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Journal articles on the topic 'Cornification'

Author: Grafiati

Published: 4 June 2021

Last updated: 18 February 2022

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1

Goldsmith, Lowell A., David H. Stein, and A. Elise Weinrich. "Cornification diseases." Journal of the American Academy of Dermatology 14, no. 1 (1986): 118–21. http://dx.doi.org/10.1016/s0190-9622(86)80382-6.

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2

Eckhart, Leopold, Saskia Lippens, Erwin Tschachler, and Wim Declercq. "Cell death by cornification." Biochimica et Biophysica Acta (BBA) - Molecular Cell Research 1833, no. 12 (2013): 3471–80. http://dx.doi.org/10.1016/j.bbamcr.2013.06.010.

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3

Alibardi, Lorenzo, Mattia Toni, and Luisa Dalla Valle. "Hard cornification in reptilian epidermis in comparison to cornification in mammalian epidermis." Experimental Dermatology 16, no. 12 (2007): 961–76. http://dx.doi.org/10.1111/j.1600-0625.2007.00609.x.

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4

Hood, David M. "Cornification of the hoof capsule." Journal of Equine Veterinary Science 33, no. 10 (2013): 856–57. http://dx.doi.org/10.1016/j.jevs.2013.08.025.

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5

Ammirati, Christie T., and Susan Bayliss Mallory. "THE MAJOR INHERITED DISORDERS OF CORNIFICATION." Dermatologic Clinics 16, no. 3 (1998): 497–508. http://dx.doi.org/10.1016/s0733-8635(05)70248-5.

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6

Williams, Mary L., and Peter M. Elias. "Genetically Transmitted, Generalized Disorders of Cornification." Dermatologic Clinics 5, no. 1 (1987): 155–78. http://dx.doi.org/10.1016/s0733-8635(18)30772-1.

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7

Resnik, Kenneth S., and Mario DiLeonardo. "Incidental Granular Parakeratotic Cornification in Carcinomas." American Journal of Dermatopathology 29, no. 3 (2007): 264–69. http://dx.doi.org/10.1097/dad.0b013e3180465860.

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8

Martin, L., and LH Crane. "Is the antiprogestin RU486 luteolytic in rats?" Reproduction, Fertility and Development 3, no. 5 (1991): 615. http://dx.doi.org/10.1071/rd9910615.

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Subcutaneous injections of RU486 on Day 1, Day 4 or Days 3 and 4 of pregnancy in rats induced abortion of the embryo and transient vaginal cornification. Nevertheless, most corpora lutea appeared to be functional at autopsy on Day 12. The same treatments in pseudopregnant rats also produced transient vaginal cornification, but did not terminate pseudopregnancy. This suggests that transient antagonism of progesterone by RU486 does not terminate function of the corpus luteum in rats and that positive feedback by progesterone is not essential for continuing luteal function in rats.

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9

Copic, Dragan, Maria Laggner, Polina Kalinina, Katharina Klas, Erwin Tschachler, and Michael Mildner. "Experimental Models for the Study of Hereditary Cornification Defects." Biomedicines 9, no. 3 (2021): 238. http://dx.doi.org/10.3390/biomedicines9030238.

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Ichthyoses comprise a broad spectrum of keratinization disorders due to hereditary defects of cornification. Until now, mutations in more than 50 genes, mostly coding for structural proteins involved in epidermal barrier formation, have been identified as causes for different types of these keratinization disorders. However, due to the high heterogeneity and difficulties in the establishment of valid experimental models, research in this field remains challenging and translation of novel findings to clinical practice is difficult. In this review, we provide an overview of existing models to st

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10

Alibardi, Lorenzo. "Review: cornification, morphogenesis and evolution of feathers." Protoplasma 254, no. 3 (2016): 1259–81. http://dx.doi.org/10.1007/s00709-016-1019-2.

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11

Williams, Mary L., and Peter M. Elias. "Enlightened therapy of the disorders of cornification." Clinics in Dermatology 21, no. 4 (2003): 269–73. http://dx.doi.org/10.1016/s0738-081x(03)00059-2.

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12

Schmuth, Matthias, Verena Martinz, Andreas R. Janecke, et al. "Inherited ichthyoses/generalized Mendelian disorders of cornification." European Journal of Human Genetics 21, no. 2 (2012): 123–33. http://dx.doi.org/10.1038/ejhg.2012.121.

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13

Itin, Peter H., Michael Moschopulos, and Gabriela Richard. "Reticular erythrokeratoderma: A new disorder of cornification." American Journal of Medical Genetics 120A, no. 2 (2003): 237–40. http://dx.doi.org/10.1002/ajmg.a.20036.

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14

Lippens, S., G. Denecker, P. Ovaere, P. Vandenabeele, and W. Declercq. "Death penalty for keratinocytes: apoptosis versus cornification." Cell Death & Differentiation 12, S2 (2005): 1497–508. http://dx.doi.org/10.1038/sj.cdd.4401722.

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15

Wruck, Christoph J., Anja Wruck, Lars-Ove Brandenburg, Mamed Kadyrov, Mersedeh Tohidnezhad, and Thomas Pufe. "Impact of Nrf2 on esophagus epithelium cornification." International Journal of Dermatology 50, no. 11 (2011): 1362–65. http://dx.doi.org/10.1111/j.1365-4632.2011.04989.x.

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16

Mauldin, Elizabeth A. "Canine Ichthyosis and Related Disorders of Cornification." Veterinary Clinics of North America: Small Animal Practice 43, no. 1 (2013): 89–97. http://dx.doi.org/10.1016/j.cvsm.2012.09.005.

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17

Fisher, C., M. R. Byers, M. J. Iadarola, and E. A. Powers. "Patterns of epithelial expression of Fos protein suggest important role in the transition from viable to cornified cell during keratinization." Development 111, no. 2 (1991): 253–58. http://dx.doi.org/10.1242/dev.111.2.253.

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An antibody directed against the DNA-binding region of c-fos was used to localize the distribution of cells positive for Fos protein in epithelial tissues. The antibody consistently bound to the nuclei of epithelial cells in the late stages of differentiation, just prior to cornification. The epidermis, palate, buccal mucosa, gingiva, tongue, forestomach and vagina in estrus all produced this type of labelling, suggesting a burst of expression immediately before cell death and cornification. The differentiating cells of the hair follicle, including the hair and inner root sheath, were also lab

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18

ISSEROFF, R. R., K. T. CHUN, and R. M. ROSENBERG. "Triiodothyronine alters the cornification of cultured human keratinocytes." British Journal of Dermatology 120, no. 4 (1989): 503–10. http://dx.doi.org/10.1111/j.1365-2133.1989.tb01323.x.

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19

Demerjian, Marianne, Jean-Pierre Hachem, Erwin Tschachler, et al. "Acute Modulations in Permeability Barrier Function Regulate Epidermal Cornification." American Journal of Pathology 172, no. 1 (2008): 86–97. http://dx.doi.org/10.2353/ajpath.2008.070161.

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20

Corrales, Rosa Maria, Cintia Sade de Paiva, De-Quan Li, et al. "Entrapment of Conjunctival Goblet Cells by Desiccation-Induced Cornification." Investigative Opthalmology & Visual Science 52, no. 6 (2011): 3492. http://dx.doi.org/10.1167/iovs.10-5782.

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21

Alibardi, Lorenzo, and Mattia Toni. "Immunolocalization and characterization of cornification proteins in snake epidermis." Anatomical Record Part A: Discoveries in Molecular, Cellular, and Evolutionary Biology 282A, no. 2 (2005): 138–46. http://dx.doi.org/10.1002/ar.a.20153.

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22

Mack, Judith A., Sanjay Anand, and Edward V. Maytin. "Proliferation and cornification during development of the mammalian epidermis." Birth Defects Research Part C: Embryo Today: Reviews 75, no. 4 (2005): 314–29. http://dx.doi.org/10.1002/bdrc.20055.

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23

Alibardi, L., and M. Toni. "Immuno-Cross reactivity of transglutaminase and cornification marker proteins in the epidermis of vertebrates suggests common processes of soft cornification across species." Journal of Experimental Zoology 302B, no. 6 (2004): 526–49. http://dx.doi.org/10.1002/jez.b.21016.

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24

Devaraj, Yogesh, Ranga Swaroop Mukunda, Rashmi R. Mallya, Taranpreet Kaur Kalra, and Shaila Shree Devendra. "Erythrokeratoderma variabilis: a case report." International Journal of Research in Dermatology 7, no. 4 (2021): 581. http://dx.doi.org/10.18203/issn.2455-4529.intjresdermatol20212558.

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<p>Erythrokeratodermia variabilis (EKV) was first described by Mendes da Costa. It is a rare heterogeneous group of inherited cornification disorders characterized by two distinct types of skin lesions: fixed hyperkeratotic plaques and sharply marginated, pruritic, migratory erythematous lesions. We report a case of EKV in a 44-year-old male patient.</p>

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25

Ladd, Patricia A., Liping Du, Jorge H. Capdevila, Raymond Mernaugh, and Diane S. Keeney. "Epoxyeicosatrienoic Acids Activate Transglutaminasesin Situand Induce Cornification of Epidermal Keratinocytes." Journal of Biological Chemistry 278, no. 37 (2003): 35184–92. http://dx.doi.org/10.1074/jbc.m301666200.

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26

Alibardi, Lorenzo, and Mattia Toni. "Localization and Characterization of Specific Cornification Proteins in Avian Epidermis." Cells Tissues Organs 178, no. 4 (2004): 204–15. http://dx.doi.org/10.1159/000083732.

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27

Kiritsi, Dimitra, Franziska Schauer, Ute Wölfle, et al. "Targeting epidermal lipids for treatment of Mendelian disorders of cornification." Orphanet Journal of Rare Diseases 9, no. 1 (2014): 33. http://dx.doi.org/10.1186/1750-1172-9-33.

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28

Kadono, Nanako, and Yohei Hirai. "Extracellular syntaxin4 accelerates epidermal cornification via its functional core (AIEPQK)." Journal of Dermatological Science 84, no. 1 (2016): e40. http://dx.doi.org/10.1016/j.jdermsci.2016.08.129.

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29

Mezzalira, G., D. Denti, S. Ferro, M. Angileri, F. Di Diodoro, and M. De Lucia. "A Congenital Cornification Disorder in a Labrador Retriever-Cross Dog." Journal of Comparative Pathology 156, no. 1 (2017): 137. http://dx.doi.org/10.1016/j.jcpa.2016.11.253.

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30

Nikoo, Azita. "Pachyonychia Congenita-Associated Alopecia." Case Reports in Pathology 2012 (2012): 1–2. http://dx.doi.org/10.1155/2012/850658.

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A 5-year-old female, known case of pachyonychia congenita, presented with diffuse hair loss; remaining hairs were easily plucked kinky hairs. Hair samples from patient were investigated using a light microscope. The hairs of the patients were mainly anagen hairs and unlike normal plucked anagen hairs, showed keratinization and cornification of their hair bulbs. No specific hair shaft abnormality was found.

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31

Ishitsuka, Yosuke, and Dennis R. Roop. "Loricrin: Past, Present, and Future." International Journal of Molecular Sciences 21, no. 7 (2020): 2271. http://dx.doi.org/10.3390/ijms21072271.

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The terminal differentiation of the epidermis is a complex physiological process. During the past few decades, medical genetics has shown that defects in the stratum corneum (SC) permeability barrier cause a myriad of pathological conditions, ranging from common dry skin to lethal ichthyoses. Contrarily, molecular phylogenetics has revealed that amniotes have acquired a specialized form of cytoprotection cornification that provides mechanical resilience to the SC. This superior biochemical property, along with desiccation tolerance, is attributable to the proper formation of the macromolecular

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32

Lima Cunha, Dulce, Amanda Oram, Robert Gruber, et al. "hiPSC-Derived Epidermal Keratinocytes from Ichthyosis Patients Show Altered Expression of Cornification Markers." International Journal of Molecular Sciences 22, no. 4 (2021): 1785. http://dx.doi.org/10.3390/ijms22041785.

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Inherited ichthyoses represent a large heterogeneous group of skin disorders characterised by impaired epidermal barrier function and disturbed cornification. Current knowledge about disease mechanisms has been uncovered mainly through the use of mouse models or human skin organotypic models. However, most mouse lines suffer from severe epidermal barrier defects causing neonatal death and human keratinocytes have very limited proliferation ability in vitro. Therefore, the development of disease models based on patient derived human induced pluripotent stem cells (hiPSCs) is highly relevant. Fo

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33

Kadono, Nanako, Tomoatsu Horigome, Hiroko Yano, Ayumi Nakashima, and Yohei Hirai. "Heparinoid facilitates functional expression of extracellular syntaxin-4 on keratinocyte cornification." Journal of Dermatological Science 86, no. 2 (2017): e42. http://dx.doi.org/10.1016/j.jdermsci.2017.02.124.

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34

Kadono, Nanako, Natsumi Hagiwara, Takashi Tagawa, Kenji Maekubo, and Yohei Hirai. "Extracellularly Extruded Syntaxin-4 Is a Potent Cornification Regulator of Epidermal Keratinocytes." Molecular Medicine 21, no. 1 (2015): 77–86. http://dx.doi.org/10.2119/molmed.2014.00234.

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35

ISHIGAMI, Akihito, Hiroaki ASAGA, Takako OHSAWA, Kyoichi AKIYAMA, and Naoki MARUYAMA. "Protein Deimination and Peptidylarginine Deiminase Expression during Cornification of Rat Epidermal Keratinocytes." Biomedical Research 23, no. 3 (2002): 145–51. http://dx.doi.org/10.2220/biomedres.23.145.

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36

Williams, Mary L., and Peter M. Elias. "Ichthyosis: Where we have been disorders of cornification: Where we are going." Current Problems in Dermatology 12, no. 4 (2000): 170–76. http://dx.doi.org/10.1016/s1040-0486(00)90011-5.

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37

Williams, Mary L., and Peter M. Elias. "Ichthyosis: Where we have been disorders of cornification: Where we are going." Current Problems in Pediatrics 30, no. 9 (2000): 298–304. http://dx.doi.org/10.1016/s0045-9380(00)80023-7.

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38

Alibardi, Lorenzo, and Mattia Toni. "Cytochemical and molecular characteristics of the process of cornification during feather morphogenesis." Progress in Histochemistry and Cytochemistry 43, no. 1 (2008): 1–69. http://dx.doi.org/10.1016/j.proghi.2008.01.001.

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39

Alibardi, Lorenzo, and Mattia Toni. "Skin structure and cornification proteins in the soft-shelled turtle Trionyx spiniferus." Zoology 109, no. 3 (2006): 182–95. http://dx.doi.org/10.1016/j.zool.2005.11.005.

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40

Cadiergues, Marie-Christine, Anita Patel, David H. Shearer, Ruth Fermor, Suhel Miah, and Anke Hendricks. "Cornification defect in the Golden retriever: clinical, histopathological, ultrastructural and genetic characterisation." Veterinary Dermatology 19, no. 3 (2008): 120–29. http://dx.doi.org/10.1111/j.1365-3164.2008.00667.x.

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41

Leclerc, E. A., A. Huchenq, S. Kezic, G. Serre, and N. Jonca. "Mice deficient for the epidermal dermokine and isoforms display transient cornification defects." Journal of Cell Science 127, no. 13 (2014): 2862–72. http://dx.doi.org/10.1242/jcs.144808.

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42

Sundberg, John P., Wesley G. Beamer, Leonard D. Shultz, and Robert W. Dunstan. "Inherited Mouse Mutations as Models of Human Adnexal, Cornification, and Papulosquamous Dermatoses." Journal of Investigative Dermatology 95, no. 5 (1990): S62—S63. http://dx.doi.org/10.1111/1523-1747.ep12505816.

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43

ALIBARDI, LORENZO. "Perspectives on Hair Evolution Based on Some Comparative Studies on Vertebrate Cornification." Journal of Experimental Zoology Part B: Molecular and Developmental Evolution 318, no. 5 (2012): 325–43. http://dx.doi.org/10.1002/jez.b.22447.

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44

Jaeger, K., S. Sukseree, S. Zhong, et al. "1344 Keratinocyte cornification requires autophagy for efficient degradation of non-cytoskeletal proteins." Journal of Investigative Dermatology 138, no. 5 (2018): S228. http://dx.doi.org/10.1016/j.jid.2018.03.1361.

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45

Guttman-Yassky, Emma, Mayte Suárez-Fariñas, Andrea Chiricozzi, et al. "Broad defects in epidermal cornification in atopic dermatitis identified through genomic analysis." Journal of Allergy and Clinical Immunology 124, no. 6 (2009): 1235–44. http://dx.doi.org/10.1016/j.jaci.2009.09.031.

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46

Alibardi, Lorenzo. "Claw development and cornification in the passeraceous bird zebrafinch (Taeniatopygia guttata castanotis)." Anatomical Science International 84, no. 3 (2009): 189–99. http://dx.doi.org/10.1007/s12565-009-0015-4.

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47

Metze, Dieter, Heiko Traupe, and Kira Süßmuth. "Ichthyoses—A Clinical and Pathological Spectrum from Heterogeneous Cornification Disorders to Inflammation." Dermatopathology 8, no. 2 (2021): 107–23. http://dx.doi.org/10.3390/dermatopathology8020017.

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Ichthyoses are inborn keratinization disorders affecting the skin only (non-syndromic) or are associated with diseases of internal organs (syndromic). In newborns, they can be life-threatening. The identification of the gene defects resulted in reclassification and a better understanding of the pathophysiology. Histopathologic patterns include orthohyperkeratosis with a reduced or well-developed stratum granulosum, hyperkeratosis with ortho- and parakeratosis with preserved or prominent stratum granulosum, and epidermolytic ichthyosis. Another pattern features “perinuclear vacuoles and binucle

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48

Kretzschmar, Kai, Kim E. Boonekamp, Margit Bleijs, et al. "Troy/Tnfrsf19 marks epidermal cells that govern interfollicular epidermal renewal and cornification." Stem Cell Reports 16, no. 9 (2021): 2379–94. http://dx.doi.org/10.1016/j.stemcr.2021.07.007.

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49

Lapatto, Risto, J. Carl Pallais, Dongsheng Zhang, et al. "Kiss1 −/− Mice Exhibit More Variable Hypogonadism than Gpr54−/− Mice." Endocrinology 148, no. 10 (2007): 4927–36. http://dx.doi.org/10.1210/en.2007-0078.

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The G protein-coupled receptor Gpr54 and its ligand metastin (derived from the Kiss1 gene product kisspeptin) are key gatekeepers of sexual maturation. Gpr54 knockout mice demonstrate hypogonadotropic hypogonadism, but until recently, the phenotype of Kiss1 knockout mice was unknown. This report describes the reproductive phenotypes of mice carrying targeted deletions of Kiss1 or Gpr54 on the same genetic background. Both Kiss1 and Gpr54 knockout mice are viable but infertile and have abnormal sexual maturation; the majority of males lack preputial separation, and females have delayed vaginal

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50

Parhizkarsup;, Saadat. "Assessing estrogenic activity of Nigella sativa in ovariectomized rats using vaginal cornification assay." African Journal of Pharmacy and Pharmacology 5, no. 2 (2011): 137–42. http://dx.doi.org/10.5897/ajpp10.276.

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