Relevant bibliographies by topics / Crystal-induced inflammation

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Crystal-induced inflammation'

Author: Grafiati
Published: 31 May 2025
Last updated: 31 July 2025

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Journal articles on the topic "Crystal-induced inflammation"

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Halverson, Paul B., and Beth A. Derfus. "Calcium crystal–induced inflammation." Current Opinion in Rheumatology 13, no. 3 (2001): 221–24. http://dx.doi.org/10.1097/00002281-200105000-00013.

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Landis, R. Clive, and Dorian O. Haskard. "Pathogenesis of crystal-induced inflammation." Current Rheumatology Reports 3, no. 1 (2001): 36–41. http://dx.doi.org/10.1007/s11926-001-0049-7.

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Lan, Zhou, Lvyi Chen, Jing Feng, et al. "Mechanosensitive TRPV4 is required for crystal-induced inflammation." Annals of the Rheumatic Diseases 80, no. 12 (2021): 1604–14. http://dx.doi.org/10.1136/annrheumdis-2021-220295.

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Crystal structures activate innate immune cells, especially macrophages and initiate inflammatory responses. We aimed to understand the role of the mechanosensitive TRPV4 channel in crystal-induced inflammation. Real-time RT-PCR, RNAscope in situ hybridisation, and Trpv4eGFP mice were used to examine TRPV4 expression and whole-cell patch-clamp recording and live-cell Ca2+ imaging were used to study TRPV4 function in mouse synovial macrophages and human peripheral blood mononuclear cells (PBMCs). Both genetic deletion and pharmacological inhibition approaches were used to investigate the role o
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Terkeltaub, Robert A. "Gout and mechanisms of crystal-induced inflammation." Current Opinion in Rheumatology 5, no. 4 (1993): 510–16. http://dx.doi.org/10.1097/00002281-199305040-00017.

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Getting, Stephen J., Connie W. Lam, Airu S. Chen, Paolo Grieco, and Mauro Perretti. "Melanocortin 3 receptors control crystal‐induced inflammation." FASEB Journal 20, no. 13 (2006): 2234–41. http://dx.doi.org/10.1096/fj.06-6339com.

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Akahoshi, Tohru, Yousuke Murakami, and Hidero Kitasato. "Recent advances in crystal-induced acute inflammation." Current Opinion in Rheumatology 19, no. 2 (2007): 146–50. http://dx.doi.org/10.1097/bor.0b013e328014529a.

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Pham, N., T. W. Chirayath, F. Castelli, et al. "POS0510 INTERMITTENT FASTING REDUCES CRYSTAL-INDUCED INFLAMMATION." Annals of the Rheumatic Diseases 82, Suppl 1 (2023): 517. http://dx.doi.org/10.1136/annrheumdis-2023-eular.5106.

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BackgroundInflammation induced by monosodium urate (MSU) and calcium pyrophosphate (CPP) crystals depends on interleukin (IL)-1β activated by the NLRP3 inflammasome. The inflammatory response can be modulated by diet, fasting, and caloric restriction.ObjectivesTo determine whether intermittent fasting reduces MSU and CPP crystal-induced inflammation.MethodsCrystal-induced inflammation was assessed using both types of crystalsin vivoin the air pouch model in 8-week-old wild-type male mice fed either with a normal ad libitum diet or intermittent fasting (IF) (every over day fast, 2 days during 1
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Tsay, Gregory, Jia-Hau Yen, Ling Chung Lin, Pui Ying Leong, Jiunn-Horng Chen та Zsuzsa Szondy. "The MTA1-TG2 is involved in self-limitation of monosodium urate crystal-induced inflammation by upregulating the levels of active TGFβ1 leading to inhibition of JAK2 signaling (HUM1P.305)". Journal of Immunology 194, № 1_Supplement (2015): 52.30. http://dx.doi.org/10.4049/jimmunol.194.supp.52.30.

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Abstract Abstract Introduction: Transglutaminase 2 (TG2), a protein crosslinking enzyme with multiple biochemical functions, has been connected to various inflammatory processes. In this study, the involvement of TG2 in monosodium urate (MSU) crystal-induced inflammation was studied. Conclusions: These findings reveal an inherent regulatory role of the MTA1-TG2 pathway in the self-limitation of MSU crystal-induced inflammation via positively regulating the levels of active TGF-b in macrophages that opposes the MSU crystal- induced JAK2 -dependent pro-inflammatory cytokine formation.
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Rasool, Mahaboobkhan, Sonal Chandal, and Evan Prince Sabina. "Inhibition of Monosodium Urate Crystal-Induced Inflammation by Withaferin A." Journal of Pharmacy & Pharmaceutical Sciences 11, no. 4 (2009): 46. http://dx.doi.org/10.18433/j35k58.

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ABSTRACT 
 Purpose. Gouty arthritis is a characteristically intense acute inflammatory reaction resulting from the formation of sodium urate crystals in the joint cavity. In the present study, the effect of withaferin A, a steroidal lactone was investigated on monosodium urate crystal-induced inflammation in mice; an experimental model for gouty arthritis and compared it with that of the non-steroidal anti-inflammatory drug, indomethacin. 
 Methods. Paw volume and levels/activities of lysosomal enzymes, lipid peroxidation, anti-oxidant status and inflammatory mediator TNF-α were dete
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Galozzi, Paola, Lucia Maschio, Samuela Carraro, Anna Scanu, Monica Facco, and Francesca Oliviero. "M2 macrophages as resolvers of crystal-induced inflammation." Rheumatology 60, no. 5 (2021): 2480–83. http://dx.doi.org/10.1093/rheumatology/keab122.

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Dissertations / Theses on the topic "Crystal-induced inflammation"

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Pham, Chinh Nghia. "Le métabolisme de l'inflammation dans les maladies microcristallines." Electronic Thesis or Diss., Université Paris Cité, 2024. http://www.theses.fr/2024UNIP5246.

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La goutte, due aux dépôts de cristaux d'urate monosodique (UMS), et la maladie liée aux dépôts des cristaux de pyrophosphate de calcium (PPC) sont les deux rhumatismes inflammatoires les plus fréquents de l'adulte. Les cristaux sont responsables de crises inflammatoires aiguës et récurrentes qui dépendent de la production de l'interleukine (IL)-1 beta via l'activation de l'inflammasome NLRP3. Ces crises inflammatoires peuvent être modulées par des facteurs environnementaux tels que les régimes alimentaires dont la restriction calorique (RC) ou le jeûne intermittent (JI). Les objectifs de ma th
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HUANG, CHUN-JUNG, та 黃桾蓉. "Cardamonin Attenuates IL-1β and Urate Crystal-Induced Joint Inflammation". Thesis, 2018. http://ndltd.ncl.edu.tw/handle/ev6gv5.

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碩士<br>國防醫學院<br>病理及寄生蟲學研究所<br>106<br>Cardamonin, a chalcone isolated mostly from Zingiberaceae plants has been reported to exert anti-inflammatory and anti-oxidant properties. Osteoarthritis (OA) is a common degenerative joint disorder with progressive cartilage destruction and joint dysfunction. Interleukin (IL)-1β is one of the potent cytokine identified in osteoarthritic synovial fluid and triggers a succession of cartilage catabolic effects. Gouty arthritis (GA) arises an acute inflammatory response results from the precipitation of monosodium urate (MSU) crystals in the joint. The uptake o
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TSAI, MING-HSIU, та 蔡明修. "Amentoflavone Attenuates IL-1β and Urate Crystal-Induced Joint Inflammation". Thesis, 2019. http://ndltd.ncl.edu.tw/handle/hwwu72.

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Books on the topic "Crystal-induced inflammation"

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Platt, Philip. Mechanisms of inflammation in the crystal induced diseases. University of Birmingham, 1987.

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Abhishek, Abhishek, and Michael Doherty. Pathophysiology of calcium pyrophosphate deposition. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0049.

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Calcium pyrophosphate (CPP) dihydrate crystals form extracellularly. Their formation requires sufficient extracellular inorganic pyrophosphate (ePPi), calcium, and pro-nucleating factors. As inorganic pyrophosphate (PPi) cannot cross cell membranes passively due to its large size, ePPi results either from hydrolysis of extracellular ATP by the enzyme ectonucleotide pyrophosphatase/phosphodiesterase 1 (also known as plasma cell membrane glycoprotein 1) or from the transcellular transport of PPi by ANKH. ePPi is hydrolyzed to phosphate (Pi) by tissue non-specific alkaline phosphatase. The level
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Book chapters on the topic "Crystal-induced inflammation"

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Terkeltaub, R. "Pathogenesis of Monosodium Urate Crystal-Induced Inflammation." In Urate Deposition in Man and its Clinical Consequences. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-84491-1_10.

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Liu-Bryan, Ru, and Robert Terkeltaub. "Tophus Biology and Pathogenesis of Monosodium Urate Crystal–Induced Inflammation." In Gout & Other Crystal Arthropathies. Elsevier, 2012. http://dx.doi.org/10.1016/b978-1-4377-2864-4.10005-3.

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Park, So-Young, Tae-Jin Ju, Kyu-Chang Won, Hyo-Jeong Kim, and Eun-Gyoung Hong. "Inhibition of Inducible Nitric Oxide Synthase Suppresses Monosodium Urate Crystal-Induced Gouty Inflammation in Mice." In BASIC/TRANSLATIONAL - Growth Factors, Cytokines & Intracellular Signaling. The Endocrine Society, 2011. http://dx.doi.org/10.1210/endo-meetings.2011.part2.p24.p2-78.

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Conference papers on the topic "Crystal-induced inflammation"

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Scanu, A., R. Luisetto, B. Raffeiner, et al. "AB0052 Treatment with bactericidal/permeability-increasing protein reduces crystal-induced inflammation and collagen-induced arthritis in mice." In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.2674.

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Zamudio-Cuevas, Y., F. Oliviero, E. Belluzzi, et al. "THU0047 Resveratrol and its precursor polydatin inhibit crystal-induced inflammation in vitro by decreasing oxidative stress and il-1beta activation pathway." In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.6830.

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You might also be interested in the extended bibliographies on the topic 'Crystal-induced inflammation' for particular source types:
Journal articles
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