–33. http://dx.doi.org/10.1378/chest.120.3.730.

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Lorenz, J., R. Bals, M. Dreher, et al. "Expertentreffen COPD: Exazerbation der COPD." Pneumologie 71, no. 05 (2017): 269–89. http://dx.doi.org/10.1055/s-0043-106559.

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Al Obaidy, Muhammed Waheeb, Adnan M. Aljubouri, and Rana Ehsan. "Noninvasive Prediction of Pulmonary Hypertension in Chronic Obstructive Pulmonary Disease by Echocardiography With & It's Co-Relation With the Disease Severity (Hospital Based Study)." Global Journal of Health Science 10, no. 7 (2018): 50. http://dx.doi.org/10.5539/gjhs.v10n7p50.

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BACKGROUND: Chronic obstructive pulmonary disease (COPD) is one of most common cause of death. Pulmonary hypertension, one of the major and under diagnosed complications of COPD which have a great impact on outcome of COAD and associated with frequent exacerbations and bad prognosis. Echocardiography provides a rapid, noninvasive, portable, and accurate method to evaluate changes related to pulmonary hypertension in COAD.OBJECTIVE OF STUDY: Study pulmonary artery systolic pressure and Tricuspid annular plane systolic excursion in patients with COAD by 2D ECHO Doppler and correlate them with COPD severity.METHOD: Cross sectional study was conducted on 50 COPD patients in Baghdad teaching hospital (age>40 year) from first of January to the end of June, 2017. First, the diagnosis of COPD was confirmed and evaluated for staging by history, clinical examination, and spirometery.All patients have undergone ECG and 2D echocardiography and systolic pulmonary artery pressure and Tricuspid Annular Plane Systolic Excursion (TAPSE) were calculated. Patients with other cardiac or respiratory problems (asthma, pulmonary TB, lung malignancy, connective tissue diseases, interstitial lung disease, and cardiac ischemia, left side heart failure) were excluded from this study.RESULTS: Study of PASP by TR jet with the use of 2D ECHO on 50 patients with COPD showed that 20 patients had normal echo study , mild increase in PASP was found in 15 patients, moderate 11, and severe increase in 4 patients.Study of TAPSE by 2D ECHO showed that 35 patients had normal TAPSE values, while others 15 had abnormal values classified as mild, moderate, and severe: 3, 9, and 3, respectively.A significant associations between echo findings of increasing PASP and abnormal TAPSE, with decrease in FEV1, and oxygen saturation measured by pulse oximeter, duration since COPD was diagnosed and MRC dyspnea scale.CONCLUSION: High incidence of pulmonary hypertension with increasing severity of COAD. Echocardiography is useful and effective tool for detection of PHT secondary to COPD.

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Scholl, Aaron, Istri Ndoja, and Lan Jiang. "Drosophila Trachea as a Novel Model of COPD." International Journal of Molecular Sciences 22, no. 23 (2021): 12730. http://dx.doi.org/10.3390/ijms222312730.

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COPD, a chronic obstructive pulmonary disease, is one of the leading causes of death worldwide. Clinical studies and research in rodent models demonstrated that failure of repair mechanisms to cope with increased ROS and inflammation in the lung leads to COPD. Despite this progress, the molecular mechanisms underlying the development of COPD remain poorly understood, resulting in a lack of effective treatments. Thus, an informative, simple model is highly valued and desired. Recently, the cigarette smoke-induced Drosophila COPD model showed a complex set of pathological phenotypes that resemble those seen in human COPD patients. The Drosophila trachea has been used as a premier model to reveal the mechanisms of tube morphogenesis. The association of these mechanisms to structural changes in COPD can be analyzed by using Drosophila trachea. Additionally, the timeline of structural damage, ROS, and inflammation can be studied in live organisms using fluorescently-tagged proteins. The related function of human COPD genes identified by GWAS can be screened using respective fly homologs. Finally, the Drosophila trachea can be used as a high-throughput drug screening platform to identify novel treatments for COPD. Therefore, Drosophila trachea is an excellent model that is complementary to rodent COPD models.

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Koskela, Heikki O., Hannu O. Tukiainen, and Anna K. Koskela. "Bronchoconstriction due to Cold Weather in COPD." Chest 110, no. 3 (1996): 632–36. http://dx.doi.org/10.1378/chest.110.3.632.

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Dissertations / Theses on the topic "COPD"

1

Hellmann, Sebastian. "COPD und Begleiterkrankungen." Doctoral thesis, Universitätsbibliothek Leipzig, 2011. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-67800.

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Die COPD ist eine Erkrankung mit hoher Prävalenz, die in den nächsten Jahren weiter zunehmen wird. Aufgrund der ausgeprägten klinischen Symptomatik, der hohen Mortalitätsrate und des stetig steigenden Alters in der Bevölkerung wird die COPD aus gesundheits- und sozio-ökonomischen Gründen in den nächsten Jahren immer mehr an Bedeutung gewinnen. Daher ist eine Risikostratifizierung der Erkrankung COPD nicht nur für die Betroffenen von starker Bedeutung. In der Robert-Koch-Klinik, dem Thoraxzentrum des Klinikums St. Georg Leipzig und Lehrkrankenhaus der Universität Leipzig wurden vom 01.01.2002 bis zum 01.03.2009 insgesamt 366 Patienten mit COPD in die Studie aufgenommen, nach international anerkannten Leitlinien in Schweregrade eingeteilt und retrospektiv hinsichtlich ihrer Begleiterkrankungen analysiert. Ziel der Arbeit war eine Evaluation von bedeutsamen Begleiterkrankungen der COPD. Dabei wurden die Häufigkeiten und Arten der Begleiterkrankungen in Zusammenhang mit den Schweregraden der COPD und deren Auswirkungen auf die Hospitalisationsrate untersucht und die Frage beantwortet, ob und wenn ja welche Risikofaktoren für das Erwerben der Begleiterkrankungen eine Rolle spielen. Das vorliegende Kollektiv war im Mittel 62,7±9,6 Jahre alt, bestand zu 64% aus Männern und hatte einen BMI von im Mittel 26,1±5,9. Die Patienten waren überwiegend Raucher oder Ex-Raucher. Als Begleiterkrankungen wurden die KHK mit 20,5%, die arterielle Hypertonie mit 58,5%, die Hyperlipoproteinämie mit 11,7%, der Diabetes Mellitus mit 21,4%, embolische Ereignisse mit 10,7% und die Osteoporose mit 15,3% nachgewiesen. Innerhalb des Beobachtungszeitraumes wurden 75,4% ein weiteres Mal in das Krankenhaus stationär aufgenommen. Nur bei der Begleiterkrankung Hyperlipoproteinämie ließ sich eine statistisch signifikante Assoziation mit den COPD-Schweregraden nachweisen. Bei der Anzahl der Wiederaufnahmen ergab sich ein statistisch signifikanter Zusammenhang mit dem Schweregrad der COPD-Erkrankung. Eine inverse Assoziation konnte zwischen dem BMI und dem ansteigenden COPD-Schweregrad berechnet werden. Das Geschlecht, das CRP und das Raucherverhalten konnten dagegen statistisch nicht mit dem steigenden Schweregrad der Erkrankung assoziiert werden. Im Einklang mit der aktuellen Literatur ließ sich in unseren Daten eine hohe Prävalenz bei Erkrankten mit COPD hinsichtlich der Begleiterkrankungen KHK, arterielle Hypertonie, Diabetes mellitus und Osteoporose nachweisen. Es ließ sich kein erhöhtes Auftreten der Anzahl von embolischen Ereignissen bei COPD-Erkrankten feststellen. Nur bei der Osteoporose konnte eine ansteigende Prävalenzhäufigkeit in den COPD-Stadien erkannt werden, die allerdings nicht statistisch signifikant war. Die Anzahl der stationären Wiederaufnahmen stiegen dagegen statistisch signifikant analog zum Schweregrad der COPD-Erkrankung an, während sich der Schweregrad der COPD invers zum BMI verhielt. In einer zusammenfassenden Bewertung konnte diese Arbeit den Zusammenhang zwischen der COPD und der Häufigkeit der aufgeführten Begleiterkrankungen zeigen. Es wurde dokumentiert, dass Patienten mit COPD für die untersuchten Begleiterkrankungen eine starke Prädisposition haben. Die Osteoporose, der BMI und die Hospitalisationsrate scheinen mit dem Schweregrad der COPD zusammenzuhängen, so dass insbesondere hinsichtlich dieser genannten Faktoren ein systematisches Screening erfolgen sollte. Die Erkenntnis der engen Beziehung der Begleiterkrankungen, insbesondere die mit dem Schweregrad zunehmende Prävalenz der Osteoporose, spielt in der Prävention COPD-Erkrankter und für deren Prognose eine große Rolle, da durch frühe Therapie Sekundärschäden verhindert werden könnten. Dies Arbeit zeigt, dass die COPD, unter Berücksichtigung der steigenden Morbidität der Grunderkrankung und der mit ihr verbundenen Begleiterkrankungen sowie der steigenden sozioökonomischen Kosten durch vermehrte Krankenhausaufenthalte, in Zukunft ein zentrales Thema in der Medizin werden wird und verdeutlicht die Forderung nach weiterer Forschung auf diesem Gebiet.

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Stone, Helen Marie. "Proteomics in COPD." Thesis, University of Birmingham, 2017. http://etheses.bham.ac.uk//id/eprint/7565/.

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In alpha-1-antitrypsin deficiency (A1ATD) there is excess neutrophil elastase activity, resulting in proteolytic destruction of the lung parenchyma. I hypothesised that the peptide fragments of proteins present in the lung might be detectable in plasma by mass spectrometry and that they might be useful biomarkers of disease activity and treatment efficacy. Calcium ionophore, neutrophil elastase and proteinase 3 were added to plasma from patients with A1ATD to create an in vitro model of the destructive processes. MALDI-based peptide profiling of plasma from patients pre and post treatment with intravenous A1AT was undertaken and MS/MS performed to identify differences. Plasma was also depleted of abundant plasma proteins, labelled with isobaric tags and analysed by shotgun proteomics. The readily detectible components of the plasma proteome remained unchanged with intravenous A1AT. Addition of ionophore, elastase and proteinase 3 to patient blood generated predominantly fragments of fibrinogen. In patients treated with intravenous A1AT, fragments of A1AT increased significantly with treatment: - 2 of these were fragments of a short C-terminal segment of the A1AT protein and were also present in healthy subjects. The shotgun experiments did not identify any robust biomarkers and illustrate the challenging nature of plasma proteomics.

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Barius, Per. "COPD och Uppdragstaktik." Thesis, Försvarshögskolan, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:fhs:diva-2542.

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Nato:s planeringsmodell Comprehensive Operations Planning Directive ska framgent användas för nationell militär planering. Anpassning till svenska förhållanden kommer att genomföras. Överbefälhavaren betonar redan i inledningen på ny Militärstrategisk doktrin att uppdragstaktik ska vara grunden för ledning i Försvarsmakten. Forskningen i uppsatsen fokuserar till mötet mellan planeringsmetod och betoning av att uppdragstaktik ska vara grunden för ledning. Syftet är att identifiera om operativ planering enligt COPD och doktrinens betoning av uppdragstaktik harmonierar. Genom det kan uppsatsen bidra med ny kunskap inom området och med ett analysverktyg för uppdragstaktik. För att genomföra analys av COPD har ett analysverktyg skapats, baserat på olika forskares teorier om vad som stödjer och motverkar uppdragstaktik. Forskningen visar att COPD i hög grad stödjer uppdragstaktik i ett filosofiskt perspektiv, men i lägre grad i praktiken. Svensk anpassning av COPD kräver ett ställningstagande om uppdragstaktik ska ses som en ledningsmetod eller filosofi.

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Liesker, Jeroen Johannes Wilhelmus. "COPD a multifaceted disease /." [S.l. : [Groningen : s.n.] ; University Library Groningen] [Host], 2008. http://irs.ub.rug.nl/ppn/.

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Raste, Yogini. "Physical activity in COPD." Thesis, Imperial College London, 2016. http://hdl.handle.net/10044/1/55133.

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Reduced physical activity is an important feature of Chronic Obstructive Pulmonary Disease (COPD). This thesis explores the importance of technical, environmental and patient factors in physical activity in COPD. Activity monitors are integral in accurately measuring physical activity in COPD but, to date, commercially available monitors had not been validated in this patient group. The first study (Chapter 3) was a multicentre validation study of 6 physical activity monitors in a laboratory setting in 40 COPD patients, against a gold standard of indirect calorimetry from a portable metabolic kit. Three triaxial accelerometers were found to be the most accurate activity monitors Chapter 4 describes a validation study of 4 activity monitors in a domestic setting in 20 UK-based patients. The gold standard of indirect calorimetry from the doubly labelled water method was used. Chapter 5 describes a multicentre longitudinal study of 236 COPD patients. Physical activity was measured at 3 different time points over 12 months with 2 triaxial monitors to assess the effect of time, geographic location and climatic conditions on physical activity. There was a significant effect of time with a decline in physical activity over time. There was also a significant effect of temperature and day length with lower levels of activity associated with lower temperatures and shorter day length. This has implications for future studies in COPD using physical activity as an outcome measure. A decline in physical activity over time across several European centres supports the importance of physical activity in the course of COPD and early intervention to attenuate its decline. The breathing response to the initiation of exercise in COPD and its association with daily physical activity levels was investigated (Chapter 6). A rapid shallow pattern of breathing on exertion is commonly found in COPD, which worsens with disease severity. However, the hypothesis that this breathing pattern would be associated with lower levels of daily physical activity was not proven.

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Dewhurst, Jennifer. "Macrophage subpopulations in COPD." Thesis, University of Manchester, 2015. https://www.research.manchester.ac.uk/portal/en/theses/macrophage-subpopulations-in-copd(e0e66fa4-ddff-4591-b785-edee74918539).html.

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Lung macrophage numbers are increased in Chronic Obstructive Pulmonary Disease (COPD). Macrophage subpopulations with differences in cell surface marker expression and function have been identified in sputum. The aim of this thesis was to investigate the role of lower airway macrophage subpopulations in COPD, focussing on their phenotype and function. Alveolar and interstitial cells from human lung resection tissue were isolated by perfusing the airways with saline. Interstitial cells were isolated by mechanically and enzymatically digesting perfused lung tissue. Alveolar and interstitial macrophages were negatively selected using a monocyte enrichment kit. Flow cytometry and PCR were used to identify and phenotype macrophage subpopulations. A flow cytometry based phagocytosis assay was used to characterise the function of macrophage subpopulations. Subpopulations of alveolar macrophages and interstitial macrophages were identified as “small macrophages” and “large macrophages” based on size and granularity. Small macrophages were further characterised as HLA-DRhigh, CD14high, CD38high, CD36high, CD206low. Large macrophages were characterised as HLA-DRlow, CD14low, CD38low, CD36low, CD206high. The percentage of large macrophages was reduced in the interstitium compared to alveolar macrophages, while the opposite pattern was observed for small macrophages (increased percentage in interstitium). No significant differences between the proportion of macrophage subpopulation from COPD patients and smokers have been observed. Large interstitial macrophages were 66% apoptotic. Small interstitial macrophages had high inflammatory potential but low phagocytic ability; small alveolar macrophages had intermediate inflammatory potential but high phagocytic ability; large alveolar macrophages had low inflammatory potential and low phagocytic ability. In addition the phenotype and function of large macrophages from COPD patients was sensitive to current smoking. I have shown phenotypically different subpopulations of alveolar and interstitial macrophages exist. The proportion of small and large macrophages varies between the interstitium and alveolar space. Small macrophages appear to polarised towards an inflammatory phenotype compared to large macrophages. Therefore targeting macrophages for COPD therapy should be tailored towards specific subpopulations.

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Lee, Jeeyoung. "Accelerated aging in COPD : the relationship of telomere length and mortality in COPD." Thesis, University of British Columbia, 2011. http://hdl.handle.net/2429/38486.

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The evidence for the role of accelerated aging in COPD progression is growing and the senescence hypothesis is one possible molecular pathway by which COPD develops. Telomeres are used as a biomarker of cellular aging, and cellular aging is accelerated by the presence of oxidative stress and inflammation. Previous studies have shown that telomeres of peripheral blood cells are significantly shorter in COPD patients, but no studies to date explored the relationship of telomere length to important health outcomes such as mortality. Using samples from Lung Health Study (LHS), we examined the role of telomere length and polymorphisms in genes involved in aging process in health outcomes in COPD patients. There were no significant differences in age, sex, BMI, race of cumulative smoking exposure (pack-years) among 4 groups, divided on basis of telomere length. However, the risk of all cause mortality was similar across the first 3 quartiles (short telomere) but dropped significantly in the 4th quartile (longest telomere, hazard ratio (HR), 1.30). Compared to individuals in the 4th quartile of relative telomere length, the remaining participants had significantly higher risk of cancer mortality (HR, 1.48). Smoking status did not make a significant difference in leukocyte telomere length but when compared to non-COPD, age matched control group, all smoker groups in LHS had shorter telomeres. We also investigated the role of SNPs that were previously associated with leukocyte telomere length in disease outcome. Although no SNPs were associated with leukocyte telomere length, several SNPs of telomere biology genes were associated with cardiovascular and lung cancer mortality. The rate of telomere attrition is influenced by both extrinsic factors, such as inflammation and oxidative stress and intrinsic factors such as genetic predisposition. Here, we have shown that accelerated aging of peripheral blood cells, indicated by short telomeres, seems to play a role in disease outcome of COPD. Although we failed to show significant associations between leukocyte telomere length and genetic polymorphisms of telomere regulatory genes, the SNPs may contribute to risk of mortality. Further research is needed to elucidate the pathways underlying these observations.

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Hackx, Maxime. "CT Airways Measurements in COPD." Doctoral thesis, Universite Libre de Bruxelles, 2017. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/248677.

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La bronchopneumopathie chronique obstructive (BPCO) est une maladie essentiellement liée au tabagisme, caractérisée par une limitation du débit aérien secondaire à deux processus histologiques : l’emphysème pulmonaire (destruction du tissu alvéolaire) et l’atteinte des voies aériennes (remodelage pariétal et rétrécissement luminal). C’est une maladie hétérogène dans laquelle deux patients avec une même limitation du débit aérien peuvent avoir des proportions différentes d’emphysème pulmonaire et d’atteinte des voies aériennes. Le phénotypage par tomodensitométrie (TDM) de ces deux processus est cliniquement important car il permet de sélectionner et suivre certains traitements, et d’identifier des patients à risques accrus de morbidité et de mortalité. Cependant, alors que les mesures TDM d’emphysème pulmonaire sont bien établies, de nombreuses questions restent en suspens concernant les mesures TDM de l’atteinte des voies aériennes, constituant un frein à l’intégration de la TDM dans le traitement de la BPCO. Notre travail a consisté en quatre études originales investiguant quatre mesures TDM de voies aériennes de 3ème et 4éme générations, corrélées aux mesures histologiques des voies aériennes distales et aux mesures spirométriques. Une première étude a testé l’effet des exacerbations de BPCO sur ces mesures. Une deuxième étude a testé l’effet de la bronchodilatation sur ces mesures et a permis de calculer le nombre de voies aériennes à mesurer. Une troisième étude a comparé des variabilités de ces mesures avec celles des mesures spirométriques. Enfin, une quatrième étude a testé les effets de la capacité pulmonaire totale, du genre et de la taille sur ces mesures. Au terme de ces investigations, nous avons proposés deux mesures TDM appropriées pour mesurer les voies aériennes chez le patient BPCO: l’épaisseur pariétale des voies aériennes de 3ème génération et la racine carrée de la surface pariétale à un périmètre interne de 10 mm.<br>Doctorat en Sciences médicales (Médecine)<br>info:eu-repo/semantics/nonPublished

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Bathoorn, Derk. "COPD exacerbations, inflammation and treatment." [S.l. : Groningen : s.n. ; University Library of Groningen] [Host], 2007. http://irs.ub.rug.nl/ppn/304982296.

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Sheikh, Omer, MohD Ibrahim, Joseph Maguire, et al. "COPD exacerbation induced Takotsubo Cardiomyopathy." Digital Commons @ East Tennessee State University, 2019. https://dc.etsu.edu/asrf/2019/schedule/75.

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Introduction: Takotsubo cardiomyopathy or stress cardiomyopathy is a syndrome of transient left ventricular (LV) dysfunction mimicking myocardial infarction, but lacking obstruction of coronary artery disease (CAD) or acute plaque rupture. A characteristic differentiation from CAD is that regional motional abnormality extends beyond a territory perfused with a single epicardial coronary artery. Clinically, it is characterized by apical ballooning of the LV due to due to depression of mid and apical segments, with hyperkinesis of cardiac basal walls. Women are affected more than men, predominantly in the postmenopausal age. Case Report: A 54-year-old Caucasian female with a history of COPD, hypertension, uncontrolled diabetes mellitus, hyperlipidemia, depression and ongoing tobacco use presented with complaints of worsening shortness of breath two days prior to admission. She denied chest pain, worsened cough, palpitations, nausea or vomiting. On examination, she was in distress and anxious, with labored breathing. Upon examining the chest, decreased air entry was present in both lung fields with bibasilar wheezing. Initial lab tests showed mild respiratory acidosis, with pH of 7.24, pCO2 of 47.4 and pO2 of 65. Troponins on the day of admission was Soon after admission, she started complaining of severe right neck pain. Repeat EKG revealed localized lateral J point, anteroseptal q waves and 4mm ST-segment elevation in leads V3 and V4 reciprocal changes and without chest pain. Repeat troponins were slightly elevated to 0.42 ng/ml and CK-MB was elevated to 20.2 ng/ml. A transthoracic echocardiogram showed regional abnormalities in left ventricle with the apex, mid to distal septum and the anterior part of septum was akinetic. Discussion: Takotsubo cardiomyopathy presents in 1 to 2 percent of troponin-positive acute coronary syndrome (ACS) with various clinical manifestations and various outcomes. Some patients have favorable outcomes based on their clinical performance and extent of cardiac muscle involvement. As in the case we presented, this syndrome can be entirely idiopathic, without a definitive underlying cause. Supportive management while hospitalized and early identification of complications improve the prognosis.

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Books on the topic "COPD"

1

Andreas, Stefan, and Henrik Watz, eds. COPD. De Gruyter, 2018. http://dx.doi.org/10.1515/9783110494341.

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Hanania, Nicola A., and Amir Sharafkhaneh, eds. COPD. Humana Press, 2011. http://dx.doi.org/10.1007/978-1-59745-357-8.

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Lee, Sang-Do, ed. COPD. Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-47178-4.

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Heleen, Silvis, ed. COPD. Veltman Uitgevers, 2007.

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Russell, Richard EK, Paul A. Ford, Peter J. Barnes, and Sarah Russell. Managing COPD. Springer Healthcare Ltd., 2013. http://dx.doi.org/10.1007/978-1-907673-52-8.

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Russell, Richard, Paul Ford, and Peter Barnes. Managing COPD. Springer Healthcare UK, 2011. http://dx.doi.org/10.1007/978-1-908517-61-6.

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Liu, Weisheng. COPD & asthma. People's Medical Publishing House, 2007.

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8

Felner, Kevin. COPD for dummies. Wiley Pub., 2008.

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van Schayck, C. P., and G. Wesseling. Leven met COPD. Bohn Stafleu van Loghum, 2010. http://dx.doi.org/10.1007/978-90-313-7580-6.

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Chavannes, N. H., and J. W. M. Muris. Het COPD Formularium. Bohn Stafleu van Loghum, 2011. http://dx.doi.org/10.1007/978-90-313-8829-5.

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Book chapters on the topic "COPD"

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Wouters, Eveline, Teatske van der Zijpp, and Marianne Nieboer. "COPD." In (B)eHealth. Bohn Stafleu van Loghum, 2017. http://dx.doi.org/10.1007/978-90-368-1073-9_3.

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Troosters, T., V. Barbier, and W. Janssens. "COPD." In Geriatrie in de fysiotherapie en kinesitherapie. Bohn Stafleu van Loghum, 2017. http://dx.doi.org/10.1007/978-90-368-1350-1_12.

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Kalmar, Jayne M., Brigid M. Lynch, Christine M. Friedenreich, et al. "COPD." In Encyclopedia of Exercise Medicine in Health and Disease. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_2256.

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Franssen, F. M. E., and R. Posthuma. "COPD." In Inleiding in de gerontologie en geriatrie. Bohn Stafleu van Loghum, 2020. http://dx.doi.org/10.1007/978-90-368-2453-8_10.

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Davies, Eryl. "Copd." In The Final FFICM Structured Oral Examination Study Guide. CRC Press, 2022. http://dx.doi.org/10.1201/9781003243694-136.

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Bell, Aaron E. "COPD." In Mechanical Ventilation in Critical Care Transport. Springer Nature Switzerland, 2024. http://dx.doi.org/10.1007/978-3-031-66782-4_13.

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Franssen, F. M. E., and M. A. Spruit. "COPD." In Kernboek. Bohn Stafleu van Loghum, 2015. http://dx.doi.org/10.1007/978-90-368-0444-8_6.

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van Gunst, S. G., and V. G. Pigmans. "COPD." In NHG-standaarden voor de praktijkassistente 2014. Bohn Stafleu van Loghum, 2013. http://dx.doi.org/10.1007/978-90-368-0485-1_19.

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van Gunst, S. G., V. G. Pigmans, and C. J. in ’t Veld. "COPD." In NHG Standaarden voor praktijkassistente en -ondersteuner 2009. Bohn Stafleu van Loghum, 2009. http://dx.doi.org/10.1007/978-90-313-6916-4_19.

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van Gunst, S. G., and V. G. Pigmans. "COPD." In NHG-Standaarden voor praktijkassistente 2010. Bohn Stafleu van Loghum, 2010. http://dx.doi.org/10.1007/978-90-313-7755-8_20.

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Conference papers on the topic "COPD"

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Muñoz Ramirez, Isabel, Alfonso Marín Andreu, Carolina España Domínguez, María Morales González, Eva Vázquez Gandullo, and Aurelio Arnedillo Muñoz. "Descriptive analysis of patients admitted due to COPD exacerbation (COPDE) and COPD-pneumonia." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.2442.

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Kaur, Amarpreet, Jacqueline Creasey, Rumina Mirza, Neelya Kumar, Kanwar Pannu, and Dipak Mukherjee. "COPD mortality - Is it all COPD?" In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa1054.

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Villamizar, J. P., A. De Diego, M. D. Schweitzer, M. Aboubkar, and S. Kosseifi. "COPD Care Program: Reducing COPD Readmissions." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a1698.

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Quint, J., D. Alber, J. Goldring, G. Donaldson, and J. Wedzicha. "IP10: A Biomarker for Cold Associated COPD Exacerbations." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a5349.

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Guillien, Alicia, Marc Puyraveau, Thibaud Soumagne, et al. "COPD or not COPD: Answer in FEV1." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa5034.

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Byun, Min Kwang, Hye Jung Park, Hyung Jung Kim, Chul Min Ahn, Kwang Ha Yoo, and Ki Suck Jung. "Asthma-COPD overlap syndrome shows favorable prognosis compared to pure COPD in Korean COPD cohort." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa4011.

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Boussoffara, Leila, Imen Touil, Soumaya Bouchareb, Nadia Boudawara, Amani Msakni, and Jalel Knani. "Features of COPD in correlation with COPD assessment test." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa2693.

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van Nijnatten, Jos, Alen Faiz, Cornelis Joseph Vermeulen, et al. "Differential DNA Methylation Associated with COPD and COPD Severity." In ERS International Congress 2023 abstracts. European Respiratory Society, 2023. http://dx.doi.org/10.1183/13993003.congress-2023.pa5209.

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Keene, Spencer, Annmariek Driessen, Rachel Jordan, et al. "Smoking and BMI in COPD and non-COPD patients." In ERS International Congress 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/13993003.congress-2021.pa2442.

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Ellis, Paul, Alice Turner, and Michael Newnham. "Prevalence of cardiovascular disease in COPD and AATD-COPD." In ERS Congress 2024 abstracts. European Respiratory Society, 2024. http://dx.doi.org/10.1183/13993003.congress-2024.pa3024.

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Reports on the topic "COPD"

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Liang, R., D. Liu, HB Li, and ZG Zhai. The efficacy and safety of traditional Chinese medicine formulas in the treatment of chronic obstructive pulmonary disease complicated with pulmonary hypertension: a systematic review and meta-analysis study. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.10.0041.

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Review question / Objective: This systematic review and meta-analysis was intended to evaluate the efficacy and safety of traditional Chinese medicine(TCM) formulas in the treatment of chronic obstructive pulmonary disease(COPD) complicated with pulmonaryhypertension (PH). Condition being studied: Chronic obstructive pulmonary disease(COPD) complicated with pulmonary hypertension(PH) is classified as the third group PH.According to epidemiology, the most common cause of PH associated with lung diseases and/or hypoxia is COPD, but the prevalence rate of COPD with PH range from 20% to 91% variously. In China, many TCM formulas are regularly used in COPD patients , thus TCM formulas therapy is worth considering.

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Liu, Lu, Wenchuan Qi, Qian Zeng, et al. Does acupuncture improve lung function in chronic obstructive pulmonary disease animal model?: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.3.0104.

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Review question / Objective: Chronic obstructive pulmonary disease (COPD) is a common, preventable, and treatable disease characterized by persistent respiratory symptoms and progressive airflow obstruction documented on spirometry. Acupuncture, as a safe and economical non-pharmacology therapy, has pronounced therapeutic effects in COPD patients. Several systematic reviews draw the conclusion that acupuncture could improve patients’ quality of life, exercise capacity and dyspnoea, however, the results about lung function were inconclusive. Recently, increasing number of animal studies has been published to illustrate the effects of acupuncture in improving lung function in COPD animal model. However, the efficacy of acupuncture for experimentally induced COPD have not been systematically investigated yet. A systematic review of animal experiments can benefit future experimental designs, promote the conduct and report of basic researches and provide some guidance to translate the achievements of basic researches to clinical application in acupuncture for COPD. Therefore, we will conduct this systematic review and meta-analysis to evaluate effects of acupuncture on COPD animal model.

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tian, hao. Acupuncture treatment for COPD: a trial sequential meta‑analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.12.0007.

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Ellerbeck, Edward, Kimber Richter, Nicole Nollen, and Milind Phadnis. Comparing Two Ways to Help People with COPD Stop Smoking. Patient-Centered Outcomes Research Institute® (PCORI), 2023. http://dx.doi.org/10.25302/12.2018.cer.130602901.

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Tachkov, Konstantin, Maria Dimitrova, Konstantin Mitov, and Alexandra Savova. Assessment of the Risk Factors for COPD Development – a Methodological Approach. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, 2019. http://dx.doi.org/10.7546/crabs.2019.06.15.

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Huang, Houqiang, Min Huang, Qi Chen, Mark Hayter, and Roger Hayter. Health-related Serious Games on the Rehabilitation for Patients with COPD: Systematic Review Protocol. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.12.0062.

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Review question / Objective: The aim of this systematic review is to identify effectiveness and patients’ demand on serious games for COPD patients as well as to recognize potential research gaps in this area by synthesizing and appraising studies examining effects of serious games on COPD patients. Eligibility criteria: OutcomesThe outcomes that include health-related endpoints such as pulmonary function, exercise capacity, dyspnea, compliance, or adverse effects, will be enrolled.Further inclusion criteriaStudies must be peer-reviewed and be in English or Chinese.Exclusion criteriaStudies will be excluded for the following reasons: (1) duplicate records;, (2) studies focused on measurement; diagnostic methods, serious game theory or game development; and (3) conference abstracts or studies that cannot find out full texts.

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Czerwaty, Katarzyna, Karolina Dżaman, Krystyna Maria Sobczyk, and Katarzyna Irmina Sikrorska. The Overlap Syndrome of Obstructive Sleep Apnea and Chronic Obstructive Pulmonary Disease: A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.11.0077.

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Review question / Objective: To provide the essential findings in the field of overlap syndrome of chronic obstructive pulmonary disease and obstructive sleep apnea, including prevalence, possible predictors, association with clinical outcomes, and severity compared to both chronic obstructive pulmonary disease and obstructive sleep apnea patients. Condition being studied: OSA is characterized by complete cessation (apnea) or significant decrease (hy-popnea) in airflow during sleep and recurrent episodes of upper airway collapse cause it during sleep leading to nocturnal oxyhemoglobin desaturations and arousals from rest. The recurrent arousals which occur in OSA lead to neurocognitive consequences, daytime sleepiness, and reduced quality of life. Because of apneas and hypopneas, patients are experiencing hypoxemia and hypercapnia, which result in increasing levels of catecholamine, oxidative stress, and low-grade inflammation that lead to the appearance of cardio-metabolic consequences of OSA. COPD is a chronic inflammatory lung disease defined by persistent, usually pro-gressive AFL (airflow limitation). Changes in lung mechanics lead to the main clini-cal manifestations of dyspnea, cough, and chronic expectoration. Furthermore, patients with COPD often suffer from anxiety and depression also, the risk of OSA and insomnia is higher than those hospitalized for other reasons. Although COPD is twice as rare as asthma but is the cause of death eight times more often.

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Ly, Lena, Jennifer Philip, Peter Hudson, and Natasha Smallwood. Singing for people with advance chronic respiratory diseases: a qualitative meta-synthesis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.8.0017.

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Review question / Objective: This study undertook a meta-synthesis of qualitative data with the aim of collating, synthesizing, and evaluating the current evidence regarding the experiences of singing for people with advanced chronic respiratory disease. Condition being studied: Advanced respiratory illnesses are disorders that impact the airways and other structures of the lung. People with lung cancer, chronic obstructive pulmonary disease (COPD) and interstitial lung disease (ILD) frequently experience progressive, frightening breathlessness, cough and fatigue, which affect their quality of life. Furthermore, people with advanced chronic respiratory disease (CRD) and their carers experience a high prevalence of loneliness and uncertainty, especially if breathlessness is felt to herald death and thus, require both psychological and practical supportive care to cope with their symptoms.

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Aboumatar, Hanan, Mohammad Naqibuddin, Suna Chung, et al. Helping Patients with COPD Transition from Hospital to Home—The BREATHE Study. Patient-Centered Outcomes Research Institute (PCORI), 2020. http://dx.doi.org/10.25302/04.2020.ih.13047118.

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Chen, Li, and Jing Su. Effect of auricular point pressing therapy on rehabilitation in patients with COPD. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.4.0051.

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