Relevant bibliographies by topics / Cow brain

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Cow brain'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Cow brain"

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Gökyar, Ahmet, and Cengiz Cokluk. "Using of Fresh Cadaveric Cow Brain in the Microsurgical Training Model for Sulcal-Cisternal and Fissural Dissection." Journal of Neurosciences in Rural Practice 09, no. 01 (January 2018): 026–29. http://dx.doi.org/10.4103/jnrp.jnrp_390_17.

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ABSTRACT Objective: The aim of this experimental study was to evaluate the feasibility of using fresh cadaveric cow brain as in a training model for microsurgical dissection of sulcus, cisterns, and fissure. Methods: Experimental microneurosurgical activities in this study were performed under the operating microscope. Bilateral sylvian cisterns, interhemispheric fissure, and hemispheric sulcus of the fresh cadaveric cow brain were used as an interested area for this experimental study. The dissection was continued reaching down to the floor of the cistern and total dissection of the middle cerebral artery inside the cisternal space. The suitability of a cow brain as a training model for sylvian fissure microdissection was evaluated as three groups; bad, good, and perfect. Results: Ten uncovered fresh cadaveric cow brains were used in this experimental feasibility study. The suitability of the experiment for training model was evaluated as bad in (1) 10% of the fresh cadaveric cow brains. The suitability was found as good in (6) 60% of the procedures. In the remaining (3) 30% of the brain dissection, the suitability of the experiment was evaluated as perfect. Conclusion: In conclusion, performing the sulcal, cisternal, and fissural dissection and protecting the neural and vascular tissue from the mechanical bruising effect of metallic microsurgical instruments are feasible as shown in this study. We believe that this training model will contribute to the practical micro-neurosurgery. Additionally, it provides adequate performance for the microsurgical intervention.
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Widhiyanuriyawan, Denny, Prihanto Trihutomo, Sudjito Soeparman, and Lilis Yuliati. "Zwitterion Effect of Cow Brain Protein towards Efficiency Improvement of Dye-Sensitized Solar Cell (DSSC)." Scientific World Journal 2020 (February 19, 2020): 1–12. http://dx.doi.org/10.1155/2020/7910702.

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Dye-Sensitized Solar Cell (DSSC) constitutes a solar cell using natural dyes from plants that are adsorbed in semiconductors to convert solar energy into electrical energy. DSSC has relatively inexpensive fabrication costs, is easy to produce, works in visible light, and is environmentally friendly. The disadvantage of DSSC is that its efficiency is still low compared to silicon solar cells. This low efficiency is due to obstacles in the flow of electric current on DSSC. In this study, DSSC has been successfully fabricated with the deposition of clathrin protein from cow brain. The zwitterions effect of protein on cow brain is able to reduce resistance and increase electric current on DSSC. The zwitterions effect of cow brain protein that fills gaps or empty spaces between TiO2 particles generates acidic reactions (capturing electrons) and bases (releasing electrons); hence, proteins in the cow brain are able to function as electron bridges between TiO2 molecules and generate an increase in electric current in DSSC. The method used in this research was to deposit clathrin protein from cow brain in a porous TiO2 semiconductor with a concentration of 0%, 25%, 50%, and 75%. Tests carried out on DSSC that have been performed were X-Ray Diffractometer (XRD) testing to determine the crystal structure formed, Fourier Transform Infrared Spectroscopy (FTIR) testing to determine the functional groups formed on DSSC, Scanning Electron Microscopy (SEM) testing to determine the surface morphological characteristics of the DSSC layer, and testing the efficiency using AM 1.5 G solar simulator (1000 W/m2) to determine the efficiency changes that occur in DSSC. From the XRD test results by increasing the concentration of cow brain protein in DSSC, the structure of amino acid crystals also increased and the crystal size increased with the largest crystal size of 42.25 nm at the addition of 75% of cow brain protein. FTIR test results show that the addition of cow brain protein will form functional protein-forming amino groups on DSSC. FTIR analysis shows the sharp absorption of energy by protein functional groups in the FTIR spectrum with increasing concentration of cow brain protein in DSSC. The SEM test results show that the concentration of additional molecules of protein deposited into TiO2 increases and the cavity or pore between the TiO2 molecules decreases. The reduction of cavities in the layers indicates that protein molecules fill cavities that exist between TiO2 molecules. From the results of testing using AM 1.5 G solar simulator (1000 W/m2), the highest efficiency value is 1.465% with the addition of 75% brain protein concentration.
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Sant'Ana, Fabiano José Ferreira de, Adriane Loy Gabriel, Glaucia Denise Kommers, and Claudio Severo Lombardo de Barros. "Choroid plexus carcinoma in a cow." Ciência Rural 39, no. 7 (July 31, 2009): 2229–32. http://dx.doi.org/10.1590/s0103-84782009005000150.

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A red, firm 3.5x3.0x3.0cm mass was found within the right lateral ventricle, compressing the hippocampus and thalamus of the brain of a 4-year-old cow. The pituitary was enlarged and firm. Histologically, both the brain and pituitary masses consisted of moderately pleomorphic epithelial cells arranged as rosettes or sheets supported by a delicate fibrovascular stroma. Based on anatomical localization, histopathological and immunohistochemical findings, a diagnosis of choroid plexus carcinoma with pituitary metastasis was made.
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Yanai, T., T. Masegi, M. Iwanaka, K. Yoshida, K. Ueda, Y. Suzuki, and S. Kamiya. "Polyglucosan bodies in the brain of a cow." Acta Neuropathologica 88, no. 1 (July 1994): 75–77. http://dx.doi.org/10.1007/bf00294362.

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Антипин, Алексей, Aleksey Antipin, Григорий Надточей, and Grigoriy Nadtochey. "Detection of prion protein, the infection agent of bovine spongiform encephalopathy, by using aptamers." Russian veterinary journal 2019, no. 6 (November 27, 2019): 5–8. http://dx.doi.org/10.32416/2500-4379-2019-2019-6-5-8.

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In the paper results of the specific interaction of selected DNA aptamers with prion protein from CNS tissues from DSE infected cows are described. The test detects amyloid epitops. The purpose of investigation. To study the possibility of detection of prion protein (BSE infectious agent in tissues CNS infected cows), using DNA aptamers. The DNA aptamers had been selected to yeast prions. Materials and methods. CNS tissues samples from a DSE experimentally infected cow were used. The infection was confirmed by histological analyses. The prion protein of BSE was isolated by ultracentrifugation in sucrose gradient and analysed, using immunoblotting and electron microscopy. Results. DSE infection in an experimentally infected cow was confirmed. The brain tissue samples were prepared. Specific effective binding of selected DNA aptamers with prions from DSE cow brain tissues was revealed. Conclusion. The possibility of detection of prion proteins in brain tissues from a DSE cow, using DNA aptamers, specifically binded with amyloid epitops was shown.
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Prisilia, Fiola Hamanda, Yhulia Praptinngsih, and Rizka Rian Fauziah. "KARAKTERISTIK SOSIS BERBAHAN BAKU CAMPURAN JAMUR TIRAM PUTIH (Pleurotus ostreatus) DAN OTAK SAPI." JURNAL AGROTEKNOLOGI 11, no. 02 (January 12, 2018): 117. http://dx.doi.org/10.19184/j-agt.v11i02.6516.

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sage is a processed product which have meat as its main ingredients and the extender ingredients are binders, filler, sugar, salt and also seasoning then mixed and put it into a casing. Commonly sausage use meat as its raw ingredient.The expensive price of meat will cause the price of sausage also increasing, so then an innovation to use another main ingredients as the alternative of meat that is cheaper, such as white oyster mushroom. Oyster mushroom have protein and fiber but also contain low level of fat, so it need to increase of fat content by cow’s brain. Cow’s brain have high of fat content so that it will be useful to increase the fat content in the oyster mushroom mixed with cow brain sausage. The purpose of this experiment is to find the right ratio in the process of mixing white oyster mushroom with cow brain. This experiment was conducted by the Complete Randomized Design. The treatment was ratio of oyster mushroom and cow brain it had 5 levels. The oyster mushroom and cow brain ratio are P1 (90:10); P2 (80:20); P3 (70:30); P4 (60:40); P5(50:50). The parameter of the observation are texture, colour, cooking loss,apperance of the slice, moisture content, fat content, and sensoric properties was the preference of colour, flavour, texture, taste and overall. The best treatment was analysis of proximate test. Based on effectiveness test from this research, the best formulation was on P5 treatment (oyster mushroom and cow meat ratio 50:50). The sausage had score of texture of 63 g/1,5mm; color 57,59; cooking loss 7,11%; moisture content 60,31%; fat content 4,22%; protein content 21,05%; ash content 2,08%; carbohydrate content 12,34%; preference of colour, smell, texture, taste and overall was 5,7;6,36;5,36;5,75;5,92 (neutral- a bit of contentment). Keywords: sausage, oyster mushrooms, cow’s brain, ratio
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Neimark, Matthew A., Angelos-Aristeidis Konstas, Andrew F. Laine, and John Pile-Spellman. "Integration of jugular venous return and circle of Willis in a theoretical human model of selective brain cooling." Journal of Applied Physiology 103, no. 5 (November 2007): 1837–47. http://dx.doi.org/10.1152/japplphysiol.00542.2007.

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A three-dimensional mathematical model was developed to examine the induction of selective brain cooling (SBC) in the human brain by intracarotid cold (2.8°C) saline infusion (ICSI) at 30 ml/min. The Pennes bioheat equation was used to propagate brain temperature. The effect of cooled jugular venous return was investigated, along with the effect of the circle of Willis (CoW) on the intracerebral temperature distribution. The complete CoW, missing A1 variant (mA1), and fetal P1 variant (fP1) were simulated. ICSI induced moderate hypothermia (defined as 32–34°C) in the internal carotid artery (ICA) territory within 5 min. Incorporation of the complete CoW resulted in a similar level of hypothermia in the ICA territory. In addition, the anterior communicating artery and ipsilateral posterior communicating artery distributed cool blood to the contralateral anterior and ipsilateral posterior territories, respectively, imparting mild hypothermia (35 and 35.5°C respectively). The mA1 and fP1 variants allowed for sufficient cooling of the middle cerebral territory (30–32°C). The simulations suggest that ICSI is feasible and may be the fastest method of inducing hypothermia. Moreover, the effect of convective heat transfer via the complete CoW and its variants underlies the important role of CoW anatomy in intracerebral temperature distributions during SBC.
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Piechna, A., and K. Cieslicki. "Reversed Robin Hood Syndrome in the Light of Nonlinear Model of Cerebral Circulation." International Journal of Applied Mechanics and Engineering 22, no. 2 (May 24, 2017): 459–64. http://dx.doi.org/10.1515/ijame-2017-0029.

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AbstractThe brain is supplied by the internal carotid and vertebro-basilar systems of vessels interconnected by arterial anastomoses and forming at the base of the brain a structure called the Circle of Willis (CoW). An active intrinsic ability of cerebral vascular bed maintains constant Cerebral Blood Flow (CBF) in a certain range of systemic pressure changes. This ability is called autoregulation and together with the redundant structure of the CoW guarantee maintaining CBF even in partial occlusion of supplying arteries. However, there are some situations when the combination of those two mechanisms causes an opposite effect called the Reversed Robin Hood Syndrome (RRHS). In this work we proposed a model of the CoW with autoregulation mechanism and investigated a RRHS which may occur in the case of Internal Carotid Artery (ICA) stenosis combined with hypercapnia. We showed and analyzed the mechanism of stealing the blood by the contralateral side of the brain. Our results were qualitatively compared with the clinical reports available in the literature.
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Strömberg, Elisabeth, and Margareta Wallin. "Differences in the effect of Ca2+on isolated microtubules from cod and cow brain." Cell Motility and the Cytoskeleton 28, no. 1 (1994): 59–68. http://dx.doi.org/10.1002/cm.970280106.

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Oruç, E. "Meningoencephalitis Tuberculosa in a Holstein Cow." Veterinary Pathology 42, no. 6 (November 2005): 856–58. http://dx.doi.org/10.1354/vp.42-6-856.

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The gross and histopathologic lesions of meningoencephalitis tuberculosa in a 4-year-old Holstein cow showing clinical signs compatible with bovine spongiform encephalopathy are described in this report. Grossly, numerous gray to yellow, firm and caseous nodules were seen on the ventral surfaces of the brain and in the lateral and fourth ventricles. Histopathologically, foci of caseation and dystrophic mineralization were surrounded by multinucleated giant cells, epitheloid macrophages, plasma cells, lymphocytes and fibrous proliferation. Ziehl-Neelsen stains of the lesions revealed masses of slender acid-fast bacilli in the necrotic centers of lesions and within surrounding giant cells.
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Dissertations / Theses on the topic "Cow brain"

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Elander, Louise. "Prostaglandin E2 in Brain-mediated Illness Responses." Doctoral thesis, Linköpings universitet, Cellbiologi, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-53701.

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We are unceasingly exposed to potentially harmful microorganisms. The battle against threatening infectious agents includes activation of both the innate and of the adaptive immune systems. Illness responses are elicited and include inflammation, fever, decreased appetite, lethargy and increased sensitivity to painful stimuli in order to defeat invaders. While many of these signs of disease are controlled by the central nervous system, it has remained an enigma how signals from the peripheral immune system reach the brain through its blood-brain barrier, which precludes macromolecules, including cytokines, from diffusing into the brain parenchyma. Previous findings indicate the existence of a pathway across the blood-brain barrier, which includes binding of the cytokine interleukin-1 (IL-1) to its receptor in the brain vessels, thereby inducing the production of the prostaglandin E2 (PGE2) synthesizing enzymes cyclooxygenase-2 (Cox-2) and microsomal prostaglandin E synthase-1 (mPGES-1), which ultimately synthesize PGE2. PGE2 subsequently binds to any of the four prostaglandin E2 (EP) -receptors. Previous results from our laboratory have suggested that this pathway plays a critical role in the febrile response to infectious stimuli. The present thesis aims at further investigating the molecular events underlying immune-to-brain signalling, with special emphasis on fever, hypothalamic-pituitary-adrenal (HPA) -axis activation and anorexia and their connection to signalling molecules of the cytokine and prostaglandin families, respectively. In paper I, the molecular processes linking the proinflammatory cytokine interleukin-6 (IL-6) and PGE2 in the febrile response were investigated. Both IL-6 and PGE2 have been shown to be critical players in the febrile response, although the molecular connections are not known, i.e. if IL-6 exerts its effects up- or downstream of PGE2. Mice deficient in IL-6 were unable to respond to bacterial lipopolysaccharide (LPS) with a febrile response, but displayed similar induction of Cox-2 and mPGES-1, and similar concentrations of PGE2 in the cerebrospinal fluid as wild-type mice. Paradoxically, the IL-6 deficient mice responded with a dose-dependent elevation of body temperature in response to intracerebroventricularly injected PGE2. Furthermore, IL-6 per se was not pyrogenic when injected peripherally in mice, and did not cause increased levels of PGE2 in cerebrospinal fluid. IL-6 deficient mice were not refractory to the action of PGE2 because of excess production of some hypothermia-producing factor, since administration of a Cox-2 inhibitor in LPS-challenged IL-6 deficient mice did not unmask any hypothermic response, and neutralization of tumor necrosis factor α (TNFα), associated with hypothermia, did not produce fever in LPS-challenged IL-6 deficient mice. These data indicate that IL-6 rather than exerting its effects up- or down-stream of PGE2 affects some process in parallel to PGE2, perhaps by influencing the diffusion and binding of PGE2 onto its target neurons. In papers II and III, we injected the proinflammatory cytokine IL-1β in free-fed wild-type mice, in mice with a deletion of the gene encoding mPGES-1, or in mice deficient in the EP1, EP2 and EP3. Food intake was continuously measured during their active period, revealing that mPGES-1 deficient mice were almost completely resistant to anorexia induced by IL-1β. However, all of the investigated EP receptor deficient mice exhibited a normal profound anorexic response to IL-1β challenge, suggesting that the EP4 is the critical receptor that mediates IL-1β-induced anorexia. We also investigated the role of mPGES-1 in anorexia induced by lipopolysaccharide (LPS) in mPGES-1 deficient mice. The profound anorexic response after LPS-challenge was similar in mPGES-1 deficient and wild-type mice. To further investigate the anorectic behaviour after LPS injection, we pre-starved the animals for 22 hours before injecting them with LPS. In this paradigm, the anorexia was less profound in mPGES-1 knock-out mice. Our results suggest that while the inflammatory anorexia elicited by peripheral IL-1β seems largely to be dependent on mPGES-1-mediated PGE2 synthesis, similar to the febrile response, the LPS-induced anorexia is independent of this mechanism in free-fed mice but not in pre-starved animals. In papers IV and V, the role of prostanoids for the immune-induced HPA-axis response was investigated in mice after genetic deletion or pharmacological inhibition of prostanoid-synthesizing enzymes, including Cox-1, Cox-2, and mPGES-1. The immediate LPS-induced release of ACTH (adrenocorticotropic hormone and corticosteroids was critically dependent on Cox-1 derived prostanoids and occurred independently of Cox-2 and mPGES-1 derived PGE2. In contrast, the delayed HPA-axis response was critically dependent on immune-induced PGE2, synthesized by Cox-2 and mPGES-1, and occurred independently of Cox-1 derived enzymes. In addition, in the mPGES-1 deficient mice, the synthesis of CRH hnRNA and mRNA was decreased in the paraventricular nucleus of the hypothalamus after LPS-challenge, indicating that the delayed hormone secretion was mediated by PGE2-induced gene-transcription of CRH in the hypothalamus. The expression of the c-fos gene and Fos protein, an index of synaptic activation, was maintained in the paraventricular nucleus and its brainstem afferents both after unselective and Cox-2 selective inhibition as well as in Cox-1, Cox-2, and mPGES-1 knock-out mice. This suggests that the immune-induced neuronal activation of autonomic relay nuclei occurs independently of prostanoid synthesis and that it is insufficient for eliciting stress hormone release.
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Salagic, Belma. "Regulation of COX-2 signaling in the blood brain barrier." Thesis, Linköping University, Linköping University, Department of Physics, Chemistry and Biology, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-18113.

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Upon an inflammation the immune system signals the brain by secreted cytokines to elicit central nervous responses such as fever, loss of appetite and secretion of stress hormones. Since the blood brain barrier, (BBB) protects the brain from unwanted material, molecules like cytokines are not allowed to cross the barrier and enter the brain. However, it is clear that they in some way can signal the brain upon an inflammation. Many suggestions concerning this signaling has been made, one being that cytokines bind to receptors on the endothelial cells of the blood vessels of the brain and trigger the production of prostaglandins that can cross the BBB. This conversion is catalyzed by the enzyme cyclooxygenase-2, (COX-2), which is induced by transcription factors like NF-κB in response to cytokines. One of the central nervous responses to inflammatory stimuli is activation of the HPA-axis whose main purpose is glucocorticoid production. Glucocorticoids inhibit the inflammatory response by suppressing gene transcription of pro-inflammatory genes including those producing prostaglandins through direct interference with transcription factors such as NF-κB or initiation of transcription of anti-inflammatory genes like IκB or IL-10. It has however not been clear if glucocorticoids can target the endothelial cells of the brain in order to provide negative feed-back on the immune-to-brain signaling, and in that way inhibit central nervous inflammatory symptoms. An anatomical prerequisite for such a mechanism would be that the induced prostaglandin production occurs in cells expressing GR. This has however never been demonstrated. Here I show that a majority of the brain endothelial cells expressing the prostaglandin synthesizing enzyme COX-2 in response to immune challenge also express the glucocorticoid receptor, (GR). This indicates that immune-to-brain signaling is a target for negative regulation of inflammatory signaling executed by glucocorticoids and identifies brain endothelial GR as a possible future drug target for treatment of central nervous responses to inflammation such as fever and pain.

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Tango, Humberto Katsuji. "Efeitos da hemodiluição normovolêmica aguda com Ringer lactato e hidroxietilamido na hipertensão intracraniana: estudo em cães com lesão criogênica do cérebro." Universidade de São Paulo, 2007. http://www.teses.usp.br/teses/disponiveis/5/5152/tde-10032008-155039/.

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INTRODUÇÃO: Em pacientes vítimas de trauma crânio-encefálico é fundamental que se restabeleça a volemia intravascular, quando associado à hipotensão arterial, com o intento de manter a pressão de perfusão e não agravar a lesão do sistema nervoso central. A hipovolemia pode ser corrigida com infusão rápida de soluções cristalóides e/ou colóides, quando hemoderivados não estão disponíveis. Nesta condição, o hematócrito (Ht) pode reduzir-se para valores muito baixos. A anemia aguda altera a viscosidade do sangue e pode interferir na reatividade vascular encefálica. O objetivo deste estudo foi avaliar a pressão intracraniana(PIC) na presença de lesão criogênica encefálica, quando se realiza hemodiluição aguda com Ringer lactato ou hidroxietilamido 450/0,7 a 6%, estabelecendo-se como meta reduzir o hematócrito de 40% para 35% ou para 27%. MÉTODOS: Foram utilizados 35 cães machos sem raça definida, cujo hematócrito inicial era superior a 40%, anestesiados e submetidos à lesão encefálica criogênica durante 20 minutos. Após foram aleatoriamente distribuídos em 5 grupos experimentais: HES35, hemodiluídos com hidroxietilamido até Ht de 35%; RL35, hemodiluídos com Ringer lactato até Ht de 35%; HES27, hemodiluídos com hidroxietilamido até Ht de 27%; RL 27, hemodiluídos com Ringer lactato até Ht de 27%; e controle, sem hemodiluição. As variáveis hemodinâmicas sistêmicas foram obtidas por meio de cateter de artéria pulmonar; a PIC foi medida por sensor introduzido no espaço subaracnóideo no hemisfério contralateral à lesão criogênica; as variáveis laboratoriais foram obtidas de amostras de sangue arterial. RESULTADOS: A lesão criogênica encefálica levou a aumento da PIC em todos os animais, sem diferença entre os grupos(p>0,5). Este aumento foi exacerbado somente nos animais hemodiluídos até hematócrito de 27%(p<0,03). A solução utilizada não influenciou o comportamento da PIC(p>0,5).
Objective: Brain injury is responsible for significant morbidity and mortality in trauma patients, but controversy still exists over optimal fluid management for these patients. This study aimed to investigate the effects of acute hemodilution with hydroxyethyl starch (HES) or lactated Ringer\'s solution (LR) in intracranial pressure(ICP) and cerebral perfusion pressure in dogs submitted to a cryogenic brain injury model. Design: Prospective laboratory animal study. Setting: Research laboratory in a teaching hospital. Subjects: Thirty-five male mongrel dogs. Interventions: Animals were enrolled to 5 groups: control, hemodilution with lactated Ringer\'s solution (RL) or hydroxyethyl starch (HES) 6% to an hematocrit target of 27% or 35%. Measurements and Main Results: ICP and CPP levels were measured after cryogenic brain injury. Hemodilution promotes an increment of ICP levels, which decreases CPP when hematocrit target was estimated in 27% after hemodilution. However, no differences were observed regarding crystalloid or colloid solution used for hemodilution in ICP and CPP levels. Conclusions: Hemodilution to a low hematocrit level increases ICP and decreases CPP scores in dogs submitted to a cryogenic brain injury. These results suggest that excessive hemodilution to a hematocrit below 30% should be avoided in traumatic brain injury patients.
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Taga, Marcio Luiz Lima. "Análise proteômica em cérebro de ratos submetidos a tratamento subcrônico com chumbo: influência da suplementação com ferro." Universidade de São Paulo, 2015. http://www.teses.usp.br/teses/disponiveis/25/25149/tde-26042016-105747/.

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O chumbo (Pb) é um metal pesado que pode ocasionar alterações em todos os sistemas. Porém os maiores danos à saúde ocorrem quando este acomete o sistema nervoso central (SNC). Muitos estudos demonstram as alterações clinicas/comportamentais causadas pela ação do Pb no SNC. Entretanto, ainda são necessários estudos que demonstrem as alterações bioquímicas causadas pelo Pb neste sistema. Por outro lado, tem sido relatado que o ferro (Fe) parece ter um efeito protetor na toxicidade cerebral causada pelo Pb. Assim, o objetivo deste estudo foi analisar a concentração de Pb no tecido cerebral, bem como realizar análise proteômica em cérebro de ratos intoxicados por Pb, submetidos à suplementação com Fe ou não. O experimento foi realizado com 30 ratos recém-desmamados (Rattus norvegicus, variedade Wistar) divididos em 6 grupos (n=5/grupo), de acordo com o tratamento recebido por 6 semanas, a saber: Controle (não exposto ao Pb ou Fe), Controle Fe (exposto à administração de 20 mg/Kg p.c. de FeSO4 a cada 2 dias, por gavagem gástrica), Pb 100 (exposto à água contendo 100 mg/L de Pb), Pb 400 (exposto à água contendo 400 mg/L de Pb) Pb100 + Fe (exposto à água contendo 100 mg/L de Pb e à gavagem com FeSO4) e Pb400 + Fe (exposto à água contendo 400 mg/L de Pb e à gavagem com FeSO4). Decorrido o período experimental, os animais foram eutanasiados e o cérebro dos animais foi removido, sendo descartados o cerebelo e o tronco encefálico. O restante foi submetido à concentração de Pb e à análise proteômica. Foi observada uma dose-resposta em relação à concentração de Pb no cérebro. A administração de FeSO4 reduziu os níveis de Pb no cérebro, embora sem significância estatística. A análise dos géis com os spots proteicos demonstrou uma redução na quantidade destes de acordo com o tratamento recebido pelos grupos. O grupo controle mostrou a maior quantidade de spots, ao passo que os grupos que receberam a maior concentração de Pb (400 mg/L) apresentaram as menores quantidade de spots. Também houve uma diminuição na quantidade de spots detectados quando administrado FeSO4. Dos spots que apresentaram diferença de expressão nas comparações entre o grupo controle e os grupos experimentais e os grupos experimentais comparados aos seus pares suplementados ou não com FeSO4, 75 proteínas foram identificadas por espectrometria de massas, sendo 20 proteínas (26,0%) relacionadas à função de metabolismo, 21 proteínas (28,0%) relacionadas a estrutura e organização das estruturas, 22 proteínas (30,0%) relacionadas à função de processos celulares, nove proteínas (12,0%) relacionadas às vias de informação e três proteínas (4,0%) pertencentes à categoria miscelânea. A expressão das proteínas dimunuiu na maioria das vezes, para todas as classificações funcionais. O presente estudo, associado a achados anteriores, aponta para um papel deletério do Pb no córtex cerebral dos animais expostos a este metal, independente da concentração utilizada, não sendo possível observar um efeito protetor do FeSO4. Este processo parece ser mediado por proteínas como Gamma-enolase, Alpha-internexin, várias isoformas de 14-3-3 e Homer protein homolog 1.
Lead (Pb) is a heavy metal that may yield changes in all body systems, yet the greatest health damages occur when it affects the central nervous system (CNS). Many studies demonstrate the clinical/behavioral changes caused by the action of Pb on the CNS. However, studies are necessary to demonstrate the biochemical changes caused by Pb in this system. Conversely, it has been reported that iron (Fe) seems to play a protective role on the brain toxicity caused by Pb. Therefore, this study analyzed the concentration of Pb in the brain tissue, and conducted proteomic analysis in the brain of rats intoxicated by Pb, submitted or not to Fe supplementation. The study was conducted on 30 weaning rats (Rattus norvegicus, Wistar type) divided in 6 groups (n=5/group), according to the treatment established for 6 weeks, as follows: Control (not exposed to Pb or Fe), Control Fe (exposed to administration of 20 mg/Kg p.c. of FeSO4 at every 2 days, by gastric gavage), Pb 100 exposed to water containing 100 mg/L of Pb), Pb 400 (exposed to water containing 400 mg/L of Pb) Pb100 + Fe (exposed to water containing 100 mg/L of Pb and gavage with FeSO4) and Pb400 + Fe (exposed to water containing 400 mg/L of Pb and gavage with FeSO4). After the experimental period, the animals were killed and the brains of animals were removed, discarding the cerebellum and brainstem. The remaining structure was submitted to analysis of Pb concentration and proteomic analysis. A dose-response relationship was observed in Pb concentration in the brain. The administration of FeSO4 reduced the levels of Pb in the brain, though without statistical significance. The analysis of gels with proteic spots demonstrated reduction in their quantity according to the treatment performed in the groups. The control group exhibited greater concentration of spots, while groups receiving higher Pb concentration (400 mg/L) presented the lowest quantity of spots. There was also reduction in the quantity of spots detected when FeSO4 was administered. Among the spots presented different expression in the comparisons between the control group and experimental groups and between the experimental groups and their counterparts supplemented or not with FeSO4, 75 proteins were identified by mass spectrometry, being 20 proteins (26.0%) related to metabolic functions, 21 proteins (28.0%) related to structure and organization of structures, 22 proteins (30.0%) related to cell functions and processes, nine proteins (12.0%) related to information pathways and three proteins (4.0%) from the miscellaneous category. The expression of proteins was reduced in most cases, for all functional classifications. The present study, combined to previous findings, indicates a harmful effect of Pb in the cerebral cortex of animals exposed to this metal, regardless of the concentration employed, without observation of a protective effect of FeSO4. This process seems to be mediated by proteins as Gamma-enolase, Alpha-internexin, several isoforms of 14-3-3 and Homer protein homolog 1.
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Marson, Francine. "Neurolinguistica discursiva : analise de praticas clinicas e escolares com a linguagem." [s.n.], 2009. http://repositorio.unicamp.br/jspui/handle/REPOSIP/270976.

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Orientador: Maria Irma Hadler Coudry
Dissertação (mestrado) - Universidade Estadual de Campinas, Instituto de Estudos da Linguagem
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Resumo: A Neurolingüística Discursiva vem sendo elaborada na área de Neurolingüística do Instituto de Estudos da Linguagem na UNICAMP, desde os primeiros trabalhos de Coudry (1986), articulando uma concepção abrangente de linguagem a uma concepção de funcionamento do cérebro baseada nos conceitos lurianos. Tomando por base essa perspectiva teórico-prática, o objetivo deste trabalho é discutir a relevância de um saber sobre a linguagem e sobre o cérebro em avaliações e práticas clínicas com a linguagem. Por meio da apresentação e análise de dados de crianças, pretende-se ilustrar de que modo o olhar da ND é capaz de orientar a prática clínica e escolar com a linguagem explicitando como as patologias e as "falsas patologias" podem afetar o sujeito e a sua relação com a linguagem.
Abstract: Discursive Neurolinguistics (ND) is beeing developed at the Language Studies Institute (IEL), since the initial works of Coudry (1986), presenting a comprehensive conception of language toward brain work based on lurian concepts. Having in mind this theoretical-practical perspective, this thesis aims at debating the importance of such knowledge on the evaluation and clinical practice on language. Through careful presentation and data analysis from children, our intention is to show how ND is capable of giving grounds to the clinical and scholar practice on language, bringing forth how pathologies and false pathologies may affect the subject and his/her relation towards language.
Mestrado
Linguistica
Mestre em Linguística
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A, Vega, Antonny G. Vega, Carlos R. Aguilar, Alfredo Barrientos, and Rosario Villalta. "Desarrollo de aplicaciones para personas con discapacidad motora utilizando Emotiv Epoc." International Institute of Informatics and Systemics, IIIS, 2018. http://hdl.handle.net/10757/624674.

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Personas con discapacidad motora presentan inconvenientes en el desarrollo de actividades tales como caminar, correr, comer. Además, en su mayoría, la visión y el intelecto no se ven afectados. Estas deficiencias no le permiten al manipular dispositivos tecnológicos que podrían ayudarlo a mejorar su calidad de vida como los smartphones. Presentamos una solución que permite superar esta limitación apoyada en la tecnología Brain Computer Interface).
Revisión por pares
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Andrade, Mara Lucia Fabricio de. "Linguagem e atenção : um estudo com sujeitos cerebro-lesados." [s.n.], 2007. http://repositorio.unicamp.br/jspui/handle/REPOSIP/270989.

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Orientador: Maria Irma Hadler Coudry
Tese (doutorado) - Universidade Estadual de Campinas, Instituto de Estudos da Linguagem
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Resumo: Nosso objetivo principal, neste trabalho, é buscar conhecer quais as inter-relações que podemos estabelecer entre o funcionamento da linguagem e da atenção, tomando por base indícios presentes na fala de sujeitos cérebro-lesados. Como fundamentação teórica, tomamos por base uma Neurolingüística discursivamente orientada (abreviada como ND). Na ND o indivíduo é visto em relação ao seu meio, ou seja, social e historicamente, conforme a perspectiva luriana. Também na ND não se considera o indivíduo como ¿amostra¿ de uma população, mas sim como um sujeito, com uma história de vida e que vive em sociedade, o que se reflete na metodologia do dado-achado. A atenção, conforme perspectiva luriana, diz respeito ao caráter direcional e à seletividade dos processos mentais, sendo parte de um sistema funcional complexo, no qual atenção e linguagem se inter-relacionam entre si e com demais processos psíquicos. Para essa investigação utilizamos transcrições e découpages de sessões do Centro de Convivência de Afásicos - Grupo II (CCA-IEL/Unicamp) que compõem o Banco de Dados em Neurolingüística (BDN), vinculado ao Projeto Integrado em Neurolingüística (CNPq 521773/95-4). O estudo foi realizado com cinco sujeitos cérebro-lesados por diversas etiologias. A análise, de natureza qualitativa, é voltada para o levantamento e descrição de diferentes aspectos envolvidos nas inter-relações entre linguagem e atenção. Consideramos, conforme teorias lurianas e vigostskianas, que a atenção se desenvolve nos períodos iniciais da vida, socialmente e pela via da linguagem, mas, tendo em vista a reorganização do processo inicial, a atenção do sujeito se reconstrói. A presente tese tem como hipótese que a atenção, após a reorganização do processo inicial, estaria fortemente envolvida no trabalho lingüístico, o que, em relação à linguagem, resultaria em uma mútua constitutividade: a linguagem constitui a atenção e esta se volta para a linguagem. No capítulo três, a análise e discussão gira em torno de cada um dos cinco sujeitos e em torno de temas relacionados à seletividade, aos sistemas de referência e à especularidade. A hipótese da mútua constitutividade nos guiou na análise dos dados e a partir dela chegamos a três outras hipóteses mais específicas, uma para cada conjunto de dados. Essas hipóteses evidenciam o trabalho com a linguagem pela via da atenção, ou seja, a atenção se voltando para a linguagem. No capítulo quatro, ainda com base na hipótese da mútua constitutividade, retomamos o caso de JS para analisar determinadas falas em que ele se refere a si mesmo como se fosse outra pessoa. Trabalhando com a hipótese de que essas falas se apresentam como uma ¿linguagem interior¿, neste caso, o que estaria em evidência é a linguagem constituindo a atenção. As relações entre linguagem e atenção não podem se limitar, acreditamos nós, às fronteiras do ¿patológico¿. O estudo com sujeitos cérebro-lesados pode conduzir a certas especificidades, mas os vários casos aqui trazidos, apesar de se constituírem de forma singular expressam alguns fenômenos da linguagem comuns a todos os sujeitos falantes. Terminamos este trabalho considerando que o estudo das relações entre linguagem e atenção, tal como o desenvolvemos permite estabelecer uma correlação entre o lingüístico e o psicológico, e, levantar, entre outros, aspectos que contribuem para desnudar todo um trabalho com a linguagem
Abstract: The main scope of this work is to know which relations can be established between the language and attention, with the basic signals presented in the speaking of those with injured brain. A basic guided Neurolinguistic (ND) was used as a theoric fundamentation. In the ND, the person is analyzed in relation to his social and historic life as the Lurian perspective. Also, in the ND the individual is not considered as a sample of the population as the statistics based in experimental tests but as an individual with a life story, reflecting on the methodology of the case. The attention, as a Lurian perspective, has a direct character and selective of mental process, being part of a complex functional system in which the attention and language are self related and also related to psycho process. Transcriptions and selections of sections of the Aphasics Acquaintanceship Center ¿ Group II (CCA-IEL/Unicamp) from the Neurolinguistic Data Bank (NDB/Integrated Project of Neuron Linguistic/CNPq 521773/95-4) were used. As we consider Luria and Vigostski theories that states that the attention starts its development in the first period of life, socially and speaking as well. However, when we consider the reorganization of the attention of the individual, it can be reconstructed.. This work studies the hypothesis that the attention, after the reorganization of the initial process, would be strongly involved in the linguistics task, which would result in a mutual constitutively: the language focus attention and attention turns back to the language. On chapter three, the analysis and discussion was based in each of the five individuals and on related themes to selectivity, reference system and specularity. The hypothesis of the mutual constitutionality guided us in the data analysis and following this hypothesis we reached three specific hypotheses being each one for each complete data. These hypotheses focus the work with the language through attention specifically linked to the language. On chapter four which still includes the hypothesis of the mutual constructivist, and we focused the case of JS to analyse some of his speaking, where he refers to himself on the third person and are directed for himself. Working with the hypothesis that theses talking show an ¿interior language¿, in this case we focus the language bringing attention. The relations between the language and attention cannot be delimited only to the pathologic aspect. The work with injured brain individuals may conduct to some specific cases, but the many cases presented in this study, although been singular cases, we can say that they express some of the language phenomena that may be common to all speaking individuals. We finish this work considering that the study of the relations between language and attention, as we considered here, permitted us to stablish a relationship between the linguistic and psyclological aspects and to focus, among others subjects, the aspects that may contribute to clarify all the work of the language
Doutorado
Doutor em Linguística
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Sokola, Brent S. "DUAL LOX/COX INHIBITION: A NOVEL STRATEGY TO PREVENT NEUROVASCULAR LEAKAGE IN EPILEPSY." UKnowledge, 2018. https://uknowledge.uky.edu/pharmacy_etds/83.

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Epilepsy affects 3.4 million patients in the USA and is characterized by recurring seizures. The blood-brain barrier is leaky in epilepsy and may contribute to seizure progression but the mechanisms which cause this leakage are not fully understood. We hypothesized that seizures trigger LOX- and COX-mediated blood-brain barrier leakage and that dual LOX/COX inhibition prevents barrier leakage in vivo. To test this hypothesis, we administered either the dual LOX/COX inhibitor licofelone or a combination of the 5-LOX inhibitor zileuton and the COX-2 inhibitor celecoxib to rats that experienced status epilepticus (SE). Serum and brain capillaries were isolated 48 hours after SE and serum S100β levels were measured and Texas Red™ leakage rates were determined. Dual inhibition of 5-LOX and COX prevented serum S100β elevations observed in SE rats in a dose-dependent manner with licofelone. Inhibition of 5-LOX and COX-2 with zileuton and celecoxib completely prevented serum S100β elevation. Texas Red™ leakage rates for SE rats were also reduced in a dose-depended manner with licofelone and reduced to control rates with zileuton and celecoxib. These data support our hypothesis that seizure-induced blood-brain barrier leakage is mediated by LOX and COX, and inhibition of these enzymes prevents barrier leakage.
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Benderro, Girriso Futara. "AMBIENT OXYGEN AVAILABILITY MODULATES EXPRESSION OF VASCULAR ANGIOGENIC FACTORS AND CAPILLARY REMODELING (ANGIOPLASTICITY) IN THE MOUSE BRAIN." Case Western Reserve University School of Graduate Studies / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=case1350159484.

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Leão, Karina Ferreira. "DESEMPENHO COGNITIVO DE PESSOAS COM ACIDENTE VASCULAR ENCEFÁLICO." Pontifícia Universidade Católica de Goiás, 2015. http://localhost:8080/tede/handle/tede/1878.

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Recent studies reveal the high frequency of individuals who are affected by stroke and the cognitive dysfunctions they feature consequently. This paper seeks to assess the cognitive impairments of experiencing a stroke and, more specifically, to evaluate the cognitive performance of people affected by stroke according to hemisphere and artery affected. The paper is divided into two main parts. At first, a theoretical review about the neuropsychological impairments in individuals with stroke was made with the goal of understanding the relationship between brain areas and the cognitive performance of these individuals. For this, a bibliographical survey was conducted in major databases on this subject, describing the theoretical aspects related to Neuroscience, stroke and resulting cognitive dysfunctions assessed by neuropsychological avaliation. It has been found that, as for the etiological description, identification of the types and anatomical structures altered, there is a wide literature, but in relation to cognitive impairment and specific brain areas of research conducted in the field of neurophysiology there is a paucity of publications. Research indicates that it is of fundamental importance more studies intending to show, more and more, the cognitive difficulties in people afflicted with a disease so common in society. Then, in Part 2, an empirical study was conducted with the objective of evaluating the cognitive performance according to hemisphere and artery affected in different neuropsychological tests. Participated in the study 30 patients with a diagnosis of stroke, with the age group between 24-60 years. According to the data, we observe a significant difference according to the lesioned hemisphere only in alternation skills, auditory span, visual-construction, lexical and semantic verbal fluency and verbal comprehension. Regarding to the affected arteries, significant differences were found in selective attention, abstraction, naming, phonetic verbal fluency, verbal comprehension, visual-constructional praxis and recognition of the mnemonic process. The present study as well as providing an understanding of neuropsychological and cognitive impairments of patients injured by stroke, can assist in the development of neuropsychological assessment batteries for people affected by stroke and rehabilitation strategies from possibilities of cognitive dysfunctions according to compromised region after stroke. In general, the two parts of the dissertation contribute to the theoretical extension about cognitive dysfunctions of patients suffering from stroke and consequently the identification of these dysfunctions may thus help to improve care for patients with this disease.
Estudos recentes revelam a alta frequência de indivíduos acometidos por acidente vascular encefálico (AVE) e as disfunções cognitivas que se apresentam em consequência. Esta dissertação buscou avaliar as disfunções cognitivas derivadas da vivência de um AVE e, de forma mais específica, avaliar o desempenho cognitivo de pessoas acometidas por AVE em virtude do hemisfério lesionado e da artéria afetada. A dissertação está dividida em duas partes. Em um primeiro momento, foi realizada uma revisão teórica sobre as alterações neuropsicológicas em indivíduos com AVE, com o objetivo de compreender as relações entre áreas cerebrais e desempenho cognitivo desses indivíduos. Para tanto, foi realizado um levantamento bibliográfico nas principais bases de dados sobre esse tema, com o objetivo de descrever os aspectos teóricos relacionados à Neurociência, o AVE e as decorrentes disfunções cognitivas mediante avaliação neuropsicológica. Verificou-se que, em referência à descrição etiológica, identificação dos tipos e a estruturas anatômicas alteradas, existe uma ampla literatura, porém, em relação a disfunções cognitivas e áreas cerebrais específicas há uma escassez de publicação na área da neuropsicologia. As pesquisas apontam que é de mais fundamental relevância estudos que se proponham a evidenciar cada vez mais as dificuldades cognitivas em pessoas acometidas com uma doença tão frequente na sociedade. Na parte 2, foi realizado um estudo empírico, com o objetivo de avaliar o desempenho cognitivo segundo hemisfério e artéria afetada em diferentes testes neuropsicológicos. Participaram da pesquisa 30 pacientes com diagnóstico médico de AVE, na faixa etária entre 24 e 60 anos. De acordo com os dados, observouse diferença significativa quanto ao hemisfério lesionado nas habilidades de alternância, span auditivo, visuoconstrução, fluência verbal léxica e semântica e compreensão verbal. No que se refere às artérias afetadas, foram encontradas diferenças significativas nas atividades de atenção seletiva, abstração, nomeação, fluência verbal fonética, compreensão verbal, praxia visuoconstrutiva e reconhecimento do processo mnemônico. O presente estudo, além de proporcionar a compreensão de alterações neuropsicológicas e cognitivas dos pacientes lesionados pelo AVE, pode auxiliar o desenvolvimento de baterias de avaliação neuropsicológica para pessoas acometidas por essa doença, bem como estratégias de reabilitação, conforme possibilidades de disfunções cognitivas na região comprometida após a ocorrência do AVE. De maneira geral, as duas partes da dissertação contribuem para a ampliação teórica sobre as disfunções cognitivas de pacientes acometidos por AVE e, consequentemente, para identificação desses comprometimentos, podendo assim auxiliar o atendimento a pacientes com essa enfermidade.
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Books on the topic "Cow brain"

1

Crasto, Audrey. Satan cows and rooster brains. Toronto: Life Rattle Press, 2005.

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Cooke, Jennifer. Cannibals, cows & the CJD catastrophe. Milsons Point, NSW: Random House Australia, 1998.

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Silvano Ambrogi (1929-1996): Per sogghignare-- : con brani tratti dall'inedito Il Novantino. Pisa: ETS, 2002.

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Vetri, Maria Luisa Basso. Karl Jaspers, o, Della filosofia come amore: Con brani scelti, tradotti e commentati. Napoli: Liguori, 2013.

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Hollywood, John S. High-priority information technology needs for law enforcement / John S. Hollywood, John E. Boon, Jr., Richard Silberglitt, Brian G. Chow, Brian A. Jackson. Santa Monica, Calif: Rand Corporation, 2015.

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Garbi, Esmeralda. Qué pasó con los mejores: La trayectoria profesional de los mejores estudiantes en ciencias básicas e ingeniería. [Venezuela]: IESA, 1989.

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Padua, Donatella. Agire creativo e senso della razionalità in Pareto: Con una selezione di brani del Trattota di sociologia. Milano: F. Angeli, 2009.

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El niño con daño cerebral: En la escuela, en el hogar y en la comunidad. 3rd ed. México: Editorial Trillas, 1992.

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Luzi, Mario. Casi e brani di adolescenza: Una prosa e una poesia rare : con dieci lettere inedite a Piero Bigongiari. Pistoia: Via del vento, 1995.

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Gasca, Omar. Efe ocho por uno: Temas de reflexión a partir de conversaciones con ocho fotógrafos : Braun, Chávez, Díaz Infante, Fematt, Meyer, Oloarte, Paz, Tzontémoc. Tuxtla Gutiérrez, Mexico: Universidad de Ciencias y Artes de Chiapas, 2000.

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Book chapters on the topic "Cow brain"

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Ebringer, Alan. "Autoantibodies to Brain Components and Antibodies to Acinetobacter Are Present in Bovine Spongiform Encephalopathy." In Multiple Sclerosis, Mad Cow Disease and Acinetobacter, 45–55. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-02735-7_7.

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McKenna, Mary C., and Irene B. Hopkins. "Determination of CO2 Production in Subcellular Preparations Like Synaptosomes and Isolated Mitochondria Using 14C-Labeled Substrates and Radioactive CO2 Measurements." In Brain Energy Metabolism, 1–24. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1059-5_1.

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Adalia, Ramón, Paola Hurtado, and Ricard Valero. "Neuroanesthesia and Brain Death." In Co-existing Diseases and Neuroanesthesia, 277–95. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-13-2086-6_15.

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Friesgaard, Kristian Dahl, and Bent Lob Dahl. "Blood Pressure, CO2, and Oxygen Saturation." In Management of Severe Traumatic Brain Injury, 239–43. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39383-0_36.

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Dahl, Bent Lob. "Blood Pressure, CO2 and Oxygen Saturation." In Management of Severe Traumatic Brain Injury, 133–37. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-28126-6_26.

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Bazan, N. G., V. L. Marcheselli, P. K. Mukherjee, W. J. Lukiw, W. C. Gordon, and D. Zhang. "COX-2 in brain and retina: role in neuronal survival." In Selective COX-2 Inhibitors, 47–53. Dordrecht: Springer Netherlands, 1998. http://dx.doi.org/10.1007/978-94-011-4872-6_4.

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Namba, Kazunori, and Hiroki Kaneko. "Co-immunoprecipitation Methods for Detection of G Protein-Coupled Receptors in Brain Tissue." In Co-Immunoprecipitation Methods for Brain Tissue, 1–8. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8985-0_1.

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Borroto-Escuela, Dasiel O., Manuel Narvaez, Martina Zannoni, Chiara Contri, Minerva Crespo-Ramírez, Michael di Palma, Patrizia Ambrogini, et al. "Isolation and Detection of G Protein-Coupled Receptor (GPCR) Heteroreceptor Complexes in Rat Brain Synaptosomal Preparation Using a Combined Brain Subcellular Fractionation/Co-immunoprecipitation (Co-IP) Procedures." In Co-Immunoprecipitation Methods for Brain Tissue, 123–35. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8985-0_10.

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Romero-Fernandez, Wilber, Maria Garcia-Barcelo, and Yunis Perez-Betancourt. "Co-immunoprecipitation of Membrane-Bound Receptors from Subsynaptic Compartments." In Co-Immunoprecipitation Methods for Brain Tissue, 137–45. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8985-0_11.

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Borroto-Escuela, Daily Y., Idania Hernández-Ramos, Kjell Fuxe, and Dasiel O. Borroto-Escuela. "Coimmunoprecipitation (co-IP) Analysis for Protein-Protein Interactions in the Neurons of the Cerebral Ganglia of the Land Snails of the Genus Polymita During Aestivation." In Co-Immunoprecipitation Methods for Brain Tissue, 147–56. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8985-0_12.

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Conference papers on the topic "Cow brain"

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Fahy, Paul, Patrick Delassus, Padraig O’Flynn, and Liam Morris. "An Experimental Study of the Effects Anatomical Variations Have on Collateral Flows Within the Circle of Willis." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53723.

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The circle of Willis (CoW) is a complex arterial network comprising of major cerebral arteries that converge to form a pentagonal arrangement as shown in Figure 1(A). This arterial network supplies oxygen-enriched blood to the brain. An incomplete CoW can exist in up to 50% of cases [1]. These missing vessels can be accommodated by the collateral flow feature within the CoW configuration. In certain circumstances, anatomical variations within the CoW can result in undesirable flow patterns [2–3]. It is unclear from the literature what effects these variations can have on blood flow collision paths within a complete CoW.
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Luppi, Luca, Quan Long, Carola S. König, and Michael W. Collins. "Numerical Study of Blood Redistribution in the Circle of Willis of Patients With Carotid Artery Stenosis." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176429.

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Stroke is an acute neurological injury in which the blood supply to a part of the brain is interrupted due to either a diminution of cerebral blood flow or a sudden occlusion of a feeding artery. Blood perfusion depends not only on the status of the diseased vessels but also on the patency of collateral pathways. The primary collateral pathway is that provided by the Circle of Willis (CoW).
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Zhu, Guangyu, Qi Yuan, and Joon Hock Yeo. "Experimental Study of Hemodynamics in the Circle of Willis." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14162.

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The Circle of Willis (CoW) is a ring like structure located at the base of brain, which is composed of a single anterior communicating artery (ACoA), paired anterior cerebral arteries (ACAs), paired internal carotid arteries (ICAs), paired posterior communicating arteries (PCoAs), paired posterior cerebral arteries (PCAs), paired vertebral arteries (VAs) and a single basilar artery (BA). It is the main cerebral blood perfusion pathway and provides an important collateral channel in patients with severe carotid or vertebral artery disease. Over 50% of stroke cases are related to the stenosis of arteries in the CoW, so the detailed information of the cerebral hemodynamics under different pathology situations is important for a variety of clinical applications. Numerous experimental studies have been performed on this field from different perspectives, include the mechanism of stenosis in the CoW [1], risk assessment of cerebral aneurysm [2] and the impact of pathological variations on the flow distribution [3]. However, none of these researches focus on the influence of ICA stenosis rates on cerebral perfusion and the specific collateral mechanism of the Circle of Willis under such situations. In this paper, an experimental study on cerebral blood perfusion and the collateral mechanism under a series of ICA stenosis rates was carried out.
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Zhu, Li, Chongwei Su, Gaochao Cui, Changle Zhou, Jianhai Zhang, and Wanzeng Kong. "Idle-State Detection in Multi-user Motor Imagery Brain Computer Interface with Cross-Brain CSP and Hyper-Brain-Network." In 2019 International Conference on Cyberworlds (CW). IEEE, 2019. http://dx.doi.org/10.1109/cw.2019.00045.

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Backsanskij, O. E., and E. A. Dergacheva. "Cognitive Processes of the Brain and Learning Theory." In International Scientific Conference "Far East Con" (ISCFEC 2020). Paris, France: Atlantis Press, 2020. http://dx.doi.org/10.2991/aebmr.k.200312.010.

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McQuighan, Joseph M., Garima Bajwa, and Jason M. Pittman. "B2CI 2019: The IEEE Brain to Computer Interface Competition’s Gaming Event." In 2019 IEEE Conference on Games (CoG). IEEE, 2019. http://dx.doi.org/10.1109/cig.2019.8848115.

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Hamadicharef, Brahim, Mufeng Xu, and Sheel Aditya. "Brain-Computer Interface (BCI) Based Musical Composition." In 2010 International Conference on Cyberworlds (CW). IEEE, 2010. http://dx.doi.org/10.1109/cw.2010.32.

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Du, Huiping, Shu Wang, Xingfu Wang, Xiaoqin Zhu, Shuangmu Zhuo, and Jianxin Chen. "Identification of the boundary between normal brain tissue and ischemia region using two-photon excitation fluorescence microscopy." In SPIE/COS Photonics Asia, edited by Qingming Luo, Xingde Li, Ying Gu, and Yuguo Tang. SPIE, 2016. http://dx.doi.org/10.1117/12.2245399.

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Liu, Bin, Xinzhu Sang, Shujun Xing, and Bo Wang. "An improved non-local means filter for denoising in brain magnetic resonance imaging based on fuzzy cluster." In SPIE/COS Photonics Asia, edited by Qingming Luo, Xingde Li, Ying Gu, and Yuguo Tang. SPIE, 2014. http://dx.doi.org/10.1117/12.2071401.

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Teland, Jan, Anders Hamberger, Morten Huseby, Annette Säljö, and Eirik Svinsås. "Numerical simulation of mechanisms of blast-induced traumatic brain injury." In 159th Meeting Acoustical Society of America/NOISE-CON 2010. ASA, 2010. http://dx.doi.org/10.1121/1.3492797.

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Reports on the topic "Cow brain"

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Olson-Madden, Jennifer H. Development of an Intervention for Soldiers and Veterans With Co-Occurring Traumatic Brain Injury and Substance Abuse Disorders. Fort Belvoir, VA: Defense Technical Information Center, October 2010. http://dx.doi.org/10.21236/ada541227.

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Goldstein, L. B., A. M. Dechovskaia, S. Bullman, K. H. Jones, and A. A. Abdel-Rahman. Daily Dermal Co-Exposure of Rats to DEET and Permethrin Produces Sensorimotor Deficit, and Changes in Blood-Brain Barrier (BBB) and Blood-Testis Barrier (BTB). Fort Belvoir, VA: Defense Technical Information Center, March 2001. http://dx.doi.org/10.21236/ada402081.

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