Relevant bibliographies by topics / COX2 inhibition

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'COX2 inhibition'

Author: Grafiati
Published: 7 June 2025
Last updated: 2 August 2025

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Journal articles on the topic "COX2 inhibition"

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Rao, Reena, Ming-Zhi Zhang, Min Zhao, et al. "Lithium treatment inhibits renal GSK-3 activity and promotes cyclooxygenase 2-dependent polyuria." American Journal of Physiology-Renal Physiology 288, no. 4 (2005): F642—F649. http://dx.doi.org/10.1152/ajprenal.00287.2004.

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The use of LiCl in clinical psychiatry is routinely complicated by overt nephrogenic diabetes insipidus (NDI), the mechanism of which is incompletely understood. In vitro studies indicate that lithium can induce renal medullary interstitial cell cyclooxygenase 2 (COX2) protein expression via inhibition of glycogen synthase kinase-3β (GSK-3β). Both COX1 and COX2 are expressed in the kidney. Renal prostaglandins have been suggested to play an important role in lithium-induced polyuria. The present studies examined whether induction of the COX2 isoform contributes to LiCl-induced polyuria. Four d
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Hao, Chuan-Ming, Martin Kömhoff, Youfei Guan, Reyadh Redha, and Matthew D. Breyer. "Selective targeting of cyclooxygenase-2 reveals its role in renal medullary interstitial cell survival." American Journal of Physiology-Renal Physiology 277, no. 3 (1999): F352—F359. http://dx.doi.org/10.1152/ajprenal.1999.277.3.f352.

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Renal medullary interstitial cells (MICs) are a major site of cyclooxygenase (COX)-mediated PG synthesis. These studies examined the role of COX in MIC survival. Immunoblot and nuclease protection demonstrate that cultured MICs constitutively express COX2, with little constitutive COX1 expression. SC-58236, a COX2-selective inhibitor, but not SC-58560, a COX1 inhibitor, preferentially blocks PGE2 synthesis in MICs. Transduction with a COX2 antisense adenovirus reduced MIC COX2 protein expression and also decreased PGE2production. Antisense downregulation of COX2 was associated with MIC death,
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Ricketts, Anthony P., Kristin M. Lundy, and Scott B. Seibel. "Evaluation of selective inhibition of canine cyclooxygenase 1 and 2 by carprofen and other nonsteroidal anti-inflammatory drugs." American Journal of Veterinary Research 59, no. 11 (1998): 1441. http://dx.doi.org/10.2460/ajvr.1998.59.11.1441.

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Abstract Objective To evaluate the activity of carprofen and other nonsteroidal anti-inflammatory drugs (NSAID) against isozymes of canine cyclooxygenases (COX1 and COX2). Procedure Constitutive COX1 was obtained from washed canine platelets, and COX2 was obtained from a canine macrophage-like cell line that was induced with endotoxin. Activity of carprofen and other NSAID against COX1 and COX2 was compared. Dose-response curves were plotted, and calculations were performed to identify concentrations that caused 50% inhibition (IC50[μM]) for each isozyme. Ratio of the COX1-to-COX2 IC50 was use
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Chen, Haolin, Lindi Luo, June Liu, and Barry R. Zirkin. "Cyclooxygenases in Rat Leydig Cells: Effects of Luteinizing Hormone and Aging." Endocrinology 148, no. 2 (2007): 735–42. http://dx.doi.org/10.1210/en.2006-0925.

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Previous studies suggested that increased Leydig cell cyclooxygenase (COX)2 expression may be involved in the reduced testosterone production that characterizes aged Leydig cells. Our objective herein was to further elucidate the relationships among LH stimulation, Leydig cell COX2 and COX1 expression, aging, and testosterone production. Incubation of Leydig cells from young or aged rats with LH or dibutyryl cAMP resulted in increases in both intracellular COX2 protein expression and testosterone production. COX1 expression did not respond to LH or dibutyryl cAMP. Incubation of adult cells wit
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Ren, Rongqin, Peter C. Charles, Chunlian Zhang, Yaxu Wu, Hong Wang, and Cam Patterson. "Gene expression profiles identify a role for cyclooxygenase 2–dependent prostanoid generation in BMP6-induced angiogenic responses." Blood 109, no. 7 (2006): 2847–53. http://dx.doi.org/10.1182/blood-2006-08-039743.

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Abstract The bone morphogenetic protein (BMP) family of proteins participates in regulation of angiogenesis in physiologic and pathologic conditions. To investigate the molecular mechanisms that contribute to BMP-dependent angiogenic signaling, we performed gene expression profiling of BMP6-treated mouse endothelial cells. We detected 77 mRNAs that were differentially regulated after BMP6 stimulation. Of these, cyclooxygenase 2 (Cox2) was among the most highly up-regulated by BMP stimulation, suggesting a role for Cox2 as a downstream regulator of BMP-induced angiogenesis. Up-regulation of Cox
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Mohammed Al bratty, Mohammed Al bratty, Neelaveni Thangavel Neelaveni Thangavel, Hassan Ahmad Alhazmi Hassan Ahmad Alhazmi, et al. "Gas Chromatography-Mass Spectrometry-based Phytochemical Analysis and In-Vitro Anti-Lipid Peroxidation, Cyclooxygenase Inhibition Activities of Saudi Eruca sativa Leaves." Journal of the chemical society of pakistan 44, no. 2 (2022): 175. http://dx.doi.org/10.52568/001001/jcsp/44.02.2022.

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Eruca sativa is a wholesome yearly shrubbery herb in Saudi Arabia. Eruca sativa leaves are a conventional food and are consumed raw in salads. The present research reports the phytochemical analysis, in-vitro anti-lipid peroxidation, total anti-oxidant capability, cyclooxygenase-1, and -2 (COX1 and COX2) inhibition activities of Saudi Eruca sativa leaves water decoction (EWD). Gas chromatography-mass spectrometry (GC-MS) of EWD revealed seventeen constituents of six different chemical groups: phenolics (23.60%), aromatic and aliphatic esters (16.97%), terpenoids (31.91%), heterocyclics (14.83%
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Upmacis, Rita K., Wendy L. Becker, Donna M. Rattendi, et al. "Analysis of Sex-Specific Prostanoid Production Using a Mouse Model of Selective Cyclooxygenase-2 Inhibition." Biomarker Insights 17 (January 2022): 117727192211421. http://dx.doi.org/10.1177/11772719221142151.

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Background: Prostanoids are a family of lipid mediators formed from arachidonic acid by cyclooxygenase enzymes and serve as biomarkers of vascular function. Prostanoid production may be different in males and females indicating that different therapeutic approaches may be required during disease. Objectives: We examined sex-dependent differences in COX-related metabolites in genetically modified mice that produce a cyclooxygenase-2 (COX2) enzyme containing a tyrosine 385 to phenylalanine (Y385F) mutation. This mutation renders the COX2 enzyme unable to form a key intermediate radical required
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Cherney, David Z. I., James W. Scholey, Rania Nasrallah, et al. "Renal hemodynamic effect of cyclooxygenase 2 inhibition in young men and women with uncomplicated type 1 diabetes mellitus." American Journal of Physiology-Renal Physiology 294, no. 6 (2008): F1336—F1341. http://dx.doi.org/10.1152/ajprenal.00574.2007.

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In experimental studies, cyclooxygenase 2 (COX2)-derived vasodilatory prostaglandins play a more prominent role in arterial vasoregulation in females. The gender-dependent effect of COX2 modulation in humans with type 1 diabetes mellitus (DM) is unknown. Accordingly, we examined the renal hemodynamic role of prostaglandins by assessing the response to COX2 inhibition in young men and women with type 1 DM. We also used a graded ANG II infusion to determine whether gender-based differences were mediated by effects of COX2 inhibition on the renin angiotensin system (RAS). We hypothesized that COX
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Stefanski, Casey D., Daniel A. Erkes, Timothy J. Purwin, et al. "Abstract A023: Arachidonic acid metabolism promoting cross resistance in melanoma cells." Cancer Research 84, no. 22_Supplement (2024): A023. http://dx.doi.org/10.1158/1538-7445.tumbody-a023.

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Abstract Melanoma patients initially respond well to first line immunotherapy. However, cross resistance to immunotherapy and BRAF/MEK inhibitor is a clinical challenge for melanoma care. To investigate drug tolerant and resistant mechanisms promoting cross resistance, we analyzed RNA-sequencing and gene set enrichment targeted therapy resistant melanoma cells revealing an enriched arachidonic acid gene signature. Intracellular arachidonic acid levels were increased in drug resistant and drug tolerant melanoma models. Arachidonic acid is enzymatically converted by COX1 and COX2 to prostaglandi
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Park, James L., Liming Shu та James A. Shayman. "Differential involvement of COX1 and COX2 in the vasculopathy associated with the α-galactosidase A-knockout mouse". American Journal of Physiology-Heart and Circulatory Physiology 296, № 4 (2009): H1133—H1140. http://dx.doi.org/10.1152/ajpheart.00929.2008.

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The lysosomal storage disorder Fabry disease is characterized by excessive globotriaosylceramide (Gb3) accumulation in major organs such as the heart and kidney. Defective lysosomal α-galactosidase A (Gla) is responsible for excessive Gb3 accumulation, and one cell sensitive to the effects of Gb3 accumulation is vascular endothelium. Endothelial dysfunction is associated with Fabry disease and excessive cellular Gb3. We previously demonstrated that excessive vascular Gb3 in a mouse model of Fabry disease, the Gla-knockout ( Gla−/0) mouse, results in abnormal vascular function, which includes a
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Dissertations / Theses on the topic "COX2 inhibition"

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Ding, Yuan. "Mechanistic Understanding of CO2 Corrosion Inhibition at Elevated Temperatures." Ohio University / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1547122087551426.

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Vajrala, Sucheta Gowthami. "Mechanism of CO2 inhibition in insect cell culture." Thesis, University of Iowa, 2010. https://ir.uiowa.edu/etd/611.

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The prominence of insect cell culture has grown rapidly due to its ability to produce baculovirus biopesticides and recombinant proteins using the Baculovirus Expression Vector System. A critical problem in the mass production of these products is CO2 accumulation to inhibitory levels within the bioreactor. The current research investigated the effect of elevated CO2 concentrations on insect cell growth and metabolism and the roles of oxidative stress and intracellular pH (pHi) in CO2 inhibition. Spodoptera frugiperda Sf-9 insect cells were cultured in a 3 L bioreactor (1.2 L working volume) c
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Zibell, Guido. "Prävention der anfallsinduzierten Pgp-Überexpression durch COX-2-Inhibition." Diss., lmu, 2009. http://nbn-resolving.de/urn:nbn:de:bvb:19-96951.

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Rattanatray, Leewen. "COX-2 inhibition during development and subsequent renal function /." Title page and abstract only, 2004. http://web4.library.adelaide.edu.au/theses/09SB/09sbr237.pdf.

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Al-Sayed, Mohd S. A. "Effect of flow and pH on CO2 corrosion and inhibition." Thesis, University of Manchester, 1989. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.488183.

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Dovedi, Simon Julian. "COX-2 inhibition : a role in the immunotherapy of urothelial carcinoma?" Thesis, University of Newcastle Upon Tyne, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.430698.

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Li, Wei. "Mechanical Effects of Flow on CO2 Corrosion Inhibition of Carbon Steel Pipelines." Ohio University / OhioLINK, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1461751721.

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Wenner, Megan M. "The role of COX-2 inhibition in salt sensitivity of blood pressure." Access to citation, abstract and download form provided by ProQuest Information and Learning Company; downloadable PDF file, 135 p, 2009. http://proquest.umi.com/pqdweb?did=1674099691&sid=3&Fmt=2&clientId=8331&RQT=309&VName=PQD.

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Benoit, Jeremiah. "SUPERCRITICAL PHASE FISCHER-TROPSCH SYNTHESIS INHIBITION OF CO2 SELECTIVITY FOR ENHANCED HYDROCARBON PRODUCTION." OpenSIUC, 2008. https://opensiuc.lib.siu.edu/theses/485.

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ABSTRACT This thesis presents the results from research conducted on Fischer-Tropsch synthesis (FTS) in supercritical CO2 from syngas (H2:CO =1:1) typically produced from coal gasification and using a Fe-Zn-K catalyst. Experiments were conducted with syngas alone at different pressures (200 psi - 1050 psi) and temperatures (275, 350 and 375 oC). Experiments were also conducted with a syngas pressure of 200 psi and at different partial pressures of an inert diluent (N2) such that the total pressure varied from 200 psi to 1050 psi. Finally, experiments were conducted with CO2 as a diluent and
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Sokola, Brent S. "DUAL LOX/COX INHIBITION: A NOVEL STRATEGY TO PREVENT NEUROVASCULAR LEAKAGE IN EPILEPSY." UKnowledge, 2018. https://uknowledge.uky.edu/pharmacy_etds/83.

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Epilepsy affects 3.4 million patients in the USA and is characterized by recurring seizures. The blood-brain barrier is leaky in epilepsy and may contribute to seizure progression but the mechanisms which cause this leakage are not fully understood. We hypothesized that seizures trigger LOX- and COX-mediated blood-brain barrier leakage and that dual LOX/COX inhibition prevents barrier leakage in vivo. To test this hypothesis, we administered either the dual LOX/COX inhibitor licofelone or a combination of the 5-LOX inhibitor zileuton and the COX-2 inhibitor celecoxib to rats that experienced s
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Books on the topic "COX2 inhibition"

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M. A. M. Abou Zour. Inhibitive effects of thiourea and mixed inhibitor systems on mild steel in CO2 solutions. UMIST, 1997.

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Arousi, A. A. Inhibition of CO2 corrosion in crude oils and brine. UMIST, 1993.

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Elaghtaa, A. R. Effect of the presence of precorrosion on the efficiency of inhibitor in CO2 corrosion. UMIST, 1995.

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Malik, H. P. The influence of pH and surface films on corrosion inhibitor performance in CO2 saturated 5% NaC1. UMIST, 1992.

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Abdulrahman, A. Influence of pre-corrosion on the inhibitor efficiency of a quaternary amine in oxygen-free CO2 containing solution. UMIST, 1996.

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Emery, Paul, John R. Vane, and Regina Botting. Clinician's Manual on COX-2 Inhibition. 2nd ed. Science Press Inc., 1999.

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COX-2 Inhibitor Research. Nova Science Publishers, 2006.

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Howardell, Maynard J. Cox-2 Inhibitor Research. Nova Science Publishers, Incorporated, 2006.

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Vane and Botting. Clinician's Manual on Cox-2 Inhibition and Arthritis. 2nd ed. Science Press Inc., 2002.

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Howardell, Maynard J. Trends in Cox-2 Inhibitor Research. Nova Science Publishers, 2006.

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Book chapters on the topic "COX2 inhibition"

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Dubois, R. N., H. Sheng, J. Shao, C. Williams, and R. D. Beauchamp. "Inhibition of intestinal tumorigenesis via selective inhibition of COX-2." In Selective COX-2 Inhibitors. Springer Netherlands, 1998. http://dx.doi.org/10.1007/978-94-011-4872-6_6.

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Mohan, Aarthi R., and Phillip R. Bennett. "Reproduction: role of COX-2 and its inhibition." In COX-2 Inhibitors. Birkhäuser Basel, 2004. http://dx.doi.org/10.1007/978-3-0348-7879-1_10.

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Furst, D. E. "Meloxicam: selective COX-2 inhibition in clinical practice." In Selective COX-2 Inhibitors. Springer Netherlands, 1998. http://dx.doi.org/10.1007/978-94-011-4872-6_14.

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Ellison, D. Hank. "COX-Inhibiting Blood Agents." In Handbook of Chemical and Biological Warfare Agents, Volume 1, 3rd ed. CRC Press, 2022. http://dx.doi.org/10.4324/9781003230571-10.

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Espie, George S., Anthony G. Miller, and David T. Canvin. "Selective Inhibition of CO2 Transport in a Cyanobacterium." In Current Research in Photosynthesis. Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-0511-5_745.

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Patrono, C., P. Patrignani, M. R. Panara, et al. "COX-2 expression and inhibition in human monocytes." In Improved Non-Steroid Anti-Inflammatory Drugs: COX-2 Enzyme Inhibitors. Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-010-9029-2_7.

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Gierse, James, Ravi Kurumbail, Mark Walker, et al. "Mechanism of Inhibition of Novel Cox-2 Inhibitors." In Advances in Experimental Medicine and Biology. Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0193-0_56.

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Pairet, M., J. Van Ryn, A. Mauz, et al. "Differential inhibition of COX-1 and COX-2 by NSAIDs: a summary of results obtained using various test systems." In Selective COX-2 Inhibitors. Springer Netherlands, 1998. http://dx.doi.org/10.1007/978-94-011-4872-6_3.

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Bruno, Annalisa, Melania Dovizio, and Paola Patrignani. "Molecular and Experimental Basis for COX Inhibition in Cancer." In NSAIDs and Aspirin. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-33889-7_12.

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Petersen, S., W. Eicheler, C. Petersen, N. Hunter, and L. Milas. "COX-2 Inhibition als Angriffspunkt in der Therapie kolorektaler Karzinome." In Deutsche Gesellschaft für Chirurgie. Springer Berlin Heidelberg, 2001. http://dx.doi.org/10.1007/978-3-642-56698-1_30.

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Conference papers on the topic "COX2 inhibition"

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Okafor, P. C., C. B. Liu, and Y. G. Zheng. "Corrosion Inhibition of Pipeline Steels in CO2-Saturate System by an Imidazoline Based Inhibitor." In CORROSION 2016. NACE International, 2016. https://doi.org/10.5006/c2016-07601.

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Abstract The corrosion inhibition characteristics of an imidazoline based inhibitor (IMI) on N80 and P110 pipeline steel in CO2-saturated simulated produced water and NaCl solutions was studied at pH 4 and at 25, 40 and 60°C using electrochemical methods. IMI was found to inhibit the corrosion of the carbon steels in the solution to a maximum of 98.8% for N80 in the simulated produce water and over 99% in the NaCl solution for both N80 and P110. Inhibition efficiency shows no significant dependence on the range of IMI concentration studied but dependent on the composition of the metal and corr
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Gulbrandsen, Egil, Rolf Nyborg, Trine Løland, and Kemal Nisancioglu. "Effect of Steel Microstructure and Composition on Inhibition of CO2 Corrosion." In CORROSION 2000. NACE International, 2000. https://doi.org/10.5006/c2000-00023.

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Abstract The effects of steel microstructure and composition on the inhibition of CO2 corrosion were studied at 25 °C, pH 5, 1 bar CO2 in 3 % NaCl solutions. Generic inhibitor compounds cocoalkyl-dimethyl-benzyl ammonium chloride (QUAT) and sodium thiosulfate (THIO) were used. Sixteen different carbon steels were tested with a blend of 40 ppm QUAT and 0.5 ppm THIO. The test specimens were precorroded for six days in the corrosive medium prior to inhibitor addition. The effect of inhibitors was assessed by electrochemical polarization tests. The inhibitor efficiency ranged from 84 to 98 %. The
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Gulbrandsen, Egil, and Jean-Hervé Morard. "Why Does Glycol Inhibit CO2 Corrosion?" In CORROSION 1998. NACE International, 1998. https://doi.org/10.5006/c1998-98221.

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Abstract CO2 corrosion of carbon steel and its inhibition in mixtures of water and ethylene glycol (MEG) or diethylene glycol (DEG) has been studied at 1 bar CO2, 25 °C, pH 5 under natural convection conditions. Corrosion rates predicted by the glycol at 1 bar CO2,25 °C, pH 5 under natural convection agreement with the experimental results. Analysis of the experimental results shows that the corrosion inhibitor effect of MEG and DEG can be correlated to changes in solution properties with increasing MEG or DEG concentration, such as decreased CO2 solubility, decreased CO2 diffusivity / increas
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Alink, B., B. Outlaw, V. Jovancicevic, S. Ramachandran, and S. Campbell. "Mechanism of CO2 Corrosion Inhibition by Phosphate Esters." In CORROSION 1999. NACE International, 1999. https://doi.org/10.5006/c1999-99037.

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Abstract The mechanisms of corrosion inhibition of mild steel by phosphate esters in a CO2 containing environment is studied by using various inhibitor solution characterization techniques (residual analysis, dynamic surface tension, emulsion tendency). Corrosion inhibition and inhibitor film persistancy is monitored over time by using a rotating cylinder electrode (RCE) system and linear polarization resistance (LPR) technique. Corrosion rate-time/concentration profiles are obtained for all phosphate esters studied. Optical profilometry is used to assess surface microstructure as related to i
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Minxu, Lu, Zhao Guoxian, Yan Milin, and Li Pingquan. "Research on the Inhibition Behavior of CO2 Corrosion Resistant Inhibitor." In CORROSION 1999. NACE International, 1999. https://doi.org/10.5006/c1999-99026.

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Abstract The inhibition efficiency and local corrosion resistance behavior were comparatively studied for three inhibitors in terms of the average corrosion rate measured by the weight-loss method. Besides, die alternating impedance and dynamic potential polarization curve were also used to investigate and analyze the electrochemical inhibition behavior for the three inhibitors.
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Pandarinathan, Vedapriya, Kateřina Lepková, and Rolf Gubner. "Inhibition of CO2 Corrosion of 1030 Carbon Steel beneath Sand-Deposits." In CORROSION 2011. NACE International, 2011. https://doi.org/10.5006/c2011-11261.

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Abstract The performance of three corrosion inhibitors was investigated at 1030 carbon steel surfaces in the presence and absence of a sand deposit. Potentiodynamic measurements showed that the inhibition efficiency to mitigate corrosion reactions decreases in the presence of sand deposit. In contrary, the inhibitor performance was found to increase with longer exposure time of the steel to the corrosive media, at sand deposited surfaces. The differences between the steels corroded with and without sand deposit in the presence of an inhibitor were confirmed using both potentiostatic polarisati
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Jovancicevic, V., Y. S. Ahn, J. Dougherty, and B. Alink. "CO2 Corrosion Inhibition by Sulfur-Containing Organic Compounds." In CORROSION 2000. NACE International, 2000. https://doi.org/10.5006/c2000-00007.

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Abstract Corrosion inhibition of mild steel in CO2 environments by sulfur-containing compounds was studied using various laboratory performance test methods, inhibitor residual analysis techniques and a surface imaging technique. Corrosion rate-time/concentration profiles were obtained for several commercially available sulfur-containing inhibitors together with a proprietary thio compound. The role of chelation and redox chemistry of these inhibitors in corrosion inhibition is discussed in terms of the structure-activity relationship. A number of formulated products with sulfur-containing com
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Bilkova, K., N. Hackerman, and M. Bartos. "Inhibition of CO2 Corrosion of Carbon Steel by Thioglycolic Acid." In CORROSION 2002. NACE International, 2002. https://doi.org/10.5006/c2002-02284.

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Abstract Corrosion inhibition of mild steel in aqueous salt environments containing CO2 by thioglycolic acid (TGA) was studied using various electrochemical and analytical techniques. The inhibitor performance and its inhibition efficiency at various concentrations and the effect of exposure time and pH of the solution were evaluated. The proposed mechanism of inhibition by TGA considers the complexity of experimental data obtained by various techniques. This mechanism is discussed in terms of adsorption, protective film formation, and especially pH effect and the chemistry of the solution. Th
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Yepez, Omar, Nihal Obeyesekere, and Jonathan Wylde. "Development of Novel Phosphate Based Inhibitors Effective for Oxygen Corrosion. Part II: Electrochemical Characterization." In CORROSION 2015. NACE International, 2015. https://doi.org/10.5006/c2015-06162.

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Abstract Corrosion inhibitors can behave as cathodic inhibitors when they mainly suppress the cathodic reaction. These can be anodic inhibitors when they mainly suppress the anodic reaction. When corrosion inhibitors interact with both cathodic and anodic reactions, they may behave as mixed inhibitors. This paper documents the electrochemical characterization of a phosphate ester based inhibitor which creates protection from oxygen corrosion. Potentiodynamic polarization is used to determine the inhibitor behavior during the anodic and cathodic processes of corrosion. A system of a carbon stee
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Gulbrandsen, Egil, and Arne Dugstad. "Corrosion Loop Studies of Preferential Weld Corrosion and its Inhibition in CO2 Environments." In CORROSION 2005. NACE International, 2005. https://doi.org/10.5006/c2005-05276.

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Abstract Preferential corrosion of carbon steel weld corrosion and its inhibition were studied by corrosion tests in a flow loop. Eight weld metal/parent metal combination were tested at 60 °C, 1 bar CO2, pH 5-6. Results from 3 selected welds are presented. The tests were carried out in high and low conductivity NaCl brines (0.35 and 35 g/L). It was found that corrosion inhibition could prevent preferential weld corrosion when inhibitor was dosed at sufficiently high concentration. A higher concentration was needed to inhibit the corrosion on some of the weld metals than the parent steel. Furt
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Reports on the topic "COX2 inhibition"

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Reddy, E. P. Novel COX-2 Inhibitor for Breast Cancer Therapy. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada444637.

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Reddy, E. P. Novel Cox-2 Inhibitor for Breast Cancer Therapy. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada431390.

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Reddy, E. P. Novel COX-2 Inhibitor for Breast Cancer Therapy. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada420352.

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Shahak, Yosepha, and Donald R. Ort. Physiological Bases for Impaired Photosynthetic Performance of Chilling-Sensitive Fruit Trees. United States Department of Agriculture, 2001. http://dx.doi.org/10.32747/2001.7575278.bard.

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Chilling-sensitivity is an important agricultural problem in both the U.S. and Israel. Most research attention has focused so far on herbaceous crop plants, even though the problem is also acute in the fruit tree industry. Under BARD funding we made substantial progress in identifying the mechanisms involved in the disruption of photosynthesis following a chill in mango. Our investigation with fruit trees has been substantially accelerated by drawing on our knowledge and experience with herbaceous crops. The four original research objectives, focused or discovering the underlying mechanisms of
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Marmol, Frederic. Effects of lithium treatment on oxidative stress markers in mitochondrial complex I and complex III inhibition and after CO2 exposure in SH-SY5Y cells. Science Repository OÜ, 2019. http://dx.doi.org/10.31487/j.nnb.2019.01.001.

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Paterek, J. Robert. L51963 Environmental Benign Mitigation of Microbiologically Influenced Corrosion (MIC). Pipeline Research Council International, Inc. (PRCI), 2002. http://dx.doi.org/10.55274/r0011299.

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The objective is to develop and evaluate environmental benign agents or products that are effective in the prevention, inhibition, and mitigation of microbially influenced corrosion (MIC) on the internal surfaces of metallic natural gas pipelines. The goal is one or more products that can be applied to maintain the structure and dependability of the natural gas infrastructure. The technical approach for this year has been to evaluate a number of methods to fractionate and concentrate the components found in the pepper extracts. The separation methods considered include size exclusion chromatog
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Alwagdani, Abdullah. Review Of mPGES-1 Inhibitors Based On The Benzoxazole And Its Isostere Scaffold For The Treatment Of Inflammatory Diseases. University of Tennessee Health Science Center, 2024. http://dx.doi.org/10.21007/com.lsp.2024.0021.

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The vital role of the prostanoid pathway in inflammation, pain, cancer, Alzheimer’s and many other diseases has attracted the drug discovery community to discover targets for therapeutic development. Although existing non-steroidal anti-inflammatory drugs (NSAIDs) inhibiting cyclooxygenases (COX) are widely used, the side effects of these NSAIDs limit the ling time medication. Microsomal prostaglandin E synthase-1 (mPGES-1) is an attractive target that is overexpressed during inflammations, and it could be a safe alternative to NSAIDs for treating inflammatory diseases.Since the discovery of m
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Moghissi. L51914 Interdependent Effects of Bacteria Gas Composition and Water Chemistry on Internal Corrosion. Pipeline Research Council International, Inc. (PRCI), 2002. http://dx.doi.org/10.55274/r0010433.

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A recent Office of Pipeline Safety survey found that corrosion caused 17 to 20 percent of pipeline failures. Of those corrosion failures, roughly half resulted from internal corrosion. In pipelines, internal corrosion is caused by produced (carry-over) or condensed water that contains dissolved gas and/or bacteria. In many cases, chemicals with inhibiting or biocidal properties are added to mitigate corrosion. The internal corrosion in many systems occurs under slowly flowing conditions at ambient temperatures (e.g., relatively low temperature of about 15.5�C (60�F)). The overall objectives of
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Lee, Yi-Fen. To Investigate the Therapeutic Efforts of the COX-2 Inhibitor NS-398 as a Single Agent, and in Combination With Vitamin D, in Vitro and in Vivo. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada448627.

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Pavasant, Prasit, and Tussanee Yongchaitrakul. Influence of mechanical stress on the expression of osteopontin in human periodontal ligament cells. Chulalongkorn University, 2008. https://doi.org/10.58837/chula.res.2008.14.

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Background: Mechanical stress such as orthodontic forces can produce mechanical damage and inflammatory reaction in periodontium. Osteopontin (OPN) is a multifunctional cytokine that found to be correlated with periodontal disease progression. As periodontal ligaments (PDL) can be affected by stress and PDL cells are involved in periodontal destruction and remodeling, we aimed to investigate the influence of mechanical stress on the expression and regulation of OPN in human PDL (HPDL) cells. Methods: The mechanical stress was generated by continuous compressive force and the expression of OPN
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