Relevant bibliographies by topics / Critérios de Simon Broome

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  2. Dissertations / Theses
  3. Book chapters

Academic literature on the topic 'Critérios de Simon Broome'

Author: Grafiati
Published: 10 September 2021
Last updated: 17 July 2025

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Journal articles on the topic "Critérios de Simon Broome"

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Prawitasari, Titis, Sudigdo Sastroasmoro, and Damayanti R. Sjarif. "Skrining Sistematik terhadap Hiperkolesterolemia Familial pada Anak Berdasarkan Kriteria MedPed, Simon Brome Register Register dan Dutch Lipid Clinic." Sari Pediatri 13, no. 2 (2016): 152. http://dx.doi.org/10.14238/sp13.2.2011.152-8.

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Latar belakang. Hiperkolesterolemia familial (HF) merupakan kelainan genetik tersering penyebab terjadinya penyakit jantung koroner/aterosklerosis. Penyakit tersebut seringkali terlambat diketahui, padahal jika dapat diketahui sejak usia muda terjadinya penyakit jantung koroner dan kematian dapat dicegah. Terdapat berbagai macam kriteria untuk dapat mendeteksi dini HF pada orang dewasa, yaitu MedPed, Simon Broome RegisterdanDucth Lipid Clinic.Tujuan. Mendeteksi secara dini HF pada anak dengan riwayat orangtua mengalami penyakit jantung koroner dini dan hiperkolesterolemia berdasarkan kriteria
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Jones, Matthew, Ralph K. Akyea, Katherine Payne, et al. "Cost-Effectiveness of Screening Algorithms for Familial Hypercholesterolaemia in Primary Care." Journal of Personalized Medicine 12, no. 3 (2022): 330. http://dx.doi.org/10.3390/jpm12030330.

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Although familial hypercholesterolemia (FH) screening within primary care is considered cost-effective, which screening approach is cost-effective has not been established. This study determines the cost-effectiveness of six case-finding strategies for screening of electronic health records to identify index patients who have genetically confirmed monogenic FH in English primary care. A decision tree was constructed to represent pathways of care for each approach (FH Case Identification Tool (FAMCAT) versions 1 and 2, cholesterol screening, Dutch Lipid Clinic Network (DLCN), Simon Broome crite
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Akyea, Ralph K., Nadeem Qureshi, Joe Kai, et al. "Evaluating a clinical tool (FAMCAT) for identifying familial hypercholesterolaemia in primary care: a retrospective cohort study." BJGP Open 4, no. 5 (2020): bjgpopen20X101114. http://dx.doi.org/10.3399/bjgpopen20x101114.

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BackgroundFamilial hypercholesterolaemia (FH) is an inherited lipid disorder causing premature heart disease, which is severely underdiagnosed. Improving the identification of people with FH in primary care settings would help to reduce avoidable heart attacks and early deaths.AimTo evaluate the accuracy of the familial hypercholesterolaemia case ascertainment identifcation tool (FAMCAT) for identifying FH in primary care.Design & settingA retrospective cohort study of 1 030 183 patients was undertaken. Data were extracted from the UK Royal College of General Practitioners (RCGP) Research
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Iyen, Barbara, Ralph Akyea, Steve E. Humphries, and Nadeem Qureshi. "CARDIOVASCULAR OUTCOMES IN SEVERE AND NON-SEVERE FH IN SIMON-BROOME REGISTER." Atherosclerosis 399 (December 2024): 118743. https://doi.org/10.1016/j.atherosclerosis.2024.118743.

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5

Pasqua, Melissa-Rosina, Aurelie Pare, David Blank, and Brian Gilfix. "The Use of Genetic Testing Panels for Dyslipidemia: A Quality Improvement Project at the McGill University Health Centre." Journal of the Endocrine Society 5, Supplement_1 (2021): A511. http://dx.doi.org/10.1210/jendso/bvab048.1046.

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Abstract Background: Genetic testing panels are used to identify the most common genetic causes of dyslipidemia, and the results of these panels can guide treatment and management. The objective of this quality improvement project was to assess the appropriateness of genetic testing panels requested by the McGill University Health Centre (MUHC). Methods: Genetic testing panels sent for analysis from January 2018 to December 2019 were identified. Ordering physician specialty, patient personal and family medical history, lipid panel results, and genetic testing results were collected. Then, vali
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Kim, Z. F., A. S. Galyavich, D. I. Sadykova, L. M. Nurieva та E. S. Kim. "Clinical characteristics of carriers of pathogenic LDLR and АРОВ mutations". Russian Journal of Cardiology 29, № 1 (2023): 5686. http://dx.doi.org/10.15829/1560-4071-2024-5686.

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Aim. To analyze the clinical characteristics of carriers of pathogenic LDLR and АРОВ mutations, as well as the prognostic value of Dutch Lipid Clinic Network Score (DLCNS) as applied to carriage of АРОВ or LDLR.Material and methods. The study included 1233 outpatient lipid clinic patients. Biomaterial samples from 421 patients with the hereditary dyslipidemia were studied using Next Generation Sequencing (NGS) to identify carriage of familial hypercholesterolemia (FH)-associated genes (LDLR, АРОВ, PCSK9, LDLRAP1), as well as polymorphism of the APOE gene. Statistical processing was performed u
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Soran, Handrean, Jackie A. Cooper, Paul N. Durrington, et al. "Non-HDL or LDL cholesterol in heterozygous familial hypercholesterolaemia: findings of the Simon Broome Register." Current Opinion in Lipidology 31, no. 4 (2020): 167–75. http://dx.doi.org/10.1097/mol.0000000000000692.

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8

Qureshi, Nadeem, Ralph K. Akyea, Brittany Dutton, et al. "Comparing the performance of the novel FAMCAT algorithms and established case-finding criteria for familial hypercholesterolaemia in primary care." Open Heart 8, no. 2 (2021): e001752. http://dx.doi.org/10.1136/openhrt-2021-001752.

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ObjectiveFamilial hypercholesterolaemia (FH) is a common inherited disorder causing premature coronary heart disease (CHD) and death. We have developed the novel Familial Hypercholesterolaemia Case Ascertainment Tool (FAMCAT 1) case-finding algorithm for application in primary care, to improve detection of FH. The performance of this algorithm was further improved by including personal history of premature CHD (FAMCAT 2 algorithm). This study has evaluated their performance, at 95% specificity, to detect genetically confirmed FH in the general population. We also compared these algorithms to e
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Humphries, S. E., J. A. Cooper, M. Seed, et al. "Coronary heart disease mortality in treated Familial Hypercholesterolaemia: Update of the UK Simon Broome FH Register." Atherosclerosis Supplements 28 (September 2017): e4. http://dx.doi.org/10.1016/j.atherosclerosissup.2017.08.006.

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Humphries, S. E., J. A. Cooper, M. Seed, et al. "Coronary heart disease mortality in treated familial hypercholesterolaemia: Update of the UK Simon Broome FH register." Atherosclerosis 274 (July 2018): 41–46. http://dx.doi.org/10.1016/j.atherosclerosis.2018.04.040.

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Dissertations / Theses on the topic "Critérios de Simon Broome"

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Albuquerque, João David Ferreira de Castro. "Classification methods applied to familial hypercholesterolemia diagnosis in pediatric age." Master's thesis, 2019. http://hdl.handle.net/10451/40378.

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Tese de mestrado, Bioestatística, Universidade de Lisboa, Faculdade de Ciências, 2019<br>Introdução: A Hipercolesterolemia Familiar (FH) é uma doença genética do metabolismo lipídico, caracterizada por níveis elevados de colesterol proveniente das lipoproteínas de baixa densidade (LDLc). A severa dislipidemia resultante leva ao desenvolvimento precoce de aterosclerose, representando um grande factor de risco de doença cardiovascular (CVD). O diagnóstico antecipado da FH encontra-se associado com uma redução significativa do risco de CVD, fundamentando a introdução de medidas terapêuticas mais
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Book chapters on the topic "Critérios de Simon Broome"

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Sadykova, Dinara, Liliia Galimova, Evgeniia Slastnikova, Zulfiia Khabibrakhmanova, and Natalya Guseva. "Arterial Stiffness Assessment in Children with Familial Hypercholesterolemia." In Management of Dyslipidemia. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.96018.

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Familial hypercholesterolemia (FH) is the genetic disease which characterized by an increase of level total cholesterol and low density lipoproteins since childhood. The aim of the study was to assess arterial stiffness in children with heterozygous FH by measuring the pulse wave velocity (PWV) in the aorta. The study involved 118 children, 60 healthy children in the control group and 58 children with heterozygous FH in the main group. Both groups were divided into 3 age subgroups: 5–7 years old, 8–12 years old and 13–17 years old. The diagnosis of FH was made using British criteria by Simon Broome. The lipid profile was determined for all children, blood pressure was monitored daily with an estimate of the minimum, mean and maximum PWV (PWVmin, mean PWV, PWVmax) in aorta using oscillometric method. Correlation analysis in patients with FH revealed direct correlation between PWVmin, mean PWV and PWVmax with total cholesterol (r = 0.46, r = 0.46 and r = 0.464, respectively, p &lt; 0.001). The study demonstrates an increase in the PWV in the aorta in children with FH compared with healthy peers from 8–12 years of age and a progression of arterial stiffness most significant in the group of 13–17 years.
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Journal articles
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