Relevant bibliographies by topics / Coronary Vessels physiology

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Coronary Vessels physiology'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Coronary Vessels physiology"

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Zamir, M. "Distributing and delivering vessels of the human heart." Journal of General Physiology 91, no. 5 (May 1, 1988): 725–35. http://dx.doi.org/10.1085/jgp.91.5.725.

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The branching characteristics of the right coronary artery, acute marginal, posterior descending, left anterior descending, circumflex, and obtuse marginal arteries are compared with those of diagonal branches, left and right ventricular branches, septal, and higher-order branches, to test a newly proposed functional classification of the coronary arteries in which the first group rank as distributing vessels and the second as delivering vessels. According to this classification, the function of the first type is merely to convey blood to the borders of myocardial zones, while the function of
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Greer, C., A. Puri, J. Sutherland, J. Blake, D. McClean, J. Elliott, and D. Smyth. "Borderline Coronary Physiology – Are All Vessels Equal?" Heart, Lung and Circulation 28 (2019): S387. http://dx.doi.org/10.1016/j.hlc.2019.06.588.

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Kassab, G. S., C. A. Rider, N. J. Tang, and Y. C. Fung. "Morphometry of pig coronary arterial trees." American Journal of Physiology-Heart and Circulatory Physiology 265, no. 1 (July 1, 1993): H350—H365. http://dx.doi.org/10.1152/ajpheart.1993.265.1.h350.

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To establish a mathematical model of the tree like arteries for the purpose of hemodynamic analysis, a complete set of morphometric data of pig coronary arteries is presented. For the purpose of mathematical modeling, three innovations in morphometry are introduced: 1) a rule for assigning the order numbers of the vessels on the basis of diameter ranges, 2) a connectivity matrix to describe asymmetric branching, and 3) a measurement of the fraction of vessel segments connected in series. The morphometric measurements were made with the silicone elastomer-casting method. Data on smaller vessels
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Kassab, Ghassan S. "Functional hierarchy of coronary circulation: direct evidence of a structure-function relation." American Journal of Physiology-Heart and Circulatory Physiology 289, no. 6 (December 2005): H2559—H2565. http://dx.doi.org/10.1152/ajpheart.00561.2005.

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The heart muscle is nourished by a complex system of blood vessels that make up the coronary circulation. Here we show that the design of the coronary circulation has a functional hierarchy. A full anatomic model of the coronary arterial tree, containing millions of blood vessels down to the capillary vessels, was simulated based on previously measured porcine morphometric data. A network analysis of blood flow through every vessel segment was carried out based on the laws of fluid mechanics and appropriate boundary conditions. Our results show an abrupt change in cross-sectional area that dem
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Hoffman, J. I., and J. A. Spaan. "Pressure-flow relations in coronary circulation." Physiological Reviews 70, no. 2 (April 1, 1990): 331–90. http://dx.doi.org/10.1152/physrev.1990.70.2.331.

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The blood vessels that run on the surface of the heart and through its muscle are compliant tubes that can be affected by the pressures external to them in at least two ways. If the pressure outside these vessels is higher than the pressure at their downstream ends, the vessels may collapse and become Starling resistors or vascular waterfalls. If this happens, the flow through these vessels depends on their resistance and the pressure drop from their inflow to the pressure around them and is independent of the actual downstream pressure. In the first part of this review, the physics of collaps
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Habazettl, H., B. Vollmar, M. Christ, H. Baier, P. F. Conzen, and K. Peter. "Heterogeneous microvascular coronary vasodilation by adenosine and nitroglycerin in dogs." Journal of Applied Physiology 76, no. 5 (May 1, 1994): 1951–60. http://dx.doi.org/10.1152/jappl.1994.76.5.1951.

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We investigated the effects of adenosine and nitroglycerin (NTG) on coronary microvessel diameters (intravital fluorescence microscopy) and coronary perfusion (radioactive microspheres). Measurements were performed during baseline conditions (intravenous piritramid) and during controlled hypotension (mean arterial pressure approximately 60 mmHg) induced by halothane, adenosine, and NTG. Coronary vascular resistance (CVR) remained unchanged during halothane (-7%) but decreased during adenosine (-76%) and NTG (-29%). Coronary arteriolar diameters increased during all experimental steps. In the s
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Räsänen, Markus, Ibrahim Sultan, Jennifer Paech, Karthik Amudhala Hemanthakumar, Wei Yu, Liqun He, Juan Tang, et al. "VEGF-B Promotes Endocardium-Derived Coronary Vessel Development and Cardiac Regeneration." Circulation 143, no. 1 (January 5, 2021): 65–77. http://dx.doi.org/10.1161/circulationaha.120.050635.

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Background: Recent discoveries have indicated that, in the developing heart, sinus venosus and endocardium provide major sources of endothelium for coronary vessel growth that supports the expanding myocardium. Here we set out to study the origin of the coronary vessels that develop in response to vascular endothelial growth factor B (VEGF-B) in the heart and the effect of VEGF-B on recovery from myocardial infarction. Methods: We used mice and rats expressing a VEGF-B transgene, VEGF-B-gene–deleted mice and rats, apelin-CreERT, and natriuretic peptide receptor 3–CreERT recombinase-mediated ge
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Lavallée, Michel, and Eric Thorin. "Role of ET-1 in the regulation of coronary circulation." Canadian Journal of Physiology and Pharmacology 81, no. 6 (June 1, 2003): 570–77. http://dx.doi.org/10.1139/y03-014.

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Given that circulating ET levels in heart failure, in particular, may reach physiological threshold for coronary constrictor responses, the primary objective of the present review is to consider coronary vessels as an important target for circulating and locally produced endothelin(s). In healthy vessels, ET-1 causes biphasic coronary responses characterized by a transient dilation of large and small arteries followed by a sustained constriction. ETB receptors are pivotal in the early dilation of resistance vessels, whereas dilation of conductance vessels may be a secondary phenomenon triggere
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Zhang, J., M. Somers, and F. R. Cobb. "Heterogeneous effects of nitroglycerin on the conductance and resistance coronary arterial vasculature." American Journal of Physiology-Heart and Circulatory Physiology 264, no. 6 (June 1, 1993): H1960—H1968. http://dx.doi.org/10.1152/ajpheart.1993.264.6.h1960.

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This study assessed the effects of nitroglycerin (NTG) on epicardial conductance and blood flow regulatory vessels over a wide dose range (10(-10) to 10(-6) mol NTG) in chronically instrumented awake mongrel dogs. NTG bolus injection caused dose-dependent dilation of both conductance and blood flow regulatory vessels. The dose-response curves for blood flow were shifted markedly to the right of the response of conductance vessels so that the proximal vessels had reached 50% of their maximum vasodilation before significant increases in blood flow. The calculated doses for half-maximal vasodilat
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Duncker, Dirk J., and Robert J. Bache. "Regulation of Coronary Blood Flow During Exercise." Physiological Reviews 88, no. 3 (July 2008): 1009–86. http://dx.doi.org/10.1152/physrev.00045.2006.

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Exercise is the most important physiological stimulus for increased myocardial oxygen demand. The requirement of exercising muscle for increased blood flow necessitates an increase in cardiac output that results in increases in the three main determinants of myocardial oxygen demand: heart rate, myocardial contractility, and ventricular work. The approximately sixfold increase in oxygen demands of the left ventricle during heavy exercise is met principally by augmenting coronary blood flow (∼5-fold), as hemoglobin concentration and oxygen extraction (which is already 70–80% at rest) increase o
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Dissertations / Theses on the topic "Coronary Vessels physiology"

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Warner, Anke Sigrid. "The expression, regulation and effects of inducible nitric oxide synthase in hibernating myocardium." Title page, contents and summary only, 2002. http://web4.library.adelaide.edu.au/theses/09PH/09phw279.pdf.

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Amendments inserted at back. "May 2002" Includes bibliographical references (leaves 237-290) Experiments described in this thesis address the potential role of inducible nitric oxide synthase (iNOS) in hibernating myocardium. Specifically it was sought to establish a cellular model of hibernating myocardium and investigate the expression, regulation and effects of iNOS in this model. Experiments were performed using primary cultures of neonatal rat ventricular myocytes.
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Wikenheiser, Jamie Christopher. "Altered Hypoxia-Inducible Factor-1 Alpha Levels Correlate with Coronary Artery Anomalies." Case Western Reserve University School of Graduate Studies / OhioLINK, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=case1216218122.

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Wang, Ruoya. "Novel theoretical and experimental frameworks for multiscale quantification of arterial mechanics." Diss., Georgia Institute of Technology, 2013. http://hdl.handle.net/1853/47718.

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The mechanical behavior of the arterial wall is determined by the composition and structure of its internal constituents as well as the applied traction-forces, such as pressure and axial stretch. The purpose of this work is to develop new theoretical frameworks and experimental methodologies to further the understanding of arterial mechanics and role of the various intrinsic and extrinsic mechanically motivating factors. Specifically, residual deformation, matrix organization, and perivascular support are investigated in the context of their effects on the overall and local mechanical behavio
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Sarkar, Nondita. "Myocardial angiogenesis induced by plasmid VEGF-A165 gene transfer : experimental and clinical studies /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-196-2/.

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"Cellular electrophysiological and mechanical effects of organ preservation solutions on endothelial function in resistance coronary and pulmonary arteries: implications in heart and lung transplantation." 2006. http://library.cuhk.edu.hk/record=b5892759.

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Wu Min.<br>Thesis (M.Phil.)--Chinese University of Hong Kong, 2006.<br>Includes bibliographical references (leaves 87-114).<br>Abstracts in English and Chinese.<br>Declaration --- p.i<br>Acknowledgement --- p.ii<br>Publication list --- p.iii<br>Abstract (English) --- p.xi<br>Abstract (Chinese) --- p.xiv<br>Abbreviations --- p.xvi<br>List of figures / tables --- p.xviii<br>Chapter Chapter 1. --- General Introduction --- p.1<br>Chapter 1.1 --- Endothelial function in the regulation of vascular tone --- p.1<br>Chapter 1.1.1 --- NO --- p.2<br>Chapter 1.1.2 --- PGI2 --- p.5<br>Chapter 1.
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Noblet, Jillian Nicole. "Coronary perivascular adipose tissue and vascular smooth muscle function: influence of obesity." Diss., 2016. http://hdl.handle.net/1805/9815.

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Indiana University-Purdue University Indianapolis (IUPUI)<br>Factors released from coronary perivascular adipose tissue (PVAT), which surrounds large coronary arteries, have been implicated in the development of coronary disease. However, the precise contribution of coronary PVAT-derived factors to the initiation and progression of coronary vascular dysfunction remains ill defined. Accordingly, this investigation was designed to delineate the mechanisms by which PVAT-derived factors influence obesity-induced coronary smooth muscle dysfunction. Isometric tension studies of coronary arteries fro
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Owen, Meredith Kohr. "Effect of coronary perivascular adipose tissue on vascular smooth muscle function in metabolic syndrome." Thesis, 2013. http://hdl.handle.net/1805/3789.

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Indiana University-Purdue University Indianapolis (IUPUI)<br>Obesity increases cardiovascular disease risk and is associated with factors of the “metabolic syndrome” (MetS), a disorder including hypertension, hypercholesterolemia and/or impaired glucose tolerance. Expanding adipose and subsequent inflammation is implicated in vascular dysfunction in MetS. Perivascular adipose tissue (PVAT) surrounds virtually every artery and is capable of releasing factors that influence vascular reactivity, but the effects of PVAT in the coronary circulation are unknown. Accordingly, the goal of this inves
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Deep, Debanjan. "A study of blood flow in normal and dilated aorta." Thesis, 2013. http://hdl.handle.net/1805/4440.

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Indiana University-Purdue University Indianapolis (IUPUI)<br>Atherosclerotic lesions of human beings are common diagnosed in regions of arte- rial branching and curvature. The prevalence of atherosclerosis is usually associated with hardening and ballooning of aortic wall surfaces because of narrowing of flow path by the deposition of fatty materials, platelets and influx of plasma through in- timal wall of Aorta. High Wall Shear Stress (WSS) is proved to be the main cause behind all these aortic diseases by physicians and researchers. Due to the fact that the atherosclerotic regions are assoc
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Books on the topic "Coronary Vessels physiology"

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Carl, Maurer Peter, and Deutsche Gesellschaft für Angiologie. Jahrestagung, eds. What is new in angiology?: Trends and controversies : proceedings, 14th World Congress, International Union of Angiology, 15th Annual Meeting, German Society of Angiology, July 6-11, 1986, Munich, West Germany. München: W. Zuckschwerdt Verlag, 1986.

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Mohl, W. Coronary sinus interventions in cardiac surgery. 2nd ed. Georgetown, Tex: Landes Bioscience, 2000.

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de, Bruyne Bernard, ed. Coronary pressure. Dordrecht: Kluwer Academic Publishers, 1997.

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de, Bruyne Bernard, ed. Coronary pressure. 2nd ed. Dordrecht: Kluwer Academic Publishers, 2000.

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V, Schaff Hartzell, ed. Vasoactive factors produced by the endothelium: Physiology and surgical implications. Austin: R.G. Landes, 1994.

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Tomanek, Robert J. Coronary Vasculature: Development, Structure-Function, and Adaptations. Springer, 2012.

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Coronary Vasculature: Development, Structure-Function, and Adaptations. Springer, 2012.

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Pijls, N. H. Coronary Pressure. Springer, 2010.

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Pijls, N. H., and B. de Bruyne. Coronary Pressure (DEVELOPMENTS IN CARDIOVASCULAR MEDICINE Volume 227) (Developments in Cardiovascular Medicine). Springer, 2000.

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Li, John K.-J. The Arterial Circulation: Physical Principles and Clinical Applications. Humana Press, 2000.

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Book chapters on the topic "Coronary Vessels physiology"

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Kozarek, Katherine, and Ryan Hood. "Cardiac Tumors." In Cardiac Anesthesia: A Problem-Based Learning Approach, edited by Mohammed M. Minhaj, 42–47. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780190884512.003.0005.

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Cardiac tumors are rarely encountered in clinical practice; however, because they affect vital structures, they can have significant consequences. Cardiac tumors are categorized as either primary tumors originating from cardiac tissue or metastatic tumors migrated from other sites; they may infiltrate the atria, ventricles, valves, and coronary arteries, resulting in considerable changes to normal physiology. They may extend beyond the myocardium to the pericardium, great vessels, and mediastinum. Advances in the development of imaging modalities have allowed for expedited diagnostic evaluation of cardiac tumors. Prompt treatment, which often includes surgical intervention, is essential to both alleviate symptoms and prevent harmful sequelae. The intraoperative anesthetic management of a patient with a cardiac mass presents a number of challenges due to altered cardiovascular physiology. Because these tumors occur so infrequently, standardized, evidence-based diagnostic and treatment guidelines have not been developed. The existing data were derived mainly from small case series and case reports. The aggressive nature of the disease warrants further investigation to improve the accuracy of diagnostic modalities and the efficacy of treatment regimens.
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Becker, Richard C., and Frederick A. Spencer. "Acute Coronary Syndromes." In Fibrinolytic and Antithrombotic Therapy. Oxford University Press, 2006. http://dx.doi.org/10.1093/oso/9780195155648.003.0025.

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For over a century astute clinicians have recognized that prodromal symptoms often precede acute myocardial infarction (MI). The evolution of symptoms was subsequently found to correlate with changes in atherosclerotic plaque composition, morphology, and thrombogenicity, leading to the classification of symptoms that are currently categorized to better delineate diagnostic and management strategies. Acute coronary syndromes (ACSs) are traditionally divided into two separate categories—ST-segment elevation and non–ST-segment elevation ACS—based on the presenting electrocardiogram. The latter category is then subdivided into unstable angina and non–ST-segment elevation MI, based on the absence or presence of elevated cardiac biomarkers, respectively. This chapter considers ST-segment elevation MI and non–ST-segment elevation ACS based on pharmacologic and clinical (diagnostics and routine management) constructs. ST-segment elevation MI (STEMI), in a vast majority of cases, is caused by occlusive thrombosis at a site of plaque rupture. In others, particularly when the stimulus for thrombosis is strong, occlusion may follow minor disruption of the plaque surface (erosion) or occur in areas of endothelial cell injury (activation with inflammatory features and concomitantly impaired vascular thromboresistance). Coronary arterial spasm, in the absence of intrinsic vascular disease (as may be seen with cocaine use), can also impair restrictive blood flow to the myocardium, resulting in cellular death. The goal of pharmacology-based therapy (and mechanical intervention) is to restore myocardial blood flow as quickly and completely as possible. The “open vessel hypotheses” predicts that rapid, complete, and sustained myocardial perfusion through the prompt restoration of physiologic blood flow will minimize (salvage) myocardium, promote ventricular performance, and reduce mortality. Strong support for the open-vessel hypothesis can be traced to the Thrombolysis and Myocardial Infarction (TIMI) trial performed in the 1980s (Dalen et al., 1988; TIMI Study Group, 1985). Patients with patent infarct-related coronary arteries 90 minutes after the initiation of fibrinolytic therapy had an 8.1% mortality at 1 year, compared to a 14.8% mortality among those with an occluded vessel. Since that time, several large-scale clinical trials have confirmed the importance of an open infarct-related coronary artery for early, intermediate, and long-term outcome.
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Becker, Richard C., and Frederick A. Spencer. "Facilitated Percutaneous Coronary Intervention." In Fibrinolytic and Antithrombotic Therapy. Oxford University Press, 2006. http://dx.doi.org/10.1093/oso/9780195155648.003.0018.

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The failure of fibrinolytic therapy to restore physiologic myocardial perfusion in upward of 40% of patients supports the development of strategies to improve response rates to percutaneous coronary intervention (PCI) in those requiring early procedures. The construct of facilitated PCI (pharmacoinvasive therapy) provides a platform for utilizing the strengths of existing therapies and treatment modalities. The Heparin in Early Patency (HEAP) trial (Zijlstra et al., 2002) included 1,702 patients treated with primary PCI for myocardial infarction (MI); 860 patients received aspirin (500 mg IV) and UFH (≥5,000 U IV) before being transported to the hospital and 842 patients received the same antithrombotic therapy in the hospital. TIMI 2 or 3 flow rates were higher in the pretreated group (31% vs. 20%; p = .001), and patients with TIMI 2 or 3 flow initially had a higher PCI success rate (94% vs. 89%; p &lt;.001) and a lower 30-day mortality (1.6% vs. 3.4%; p = .04). The Plasminogen Activator Angioplasty Compatibility Trial (PACT) randomized 606 patients to receive a 50-mg bolus of alteplase or placebo, followed by immediate angiography and angioplasty if needed (Ross et al., 1999). TIMI flow rates on arrival to the catheterization laboratory were 33% and 15%, respectively. Facilitated PCI and primary PCI restored TIMI 3 flow in occluded vessels equally (77% and 79%, respectively). There were no differences in major bleeding. Left ventricular ejection fraction was highest in those with TIMI 3 flow on arrival to the catheterization laboratory or following PCI within 1 hour of alteplase administration. Full-dose fibrinolytic therapy with alteplase or reteplase followed by coronary angiography and PCI (if no clinical evidence of reperfusion) was evaluated retrospectively in the Global Use of Strategies to Open Occluded Arteries (GUSTO) III trial (Miller et al., 1999). Among those undergoing PCI (n = 392), 87 patients received in-laboratory abciximab. A trend toward reduced mortality was observed in abciximab-treated patients, but at a higher cost of hemorrhagic complications. In the Strategies for Patency Enhancement in the Emergency Department (SPEED) trial (Herrmann et al., 2000), 323 patients who underwent PCI had an 88% procedural success rate and a 30-day composite of death, reinfarction, or revascularization of 5.6%.
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Conference papers on the topic "Coronary Vessels physiology"

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Biglino, Giovanni, Daria Cosentino, Matteo Castelli, Lorenzo De Nova, Hopewell N. Ntsinjana, Jennifer A. Steeden, Andrew M. Taylor, and Silvia Schievano. "Combining 4D MR Flow Experimental Data and Computational Fluid Dynamics to Study the Neoaorta in Patients With Repaired Transposition of the Great Arteries." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14456.

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Transposition of the great arteries (TGA) is a congenital heart disease characterized by abnormal spatial arrangement of the two main vessels, with the aorta arising from the pulmonary valve and the main pulmonary artery arising from the aortic valve. TGA surgical repair with the arterial switch operation (ASO) involves physically repositioning the aorta and the pulmonary artery in their correct anatomical location, as well as separately moving the coronary arteries. Following ASO, decreased aortic distensibility and enlarged aortic root have been observed, together with late complications suc
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Albal, Priti G., and Prahlad G. Menon. "MRI and CT Image-Fusion Based Aorta and Coronary Artery Model for In-Silico Feasibility Evaluation of Perfusion With an Ascending Aortic Pump, Using Computational Fluid Dynamics." In ASME 2013 Conference on Frontiers in Medical Devices: Applications of Computer Modeling and Simulation. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/fmd2013-16105.

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Left ventricular assist devices (LVADs) are mechanical pumps that provide full or partial support of the circulation in patients with varying degrees of heart failure (HF). This simulation study explores the hemodynamic effects of a continuous flow pump deployed in the Ascending Aorta (AAo) specifically focusing on: (a) perfusion of the coronary arterial circulation and (b) the effect of induced non-physiologic, swirling flow discharged by the pump on perfusion to head-neck vessels of the aortic arch.
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Fayssal, Iyad, Fadl Moukalled, Samir Alam, Robert Habib, and Hussain Ismaeel. "The Development of a Robust Low Computational Cost Diagnostic Tool to Evaluate Stenosis Functional Significance in Human Coronary Arteries." In ASME 2015 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2015. http://dx.doi.org/10.1115/imece2015-51532.

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There is discordance between the anatomic severities of the coronary narrowing and their corresponding functional significance. Fractional Flow Reserve (FFR) is among the physiological parameters invasively measured to assess the hemodynamic significance of a stenosis during maximal hyperemia. FFR values ≤ 0.8 indicate that the downstream heart tissue perfused by this vessel is at risk for ischemia. While measuring FFR is an invasive procedure that is expensive, time consuming, and not without complications, recently, noninvasive estimation of FFR was shown to be possible from comprehensive pr
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