Relevant bibliographies by topics / CSK

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'CSK'

Author: Grafiati
Published: 4 June 2021
Last updated: 9 June 2025

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Journal articles on the topic "CSK"

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Nagy, Zoltan, Jun Mori, Vanesa-Sindi Ivanova, Alexandra Mazharian, and Yotis A. Senis. "Interplay between the tyrosine kinases Chk and Csk and phosphatase PTPRJ is critical for regulating platelets in mice." Blood 135, no. 18 (April 30, 2020): 1574–87. http://dx.doi.org/10.1182/blood.2019002848.

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Abstract The Src family kinases (SFKs) Src, Lyn, and Fyn are essential for platelet activation and also involved in megakaryocyte (MK) development and platelet production. Platelet SFKs are inhibited by C-terminal Src kinase (Csk), which phosphorylates a conserved tyrosine in their C-terminal tail, and are activated by the receptor-type tyrosine phosphatase PTPRJ (CD148, DEP-1), which dephosphorylates the same residue. Deletion of Csk and PTPRJ in the MK lineage in mice results in increased SFK activity, but paradoxically hypoactive platelets resulting from negative feedback mechanisms, including upregulation of Csk homologous kinase (Chk) expression. Here, we investigate the role of Chk in platelets, functional redundancy with Csk, and the physiological consequences of ablating Chk, Csk, and PTPRJ in mice. Platelet count was normal in Chk knockout (KO) mice, reduced by 92% in Chk;Csk double KO (DKO) mice, and partially rescued in Chk;Csk;Ptprj triple KO (TKO) mice. Megakaryocyte numbers were significantly increased in both DKO and TKO mice. Phosphorylation of the inhibitory tyrosine of SFKs was almost completely abolished in DKO platelets, which was partially rescued in Src and Fyn in TKO platelets. This residual phosphorylation was abolished by Src inhibitors, revealing an unexpected mechanism in which SFKs autoinhibit their activity by phosphorylating their C-terminal tyrosine residues. We demonstrate that reduced inhibitory phosphorylation of SFKs leads to thrombocytopenia, with Csk being the dominant inhibitor in platelets and Chk having an auxiliary role. PTPRJ deletion in addition to Chk and Csk ameliorates the extent of thrombocytopenia, suggesting targeting it may have therapeutic benefits in such conditions.
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Sabe, H., M. Okada, H. Nakagawa, and H. Hanafusa. "Activation of c-Src in cells bearing v-Crk and its suppression by Csk." Molecular and Cellular Biology 12, no. 10 (October 1992): 4706–13. http://dx.doi.org/10.1128/mcb.12.10.4706-4713.1992.

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The protein product of the CT10 virus, p47gag-crk (v-Crk), which contains Src homology region 2 (SH2) and 3 (SH3) domains but lacks a kinase domain, is believed to cause an increase in cellular protein tyrosine phosphorylation. A candidate tyrosine kinase, Csk (C-terminal Src kinase), has been implicated in c-Src Tyr-527 phosphorylation, which negatively regulates the protein tyrosine kinase of pp60c-src (c-Src). To investigate how c-Src kinase activity is regulated in vivo, we first looked at whether v-Crk can activate c-Src kinase. We found that cooverexpression of v-Crk and c-Src caused elevation of c-Src kinase activity, resulting in an increase of tyrosine phosphorylation of cellular proteins and morphological transformation of rat 3Y1 fibroblasts. v-Crk and c-Src complexes were not detected, although v-Crk bound to a variety of tyrosine-phosphorylated proteins in cells overexpressing v-Crk and c-Src. Overexpression of Csk in these transformed cells caused reversion to normal phenotypes and also reduced the level of c-Src kinase activity. However, Csk did not cause reversion of cells transformed by v-Src or c-Src527F, in which Tyr-527 was changed to Phe. These results strongly suggest that Csk acts on Tyr-527 of c-Src and suppresses c-Src kinase activity in vivo. Because Csk can suppress transformation by cooverexpression of v-Crk and c-Src, we suggest that v-Crk causes activation of c-Src in vivo by altering the phosphorylation state of Tyr-527.
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Sabe, H., M. Okada, H. Nakagawa, and H. Hanafusa. "Activation of c-Src in cells bearing v-Crk and its suppression by Csk." Molecular and Cellular Biology 12, no. 10 (October 1992): 4706–13. http://dx.doi.org/10.1128/mcb.12.10.4706.

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The protein product of the CT10 virus, p47gag-crk (v-Crk), which contains Src homology region 2 (SH2) and 3 (SH3) domains but lacks a kinase domain, is believed to cause an increase in cellular protein tyrosine phosphorylation. A candidate tyrosine kinase, Csk (C-terminal Src kinase), has been implicated in c-Src Tyr-527 phosphorylation, which negatively regulates the protein tyrosine kinase of pp60c-src (c-Src). To investigate how c-Src kinase activity is regulated in vivo, we first looked at whether v-Crk can activate c-Src kinase. We found that cooverexpression of v-Crk and c-Src caused elevation of c-Src kinase activity, resulting in an increase of tyrosine phosphorylation of cellular proteins and morphological transformation of rat 3Y1 fibroblasts. v-Crk and c-Src complexes were not detected, although v-Crk bound to a variety of tyrosine-phosphorylated proteins in cells overexpressing v-Crk and c-Src. Overexpression of Csk in these transformed cells caused reversion to normal phenotypes and also reduced the level of c-Src kinase activity. However, Csk did not cause reversion of cells transformed by v-Src or c-Src527F, in which Tyr-527 was changed to Phe. These results strongly suggest that Csk acts on Tyr-527 of c-Src and suppresses c-Src kinase activity in vivo. Because Csk can suppress transformation by cooverexpression of v-Crk and c-Src, we suggest that v-Crk causes activation of c-Src in vivo by altering the phosphorylation state of Tyr-527.
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Karyati, Nur Etika, Latief Mahir Rachman, and Dwi Putro Tejo Baskoro. "Penetapan Alokasi Sawah Penerima Air Irigasi Berdasarkan Kondisi Hidroklimatologi di Daerah Irigasi Cihea." Jurnal Ilmu Pertanian Indonesia 27, no. 2 (April 13, 2022): 216–25. http://dx.doi.org/10.18343/jipi.27.2.216.

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One of the main supporting factors for the agricultural sector in the Cihea Irrigation Area is the availability of sufficient water resources. However, uncertain climatic conditions due to climate change lead the changes in the rain distribution and affect the availability of water resources. The study aims to (1) assess the land water balance and drought index in Cihea Irrigation Area and (2) develop alternatives related to irrigation priority locations based on hydro climatological conditions. The water balance was analyzed using the Thornthwaite-Mather method (1957), while the irrigation priority locations were determined using the composite performance index method. Based on the annual water balance analysis, this irrigation area had a 568 mm/year water surplus. However, there was a water shortage from July to October, at 132 mm per month. From ten land systems,the highest deficit was in SL_9, while the lowest was in SL_1. These conditions triggered drought in some locations. The highest drought index occurred in August (56,89%), while the lowest occurred in July (3.98%). Based on hidroclimatological ,the prioritised locations for escalated productivity are paddy fields in CDK.16 Ka, CDK.17 Ki, PD.1 Ka, PD.1 Ki, PD.2 Ka, PD.2 Ki, CRJ.6 Ki, CRJ.7 Ki, CRJ.8 Ki, CRJ.9 Ki, CRJ.10 Ki, and CKR.1 Ki in the Ciranjang subdistrict. Meanwhile, to gain the equal irrigation distribution, the priority locations consist of paddy fields in CSK.1 Ki to CSK.5 ki, CSK.6 Ki, CSK.6 Ka, CSK.7 Ki to CSK.9 Ki, CSK.10 Ki, and CSK.10 Ka, situated in Bocongpicung District.
 
 Keywords: drought index, irrigation priority, water balance
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Li, Leiming, Masaya Okura, and Akira Imamoto. "Focal Adhesions Require Catalytic Activity of Src Family Kinases To Mediate Integrin-Matrix Adhesion." Molecular and Cellular Biology 22, no. 4 (February 15, 2002): 1203–17. http://dx.doi.org/10.1128/mcb.22.4.1203-1217.2002.

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ABSTRACT Members of the Src family of tyrosine kinases function to phosphorylate focal adhesion (FA) proteins. To explore the overlapping functions of Src kinases, we have targeted Csk, a negative regulator of the Src family, to FA structures. Expression of FA-targeted Csk (FA-Csk) effectively reduced the active form (nonphosphorylated at the C-terminal regulatory tyrosine) of Src members in the cell. We found that fibroblasts expressing FA-Csk lost integrin-mediated adhesion. Activated Src (SrcY529F) as well as activation of putative Src signaling mediators (Fak, Cas, Crk/CrkL, C3G, and Rap1) blocked the effect of FA-Csk in a manner dependent on Rap1. SrcY529F also inhibited activated Ras-induced cell detachment but failed to rescue detachment caused by an activated mutant of Raf1 (Raf-BXB) that Rap1 cannot inhibit. Although normal spreading onto fibronectin was restored by the β1 integrin affinity-activating antibody TS2/16 in cells expressing FA-Csk or Raf-BXB, FAs were lost in these cells. On the other hand, Rap1 activation could restore FAs in cells expressing FA-Csk. Activation of the executioner caspase, caspase 3, is essential for many forms of apoptosis. While a caspase 3 inhibitor (Z-DEVD-FMK) inhibited cell detachment triggered by activation of caspase 8, this inhibitor had no effect on cell detachment caused by FA-Csk. Likewise, overexpression of an activated Akt made cells resistant to the effect of caspase 8 activation, but not to the effect of FA-Csk. It is therefore likely that the primary cause of cell rounding and detachment induced by FA-Csk involves dysfunction of FAs rather than caspase-mediated apoptosis that may result from possible loss of survival signals mediated by Src family kinases. We suggest that endogenous Src family kinases are essential for FAs through activation of Rap1 in fibroblasts.
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Yamaguchi, N., Y. Nakayama, T. Urakami, S. Suzuki, T. Nakamura, T. Suda, and N. Oku. "Overexpression of the Csk homologous kinase (Chk tyrosine kinase) induces multinucleation: a possible role for chromosome-associated Chk in chromosome dynamics." Journal of Cell Science 114, no. 9 (May 1, 2001): 1631–41. http://dx.doi.org/10.1242/jcs.114.9.1631.

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The Csk family of non-receptor-type tyrosine kinases consists of Csk and the Csk homologous kinase Chk. Each enzyme suppresses the catalytic activity of Src family kinases by phosphorylating their C-terminal negative regulatory tyrosine residues. Ectopic and transient expression of Chk in COS-1 cells showed nuclear localization of Chk and growth inhibition. To further explore the role of Chk in cell growth, we overexpressed Chk in human immature myeloid KMT-2 cells. Chk overexpression brought about growth retardation and aberrant chromosome movement leading to multinucleation, and these events were accompanied by insufficient formation of mitotic spindles. In vitro kinase assays showed that Chk overexpression suppressed the tyrosine kinase activity of Lyn, a member of the Src family, immunoprecipitated from Triton X-100 lysates. Subcellular fractionation studies revealed that fractions of Chk and Lyn, resistant to Triton X-100 solubilization, are associated with mitotic chromosome scaffolds and spindles. Chk overexpression induced a decrease in autophosphorylation of Lyn and concomitant changes in levels of tyrosine phosphorylation of proteins associated with both fractions. These results indicate that Chk, Lyn and the tyrosine-phosphorylated proteins localize to mitotic chromosomes and spindles, suggesting that Chk-dependent tyrosine phosphorylation, presumably through Lyn, may be involved in chromosome dynamics.
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Howell, B. W., and J. A. Cooper. "Csk suppression of Src involves movement of Csk to sites of Src activity." Molecular and Cellular Biology 14, no. 8 (August 1994): 5402–11. http://dx.doi.org/10.1128/mcb.14.8.5402-5411.1994.

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Csk phosphorylates Src family members at a key regulatory tyrosine in the C-terminal tail and suppresses their activities. It is not known whether Csk activity is regulated. To examine the features of Csk required for Src suppression, we expressed Csk mutants in a cell line with a disrupted csk gene. Expression of wild-type Csk suppressed Src, but Csk with mutations in the SH2, SH3, and catalytic domains did not suppress Src. An SH3 deletion mutant of Csk was fully active against in vitro substrates, but two SH2 domain mutants were essentially inactive. Whereas Src repressed by Csk was predominantly perinuclear, the activated Src in cells lacking Csk was localized to structures resembling podosomes. Activated mutant Src was also in podosomes, even in the presence of Csk. When Src was not active, Csk was diffusely located in the cytosol, but when Src was active, Csk colocalized with activated Src to podosomes. Csk also localizes to podosomes of cells transformed by an activated Src that lacks the major tyrosine autophosphorylation site, suggesting that the relocalization of Csk is not a consequence of the binding of the Csk SH2 domain to phosphorylated Src. A catalytically inactive Csk mutant also localized with Src to podosomes, but SH3 and SH2 domain mutants did not, suggesting that the SH3 and SH2 domains are both necessary to target Csk to places where Src is active. The failure of the catalytically active SH3 mutant of Csk to regulate Src may be due to its inability to colocalize with active Src.
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Howell, B. W., and J. A. Cooper. "Csk suppression of Src involves movement of Csk to sites of Src activity." Molecular and Cellular Biology 14, no. 8 (August 1994): 5402–11. http://dx.doi.org/10.1128/mcb.14.8.5402.

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Csk phosphorylates Src family members at a key regulatory tyrosine in the C-terminal tail and suppresses their activities. It is not known whether Csk activity is regulated. To examine the features of Csk required for Src suppression, we expressed Csk mutants in a cell line with a disrupted csk gene. Expression of wild-type Csk suppressed Src, but Csk with mutations in the SH2, SH3, and catalytic domains did not suppress Src. An SH3 deletion mutant of Csk was fully active against in vitro substrates, but two SH2 domain mutants were essentially inactive. Whereas Src repressed by Csk was predominantly perinuclear, the activated Src in cells lacking Csk was localized to structures resembling podosomes. Activated mutant Src was also in podosomes, even in the presence of Csk. When Src was not active, Csk was diffusely located in the cytosol, but when Src was active, Csk colocalized with activated Src to podosomes. Csk also localizes to podosomes of cells transformed by an activated Src that lacks the major tyrosine autophosphorylation site, suggesting that the relocalization of Csk is not a consequence of the binding of the Csk SH2 domain to phosphorylated Src. A catalytically inactive Csk mutant also localized with Src to podosomes, but SH3 and SH2 domain mutants did not, suggesting that the SH3 and SH2 domains are both necessary to target Csk to places where Src is active. The failure of the catalytically active SH3 mutant of Csk to regulate Src may be due to its inability to colocalize with active Src.
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Chong, Yuh-Ping, Terrence D. Mulhern, and Heung-Chin Cheng. "C-terminal Src kinase (CSK) and CSK-homologous kinase (CHK)—endogenous negative regulators of Src-family protein kinases." Growth Factors 23, no. 3 (January 2005): 233–44. http://dx.doi.org/10.1080/08977190500178877.

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Zagozdzon, Radoslaw, Rafal Kaminski, Yigong Fu, Wei Fu, Cecile Bougeret, and Hava Karsenty Avraham. "Csk homologous kinase (CHK), unlike Csk, enhances MAPK activation via Ras-mediated signaling in a Src-independent manner." Cellular Signalling 18, no. 6 (June 2006): 871–81. http://dx.doi.org/10.1016/j.cellsig.2005.07.016.

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Dissertations / Theses on the topic "CSK"

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Thomas, R. M. "Csk is an important negative regulator of phagocyte responsiveness in vivo : characterisation of myeloid cell-specific Csk deficiency in mice by conditional mutagenesis (Cre/loxP)." Thesis, University College London (University of London), 2007. http://discovery.ucl.ac.uk/1445173/.

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Whilst the recruitment of phagocytic leukocytes is fundamental to the innate response against pathogenic infection, the inappropriate mobilisation of their cytotoxic potential can also lead to fatal tissue injury. To determine the contribution of Csk, a negative regulator of Src family kinases, to the regulation of phagocyte recruitment and activation in vivo, mice lacking Csk in the myeloid lineage were generated using conditional mutagenesis (cre/loxP). This Csk deficiency resulted in acute multifocal inflammation in skin and lung, accompanied by extramedullary haematopoiesis in the spleen and liver, and increased myelopoiesis in bone marrow. Animals were protected from the disease in a microbiologically controlled environment, but remained hypersensitive to LPS-induced shock. Csk-deficient granulocytes showed enhanced spontaneous and ligand-induced degranulation accompanied by hyperinduction of integrins. Hyperresponsiveness was associated with hyperadhesion and impaired migratory responses in vitro. Biochemical studies revealed spontaneous accumulation of tyrosine-phosphorylated proteins, including hyperphosphorylation of key signalling proteins including Syk and paxillin. These data support a breakdown of the activation threshold set by Csk. Thus, Csk is critical in preventing premature granulocyte recruitment through enforcing the requirement for ligand engagement while supporting the migratory capacity of activated cells through negative regulation of cell adhesion. To address the incomplete Cre mediated deletion of floxed genes in vivo, a genetic approach to elevate Cre recombinase gene expression was developed. Whilst manipulation of regulatory elements including promoter, enhancer, and untranslated regions has yielded enhanced and sustained expression in vitro, this has been difficult to achieve in vivo. Here, it is reported that construction of artificial exons through insertion of short heterologous intron sequences into the open reading frames of the Cre recombinase and enhanced green fluorescent protein results in functional expression accompanied by a 30-fold increase in transcription levels in vitro. Furthermore, green fluorescence levels were enhanced five-fold in cell lines and enhanced considerably in the rat brain after transduction with a herpes simplex virus-based vector. These data define a method of improving both the level and duration of recombinant gene expression, in addition to and independently of surrounding regulatory elements. Significantly, the method should help to increase Cre recombinase expression from weak or transiently expressed promoters thus overcoming an important limitation of Cre/loxP technology incomplete deletion. Furthermore, this method may also be applicable in gene therapy to obtain sustained and effective expression of recombinant proteins in vivo.
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Correia, Lúcio José Herculano. "CSK: uma abordagem para estruturação de kernel de tempo real em componentes." Universidade Federal de São Carlos, 2004. https://repositorio.ufscar.br/handle/ufscar/520.

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Made available in DSpace on 2016-06-02T19:06:02Z (GMT). No. of bitstreams: 1 DissLJHC.pdf: 911507 bytes, checksum: 0af483c56c4aeb6304f9100fe71cccb9 (MD5) Previous issue date: 2004-05-27<br>Financiadora de Estudos e Projetos<br>Generally, operating systems offer a set of services through primitives that are used on demand by applications. Similarly, software components implement and provide services through well-defined interfaces, which are reused in different software projects of a problem domain. Based upon this similarity, it is researched the reuse of software components in real time operating systems projects. It is presented CSK (Computer Structured Kernel) approach for structuring a real time kernel with software components. The approach is divided in two phases. In the first one software components are built from the legacy code and documentation of a preexisting kernel. In the second one, that kernel is rebuilt by reusing the produced components. A case study applies CSK to real time kernel Virtuoso.<br>Sistemas operacionais em geral oferecem um conjunto de serviços através de primitivas, as quais são utilizadas sob demanda pelas aplicações. Similarmente, componentes de software implementam e disponibilizam serviços através de interfaces bem definidas, sendo reutilizados em diferentes projetos de software de um domínio de problema. Baseado nesta similaridade, é pesquisado o reuso de componentes de software na área de tempo-real, nos projetos de sistemas operacionais. É apresentada uma abordagem para a estruturação de kernels de tempo-real utilizando componentes de software, denominada CSK (Component Structured Kernel). Esta abordagem é dividida em duas grandes fases. Na primeira fase, constroem-se os componentes a partir do código legado e da documentação de um kernel já existente. Na segunda fase é feita a reestruturação do kernel, através do reúso de componentes. Um estudo de caso aplica a abordagem CSK ao kernel Virtuoso.
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Ayrapetov, Marina K. "Structural and functional studies of the Csk and Src family protein tyrosine kinases /." View online ; access limited to URI, 2006. http://0-wwwlib.umi.com.helin.uri.edu/dissertations/dlnow/3225312.

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Lee, Sungsoo. "Functional and structural study of the protein tyrosine kinase CSK, as a model system /." View online ; access limited to URI, 2005. http://0-wwwlib.umi.com.helin.uri.edu/dissertations/dlnow/3188063.

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Lau, Yuu Seng, and lauje@rocketmail com. "Techniques in Secure Chaos Communication." RMIT University. Electrical and Computer Engineering, 2006. http://adt.lib.rmit.edu.au/adt/public/adt-VIT20070116.151025.

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In today's climate of increased criminal attacks on the privacy of personal or confidential data over digital communication systems, a more secure physical communication link is required. Chaotic signals which have bifurcation behavior (depending on some initial condition) can readily be exploited to enhance the security of communication systems. A chaotic generator produces disordered sequences that provide very good auto- and cross- correlation properties similar to those of random white noise. This would be an important feature in multiple access environments. These sequences are used to scramble data in spread spectrum systems as they can produce low co-channel interference, hence improve the system capacity and performance. The chaotic signal can be created from only a single mathematical relationship and is neither restricted in length nor is repetitive/ cyclic. On the other hand, with the progress in digital signal processing and digital hardware, there has been an increased interest in using adaptive algorithms to improve the performance of digital systems. Adaptive algorithms provide the system with the ability to self-adjust its coefficients according to the signal condition, and can be used with linear or non-linear systems; hence, they might find application in chaos communication. There has been a lot of literature that proposed the use of LMS adaptive algorithm in the communication arena for a variety of applications such as (but not limited to): channel estimation, channel equalization, demodulation, de-noising, and beamforming. In this thesis, we conducted a study on the application of chaos theory in communication systems as well as the application of adaptive algorithms in chaos communication. The First Part of the thesis tackled the application of chaos theory in com- munication. We examined different types of communication techniques utilizing chaos theory. In particular, we considered chaos shift keying (CSK) and mod- ified kind of logistic map. Then, we applied space-time processing and eigen- beamforming technique to enhance the performance of chaos communication. Following on, we conducted a study on CSK and Chaos-CDMA in conjunction with multi-carrier modulation (MCM) techniques such as OFDM (FFT/ IFFT) and wavelet-OFDM. In the Second Part of the thesis, we tried to apply adaptivity to chaos com- munication. Initially, we presented a study of multi-user detection utilizing an adaptive algorithm in a chaotic CDMA multi-user environment, followed by a study of adaptive beamforming and modified weight-vector adaptive beam- forming over CSK communication. At last, a study of modified time-varying adaptive filtering is presented and a conventional adaptive filtering technique is applied in chaotic signal environment. Twelve papers have been published during the PhD candidature, include two journal papers and ten refereed conference papers.
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Seet, Siong Leng Henry. "Analysis of noncoherent orthogonal modulation for mobile computing." Thesis, Monterey, California. Naval Postgraduate School, 2010. http://hdl.handle.net/10945/55206.

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Approved for public release; distribution is unlimited.<br>Wireless communication is employed to connect mobile computers in a networked environment for information exchange. In a tactical space, sensors and computers typically need to operate on-the-move while transmitting data over both short and long distances in different terrain and conditions. The wireless communication is thus susceptible to effects of Doppler shift and channel fading. In addition, when security and anti jamming features are required, such as frequency-hopping techniques, then coherent signal detection is difficult and noncoherent modulation is used instead. Our study will focus on the bit error rate (BER) performance analysis of noncoherent orthogonal modulation, specifically M-ary frequency-shift keying (MFSK) and code-shift keying (CSK) modulation, in both additive white Gaussian noise (AWGN) and for a Rayleigh fading channel with Doppler shift. The potential applications include communications between mobile computer-sensor devices, such as a mobile ground control station maintaining a datalink with UAV.<br>Civilian
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Baumeister, Ulf. "Identifizierung der zytosolischen Kinase CSK als Bindungspartner von VE-Cadherin Auswirkung dieser Assoziation auf das Zellwachstum /." [S.l. : s.n.], 2004. http://deposit.ddb.de/cgi-bin/dokserv?idn=97146720X.

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Gregorieff, Alexander. "Determinants of the Csk-PEP complex and translational regulation of suppressor of cytokine signalling (SOCS)-1." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape7/PQDD_0017/MQ55063.pdf.

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Lieser, Scot A. "Mechanistic characterization of Csk the role of structural transitions and inter-domain crosstalk in enzyme function /." Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2007. http://wwwlib.umi.com/cr/ucsd/fullcit?p3277705.

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Thesis (Ph. D.)--University of California, San Diego, 2007.<br>Title from first page of PDF file (viewed October 10, 2007). Available via ProQuest Digital Dissertations. Vita. Includes bibliographical references (p. 143-153).
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Lin, Xiaofeng. "Probing the regulatory mechanisms of protein tyrosine kinases, using C-terminal SRC kinase (CSK) as a model system /." View online ; access limited to URI, 2005. http://0-wwwlib.umi.com.helin.uri.edu/dissertations/dlnow/3188064.

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Books on the topic "CSK"

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name, No. The CDK-activating kinase (CAK). Georgetown, TX: Landes Bioscience, 2003.

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Tournier, Laurent. GREP et InDesign CS3/CS4. Paris: Dunod, 2009.

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Philipp, Kaldis, ed. The CDK-activating kinase (CAK). Georgetown, Tex., U.S.A: Landes Bioscience/Eurekah.com, 2002.

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Wang, Qing. Adobe InDesign zhong wen ban cong ru men dao jing tong: Shi he CS3,CS4,CS5 ban ben. Beijing: Qing hua da xue chu ban she, 2011.

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Kompare, Derek. CSI. Oxford, UK: Wiley-Blackwell, 2010. http://dx.doi.org/10.1002/9781444328028.

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Muraḷi, Mērlapāka. Cek. Vijayavāḍa: Navajyōti Pablikēṣans, 1991.

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Bishop, Sherry. The Web collection revealed: Adobe Flash CS4, Dreamweaver CS4, & Photoshop CS4. Clifton Park, NY: Delmar Cengage Learning, 2010.

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Bishop, Sherry. The Web collection revealed: Adobe Flash CS4, Dreamweaver CS4, & Photoshop CS4. Clifton Park, NY: Delmar Cengage Learning, 2010.

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E, Shuman James, and Reding Elizabeth Eisner, eds. The Web collection revealed: Adobe Flash CS4, Dreamweaver CS4, & Photoshop CS4. Clifton Park, NY: Delmar Cengage Learning, 2010.

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Eisner, Reding Elizabeth, ed. The design collection revealed: Adobe InDesign CS4, Photoshop CS4 & Illustrator CS4. Clifton Park, NY: Delmar, 2010.

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Book chapters on the topic "CSK"

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Okada, Masato. "Csk." In Encyclopedia of Signaling Molecules, 1210–14. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_264.

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van Roy, Frans, Volker Nimmrich, Anton Bespalov, Achim Möller, Hiromitsu Hara, Jacob P. Turowec, Nicole A. St. Denis, et al. "Csk." In Encyclopedia of Signaling Molecules, 458–63. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_264.

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Cheng, Heung-Chin, Gahana Advani, Mohammed Iqbal Hossain, Nadia L. Y. Ng, Ya Chee Lim, Anderly C. Chüeh, Mohd Aizuddin Kamaruddin, and Yuh-Ping Chong. "CSK-Homologous Kinase." In Encyclopedia of Signaling Molecules, 1215–30. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_185.

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Cheng, Heung-Chin, Gahana Advani, Mohammed Iqbal Hossain, Nadia LY Ng, Ya Chee Lim, Anderly C. Chüeh, Mohd Aizuddin Kamaruddin, and Yuh-Ping Chong. "CSK-Homologous Kinase." In Encyclopedia of Signaling Molecules, 1–17. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4614-6438-9_185-1.

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van Roy, Frans, Volker Nimmrich, Anton Bespalov, Achim Möller, Hiromitsu Hara, Jacob P. Turowec, Nicole A. St. Denis, et al. "CSK-Homologous Kinase." In Encyclopedia of Signaling Molecules, 463–72. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_185.

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Shang, Ying. "Performance of Chirp CSK for Integrated Wideband Communication." In Advances in Intelligent Systems and Computing, 666–73. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-53980-1_98.

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Chen, Siyuan, Xiaohui Ba, Baigen Cai, Wei Jiang, Jian Wang, and Xu Li. "Spreading Code Authentication Technique Based on CSK Modulation." In Lecture Notes in Electrical Engineering, 322–33. Singapore: Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-99-6932-6_26.

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Chen, Yaohui, Dun Wang, Siyuan Chen, Wencong Ma, Dongjun Li, and Qijia Dong. "Research on Receiving Method of Code Shift Keying (CSK) Signal." In China Satellite Navigation Conference (CSNC) 2020 Proceedings: Volume III, 298–309. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-3715-8_28.

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Yan, Tao, Ying Wang, Tian Li, Ye Tian, Lang Bian, and Yansong Meng. "Low Complexity Acquisition and Tracking Methods for CSK Modulated Signals." In Lecture Notes in Electrical Engineering, 193–202. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-3146-7_19.

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Barreto, Sara, and Damien Lacroix. "Quantification of CSK Mechanics and Deformation in Relation to Cellular Functioning." In Frontiers of Biomechanics, 181–93. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-8075-3_10.

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Conference papers on the topic "CSK"

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Rodrigues, Igor S. C., Leandro R. Ximenes, and Rangel Arthur. "Color-Selective Interference Equalization in Color-Shift Keying (CSK) Systems." In 2024 19th International Symposium on Wireless Communication Systems (ISWCS), 1–6. IEEE, 2024. http://dx.doi.org/10.1109/iswcs61526.2024.10639056.

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Wu, Songwen, Feng Zhou, Yinheng Lu, and Haoxuan Yue. "Self-Interference Suppression of Full-Duplex Spread Spectrum Underwater Acoustic Communication Based on CSK Modulation." In 2024 OES China Ocean Acoustics (COA), 1–4. IEEE, 2024. http://dx.doi.org/10.1109/coa58979.2024.10723370.

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Liping Du, Xiangyi Hu, Ying Li, and Guifen Zhao. "A CSK based SSL handshake protocol." In 2009 IEEE International Conference on Network Infrastructure and Digital Content (IC-NIDC 2009). IEEE, 2009. http://dx.doi.org/10.1109/icnidc.2009.5360980.

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Chernyavsky, Irina, Aparna S. Varde, and Simon Razniewski. "CSK-Detector: Commonsense in object detection." In 2022 IEEE International Conference on Big Data (Big Data). IEEE, 2022. http://dx.doi.org/10.1109/bigdata55660.2022.10020915.

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Yoshinaga, Honoka, Yusuke Kozawa, and Hiromasa Habuchi. "Enhancement of Optimized CSK for USLIPT." In GLOBECOM 2023 - 2023 IEEE Global Communications Conference. IEEE, 2023. http://dx.doi.org/10.1109/globecom54140.2023.10436855.

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Li, Jiangnan, Fandong Meng, Zheng Lin, Rui Liu, Peng Fu, Yanan Cao, Weiping Wang, and Jie Zhou. "Neutral Utterances are Also Causes: Enhancing Conversational Causal Emotion Entailment with Social Commonsense Knowledge." In Thirty-First International Joint Conference on Artificial Intelligence {IJCAI-22}. California: International Joint Conferences on Artificial Intelligence Organization, 2022. http://dx.doi.org/10.24963/ijcai.2022/584.

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Conversational Causal Emotion Entailment aims to detect causal utterances for a non-neutral targeted utterance from a conversation. In this work, we build conversations as graphs to overcome implicit contextual modelling of the original entailment style. Following the previous work, we further introduce the emotion information into graphs. Emotion information can markedly promote the detection of causal utterances whose emotion is the same as the targeted utterance. However, it is still hard to detect causal utterances with different emotions, especially neutral ones. The reason is that models are limited in reasoning causal clues and passing them between utterances. To alleviate this problem, we introduce social commonsense knowledge (CSK) and propose a Knowledge Enhanced Conversation graph (KEC). KEC propagates the CSK between two utterances. As not all CSK is emotionally suitable for utterances, we therefore propose a sentiment-realized knowledge selecting strategy to filter CSK. To process KEC, we further construct the Knowledge Enhanced Directed Acyclic Graph networks. Experimental results show that our method outperforms baselines and infers more causes with different emotions from the targeted utterance.
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Ndjiongue, A. R., H. C. Ferreira, and Telex M. N. Ngatched. "Constellation design for cascaded MPSK-CSK systems." In 2017 IEEE International Conference on Communications Workshops (ICC Workshops). IEEE, 2017. http://dx.doi.org/10.1109/iccw.2017.7962627.

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Cho, Hyunwoo, Hyojeong Choi, and Hong-Yeop Song. "Performance of CSK Modulation with Various Lengths." In 2022 27th Asia Pacific Conference on Communications (APCC). IEEE, 2022. http://dx.doi.org/10.1109/apcc55198.2022.9943720.

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Корнеева, Е. В., М. И. Воевода, С. Е. Семаев та В. Н. Максимов. "ГЕНЕТИЧЕСКИЕ ИССЛЕДОВАНИЯ МЕТАБОЛИЧЕСКОГО СИНДРОМА У МОЛОДЫХ ЛИЦ, ПРОЖИВАЮЩИХ В СЕВЕРНЫХ УСЛОВИЯХ". У Сборник тезисов III Конференции по лечению и диагностике сахарного диабета «Фундаментальная и клиническая диабетология в 21 веке: от теории к практике». ФГБУ «НМИЦ эндокринологии» Минздрава России, 2023. http://dx.doi.org/10.14341/diaconfiii25-26.05.23-47.

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ЦЕЛЬ: изучить влияние генов-кандидатов на развитие метаболического синдрома у молодых лиц, про-&#x0D; живающих в северных условиях.&#x0D; МАТЕРИАЛЫ И МЕТОДЫ: проведено когортное исследование 2354 молодых людей, проживаю-&#x0D; щих в северных условиях, из них 749 человек с метаболическим синдромом (31,8%). Средний возраст -&#x0D; 36,6±0,00 лет. Изучены полиморфизмы генов: rs1378942 гена CSK, rs1801133 (С677Т) гена MTHFR, гена ITGA2B,&#x0D; rs7903146 гена TCF7L2, rs1799752 гена АСЕ. Геномную ДНК выделяли из венозной крови методом фенол-&#x0D; хлороформной экстракции. Полиморфизм генов тестировали с помощью полимеразной цепной реакции&#x0D; с полиморфизмом длин рестрикционных фрагментов.&#x0D; РЕЗУЛЬТАТЫ: метаболический синдром был выявлен у 34,1% некоренных жителей, из них среди го-&#x0D; родских жителей – у 33,1%, среди сельских жителей – у 34,9%. Коренные жители имели метаболические&#x0D; нарушения 27,9% человек. По гендерному признаку: среди мужского населения метаболический синдром&#x0D; встречался у 30,4%, среди женского населения – у 32,5%. При анализе частоты однонуклеотидных полимор-&#x0D; физмов исследуемых генов выявлено гетерозиготное носительство (генотип TG гена CSK - у 50,6% обсле-&#x0D; дованных, генотип ID гена АСЕ – у 51,7%, генотип ID гена ADRα2B - 44,5%). Гомозиготные генотипы СС гена&#x0D; MTHFR были распространены у 52,9% и СС гена TCF7L2 – у 61,8% пациентов. Патологические гомозиготные&#x0D; генотипы встречались от 5,8% носителей полиморфизма гена TCF7L2 до 21,6% носителей полиморфизма&#x0D; гена АСЕ. Носительство генотипа ТТ и аллеля Т гена CSK чаще всего ассоциируется с развитием ожирения&#x0D; (ОШ 1.222 95% ДИ 0.854-1.749, p=0.183). У большинства пациентов встречаемость гетерозиготных генотипов&#x0D; генов CSK и MTHFR увеличивалась к III степени ожирения на 3,1% и 8,0%. Частота гетерозиготных геноти-&#x0D; пов генов АСЕ, ADRα2B и TCF7L2 имела тенденцию к росту при II степени ожирения на 7,7%, 8,5% и 2,6%.&#x0D; Самым распространенным среди коренных жителей был патологический генотип ТТ и аллель Т гена CSK,&#x0D; 36,5% и 60,0%. Среди женского населения выявлен гомозиготный генотип ТТ гена CSK (42,9%) и аллель&#x0D; Т (64,3%). Аллель Т гена MTHFR чаще распространена среди коренных жительниц (62,5%) с III степенью&#x0D; ожирения. В отличие от женского населения патологический генотип ТТ и аллель Т гена CSK встречался&#x0D; среди некоренных мужчин села (75,0% и 87,5%) с избытком массы тела (66,7%) и мужчин-ханты со II сте-&#x0D; пенью ожирения (62,5%). Гиперхолестеринемия среди коренных жителей ассоциировалась с наличием&#x0D; патологических аллелей генов MTHFR, АСЕ, TCF7L2. Встречаемость патологических генотипов ТТ и алелля Т&#x0D; гена TCF7L2 имела статистически значимые значения у пациентов с гипертриглицеридемией в сравнении&#x0D; с группой лиц с нормальными показателями триглицеридов (p=0,056). Статистически значимые значения&#x0D; у пациентов с гипергликемией и гиперинсулинемией были определены при носительстве патологиче-&#x0D; ского генотипа ТТ гена TCF7L2 (ОШ 2.042, 95%ДИ 1.050–3.970, p=0,046). При анализе распределения пар&#x0D; генотипов среди обследованных пациентов с МС в общей когорте наиболее распространенными были&#x0D; сочетания полиморфизма rs1799752 гена АСЕ и полиморфного локуса rs1378942 гена CSK (15,5%) (среди&#x0D; женщин - 16,0%). Среди мужчин - комбинации полиморфизма гена TCF7L2, гена ADRα2B (15,1%). Таким об-&#x0D; разом, необходимость комплексного обследования молодых людей, включая определение генетических&#x0D; детерминант развития метаболических нарушений, позволит персонализировано подойти к терапии вы-&#x0D; явленных метаболических изменений.
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Mennella, Sabrina, Maria Di Maro, and Martina Di Bratto. "Estimating Commonsense Knowledge from a Linguistic Analysis on Information Distribution." In Computational Linguistics in Bulgaria, 257–63. Institute for Bulgarian Language, 2024. https://doi.org/10.47810/clib.24.28.

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Commonsense Knowledge (CSK) is defined as a complex and multifaceted structure, encompassing a wide range of knowledge and reasoning generally acquired through everyday experiences. As CSK is often implicit in communication, it poses a challenge for AI systems to simulate human-like interaction. This work aims to deepen the CSK information structure from a linguistic perspective, starting from its organisation in conversations. To achieve this goal, we developed a three-level analysis model to extract more insights about this knowledge, focusing our attention on the second level. In particular, we aimed to extract the distribution of explicit actions and their execution order in the communicative flow. We built an annotation scheme based on FrameNet and applied it to a dialogical corpus on the culinary domain. Preliminary results indicate that certain frames occur earlier in the dialogues, while others occur towards the process’s end. These findings contribute to the systematic nature of actions by establishing clear patterns and relationships between frames.
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Reports on the topic "CSK"

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Lee, Byeong-Chei. Csk Homologous Kinase, a Potential Regulator of CXCR4-mediated Breast Cancer Cell Metastasis. Fort Belvoir, VA: Defense Technical Information Center, August 2010. http://dx.doi.org/10.21236/ada538886.

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Lee, Byeong-Chel. Csk Homologous Kinase, a Potential Regulator of CXCR4-Medicated Breast Cancer Cell Metastasis. Fort Belvoir, VA: Defense Technical Information Center, August 2011. http://dx.doi.org/10.21236/ada554270.

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Zagozdzon, Radoslaw, and Hava Avraham. Effects of Csk Homologous Kinase Overexpression on HER2/Neu-Mediated Signal Transduction Pathways in Breast Cancer Cells. Fort Belvoir, VA: Defense Technical Information Center, April 2005. http://dx.doi.org/10.21236/ada436916.

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Zagozdzon, Radoslaw, and Hava Avraham. Effects of CSK Homologous Kinase Overexpression on HER2/Neu-Mediated Signal Transduction Pathways in Breast Cancer Cells. Fort Belvoir, VA: Defense Technical Information Center, April 2003. http://dx.doi.org/10.21236/ada416967.

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Zagozdzon, Radoslaw, and Hava Avraham. Effects of CSK Homologous Kinase Overexpression on HER2/Neu-Mediated Signal Transduction Pathways in Breast Cancer Cells. Fort Belvoir, VA: Defense Technical Information Center, April 2004. http://dx.doi.org/10.21236/ada425671.

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REED, A. V. Evaluation of CSB Cask Receipt Pressure. Office of Scientific and Technical Information (OSTI), August 1999. http://dx.doi.org/10.2172/797705.

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Tengamnuay, Parkpoom. Efficacy and mechanistic studies of chitosan as nasal absorption enhancer of peptide drugs : research report. Chulalongkorn University, 1999. https://doi.org/10.58837/chula.res.1999.27.

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Objective. To evaluation the in vivo efficacy of chitosan as nasal absorption enhancers of peptides in rats and compare the results with that of hydroxypropyl- and dimethyl-Beta-cyclodextrins (HPBetaCD and DMBetaCD). Methods. Two types of chitosan. i.e., the free base (CSJ) and the glutamate salt form (CSG) were evaluated for their nasal absorption enhancing effect on salmon calcitonin (sCT) using an in vibo rat absorption technique. Solutions containing sCT and chitosan (0 to 1.25 % w/v) in isotonic phosphate buffers (pH 3.0 to 6.0) were nasally administered at the dose of 10 IU/kg. The plasma calcium lowering effect in each sCT-treated rat was determined by calculating the total percent decrease in plasma calcium (%D). Results. CSJ showed an increase in %D as the solution pH was decreased in accordance with the increased ionization and hydration of the free base chitosan at the more acidic pH. However, CSG showed an increase in %D with increasing pH, with maximum calcium lowereing effect observed at pH 6.0. At their optimal pH (4.0 for CSJ and 6.0 for CSG), the absorption enhancing effect of both chitosans was concentration dependent from 0.25 to 1.0 % w/v and leveled off at 1.25% w/v. Using specific RIA, the absolute bioavailability of plasma sCT was determined to be 2.45, 1.91, and 1.22 % for 1% CSJ, 5% DMBetaCD, and control group (intranasal sCt alone). Respectively. All the enhancers showed significant absorption enhancement with the highest effect observed with CSJ and DMBetaCD wheras the effect of HPBetaCD was the smallest. Also, the two chitosan did not possess any inhibitory effect on the in vitro activities of trypsin and leucine aminopeptidase, two major nasal proteolytic enzymes responsible for the degradation of sCT in the nasal cavity. Thus, the nasal absorption enhancement of chitosans may not involve protection of the peptide against proteolytic degradation in the nasal cavity. In conclusion, cationic polymer chitosans may have promising potential as an effective nasal absorption enhancer ofsCT.
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Beccali, Marco, Marina Bonomolo, Francesca Martorana, Ben Alex Baby, Marco Pellegrini, and Salvatore Vasta. Show Cases on System and Component Level & Adapted Components. IEA SHC Task 65, April 2024. http://dx.doi.org/10.18777/ieashc-task65-2024-0001.

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This document is the final report for joint activities A2, “Adapted components”, and B1, “Show cases on system and component level” of the IEA SHC Task 65, “Solar Cooling for the Sunbelt Regions. The first part of the report presents results from 32 investigated projects across 18 countries representing a range of 10 weather profiles such as the tropical wet and dry (Aw), hot desert (BWh), hot semi-arid (BSh), hot summer-Mediterranean (Csa), warmsummer Mediterranean (Csb), humid subtropic (Cfa), monsoon-influenced humid subtropical (Cwa), hot summer humid continental climate zones.
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Khadr, Ali, Oliver Peña-Habib, and Stefania De Santis. Independent Country Program Review Trinidad and Tobago 2016-2020. Inter-American Development Bank, April 2021. http://dx.doi.org/10.18235/0003852.

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This Independent Country Program Review (ICPR) covers the Inter-American Development Bank (IDB) Group's country strategy (CS) and program in Trinidad and Tobago (T&amp;T) over the period 2016-2020. ICPRs assess the relevance of a CS and, data permitting, provide aggregate information on the alignment and execution of the corresponding country program. ICPRs are primarily addressed to the IDB Group's Boards of Executive Directors (BoD). They seek to provide the BoD with relevant information, otherwise not readily available to them, to inform their consideration of the upcoming IDB Group CSs.
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Baich, M. A. CST/FRIT Settling, CST Particle Size Reduction and CST Loading. Office of Scientific and Technical Information (OSTI), August 2000. http://dx.doi.org/10.2172/761150.

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