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Journal articles on the topic 'Creutzfeldt-Jakob disease'

Author: Grafiati

Published: 4 June 2021

Last updated: 17 June 2025

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1

Kovacs, G. G., S. Horvath, T. Strobel, et al. "Genetic Creutzfeldt-Jakob disease mimicking variant Creutzfeldt-Jakob disease." Journal of Neurology, Neurosurgery & Psychiatry 80, no. 12 (2009): 1410–11. http://dx.doi.org/10.1136/jnnp.2008.163733.

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2

Martindale, Jennifer, Michael D. Geschwind, Stephen De Armond, et al. "Sporadic Creutzfeldt-Jakob Disease Mimicking Variant Creutzfeldt-Jakob Disease." Archives of Neurology 60, no. 5 (2003): 767. http://dx.doi.org/10.1001/archneur.60.5.767.

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3

Stricof, Rachel L., Patricia P. Lillquist, Nadia Thomas, Ermias D. Belay, Lawrence B. Schonberger, and Dale L. Morse. "An Investigation of Potential Neurosurgical Transmission of Creutzfeldt-Jakob Disease: Challenges and Lessons Learned." Infection Control & Hospital Epidemiology 27, no. 3 (2006): 302–4. http://dx.doi.org/10.1086/503017.

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In 2001, New York State health officials were notified about 2 patients with Creutzfeldt-Jakob disease who had undergone neurosurgical procedures at the same hospital within 43 days of each other. One patient had Creutzfeldt-Jakob disease at the time of surgery; the other patient developed Creutzfeldt-Jakob disease 6.5 years later. This investigation highlights the difficulties in assessing possible transmission of Creutzfeldt-Jakob disease.

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4

Yash Srivastav and Abdul Hameed. "Creutzfeldt-Jakob Disease (CJD): A Deadly Neurodegenerative Condition; Its Epidemiology, Monitoring, Current Status of Prevention and Circumvention." Asian Journal of Pharmaceutical Research and Development 12, no. 6 (2024): 125–30. https://doi.org/10.22270/ajprd.v12i6.1491.

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Creutzfeldt-Jakob Disease CJDis a rare brain ailment that causes dementia. It is often referred to as Creutzfeldt-Jakob. It is a member of a class of illnesses called prion disorders that affect both humans and animals. Alzheimer's disease and Creutzfeldt-Jakob disease symptoms can be comparable.A rare yet deadly neurodegenerative condition is Creutzfeldt-Jakob disease (CJD). According to reports, it affects one in one to one and a half million people worldwide each year, with less than 1,000 cases occurring in the US.A deadly neurodegenerative condition, Creutzfeldt-Jakob disease (CJD) is also referred to as subacute spongiform encephalopathy or neurocognitive dysfunction caused by prion disease. Memory issues, behavioural changes, poor coordination, and visual impairments are some of the early indications. Dementia, uncontrollable movements, blindness, paralysis, and coma are later signs. Within a year, almost 70% of people pass away.In 1922, Walther Spielmeyer coined the term "Creutzfeldt–Jakob disease" in honour of the German neurologists Hans Gerhard Creutzfeldt and Alfons Maria Jakob. A protein called a prion folds abnormally to cause CJD. Misfolded proteins known as infectious prions can cause other misfolded proteins to do the same.People with CJD experience a variety of symptoms, such as uncontrollable muscle spasms or mobility issues, memory loss, and cognitive challenges. Because CJD causes so much damage, it is ultimately fatal.The diseases CJD and variant CJD (vCJD) are not the same, despite their very similar names. They're both prion illnesses. But eating meat from cows with bovine spongiform encephalopathy, also known as Mad Cow Disease, is linked to variant CJD. CJD mostly happens seldom and is sometimes referred to as "classic CJD" to prevent confusion.Although there isn't a known cure for Creutzfeldt-Jakob disease (CJD), the National Prion Clinic is doing clinical research to look at potential therapies. Currently, the goal of treatment is to keep the patient as comfortable as possible while using medications to lessen symptoms.The present state of Creutzfeldt-Jakob disease (CJD), its underlying causes, and possible treatments are assessed in this article.

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5

Le, Viet H. "Creutzfeldt-Jakob Disease." Journal of the American Osteopathic Association 119, no. 1 (2019): 63. http://dx.doi.org/10.7556/jaoa.2019.010.

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6

Coulthart, Michael B., and Beau M. Ances. "Creutzfeldt-Jakob disease." Neurology: Clinical Practice 5, no. 2 (2015): 99–101. http://dx.doi.org/10.1212/cpj.0000000000000103.

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7

Maldonado, Sonny, James B. Gross, and Eileen H. Bigio. "Creutzfeldt-Jakob Disease." Baylor University Medical Center Proceedings 11, no. 2 (1998): 89–98. http://dx.doi.org/10.1080/08998280.1998.11930089.

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8

MASTERS, COLIN L. "CREUTZFELDT-JAKOB DISEASE." Alzheimer Disease & Associated Disorders 3, no. 1 (1989): 46–51. http://dx.doi.org/10.1097/00002093-198903010-00006.

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9

Su, Ming-Shung, Shuu-Jiun Wang, Hung Chiang, et al. "Creutzfeldt- Jakob Disease." European Neurology 33, no. 3 (1993): 241–43. http://dx.doi.org/10.1159/000116945.

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10

Finkelstein, Lori E., Meryl H. Mendelson, Carmelita Tejero, and Jeanine Troccoli. "Creutzfeldt-Jakob Disease." American Journal of Nursing 98, no. 9 (1998): 66–67. http://dx.doi.org/10.1097/00000446-199809000-00054.

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11

Collie, Donald. "Creutzfeldt-Jakob Disease." Practical Neurology 2, no. 3 (2002): 168–72. http://dx.doi.org/10.1046/j.1474-7766.2002.05057.x.

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12

GENÇER, Ali Görkem, Zerrin PELIN, Cem İsmail KÜÇÜKALI, Özgür Bilgin TOPÇUOĞLU, and Nuriye YILMAZ. "Creutzfeldt-Jakob disease." Psychogeriatrics 11, no. 2 (2011): 119–24. http://dx.doi.org/10.1111/j.1479-8301.2011.00361.x.

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13

Urbach, H. "Creutzfeldt-Jakob disease." NeuroReport 11, no. 17 (2000): L5. http://dx.doi.org/10.1097/00001756-200011270-00001.

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14

Tyler, Kenneth L. "Creutzfeldt–Jakob Disease." New England Journal of Medicine 348, no. 8 (2003): 681–82. http://dx.doi.org/10.1056/nejmp020187.

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15

Weinstein, Martin. "Creutzfeldt-Jakob Disease." Neurological Research 14, no. 4 (1992): 355–56. http://dx.doi.org/10.1080/01616412.1992.11740084.

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16

Davanipour, Zoreh, Milton Alter, and Eugene Sobel. "Creutzfeldt-Jakob Disease." Neurologic Clinics 4, no. 2 (1986): 415–26. http://dx.doi.org/10.1016/s0733-8619(18)30978-2.

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17

McLean, Catriona. "Creutzfeldt-Jakob disease." Pathology 43 (2011): S26. http://dx.doi.org/10.1016/s0031-3025(16)33139-7.

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18

Verdrager, J., Chris MacKnight, Kenneth Rockwood, and A. R. Michell. "Creutzfeldt-Jakob disease." Lancet 347, no. 9016 (1996): 1704–5. http://dx.doi.org/10.1016/s0140-6736(96)91540-8.

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19

McPherson, Klim, RichardH Kimberlin, John Jacobs, et al. "Creutzfeldt-Jakob disease." Lancet 347, no. 8993 (1996): 64–66. http://dx.doi.org/10.1016/s0140-6736(96)91601-3.

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20

Clement. "Creutzfeldt-Jakob Disease." Haemophilia 6, s1 (2000): 68–78. http://dx.doi.org/10.1046/j.1365-2516.2000.00049.x.

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21

Narula, Reshma, and Sule Tinaz. "Creutzfeldt–Jakob Disease." New England Journal of Medicine 378, no. 4 (2018): e7. http://dx.doi.org/10.1056/nejmicm1710121.

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22

PEARL, GARY S., and ROBERT E. ANDERSON. "Creutzfeldt-Jakob Disease." Southern Medical Journal 82, no. 9 (1989): 1177–79. http://dx.doi.org/10.1097/00007611-198909000-00031.

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23

Zeidler, Martin. "Creutzfeldt-Jakob Disease." Scottish Medical Journal 47, no. 5 (2002): 100–104. http://dx.doi.org/10.1177/003693300204700502.

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24

Neatherlin, Jacquelin S. "Creutzfeldt-Jakob Disease." Journal of Neuroscience Nursing 20, no. 5 (1988): 309–13. http://dx.doi.org/10.1097/01376517-198810000-00008.

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25

Bailes, Barbara K. "Creutzfeldt-Jakob Disease." AORN Journal 52, no. 5 (1990): 976–85. http://dx.doi.org/10.1016/s0001-2092(07)69158-3.

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26

Foncin, Jean-François, Jean-Philippe Deslys, Corinne Ida Lasmezas, et al. "Creutzfeldt-Jakob disease." Lancet 347, no. 9011 (1996): 1332–33. http://dx.doi.org/10.1016/s0140-6736(96)90979-4.

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27

Kitamoto, Tetsuyuki. "Creutzfeldt-Jakob disease." Neuropathology 20, s1 (2000): 52–54. http://dx.doi.org/10.1046/j.1440-1789.2000.00308.x.

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28

Porter, Marie-Clare, and Michelle Leemans. "Creutzfeldt–Jakob disease." Continuing Education in Anaesthesia Critical Care & Pain 13, no. 4 (2013): 119–24. http://dx.doi.org/10.1093/bjaceaccp/mkt002.

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29

Wood, JamesL N., and AndrewJ Hall. "Creutzfeldt-Jakob disease." Lancet 338, no. 8760 (1991): 182–83. http://dx.doi.org/10.1016/0140-6736(91)90168-o.

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30

Iwasaki, Yasushi. "Creutzfeldt-Jakob disease." Neuropathology 37, no. 2 (2016): 174–88. http://dx.doi.org/10.1111/neup.12355.

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31

Pearce, J. M. S. "Jakob-Creutzfeldt Disease." European Neurology 52, no. 3 (2004): 129–31. http://dx.doi.org/10.1159/000081462.

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32

Kernich, Catherine A. "Creutzfeldt-Jakob Disease." Neurologist 13, no. 5 (2007): 329–30. http://dx.doi.org/10.1097/nrl.0b013e31814b8ef0.

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33

Matthews, W. B. "Creutzfeldt-Jakob disease." BMJ 291, no. 6493 (1985): 483. http://dx.doi.org/10.1136/bmj.291.6493.483-a.

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34

Baker, H. F., R. M. Ridley, and T. J. Crow. "Creutzfeldt-Jakob disease." BMJ 291, no. 6498 (1985): 822–23. http://dx.doi.org/10.1136/bmj.291.6498.822-a.

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35

Gasser, G. "Creutzfeldt-Jakob disease." BMJ 300, no. 6738 (1990): 1523. http://dx.doi.org/10.1136/bmj.300.6738.1523.

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36

Harries-Jones, R. "Creutzfeldt-Jakob disease." BMJ 301, no. 6742 (1990): 46. http://dx.doi.org/10.1136/bmj.301.6742.46-c.

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37

MANUELIDIS, ELIAS E. "Creutzfeldt-Jakob Disease." Journal of Neuropathology and Experimental Neurology 44, no. 1 (1985): 1–17. http://dx.doi.org/10.1097/00005072-198501000-00001.

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38

&NA;. "Creutzfeldt-Jakob disease." Advances in Anatomic Pathology 4, no. 6 (1997): 383. http://dx.doi.org/10.1097/00125480-199711000-00038.

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39

Benninger, Felix F., and Israel Steiner. "Creutzfeldt-Jakob Disease." JAMA Neurology 70, no. 12 (2013): 1588. http://dx.doi.org/10.1001/jamaneurol.2013.4412.

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40

Zhelyazkova, S., S. Nachev, O. Kalev, and I. Tournev. "Sporadic Creutzfeldt–Jakob Disease." Acta Medica Bulgarica 51, s1 (2024): 36–40. http://dx.doi.org/10.2478/amb-2024-0032.

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Abstract Sporadic Creutzfeldt–Jakob disease is a rare and fatal human prion disease characterized by a rapidly progressive dementia, myoclonus, cerebellar, pyramidal, extrapyramidal, visual, and psychiatric symptoms. These findings are all non-specific and making diagnosis is often difficult at the symptoms onset, especially in case of atypical clinical and radiological presentation. This case report describes a woman in her 60s, who presented with rapid cognitive decline, confusion, ataxia and electroencephalographic changes compatible with nonconvulsive status epilepticus. Her symptoms progressively worsened and she died 8 weeks after the onset. The two cerebrospinal fluid analyses were normal and no 14-3-3 protein was detected. The brain MRls revealed areas of cortical restricted diffusion involving the right frontal and parietal lobe. The electroencephalographic findings of continuous periodic generalized bi-triphasic complexes, typical for sporadic Creutzfeldt–Jakob disease, were detected 1 month after the onset. Sporadic Creutzfeldt–Jakob disease was neuropathologically confirmed. Although sporadic Creutzfeldt–Jakob disease is a rare neurodegenerative disease, it should be considered in the differential diagnosis of all cases with unexplained and rapid cognitive decline and confusion, along with ataxia, pyramidal/ extrapyramidal signs, myoclonus and dysphagia.

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41

Shkrobot, S. I., R. B. Nasalyk, Kh V. Duve, and N. T. Shalabai. "Sporadic variant of Creutzfeldt-Jakob disease and modern possibilities of lifetime diagnosis (a case from clinical practice)." Psychiatry Neurology and Medical Psychology, no. 27 (February 28, 2025): 122–32. https://doi.org/10.26565/2312-5675-2025-27-10.

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Background. Creutzfeldt–Jakob Disease is a neurodegenerative disorder characterized by relentless rapid progression and structural damage to the brain, belonging to the group of prion infections. The first description of the disease was made by the German neurologist Alfons Jakob in 1920, based on three patient cases. Independently, in 1921, neurologist Hans Gerhard Creutzfeldt, analyzing a clinical case of a single patient, reported the identification of a new neurological disorder characterized by rapidly progressing psychiatric disturbances, involvement of the extrapyramidal and pyramidal systems, and a fatal outcome. Purpose – of the study was to present a case of sporadic variant of CREUTZFELDT–JAKOB disease and modern possibilities of lifetime diagnosis Materials and methods. A clinical case of a patient with a confirmed diagnosis of Creutzfeldt–Jakob disease during life is presented for consideration, according to the 2017-th diagnostic criteria. Results and discussions. Clinical features of a 66-year-old patient with Creutzfeldt–Jakob disease, typical changes detected by brain MRI, typical EEG characteristics are presented, and the use of modern laboratory biomarkers of the disease, which provide the greatest opportunities for intravital diagnosis of this disease, is considered. Conclusions. We describe a rare case of sporadic variant of CREUTZFELDT–JAKOB disease and modern possibilities of lifetime diagnosis.

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42

Guan, J. J., B. Policeni, G. Bathla, A. A. Capizzano, and T. Moritani. "Heidenhain Variant of Creutzfeldt-Jakob Disease with Concurrent Findings of Posterior Reversible Encephalopathy Syndrome: A Case Report and Literature Review." Neurographics 11, no. 1 (2021): 26–29. http://dx.doi.org/10.3174/ng.2000059.

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Although Creutzfeldt-Jakob disease is not yet proved to be related to the development of posterior reversible encephalopathy syndrome, prion invasion to the endothelium has been shown to change blood-brain barrier permeability. We present a case of Heidenhain variant of Creutzfeldt-Jakob disease that had associated imaging findings of posterior reversible encephalopathy syndrome, with a review of the literature, raising the question of whether Creutzfeldt-Jakob disease instead represents another cause for the development of posterior reversible encephalopathy syndrome.

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43

Rakitin, Aleksei, Riina Vibo, Vaiko Veikat, Anne Õunapuu, Aive Liigant, and Sulev Haldre. "Creutzfeldt-Jakob Disease Presenting as Nonconvulsive Status Epilepticus." Case Reports in Neurological Medicine 2018 (November 4, 2018): 1–4. http://dx.doi.org/10.1155/2018/3092018.

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Creutzfeldt-Jakob disease is a rare, rapidly progressive spongiform encephalopathy in humans. EEG plays an important role in diagnosing this disease. In some patients, epileptic activity and encephalopathy from various aetiologies may share morphological features on EEG. This similarity could create difficulties in EEG interpretation, especially if the patient presents with disturbed consciousness. In this case report, a 74-year-old female with Creutzfeldt-Jakob disease presented initially with rapidly progressive impairment of consciousness and focal epileptiform activity on EEG. An EEG performed 25 days later showed periodic sharp-wave complexes with triphasic morphology at a rate of 0.5 Hz, compatible with a diagnosis of Creutzfeldt-Jakob disease. Based on these results, we recommend that a diagnosis of Creutzfeldt-Jakob disease be considered in patients presenting with a rapid deterioration of consciousness and a clinical presentation of nonconvulsive status epilepticus. Monitoring these patients with serial EEGs could be useful to establish an accurate diagnosis.

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44

Karatas, Hulya, Nese Dericioglu, Oguzhan Kursun, and Serap Saygi. "Creutzfeldt-Jakob Disease Presenting as Hyperparathyroidism and Generalized Tonic Status Epilepticus." Clinical EEG and Neuroscience 38, no. 4 (2007): 203–6. http://dx.doi.org/10.1177/155005940703800404.

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Creutzfeldt-Jakob disease is the most common disorder among the very rare human transmissible subacute spongiform encephalopathies. Sporadic, familial or iatrogenic forms of the disease can be seen. The common presentations of the disease include rapidly progressive cognitive decline, behavioral changes, visual disturbances and cerebellar dysfunction. Hyperparathyroidism and toxicity of lithium and bismuth have been reported to induce similar symptoms and EEG findings, leading to a Creutzfeldt-Jakob like syndrome. We report a very rare case of Creutzfeldt-Jakob disease presenting with hyperparathyroidism and generalized tonic status epilepticus.

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45

Pauranik, Apoorva. "Sporadic Creutzfeldt Jakob disease." Neurology India 64, no. 3 (2016): 380. http://dx.doi.org/10.4103/0028-3886.181563.

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46

Danish, SMA, JD Pearson, and AB Murgatroyd. "Creutzfeldt-Jakob disease (CJD)." Morecambe Bay Medical Journal 2, no. 5 (1995): 106–7. http://dx.doi.org/10.48037/mbmj.v2i5.963.

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47

Henry, Ronnie, and Frederick A. Murphy. "Etymologia: Creutzfeldt-Jakob Disease." Emerging Infectious Diseases 23, no. 6 (2017): 956. http://dx.doi.org/10.3201/eid2306.et2306.

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48

Lanska, Douglas John. "Etymologia: Creutzfeldt-Jakob Disease." Emerging Infectious Diseases 23, no. 10 (2017): 1760–61. http://dx.doi.org/10.3201/eid2310.171142.

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49

Taylor, David M. "Creutzfeldt-Jakob Disease Agent." Infection Control and Hospital Epidemiology 9, no. 12 (1988): 526. http://dx.doi.org/10.2307/30144188.

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50

Nguyen, Steve T., and Leland S. Rickman. "UNDERSTANDING Creutzfeldt-Jakob DISEASE." Journal of Gerontological Nursing 23, no. 11 (1997): 22–27. http://dx.doi.org/10.3928/0098-9134-19971101-10.

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