Relevant bibliographies by topics / Corpus luteum hormones

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Academic literature on the topic 'Corpus luteum hormones'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Corpus luteum hormones"

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Duncan, W. Colin. "The inadequate corpus luteum." Reproduction and Fertility 2, no. 1 (February 26, 2021): C1—C7. http://dx.doi.org/10.1530/raf-20-0044.

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The corpus luteum is the source of progesterone in the luteal phase of the cycle and the initial two-thirds of the first trimester of pregnancy. Normal luteal function is required for fertility and the maintenance of pregnancy. Progesterone administration is increasingly used during fertility treatments and in early pregnancy to mitigate potentially inadequate corpus luteum function. This commentary considers the concept of the inadequate corpus luteum and the role and effects of exogenous progesterone. Progesterone supplementation does have important beneficial effects but we should be wary of therapeutic administration beyond or outside the evidence base. Lay summary After an egg is released a structure is formed on the ovary called a corpus luteum (CL). This produces a huge amount of a hormone called progesterone. Progesterone makes the womb ready for pregnancy but if a pregnancy does not happen the CL disappears after 12–14 days and this causes a period. If a pregnancy occurs, then the pregnancy hormone (hCG) keeps the CL alive and its progesterone supports the pregnancy for the next 6–8 weeks until the placenta takes over and the corpus luteum disappears. That means that if the CL is not working correctly there could be problems getting pregnant or staying pregnant. If a CL is not producing enough progesterone it usually means there is a problem with the growing or releasing of the egg and treatment should focus on these areas. In IVF cycles, where normal hormones are switched off, the CL does not produce quite enough progesterone before the pregnancy test and extra progesterone is needed at this time. In recurrent or threatened miscarriage, however, there is not any evidence that the CL is not working well or progesterone is low. However, there is benefit in taking extra progesterone if there is bleeding in early pregnancy in women with previous miscarriages. This might be because of the effects of high-dose progesterone on the womb or immune system. As changes to the hormone environment in pregnancy may have some life-long consequences for the offspring we have to be careful only to give extra progesterone when we are sure it is needed.
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Niswender, Gordon D., Jennifer L. Juengel, Patrick J. Silva, M. Keith Rollyson, and Eric W. McIntush. "Mechanisms Controlling the Function and Life Span of the Corpus Luteum." Physiological Reviews 80, no. 1 (January 1, 2000): 1–29. http://dx.doi.org/10.1152/physrev.2000.80.1.1.

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The primary function of the corpus luteum is secretion of the hormone progesterone, which is required for maintenance of normal pregnancy in mammals. The corpus luteum develops from residual follicular granulosal and thecal cells after ovulation. Luteinizing hormone (LH) from the anterior pituitary is important for normal development and function of the corpus luteum in most mammals, although growth hormone, prolactin, and estradiol also play a role in several species. The mature corpus luteum is composed of at least two steroidogenic cell types based on morphological and biochemical criteria and on the follicular source of origin. Small luteal cells appear to be of thecal cell origin and respond to LH with increased secretion of progesterone. LH directly stimulates the secretion of progesterone from small luteal cells via activation of the protein kinase A second messenger pathway. Large luteal cells are of granulosal cell origin and contain receptors for PGF2αand appear to mediate the luteolytic actions of this hormone. If pregnancy does not occur, the corpus luteum must regress to allow follicular growth and ovulation and the reproductive cycle begins again. Luteal regression is initiated by PGF2αof uterine origin in most subprimate species. The role played by PGF2αin primates remains controversial. In primates, if PGF2αplays a role in luteolysis, it appears to be of ovarian origin. The antisteroidogenic effects of PGF2αappear to be mediated by the protein kinase C second messenger pathway, whereas loss of luteal cells appears to follow an influx of calcium, activation of endonucleases, and an apoptotic form of cell death. If the female becomes pregnant, continued secretion of progesterone from the corpus luteum is required to provide an appropriate uterine environment for maintenance of pregnancy. The mechanisms whereby the pregnant uterus signals the corpus luteum that a conceptus is present varies from secretion of a chorionic gonadotropin (primates and equids), to secretion of an antiluteolytic factor (domestic ruminants), and to a neuroendocrine reflex arc that modifies the secretory patterns of hormones from the anterior pituitary (most rodents).
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Bramley, T. A., G. S. Menzies, A. S. McNeilly, and H. G. Friesen. "Receptors for lactogenic hormones in the ovine corpus luteum. I: A major discrepancy in the specific binding of radiolabelled ovine prolactin and human growth hormone." Journal of Endocrinology 113, no. 3 (June 1987): 365–74. http://dx.doi.org/10.1677/joe.0.1130365.

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ABSTRACT The characteristics of the binding of 125I-labelled human GH (hGH) and ovine prolactin (oPRL) were studied in the ovine corpus luteum. Although oPRL is the homologous ligand for sheep lactogenic receptors, its binding was significantly and consistently lower than that of 125I-labelled hGH. This was not due to iodination damage of oPRL since: (1) 125I-labelled oPRL tracers which bound poorly relative to 125I-labelled hGH in the ovine corpus luteum were equipotent in the pig and rat corpus luteum, (2) the differences between 125I-labelled hGH and oPRL binding persisted with tracers of equivalent biopotency and (3) the iodination procedure affected neither oPRL bioactivity in the Nb2 tumour assay nor its binding activity with ovine corpus luteum receptors. Ovine luteal receptors were specific for lactogenic hormones. The specific binding of 125I-labelled hGH or oPRL could be inhibited completely by incubation with either unlabelled hormone, with similar potencies. However, oGH inhibited binding only at much higher concentrations, consistent with its known contamination with oPRL. Moreover, 125I-labelled oGH was not bound specifically to sheep luteal tissue. Fractionation of sheep luteal homogenates on sucrose density gradients (with or without cell-surface membrane perturbation by digitonin) demonstrated that binding of 125I-labelled hGH and 125I-labelled oPRL peaked in the same regions of the gradients, coincident with a number of luteal cell-surface membrane markers. We conclude that the marked discrepancy between the binding of hGH and oPRL tracers by sheep luteal tissue was not due to iodination damage of oPRL, binding of 125I-labelled hGH to somatogenic receptors or differential binding to luteal cell-surface versus intracellular receptors. J. Endocr. (1987) 113, 365–374
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Gemmell, RT. "A comparative study of the corpus luteum." Reproduction, Fertility and Development 7, no. 3 (1995): 303. http://dx.doi.org/10.1071/rd9950303.

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The corpus luteum (CL) is a transitory organ which has a regulatory role in reproduction. Sharks, amphibians and reptiles have corpora lutea that produce progesterone which influences the rate of embryonic development. The egg-laying monotremes and the two major mammalian groups, eutherian and marsupial, have a CL that secretes progesterone. Most eutherians have allowed for the uterine development of their young by extending the length of the oestrous cycle and the CL or placenta actively secretes progesterone until birth. Gestation in the marsupial does not extend beyond the length of an oestrous cycle and the major part of fetal development takes place in the pouch. Where the extension of the post-luteal phase in the eutherian has allowed for the uterine development of young, the marsupial has extended the pre-luteal phase of the oestrous cycle and has evolved an alternative reproductive strategy, embryonic diapause. The mechanism for the secretion of hormones from the CL has been controversial for many years. Densely-staining secretory granules have been observed in the CL of sharks, marsupials and eutherians. These granules have been reported to contain relaxin, oxytocin or mesotocin, and progesterone. A hypothesis to suit all available data is that all hormones secreted by the CL are transported within such granules. In conclusion, although there are obvious differences in the mode of reproduction in the two main mammalian groups, it is apparent that there is a great deal of similarity in the hormonal control of regression of the CL and parturition.
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Bramley, T. A., and G. S. Menzies. "Receptors for lactogenic hormones in the porcine corpus luteum: properties and luteal phase concentrations." Journal of Endocrinology 113, no. 3 (June 1987): 355–64. http://dx.doi.org/10.1677/joe.0.1130355.

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ABSTRACT Homogenates of pig corpora lutea contained specific, high-affinity receptors for ovine prolactin (oPRL) and human GH (hGH). Specific hormone binding was enhanced by divalent metal ions, but only when included in the binding reaction. Divalent metal ions did not act by increasing the recovery of bound hormone by low-speed centrifugation, but appeared to promote the formation of a more stable hormone– receptor complex. Both oPRL and hGH tracers were bound in similar amounts and with similar affinities by pig luteal homogenates and the concentrations of either unlabelled hormone required to displace specific binding of either tracer by 50% were identical. In contrast, 125I-labelled oGH failed to bind to pig luteal homogenates and oGH competed poorly for hGH or oPRL binding. Only hormones with prolactin-like activity competed for 125I-labelled oPRL binding. Specific prolactin binding was low in recently ovulated and early luteal phase corpora lutea, increased significantly in the mid-luteal phase and declined once more in the late luteal phase. Receptor concentrations increased with increasing gestational age. J. Endocr. (1987) 113, 355–364
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Yang, Ya-Li, Li-Rong Ren, Li-Feng Sun, Chen Huang, Tian-Xia Xiao, Bao-Bei Wang, Jie Chen, Brian A. Zabel, Peigen Ren, and Jian V. Zhang. "The role of GPR1 signaling in mice corpus luteum." Journal of Endocrinology 230, no. 1 (July 2016): 55–65. http://dx.doi.org/10.1530/joe-15-0521.

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Chemerin, a chemokine, plays important roles in immune responses, inflammation, adipogenesis, and carbohydrate metabolism. Our recent research has shown that chemerin has an inhibitory effect on hormone secretion from the testis and ovary. However, whether G protein-coupled receptor 1 (GPR1), the active receptor for chemerin, regulates steroidogenesis and luteolysis in the corpus luteum is still unknown. In this study, we established a pregnant mare serum gonadotropin-human chorionic gonadotropin (PMSG-hCG) superovulation model, a prostaglandin F2α (PGF2α) luteolysis model, and follicle and corpus luteum culture models to analyze the role of chemerin signaling through GPR1 in the synthesis and secretion of gonadal hormones during follicular/luteal development and luteolysis. Our results, for the first time, show that chemerin and GPR1 are both differentially expressed in the ovary over the course of the estrous cycle, with highest levels in estrus and metestrus. GPR1 has been localized to granulosa cells, cumulus cells, and the corpus luteum by immunohistochemistry (IHC). In vitro, we found that chemerin suppresses hCG-induced progesterone production in cultured follicle and corpus luteum and that this effect is attenuated significantly by anti-GPR1 MAB treatment. Furthermore, when the phosphoinositide 3-kinase (PI3K) pathway was blocked, the attenuating effect of GPR1 MAB was abrogated. Interestingly, PGF2α induces luteolysis through activation of caspase-3, leading to a reduction in progesterone secretion. Treatment with GPR1 MAB blocked the PGF2α effect on caspase-3 expression and progesterone secretion. This study indicates that chemerin/GPR1 signaling directly or indirectly regulates progesterone synthesis and secretion during the processes of follicular development, corpus luteum formation, and PGF2α-induced luteolysis.
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Auletta, F. J., D. S. C. Jones, and A. P. F. Flint. "Does the human corpus luteum synthesize neurohypophysial hormones?" Journal of Endocrinology 116, no. 2 (February 1988): 163–65. http://dx.doi.org/10.1677/joe.0.1160163.

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Thordarson, G., S. Galosy, G. O. Gudmundsson, B. Newcomer, R. Sridaran, and F. Talamantes. "Interaction of Mouse Placental Lactogens and Androgens in Regulating Progesterone Release in Cultured Mouse Luteal Cells." Endocrinology 138, no. 8 (August 1, 1997): 3236–41. http://dx.doi.org/10.1210/endo.138.8.5309.

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Abstract Pituitary hormones are essential for the maintenance of the corpus luteum in the pregnant mouse during the first half of gestation. Thereafter, hormones from the placenta take over the luteotropic role of the pituitary hormones. Mouse placental lactogen-I (mPL-I) and mPL-II, two PRL-like hormones produced in the placenta, are probably necessary for the maintenance of the corpus luteum in the latter half of pregnancy. A culture system of luteal cells from pregnant mice was developed to investigate the role of hormones from the placenta that may be important for the function of the corpus luteum. Mice were killed on days 10, 14, and 18 of pregnancy, and the corpora lutea were excised from the ovaries and digested in 0.1% collagenase, 0.002% DNase for 1 h. The resulting luteal cell suspension was plated onto 96-well plates coated with fibronectin (1 × 105 cells/well) and cultured for 1–3 days. Medium was changed daily. The cells were treated with various concentrations and combinations of mPL-I, mPL-II, mouse PRL, androstenedione, dihydrotestosterone, 17β-estradiol (E2), testosterone, hydroxyflutamide, cycloheximide, actinomycin D, and fadrozole to study the effects of these different treatments on progesterone (P4) production. The three lactogens (mPL-I, mPL-II, and mouse PRL) all stimulated the release of P4 from the luteal cells. The potency of the lactogens was similar and did not depend on the stage of pregnancy at which the luteal tissue was obtained. However, the responsiveness of the cells to all hormone-stimulated P4 release was gradually reduced the later in pregnancy the tissue was collected. Androgens also stimulated the release of P4 from the luteal cells, and when administered together, the lactogens and the androgens acted synergistically to stimulate P4 release. The androgens acted directly but not through conversion to E2, as determined by the findings that 1) the effects of the androgens could not be reproduced by E2 administration, 2) nonaromatizable androgen dihydrotestosterone was as effective as aromatizable androgens, and 3) aromatase inhibitor did not prevent the action of the androgens to stimulate the P4 release. The effect of the androgens on the P4 release was rapid, occurring within 15 min of hormone administration. It was not prevented by inhibitors of protein and RNA synthesis, and the intracellular androgen receptor antagonist hydroxyflutamide did not affect the androgen action. Therefore, the androgen effects were not mediated through the intracellular androgen receptor and de novo protein synthesis was not needed for androgen-stimulated P4 release.
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Hearn, J. P., and G. E. Webley. "Regulation of the corpus luteum of early pregnancy in the marmoset monkey: local interactions of luteotrophic and luteolytic hormones in vivo and their effects on the secretion of progesterone." Journal of Endocrinology 114, no. 2 (August 1987): 231–39. http://dx.doi.org/10.1677/joe.0.1140231.

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ABSTRACT The interaction between luteotrophic and luteolytic agents in controlling progesterone production by the marmoset corpus luteum in the late luteal phase/early pregnancy was investigated at the local level in vivo using a perfusion cannula system. Perfusion of the prostaglandin F2α(PGF2α) analogue, cloprostenol (0·5 μg/ml), resulted in an immediate fall in progesterone production. This response was not sustained in two out of five corpora lutea but pregnancy was terminated in all animals exposed to PGF2α. Perfusion of human chorionic gonadotrophin (hCG) (4 μg/ml) alone significantly stimulated progesterone secretion but there was no response to hCG when the corpus luteum had previously been perfused with PGF2α. Perfusion with hCG together with PGF2α prevented a fall in progesterone secretion. The results suggest that the luteolytic action of PGF2α in the marmoset may be to prevent luteotrophic support of the corpus luteum. Melatonin (860 pmol/l), perfused either with PGF2α or after PGF2α, stimulated progesterone production. The ability of melatonin to influence progesterone production by the primate corpus luteum may therefore be by both a direct luteotrophic action and the prevention of luteolysis. Application of the perfusion system in order to investigate the ability of deglycosylated hCG to antagonize the action of hCG at the corpus luteum showed the necessity of testing pure preparations of hormones. J. Endocr. (1987) 114, 231–239
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Journal, Baghdad Science. "The effect of aqueous crude extract of ginger on the histology of corpus luteum and the concentration of hormones estrogen and progesterone in pregnant mice." Baghdad Science Journal 12, no. 4 (December 6, 2015): 638–44. http://dx.doi.org/10.21123/bsj.12.4.638-644.

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This study was designed to investigate the effect of aqueous extract of ginger Zingiber officinale Roscoe on the histology of corpus luteum and the concentration of the hormones progesterone and estrogen during the first trimester of pregnancy (0 - 7) days from fertilization. 30 pregnant mice were divided into five experimental groups: control group (administrated with distilled water), and four groups treated at doses (284, 568, 1136,1420 mg / kg), orally administrated , daily with (0.1 ml). Microscopic examination results have shown histopathological changes in corpus luteum included: Pyknosis in some nuclei of granulosa cells, Karyorrhexis, Karyolysis in some granulosa cells, and necrosis in corpus luteum, with additional significant decrease in the average of diameters of corpus luteum at level (P
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Dissertations / Theses on the topic "Corpus luteum hormones"

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Prata, Alexandre Barbieri. "Ovarian function, steroides hormones and fertility in cows stimulated with gonadotropins." Universidade de São Paulo, 2018. http://www.teses.usp.br/teses/disponiveis/11/11139/tde-02052018-182855/.

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To increase profitability in beef and dairy cattle operations, adequate reproductive management strategies that provide high service and conception rates, especially at the beginning of the breeding season for beef cattle, and after the voluntary waiting period for dairy herds, are necessary. To achieve these goals various hormonal protocols have been developed to synchronize the emergence of a new follicular wave, estrus and ovulation, thus allowing fixed time artificial insemination (FTAI). Treatment with eCG has been included in FTAI protocols. Considering that eCG is an indispensable tool for reproductive management, a better understanding of its biological action in the final follicular growth process, ovulation and luteal development is crucial to optimize its use in hormonal protocols. At the same time, alternatives for eCG need to be tested. In this regard, it is important a better understanding of how FSH and LH act in the dominant follicle. Based on that, three studies were performed. The first study evaluated effects of eCG on fertility of 679 crossbred lactating grazing cows synchronized for FTAI. Treatment with eCG tended to increase P/AI at 30 and 60 days and increased P/AI at 30 and 60 days for cows inseminated at ≤ 70 DIM but had no effect in cows receiving AI after 70 DIM. The second study evaluated the effect of eCG or different doses of hCG on the final growth of the dominant follicle in 84 Nelore cows submitted FTAI. No differences were observed for the diameter of the largest follicle on D8 or D10. However, the growth rate of the dominant follicle between D8 and D10 was greater for the groups eCG and hCG 300. In addition, more cows from the Groups hCG 300 and hCG 200 SC presented premature ovulation. Treatment with different hCG doses on D8 of a FTAI protocol failed to produce similar effects compared to eCG. The third study evaluated the effect of eCG, FSH, or hCG on follicular growth rate, ovulatory follicle size, CL volume and circulating E2 and P4 concentrations, as well as the number of large and small luteal cells in cows submitted to a protocol for synchronization of ovulation. Seventeen non-lactating Nelore cows were used. Two Latin squares were done, totaling eight replicates. The gonadotropin treatments, eCG, FSH, or hCG, were effective in increasing the follicular growth rate between D7-10 and consequently the follicular diameter on D10 and ovulatory follicle diameter in comparison to Control. In addition, treatment with different gonadotropins increased the number of large and small luteal cells, however, there was no difference in preovulatory E2 peak concentration, CL volume and circulating P4 concentration post ovulation.
Para aumentar a rentabilidade nos sistemas de produção de bovinos de corte e leiteiro, são necessárias estratégias de manejo reprodutivo que proporcionem elevadas taxas de serviço e concepção, especialmente no início da estação de monta em bovinos de corte e após o período de espera voluntária para rebanhos leiteiros. Para atingir esses objetivos, vários protocolos hormonais foram desenvolvidos com o intuito de sincronizar a onda folicular, o estro e a ovulação, permitindo assim inseminação artificial em tempo fixo (IATF). Considerando que a eCG é uma ferramenta indispensável para o manejo reprodutivo, é fundamental uma melhor compreensão de sua ação biológica no processo de crescimento folicular final, ovulação e desenvolvimento luteal, otimizando seu uso em protocolos hormonais. Além disso, alternativas para a eCG precisam ser testadas. Dessa forma é importante uma melhor compreensão de como FSH e LH atuam no folículo dominante. Com base nisso, três estudos foram realizados. O primeiro avaliou os efeitos da eCG na fertilidade de 679 vacas lactantes mestiças em sistema de pastejo sincronizadas para IATF. O tratamento com eCG tendeu em aumentar a P/IA aos 30 e 60 dias e aumentou a P/IA aos 30 e 60 dias para vacas inseminadas com DEL ≤ 70, mas não houve efeito nas vacas que receberam IA após 70 DEL. O segundo avaliou o efeito da eCG ou diferentes doses de hCG no crescimento final do folículo dominante em 84 vacas Nelore submetidas a IATF. Não houve diferença quanto ao diâmetro do maior folículo no D8 ou D10. No entanto, a taxa de crescimento folicular entre D8 e D10 foi maior para os grupos eCG e hCG 300. No entanto, mais vacas dos Grupos hCG 300 e hCG 200 SC apresentaram ovulação precoce. O tratamento com diferentes doses de hCG no D8 do protocolo de IATF não produziu efeitos semelhantes em relação à eCG. O terceiro estudo avaliou o efeito de eCG, FSH ou hCG na taxa de crescimento folicular, tamanho do folículo ovulatório, volume de CL e concentrações circulantes de E2 e P4, bem como o número de células lúteas grandes e pequenas em vacas submetidas a sincronização da ovulação. Foram utilizadas 17 vacas Nelore não lactantes em delineamento com dois quadrados latinos, totalizando oito réplicas. Os tratamentos com as gonadotrofinas eCG, FSH, ou hCG foram efetivos em aumentar a taxa de crescimento folicular entre D7-10 e consequentemente o diâmetro folicular no D10 e o diâmetro do folículo ovulatório em relação ao Controle. Além disso, o tratamento com diferentes gonadotrofinas aumentou o número de células lúteas grandes e pequenas sem, entretanto, se detectar diferenças no pico pré-ovulatório de E2, volume luteal e concentração circulante de P4 após a ovulação.
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Smith, George W. "Local regulators of corpus luteum function /." free to MU campus, to others for purchase, 1996. http://wwwlib.umi.com/cr/mo/fullcit?p9717154.

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Petroff, Brian Kelli. "Mechanisms of Hormone Action in the Porcine Corpus Luteum /." The Ohio State University, 1996. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487934589976858.

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Xu, Jing. "Luteinizing hormone-regulated genes and corticotropin releasing hormone/urocortin-receptor-binding protein system in the primate corpus luteum during the menstrual cycle : a dissertation /." Restricted access until December 2006 at:, 2006. http://content.ohsu.edu/u?/etd,148.

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Gåfvels, Mats. "Blood flow and metabolism in the corpus luteum of the rat : in vivo and in vitro studies on the ovarian luteal and follicular compartment of the rat." Doctoral thesis, Umeå universitet, Fysiologi, 1987. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-102824.

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The ovary undergoes cyclic changes in follicular growth and luteogenesis due to the action of gonadotropins and steroids. The ovary and especially the corpus luteum has an exteremely high blood flow. There is a gap in our knowledge about the physiological role of the high blood flow of the corpus luteum. The production of lactate, progesterone and cyclic AMP of follicles and corpora lutea incubated in vitro was analyzed and related to the tissue content of ATP to elucidate possible connections between oxygen and substrate levels and energy consumption, steroid output and LH responsiveness in vitro. It was also considered of interest to investigate if the oxygen tensions needed for ATP and progesterone production of the follicle and the corpus luteum differed. A corpus luteum model using adult pseudopregnant rats was developed and characterized according to criteria for identification of corpora lutea as well as levels of plasma steroids and gonadotropins. In vitro progesterone production was compared to plasma progesterone levels. The absolute blood flow of corpora lutea of different ages and the response to injection of hCG, noradrenaline and antidiuretic hormone was investigated with the microsphere technique. Relative blood flow changes of follicles and corpora lutea during follicular growth and luteogenesis in vivo were studied by injecting radiolabelled microspheres to anaesthetized immature rats at different time periods after injection of an ovulatory dose of pregnant mare serum gonadotropin. This approach was chosen to investigate the possible relation between follicular/luteal blood flow, steroid output and morphology in relation to the endogenous gonadotropin surge, ovulation and luteogenesis. Hormonal stimulation by injection of hCG and noradrenaline increased total ovarian blood flow but no evidence was found for a parallelism between luteotropism and blood flow. The increasing effect of hCG on ovarian blood flow was partly due to a shunting of blood from the uterus towards the ovary. The antidiuretic hormone potently decreased ovarian and uterine blood flow by 80-90% while blood flow of some other organs (e.g. kidney and spleen) were hardly affected. The corpus luteum of pseudopregnancy was found to produce 15“ 20 times more progesterone in vitro as compared to the preovulatory follicle. The steroidogenesis and energy production of corpora lutea was found to be more sensitive to decreases in oxygen tension in terms of tissue ATP levels and LH responsiveness of progesterone production while the follicle could compensate by increasing glycolysis. A parallelism between follicular/luteal blood flow and progesterone production in vivo was found. It was shown that the formation, growth and progesterone production of the corpus luteum was accompanied by an increase in blood flow as well as vascularization as seen under the light microscope. The endogenous gonadotropin surge did not change follicular blood flow due to the development of a follicular oedema. We hypothesize that the corpus luteum function in vivo and in vitro is dependent on higher energy levels than the preovulatory follicle and that the transformation of the follicle to a corpus luteum is supported by a high nutritive blood flow possibly to support a high demand for energy-rich substrates.

Diss. (sammanfattning) Umeå : Umeå universitet, 1987, härtill 7 uppsatser.


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Salfen, Brent Edward. "Effect of the dominant ovarian follicle on the establishment and regulation of postpartum estrous cycles in dairy and beef animals /." free to MU campus, to others for purchase, 2000. http://wwwlib.umi.com/cr/mo/fullcit?p9974683.

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Kelly, Christopher Mark 1962. "THE EQUINE CORPUS LUTEUM: IN VIVO AND IN VITRO RESPONSIVENESS TO GONADOTROPIN STIMULATION." Thesis, The University of Arizona, 1987. http://hdl.handle.net/10150/291492.

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Gonadotropins were used to stimulate luteal function, as determined by progesterone secretion, in both in vitro and in vivo systems. LH and hCG were capable of significantly stimulating progesterone secretion in the in vivo systems. Stimulation of progesterone secretion by hCG was greater than that for LH. PMSG failed to increase progesterone production at any level of treatment. hCG was also used to stimulate progesterone production by the corpus luteum in mares during early gestation. hCG administration resulted in a significant (p < 0.10) increase in peripheral progesterone levels in treatment mares through day 14 post-estrus. Peripheral progesterone concentrations were also higher in hCG treated mares for days 15 through 30 post-estrus in mares that conceived. hCG treatment had no influence on anterior pituitary release of LH.
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McIntush, Eric W. "Tissue inhibitor of metalloproteinases (TIMP-1) in luteal function /." free to MU campus, to others for purchase, 1996. http://wwwlib.umi.com/cr/mo/fullcit?p9737849.

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Davis, Tracy Leigh. "Role of the endocrine and immune systems in the developing and regressing corpus luteum." Connect to this title online, 2004. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1085166703.

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Thesis (Ph. D.)--Ohio State University, 2004.
Title from first page of PDF file. Document formatted into pages; contains xvi, 202 p.; also includes graphics (some col.). Includes bibliographical references (p. 151-185). Available online via OhioLINK's ETD Center
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Hild-Petito, Sheri Ann. "Distribution of estrogen and progesterone receptors in the primate ovary, with emphasis on subpopulations of cells within the corpus luteum." Diss., The University of Arizona, 1988. http://hdl.handle.net/10150/184485.

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Both estradiol and progeterone are proposed autocrine or paracrine regulators of ovarian function in primate species. However, specific receptors for these steroids have not been localized to individual compartments of the primate ovary. Using immunocytochemical techniques, estradiol receptors were detected in the germinal epithelium, but not other structures, of ovaries obtained from rhesus or cynomolgus monkeys during the follicular and luteal phases of the menstrual cycle. In contrast, progesterone receptors were present in stromal and interstitial tissue, the thecal layers of healthy and atretic follicles, as well as the functional corpus luteum. These results are consistent with the concept of a receptor-mediated role for progesterone, but not estrogen, within the predominant gametogenic and endocrine structures, e.g., the follicle and corpus luteum, of the primate ovary. The recent discovery of distinct cell types in the corpus luteum of domestic ungulates has revised concepts on the control of luteal function in these species. Studies were designed to test the hypothesis that the primate corpus luteum consists of cell subpopulations that differ in physical characteristics, function and regulation. Cells enzymatically-dispersed from the monkey corpus luteum at mid-luteal phase of the menstrual cycle differed in size (diameter) and the presence of the steroidogenic enzyme, 3β-hydroxysteroid dehydrogenase (3β-HSD). Analysis of dispersed cells for forward and 90° light scatter properties by flow cytometry revealed two distinct continua (Cα and Cβ). These continua were isolated using the sorting capabilities of the flow cytometer. Cα contained single cells of ≤ 15 μm and cell clusters; the cells were typically 3β-HSD-negative nonsteroidogenic. Cβ consisted of single cells that increased in size up to 40 μm and were 3β-HSD-positive. Cβ was divided into two regions (R₁ and R₃) and the cells isolated. R₁ cells were ≤ 15 μm whereas R₃ cells were ≥ 20 μm. Basal progesterone and estrogen production by R₃ cells was greater than that produced by R₁ cells (as determined by radioimmunoassay of the incubation media). Relative stimulation of progesterone production by hCG, cAMP or PGE₂ was not different between R₁ and R₃ luteal cells. These results support the hypothesis that the primate corpus luteum consists of distinct cell subpopulations which differ in size and steroidogenic capacity. However, the cell types which secrete progesterone are typically responsive to gonadotropin and PGE₂, possibly via a cAMP-mediated pathway.
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Books on the topic "Corpus luteum hormones"

1

Primate Ovary Symposium (1987 Beaverton, Or.). The primate ovary. New York: Plenum Press, 1987.

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The Primate Ovary (Serona Symposiausa). Springer, 1988.

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Rodger, Lynn Dian. Gonadotropin releasing hormone-induced alteration of corpus luteum function in beef heifers. 1985.

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Martin, Teri L. Response of the bovine corpus luteum to exogenous gonadotropin-releasing hormone during the estrous cycle. 1988.

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Whitmore, Diana L. Corpus luteum function in hysterectomized and unilaterally hysterectomized ewes treated with gonadotropin-releasing hormone. 1995.

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Using gonadotropin-releasing hormone (GnRH) at breeding to enhance corpus luteum function in dairy cows. Honolulu, Hawaii: HITAHR, College of Tropical Agriculture and Human Resources, University of Hawaii, 1990.

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Bertrand, Jennifer Elaine. Cellular mechanisms of altered bovine luteal function in response to exogenous gonadotropin-releasing hormone. 1995.

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Book chapters on the topic "Corpus luteum hormones"

1

Xavier, Françoise. "Functional Morphology and Regulation of the Corpus Luteum." In Hormones and Reproduction in Fishes, Amphibians, and Reptiles, 241–82. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1869-9_9.

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Rekawiecki, Robert, Magdalena K. Kowalik, and Jan Kotwica. "Steroid Hormone Receptors in the Corpus Luteum." In The Life Cycle of the Corpus Luteum, 79–97. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-43238-0_5.

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Stouffer, Richard L. "Corpus Luteum in Primates." In Encyclopedia of Hormones, 288–97. Elsevier, 2003. http://dx.doi.org/10.1016/b0-12-341103-3/00051-6.

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Zeleznik, Anthony J. "Corpus Luteum: Regression and Rescue." In Encyclopedia of Hormones, 284–88. Elsevier, 2003. http://dx.doi.org/10.1016/b0-12-341103-3/00050-4.

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Wuttke, W., L. Pitzel, D. Seidlová-Wuttke, and B. Hinney. "LH pulses and the corpus luteum: The luteal phase deficiency (LPD)." In Vitamins & Hormones, 131–58. Elsevier, 2001. http://dx.doi.org/10.1016/s0083-6729(01)63005-x.

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Gangat, Naseema. "Benign Hematologic Disorders." In Mayo Clinic Internal Medicine Board Review, 399–414. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190464868.003.0037.

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The menstrual cycle is composed of the follicular (proliferative), periovulatory, and luteal (secretory) phases. At periovulation, the mature follicle triggers a surge in luteinizing hormone level, causing ovum release and stimulating the residual ovarian follicle to transform into a corpus luteum. Circulating estrogen and progestin levels increase. A thickened, enriched endometrium develops owing to progestin secretion from the corpus luteum. Without fertilization, the corpus luteum atrophies, estrogen and progestin levels decline, follicle-stimulating hormone release is stimulated, and the endometrium sloughs.
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"Corpus-luteum-Hormon." In Springer Reference Medizin, 623. Berlin, Heidelberg: Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-48986-4_310891.

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Ojeda, Sergio R. "Female Reproductive Function." In Textbook of Endocrine Physiology. Oxford University Press, 2011. http://dx.doi.org/10.1093/oso/9780199744121.003.0011.

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The production of germ cells is essential for the continuation of a species. In the female this function is accomplished by the ovaries. In addition, the ovaries secrete steroids and nonsteroidal hormones that not only regulate the secretion of anterior pituitary hormones but also act on various target organs, including the ovaries themselves, the uterus, fallopian tubes, vagina, mammary gland, and bone. Morphologically, the ovary has three regions: an outer cortex that contains the oocytes and represents most of the mass of the ovary; the inner medulla, formed by stromal cells and cells with steroid-producing characteristics; and the hilum, which, in addition to serving as the point of entry of the nerves and blood vessels, represents the attachment region of the gland to the mesovarium. The cortex, which is enveloped by the germinal epithelium, contains the follicles, which are the functional units of the ovary. They are present in different states of development or degeneration (atresia), each enclosing an oocyte. In addition to the oocyte, ovarian follicles have two other cellular components: granulosa cells, which surround the oocyte, and thecal cells, which are separated from the granulosa cells by a basal membrane and are arranged in concentric layers around this membrane. The follicles are embedded in the stroma, which is composed of supportive connective cells similar to that of other tissues, interstitial secretory cells, and neurovascular elements. The medulla has a heterogeneous population of cells, some of which are morphologically similar to the Leydig cells in the testes. These cells predominate in the ovarian hilum; their neoplastic transformation results in excess androgen production. The ovary produces both steroids and peptidergic hormones. Whereas the steroids are synthesized in both interstitial and follicular cells, peptidergic hormones are primarily produced in follicular cells and, after ovulation, by cells of the corpus luteum. The initial precursor for steroid biosynthesis is cholesterol, which derives from animal fats of the diet or from local synthesis.
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Mitchell, Graham. "Reproduction and the Fetus." In How Giraffes Work, 433–80. Oxford University Press, 2021. http://dx.doi.org/10.1093/oso/9780197571194.003.0017.

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The reproductive organs of male and female giraffes are similar to those of all other artiodactyls. Giraffes have 14 pairs of autosomes and a pair of sex chromosomes. A constant testis temperature depends on countercurrent heat exchange between a large pampiniform venous plexus and the testicular artery. The onset of puberty in males and females occurs at ~3 years of age and is marked by enlargement of testes and the onset of oestrous cycles. Oestrus cycles are ~15 days long. Courtship, conception, and pregnancy are delayed until ~5 years of age. The giraffe placenta is polycotyledonous and epitheliochorial. Pregnancy is sustained by progesterone secreted by a single corpus luteum, the placenta and fetal ovaries and testes. Gestation lasts ~450 days and is ended by hormones secreted by the hypothalamic-pituitary axis of a mature fetus. Birth takes ~30 minutes. Daily milk yield ranges between 2.5 L and 10 L. Protein, fat, and lactose are higher than in cow’s milk. Weaning occurs at ~6 months of age. The average calving interval is ~20 months but varies according to the degree of lactational stress itself partly determined by whether a calf survives. Female giraffes have ~8 calves of which ~3 will reach adulthood. Survival of giraffes as a species depends on fewer adults dying each year than the number of calves reaching adulthood.
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Yonezawa, Tomohiro, Atsushi Shioya, Shiro Kurusu, and Mitsumori Kawaminami. "Changes in Corpus Luteal Expression of Prolactin Receptors during Luteal Phase of Rats: Implication of Local Gonadotropin-Releasing Hormone (GnRH) Expression." In BASIC/TRANSLATIONAL - Female Reproduction & Mammary Gland, P2–202—P2–202. The Endocrine Society, 2011. http://dx.doi.org/10.1210/endo-meetings.2011.part2.p30.p2-202.

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