Relevant bibliographies by topics / Creatine phosphokinase (CPK)

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers

Academic literature on the topic 'Creatine phosphokinase (CPK)'

Author: Grafiati
Published: 4 June 2021
Last updated: 27 July 2025

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Journal articles on the topic "Creatine phosphokinase (CPK)"

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Lemos, Joshua, Cameron Gibson, Arek Wiktor, and Scott Mueller. "567 Creatine Phosphokinase: A Novel Biomarker for Frostbite Severity." Journal of Burn Care & Research 46, Supplement_1 (2025): S153. https://doi.org/10.1093/jbcr/iraf019.196.

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Abstract Introduction Serum creatine phosphokinase (CPK) levels in injuries involving muscular damage, such as trauma, are used as an indicator of severity of injury. The role of CPK in frostbite injuries is unknown. We hypothesize that there is an association between serum CPK levels and the severity of frostbite injury. Methods This is a single center retrospective chart review of patients admitted to an ABA verified burn center with an ICD-10 diagnosis of frostbite from October 2022 to July 2024. Patients were excluded from the study if they were under the age of 18, had end stage renal dis
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Ellington, W. R. "Phosphocreatine represents a thermodynamic and functional improvement over other muscle phosphagens." Journal of Experimental Biology 143, no. 1 (1989): 177–94. http://dx.doi.org/10.1242/jeb.143.1.177.

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In vertebrate tissues, the only phosphagen is phosphocreatine (PC), and the corresponding phosphotransferase is creatine phosphokinase (CPK). Among invertebrates, a variety of phosphotransferase reactions are found in addition to CPK, including arginine phosphokinase (APK), glycocyamine phosphokinase (GPK), taurocyamine phosphokinase (TPK) and lombricine phosphokinase (LPK). Although there is some uncertainty about the exact value, the apparent equilibrium constant for the CPK reaction (K′cpk = [creatine][ATP]/[PC][ADP]), under physiological conditions similar to those of vertebrate muscle, ra
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Egan, Michael F., Anissa Abi Dargham, Darrelll G. Kirch, and Richard Jed Wyatt. "Serum Creatine Phosphokinase Elevations in Patients with Tardive Dyskinesia." British Journal of Psychiatry 162, no. 4 (1993): 551–53. http://dx.doi.org/10.1192/bjp.162.4.551.

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We describe a case of marked increases in serum creatine phosphokinase (CPK) associated with a dramatic exacerbation of tardive dyskinesia (TD). Chart review of eight additional patients with severe TD revealed three cases with mild CPK elevations. These cases suggest that TD, especially when accompanied by dystonia, may be associated with pathology of striated muscle.
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Santos, André L. F., Guilherme G. Pereira, Mário M. Santos, Vanda C. R. Gutierrez, Priscila C. Calyjur, and Maria Helena M. A. Larsson. "Serum dosage of CPK-MB in dogs with ST deviation by chemiluminescence." Pesquisa Veterinária Brasileira 34, suppl 1 (2014): 74–78. http://dx.doi.org/10.1590/s0100-736x2014001300014.

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Abstract: Although frequently in humans, hypoxic and ischemic heart diseases are poorly documented in dogs, with only few reports of acute myocardial infarction (AMI) in this species. Some electrocardiographic findings might suggest myocardium hypoxia/ischemia, like ST segment elevation or depression, but there are no studies showing whether deviations in ST segment are associated to myocardial injury and serum increase of creatine phosphokinase (CPK-MB). In order to investigate possible myocardial cells injury in poor perfusion conditions, 38 dogs were studied, 20 with normal electrocardiogra
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Sharma, Nalini, Dimple Kharkongor, Ritisha Basu, et al. "Role of creatine phosphokinase as a diagnostic marker in tubal ectopic pregnancy." Journal of Family Medicine and Primary Care 12, no. 11 (2023): 2774–79. http://dx.doi.org/10.4103/jfmpc.jfmpc_2483_22.

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Background and Aim: Ectopic pregnancy (EP) is still one of the leading preventable causes of maternal morbidity and mortality in the first trimester. Amidst the use of sensitive assays for β-HCG and high-definition ultrasonography for the identification of EP, the search for a more reliable and sensitive marker remains a challenge till date. Our aim was to determine the validity of creatine phosphokinase (CPK) and its isoenzyme (CPK-MB) in the prediction of tubal EP. Materials and Methods: A prospective and comparative diagnostic accuracy study was conducted among 105 pregnant women in the fir
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Vasileva, S., R. Vasilev, A. Prusakov, and M. Golodyaeva. "Comparative assessment of the most important markers of equine myopathy in connection with creatine kinase activity." Genetics and breeding of animals, no. 2 (November 5, 2024): 44–50. http://dx.doi.org/10.31043/2410-2733-2024-2-44-50.

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Purpose: to assess the activity of AST, LDH and HBDH enzymes in relation to different levels of total creatine phosphokinase activity to understand the degree of their diagnostic value in determining the severity of myopathy in horses.Materials and methods. For the study, statistical processing of a data array of the results of a biochemical study of blood serum from 247 horses was carried out. Taking into account the reference intervals of CPK activity, all results were ranked by increasing creatine phosphokinase activity and divided into four groups. In each group, the average values for AST
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C., Deepika, P.Mohanapriya, Ramprasath R., et al. "Correlation between Respiratory Failure and Serum Creatine Phosphokinase Levels in Organophosphate Poisoning." International Journal of Pharmaceutical and Clinical Research 16, no. 11 (2024): 241–47. https://doi.org/10.5281/zenodo.14250239.

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<strong>Background:</strong>&nbsp;Organophosphate (OP) poisoning is a significant global health issue, particularly in agricultural settings, and is associated with severe toxicity, including respiratory distress, neurological impairment, and, in extreme cases, death. Early prognosis is critical in managing OP poisoning, and biomarkers such as serum creatine phosphokinase (CPK) have been suggested to predict patient outcomes. This study aims to evaluate the role of CPK levels as a prognostic indicator in organophosphate poisoning.&nbsp;<strong>Methods:</strong>&nbsp;A cross-sectional, prospect
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Badiger, Sharan, and Prashant Badiger. "A study of serum creatine phosphokinase and severity of organophosphorus poisoning." Annals of Geriatric Education and Medical Sciences 9, no. 2 (2023): 70–74. http://dx.doi.org/10.18231/j.agems.2022.015.

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Organophosphorus (OP) poisoning is an important global health problem especially in developing countries. Estimation of erythrocyte cholinesterase (EChE) and plasma cholinesterase (BChE) as an evidence of OP poisoning is costly, not regularly performed and shows wide inter-individual variability. To study the feasibility of using serum creatine phosphokinase (CPK) as a predictor for severity of OP poisoning. Thirty four patients were included in this study who were admitted to tertiary care centre with history of OP poisoning less than 24hrs. The clinical severity of patients categorized accor
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Hochachka, Peter W., and Mark K. P. Mossey. "Does muscle creatine phosphokinase have access to the total pool of phosphocreatine plus creatine?" American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 274, no. 3 (1998): R868—R872. http://dx.doi.org/10.1152/ajpregu.1998.274.3.r868.

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Two fundamental assumptions underlie currently accepted dogma on creatine phosphokinase (CPK) function in phosphagen-containing cells: 1) CPK always operates near equilibrium and 2) CPK has access to, and reacts with, the entire pool of phosphocreatine (PCr) and creatine (Cr). We tested the latter assumption in fish fast-twitch or white muscle (WM) by introducing [14C]Cr into the WM pool in vivo. To avoid complications arising from working with muscles formed from a mixture of fast and slow fibers, it was advantageous to work with fish WM because it is uniformly fast twitch and is anatomically
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K., Dhanalakshmi, Febina K., Sarala Sabapathy, Chaithra R., and Thileepan N. "Correlation of serum creatine phosphokinase and serum cholinesterase in organophosphate poisoning in children." International Journal of Contemporary Pediatrics 6, no. 6 (2019): 2352. http://dx.doi.org/10.18203/2349-3291.ijcp20194546.

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Background: Organophosphate (OP) poisoning is one of the most common pesticide poisoning in India in adolescents because of its easy availability. Serum pseudocholinesterase levels are commonly used to assess the severity and to know the prognosis in OP compound poisoning. Serum creatine phosphokinase (CPK) levels is another lab parameter which gets deranged in OP poisoning and has been tried in adults to assess the severity and to know the prognosis. Authors objective was to study the correlation of serum pseudocholinesterase and serum CPK in organophosphate poisoning at admission and to comp
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Dissertations / Theses on the topic "Creatine phosphokinase (CPK)"

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Bourdenet, Gwladys. "Étude physiopathologique de la myopathie auto-immune des souris NOD invalidées pour la voie de costimulation ICOS/ICOSL." Thesis, Normandie, 2017. http://www.theses.fr/2017NORMR062/document.

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Les myopathies inflammatoires (MI) représentent un groupe hétérogène de maladies caractérisépar une faiblesse musculaire chronique et symétrique associée à une augmentation du taux sérique decréatine phosphokinase (CPK). Les MI sont actuellement subdivisées en 5 entitées : les dermatomyosites,les myopathies nécrosantes auto-immunes, la myosite à inclusion, la polymyosite et les myosites dechevauchement. A ce jour, le diagnostic des MI repose sur l’association de signes cliniques, decaractéristiques anatomopathologiques sur la biopsie musculaire et la présence d’auto-anticorps (aAc). Eneffet, l
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Book chapters on the topic "Creatine phosphokinase (CPK)"

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Liveson, Jay Allan. "Hyper-CPK-emia and Muscle Aches." In Peripheral Neurology. Oxford University PressNew York, NY, 2000. http://dx.doi.org/10.1093/oso/9780195135633.003.0106.

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Abstract A 48-year-old woman complained of diffuse muscle aches maximal in the lower limbs during the previous 7 years. She was found to have markedly increased serum creatine phosphokinase (CPK) levels and was referred for evaluation. She admitted to mild generalized weakness and some numbness of her fingers. There was no previous or family medical history. Physical Examination. She had normal cranial nerves, cerebellar function, sensation, and even strength. DTRs were symmetrically active in the upper limbs and absent in the lower limbs. No pathologic reflexes were present. Nerves were norma
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Kappagoda, Shanthi, and Upinder Singh. "Infectious polymyositis." In Schlossberg's Clinical Infectious Disease, edited by Cheston B. Cunha. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780190888367.003.0072.

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This chapter discusses infectious polymyositis, which is a rare entity in which there is generalized muscle damage (rhabdomyolysis) caused by an infectious agent. The syndrome of rhabdomyolysis results from muscle necrosis and is characterized by muscle pain, elevated serum creatinine phosphokinase (CPK) concentrations, and myoglobinuria which may lead to renal dysfunction. A variety of factors can lead to rhabdomyolysis. These include crush and compression injuries, drug and alcohol ingestion, metabolic and electrolyte disturbances, hypothermia and hyperthermia, envenomation, and a variety of
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Conference papers on the topic "Creatine phosphokinase (CPK)"

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Sangam, S., S. Sharma, B. Patel, and V. Bengualid. "Legionella Pneumonia With Rhabdomyolysis With Extremely High Creatine Phosphokinase (CPK) Without Acute Kidney Injury (AKI)." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a4161.

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Hanss, M., D. Rousson, P. Touboul, and M. Dechavanne. "EARLY SPONTANEOUS FIBRINOLYSIS IS RELATED TO THE EXTENSION OF ACUTE MYOCARDIAL INFARCTION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643015.

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The relative importance of spontaneous fibrinolysis was studied during the early stage of acute myocardial infarction (AMI). Ten consecutive male patients (52.1 ± 5.7 years old) with AMI and without haemodynamic or rythmic complications were selected. Blood samples were obtained less than 5 hours (3.4 ± 0.8 hours) after the onset of symptoms. Enzyme linked immunosorbent assay procedures were performed to quantify the D-dimer antigen (Dd-Ag) and tissue plasminogen activator antigen (tPA-Ag) plasma levels. The creatine phosphokinase (CPK) peak level was measured in serum as an index of the AMI e
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Oura, Y., N. Sakiyama, R. Ueshima, M. Higuchi, E. Kakishitha, and K. Nagai. "Effect of Platelet Activating Factor(PAF) on the collagen induced platelet aggregation in whole blood." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643481.

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Studies of platelet aggregation are generally performed in p1ate1et-rich plasma(PRP) by the transmittance method. Recently, impedance aggregometry has been introduced which shows the platelet aggregability in whole blood. We compared the impedance aggregometry in whole blood with the transmittance method in PRP, with regard to collagen induced platelet aggregation. The aggregation rate in whole blood increased with increasing concentration of collagen, but remained unchanged in PRP. The factors which influence the platelet aggregation rate in whole blood were studied. CV-3988, that is the spec
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Carvalho, Ana Cláudia Pires, Fernanda Rezende Dias, Luisa Crevelin Costa, and Natália de Castro Fim Nakao. "Myopathy following statin use." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.512.

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Introduction: Myopathies can be caused by various drugs, including statins and corticosteroids, and can be toxic or inflammatory, one example being necrotizing myositis triggered by statins. Objectives: Describe the case of a patient with weakness after statin use. Design and setting: Case report Methods: Analysis of medical record, photographic record of the diagnostic methods and literature review. Case description: 69-year-old female, obese, hypertensive, diabetic, dyslipidemic and hypothyroid, taking atorvastatin since 2017, referred by endocrinology for generalized myalgia in 2019, with i
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Winocour, P. P., P. D. Rand, J. D. Vickers, R. L. Kinlough-Rathbone, and J. F. Mustard. "ENHANCED THROMBIN-INDUCED AGGREGATION AND INOSITOL TRISPHOSPHATE FORMATION OF PLATELETS FROM SPONTANEOUSLY HYPERCHOLESTEROLEMIC RATS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643811.

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Platelets from rats with diet-induced hypercholesterolemia are hypersensitive to thrombin through a pathway independent of released ADP or thromboxane A2 (TXA2) formation. We examined if platelets from rats with spontaneous hypercholesterolemia (HC) are similarly hypersensitive. HC rats (plasma cholesterol: 130±4 mg/dl, n=15) were compared with their normocholesterolemic genetic controls (NC) (87±4 mg/dl, p&lt;0.001, n=16). Total cholesterol/109 platelets was not different between the groups (HC: 0.314±0.032 μmole, n=7; NC: 0.357±0.046 ymole, n=7). Washed platelets were prelabelled with 14c-se
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