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Books on the topic 'Cow brain'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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1

Crasto, Audrey. Satan cows and rooster brains. Toronto: Life Rattle Press, 2005.

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2

Cooke, Jennifer. Cannibals, cows & the CJD catastrophe. Milsons Point, NSW: Random House Australia, 1998.

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3

Silvano Ambrogi (1929-1996): Per sogghignare-- : con brani tratti dall'inedito Il Novantino. Pisa: ETS, 2002.

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4

Vetri, Maria Luisa Basso. Karl Jaspers, o, Della filosofia come amore: Con brani scelti, tradotti e commentati. Napoli: Liguori, 2013.

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5

Hollywood, John S. High-priority information technology needs for law enforcement / John S. Hollywood, John E. Boon, Jr., Richard Silberglitt, Brian G. Chow, Brian A. Jackson. Santa Monica, Calif: Rand Corporation, 2015.

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6

Garbi, Esmeralda. Qué pasó con los mejores: La trayectoria profesional de los mejores estudiantes en ciencias básicas e ingeniería. [Venezuela]: IESA, 1989.

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7

Padua, Donatella. Agire creativo e senso della razionalità in Pareto: Con una selezione di brani del Trattota di sociologia. Milano: F. Angeli, 2009.

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8

El niño con daño cerebral: En la escuela, en el hogar y en la comunidad. 3rd ed. México: Editorial Trillas, 1992.

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9

Luzi, Mario. Casi e brani di adolescenza: Una prosa e una poesia rare : con dieci lettere inedite a Piero Bigongiari. Pistoia: Via del vento, 1995.

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10

Gasca, Omar. Efe ocho por uno: Temas de reflexión a partir de conversaciones con ocho fotógrafos : Braun, Chávez, Díaz Infante, Fematt, Meyer, Oloarte, Paz, Tzontémoc. Tuxtla Gutiérrez, Mexico: Universidad de Ciencias y Artes de Chiapas, 2000.

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11

Caproni, Giorgio. Il Girasole. Edited by Giada Baragli. Florence: Firenze University Press, 2017. http://dx.doi.org/10.36253/978-88-6453-495-4.

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«Apriamo a caso il libro». È così che si eclissava tra il ’72 e il ’75 la voce-guida di Caproni per lasciare ad altri il microfono della trasmissione radiofonica «Il girasole». Una rubrica destinata alla divulgazione, trasmessa nelle ore di massimo ascolto del programma nazionale, con lo scopo di offrire ad un pubblico largo e diversificato una sorta di ‘sussidiario’ letterario che desse ai colti il piacere della rilettura e ai più giovani e sprovveduti quello della scoperta. Un grande poeta, Giorgio Caproni, aveva il compito di scegliere i passi da testi di ogni tempo e paese (poesia, narrativa, teatro…) e di collegarli tra loro con poche parole di presentazione in grado di ricreare un ambiente, un’epoca, insomma quanto sta intorno all’opera d’arte e la rende per tutti comprensibile e umana. È questa singolare avventura che – perduti/distrutti i materiali radiofonici – riusciamo infine a conoscere grazie all’attenta cura di Giada Baragli, che da copioni spesso lacunosi ha ricostruito il florilegio di un centinaio di brani che non solo offrono una preziosa antologia comparata della letteratura, ma ci parlano delle predilezioni e delle letture di uno straordinario, indimenticabile autore che ha anche collaborato con la radio.
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12

Guarnieri, Patrizia. Intellettuali in fuga dall’Italia fascista. Florence: Firenze University Press, 2019. http://dx.doi.org/10.36253/978-88-6453-872-3.

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Il progetto richiama l’attenzione sull’emigrazione intellettuale dal fascismo. L’Italia è di solito considerata terra di migranti poveri e senza istruzione. Ma durante il ventennio, specie dopo le leggi antiebraiche, professionisti, studenti e studiosi anche stranieri espatriarono soli o con le famiglie nelle Americhe, in Inghilterra, in Palestina, in Svizzera. È un fenomeno limitato ma importante di brain drain, per l’Italia non ancora indagato. Chi erano e cosa fecero quanti forzatamente partirono in cerca di libertà, lavoro, e poi salvezza? I loro nomi, le loro storie vennero cancellate. Qui si cerca di ricostruirle grazie ad archivi esteri, a lettere e memorie disperse. Quali difficoltà incontrarono nei paesi di accoglienza? Quanti tornarono? Le storie parlano di perdite irreparabili a danno del paese, di responsabilità e ingiustizie, ma anche di risorse e talenti della cultura italiana, di impegno e determinazione. Il progetto promosso dall’Università di Firenze è finanziato dalla Regione Toscana e patrocinato da New York Public Library, Council for At-Risk Academics, e J. Calandra Italian American Institute.
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13

Mace, Nancy L. Cuando el día tiene 36 horas: Una guía para cuidar a enfermos con alzheimer, pérdida de memoria y demencia senil. 2nd ed. México, D.F. [México]: Editorial Pax Mexico, 1997.

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14

King, Stephen. The Bachman Books: Four Early Novels by Stephen King. New York, USA: New American Library, 1986.

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15

King, Stephen. The Bachman books: Four novels ; Rage ; The long walk ; Roadwork ; The running man. Sevenoaks: New English Library, 1986.

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16

King, Stephen. The Bachman Books. New York, USA: New American Library, 1985.

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17

King, Stephen. The Bachman Books. London, England: New English Library, 1987.

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18

King, Stephen. The Bachman Books: Four Early Novels by Stephen King. New York, USA: Signet, 1996.

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19

King, Stephen. The Bachman Books: Four Early Novels by Stephen King. New York, USA: New American Library, 1986.

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20

King, Stephen. Bachman Books: Four Early Novels by Stephen King. New York, USA: New American Library, 1986.

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21

King, Stephen. The Bachman books: Four early novels. New York: Plume, 1996.

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22

Brain Trust: The Hidden Connection Between Mad Cow and Misdiagnosed Alzheimer's Disease. Paraview Pocket Books, 2004.

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23

Wijdicks, Eelco F. M. Neurology of Brain Death. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190662493.003.0002.

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The diagnosis of brain death is based on a comprehensive neurological evaluation. First, physicians need to eliminate confounders to the clinical examination. Once excluded, a set of neurological tests and a formal apnea test (to document absent respiratory drive after CO2 challenge) often will suffice. Second, ancillary tests may be needed if some parts of the neurological examination cannot be accurately assessed. This chapter provides a full discussion of the clinical criteria in adults and children and is modeled after the guidelines of the American Academy of Neurology and the 2011 definition of pediatric brain death by a joint task force of the Society of Critical Care Medicine, the American Academy of Pediatrics, and the Child Neurology Society.
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24

Meditando Com Brian Weiss - C/ Cd. Editora Sextante, 2004.

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25

Eva Braun : una vida con Hitler. Debate, 2012.

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26

Diamond, Pamela M. Traumatic brain injury. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199360574.003.0053.

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During the past decade, traumatic brain injury (TBI) has become a frequent topic in the media. It has been a decade of expanding awareness, increased research, and growing concern about TBI of all severity levels. Consistent with this increased attention, researchers and policymakers have made strides toward greater understanding of the risks of TBI, the scope and complexity of the symptom profiles seen after TBI, and the types of treatments that optimize recovery. Recent studies have confirmed a 50 to 60% prevalence of TBI among prisoners. Most have experienced multiple injuries and experienced their first TBI in their mid-teens. Routine screening for TBI is rarely done in these settings in spite of there being a number of tested instruments available. The cognitive deficits associated with mild to moderate TBI are often indistinguishable from those associated with many mental illnesses and substance abuse. Etiology is difficult to establish; nevertheless, the common symptom patterns often make adjustment to jail or prison difficult. Educational interventions designed to improve staff knowledge of the prevalence of TBI and frequent symptom patterns are important first steps. Training staff how to modify their behavior and facilitate communication with inmates expressing these symptoms may reduce episodes of misunderstanding and potential aggression. Similarly, current programming may be modified to accommodate the cognitive deficits suffered by inmates with TBI as well as other disorders. This chapter reviews the prevalence of TBI in correctional settings, its impact on co-occurring mental illness and substance use, and opportunities to recognize, intervene, and treat patients with TBI.
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27

Cooke, Jennifer. Cannibals, Cows and the CJD Catastrophe. Random House USA Inc, 2000.

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28

Cooke, Jennifer. Cannibals, Cows and the CJD Catastrophe. Random House USA Inc, 2000.

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29

Seeck, Margitta, L. Spinelli, Jean Gotman, and Fernando H. Lopes da Silva. Combination of Brain Functional Imaging Techniques. Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0046.

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Several tools are available to map brain electrical activity. Clinical applications focus on epileptic activity, although electric source imaging (ESI) and electroencephalography-coupled functional magnetic resonance imaging (EEG–fMRI) are also used to investigate non-epileptic processes in healthy subjects. While positron-emission tomography (PET) reflects glucose metabolism, strongly linked with synaptic activity, and single-photon-emission computed tomography (SPECT) reflects blood flow, fMRI (BOLD) signals have a hemodynamic component that is a surrogate signal of neuronal (synaptic) activity. The exact interpretation of BOLD signals is not completely understood; even in unifocal epilepsy, more than one region of positive or negative BOLD is often observed. Co-registration of medical images is essential to answer clinical questions, particularly for presurgical epilepsy evaluations. Multimodal imaging can yield information about epileptic foci and underlying networks. Co-registering MRI, PET, SPECT, fMRI, and ESI (or magnetic source imaging) provides information to estimate the epileptogenic zone and can help optimize surgical results.
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30

Management COB 300A with Brian K. Miller, PH.D. Thomas Learning, 2004.

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31

Schechter, Elizabeth. Self and Other in the Split-Brain Subject. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198809654.003.0007.

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This chapter concerns self-consciousness in split-brain subjects. I argue, first, that R and L are both capable of thinking I-thoughts: thoughts containing the mental or conceptual analogue of the English word “I.” On the other hand, R’s and L’s self-consciousness differs, in its operative dynamics, from self-consciousness in, say, my sister and me. First of all, neither R nor L recognizes the existence of a second thinker sharing its body. I call this lack of mutual recognition. Second, L seems to assume that its I-thoughts refer to S, and R seems to assume the same of its I-thoughts. I call this (subjective) co-identification as S. I then argue that lack of mutual recognition and co-identification as S are explained by the fact that R and L lack the capacity for self-distinction: neither can first-personally distinguish itself from the other.
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32

Cristoforo, Buscarini, ed. Retrosi: Antologia di una tempera : con brani sulla storia della Repubblica. Cinisello Balsamo (Milano): Silvana, 2001.

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33

Retrosi: Antologia di una tempera : con brani sulla storia della Repubblica. [Cinisello Balsamo, Milano?]: Silvana, 2001.

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34

translator, Pons Pradilla Sílvia, ed. Eres lo que dices: Cambia tu vida con el poder de tus palabras. 2013.

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35

Pangarkar, Sanjog S. Pain and Addiction in Patients with Traumatic Brain Injury (DRAFT). Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190265366.003.0027.

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Distinct from Chapter 24, on co-occurring psychiatric disorders, this chapter addresses common physical comorbidities that give rise to chronic pain and are notorious for associated substance use disorders. The concept of “pseudo-addiction” is explored as one of several contributors to common misperceptions of the analgesic needs of such patients. Examples of entities discussed are chronic low back pain, sleep apnea, chronic pancreatitis, cirrhosis, and HIV infection or AIDS-related pain. While not intrinsically painful, sleep apnea merits inclusion as it arises in conjunction with sedative-hypnotic, opioid, or nicotine use. Cirrhosis likewise creates obstacles to successful pain or addiction management resulting from altered metabolism of medications and enhanced susceptibility to potentially lethal syndromes (hepato-renal syndrome, gastric hemorrhage, etc.). The management of neuropathic pain in HIV infection (Chapter 15) is amplified here.
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36

El Espejo Social de La Mujer Con Gran Discapacidad: Barreras Sociales Para Retornar a Una Vida Normal. Fundamentos Editorial, 2004.

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37

Rowena, Coles, ed. Guida alla traduzione inglese: Brani di autori italiani contemporanei con testo a fronte. Urbino: QuattroVenti, 1992.

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38

Schechter, Elizabeth. Bodies and Being One. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198809654.003.0006.

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This chapter concerns the relationship between the split-brain case and the non-split case. In the first half of the chapter, I consider arguments to the effect that if split-brain subjects have two minds apiece, then so do non-split subjects. Sometimes these arguments have taken the form of a reductio against the 2-thinkers claim for split-brain subjects. These arguments do not work: that a split-brain subject has two minds does not mean that I have two minds, although it does mean that I could. The second half of the chapter offers my own proposal for the respect in which R’s and L’s co-embodiment as one animal, S, makes a split-brain subject one of us: I argue that S must be the single object of both R’s and L’s implicit bodily self-awareness.
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39

Hyona, J., ed. The Brain's Eye: Neurobiological and Clinical Aspects of Oculomotor Research (Progress in Brain Research). Elsevier Science, 2002.

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40

Moore, Michael S. Mechanical Choices. Oxford University Press, 2020. http://dx.doi.org/10.1093/oso/9780190863999.001.0001.

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This book assays how the remarkable discoveries of contemporary neuroscience impact our conception of ourselves and our responsibility for our choices and our actions. Dramatic (and indeed revolutionary) changes in how we think of ourselves as agents and as persons are commonly taken to be the implications of those discoveries of neuroscience. Indeed, the very notions of responsibility and of deserved punishment are thought to be threatened by these discoveries. Such threats are collected into four groupings: (1) the threat from determinism, that neurosciences shows us that all of our choices and actions are caused by events in the brain that precede choice; (2) the threat from epiphenomenalism, that our choices are shown by experiment not to cause the actions that are the objects of such choice but are rather mere epiphenomena, co-effects of common causes in the brain; (3) the threat from reductionist mechanism, that we and everything we value is nothing but a bunch of two-valued switches going off in our brains; and (4) the threat from fallibilism, that we are not masters in our own house because we lack the privileged knowledge of our own minds needed to be such masters. The book seeks to blunt such radical challenges while nonetheless detailing how law, morality, and common-sense psychology can harness the insights of an advancing neuroscience to more accurately assign moral blame and legal punishment to the truly deserving.
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41

Schechter, Elizabeth. How Many Minds? Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198809654.003.0004.

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The previous chapters argued that within a split-brain subject there are two subjects of conscious experience and intentional agents, R and L. This chapter explains who these two thinking beings are and how it is possible for two thinkers to be co-embodied. The basis of the 2-thinkers claim is, naturally, that R and L think, feel, decide, and so on, independently of each other. Of course, this does not mean that they do not causally interact; since they are co-embodied, they interact all the time. What split-brain experiments show, however, is that R’s mental activities interact with L’s largely only indirectly: one of them acts or reacts in some way, and the other senses or perceives this re/action. Mental activities are causal activities whose psychological kinds are defined by their powers to interact directly. Thus the thinking things in the split-brain case are R and L, and only derivatively S.
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42

Paul, Heiney, ed. Can cows walk down stairs?: The best brains answer the biggest and smallest scientific questions. Stroud: Sutton Pub., 2005.

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43

Noël, Marie-Pascale. When Number Processing and Calculation is Not Your Cup of Tea. Edited by Roi Cohen Kadosh and Ann Dowker. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199642342.013.62.

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This section of this volume deals with the study of numerical impairment occurring either after brain damage (i.e., acquired acalculia) or during development without any known brain damage (i.e., dyscalculia). The chapters in this section will report the research aiming at characterizing those difficulties. The study of atypical number processing and calculation in acalculia has contributed importantly to the understanding of how our brain is structured to process number and to calculate. The study of dyscalculia has shed light on the numerical bases for arithmetic learning. This research has also helped us in determining how other cognitive functions such as working memory, visuospatial processing, or phonological awareness have an impact on numerical cognition. These relations between different cognitive domains could partly explain the co-morbidities that are often observed in developmental disorders. Finally, this section also reviews the few attempts that have been made to enhance those numerical capacities.
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44

Butz, Martin V., and Esther F. Kutter. Cognitive Development and Evolution. Oxford University Press, 2017. http://dx.doi.org/10.1093/acprof:oso/9780198739692.003.0004.

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When acknowledging that the mind is embodied, cognitive development and evolution must determine how the body and environment shape the mind. Evolution has evolved structures and computational mechanisms in the body, and the brain that predispose ontogenetic development. Starting with conception, brain, body, and mind co-develop, and shape each other. An infant first develops rudimentary bodily representation and control capabilities, and concurrently uses them to abstract from and generalize over the gathered sensorimotor experiences to develop conceptual understandings and language. Evolution, on the other hand, works on a different time scale. Evolutionary pressures towards survival-suitable cell and bodily structures have dominated much of evolutionary progression. Benefits due to social interactions and coordinated cooperation have led to the evolution of the human brain, enabling the development of human minds. Some details on genetics and on evolutionary computation shed further light on how evolution must have brought about human minds. Thereby, the evolution of suitable bodily structures, of brain modularizations, developmental pathways, adaptive behavioral capabilities, and predispositions for social interactions constitute critical components. Subsequent chapters focus on the computational mechanisms behind embodied cognitive development.
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45

Gaitanis, John, Phillip L. Pearl, and Howard Goodkin. The EEG in Degenerative Disorders of the Central Nervous System. Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0013.

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Nervous system alterations can occur at any stage of prenatal or postnatal development. Any of these derangements, whether environmental or genetic, will affect electrical transmission, causing electroencephalogram (EEG) alteration and possibly epilepsy. Genetic insults may be multisystemic (for example, neurocutaneous syndromes) or affect only the brain. Gene mutations account for inborn errors of metabolism, channelopathies, brain malformations, and impaired synaptogenesis. Inborn errors of metabolism cause seizures and EEG abnormalities through a variety of mechanisms, including disrupted energy metabolism (mitochondrial disorders, glucose transporter defect), neuronal toxicity (amino and organic acidopathies), impaired neuronal function (lysosomal and peroxisomal disorders), alteration of neurotransmitter systems (nonketotic hyperglycinemia), and vitamin and co-factor dependency (pyridoxine-dependent seizures). Environmental causes of perinatal brain injury often result in motor or intellectual impairment (cerebral palsy). Multiple proposed etiologies exist for autism, many focusing on synaptic development. This chapter reviews the EEG findings associated with this myriad of pathologies occurring in childhood.
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46

Gallagher, Shaun. Perception without Inferences. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198794325.003.0006.

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This chapter examines inference models of perception, including predictive coding models, and offers an enactivist alternative. It explores how the enactivist approach can respond to issues related to cognitive penetration and the effects of culture on perception. The enactivist view appeals to an account of the co-variant coupling of brain–body–environment, structured by the physical aspects of neuronal processes, bodily movements, affects, anatomy and function, and environmental regularities. Changes in any of these factors mean that perception changes. Processes involved in neural plasticity (where the brain is ‘set up to be set off’), and metaplasticity, where material and cultural practices define environmental regularities, mitigate the need to think that subpersonal perceptual processes are inferential.
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47

Hasbun, Rodrigo, Richard Dunham, Joseph S. Kass, Rituparna Das, Karen Nunez-Wallace, Lydia J. Sharp, and Doris Kung. HIV-Associated Neurocognitive Disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190493097.003.0038.

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HIV causes a chronic form of encephalitis (HIVE) that is clinically characterized by either dementia or mild neurocognitive impairment. Since the introduction of antiretroviral therapy in 1996, the incidence of HIV dementia has decreased by 50%, but the prevalence of mild neurocognitive disorder has increased up to 39%. HIVE is the result of direct microglial infection, interruption of trophic factors, or caused by inflammatory cytokines. HIV enters the brain primarily by the “Trojan horse mechanism”; it is carried by monocytes and lymphocytes that cross the blood–brain barrier. HIV has a predilection for the basal ganglia, deep white matter, and hippocampus, resulting in a subcortical dementia. HIV dementia is a diagnosis of exclusion and other co-infections, cerebrovascular disease, malnutrition, and drug abuse should be ruled out before making the diagnosis. In patients receiving antiretroviral therapy with immunological response, a novel condition termed CD8+ T cell encephalitis was recently described.
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48

Lavigne, Gilles J., Samar Khoury, Caroline Arbour, and Nadia Gosselin. Sleep and pain. Edited by Sudhansu Chokroverty, Luigi Ferini-Strambi, and Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0046.

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While sleep disturbances are highly prevalent in primary care populations, the patients with the highest rate of poor sleep complaints, including insomnia and nonrestorative sleep, are those with pain. In this chapter, a summary of the potential shared or interactive mechanisms underlying the coexistence of sleep and pain in chronic pain conditions is presented. Theoretical perspectives illustrating sleep–pain interactions are described, as well as the latest empirical evidence regarding sleep disruptions in the context of chronic widespread musculoskeletal pain, fibromyalgia, temporomandibular disorders, headaches, and mild traumatic brain injury. Finally, multidimensional strategies for the co-management of sleep and pain are proposed and discussed.
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49

Wyatt, Tristram D. 3. How behaviour develops. Oxford University Press, 2017. http://dx.doi.org/10.1093/actrade/9780198712152.003.0003.

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Behaviours evolve by natural selection. As genes influence how behaviours develop, selection on behaviour will alter gene frequencies in subsequent generations: genes that lead to successful behaviours in foraging, parental care, or mate choice, for example, will be represented in more individuals in future generations. If conditions change, then mutations of the genes that give rise to advantageous behaviours will be favoured by selection. ‘How behaviour develops’ explains that the environment is equally important: both genes and environment are intimately and interactively involved in behaviour development. Behavioural imprinting is also discussed along with co-opting genes, gene regulation, social influences on brain gene expression, phenotypic plasticity, and play.
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50

Omar, Gasca, ed. Efe ocho por uno: Temas de reflexión a partir de conversaciones con ocho fotógrafos : Braun, Chávez, Díaz Infante, Fematt, Meyer, Oloarte, Paz, Tzontémoc. Tuxtla Gutiérrez, Chiapas: Universidad de Ciencias y Artes de Chiapas, 2000.

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